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Sweroside Alleviates Pressure Overload-Induced Heart Failure Through Targeting Camkiiδ to Inhibit Ros-Mediated Nf-Κb/Nlrp3 in Cardiomyocytes

https://doi.org/10.2139/ssrn.4766307
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The Innate Immune System in Chronic Cardiomyopathy: A European Society of Cardiology (Esc) Scientific Statement from the Working Group on Myocardial Function of the ESC
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AutoDock Vina: Improving the speed and accuracy of docking with a new scoring function, efficient optimization, and multithreading
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Inflammation and fibrosis in murine models of heart failure
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Various aspects of inflammation in heart failure
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Anti-inflammatory Effects of Sweroside on LPS-Induced ALI in Mice Via Activating SIRT1
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Captopril attenuates TAC-induced heart failure via inhibiting Wnt3a/β-catenin and Jak2/Stat3 pathways
inferredcites →https://doi.org/10.1016/j.biopha.2022.113075openalex-extracted
Corynoline protects ang II-induced hypertensive heart failure by increasing PPARα and Inhibiting NF-κB pathway
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Markers of fibrosis, inflammation, and remodeling pathways in heart failure
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CaMKIIδ interacts directly with IKKβ and modulates NF-κB signalling in adult cardiac fibroblasts
inferredcites →https://doi.org/10.1016/j.coph.2005.10.008openalex-extracted
NADPH oxidase and heart failure
inferredcites →https://doi.org/10.1016/j.ebiom.2022.103992openalex-extracted
Inflammation and resolution signaling in cardiac repair and heart failure
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Targeting the Inflammasome in Cardiovascular Disease
inferredcites →https://doi.org/10.1016/j.jacc.2010.11.022openalex-extracted
The Primary Benefits of Angiotensin-Converting Enzyme Inhibition on Cardiac Remodeling Occur During Sleep Time in Murine Pressure Overload Hypertrophy
inferredcites →https://doi.org/10.1016/j.jacc.2018.02.021openalex-extracted
Myocardial Interstitial Fibrosis in Heart Failure
inferredcites →https://doi.org/10.1016/j.jacc.2020.03.019openalex-extracted
Myocardial Interstitial Fibrosis in Nonischemic Heart Disease, Part 3/4
inferredcites →https://doi.org/10.1016/j.jep.2021.113833openalex-extracted
Deciphering the effective combinatorial components from Si-Miao-Yong-An decoction regarding the intervention on myocardial hypertrophy
inferredcites →https://doi.org/10.1016/j.phrs.2019.104318openalex-extracted
Si-Miao-Yong-An decoction ameliorates cardiac function through restoring the equilibrium of SOD and NOX2 in heart failure mice
inferredcites →https://doi.org/10.1016/j.redox.2021.102020openalex-extracted
Lutein attenuates angiotensin II- induced cardiac remodeling by inhibiting AP-1/IL-11 signaling
inferredcites →https://doi.org/10.1016/j.redox.2022.102511openalex-extracted
A novel anti-atherosclerotic mechanism of quercetin: Competitive binding to KEAP1 via Arg483 to inhibit macrophage pyroptosis
inferredcites →https://doi.org/10.1016/j.redox.2023.102610openalex-extracted
Ginsenoside Rd promotes omentin secretion in adipose through TBK1-AMPK to improve mitochondrial biogenesis via WNT5A/Ca2+ pathways in heart failure
inferredcites →https://doi.org/10.1016/j.redox.2023.102702openalex-extracted
BNP protects against diabetic cardiomyopathy by promoting Opa1-mediated mitochondrial fusion via activating the PKG-STAT3 pathway
inferredcites →https://doi.org/10.1016/j.redox.2023.102937openalex-extracted
Cardiomyocyte NOX4 regulates resident macrophage-mediated inflammation and diastolic dysfunction in stress cardiomyopathy
inferredcites →https://doi.org/10.1016/j.yjmcc.2012.01.021openalex-extracted
MyD88 mediated inflammatory signaling leads to CaMKII oxidation, cardiac hypertrophy and death after myocardial infarction
inferredcites →https://doi.org/10.1016/j.yjmcc.2017.01.002openalex-extracted
CaMKIIδ subtypes differentially regulate infarct formation following ex vivo myocardial ischemia/reperfusion through NF-κB and TNF-α
inferredcites →https://doi.org/10.1038/s41569-019-0315-xopenalex-extracted
Reappraising the role of inflammation in heart failure
inferredcites →https://doi.org/10.1093/cvr/cvac013openalex-extracted
Global burden of heart failure: a comprehensive and updated review of epidemiology
inferredcites →https://doi.org/10.1093/eurheartj/ehac362openalex-extracted
Scleraxis and fibrosis in the pressure-overloaded heart
inferredcites →https://doi.org/10.1152/ajpheart.00223.2019openalex-extracted
Inflammation in nonischemic heart disease: initiation by cardiomyocyte CaMKII and NLRP3 inflammasome signaling
inferredcites →https://doi.org/10.1152/ajpheart.00795.2013openalex-extracted
Interleukin-18 mediates interleukin-1-induced cardiac dysfunction
inferredcites →https://doi.org/10.1155/2019/7092151openalex-extracted
Vascular Inflammation and Oxidative Stress: Major Triggers for Cardiovascular Disease
inferredcites →https://doi.org/10.1161/circresaha.112.276915openalex-extracted
Ca 2+ /Calmodulin-Dependent Protein Kinase II δ Mediates Myocardial Ischemia/Reperfusion Injury Through Nuclear Factor-κB
inferredcites →https://doi.org/10.1161/circresaha.116.304682openalex-extracted
Mitochondrial Reprogramming Induced by CaMKIIδ Mediates Hypertrophy Decompensation
inferredcites →https://doi.org/10.1161/circresaha.117.312317openalex-extracted
AMPKα2 Protects Against the Development of Heart Failure by Enhancing Mitophagy via PINK1 Phosphorylation
inferredcites →https://doi.org/10.1161/circresaha.119.315861openalex-extracted
Celastrol Attenuates Angiotensin II–Induced Cardiac Remodeling by Targeting STAT3
inferredcites →https://doi.org/10.1161/circresaha.122.322259openalex-extracted
Dectin-1 Acts as a Non-Classical Receptor of Ang II to Induce Cardiac Remodeling
inferredcites →https://doi.org/10.1161/circulationaha.118.034621openalex-extracted
Inflammation and NLRP3 Inflammasome Activation Initiated in Response to Pressure Overload by Ca 2+ /Calmodulin-Dependent Protein Kinase II δ Signaling in Cardiomyocytes Are Essential for Adverse Cardiac Remodeling
inferredcites →https://doi.org/10.1161/circulationaha.118.038010openalex-extracted
Anti-Inflammatory Therapy With Canakinumab for the Prevention of Hospitalization for Heart Failure
inferredcites →https://doi.org/10.1161/circulationaha.122.060860openalex-extracted
NLRP3 Inflammasome Activation Through Heart-Brain Interaction Initiates Cardiac Inflammation and Hypertrophy During Pressure Overload
inferredcites →https://doi.org/10.1172/jci38022openalex-extracted
Requirement for Ca2+/calmodulin–dependent kinase II in the transition from pressure overload–induced cardiac hypertrophy to heart failure in mice
inferredcites →https://doi.org/10.1177/08853282231194317openalex-extracted
Sweroside functionalized with Mesenchymal Stem cells derived exosomes attenuates sepsis-induced myocardial injury by modulating oxidative stress and apoptosis in rats
inferredcites →https://doi.org/10.2147/vhrm.s327661openalex-extracted
The Main Causes and Mechanisms of Increase in Cardiac Troponin Concentrations Other Than Acute Myocardial Infarction (Part 1): Physical Exertion, Inflammatory Heart Disease, Pulmonary Embolism, Renal Failure, Sepsis
inferredcites →https://doi.org/10.2967/jnumed.119.232488openalex-extracted
Multimodality Imaging of Inflammation and Ventricular Remodeling in Pressure-Overload Heart Failure
inferredcites →https://doi.org/10.3389/fcell.2022.809996openalex-extracted
Higenamine Attenuates Doxorubicin-Induced Cardiac Remodeling and Myocyte Apoptosis by Suppressing AMPK Activation
inferredcites →https://doi.org/10.3389/fcvm.2021.650368openalex-extracted
Sweroside Protects Against Myocardial Ischemia–Reperfusion Injury by Inhibiting Oxidative Stress and Pyroptosis Partially via Modulation of the Keap1/Nrf2 Axis
inferredcites →https://doi.org/10.3390/ijms20184374openalex-extracted
CaMKII Activity in the Inflammatory Response of Cardiac Diseases
inferredcites →https://doi.org/10.3390/ijms21082790openalex-extracted
Sweroside Prevents Non-Alcoholic Steatohepatitis by Suppressing Activation of the NLRP3 Inflammasome
inferredcites →https://doi.org/10.3390/ijms231911025openalex-extracted
Molecular Mechanisms Underlying Ca2+/Calmodulin-Dependent Protein Kinase Kinase Signal Transduction
inferredcites →https://doi.org/10.3390/jcm9030892openalex-extracted
Mitophagy in Cardiovascular Diseases
inferredcites →https://doi.org/10.3791/55293openalex-extracted
Minimally Invasive Transverse Aortic Constriction in Mice
inferredcites →https://doi.org/10.3791/55293-vopenalex-extracted
Minimally Invasive Transverse Aortic Constriction in Mice
inferredcites →https://doi.org/10.7150/thno.83340openalex-extracted
OTUD1 promotes pathological cardiac remodeling and heart failure by targeting STAT3 in cardiomyocytes
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