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c-Jun Is Required for Nuclear Factor-κB–Dependent, LPS-Stimulated Fos-Related Antigen-1 Transcription in Alveolar Macrophages

https://doi.org/10.1165/rcmb.2016-0028oc
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/j.1460-2075.1993.tb06066.xopenalex-snapshot
Cross‐coupling of the NF‐kappa B p65 and Fos/Jun transcription factors produces potentiated biological function.
inferredcites →https://doi.org/10.1016/j.bbrc.2008.04.063openalex-snapshot
ERK signaling regulates tumor promoter induced c-Jun recruitment at the Fra-1 promoter
inferredcites →https://doi.org/10.1016/j.tibs.2004.11.009openalex-snapshot
Phosphorylation of NF-κB and IκB proteins: implications in cancer and inflammation
inferredcites →https://doi.org/10.1038/35021228openalex-snapshot
Toll-like receptors in the induction of the innate immune response
inferredcites →https://doi.org/10.1038/nature03963openalex-snapshot
Psoriasis-like skin disease and arthritis caused by inducible epidermal deletion of Jun proteins
inferredcites →https://doi.org/10.1038/nrc1209openalex-snapshot
AP-1: a double-edged sword in tumorigenesis
inferredcites →https://doi.org/10.1038/nri2655openalex-snapshot
Regulation of tissue homeostasis by NF-κB signalling: implications for inflammatory diseases
inferredcites →https://doi.org/10.1038/onc.2011.101openalex-snapshot
Interstitial lung disease induced by gefitinib and Toll-like receptor ligands is mediated by Fra-1
inferredcites →https://doi.org/10.1038/sj.onc.1208583openalex-snapshot
Mitogen regulated induction of FRA-1 proto-oncogene is controlled by the transcription factors binding to both serum and TPA response elements
inferredcites →https://doi.org/10.1074/jbc.m303505200openalex-snapshot
Multiple cis-Elements Mediate the Transcriptional Activation of Human fra-1 by 12-O-Tetradecanoylphorbol-13-acetate in Bronchial Epithelial Cells
inferredcites →https://doi.org/10.1111/j.1600-065x.2012.01104.xopenalex-snapshot
IκB kinase regulation of the TPL‐2/ERK MAPK pathway
inferredcites →https://doi.org/10.1128/mcb.01065-12openalex-snapshot
IκB Kinase 2 Regulates TPL-2 Activation of Extracellular Signal-Regulated Kinases 1 and 2 by Direct Phosphorylation of TPL-2 Serine 400
inferredcites →https://doi.org/10.1128/mcb.24.17.7806-7819.2004openalex-snapshot
NF-κB and AP-1 Connection: Mechanism of NF-κB-Dependent Regulation of AP-1 Activity
inferredcites →https://doi.org/10.1146/annurev.immunol.021908.132641openalex-snapshot
Regulation and Function of NF-κB Transcription Factors in the Immune System
inferredcites →https://doi.org/10.1152/ajplung.00315.2014openalex-snapshot
Visualization of Fra-1/AP-1 activation during LPS-induced inflammatory lung injury using fluorescence optical imaging
inferredcites →https://doi.org/10.1165/rcmb.2009-0136ocopenalex-snapshot
Ablation of Glutaredoxin-1 Attenuates Lipopolysaccharide-Induced Lung Inflammation and Alveolar Macrophage Activation
inferredcites →https://doi.org/10.1165/rcmb.2011-0169ocopenalex-snapshot
Genetic Disruption of Fra-1 Decreases Susceptibility to Endotoxin-Induced Acute Lung Injury and Mortality in Mice
inferredcites →https://doi.org/10.1186/ar2338openalex-snapshot
Activator protein 1 (Fos/Jun) functions in inflammatory bone and skin disease
inferredcites →https://doi.org/10.1371/journal.pone.0084062openalex-snapshot
Regulation of c-Fos Gene Expression by NF-κB: A p65 Homodimer Binding Site in Mouse Embryonic Fibroblasts but Not Human HEK293 Cells
inferredcites →https://doi.org/10.4049/jimmunol.0901196openalex-snapshot
Fos Proteins Suppress Dextran Sulfate Sodium-Induced Colitis through Inhibition of NF-κB
inferredcites →https://doi.org/10.4049/jimmunol.177.10.7193openalex-snapshot
A JNK-Independent Signaling Pathway Regulates TNFα-Stimulated, c-Jun-Driven FRA-1 Protooncogene Transcription in Pulmonary Epithelial Cells
inferredcites →https://doi.org/10.5858/2009-0660-oar1.1openalex-snapshot
Increased Levels of Nuclear Factor κB and Fos-Related Antigen 1 in Lung Tissues From Patients With Acute Respiratory Distress Syndrome

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Peroxiredoxin-1 is an H2o2 Safe-Guard Antioxidant and Signalling Enzyme in Macrophages Independent of Their Polarization State
inferred← citeshttps://doi.org/10.1007/s10753-017-0535-3openalex-snapshot
Anti-inflammatory Role of Pilose Antler Peptide in LPS-Induced Lung Injury
inferred← citeshttps://doi.org/10.1016/j.arres.2023.100083openalex-snapshot
Peroxiredoxin-1 is an H2O2 safe-guard antioxidant and signalling enzyme in M1 macrophages
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inferred← citeshttps://doi.org/10.1038/s41467-026-69000-3openalex-snapshot
Microbiota-induced EI24 improves homeostasis but impedes function of alveolar macrophages via metabolic regulation
inferred← citeshttps://doi.org/10.1038/s41598-020-69735-zopenalex-snapshot
FOSL1 is a novel mediator of endotoxin/lipopolysaccharide-induced pulmonary angiogenic signaling
inferred← citeshttps://doi.org/10.1080/00450618.2017.1334824openalex-snapshot
Measuring temporal expression, systematic response, and post-mortem stability to assess potential markers for estimating wound age: an example of Fosl1 in contused skeletal muscle
inferred← citeshttps://doi.org/10.1093/jimmun/vkae029openalex-snapshot
Absence of c-Maf and IL-10 enables type I IFN enhancement of innate responses to LPS in alveolar macrophages
inferred← citeshttps://doi.org/10.1101/2024.05.22.594428openalex-snapshot
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Cancer cell phagocytosis induces an anti-inflammatory gene regulatory program in macrophages
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Fra‐1 induces apoptosis and neuroinflammation by targeting S100A8 to modulate TLR4 pathways in spinal cord ischemia/reperfusion injury
inferred← citeshttps://doi.org/10.1155/2021/5692039openalex-snapshot
Network Pharmacological Study of Achyranthis bidentatae Radix Effect on Bone Trauma
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inferred← citeshttps://doi.org/10.1172/jci96832openalex-snapshot
Transcription factor Fra-1 targets arginase-1 to enhance macrophage-mediated inflammation in arthritis
inferred← citeshttps://doi.org/10.3389/fimmu.2021.701675openalex-snapshot
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inferred← citeshttps://doi.org/10.3389/fimmu.2022.954744openalex-snapshot
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Glabrol—an impurity in licorice extract—causes toxicity in muscle, bone, and immune tissues through activation of the AP-1 signaling pathway
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inferred← citeshttps://doi.org/10.3390/antiox10060847openalex-snapshot
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inferred← citeshttps://doi.org/10.3390/biom11121801openalex-snapshot
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inferred← citeshttps://doi.org/10.3390/ijms20081912openalex-snapshot
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