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A Genome-Wide Association Study Identifies IL23R as an Inflammatory Bowel Disease Gene
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/s00251-005-0067-0openalex-snapshot
Identification and expression analysis of alternatively spliced isoforms of human interleukin-23 receptor gene in normal lymphoid cells and selected tumor cells
inferredcites →https://doi.org/10.1016/j.immuni.2006.01.001openalex-snapshot
TGFβ in the Context of an Inflammatory Cytokine Milieu Supports De Novo Differentiation of IL-17-Producing T Cells
inferredcites →https://doi.org/10.1016/j.it.2005.10.003openalex-snapshot
Understanding the IL-23–IL-17 immune pathway
inferredcites →https://doi.org/10.1016/s1074-7613(00)00070-4openalex-snapshot
Novel p19 Protein Engages IL-12p40 to Form a Cytokine, IL-23, with Biological Activities Similar as Well as Distinct from IL-12
inferredcites →https://doi.org/10.1038/35079107openalex-snapshot
Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn's disease
inferredcites →https://doi.org/10.1038/35079114openalex-snapshot
A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease
inferredcites →https://doi.org/10.1038/nature01355openalex-snapshot
Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain
inferredcites →https://doi.org/10.1038/nature04753openalex-snapshot
Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells
inferredcites →https://doi.org/10.1038/nature04754openalex-snapshot
Transforming growth factor-β induces development of the TH17 lineage
inferredcites →https://doi.org/10.1038/ng1001-223openalex-snapshot
Genetic variation in the 5q31 cytokine gene cluster confers susceptibility to Crohn disease
inferredcites →https://doi.org/10.1038/ng1001-229openalex-snapshot
High-resolution haplotype structure in the human genome
inferredcites →https://doi.org/10.1038/ng1339openalex-snapshot
Functional variants of OCTN cation transporter genes are associated with Crohn disease
inferredcites →https://doi.org/10.1038/sj.gene.6364257openalex-snapshot
Current status of genetics research in inflammatory bowel disease
inferredcites →https://doi.org/10.1053/j.gastro.2004.01.063openalex-snapshot
Clinical epidemiology of inflammatory bowel disease: incidence, prevalence, and environmental influences
inferredcites →https://doi.org/10.1056/nejmoa033402openalex-snapshot
Anti–Interleukin-12 Antibody for Active Crohn's Disease
inferredcites →https://doi.org/10.1073/pnas.0400983101openalex-snapshot
Human IL-23-producing type 1 macrophages promote but IL-10-producing type 2 macrophages subvert immunity to (myco)bacteria
inferredcites →https://doi.org/10.1074/jbc.m207577200openalex-snapshot
Interleukin-23 Promotes a Distinct CD4 T Cell Activation State Characterized by the Production of Interleukin-17
inferredcites →https://doi.org/10.1084/jem.20030896openalex-snapshot
Divergent Pro- and Antiinflammatory Roles for IL-23 and IL-12 in Joint Autoimmune Inflammation
inferredcites →https://doi.org/10.1084/jem.20041257openalex-snapshot
IL-23 drives a pathogenic T cell population that induces autoimmune inflammation
inferredcites →https://doi.org/10.1097/01.mib.0000194183.92671.b6openalex-snapshot
Both IL-12p70 and IL-23 are synthesized during active Crohnʼs disease and are down-regulated by treatment with anti-IL-12 p40 monoclonal antibody
inferredcites →https://doi.org/10.1136/gut.52.1.65openalex-snapshot
Increased expression of interleukin 17 in inflammatory bowel disease
inferredcites →https://doi.org/10.1172/jci17464openalex-snapshot
Constitutive p40 promoter activation and IL-23 production in the terminal ileum mediated by dendritic cells
inferredcites →https://doi.org/10.1172/jci21404openalex-snapshot
IL-23 is essential for T cell–mediated colitis and promotes inflammation via IL-17 and IL-6
inferredcites →https://doi.org/10.4049/jimmunol.166.12.7563openalex-snapshot
Ubiquitous Transgenic Expression of the IL-23 Subunit p19 Induces Multiorgan Inflammation, Runting, Infertility, and Premature Death
inferredcites →https://doi.org/10.4049/jimmunol.168.11.5699openalex-snapshot
A Receptor for the Heterodimeric Cytokine IL-23 Is Composed of IL-12Rβ1 and a Novel Cytokine Receptor Subunit, IL-23R
inferredcites →https://doi.org/10.4049/jimmunol.168.3.1322openalex-snapshot
Mice Lacking Bioactive IL-12 Can Generate Protective, Antigen-Specific Cellular Responses to Mycobacterial Infection Only if the IL-12 p40 Subunit Is Present
inferredcites →https://doi.org/10.4049/jimmunol.177.5.2760openalex-snapshot
Cutting Edge: IL-23 Cross-Regulates IL-12 Production in T Cell-Dependent Experimental Colitis
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Serologic and genetic markers do not aid in the determination of the clinical course and severity of patients with inflammatory bowel disease
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Association of IL23R, TNFRSF1A, and HLA-DRB1*0103 allele variants with inflammatory bowel disease phenotypes in the Finnish population
inferred← citeshttps://doi.org/10.1002/ibd.20441openalex-snapshot
NOD2/CARD15 genotype influences MDP-induced cytokine release and basal IL-12p40 levels in primary isolated peripheral blood monocytes
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Probiotic Lactobacillus reuteri suppress proinflammatory cytokines via c-Jun
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Lymphocyte homing and its role in the pathogenesis of IBD
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IL23R and IL12B polymorphisms in spanish IBD patients: No evidence of interaction
inferred← citeshttps://doi.org/10.1002/ibd.20475openalex-snapshot
IL-23/IL-17 immunity as a hallmark of Crohnʼs disease
inferred← citeshttps://doi.org/10.1002/ibd.20478openalex-snapshot
IL23R haplotypes provide a large population attributable risk for Crohnʼs disease
inferred← citeshttps://doi.org/10.1002/ibd.20488openalex-snapshot
Role of functional polymorphisms of NRAMP1 gene for the development of Crohnʼs disease
inferred← citeshttps://doi.org/10.1002/ibd.20512openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/ibd.20517openalex-snapshot
Clinical and molecular characteristics of isolated colonic Crohnʼs disease
inferred← citeshttps://doi.org/10.1002/ibd.20611openalex-snapshot
MAGI2 genetic variation and inflammatory bowel disease
inferred← citeshttps://doi.org/10.1002/ibd.20625openalex-snapshot
IL-22: A Two-Headed Cytokine in IBD?
inferred← citeshttps://doi.org/10.1002/ibd.20646openalex-snapshot
Family history of Crohnʼs disease is associated with an increased risk for Crohnʼs disease of the pouch
inferred← citeshttps://doi.org/10.1002/ibd.20808openalex-snapshot
Reduced expression of basal and probiotic-inducible G-CSF in intestinal mononuclear cells is associated with inflammatory bowel disease
inferred← citeshttps://doi.org/10.1002/ibd.20849openalex-snapshot
Associations between ABCB1/MDR1 gene polymorphisms and Crohnʼs disease: A gene-wide study in a pediatric population
inferred← citeshttps://doi.org/10.1002/ibd.20855openalex-snapshot
Genetic epistasis of IL23/IL17 pathway genes in Crohnʼs disease
inferred← citeshttps://doi.org/10.1002/ibd.20885openalex-snapshot
Association between genetic variants in myosin IXB and Crohnʼs disease
inferred← citeshttps://doi.org/10.1002/ibd.20894openalex-snapshot
Interleukin-23/Th17 pathways and inflammatory bowel disease
inferred← citeshttps://doi.org/10.1002/ibd.20921openalex-snapshot
Contribution of IL23R but not ATG16L1 to Crohnʼs disease susceptibility in Koreans
inferred← citeshttps://doi.org/10.1002/ibd.20922openalex-snapshot
Established genetic risk factors do not distinguish early and later onset Crohnʼs disease
inferred← citeshttps://doi.org/10.1002/ibd.20936openalex-snapshot
Genetic variants in surfactant, pulmonary-associated protein D (SFTPD) and Japanese susceptibility to ulcerative colitis
inferred← citeshttps://doi.org/10.1002/ibd.20941openalex-snapshot
Immunizations in patients with inflammatory bowel disease
inferred← citeshttps://doi.org/10.1002/ibd.20963openalex-snapshot
PepT1 oligopeptide transporter (SLC15A1) gene polymorphism in inflammatory bowel disease
inferred← citeshttps://doi.org/10.1002/ibd.20968openalex-snapshot
Role of diet in the development of inflammatory bowel disease
inferred← citeshttps://doi.org/10.1002/ibd.21001openalex-snapshot
Role of ATG16L1 Thr300Ala polymorphism in inflammatory bowel disease: A Study in the Spanish population and a meta-analysis
inferred← citeshttps://doi.org/10.1002/ibd.21159openalex-snapshot
Randomized, double-blind, placebo-controlled trial of the oral interleukin-12/23 inhibitor apilimod mesylate for treatment of active Crohn's disease
inferred← citeshttps://doi.org/10.1002/ibd.21171openalex-snapshot
The path to Crohnʼs disease: Is mucosal pathology a secondary event?
inferred← citeshttps://doi.org/10.1002/ibd.21176openalex-snapshot
Variants at the 3p21 locus influence susceptibility and phenotype both in adults and early-onset patients with inflammatory bowel disease
inferred← citeshttps://doi.org/10.1002/ibd.21263openalex-snapshot
Characterization of intestinal gene expression profiles in Crohnʼs disease by genome-wide microarray analysis
inferred← citeshttps://doi.org/10.1002/ibd.21283openalex-snapshot
Genetics of childhood-onset inflammatory bowel disease
inferred← citeshttps://doi.org/10.1002/ibd.21312openalex-snapshot
Strong overexpression of CXCR3 axis components in childhood inflammatory bowel disease
inferred← citeshttps://doi.org/10.1002/ibd.21352openalex-snapshot
Distinct IFNG methylation in a subset of ulcerative colitis patients based on reactivity to microbial antigens
inferred← citeshttps://doi.org/10.1002/ibd.21384openalex-snapshot
Comparative analysis of the effects of anti-IL-6 receptor mAb and anti-TNF mAb treatment on CD4+ T-cell responses in murine colitis
inferred← citeshttps://doi.org/10.1002/ibd.21390openalex-snapshot
Inflammatory bowel disease aggregation in Utah kindreds
inferred← citeshttps://doi.org/10.1002/ibd.21532openalex-snapshot
Two independent genetic factors responsible for the associations of the IBD5 locus with Crohnʼs disease in the Czech population
inferred← citeshttps://doi.org/10.1002/ibd.21562openalex-snapshot
Pregnane X receptor (PXR/NR1I2) gene haplotypes modulate susceptibility to inflammatory bowel disease
inferred← citeshttps://doi.org/10.1002/ibd.21608openalex-snapshot
Associations between variants in the ABCB1 (MDR1) gene and corticosteroid dependence in children with Crohnʼs disease
inferred← citeshttps://doi.org/10.1002/ibd.21630openalex-snapshot
Differential association of two PTPN22 coding variants with Crohnʼs disease and ulcerative colitis
inferred← citeshttps://doi.org/10.1002/ibd.21638openalex-snapshot
SAMP1/YitFc mouse strain: A spontaneous model of Crohnʼs disease-like ileitis
inferred← citeshttps://doi.org/10.1002/ibd.21651openalex-snapshot
Meta-analysis of published studies identified eight additional common susceptibility loci for Crohnʼs disease and ulcerative colitis
inferred← citeshttps://doi.org/10.1002/ibd.21656openalex-snapshot
Are NOD2 polymorphisms linked to a specific disease endophenotype of Crohnʼs disease?
inferred← citeshttps://doi.org/10.1002/ibd.21677openalex-snapshot
Functional relevance of T helper 17 (Th17) cells and the IL-17 cytokine family in inflammatory bowel disease
inferred← citeshttps://doi.org/10.1002/ibd.21728openalex-snapshot
Is there evidence to claim or deny association between variants of the multidrug resistance gene (MDR1 or ABCB1) and inflammatory bowel disease?
inferred← citeshttps://doi.org/10.1002/ibd.21735openalex-snapshot
New Pathogenic Paradigms in Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1002/ibd.21750openalex-snapshot
Involvement of mycobacterium avium subspecies paratuberculosis in TNF-α production from macrophage: Possible link between MAP and immune response in Crohnʼs disease
inferred← citeshttps://doi.org/10.1002/ibd.21845openalex-snapshot
Genetics of inflammatory bowel disease in Asia: Systematic review and meta-analysis
inferred← citeshttps://doi.org/10.1002/ibd.21905openalex-snapshot
Compartmentalized Expression of Th1 and Th17 Cytokines in Pediatric Inflammatory Bowel Diseases
inferred← citeshttps://doi.org/10.1002/ibd.22884openalex-snapshot
Pattern recognition receptor and autophagy gene variants are associated with development of antimicrobial antibodies in Crohnʼs disease
inferred← citeshttps://doi.org/10.1002/ibd.22931openalex-snapshot
Contribution of higher risk genes and European admixture to Crohnʼs disease in African Americans
inferred← citeshttps://doi.org/10.1002/ibd.22942openalex-snapshot
Mucosal genome-wide methylation changes in inflammatory bowel disease
inferred← citeshttps://doi.org/10.1002/ibd.22972openalex-snapshot
Associations Between Genetic Variants in the IRGM Gene and Inflammatory Bowel Diseases in the Korean Population
inferred← citeshttps://doi.org/10.1002/ijc.23666openalex-snapshot
Cancer risks in ulcerative colitis patients
inferred← citeshttps://doi.org/10.1002/ijc.26067openalex-snapshot
Host immune gene polymorphisms were associated with the prognosis of non‐small‐cell lung cancer in Chinese
inferred← citeshttps://doi.org/10.1002/ijc.26130openalex-snapshot
Potentially functional polymorphisms in IL‐23 receptor and risk of esophageal cancer in a Chinese population
inferred← citeshttps://doi.org/10.1002/ijc.27660openalex-snapshot
Interleukin genes and associations with colon and rectal cancer risk and overall survival
inferred← citeshttps://doi.org/10.1002/jbmr.186openalex-snapshot
Polymorphisms in predicted miRNA binding sites and osteoporosis
inferred← citeshttps://doi.org/10.1002/jcc5.70086openalex-snapshot
Early and Sustained Comprehensive Symptom Control, Including Bowel Urgency and Fatigue, With Mirikizumab in Crohn's Disease: An Exploratory Post Hoc Analysis of the Phase 3 VIVID‐1 Study
inferred← citeshttps://doi.org/10.1002/jcc5.70088openalex-snapshot
Risankizumab in a Japanese Real‐World Crohn's Disease Cohort: Clinical Effectiveness in Moderate‐to‐Severely Active Disease, Endoscopic Outcomes, and Persistence
inferred← citeshttps://doi.org/10.1002/jcla.23472openalex-snapshot
Interleukin‐12B gene rs6887695 and rs2288831 polymorphisms are associated with an increased risk of ulcerative colitis development in Chinese Han population: A case‐control study
inferred← citeshttps://doi.org/10.1002/jcp.26250openalex-snapshot
The role of innate lymphoid cells in health and disease
inferred← citeshttps://doi.org/10.1002/jgh3.12398openalex-snapshot
TL1A ( TNFSF15 ) genotype affects the long‐term therapeutic outcomes of anti‐TNFα antibodies for Crohn's disease patients
inferred← citeshttps://doi.org/10.1002/jms.1858openalex-snapshot
Glycosylation analysis of interleukin‐23 receptor: elucidation of glycosylation sites and characterization of attached glycan structures
inferred← citeshttps://doi.org/10.1002/jpn3.70275openalex-snapshot
Reshaping study design for faster extrapolation‐based drug approval in pediatric inflammatory bowel diseases: An ESPGHAN–NASPGHAN position paper
inferred← citeshttps://doi.org/10.1002/jso.24505openalex-snapshot
Increased expression of interleukin‐23 associated with progression of colorectal cancer
inferred← citeshttps://doi.org/10.1002/mc.20661openalex-snapshot
A nonsynonymous polymorphism in IL23R gene is associated with risk of gastric cancer in a Chinese population
inferred← citeshttps://doi.org/10.1002/path.2286openalex-snapshot
Innate and adaptive mechanisms to control of pathological intestinal inflammation
inferred← citeshttps://doi.org/10.1002/path.2291openalex-snapshot
Innate immunity in inflammatory bowel disease: a disease hypothesis
inferred← citeshttps://doi.org/10.1002/path.4277openalex-snapshot
Dendritic cells in IBD pathogenesis: an area of therapeutic opportunity?
inferred← citeshttps://doi.org/10.1002/path.4670openalex-snapshot
NUR77 exerts a protective effect against inflammatory bowel disease by negatively regulating the TRAF6 / TLR–IL‐1R signalling axis
inferred← citeshttps://doi.org/10.1002/path.4817openalex-snapshot
The genetic background of inflammatory bowel disease: from correlation to causality
inferred← citeshttps://doi.org/10.1002/pauz.200790023openalex-snapshot
Die Früchte der Postgenom‐Analyse: Identifizierung von Risikogenen für Morbus Crohn und Typ‐2‐Diabetes
inferred← citeshttps://doi.org/10.1002/prot.24472openalex-snapshot
Human interleukin‐23 receptor antagonists derived from an albumin‐binding domain scaffold inhibit IL‐23‐dependent ex vivo expansion of IL‐17‐producing T‐cells
inferred← citeshttps://doi.org/10.1002/wsbm.48openalex-snapshot
Host‐bacteria interactions in the intestine: homeostasis to chronic inflammation
inferred← citeshttps://doi.org/10.1007/400_2008_17openalex-snapshot
Immunological Basis for the Development of Tissue Inflammation and Organ-Specific Autoimmunity in Animal Models of Multiple Sclerosis
inferred← citeshttps://doi.org/10.1007/978-0-387-09550-9_6openalex-snapshot
Negative Interactions with the Microbiota: IBD
inferred← citeshttps://doi.org/10.1007/978-0-387-30440-3_82openalex-snapshot
Complexity in Systems Level Biology and Genetics: Statistical Perspectives
inferred← citeshttps://doi.org/10.1007/978-0-387-73481-1_1openalex-snapshot
Genetics of Inflammatory Bowel Diseases
inferred← citeshttps://doi.org/10.1007/978-0-387-73481-1_3openalex-snapshot
Cytokines and Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1007/978-0-387-79251-4_18openalex-snapshot
DNA Biomarkers for Pharmacogenomics and Personalized Medicine
inferred← citeshttps://doi.org/10.1007/978-0-387-79311-5_1openalex-snapshot
THE PROTECTIVE EFFECTS OF TYPE-1 INTERFERON IN MODELS OF INTESTINAL INFLAMMATION
inferred← citeshttps://doi.org/10.1007/978-0-387-87429-6_63openalex-snapshot
Genomics of Acute Lung Injury and Vascular Barrier Dysfunction
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