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Inflammation and NLRP3 Inflammasome Activation Initiated in Response to Pressure Overload by Ca 2+ /Calmodulin-Dependent Protein Kinase II δ Signaling in Cardiomyocytes Are Essential for Adverse Cardiac Remodeling
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/s00418-008-0541-5openalex-snapshot
Characterization of the inflammatory and fibrotic response in a mouse model of cardiac pressure overload
inferredcites →https://doi.org/10.1016/j.bbrc.2017.02.021openalex-snapshot
Triptolide attenuates pressure overload-induced myocardial remodeling in mice via the inhibition of NLRP3 inflammasome expression
inferredcites →https://doi.org/10.1016/j.jacbts.2017.12.006openalex-snapshot
CCR2+ Monocyte-Derived Infiltrating Macrophages Are Required for Adverse Cardiac Remodeling During Pressure Overload
inferredcites →https://doi.org/10.1016/j.jacc.2016.01.073openalex-snapshot
Inflammation as a Driver of Adverse Left Ventricular Remodeling After Acute Myocardial Infarction
inferredcites →https://doi.org/10.1016/j.yjmcc.2015.06.007openalex-snapshot
CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca2+ leak and the pathophysiological response to chronic β-adrenergic stimulation
inferredcites →https://doi.org/10.1016/j.yjmcc.2015.11.015openalex-snapshot
Monocyte and macrophage contributions to cardiac remodeling
inferredcites →https://doi.org/10.1016/j.yjmcc.2016.12.007openalex-snapshot
CaMKII is a nodal signal for multiple programmed cell death pathways in heart
inferredcites →https://doi.org/10.1038/labinvest.2012.97openalex-snapshot
Pressure overload-induced cardiac remodeling and dysfunction in the absence of interleukin 6 in mice
inferredcites →https://doi.org/10.1038/nature10992openalex-snapshot
Mitochondrial DNA that escapes from autophagy causes inflammation and heart failure
inferredcites →https://doi.org/10.1038/nature11444openalex-snapshot
CaMKII determines mitochondrial stress responses in heart
inferredcites →https://doi.org/10.1038/nm.3893openalex-snapshot
Inflammasomes: mechanism of action, role in disease, and therapeutics
inferredcites →https://doi.org/10.1038/nm.4428openalex-snapshot
IRF3 and type I interferons fuel a fatal response to myocardial infarction
inferredcites →https://doi.org/10.1038/nrcardio.2017.161openalex-snapshot
The NLRP3 inflammasome in acute myocardial infarction
inferredcites →https://doi.org/10.1038/nri2725openalex-snapshot
NLRP3 inflammasome activation: the convergence of multiple signalling pathways on ROS production?
inferredcites →https://doi.org/10.1038/nri3800openalex-snapshot
Role of innate and adaptive immune mechanisms in cardiac injury and repair
inferredcites →https://doi.org/10.1056/nejmoa1707914openalex-snapshot
Antiinflammatory Therapy with Canakinumab for Atherosclerotic Disease
inferredcites →https://doi.org/10.1073/pnas.1108586108openalex-snapshot
The inflammasome promotes adverse cardiac remodeling following acute myocardial infarction in the mouse
inferredcites →https://doi.org/10.1074/jbc.r114.619304openalex-snapshot
Complexity of Danger: The Diverse Nature of Damage-associated Molecular Patterns
inferredcites →https://doi.org/10.1084/jem.20171274openalex-snapshot
Cardiac macrophages promote diastolic dysfunction
inferredcites →https://doi.org/10.1089/ars.2014.5989openalex-snapshot
The Inflammasome in Myocardial Injury and Cardiac Remodeling
inferredcites →https://doi.org/10.1093/cvr/cvt091openalex-snapshot
The NLRP3 inflammasome is up-regulated in cardiac fibroblasts and mediates myocardial ischaemia–reperfusion injury
inferredcites →https://doi.org/10.1093/cvr/cvu030openalex-snapshot
Myocyte signalling in leucocyte recruitment to the heart
inferredcites →https://doi.org/10.1093/eurheartj/ehx261openalex-snapshot
IL-18 cleavage triggers cardiac inflammation and fibrosis upon β-adrenergic insult
inferredcites →https://doi.org/10.1093/eurjhf/hfn043openalex-snapshot
Inflammation as a Therapeutic Target in Heart Failure? A Scientific Statement from the Translational Research Committee of the Heart Failure Association of the European Society of Cardiology
inferredcites →https://doi.org/10.1111/eci.12118openalex-snapshot
Targeting inflammatory pathways in myocardial infarction
inferredcites →https://doi.org/10.1152/ajpcell.00338.2010openalex-snapshot
mTOR attenuates the inflammatory response in cardiomyocytes and prevents cardiac dysfunction in pathological hypertrophy
inferredcites →https://doi.org/10.1152/ajpheart.00795.2013openalex-snapshot
Interleukin-18 mediates interleukin-1-induced cardiac dysfunction
inferredcites →https://doi.org/10.1161/01.res.0000069686.31472.c5openalex-snapshot
The δ C Isoform of CaMKII Is Activated in Cardiac Hypertrophy and Induces Dilated Cardiomyopathy and Heart Failure
inferredcites →https://doi.org/10.1161/circheartfailure.115.002225openalex-snapshot
Left Ventricular T-Cell Recruitment Contributes to the Pathogenesis of Heart Failure
inferredcites →https://doi.org/10.1161/circheartfailure.117.003840openalex-snapshot
Calcium/Calmodulin-Dependent Protein Kinase II Activity Persists During Chronic β-Adrenoceptor Blockade in Experimental and Human Heart Failure
inferredcites →https://doi.org/10.1161/circheartfailure.117.004373openalex-snapshot
Interleukin-1 Blockade in Recently Decompensated Systolic Heart Failure
inferredcites →https://doi.org/10.1161/circresaha.109.207423openalex-snapshot
Phospholamban Ablation Rescues Sarcoplasmic Reticulum Ca 2+ Handling but Exacerbates Cardiac Dysfunction in CaMKIIδ C Transgenic Mice
inferredcites →https://doi.org/10.1161/circresaha.112.276915openalex-snapshot
Ca 2+ /Calmodulin-Dependent Protein Kinase II δ Mediates Myocardial Ischemia/Reperfusion Injury Through Nuclear Factor-κB
inferredcites →https://doi.org/10.1161/circresaha.116.304682openalex-snapshot
Mitochondrial Reprogramming Induced by CaMKIIδ Mediates Hypertrophy Decompensation
inferredcites →https://doi.org/10.1161/circresaha.116.308030openalex-snapshot
Chronic Heart Failure and Inflammation
inferredcites →https://doi.org/10.1161/circresaha.116.308688openalex-snapshot
Deletion of Interleukin-6 Attenuates Pressure Overload-Induced Left Ventricular Hypertrophy and Dysfunction
inferredcites →https://doi.org/10.1161/circulationaha.110.950543openalex-snapshot
Distinct Effects of Leukocyte and Cardiac Phosphoinositide 3-Kinase γ Activity in Pressure Overload–Induced Cardiac Failure
inferredcites →https://doi.org/10.1161/circulationaha.110.982777openalex-snapshot
Inflammasome Activation of Cardiac Fibroblasts Is Essential for Myocardial Ischemia/Reperfusion Injury
inferredcites →https://doi.org/10.1161/circulationaha.113.007101openalex-snapshot
CD4 + T Cells Promote the Transition From Hypertrophy to Heart Failure During Chronic Pressure Overload
inferredcites →https://doi.org/10.1161/circulationaha.114.006185openalex-snapshot
Cardiac CaM Kinase II Genes δ and γ Contribute to Adverse Remodeling but Redundantly Inhibit Calcineurin-Induced Myocardial Hypertrophy
inferredcites →https://doi.org/10.1161/jaha.115.003126openalex-snapshot
Intercellular Adhesion Molecule 1 Regulates Left Ventricular Leukocyte Infiltration, Cardiac Remodeling, and Function in Pressure Overload–Induced Heart Failure
inferredcites →https://doi.org/10.1172/jci.insight.97054openalex-snapshot
CaMKIIδ-mediated inflammatory gene expression and inflammasome activation in cardiomyocytes initiate inflammation and induce fibrosis
inferredcites →https://doi.org/10.1172/jci35814openalex-snapshot
Ca2+/calmodulin-dependent kinase II triggers cell membrane injury by inducing complement factor B gene expression in the mouse heart
inferredcites →https://doi.org/10.1172/jci38022openalex-snapshot
Requirement for Ca2+/calmodulin–dependent kinase II in the transition from pressure overload–induced cardiac hypertrophy to heart failure in mice
inferredcites →https://doi.org/10.1172/jci43171openalex-snapshot
Cyclophilin D controls mitochondrial pore–dependent Ca2+ exchange, metabolic flexibility, and propensity for heart failure in mice
inferredcites →https://doi.org/10.1371/journal.pone.0112710openalex-snapshot
Ly6Clow and Not Ly6Chigh Macrophages Accumulate First in the Heart in a Model of Murine Pressure-Overload
inferredcites →https://doi.org/10.1371/journal.pone.0142921openalex-snapshot
Tlr4 Deficiency Protects against Cardiac Pressure Overload Induced Hyperinflammation
inferredcites →https://doi.org/10.1371/journal.pone.0170781openalex-snapshot
Mononuclear Phagocytes Are Dispensable for Cardiac Remodeling in Established Pressure-Overload Heart Failure
inferredcites →https://doi.org/10.15252/emmm.201403848openalex-snapshot
CaM Kinase II mediates maladaptive post‐infarct remodeling and pro‐inflammatory chemoattractant signaling but not acute myocardial ischemia/reperfusion injury
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4636708openalex-extracted
Loss Of Beta2-Adrenoceptor Triggers Ca v1.2-CaNKII-Dependent Inflammation and Fibrosis in the Heart
inferred← citeshttps://doi.org/10.2139/ssrn.4766307openalex-extracted
Sweroside Alleviates Pressure Overload-Induced Heart Failure Through Targeting Camkiiδ to Inhibit Ros-Mediated Nf-Κb/Nlrp3 in Cardiomyocytes
inferred← citeshttps://doi.org/10.1002/advs.202502395openalex-snapshot
Calhm6 Governs Macrophage Polarization Through Chp1‐Camk4‐Creb1 Axis and Ectosomal Delivery in Inflammatory Responses
inferred← citeshttps://doi.org/10.1002/advs.76370openalex-snapshot
Targeting NSUN2‐Mediated m 5 C Modification Attenuates Chondrocyte Senescence and NLRP3 Activation in Osteoarthritis
inferred← citeshttps://doi.org/10.1002/cbf.3882openalex-snapshot
Crosstalk of NLRP3 inflammasome and noncoding RNAs in cardiomyopathies
inferred← citeshttps://doi.org/10.1002/ctm2.13openalex-snapshot
NLRP3 inflammasome, an immune‐inflammatory target in pathogenesis and treatment of cardiovascular diseases
inferred← citeshttps://doi.org/10.1002/ddr.70283openalex-snapshot
Targeting the NLRP3 Inflammasome: Novel Inhibitors for Cardiovascular Diseases Management — A Narrative Review
inferred← citeshttps://doi.org/10.1002/ehf2.14772openalex-snapshot
Ca2+/Calmodulin-Dependent Kinase IIδC-Induced Chronic Heart Failure Does Not Depend on Sarcoplasmic Reticulum Ca2+ Leak
inferred← citeshttps://doi.org/10.1002/ejhf.3703openalex-snapshot
The Crosstalk Between Immune Activation and Metabolism in Heart Failure. A Scientific Statement of the Heart Failure Association of the ESC
inferred← citeshttps://doi.org/10.1002/jcp.30285openalex-snapshot
The NLRP3 inflammasome: Multiple activation pathways and its role in primary cells during ventricular remodeling
inferred← citeshttps://doi.org/10.1002/jcsm.12763openalex-snapshot
Inhibition of the NLRP3/IL‐1β axis protects against sepsis‐induced cardiomyopathy
inferred← citeshttps://doi.org/10.1002/kjm2.70126openalex-snapshot
MiR ‐145‐5p Attenuates Doxorubicin‐Induced Heart Injury Through Targeting Cardiomyocyte Pyroptosis
inferred← citeshttps://doi.org/10.1002/mco2.418openalex-snapshot
The pathogenesis and potential therapeutic targets in sepsis
inferred← citeshttps://doi.org/10.1002/ptr.70011openalex-snapshot
Berberine Ameliorates Atrial Remodeling and Inhibits the Atrial Fibrillation of Mice via Regulating NLRP3 Inflammasome
inferred← citeshttps://doi.org/10.1002/ptr.7701openalex-snapshot
Salidroside ameliorates pathological cardiac hypertrophy via TLR4‐TAK1 ‐dependent signaling
inferred← citeshttps://doi.org/10.1007/978-3-030-78799-8_7openalex-snapshot
Fibroblasts, Fibrosis and Autophagy
inferred← citeshttps://doi.org/10.1007/978-3-031-08309-9_9openalex-snapshot
Cardiac Inflammasome and Arrhythmia
inferred← citeshttps://doi.org/10.1007/978-3-031-33588-4_22openalex-snapshot
Calmodulin Kinase II Regulation of Heart Rhythm and Disease
inferred← citeshttps://doi.org/10.1007/978-3-031-97517-2_7openalex-snapshot
Optimizing Cardiovascular Safety: Cardiotoxicity Prevention in Tyrosine Kinase Inhibitor, Monoclonal Antibody, Proteasome Inhibitor, and 5-Fluorouracil Anticancer Therapies
inferred← citeshttps://doi.org/10.1007/s00109-020-02002-wopenalex-snapshot
Critical roles of macrophages in pressure overload-induced cardiac remodeling
inferred← citeshttps://doi.org/10.1007/s00210-020-01979-yopenalex-snapshot
Cardioprotective effect of pioglitazone and curcumin against diabetic cardiomyopathy in type 1 diabetes mellitus: impact on CaMKII/NF-κB/TGF-β1 and PPAR-γ signaling pathway
inferred← citeshttps://doi.org/10.1007/s00395-025-01114-zopenalex-snapshot
CD163+ macrophages attenuate pressure overload-induced left ventricular systolic dysfunction and cardiac mitochondrial dysfunction via interleukin-10
inferred← citeshttps://doi.org/10.1007/s00424-021-02515-4openalex-snapshot
The crosstalk between cardiomyocyte calcium and inflammasome signaling pathways in atrial fibrillation
inferred← citeshttps://doi.org/10.1007/s00424-024-02965-6openalex-snapshot
Cardiac hypertrophy that affects hyperthyroidism occurs independently of the NLRP3 inflammasome
inferred← citeshttps://doi.org/10.1007/s10072-025-08040-xopenalex-snapshot
Beyond the beats: a systematic review of the underlying inflammatory pathways between atrial fibrillation and cognitive decline
inferred← citeshttps://doi.org/10.1007/s10517-021-05216-1openalex-snapshot
Activation of Ca2+/Calmodulin-Dependent Protein Kinase II (CaMKII) with Lidocaine Provokes Pyroptosis of Glioblastoma Cells
inferred← citeshttps://doi.org/10.1007/s10557-020-07071-0openalex-snapshot
Inflammatory Cytokines and Chemokines as Therapeutic Targets in Heart Failure
inferred← citeshttps://doi.org/10.1007/s10735-024-10264-8openalex-snapshot
Bleomycin induces endothelial cell pyroptosis and regulates fibrosis by activating the NLRP3/caspase-1/GSDMD pathway: a possible mechanism contributing to the sclerotherapy of venous malformations
inferred← citeshttps://doi.org/10.1007/s10741-020-09916-0openalex-snapshot
High-density lipoprotein-mediated cardioprotection in heart failure
inferred← citeshttps://doi.org/10.1007/s10787-022-01086-9openalex-snapshot
Cellular mechanisms and molecular pathways linking bitter taste receptor signalling to cardiac inflammation, oxidative stress, arrhythmia and contractile dysfunction in heart diseases
inferred← citeshttps://doi.org/10.1007/s11010-022-04602-1openalex-snapshot
The role of Nod-like receptor protein 3 inflammasome activated by ion channels in multiple diseases
inferred← citeshttps://doi.org/10.1007/s11010-023-04812-1openalex-snapshot
Mitochondria and NLRP3 inflammasome in cardiac hypertrophy
inferred← citeshttps://doi.org/10.1007/s11033-026-12556-6openalex-snapshot
MicroRNA-223-3p/NLRP3 axis attenuates LPS-induced sepsis-like myocardial dysfunction by suppressing cardiomyocyte pyroptosis
inferred← citeshttps://doi.org/10.1007/s11427-025-2939-1openalex-snapshot
Danhong Injection targets CaMKII through Dihydrotanshinone I to alleviate cardiomyocyte death and inflammation in viral myocarditis
inferred← citeshttps://doi.org/10.1007/s11864-026-01402-3openalex-snapshot
Cardiotoxicity of Targeted Therapies in Hematologic Malignancies: From Molecular Mechanisms to Clinical Management
inferred← citeshttps://doi.org/10.1007/s11886-023-01854-1openalex-snapshot
Immune Cells in Cardiac Injury Repair and Remodeling
inferred← citeshttps://doi.org/10.1007/s11936-024-01067-xopenalex-snapshot
Clinical Relevance and Mechanistic Underpinnings of Tyrosine Kinase Inhibitor Associated Cardiotoxicities
inferred← citeshttps://doi.org/10.1007/s12265-023-10445-yopenalex-snapshot
Unraveling the Molecular Mechanisms of Activated Protein C (APC) in Mitigating Reperfusion Injury and Cardiac Ischemia: a Promising Avenue for Novel Therapeutic Interventions
inferred← citeshttps://doi.org/10.1007/s12272-019-01199-wopenalex-snapshot
RETRACTED ARTICLE: MiR-338-5p ameliorates pathological cardiac hypertrophy by targeting CAMKIIδ
inferred← citeshttps://doi.org/10.1007/s13577-026-01425-wopenalex-snapshot
Exploring the role of CaMKII in exercise-induced cardioprotection
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CaMKII at the Crossroads of Cardiac Ageing: A Central Integrator of Molecular Stress Signaling in Age-Related Cardiac Disease
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The inflammasome in cardiovascular diseases
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Blocking the NLRP3 inflammasome reduces osteogenic calcification and M1 macrophage polarization in a mouse model of calcified aortic valve stenosis
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Anti-inflammatory therapies were associated with reduced risk of myocardial infarction in patients with established cardiovascular disease or high cardiovascular risks: A systematic review and meta-analysis of randomized controlled trials
inferred← citeshttps://doi.org/10.1016/j.bbapap.2021.140622openalex-snapshot
Pathological cardiac remodeling seen by the eyes of proteomics
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Paeonol protects against acute pancreatitis by inhibiting M1 macrophage polarization via the NLRP3 inflammasomes pathway
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FGF5 protects heart from sepsis injury by attenuating cardiomyocyte pyroptosis through inhibiting CaMKII/NFκB signaling
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Astragaloside IV alleviates LPS-induced cardiomyocyte hypertrophy and collagen expression associated with CCL2-mediated activation of NF-κB signaling pathway
inferred← citeshttps://doi.org/10.1016/j.bcp.2024.116072openalex-snapshot
IL-23p19 deficiency reduces M1 macrophage polarization and improves stress-induced cardiac remodeling by alleviating macrophage ferroptosis in mice
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Ammonium tetrathiomolybdate attenuates acetaminophen-induced acute liver failure by inhibiting the TRPV4/Calcium/NF-κB signaling pathway
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Epigenetic regulation of extracellular matrix mechanotransduction in cardiac fibrosis
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Hypertensive vascular and cardiac remodeling protection by allicin in spontaneous hypertension rats via CaMK Ⅱ/NF-κB pathway
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Regulatory mechanism of CaMKII δ mediated by RIPK3 on myocardial fibrosis and reversal effects of RIPK3 inhibitor GSK'872
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Advances in cardiac cell interaction studies: Implications for heart disease treatment
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The Immune System as a Proarrhythmic Mediator: A Novel Paradigm for the Development of New Antiarrhythmic Therapy
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The inflammasome in heart failure
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Chemokines in cardiac fibrosis
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The heart under pressure: immune cells in fibrotic remodeling
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Diminished arachidonate 5-lipoxygenase perturbs phase separation and transcriptional response of Runx2 to reverse pathological ventricular remodeling
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Angiopoietins, vascular endothelial growth factors and secretory phospholipase A2 in heart failure patients with preserved ejection fraction
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NLRX1 knockout aggravates lipopolysaccharide (LPS)-induced heart injury and attenuates the anti-LPS cardioprotective effect of CYP2J2/11,12-EET by enhancing activation of NF-κB and NLRP3 inflammasome
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Maresin 1 attenuates myocardium ischemia/reperfusion injury via SIRT1/HMGB-1/NLRP-3-related mechanisms
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Research advances in protein prenylation in cardiovascular diseases
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Oil mist particulate matter induces myocardial tissue injury by impairing fatty acid metabolism and mitochondrial bioenergetics function via inhibiting the PPAR alpha signaling pathway in rats
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Loss of Camk2n1 aggravates cardiac remodeling and malignant ventricular arrhythmia after myocardial infarction in mice via NLRP3 inflammasome activation
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Myeloid specific knockout of Piezo1 alleviates ang Ⅱ-induced cardiac remodeling
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Dexmedetomidine alleviates myocardial ischemia/reperfusion-induced injury and Ca2+ overload via the microRNA-346-3p/CaMKIId axis
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Kirenol attenuates pressure overload-induced heart failure by enhancing autophagy in macrophages
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Selective Inhibition of NLRP3 Inflammasome Reverses Pressure Overload-Induced Pathological Cardiac Remodeling by Attenuating Hypertrophy, Fibrosis, and Inflammation
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GTS-21 alleviates sepsis-induced atrial fibrillation susceptibility by modulating macrophage polarization and Neuregulin-1 secretion
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Neuroepithelial cell-transforming 1 promotes cardiac fibrosis via the Wnt/β-catenin signaling pathway
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Treatment Targets for Right Ventricular Dysfunction in Pulmonary Arterial Hypertension
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Absence of NLRP3 Inflammasome in Hematopoietic Cells Reduces Adverse Remodeling After Experimental Myocardial Infarction
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Targeting the Inflammasome in Cardiovascular Disease
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Immunomodulation of Myocardial Fibrosis
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TAK1 Activation by NLRP3 Deficiency Confers Cardioprotection Against Pressure Overload-Induced Cardiomyocyte Pyroptosis and Hypertrophy
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Targeting the Gut-Heart Axis Improves Cardiac Remodeling in a Clinical Scale Model of Cardiometabolic Syndrome
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Periodontitis Accelerates Progression of Heart Failure With Preserved Ejection Fraction in Mice
inferred← citeshttps://doi.org/10.1016/j.jacc.2022.03.379openalex-snapshot
Inflammasome Signaling in Atrial Fibrillation
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Oxidative stress–induced autonomous activation of the calcium/calmodulin-dependent kinase II involves disulfide formation in the regulatory domain
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Jiedu Quyu Decoction mitigates monocrotaline-induced right-sided heart failure associated with pulmonary artery hypertension by inhibiting NLRP3 inflammasome in rats
inferred← citeshttps://doi.org/10.1016/j.jep.2024.118820openalex-snapshot
Qian Yang Yu Yin Granule prevents hypertensive cardiac remodeling by inhibiting NLRP3 inflammasome activation via Nrf2
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Identification of anti-inflammatory and anti-cancer compounds targeting the NF-κB-NLRP3 inflammasome pathway from a phytochemical library of the Sideritis genus
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Combining metabolomics and network pharmacology to clarify the mechanism of Angelica sinensis-Sophora flavescens pills in treating atopic dermatitis through the sweet-bitter harmonization theory
inferred← citeshttps://doi.org/10.1016/j.jep.2026.122447openalex-snapshot
Fuling Xingren Gancao decoction ameliorates myocardial ischemia/reperfusion injury by attenuating calcium overload and modulating inflammatory signaling mediated by calcium/calmodulin-dependent protein kinase II delta
inferred← citeshttps://doi.org/10.1016/j.jlr.2024.100679openalex-snapshot
Resolvin D2/GPR 18 axis ameliorates pressure overload-induced heart failure by inhibiting pro-inflammatory macrophage polarization
inferred← citeshttps://doi.org/10.1016/j.lfs.2023.122185openalex-snapshot
NLRP3-inflammasome inhibition by MCC950 attenuates cardiac and pulmonary artery remodelling in heart failure with preserved ejection fraction
inferred← citeshttps://doi.org/10.1016/j.lfs.2026.124281openalex-snapshot
Oxytocin ameliorates cardiac hypertrophy by inhibiting mitochondrial dysfunction and pyroptosis via AMPK/PGC-1α /TFAM pathway
inferred← citeshttps://doi.org/10.1016/j.molimm.2025.05.022openalex-snapshot
SAA1 deletion ameliorates cardiac injury after myocardial infarction by promoting macrophage transformation to reparative subtype
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2021.108053openalex-snapshot
Targeting the NLRP3 inflammasome in cardiovascular diseases
inferred← citeshttps://doi.org/10.1016/j.phrs.2024.107077openalex-snapshot
Drug-induced atrial fibrillation. A narrative review of a forgotten adverse effect
inferred← citeshttps://doi.org/10.1016/j.phrs.2025.107744openalex-snapshot
Modulation of GLP-1 signalling as an innovative strategy counteracting the onset of heart failure: Potential for natural compound supplementation
inferred← citeshttps://doi.org/10.1016/j.phymed.2022.154603openalex-snapshot
Guhong injection mitigates myocardial ischemia/reperfusion injury by activating GST P to inhibit ASK1-JNK/p38 pathway
inferred← citeshttps://doi.org/10.1016/j.phymed.2024.156326openalex-snapshot
Downregulation of cardiac inflammation via the CaMKII δ/NF-κB pathway in heart failure by Lonicerae Japonicae Flos and Angelicae Sinensis Radix
inferred← citeshttps://doi.org/10.1016/j.phymed.2025.157156openalex-snapshot
The total xanthones from Gentianella acuta alleviate acute myocardial infarction by targeting BRD4-mediated cardiomyocyte pyroptosis and inflammation
inferred← citeshttps://doi.org/10.1016/j.phymed.2025.157625openalex-snapshot
Chlorogenic acid targets STING to alleviate pressure overload-induced heart failure and myocardial fibrosis
inferred← citeshttps://doi.org/10.1016/j.redox.2023.102937openalex-snapshot
Cardiomyocyte NOX4 regulates resident macrophage-mediated inflammation and diastolic dysfunction in stress cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.redox.2024.103223openalex-snapshot
Sweroside alleviates pressure overload-induced heart failure through targeting CaMKⅡδ to inhibit ROS-mediated NF-κB/NLRP3 in cardiomyocytes
inferred← citeshttps://doi.org/10.1016/j.redox.2024.103471openalex-snapshot
Cardiomyocyte-specific Piezo1 deficiency mitigates ischemia-reperfusion injury by preserving mitochondrial homeostasis
inferred← citeshttps://doi.org/10.1016/j.scitotenv.2022.155733openalex-snapshot
Silica nanoparticles induce cardiac injury and dysfunction via ROS/Ca2+/CaMKII signaling
inferred← citeshttps://doi.org/10.1016/j.tem.2021.07.006openalex-snapshot
Ketone bodies for the failing heart: fuels that can fix the engine?
inferred← citeshttps://doi.org/10.1016/j.trsl.2022.08.004openalex-snapshot
NLRP3-mediated inflammation in cardio-oncology: sterile yet harmful
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2020.07.008openalex-snapshot
Systolic overload-induced pulmonary inflammation, fibrosis, oxidative stress and heart failure progression through interleukin-1β
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2020.09.007openalex-snapshot
CaMKII exacerbates heart failure progression by activating class I HDACs
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2022.03.012openalex-snapshot
Cardio-oncology imaging tools at the translational interface
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2022.07.005openalex-snapshot
Current knowledge of pyroptosis in heart diseases
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2023.10.003openalex-snapshot
CaMKII, ‘jack of all trades’ in inflammation during cardiac ischemia/reperfusion injury
inferred← citeshttps://doi.org/10.1021/acsbiomaterials.5c02094openalex-snapshot
Tailorable Metal–Polyphenol Nanoparticles for Targeted Treatment of Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1038/nrcardio.2017.161openalex-snapshot
The NLRP3 inflammasome in acute myocardial infarction
inferred← citeshttps://doi.org/10.1038/s41390-025-04081-2openalex-snapshot
The role of NLRP3 inflammasome in necrotizing enterocolitis
inferred← citeshttps://doi.org/10.1038/s41392-021-00650-zopenalex-snapshot
Inflammasomes as therapeutic targets in human diseases
inferred← citeshttps://doi.org/10.1038/s41392-023-01687-yopenalex-snapshot
The role of inflammasomes in human diseases and their potential as therapeutic targets
inferred← citeshttps://doi.org/10.1038/s41392-024-01840-1openalex-snapshot
Macrophages in cardiovascular diseases: molecular mechanisms and therapeutic targets
inferred← citeshttps://doi.org/10.1038/s41392-024-01962-6openalex-snapshot
Characterization of ferroptosis-triggered pyroptotic signaling in heart failure
inferred← citeshttps://doi.org/10.1038/s41401-020-00587-zopenalex-snapshot
6-Gingerol protects against cardiac remodeling by inhibiting the p38 mitogen-activated protein kinase pathway
inferred← citeshttps://doi.org/10.1038/s41418-019-0286-9openalex-snapshot
Autophagy and cardiac aging
inferred← citeshttps://doi.org/10.1038/s41419-020-02777-3openalex-snapshot
Pyroptosis and ferroptosis induced by mixed lineage kinase 3 (MLK3) signaling in cardiomyocytes are essential for myocardial fibrosis in response to pressure overload
inferred← citeshttps://doi.org/10.1038/s41419-023-05960-4openalex-snapshot
Macrophage DCLK1 promotes obesity-induced cardiomyopathy via activating RIP2/TAK1 signaling pathway
inferred← citeshttps://doi.org/10.1038/s41419-024-06805-4openalex-snapshot
TNIP3 protects against pathological cardiac hypertrophy by stabilizing STAT1
inferred← citeshttps://doi.org/10.1038/s41420-022-00967-wopenalex-snapshot
ATP6AP2 knockdown in cardiomyocyte deteriorates heart function via compromising autophagic flux and NLRP3 inflammasome activation
inferred← citeshttps://doi.org/10.1038/s41420-024-01840-8openalex-snapshot
IP3R2-mediated Ca2+ release promotes LPS-induced cardiomyocyte pyroptosis via the activation of NLRP3/Caspase-1/GSDMD pathway
inferred← citeshttps://doi.org/10.1038/s41467-021-23549-3openalex-snapshot
CaMKII oxidation is a critical performance/disease trade-off acquired at the dawn of vertebrate evolution
inferred← citeshttps://doi.org/10.1038/s41467-026-73125-wopenalex-snapshot
LTBP4 deficiency inhibits NLRP3 inflammasome activation in cardiomyocytes and attenuates heart failure in male mice
inferred← citeshttps://doi.org/10.1038/s41569-020-00480-6openalex-snapshot
Cellular and molecular pathobiology of heart failure with preserved ejection fraction
inferred← citeshttps://doi.org/10.1038/s41569-022-00759-wopenalex-snapshot
Inflammatory signalling in atrial cardiomyocytes: a novel unifying principle in atrial fibrillation pathophysiology
inferred← citeshttps://doi.org/10.1038/s41569-023-00946-3openalex-snapshot
The role of the NLRP3 inflammasome and pyroptosis in cardiovascular diseases
inferred← citeshttps://doi.org/10.1038/s41598-021-83022-5openalex-snapshot
CXCR7 ameliorates myocardial infarction as a β-arrestin-biased receptor
inferred← citeshttps://doi.org/10.1038/s41598-024-55996-5openalex-snapshot
HFpEF as systemic disease, insight from a diagnostic prediction model reminiscent of systemic inflammation and organ interaction in HFpEF patients
inferred← citeshttps://doi.org/10.1038/s41598-025-28808-7openalex-snapshot
Protective role of the C-reactive protein-albumin-lymphocyte (CALLY) index for cardiovascular risk and mortality
inferred← citeshttps://doi.org/10.1038/s42003-026-10207-xopenalex-snapshot
Sensing the spectrum: diverse stimuli for the NLRP3 inflammasome
inferred← citeshttps://doi.org/10.1038/s44321-024-00132-zopenalex-snapshot
An intrinsic mechanism of metabolic tuning promotes cardiac resilience to stress
inferred← citeshttps://doi.org/10.1039/9781837675739-00202openalex-snapshot
Immunomodulatory Biomaterials for Cardiac Repair
inferred← citeshttps://doi.org/10.1080/00015385.2025.2515322openalex-snapshot
NLR/inflammasome pathway in cardiovascular disorders: from pathogenesis to potential therapies
inferred← citeshttps://doi.org/10.1080/0886022x.2024.2354444openalex-snapshot
Bushen Huoxue formula protects against renal fibrosis and pyroptosis in chronic kidney disease by inhibiting ROS/NLRP3-mediated inflammasome activation
inferred← citeshttps://doi.org/10.1080/0886022x.2024.2387432openalex-snapshot
Verapamil inhibits inflammation and promotes autophagy to alleviate ureteral scar by regulation of CaMK IIδ/STAT3 axis
inferred← citeshttps://doi.org/10.1080/10641963.2021.1996588openalex-snapshot
AT1 receptor blocker inhibits HMGB1 expression in pressure overload-induced acute cardiac dysfunction by suppressing the MAPK/NF-κB signaling pathway
inferred← citeshttps://doi.org/10.1080/10641963.2023.2189138openalex-snapshot
GSDMD-mediated pyroptosis promotes cardiac remodeling in pressure overload
inferred← citeshttps://doi.org/10.1080/13880209.2023.2224403openalex-snapshot
Punicalagin attenuates ventricular remodeling after acute myocardial infarction via regulating the NLRP3/caspase-1 pathway
inferred← citeshttps://doi.org/10.1080/21655979.2022.2053813openalex-snapshot
MCC950 ameliorates ventricular arrhythmia vulnerability induced by heart failure
inferred← citeshttps://doi.org/10.1080/21655979.2022.2054913openalex-snapshot
The crystallin alpha B (HSPB5)-tripartite motif containing 33 (TRIM33) axis mediates myocardial fibrosis induced by angiotensinogen II through transforming growth factor-β (TGF-β1)-Smad3/4 signaling
inferred← citeshttps://doi.org/10.1080/27694127.2024.2320605openalex-snapshot
Crosstalk between myocardial autophagy and sterile inflammation in the development of heart failure
inferred← citeshttps://doi.org/10.1084/jem.20192378openalex-snapshot
Spatiotemporal restriction of endothelial cell calcium signaling is required during leukocyte transmigration
inferred← citeshttps://doi.org/10.1084/jem.20211098openalex-snapshot
Macrophage-adipocyte communication and cardiac remodeling
inferred← citeshttps://doi.org/10.1089/ars.2020.8138openalex-snapshot
Persulfide Signaling in Stress-Initiated Calmodulin Kinase Response
inferred← citeshttps://doi.org/10.1093/ckj/sfad135openalex-snapshot
Leukocyte–endothelial interaction in CKD
inferred← citeshttps://doi.org/10.1093/cvr/cvab192openalex-snapshot
Non-coding RNAs in cardiac inflammation: key drivers in the pathophysiology of heart failure
inferred← citeshttps://doi.org/10.1093/cvr/cvab202openalex-snapshot
AIM2-driven inflammasome activation in heart failure
inferred← citeshttps://doi.org/10.1093/cvr/cvae045openalex-snapshot
Large animal models of pressure overload-induced cardiac left ventricular hypertrophy to study remodelling of the human heart with aortic stenosis
inferred← citeshttps://doi.org/10.1093/eschf/xvaf041openalex-snapshot
Arterio-venous gradient of active interleukin-18 is associated with diastolic dysfunction: a cross-sectional study
inferred← citeshttps://doi.org/10.1093/infdis/jiaa328openalex-snapshot
Human Immunodeficiency Virus–Associated Myocardial Diastolic Dysfunction and Soluble ST2 Concentration in Tanzanian Adults: A Cross-Sectional Study
inferred← citeshttps://doi.org/10.1096/fj.201902692ropenalex-snapshot
Melatonin alleviates cardiac fibrosis via inhibiting lncRNA MALAT1/miR‐141‐mediated NLRP3 inflammasome and TGF‐β1/Smads signaling in diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1096/fj.202502632ropenalex-snapshot
E3 Ubiquitin Ligase TRIM21 Exacerbates Pathological Cardiac Hypertrophy Through ASK1 K63‐Linked Polyubiquitination
inferred← citeshttps://doi.org/10.1097/cp9.0000000000000035openalex-snapshot
Inflammation macrophages contribute to cardiac homeostasis
inferred← citeshttps://doi.org/10.1097/fjc.0000000000000717openalex-snapshot
NLRP3 Inflammasome in Acute Myocardial Infarction
inferred← citeshttps://doi.org/10.1097/fjc.0000000000000858openalex-snapshot
Insight into the Pro-inflammatory and Profibrotic Role of Macrophage in Heart Failure With Preserved Ejection Fraction
inferred← citeshttps://doi.org/10.1097/fjc.0000000000001757openalex-snapshot
Omaveloxolone Promotes Macrophage M2 Polarization by Activating the NRF2–Keap1 Pathway and Improves Myocardial Remodeling Induced by Pressure Overload
inferred← citeshttps://doi.org/10.1097/md.0000000000035958openalex-snapshot
A bibliometric analysis of cardiomyocyte apoptosis from 2014 to 2023: A review
inferred← citeshttps://doi.org/10.1101/2025.06.16.659963openalex-snapshot
Multimodal Analysis of Sepsis-induced Cardiomyopathy in a Baboon Model
inferred← citeshttps://doi.org/10.1111/apha.13964openalex-snapshot
High‐phosphate diet causes atrial remodeling and increases atrial fibrillation vulnerability via STAT3/NF‐κB signaling and oxidative stress
inferred← citeshttps://doi.org/10.1111/apha.13978openalex-snapshot
Increases in dietary phosphate levels can augment atrial arrhythmias
inferred← citeshttps://doi.org/10.1111/apha.70280openalex-snapshot
P2X7 ‐ NEK7 ‐ NLRP3 Axis Drives Cardiac Inflammation and Fibrosis in Isoproterenol‐Induced Cardiac Remodeling in Mice
inferred← citeshttps://doi.org/10.1111/bph.16001openalex-snapshot
Drug repurposing for cardiovascular diseases: New targets and indications for probenecid
inferred← citeshttps://doi.org/10.1111/bph.70481openalex-snapshot
Drugs that act on both G protein‐coupled receptors (GPCRs) and kinases: potentiation of effects, side effects and general aspects of drug pleiotropy
inferred← citeshttps://doi.org/10.1111/cns.70590openalex-snapshot
Targeting Drp1 in Cerebral Ischemia–Reperfusion Injury: Mechanisms and Therapeutic Implications
inferred← citeshttps://doi.org/10.1111/jcmm.15955openalex-snapshot
CXADR‐like membrane protein protects against heart injury by preventing excessive pyroptosis after myocardial infarction
inferred← citeshttps://doi.org/10.1111/jcmm.17081openalex-snapshot
CaMKII inhibitor KN‐93 impaired angiogenesis and aggravated cardiac remodelling and heart failure via inhibiting NOX2/mtROS/p‐VEGFR2 and STAT3 pathways
inferred← citeshttps://doi.org/10.1111/sji.70020openalex-snapshot
Inflammasomes and Cardiovascular Disease: Linking Inflammation to Cardiovascular Pathophysiology
inferred← citeshttps://doi.org/10.1113/ep088390openalex-snapshot
Knockout of MD1 contributes to sympathetic hyperactivity and exacerbates ventricular arrhythmias following heart failure with preserved ejection fraction via NLRP3 inflammasome activation
inferred← citeshttps://doi.org/10.1113/jp285941openalex-snapshot
CaMKII at the crossroads: calcium dysregulation, and post‐translational modifications driving cell death
inferred← citeshttps://doi.org/10.1113/jp290180openalex-snapshot
Translating cardiovascular ion channel and Ca 2+ signalling mechanisms into therapeutic insights
inferred← citeshttps://doi.org/10.1136/bmjdrc-2024-004478openalex-snapshot
Association between RDW-SD and prognosis across glycemic status in patients with dilated cardiomyopathy
inferred← citeshttps://doi.org/10.1142/s0192415x25500697openalex-snapshot
Poria cocos Polysaccharides Attenuate Cardiac Injury by Inhibiting CaMKII-Mediated P38/NF-κB/NLRP3 Signaling Pathway to Reduce Sepsis-Induced Apoptosis and Inflammation
inferred← citeshttps://doi.org/10.1146/annurev-pharmtox-051421-111814openalex-snapshot
CaMKII as a Therapeutic Target in Cardiovascular Disease
inferred← citeshttps://doi.org/10.1152/ajpcell.00421.2022openalex-snapshot
Red light-green light: T-cell trafficking in cardiac and vascular inflammation
inferred← citeshttps://doi.org/10.1152/ajpcell.00924.2024openalex-snapshot
Activation of Smo/Gli1 pathway attenuates cardiac fibrosis by suppressing G1/S phase transition and cell proliferation in cardiac fibroblasts in mice
inferred← citeshttps://doi.org/10.1152/ajpheart.00097.2020openalex-snapshot
Cytosolic DNA sensor cGAS plays an essential pathogenetic role in pressure overload-induced heart failure
inferred← citeshttps://doi.org/10.1152/ajpheart.00223.2019openalex-snapshot
Inflammation in nonischemic heart disease: initiation by cardiomyocyte CaMKII and NLRP3 inflammasome signaling
inferred← citeshttps://doi.org/10.1152/ajpheart.00589.2025openalex-snapshot
Cardiomyocyte β-arrestins mediate inflammation and cGAS-STING activation in CVB3 viral myocarditis
inferred← citeshttps://doi.org/10.1152/physrev.00022.2018openalex-snapshot
Fundamental Mechanisms of Regulated Cell Death and Implications for Heart Disease
inferred← citeshttps://doi.org/10.1155/2020/4293206openalex-snapshot
NLRP3 Inflammasome and Its Central Role in the Cardiovascular Diseases
inferred← citeshttps://doi.org/10.1155/2020/7956274openalex-snapshot
Inhibition of P2X7 Purinergic Receptor Ameliorates Cardiac Fibrosis by Suppressing NLRP3/IL-1β Pathway
inferred← citeshttps://doi.org/10.1155/2022/6235747openalex-snapshot
Mitochondrial DNA Is a Vital Driving Force in Ischemia‐Reperfusion Injury in Cardiovascular Diseases
inferred← citeshttps://doi.org/10.1155/2022/7250578openalex-snapshot
A Novel Inhibitor INF 39 Promotes Osteogenesis via Blocking the NLRP3/IL‐1β Axis
inferred← citeshttps://doi.org/10.1155/2022/8593176openalex-snapshot
Exploring the Mechanism of Ling-Gui-Zhu-Gan Decoction in Ventricular Remodeling after Acute Myocardial Infarction Based on UPLC and In Vivo Experiments
inferred← citeshttps://doi.org/10.1155/2022/9205908openalex-snapshot
Traditional Chinese Medicine Ginseng Dingzhi Decoction Ameliorates Myocardial Fibrosis and High Glucose‐Induced Cardiomyocyte Injury by Regulating Intestinal Flora and Mitochondrial Dysfunction
inferred← citeshttps://doi.org/10.1155/mi/4330574openalex-snapshot
NLRP3 Inflammasome: A New Target for the Treatment of CVD and Depression Comorbidity
inferred← citeshttps://doi.org/10.1158/2326-6066.cir-23-0903openalex-snapshot
A Bifunctional Antibody Targeting PD-1 and TGF-β Signaling Has Antitumor Activity in Combination with Radiotherapy and Attenuates Radiation-Induced Lung Injury
inferred← citeshttps://doi.org/10.1161/circheartfailure.119.006573openalex-snapshot
Chronically Elevating Circulating Ketones Can Reduce Cardiac Inflammation and Blunt the Development of Heart Failure
inferred← citeshttps://doi.org/10.1161/circresaha.119.311148openalex-snapshot
The Extracellular Matrix in Ischemic and Nonischemic Heart Failure
inferred← citeshttps://doi.org/10.1161/circresaha.119.312321openalex-snapshot
Obesity, Hypertension, and Cardiac Dysfunction
inferred← citeshttps://doi.org/10.1161/circresaha.119.316288openalex-snapshot
Hyperglycemia Acutely Increases Cytosolic Reactive Oxygen Species via O -linked GlcNAcylation and CaMKII Activation in Mouse Ventricular Myocytes
inferred← citeshttps://doi.org/10.1161/circresaha.120.316363openalex-snapshot
Molecular Basis of Atrial Fibrillation Pathophysiology and Therapy
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