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The Severity of CVB3‐Induced Myocarditis Can Be Improved by Blocking the Orchestration of NLRP3 and Th17 in Balb/c Mice

https://doi.org/10.1155/2021/5551578

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1 of the 44 references listed on this page is in the Retraction Watch database as retracted (export 2026-10-28): 10.3892/mmr.2013.1507 (notice 2017-09-20).

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1016/j.bbi.2016.12.014openalex-snapshot

Inhibiting the NLRP3 inflammasome with MCC950 promotes non-phlogistic clearance of amyloid-β and cognitive function in APP/PS1 mice

inferredcites →https://doi.org/10.1016/j.cell.2006.07.035openalex-snapshot

The Orphan Nuclear Receptor RORγt Directs the Differentiation Program of Proinflammatory IL-17+ T Helper Cells

inferredcites →https://doi.org/10.1016/j.cell.2014.04.007openalex-snapshot

Mechanisms and Functions of Inflammasomes

inferredcites →https://doi.org/10.1016/j.immuni.2007.11.016openalex-snapshot

T Helper 17 Lineage Differentiation Is Programmed by Orphan Nuclear Receptors RORα and RORγ

inferredcites →https://doi.org/10.1016/j.immuni.2020.01.002openalex-snapshot

Interleukin-17A Serves a Priming Role in Autoimmunity by Recruiting IL-1β-Producing Myeloid Cells that Promote Pathogenic T Cells

inferredcites →https://doi.org/10.1016/j.jhep.2017.01.022openalex-snapshot

NLRP3 inflammasome blockade reduces liver inflammation and fibrosis in experimental NASH in mice

inferredcites →https://doi.org/10.1016/j.molimm.2019.07.012openalex-snapshot

Macrophage NLRP3 inflammasome activated by CVB3 capsid proteins contributes to the development of viral myocarditis

inferredcites →https://doi.org/10.1016/j.ymeth.2006.07.009openalex-snapshot

Coxsackievirus-induced myocarditis in mice: A model of autoimmune disease for studying immunotoxicity

inferredcites →https://doi.org/10.1016/s0140-6736(15)60692-4openalex-snapshot

Global, regional, and national incidence, prevalence, and years lived with disability for 301 acute and chronic diseases and injuries in 188 countries, 1990–2013: a systematic analysis for the Global Burden of Disease Study 2013

inferredcites →https://doi.org/10.1038/ncomms13344openalex-snapshot

Macrophage-dependent IL-1β production induces cardiac arrhythmias in diabetic mice

inferredcites →https://doi.org/10.1038/nm.3806openalex-snapshot

A small-molecule inhibitor of the NLRP3 inflammasome for the treatment of inflammatory diseases

inferredcites →https://doi.org/10.1038/nm.3893openalex-snapshot

Inflammasomes: mechanism of action, role in disease, and therapeutics

inferredcites →https://doi.org/10.1038/nm960openalex-snapshot

Dendritic cell–induced autoimmune heart failure requires cooperation between adaptive and innate immunity

inferredcites →https://doi.org/10.1038/nrcardio.2015.108openalex-snapshot

Viral myocarditis—diagnosis, treatment options, and current controversies

inferredcites →https://doi.org/10.1038/s41467-018-04947-6openalex-snapshot

Oridonin is a covalent NLRP3 inhibitor with strong anti-inflammasome activity

inferredcites →https://doi.org/10.1038/s41577-019-0165-0openalex-snapshot

The NLRP3 inflammasome: molecular activation and regulation to therapeutics

inferredcites →https://doi.org/10.1038/s41598-018-20686-6openalex-snapshot

Mesenchymal stromal cells inhibit NLRP3 inflammasome activation in a model of Coxsackievirus B3-induced inflammatory cardiomyopathy

inferredcites →https://doi.org/10.1038/s41598-018-26775-wopenalex-snapshot

MCC950, a specific small molecule inhibitor of NLRP3 inflammasome attenuates colonic inflammation in spontaneous colitis mice

inferredcites →https://doi.org/10.1073/pnas.1108586108openalex-snapshot

The inflammasome promotes adverse cardiac remodeling following acute myocardial infarction in the mouse

inferredcites →https://doi.org/10.1073/pnas.1716095115openalex-snapshot

OLT1177, a β-sulfonyl nitrile compound, safe in humans, inhibits the NLRP3 inflammasome and reverses the metabolic cost of inflammation

inferredcites →https://doi.org/10.1074/jbc.ac118.002127openalex-snapshot

RORγ regulates the NLRP3 inflammasome

inferredcites →https://doi.org/10.1074/jbc.m113.515080openalex-snapshot

3,4-Methylenedioxy-β-nitrostyrene Inhibits NLRP3 Inflammasome Activation by Blocking Assembly of the Inflammasome

inferredcites →https://doi.org/10.1084/jem.20130084openalex-snapshot

The Nlrp3 inflammasome regulates acute graft-versus-host disease

inferredcites →https://doi.org/10.1084/jem.20171419openalex-snapshot

Identification of a selective and direct NLRP3 inhibitor to treat inflammatory disorders

inferredcites →https://doi.org/10.1089/neu.2017.5344openalex-snapshot

MCC950, the Selective Inhibitor of Nucleotide Oligomerization Domain-Like Receptor Protein-3 Inflammasome, Protects Mice against Traumatic Brain Injury

inferredcites →https://doi.org/10.1093/eurheartj/ehw247openalex-snapshot

The selective NLRP3-inflammasome inhibitor MCC950 reduces infarct size and preserves cardiac function in a pig model of myocardial infarction

inferredcites →https://doi.org/10.1093/jmcb/mjr042openalex-snapshot

Inflammasome-IL-1-Th17 response in allergic lung inflammation

inferredcites →https://doi.org/10.1097/bor.0b013e328353372dopenalex-snapshot

Update on coxsackievirus B3 myocarditis

inferredcites →https://doi.org/10.1146/annurev.pathmechdis.3.121806.151534openalex-snapshot

Molecular Biology and Pathogenesis of Viral Myocarditis

inferredcites →https://doi.org/10.1146/annurev.pathol.3.121806.151534openalex-snapshot

Molecular Biology and Pathogenesis of Viral Myocarditis

inferredcites →https://doi.org/10.1152/ajpheart.00441.2014openalex-snapshot

Involvement of NLRP3 inflammasome in CVB3-induced viral myocarditis

inferredcites →https://doi.org/10.1161/atvbaha.117.309575openalex-snapshot

NLRP3 Inflammasome Inhibition by MCC950 Reduces Atherosclerotic Lesion Development in Apolipoprotein E–Deficient Mice—Brief Report

inferredcites →https://doi.org/10.1161/circheartfailure.117.003870openalex-snapshot

NOD2 (Nucleotide-Binding Oligomerization Domain 2) Is a Major Pathogenic Mediator of Coxsackievirus B3-Induced Myocarditis

inferredcites →https://doi.org/10.1161/circulationaha.110.982777openalex-snapshot

Inflammasome Activation of Cardiac Fibroblasts Is Essential for Myocardial Ischemia/Reperfusion Injury

inferredcites →https://doi.org/10.1186/1743-422x-8-17openalex-snapshot

Treatment with a neutralizing anti-murine interleukin-17 antibody after the onset of coxsackievirus b3-induced viral myocarditis reduces myocardium inflammation

inferredcites →https://doi.org/10.1186/1743-422x-8-301openalex-snapshot

Expression of il-23/th17 pathway in a murine model of coxsackie virus b3-induced viral myocarditis

inferredcites →https://doi.org/10.1371/journal.pone.0032270openalex-snapshot

NLRP3 Inflammasome: Key Mediator of Neuroinflammation in Murine Japanese Encephalitis

inferredcites →https://doi.org/10.15252/emmm.201708689openalex-snapshot

Tranilast directly targets NLRP3 to treat inflammasome‐driven diseases

inferredcites →https://doi.org/10.2337/db12-0420openalex-snapshot

Upregulated NLRP3 Inflammasome Activation in Patients With Type 2 Diabetes

inferredcites →https://doi.org/10.3390/molecules23030522openalex-snapshot

Inhibiting the NLRP3 Inflammasome Activation with MCC950 Ameliorates Diabetic Encephalopathy in db/db Mice

inferredcites →https://doi.org/10.3892/mmr.2013.1507openalex-snapshot

Cathepsin B inhibition attenuates cardiac dysfunction and remodeling following myocardial infarction by inhibiting the NLRP3 pathway

inferredcites →https://doi.org/10.3892/mmr.2017.7551openalex-snapshot

Cathepsin B inhibition attenuates cardiac dysfunction and remodeling following myocardial infarction by inhibiting the NLRP3 pathway

inferredcites →https://doi.org/10.4049/jimmunol.0904145openalex-snapshot

NLRP3 Plays a Critical Role in the Development of Experimental Autoimmune Encephalomyelitis by Mediating Th1 and Th17 Responses

inferredcites →https://doi.org/10.4049/jimmunol.1003597openalex-snapshot

Caspase-1–Processed Cytokines IL-1β and IL-18 Promote IL-17 Production by γδ and CD4 T Cells That Mediate Autoimmunity

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Shengxian Decoction Protects Against Obesity-Induced Cardiac Injuryin Rats by Regulating the Hk1/Nlrp3 Signalling Pathway

inferred← citeshttps://doi.org/10.1007/s11427-022-2273-3openalex-snapshot

Immunopathogenesis and immunomodulatory therapy for myocarditis

inferred← citeshttps://doi.org/10.1016/j.bbrep.2026.102447openalex-snapshot

NLRP3 inflammasome regulates Th17/Treg cell balance in experimental autoimmune myocarditis

inferred← citeshttps://doi.org/10.1016/j.intimp.2022.109024openalex-snapshot

SGLT2 inhibitor, canagliflozin, ameliorates cardiac inflammation in experimental autoimmune myocarditis

inferred← citeshttps://doi.org/10.1016/j.virusres.2025.199635openalex-snapshot

Effect of interleukin-17A on anti-ANT antibodies as well as cytokines in viral myocarditis mice

inferred← citeshttps://doi.org/10.1093/jleuko/qiad143openalex-snapshot

Role of immune cells in the pathogenesis of myocarditis

inferred← citeshttps://doi.org/10.1111/imr.70111openalex-snapshot

Immune Mechanisms of Viral, Autoimmune, and Immune Checkpoint Inhibitor‐Associated Myocarditis

inferred← citeshttps://doi.org/10.14715/cmb/2023.69.15.31openalex-snapshot

Analysis of the improvement effect of Astragalus extract on oxidative stress injury in viral myocarditis through STAT3/IL-6

inferred← citeshttps://doi.org/10.3390/microorganisms13102294openalex-snapshot

Cyclodextrin Counteracts Coxsackievirus-Induced Cardiac Damage by Protecting Desmosome Integrity and Suppressing Proinflammatory Cytokine Expression

inferred← citeshttps://doi.org/10.3390/v15051137openalex-snapshot

Inhibitor of CD147 Suppresses T Cell Activation and Recruitment in CVB3-Induced Acute Viral Myocarditis

inferred← citeshttps://doi.org/10.70675/e6901139zdba3z455bz9899z0821c7851b2eopenalex-snapshot

Impact des ARN génomiques des Entérovirus-B tronqués en région 5’non-codante sur leur pathogénicité et sur la réponse interféron de type 1 dans les cardiomyocytes et les cellules bêta-pancréatiques in vivo et in vitro

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