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Alterations in Multi‐Scale Cardiac Architecture in Association With Phosphorylation of Myosin Binding Protein‐C
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
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Three‐dimensional myoarchitecture of the bovine tongue demonstrated by diffusion spectrum magnetic resonance imaging with tractography
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Sarcomere alignment is regulated by myocyte shape
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Phosphorylation switches specific for the cardiac isoform of myosin binding protein‐C: a modulator of cardiac contraction?
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Determination of three‐dimensional ventricular strain distributions in gene‐targeted mice using tagged MRI
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Cardiac Myosin-Binding Protein C (MyBP-C): Identification of Protein Kinase A and Protein Kinase C Phosphorylation Sites
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Mutations in β-myosin S2 that cause familial hypertrophic cardiomyopathy (FHC) abolish the interaction with the regulatory domain of myosin-binding protein-C 1 1Edited by J. Karn
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Regional variation in myofilament length-dependent activation
inferredcites →https://doi.org/10.1007/s00424-014-1451-0openalex-snapshot
Cardiac myosin binding protein-C: a structurally dynamic regulator of myocardial contractility
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Resolving Myoarchitectural Disarray in the Mouse Ventricular Wall with Diffusion Spectrum Magnetic Resonance Imaging
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Effect of Fiber Geometry on Pulsatile Pumping and Energy Expenditure
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Sarcomeric protein isoform transitions in cardiac muscle: A journey to heart failure
inferredcites →https://doi.org/10.1016/j.bpj.2015.03.061openalex-snapshot
Patterns of Intersecting Fiber Arrays Revealed in Whole Muscle with Generalized Q-Space Imaging
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Towards new understanding of the heart structure and function
inferredcites →https://doi.org/10.1016/j.ejcts.2006.02.064openalex-snapshot
MRI myocardial motion and fiber tracking: a confirmation of knowledge from different imaging modalities
inferredcites →https://doi.org/10.1016/j.jacc.2004.07.045openalex-snapshot
Myosin binding protein C mutations and compound heterozygosity in hypertrophic cardiomyopathy
inferredcites →https://doi.org/10.1016/j.jmb.2008.09.013openalex-snapshot
Understanding the Organisation and Role of Myosin Binding Protein C in Normal Striated Muscle by Comparison with MyBP-C Knockout Cardiac Muscle
inferredcites →https://doi.org/10.1016/j.yjmcc.2008.05.020openalex-snapshot
Myosin binding protein C phosphorylation in normal, hypertrophic and failing human heart muscle
inferredcites →https://doi.org/10.1016/j.yjmcc.2009.11.014openalex-snapshot
Phosphorylation and function of cardiac myosin binding protein-C in health and disease
inferredcites →https://doi.org/10.1016/j.yjmcc.2010.09.007openalex-snapshot
Analysis of cardiac myosin binding protein-C phosphorylation in human heart muscle
inferredcites →https://doi.org/10.1016/j.yjmcc.2012.01.006openalex-snapshot
The dynamic role of cardiac myosin binding protein-C during ischemia
inferredcites →https://doi.org/10.1016/j.yjmcc.2013.08.006openalex-snapshot
Functional dissection of myosin binding protein C phosphorylation
inferredcites →https://doi.org/10.1016/j.yjmcc.2014.11.018openalex-snapshot
Haploinsufficiency of MYBPC3 exacerbates the development of hypertrophic cardiomyopathy in heterozygous mice
inferredcites →https://doi.org/10.1016/s0006-3495(01)75764-5openalex-snapshot
Multiple Structures of Thick Filaments in Resting Cardiac Muscle and Their Influence on Cross-Bridge Interactions
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The folding and stability of titin immunoglobulin-like modules, with implications for the mechanism of elasticity
inferredcites →https://doi.org/10.1021/bi700467kopenalex-snapshot
PKCε Increases Phosphorylation of the Cardiac Myosin Binding Protein C at Serine 302 both in Vitro and in Vivo
inferredcites →https://doi.org/10.1038/ng.309openalex-snapshot
A common MYBPC3 (cardiac myosin binding protein C) variant associated with cardiomyopathies in South Asia
inferredcites →https://doi.org/10.1038/sj.cr.7290146openalex-snapshot
Hypertrophic cardiomyopathy: from gene defect to clinical disease
inferredcites →https://doi.org/10.1042/bj2490709openalex-snapshot
Phosphorylation of C-protein, troponin I and phospholamban in isolated rabbit hearts
inferredcites →https://doi.org/10.1073/pnas.0607069103openalex-snapshot
Cardiac myosin binding protein c phosphorylation is cardioprotective
inferredcites →https://doi.org/10.1073/pnas.1103216108openalex-snapshot
Direct visualization of myosin-binding protein C bridging myosin and actin filaments in intact muscle
inferredcites →https://doi.org/10.1073/pnas.93.17.8999openalex-snapshot
Alteration of myosin cross bridges by phosphorylation of myosin-binding protein C in cardiac muscle.
inferredcites →https://doi.org/10.1074/jbc.m109.066456openalex-snapshot
Distinct Sarcomeric Substrates Are Responsible for Protein Kinase D-mediated Regulation of Cardiac Myofilament Ca2+ Sensitivity and Cross-bridge Cycling
inferredcites →https://doi.org/10.1074/jbc.m110.202713openalex-snapshot
Novel Role for p90 Ribosomal S6 Kinase in the Regulation of Cardiac Myofilament Phosphorylation
inferredcites →https://doi.org/10.1074/jbc.m808850200openalex-snapshot
The Myosin-binding Protein C Motif Binds to F-actin in a Phosphorylation-sensitive Manner
inferredcites →https://doi.org/10.1074/jbc.r110.171801openalex-snapshot
Signaling and Myosin-binding Protein C
inferredcites →https://doi.org/10.1093/hmg/ddu091openalex-snapshot
Rbm20-deficient cardiogenesis reveals early disruption of RNA processing and sarcomere remodeling establishing a developmental etiology for dilated cardiomyopathy
inferredcites →https://doi.org/10.1109/tmi.2010.2045126openalex-snapshot
Generalized ${ q}$-Sampling Imaging
inferredcites →https://doi.org/10.1111/j.1540-8175.2010.01287.xopenalex-snapshot
Transmural Myocardial Strain Gradient: A New and Robust Quantitative Index of Left Ventricular Wall Motion Based on Myocardial Strain Imaging
inferredcites →https://doi.org/10.1113/jphysiol.2002.022707openalex-snapshot
Troponin I phosphorylation enhances crossbridge kinetics during β‐adrenergic stimulation in rat cardiac tissue
inferredcites →https://doi.org/10.1117/1.3046724openalex-snapshot
Multiscale structural analysis of mouse lingual myoarchitecture employing diffusion spectrum magnetic resonance imaging and multiphoton microscopy
inferredcites →https://doi.org/10.1126/sciadv.1400205openalex-snapshot
Myosin-binding protein C corrects an intrinsic inhomogeneity in cardiac excitation-contraction coupling
inferredcites →https://doi.org/10.1126/science.1223602openalex-snapshot
Molecular Mechanics of Cardiac Myosin-Binding Protein C in Native Thick Filaments
inferredcites →https://doi.org/10.1148/rg.26si065510openalex-snapshot
Understanding Diffusion MR Imaging Techniques: From Scalar Diffusion-weighted Imaging to Diffusion Tensor Imaging and Beyond
inferredcites →https://doi.org/10.1152/ajpheart.00913.2013openalex-snapshot
Contractile dysfunction in a mouse model expressing a heterozygousMYBPC3mutation associated with hypertrophic cardiomyopathy
inferredcites →https://doi.org/10.1152/ajpheart.01012.2010openalex-snapshot
Left ventricular strain, rotation, and torsion as markers of acute myocardial ischemia
inferredcites →https://doi.org/10.1161/01.cir.89.1.142openalex-snapshot
Alterations in left ventricular twist mechanics with inotropic stimulation and volume loading in human subjects.
inferredcites →https://doi.org/10.1161/01.res.0000036750.81083.83openalex-snapshot
Identification of Novel Interactions Between Domains of Myosin Binding Protein-C That Are Modulated by Hypertrophic Cardiomyopathy Missense Mutations
inferredcites →https://doi.org/10.1161/01.res.0000190605.79013.4dopenalex-snapshot
Cardiac Myosin-Binding Protein-C Phosphorylation and Cardiac Function
inferredcites →https://doi.org/10.1161/01.res.24.3.339openalex-snapshot
Fiber Orientation in the Canine Left Ventricle during Diastole and Systole
inferredcites →https://doi.org/10.1161/01.res.64.4.721openalex-snapshot
Regional left ventricular epicardial deformation in the passive dog heart.
inferredcites →https://doi.org/10.1161/01.res.77.1.182openalex-snapshot
Transverse Shear Along Myocardial Cleavage Planes Provides a Mechanism for Normal Systolic Wall Thickening
inferredcites →https://doi.org/10.1161/01.res.84.10.1117openalex-snapshot
Cardiac Myosin Binding Protein C
inferredcites →https://doi.org/10.1161/01.res.86.1.6openalex-snapshot
Myosin Binding Protein C, a Potential Regulator of Cardiac Contractility
inferredcites →https://doi.org/10.1161/circimaging.108.815050openalex-snapshot
Diffusion Spectrum MRI Tractography Reveals the Presence of a Complex Network of Residual Myofibers in Infarcted Myocardium
inferredcites →https://doi.org/10.1161/circresaha.108.177683openalex-snapshot
Acceleration of Crossbridge Kinetics by Protein Kinase A Phosphorylation of Cardiac Myosin Binding Protein C Modulates Cardiac Function
inferredcites →https://doi.org/10.1161/circresaha.111.242560openalex-snapshot
A Critical Function for Ser-282 in Cardiac Myosin Binding Protein-C Phosphorylation and Cardiac Function
inferredcites →https://doi.org/10.1161/circresaha.113.301225openalex-snapshot
An Endogenously Produced Fragment of Cardiac Myosin-Binding Protein C Is Pathogenic and Can Lead to Heart Failure
inferredcites →https://doi.org/10.1161/jaha.115.002836openalex-snapshot
Alterations in Multi‐Scale Cardiac Architecture in Association With Phosphorylation of Myosin Binding Protein‐C
inferredcites →https://doi.org/10.1172/jci7377openalex-snapshot
Dilated cardiomyopathy in homozygous myosin-binding protein-C mutant mice
inferredcites →https://doi.org/10.1529/biophysj.105.068015openalex-snapshot
Mapping Complex Myoarchitecture in the Bovine Tongue with Diffusion-Spectrum Magnetic Resonance Imaging
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Proteomic analysis of cardiac ventricles: baso-apical differences
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Genetic, clinical, molecular, and pathogenic aspects of the South Asian–specific polymorphic MYBPC3Δ25bp variant
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Characterizing collagen scaffold compliance with native myocardial strains using an ex-vivo cardiac model: The physio-mechanical influence of scaffold architecture and attachment method
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Amino terminus of cardiac myosin binding protein-C regulates cardiac contractility
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Myoarchitectural disarray of hypertrophic cardiomyopathy begins pre‐birth
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Myocardial dysfunction caused by MyBPC3 P459fs mutation in hypertrophic cardiomyopathy: evidence from multi-omics approaches and super-resolution imaging
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The Junctophilin-2 Mutation p.(Thr161Lys) Is Associated with Hypertrophic Cardiomyopathy Using Patient-Specific iPS Cardiomyocytes and Demonstrates Prolonged Action Potential and Increased Arrhythmogenicity
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Sarcomeres Morphology and Z-Line Arrangement Disarray Induced by Ventricular Premature Contractions through the Rac2/Cofilin Pathway
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Abnormal ventricular wall patterning precedes and drives MYBPC3 hypertrophic cardiomyopathy
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