On the public citation graph
Premature Aging in Mice Deficient in DNA Repair and Transcription
0 signed · 574 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
2 of the 47 references listed on this page are in the Retraction Watch database as retracted (export 2026-10-28): 10.1016/s0092-8674(00)80827-2 (notice 2005-11-18), 10.1126/science.275.5302.990 (notice 2005-06-17).
Dataset join, best-effort; this page does not check the publisher record. Retraction Watch data via Crossref Labs, used with credit; OpenCitations Index (CC0) and Meta (CC BY 4.0); dataset doi:10.7910/DVN/WT8VOA (CC BY 4.0).
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/j.1460-2075.1994.tb06522.xopenalex-snapshot
The ERCC2/DNA repair protein is associated with the class II BTF2/TFIIH transcription factor.
inferredcites →https://doi.org/10.1002/j.1460-2075.1996.tb00512.xopenalex-snapshot
Opening of an RNA polymerase II promoter occurs in two distinct steps and requires the basal transcription factors IIE and IIH.
inferredcites →https://doi.org/10.1006/taap.1996.0201openalex-snapshot
A Metalloporphyrin Superoxide Dismutase Mimetic Protects against Paraquat-Induced Lung Injuryin Vivo
inferredcites →https://doi.org/10.1007/bf00282072openalex-snapshot
Xeroderma pigmentosum (complementation group D) mutation is present in patients affected by trichothiodystrophy with photosensitivity
inferredcites →https://doi.org/10.1007/s004310100845openalex-snapshot
Trichothiodystrophy with severe cardiac and neurological involvement in two sisters
inferredcites →https://doi.org/10.1016/0092-8674(92)90390-xopenalex-snapshot
ERCC6, a member of a subfamily of putative helicases, is involved in Cockayne's syndrome and preferential repair of active genes
inferredcites →https://doi.org/10.1016/0092-8674(95)90105-1openalex-snapshot
The Bloom's syndrome gene product is homologous to RecQ helicases
inferredcites →https://doi.org/10.1016/0190-9622(90)70096-zopenalex-snapshot
Trichothiodystrophy: Review of sulfur-deficient brittle hair syndromes and association with the ectodermal dysplasias
inferredcites →https://doi.org/10.1016/0190-9622(93)70109-7openalex-snapshot
Trichothiodystrophy associated with photosensitivity, gonadal failure, and striking osteosclerosis
inferredcites →https://doi.org/10.1016/0921-8777(94)00059-fopenalex-snapshot
Biological effects of active oxygen on an X-ray-sensitive mutant mouse cell line (SL3-147)
inferredcites →https://doi.org/10.1016/j.cell.2005.11.005openalex-snapshot
Transcription-Coupled Repair of 8-oxoguanine: Requirement for XPG, TFIIH, and CSB and Implications for Cockayne Syndrome
inferredcites →https://doi.org/10.1016/s0027-5107(99)00202-xopenalex-snapshot
Somatic mutations and aging: a re-evaluation
inferredcites →https://doi.org/10.1016/s0047-6374(00)00095-6openalex-snapshot
Telomerase and mammalian ageing: a critical appraisal
inferredcites →https://doi.org/10.1016/s0092-8674(00)80223-8openalex-snapshot
Defective Transcription-Coupled Repair in Cockayne Syndrome B Mice Is Associated with Skin Cancer Predisposition
inferredcites →https://doi.org/10.1016/s0092-8674(00)80567-xopenalex-snapshot
Molecular Biology of Aging
inferredcites →https://doi.org/10.1016/s0092-8674(00)80580-2openalex-snapshot
Longevity, Stress Response, and Cancer in Aging Telomerase-Deficient Mice
inferredcites →https://doi.org/10.1016/s0092-8674(00)80827-2openalex-snapshot
RETRACTED: Transcription-Coupled Repair of 8-oxoGuanine
inferredcites →https://doi.org/10.1016/s0092-8674(00)80854-5openalex-snapshot
Transcriptional Healing
inferredcites →https://doi.org/10.1016/s0960-9822(06)00190-4openalex-snapshot
Disruption of mouse ERCC1 results in a novel repair syndrome with growth failure, nuclear abnormalities and senescence
inferredcites →https://doi.org/10.1016/s1097-2765(00)80098-2openalex-snapshot
A Mouse Model for the Basal Transcription/DNA Repair Syndrome Trichothiodystrophy
inferredcites →https://doi.org/10.1038/35041705openalex-snapshot
Lessons from human progeroid syndromes
inferredcites →https://doi.org/10.1038/35077232openalex-snapshot
Genome maintenance mechanisms for preventing cancer
inferredcites →https://doi.org/10.1038/377165a0openalex-snapshot
High incidence of ultraviolet-B-or chemical-carcinogen-induced skin tumours in mice lacking the xeroderma pigmentosum group A gene
inferredcites →https://doi.org/10.1038/377169a0openalex-snapshot
Increased susceptibility to ultraviolet-B and carcinogens of mice lacking the DNA excision repair gene XPA
inferredcites →https://doi.org/10.1038/415045aopenalex-snapshot
p53 mutant mice that display early ageing-associated phenotypes
inferredcites →https://doi.org/10.1038/ng0596-25openalex-snapshot
Genetic analysis of ageing: role of oxidative damage and environmental stresses
inferredcites →https://doi.org/10.1038/ng1297-431openalex-snapshot
Rapid accumulation of genome rearrangements in liver but not in brain of old mice
inferredcites →https://doi.org/10.1038/sj.onc.1203583openalex-snapshot
Proneness to UV-induced apoptosis in human fibroblasts defective in transcription coupled repair is associated with the lack of Mdm2 transactivation
inferredcites →https://doi.org/10.1073/pnas.070471597openalex-snapshot
Removal of oxygen free-radical-induced 5′,8-purine cyclodeoxynucleosides from DNA by the nucleotide excision-repair pathway in human cells
inferredcites →https://doi.org/10.1073/pnas.90.22.10499openalex-snapshot
Preferential repair of ionizing radiation-induced damage in the transcribed strand of an active human gene is defective in Cockayne syndrome.
inferredcites →https://doi.org/10.1073/pnas.96.19.10770openalex-snapshot
Deletion of Ku86 causes early onset of senescence in mice
inferredcites →https://doi.org/10.1074/jbc.m002259200openalex-snapshot
The Oxidative DNA Lesion 8,5′-(S)-Cyclo-2′-deoxyadenosine Is Repaired by the Nucleotide Excision Repair Pathway and Blocks Gene Expression in Mammalian Cells
inferredcites →https://doi.org/10.1086/302063openalex-snapshot
Analysis of Mutations in the XPD Gene in Italian Patients with Trichothiodystrophy: Site of Mutation Correlates with Repair Deficiency, but Gene Dosage Appears to Determine Clinical Severity
inferredcites →https://doi.org/10.1093/emboj/16.21.6559openalex-snapshot
Mechanism of open complex and dual incision formation by human nucleotide excision repair factors
inferredcites →https://doi.org/10.1095/biolreprod28.2.255openalex-snapshot
Imminent Oocyte Exhaustion and Reduced Follicular Recruitment Mark the Transition to Acyclicity in Aging C57BL/6J Mice
inferredcites →https://doi.org/10.1101/gad.13.7.768openalex-snapshot
Molecular mechanism of nucleotide excision repair
inferredcites →https://doi.org/10.1101/gad.859501openalex-snapshot
The xeroderma pigmentosum group D (XPD) gene: one gene, two functions, three diseases
inferredcites →https://doi.org/10.1101/sqb.1994.059.01.036openalex-snapshot
Three Unusual Repair Deficiencies Associated with Transcription Factor BTF2(TFIIH): Evidence for the Existence of a Transcription Syndrome
inferredcites →https://doi.org/10.1111/j.1365-2133.1988.tb02434.xopenalex-snapshot
Tissue measurements in senile sebaceous gland hyperplasia
inferredcites →https://doi.org/10.1111/j.1749-6632.2000.tb06365.xopenalex-snapshot
Lipoproteins in the Central Nervous System
inferredcites →https://doi.org/10.1111/j.1749-6632.2000.tb06645.xopenalex-snapshot
The Werner Syndrome: A Model for the Study of Human Aging
inferredcites →https://doi.org/10.1126/science.272.5259.258openalex-snapshot
Positional Cloning of the Werner's Syndrome Gene
inferredcites →https://doi.org/10.1126/science.275.5302.990openalex-snapshot
Defective Transcription-Coupled Repair of Oxidative Base Damage in Cockayne Syndrome Patients from XP Group G
inferredcites →https://doi.org/10.1126/science.286.5446.1897openalex-snapshot
Quality Control by DNA Repair
inferredcites →https://doi.org/10.1126/science.8465201openalex-snapshot
DNA Repair Helicase: a Component of BTF2 (TFIIH) Basic Transcription Factor
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4509193openalex-extracted
Matters Arising: The Information Theory of Aging Has Not Been Tested
inferred← citeshttps://doi.org/10.1001/archsurg.138.10.1051openalex-snapshot
Molecular Biology of Aging
inferred← citeshttps://doi.org/10.1002/0471684228.egp12930openalex-snapshot
Trichothiodystrophy ( TTD , 19 q 13.2‐ q 13.3)
inferred← citeshttps://doi.org/10.1002/3527600906.mcb.200200009openalex-snapshot
Aging and Sex, DNA Repair in
inferred← citeshttps://doi.org/10.1002/ajmg.a.32902openalex-snapshot
New clinico‐genetic classification of trichothiodystrophy
inferred← citeshttps://doi.org/10.1002/ar.20713openalex-snapshot
Immunohistochemical Analysis of Nucleotide Excision Repair Factors XPA and XPB in Adult Rat Brain
inferred← citeshttps://doi.org/10.1002/bies.10150openalex-snapshot
When machines get stuck—obstructed RNA polymerase II: displacement, degradation or suicide
inferred← citeshttps://doi.org/10.1002/bies.201800201openalex-snapshot
Nucleotide Excision Repair and Transcription‐Associated Genome Instability
inferred← citeshttps://doi.org/10.1002/bies.20760openalex-snapshot
Cell divisions and mammalian aging: integrative biology insights from genes that regulate longevity
inferred← citeshttps://doi.org/10.1002/biof.5520270105openalex-snapshot
Thioredoxin‐binding protein‐2 (TBP‐2): Its potential roles in the aging process
inferred← citeshttps://doi.org/10.1002/em.20597openalex-snapshot
The DNA damage response—Repair or despair?
inferred← citeshttps://doi.org/10.1002/humu.20912openalex-snapshot
Genotype-phenotype relationships in trichothiodystrophy patients with novel splicing mutations in theXPDgene
inferred← citeshttps://doi.org/10.1002/jbmr.3564openalex-snapshot
The Spectrum of Fundamental Basic Science Discoveries Contributing to Organismal Aging
inferred← citeshttps://doi.org/10.1002/jnr.10454openalex-snapshot
Transcripts of damaged genes in the brain during cerebral oxidative stress
inferred← citeshttps://doi.org/10.1002/jor.22204openalex-snapshot
An overview of underlying causes and animal models for the study of age‐related degenerative disorders of the spine and synovial joints
inferred← citeshttps://doi.org/10.1007/1-4020-3764-3_3openalex-snapshot
Nucleotide Excision Repair and its Connection with Cancer and Ageing
inferred← citeshttps://doi.org/10.1007/978-0-387-74072-0_14openalex-snapshot
Translational Research: Gene, Pharmacogenomics and Cell-Based Therapy in the Aging Heart
inferred← citeshttps://doi.org/10.1007/978-0-387-74072-0_4openalex-snapshot
Molecular and Cellular Phenotypes of Cardiovascular Aging
inferred← citeshttps://doi.org/10.1007/978-1-4020-9063-9_3openalex-snapshot
Mouse Models and Genetics of Immunosenescence
inferred← citeshttps://doi.org/10.1007/978-1-4419-6448-9_10openalex-snapshot
Trichothiodystrophy: Photosensitive, TTD-P, TTD, Tay Syndrome
inferred← citeshttps://doi.org/10.1007/978-1-4419-6857-9_33openalex-snapshot
Postnatal Effects of Sperm Chromatin Damage
inferred← citeshttps://doi.org/10.1007/978-1-4419-9032-7_2openalex-snapshot
Biochemical Roles of RecQ Helicases
inferred← citeshttps://doi.org/10.1007/978-1-4614-0332-6_5openalex-snapshot
Spatiotemporal Aspects of MicroRNA-Mediated Gene Regulation
inferred← citeshttps://doi.org/10.1007/978-1-4614-1531-2_62openalex-snapshot
Nucleotide Excision Repair
inferred← citeshttps://doi.org/10.1007/978-1-4614-6436-5_62-3openalex-snapshot
Nucleotide Excision Repair
inferred← citeshttps://doi.org/10.1007/978-1-4614-7843-0_15openalex-snapshot
Postnatal Effects of Sperm Chromatin Damage
inferred← citeshttps://doi.org/10.1007/978-1-59745-458-2_24openalex-snapshot
Xeroderma Pigmentosum and the DNA Damage Response to Ultraviolet Light
inferred← citeshttps://doi.org/10.1007/978-3-319-64597-1_3-1openalex-snapshot
Mouse Models and Genetics of Immunosenescence
inferred← citeshttps://doi.org/10.1007/978-3-319-94950-5openalex-snapshot
What Does it Mean to be Human? Life, Death, Personhood and the Transhumanist Movement
inferred← citeshttps://doi.org/10.1007/978-3-319-99375-1_3openalex-snapshot
Mouse Models and Genetics of Immunosenescence
inferred← citeshttps://doi.org/10.1007/978-3-540-73709-4_4openalex-snapshot
Initiation of Genomic Instability, Cellular Senescence, and Organismal Aging by Dysfunctional Telomeres
inferred← citeshttps://doi.org/10.1007/978-3-642-02112-1_21openalex-snapshot
Aging and Cancer: Caretakers and Gatekeepers
inferred← citeshttps://doi.org/10.1007/978-3-642-18741-4_13openalex-snapshot
Molekulare Mechanismen altersspezifischer Veränderungen des Kalzium- und Knochenstoffwechsels
inferred← citeshttps://doi.org/10.1007/978-3-642-34303-2_16openalex-snapshot
Basics of Molecular Biology
inferred← citeshttps://doi.org/10.1007/978-3-642-55661-6_51openalex-snapshot
Hauterkrankungen mit genetischer Instabilität
inferred← citeshttps://doi.org/10.1007/978-3-7091-1232-8_9openalex-snapshot
Aging of Mammalian Intestinal Stem Cells
inferred← citeshttps://doi.org/10.1007/978-94-017-0667-4_2openalex-snapshot
Oxidative DNA Damage and Repair — Implications for Aging
inferred← citeshttps://doi.org/10.1007/978-94-017-0669-8_5openalex-snapshot
In Vitro Senescence of Human Osteoblasts
inferred← citeshttps://doi.org/10.1007/978-94-017-0669-8_7openalex-snapshot
Aging of Premature-Aging Syndrome Cells
inferred← citeshttps://doi.org/10.1007/978-94-017-0673-5_4openalex-snapshot
Aging of the Hematopoietic System
inferred← citeshttps://doi.org/10.1007/bf02711481openalex-snapshot
Exploring cellular senescence as a tumor suppressor mechanism
inferred← citeshttps://doi.org/10.1007/bf02982600openalex-snapshot
Genetic Control of Stem Cells: Implications for Aging
inferred← citeshttps://doi.org/10.1007/s00018-006-6349-3openalex-snapshot
Human progeroid syndromes, aging and cancer: new genetic and epigenetic insights into old questions
inferred← citeshttps://doi.org/10.1007/s00018-018-2833-9openalex-snapshot
The capacity of oocytes for DNA repair
inferred← citeshttps://doi.org/10.1007/s00018-025-05730-9openalex-snapshot
DNA glycosylases Ogg1 and Mutyh influence gene expression of PRC2 targets associated with cognition
inferred← citeshttps://doi.org/10.1007/s00105-003-0564-8openalex-snapshot
Genetisch bedingte Haarerkrankungen
inferred← citeshttps://doi.org/10.1007/s00247-004-1207-7openalex-snapshot
Central osteosclerosis with trichothiodystrophy
inferred← citeshttps://doi.org/10.1007/s00586-010-1606-4openalex-snapshot
Back muscle strength and spinal mobility are predictors of quality of life in middle-aged and elderly males
inferred← citeshttps://doi.org/10.1007/s10439-020-02710-xopenalex-snapshot
Review of Wearable and Portable Sensors for Monitoring Personal Solar UV Exposure
inferred← citeshttps://doi.org/10.1007/s10522-007-9104-8openalex-snapshot
Heritability of a skeletal biomarker of biological aging
inferred← citeshttps://doi.org/10.1007/s10522-009-9246-yopenalex-snapshot
Decreased expression and the Lys751Gln polymorphism of the XPD gene are associated with extreme longevity
inferred← citeshttps://doi.org/10.1007/s11033-011-1333-xopenalex-snapshot
Molecular cloning and expression analysis of xpd from zebrafish (Danio rerio)
inferred← citeshttps://doi.org/10.1007/s11248-005-1772-zopenalex-snapshot
The Effect of DNA Repair Defects on Reproductive Performance in Nucleotide Excision Repair (NER) Mouse Models: An Epidemiological Approach
inferred← citeshttps://doi.org/10.1007/s11307-024-01902-4openalex-snapshot
Hybrid Molecular and Functional Micro-CT Imaging Reveals Increased Myocardial Apoptosis Preceding Cardiac Failure in Progeroid Ercc1 Mice
inferred← citeshttps://doi.org/10.1007/s11356-023-28065-zopenalex-snapshot
Joint effects of phenol, chlorophenol pesticide, phthalate, and polycyclic aromatic hydrocarbon on bone mineral density: comparison of four statistical models
inferred← citeshttps://doi.org/10.1007/s11357-005-4556-8openalex-snapshot
Twenty years of progress in biogerontology research
inferred← citeshttps://doi.org/10.1007/s11357-010-9175-3openalex-snapshot
Analysis of osteoarthritis in a mouse model of the progeroid human DNA repair syndrome trichothiodystrophy
inferred← citeshttps://doi.org/10.1007/s11357-011-9291-8openalex-snapshot
Bone fragility and decline in stem cells in prematurely aging DNA repair deficient trichothiodystrophy mice
inferred← citeshttps://doi.org/10.1007/s11357-014-9631-6openalex-snapshot
Stress, ageing and their influence on functional, cellular and molecular aspects of the immune system
inferred← citeshttps://doi.org/10.1007/s11357-014-9675-7openalex-snapshot
Long-term α1B-adrenergic receptor activation shortens lifespan, while α1A-adrenergic receptor stimulation prolongs lifespan in association with decreased cancer incidence
inferred← citeshttps://doi.org/10.1007/s11655-013-1333-2openalex-snapshot
Effect of curcumin on aged Drosophila Melanogaster: A pathway prediction analysis
inferred← citeshttps://doi.org/10.1007/s11914-011-0054-3openalex-snapshot
FoxOs: Unifying Links Between Oxidative Stress and Skeletal Homeostasis
inferred← citeshttps://doi.org/10.1007/s12038-017-9684-7openalex-snapshot
Inactivation of RAD52 and HDF1 DNA repair genes leads to premature chronological aging and cellular instability
inferred← citeshttps://doi.org/10.1007/s13659-017-0135-9openalex-snapshot
Current Perspective in the Discovery of Anti-aging Agents from Natural Products
inferred← citeshttps://doi.org/10.1016/b0-12-475570-4/00053-6openalex-snapshot
Premature Aging Syndromes
inferred← citeshttps://doi.org/10.1016/b978-0-12-396971-2.00009-9openalex-snapshot
The Circadian Clock in Cancer Development and Therapy
inferred← citeshttps://doi.org/10.1016/b978-0-12-397920-9.00003-2openalex-snapshot
Mouse Models and Aging
inferred← citeshttps://doi.org/10.1016/b978-0-12-799941-8.00027-4openalex-snapshot
Environmental Determinants of Lung Aging
inferred← citeshttps://doi.org/10.1016/b978-0-12-821618-7.00245-5openalex-snapshot
Role of DNA Damage, Somatic Mutations, Telomere Shortening, and Epigenetic Alterations in Aging and Age-Related Disease
inferred← citeshttps://doi.org/10.1016/b978-0-323-85654-6.00057-5openalex-snapshot
The neurobiology of aging
inferred← citeshttps://doi.org/10.1016/b978-0-323-91824-4.00013-7openalex-snapshot
Environmental determinants of lung aging
inferred← citeshttps://doi.org/10.1016/b978-0-443-15500-0.00017-7openalex-snapshot
Age-related disease: Bones
inferred← citeshttps://doi.org/10.1016/b978-012088387-5/50009-1openalex-snapshot
p53 and Mouse Aging Models
inferred← citeshttps://doi.org/10.1016/b978-012088387-5/50010-8openalex-snapshot
Complex Genetic Architecture of Drosophila Longevity
inferred← citeshttps://doi.org/10.1016/b978-012324751-3/50062-0openalex-snapshot
Environmental Determinants of Lung Aging
inferred← citeshttps://doi.org/10.1016/b978-012336425-8/50059-5openalex-snapshot
The Mouse as an Animal Model for Human Diseases
inferred← citeshttps://doi.org/10.1016/b978-012369391-4/50050-3openalex-snapshot
Mouse Models of Accelerated Aging
inferred← citeshttps://doi.org/10.1016/b978-1-4377-0637-6.00059-3openalex-snapshot
Aging and the Cardiovascular System
inferred← citeshttps://doi.org/10.1016/b978-84-458-1536-6.50052-xopenalex-snapshot
Envejecimiento y sistema cardiovascular
inferred← citeshttps://doi.org/10.1016/j.arr.2003.08.006openalex-snapshot
How bioinformatics can help reverse engineer human aging
inferred← citeshttps://doi.org/10.1016/j.arr.2004.05.003openalex-snapshot
Open-minded scepticism: inferring the causal mechanisms of human ageing from genetic perturbations
inferred← citeshttps://doi.org/10.1016/j.arr.2005.06.008openalex-snapshot
Gene expression and DNA repair in progeroid syndromes and human aging
inferred← citeshttps://doi.org/10.1016/j.arr.2012.02.001openalex-snapshot
The role of DNA damage and repair in aging through the prism of Koch-like criteria
inferred← citeshttps://doi.org/10.1016/j.arr.2012.03.010openalex-snapshot
The mouse as a model organism in aging research: Usefulness, pitfalls and possibilities
inferred← citeshttps://doi.org/10.1016/j.bbcan.2009.02.002openalex-snapshot
Should I stay or should I go: β-catenin decides under stress
inferred← citeshttps://doi.org/10.1016/j.bbrc.2004.12.041openalex-snapshot
Aging bone and cartilage: cross-cutting issues
inferred← citeshttps://doi.org/10.1016/j.bcp.2021.114651openalex-snapshot
p53 regulated senescence mechanism and role of its modulators in age-related disorders
inferred← citeshttps://doi.org/10.1016/j.biocel.2008.05.009openalex-snapshot
Oxidative damage induced genotoxic effects in human fibroblasts from Xeroderma Pigmentosum group A patients
inferred← citeshttps://doi.org/10.1016/j.biochi.2003.09.010openalex-snapshot
DNA repair-deficient diseases, xeroderma pigmentosum, Cockayne syndrome and trichothiodystrophy
inferred← citeshttps://doi.org/10.1016/j.biochi.2003.11.005openalex-snapshot
Transcription-coupled repair of 8-oxoguanine in human cells and its deficiency in some DNA repair diseases
inferred← citeshttps://doi.org/10.1016/j.biopha.2023.114384openalex-snapshot
Bazi Bushen mitigates epigenetic aging and extends healthspan in naturally aging mice
inferred← citeshttps://doi.org/10.1016/j.bone.2011.05.023openalex-snapshot
Unraveling the role of FoxOs in bone—Insights from mouse models
inferred← citeshttps://doi.org/10.1016/j.bone.2019.03.002openalex-snapshot
Murine models of accelerated aging and musculoskeletal disease
inferred← citeshttps://doi.org/10.1016/j.cardiores.2005.03.003openalex-snapshot
Aging and biomedicine 2005: Where should we go from here?
inferred← citeshttps://doi.org/10.1016/j.cccn.2005.04.014openalex-snapshot
Ovariectomy induces oxidative stress and impairs bone antioxidant system in adult rats
inferred← citeshttps://doi.org/10.1016/j.cclet.2021.10.053openalex-snapshot
Recent advances in DNA glycosylase assays
inferred← citeshttps://doi.org/10.1016/j.ccr.2006.05.027openalex-snapshot
An Xpd mouse model for the combined xeroderma pigmentosum/Cockayne syndrome exhibiting both cancer predisposition and segmental progeria
inferred← citeshttps://doi.org/10.1016/j.cej.2026.172885openalex-snapshot
Dual-functional tandem luminescent solar concentrators with photovoltaics and linear ultraviolet response for self-powered ultraviolet monitoring
inferred← citeshttps://doi.org/10.1016/j.cell.2005.01.028openalex-snapshot
DNA Repair, Genome Stability, and Aging
inferred← citeshttps://doi.org/10.1016/j.cell.2005.11.044openalex-snapshot
Genomic Instability and Aging-like Phenotype in the Absence of Mammalian SIRT6
inferred← citeshttps://doi.org/10.1016/j.cell.2008.04.029openalex-snapshot
Structure of the DNA Repair Helicase XPD
inferred← citeshttps://doi.org/10.1016/j.cell.2008.04.030openalex-snapshot
XPD Helicase Structures and Activities: Insights into the Cancer and Aging Phenotypes from XPD Mutations
inferred← citeshttps://doi.org/10.1016/j.cell.2009.03.037openalex-snapshot
Genotoxic Stress Abrogates Renewal of Melanocyte Stem Cells by Triggering Their Differentiation
inferred← citeshttps://doi.org/10.1016/j.cell.2017.02.031openalex-snapshot
Targeted Apoptosis of Senescent Cells Restores Tissue Homeostasis in Response to Chemotoxicity and Aging
inferred← citeshttps://doi.org/10.1016/j.cellsig.2008.04.014openalex-snapshot
Wnt and beyond Wnt: Multiple mechanisms control the transcriptional property of β-catenin
inferred← citeshttps://doi.org/10.1016/j.celrep.2015.05.009openalex-snapshot
mtDNA Mutagenesis Disrupts Pluripotent Stem Cell Function by Altering Redox Signaling
inferred← citeshttps://doi.org/10.1016/j.celrep.2026.117600openalex-snapshot
Paf1 Counteracts transcriptional arrest to maintain rDNA stability during pol I elongation
inferred← citeshttps://doi.org/10.1016/j.cmet.2009.12.009openalex-snapshot
FoxO-Mediated Defense against Oxidative Stress in Osteoblasts Is Indispensable for Skeletal Homeostasis in Mice
inferred← citeshttps://doi.org/10.1016/j.cmet.2010.01.001openalex-snapshot
FoxO1 Is a Positive Regulator of Bone Formation by Favoring Protein Synthesis and Resistance to Oxidative Stress in Osteoblasts
inferred← citeshttps://doi.org/10.1016/j.cmet.2011.11.012openalex-snapshot
Somatic Progenitor Cell Vulnerability to Mitochondrial DNA Mutagenesis Underlies Progeroid Phenotypes in Polg Mutator Mice
inferred← citeshttps://doi.org/10.1016/j.coph.2018.05.007openalex-snapshot
Musculoskeletal senescence: a moving target ready to be eliminated
inferred← citeshttps://doi.org/10.1016/j.ddmod.2004.11.023openalex-snapshot
Klotho deficient mouse: an in vivo model for human aging
inferred← citeshttps://doi.org/10.1016/j.ddmod.2014.11.001openalex-snapshot
Impact of senescence on bone quality: lessons from animal models of aging
inferred← citeshttps://doi.org/10.1016/j.dnarep.2004.10.009openalex-snapshot
Severe growth retardation and short life span of double-mutant mice lacking Xpa and exon 15 of Xpg
inferred← citeshttps://doi.org/10.1016/j.dnarep.2005.07.002openalex-snapshot
Accelerated aging pathology in ad libitum fed XpdTTD mice is accompanied by features suggestive of caloric restriction
inferred← citeshttps://doi.org/10.1016/j.dnarep.2006.01.006openalex-snapshot
Cellular responses to DNA damage: Current state of the field and review of the 52nd Benzon Symposium
inferred← citeshttps://doi.org/10.1016/j.dnarep.2006.10.017openalex-snapshot
The mechanics of base excision repair, and its relationship to aging and disease
inferred← citeshttps://doi.org/10.1016/j.dnarep.2007.01.014openalex-snapshot
RNA polymerase II bypasses 8-oxoguanine in the presence of transcription elongation factor TFIIS
inferred← citeshttps://doi.org/10.1016/j.dnarep.2007.12.006openalex-snapshot
Nucleotide excision repair deficient mouse models and neurological disease
inferred← citeshttps://doi.org/10.1016/j.dnarep.2008.06.005openalex-snapshot
8,5′-Cyclopurine-2′-deoxynucleosides in DNA: Mechanisms of formation, measurement, repair and biological effects
inferred← citeshttps://doi.org/10.1016/j.dnarep.2008.09.009openalex-snapshot
Accumulation of (5′S)-8,5′-cyclo-2′-deoxyadenosine in organs of Cockayne syndrome complementation group B gene knockout mice
inferred← citeshttps://doi.org/10.1016/j.dnarep.2009.10.005openalex-snapshot
Trichothiodystrophy: From basic mechanisms to clinical implications
inferred← citeshttps://doi.org/10.1016/j.dnarep.2011.04.025openalex-snapshot
Premature aging and cancer in nucleotide excision repair-disorders
inferred← citeshttps://doi.org/10.1016/j.dnarep.2011.04.029openalex-snapshot
Nucleotide excision repair in chromatin: Damage removal at the drop of a HAT
inferred← citeshttps://doi.org/10.1016/j.ejmg.2018.10.009openalex-snapshot
A case of severe trichothiodystrophy 3 in a neonate due to mutation in the GTF2H5 gene: Clinical report
inferred← citeshttps://doi.org/10.1016/j.envpol.2018.04.108openalex-snapshot
Associations of urinary polycyclic aromatic hydrocarbons with bone mass density and osteoporosis in U.S. adults, NHANES 2005–2010
inferred← citeshttps://doi.org/10.1016/j.exger.2003.09.006openalex-snapshot
Photoaging as a consequence of natural and therapeutic ultraviolet irradiation—studies on PUVA-induced senescence-like growth arrest of human dermal fibroblasts
inferred← citeshttps://doi.org/10.1016/j.exger.2004.04.011openalex-snapshot
The Bcl-2 protein family and its role in the development of neoplastic disease
inferred← citeshttps://doi.org/10.1016/j.exger.2004.07.010openalex-snapshot
Poly(ADP-ribosyl)ation and aging
inferred← citeshttps://doi.org/10.1016/j.exger.2005.09.002openalex-snapshot
Cells discover fire: Employing reactive oxygen species in developmentand consequences for aging
inferred← citeshttps://doi.org/10.1016/j.exger.2006.06.057openalex-snapshot
Genetics of proliferative aging
inferred← citeshttps://doi.org/10.1016/j.exger.2008.03.010openalex-snapshot
Apolipoprotein E, an important player in longevity and age-related diseases
inferred← citeshttps://doi.org/10.1016/j.exger.2009.12.003openalex-snapshot
Effect of caloric restriction on base-excision repair (BER) in the aging rat brain
inferred← citeshttps://doi.org/10.1016/j.exphem.2008.07.009openalex-snapshot
Final checkup of neoplastic DNA replication: Evidence for failure in decision-making at the mitotic cell cycle checkpoint G1/S
inferred← citeshttps://doi.org/10.1016/j.foodres.2009.07.031openalex-snapshot
Pinto bean hull extract supplementation favorably affects markers of bone metabolism and bone structure in mice
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2023.03.262openalex-snapshot
Mitochondrial OGG1 expression reduces age-associated neuroinflammation by regulating cytosolic mitochondrial DNA
inferred← citeshttps://doi.org/10.1016/j.gde.2014.05.003openalex-snapshot
Genetics of aging, progeria and lamin disorders
inferred← citeshttps://doi.org/10.1016/j.ics.2006.08.001openalex-snapshot
Selenium deficiency as a putative risk factor for osteoporosis
inferred← citeshttps://doi.org/10.1016/j.ijbiomac.2017.10.080openalex-snapshot
MiR-455-3p activates Nrf2/ARE signaling via HDAC2 and protects osteoblasts from oxidative stress
inferred← citeshttps://doi.org/10.1016/j.isci.2022.104957openalex-snapshot
Ercc2/Xpd deficiency results in failure of digestive organ growth in zebrafish with elevated nucleolar stress
inferred← citeshttps://doi.org/10.1016/j.it.2004.11.005openalex-snapshot
Chronic antigenic load and apoptosis in immunosenescence
inferred← citeshttps://doi.org/10.1016/j.jep.2025.120894openalex-snapshot
Aqueous extract of Alstonia boonei de Wild. (Apocynaceae) improves bone health in a model of diabetes-related bone complications in rats: in vivo and in silico studies
inferred← citeshttps://doi.org/10.1016/j.jtbi.2005.05.007openalex-snapshot
The evolution of anisogamy: The adaptive significance of damage, repair and mortality
inferred← citeshttps://doi.org/10.1016/j.mad.2004.08.015openalex-snapshot
Longevity genes: from primitive organisms to humans
inferred← citeshttps://doi.org/10.1016/j.mad.2004.09.012openalex-snapshot
A genetic and genomic approach to identify longevity genes in mice
inferred← citeshttps://doi.org/10.1016/j.mad.2004.09.036openalex-snapshot
The impact of DNA damage, genetic mutation and cellular responses on cancer prevention, longevity and aging: observations in humans and mice
inferred← citeshttps://doi.org/10.1016/j.mad.2004.10.003openalex-snapshot
Multiple roads to the aging phenotype: insights from the molecular dissection of progerias through DNA microarray analysis
inferred← citeshttps://doi.org/10.1016/j.mad.2004.10.006openalex-snapshot
A-type nuclear lamins, progerias and other degenerative disorders
inferred← citeshttps://doi.org/10.1016/j.mad.2006.11.004openalex-snapshot
Is cell death and replacement a factor in aging?
inferred← citeshttps://doi.org/10.1016/j.mad.2006.11.011openalex-snapshot
Extended longevity mechanisms in short-lived progeroid mice: Identification of a preservative stress response associated with successful aging
inferred← citeshttps://doi.org/10.1016/j.mad.2007.05.002openalex-snapshot
Genome maintenance mechanisms are critical for preventing cancer as well as other aging-associated diseases
inferred← citeshttps://doi.org/10.1016/j.mad.2007.05.005openalex-snapshot
Protagonistic pleiotropy: Why cancer may be the only pathogenic effect of accumulating nuclear mutations and epimutations in aging
inferred← citeshttps://doi.org/10.1016/j.mad.2007.08.006openalex-snapshot
Deletion of Ku80 causes early aging independent of chronic inflammation and Rag-1-induced DSBs
inferred← citeshttps://doi.org/10.1016/j.mad.2008.01.005openalex-snapshot
Clinical implications of the basic defects in Cockayne syndrome and xeroderma pigmentosum and the DNA lesions responsible for cancer, neurodegeneration and aging
inferred← citeshttps://doi.org/10.1016/j.mad.2008.02.002openalex-snapshot
DNA double-strand breaks: A potential causative factor for mammalian aging?
inferred← citeshttps://doi.org/10.1016/j.mad.2008.03.009openalex-snapshot
Replicative stress, stem cells and aging
inferred← citeshttps://doi.org/10.1016/j.mad.2008.04.003openalex-snapshot
HGPS and related premature aging disorders: From genomic identification to the first therapeutic approaches
inferred← citeshttps://doi.org/10.1016/j.mad.2008.04.010openalex-snapshot
Tissue-specific accelerated aging in nucleotide excision repair deficiency
inferred← citeshttps://doi.org/10.1016/j.mad.2009.01.001openalex-snapshot
Altered senescence, apoptosis, and DNA damage response in a mutant p53 model of accelerated aging
inferred← citeshttps://doi.org/10.1016/j.mad.2010.05.005openalex-snapshot
Inflammatory and age-related pathologies in mice with ectopic expression of human PARP-1
inferred← citeshttps://doi.org/10.1016/j.mad.2013.03.003openalex-snapshot
Arresting transcription and sentencing the cell: The consequences of blocked transcription
inferred← citeshttps://doi.org/10.1016/j.mad.2013.03.004openalex-snapshot
Cockayne syndrome group B (CSB) protein: At the crossroads of transcriptional networks
inferred← citeshttps://doi.org/10.1016/j.mad.2013.04.003openalex-snapshot
Cockayne syndrome pathogenesis: Lessons from mouse models
inferred← citeshttps://doi.org/10.1016/j.mad.2015.03.006openalex-snapshot
MARK-AGE biomarkers of ageing
inferred← citeshttps://doi.org/10.1016/j.mad.2015.03.012openalex-snapshot
NRMT1 knockout mice exhibit phenotypes associated with impaired DNA repair and premature aging
inferred← citeshttps://doi.org/10.1016/j.mad.2016.10.001openalex-snapshot
Mouse models of ageing and their relevance to disease
inferred← citeshttps://doi.org/10.1016/j.mad.2025.112059openalex-snapshot
Changing landscape of hematopoietic and mesenchymal cells and their interactions during aging and in age-related skeletal pathologies
inferred← citeshttps://doi.org/10.1016/j.maturitas.2016.07.006openalex-snapshot
Menopause: Genome stability as new paradigm
inferred← citeshttps://doi.org/10.1016/j.mce.2008.10.031openalex-snapshot
Sealing the gap between nuclear DNA damage and longevity
inferred← citeshttps://doi.org/10.1016/j.mce.2015.01.034openalex-snapshot
Oxidation of the aromatic amino acids tryptophan and tyrosine disrupts their anabolic effects on bone marrow mesenchymal stem cells
inferred← citeshttps://doi.org/10.1016/j.mehy.2005.02.015openalex-snapshot
Pharmacologic manipulation of the ataxia–telangiectasia mutated gene product as an intervention in age-related disease
inferred← citeshttps://doi.org/10.1016/j.molmed.2006.05.002openalex-snapshot
Hypervitaminosis D and premature aging: lessons learned from Fgf23 and Klotho mutant mice
inferred← citeshttps://doi.org/10.1016/j.molmed.2016.11.006openalex-snapshot
The Fountain of Youth by Targeting Senescent Cells?
inferred← citeshttps://doi.org/10.1016/j.molmed.2021.06.011openalex-snapshot
Molecular pathology of rare progeroid diseases
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2003.07.003openalex-snapshot
Substrate specificities and excision kinetics of DNA glycosylases involved in base-excision repair of oxidative DNA damage
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2004.08.020openalex-snapshot
Modeling aging and cancer in the telomerase knockout mouse
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2005.04.004openalex-snapshot
Transcription-coupled repair and premature ageing
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2005.11.008openalex-snapshot
Increased genomic instability is not a prerequisite for shortened lifespan in DNA repair deficient mice
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2005.12.018openalex-snapshot
Tissue specific mutagenic and carcinogenic responses in NER defective mouse models
inferred← citeshttps://doi.org/10.1016/j.mrrev.2011.05.001openalex-snapshot
A review and appraisal of the DNA damage theory of ageing
inferred← citeshttps://doi.org/10.1016/j.mrrev.2025.108555openalex-snapshot
Trichothiodystrophy: Molecular insights and mechanisms of pathogenicity
inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2006.05.012openalex-snapshot
Mutant mice with small amounts of BubR1 display accelerated age-related gliosis
inferred← citeshttps://doi.org/10.1016/j.neuroscience.2006.08.074openalex-snapshot
Cockayne syndrome exhibits dysregulation of p21 and other gene products that may be independent of transcription-coupled repair
inferred← citeshttps://doi.org/10.1016/j.ophtha.2011.05.036openalex-snapshot
Ocular Manifestations of Trichothiodystrophy
inferred← citeshttps://doi.org/10.1016/j.pbiomolbio.2009.03.001openalex-snapshot
The rules of disorder or why disorder rules
inferred← citeshttps://doi.org/10.1016/j.preteyeres.2026.101442openalex-snapshot
Epigenetic reprogramming for ocular aging and disease: Mechanisms, biomarkers, and the road to the clinic
inferred← citeshttps://doi.org/10.1016/j.pt.2025.09.008openalex-snapshot
Exploring erythropoiesis: aging through the lens of malaria and microgravity
inferred← citeshttps://doi.org/10.1016/j.rbmo.2014.11.010openalex-snapshot
Changes in the expression of DNA double strand break repair genes in primordial follicles from immature and aged rats
inferred← citeshttps://doi.org/10.1016/j.regg.2011.02.010openalex-snapshot
Osteoporosis senil: una actualización
inferred← citeshttps://doi.org/10.1016/j.semcancer.2008.03.019openalex-snapshot
The microenvironments of multistage carcinogenesis
inferred← citeshttps://doi.org/10.1016/j.stem.2007.03.002openalex-snapshot
Deletion of the Developmentally Essential Gene ATR in Adult Mice Leads to Age-Related Phenotypes and Stem Cell Loss
showing the first 200 of 527 · page through all 527
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.