On the public citation graph

Genetic Disruption of Fra-1 Decreases Susceptibility to Endotoxin-Induced Acute Lung Injury and Mortality in Mice

https://doi.org/10.1165/rcmb.2011-0169oc

0 signed · 59 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.

Claim with ORCID

Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.

See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/(sici)1526-968x(200002)26:2<113::aid-gene3>3.0.co;2-2openalex-snapshot

Epiblast-restricted Cre expression in MORE mice: A tool to distinguish embryonic vs. extra-embryonic gene function

inferredcites →https://doi.org/10.1016/s0034-5687(01)00260-2openalex-snapshot

Regulation of experimental lung inflammation

inferredcites →https://doi.org/10.1016/s0039-6060(99)70161-3openalex-snapshot

Deficiency of the transcription factor c-fos increases lipopolysaccharide-induced macrophage interleukin 12 production

inferredcites →https://doi.org/10.1016/s0955-0674(97)80068-3openalex-snapshot

AP-1 function and regulation

inferredcites →https://doi.org/10.1038/35021228openalex-snapshot

Toll-like receptors in the induction of the innate immune response

inferredcites →https://doi.org/10.1038/35100529openalex-snapshot

Toll-like receptors and innate immunity

inferredcites →https://doi.org/10.1038/cr.2010.52openalex-snapshot

Fra-1 protooncogene regulates IL-6 expression in macrophages and promotes the generation of M2d macrophages

inferredcites →https://doi.org/10.1038/nature03963openalex-snapshot

Psoriasis-like skin disease and arthritis caused by inducible epidermal deletion of Jun proteins

inferredcites →https://doi.org/10.1038/ncb1761openalex-snapshot

Epidermal JunB represses G-CSF transcription and affects haematopoiesis and bone formation

inferredcites →https://doi.org/10.1038/nrc1209openalex-snapshot

AP-1: a double-edged sword in tumorigenesis

inferredcites →https://doi.org/10.1038/onc.2011.101openalex-snapshot

Interstitial lung disease induced by gefitinib and Toll-like receptor ligands is mediated by Fra-1

inferredcites →https://doi.org/10.1038/sj.emboj.7600282openalex-snapshot

The Fos‐related antigen Fra‐1 is an activator of bone matrix formation

inferredcites →https://doi.org/10.1038/sj.onc.1204383openalex-snapshot

AP-1 in cell proliferation and survival

inferredcites →https://doi.org/10.1046/j.1365-4624.1997.00007.xopenalex-snapshot

Reprogramming the signalling requirement for AP‐1 (activator protein‐1) activation during differentiation of precursor CD4+ T‐cells into effector Th1 and Th2 cells

inferredcites →https://doi.org/10.1056/nejmoa050333openalex-snapshot

Incidence and Outcomes of Acute Lung Injury

inferredcites →https://doi.org/10.1073/pnas.0910371106openalex-snapshot

Epidermal loss of JunB leads to a SLE phenotype due to hyper IL-6 signaling

inferredcites →https://doi.org/10.1073/pnas.92.11.4972openalex-snapshot

Antitumor promotion by phenolic antioxidants: inhibition of AP-1 activity through induction of Fra expression.

inferredcites →https://doi.org/10.1074/jbc.m407071200openalex-snapshot

MEK1-dependent Delayed Expression of Fos-related Antigen-1 Counteracts c-Fos and p65 NF-κB-mediated Interleukin-8 Transcription in Response to Cytokines or Growth Factors

inferredcites →https://doi.org/10.1093/emboj/cdf648openalex-snapshot

Th2 cell‐specific cytokine expression and allergen‐induced airway inflammation depend on JunB

inferredcites →https://doi.org/10.1093/intimm/dxl004openalex-snapshot

c-Fos suppresses systemic inflammatory response to endotoxin

inferredcites →https://doi.org/10.1093/intimm/dxp015openalex-snapshot

Fra-1 negatively regulates lipopolysaccharide-mediated inflammatory responses

inferredcites →https://doi.org/10.1093/nar/19.20.5537openalex-snapshot

Difference in transcriptional regulatory function between c-Fos and Fra-2

inferredcites →https://doi.org/10.1111/j.0105-2896.2005.00332.xopenalex-snapshot

Fos/AP‐1 proteins in bone and the immune system

inferredcites →https://doi.org/10.1128/mcb.24.17.7806-7819.2004openalex-snapshot

NF-κB and AP-1 Connection: Mechanism of NF-κB-Dependent Regulation of AP-1 Activity

inferredcites →https://doi.org/10.1128/mcb.8.5.2063openalex-snapshot

fra-1: a serum-inducible, cellular immediate-early gene that encodes a fos-related antigen.

inferredcites →https://doi.org/10.1136/ard.2010.140533openalex-snapshot

Targeting inflammation by modulating the Jun/AP-1 pathway

inferredcites →https://doi.org/10.1152/ajplung.00140.2002openalex-snapshot

Role and regulation of activator protein-1 in toxicant-induced responses of the lung

inferredcites →https://doi.org/10.1152/ajplung.00221.2003openalex-snapshot

DEP-inducedfra-1expression correlates with a distinct activation of AP-1-dependent gene transcription in the lung

inferredcites →https://doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1801openalex-snapshot

GENETIC DISRUPTION OF FRA-1/AP-1 TRANSCRIPTION FACTOR DECREASES SUSCEPTIBILITY TO ENDOTOXIN-INDUCED LUNG INFLAMMATION AND DEATH IN MICE

inferredcites →https://doi.org/10.1186/ar2338openalex-snapshot

Activator protein 1 (Fos/Jun) functions in inflammatory bone and skin disease

inferredcites →https://doi.org/10.1242/dev.127.22.4937openalex-snapshot

Placental vascularisation requires the AP-1 component Fra1

inferredcites →https://doi.org/10.4049/jimmunol.0901196openalex-snapshot

Fos Proteins Suppress Dextran Sulfate Sodium-Induced Colitis through Inhibition of NF-κB

inferredcites →https://doi.org/10.4049/jimmunol.158.12.5934openalex-snapshot

Impaired activation of nuclear factor-kappaB in endotoxin-tolerant rats is associated with down-regulation of chemokine gene expression and inhibition of neutrophilic lung inflammation

inferredcites →https://doi.org/10.4049/jimmunol.172.12.7377openalex-snapshot

Simvastatin Augments Lipopolysaccharide-Induced Proinflammatory Responses in Macrophages by Differential Regulation of the c-Fos and c-Jun Transcription Factors

inferredcites →https://doi.org/10.4049/jimmunol.176.8.4995openalex-snapshot

Duration and Intensity of NF-κB Activity Determine the Severity of Endotoxin-Induced Acute Lung Injury

inferredcites →https://doi.org/10.4049/jimmunol.178.10.6504openalex-snapshot

Airway Epithelium Controls Lung Inflammation and Injury through the NF-κB Pathway

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4016972openalex-extracted

Alpha-MMC Suppresses the LPS-Induced Inflammatory Cytokine Storm by Inhibiting TAK1 in the TLR4-MAPK/NF-κB Signaling Pathway

inferred← citeshttps://doi.org/10.1016/j.bbi.2022.11.011openalex-snapshot

The PMN-MDSC – A key player in glucocorticoid resistance following combined physical and psychosocial trauma

inferred← citeshttps://doi.org/10.1016/j.biopha.2016.05.025openalex-snapshot

Tetrahydroberberrubine attenuates lipopolysaccharide-induced acute lung injury by down-regulating MAPK, AKT, and NF-κB signaling pathways

inferred← citeshttps://doi.org/10.1016/j.etap.2026.104954openalex-snapshot

Assessment of the toxicity of acrolein on an alveolus microphysiological system

inferred← citeshttps://doi.org/10.1016/j.intimp.2021.108492openalex-snapshot

Identification of the Fosl1/AMPK/autophagy axis involved in apoptotic and inflammatory effects following spinal cord injury

inferred← citeshttps://doi.org/10.1016/j.omtn.2019.12.036openalex-snapshot

Silencing lncRNA AGAP2-AS1 Upregulates miR-195-5p to Repress Migration and Invasion of EC Cells via the Decrease of FOSL1 Expression

inferred← citeshttps://doi.org/10.1038/s41598-020-69735-zopenalex-snapshot

FOSL1 is a novel mediator of endotoxin/lipopolysaccharide-induced pulmonary angiogenic signaling

inferred← citeshttps://doi.org/10.1128/mcb.06390-11openalex-snapshot

Oxidant-Induced Cell Death and Nrf2-Dependent Antioxidative Response Are Controlled by Fra-1/AP-1

inferred← citeshttps://doi.org/10.1152/ajplung.00315.2014openalex-snapshot

Visualization of Fra-1/AP-1 activation during LPS-induced inflammatory lung injury using fluorescence optical imaging

inferred← citeshttps://doi.org/10.1165/rcmb.2014-0118ocopenalex-snapshot

Myeloid-Specific Fos-Related Antigen-1 Regulates Cigarette Smoke–Induced Lung Inflammation, Not Emphysema, in Mice

inferred← citeshttps://doi.org/10.1165/rcmb.2016-0028ocopenalex-snapshot

c-Jun Is Required for Nuclear Factor-κB–Dependent, LPS-Stimulated Fos-Related Antigen-1 Transcription in Alveolar Macrophages

inferred← citeshttps://doi.org/10.1166/jbt.2021.2557openalex-snapshot

Serum Amyloid A Aggravates Lipopolysaccharide-Induced Injury of BEAS-2B Cells by Activating Toll-Like Receptor 2/Activator Protein-1 Signaling

inferred← citeshttps://doi.org/10.1172/jci.insight.154183openalex-snapshot

Functional reprogramming of monocytes in patients with acute and convalescent severe COVID-19

inferred← citeshttps://doi.org/10.1172/jci96832openalex-snapshot

Transcription factor Fra-1 targets arginase-1 to enhance macrophage-mediated inflammation in arthritis

inferred← citeshttps://doi.org/10.1186/s13287-026-04945-2openalex-snapshot

Mesenchymal stem cells-derived TGF-β1 promotes polarization of M2 macrophages in mice with acute-on-chronic liver failure via FOSL1/MERTK axis

inferred← citeshttps://doi.org/10.1371/journal.pone.0041611openalex-snapshot

Fra-1/AP-1 Transcription Factor Negatively Regulates Pulmonary Fibrosis In Vivo

inferred← citeshttps://doi.org/10.20944/preprints202312.1378.v1openalex-snapshot

FOSL1’s Diverse Roles in Glioma/Glioma Stem Cells and Tu-morigenesis: A Comprehensive Review

inferred← citeshttps://doi.org/10.21203/rs.3.rs-105918/v1openalex-snapshot

Fosl1 Targets AMPK to Inhibit Autophagy-Mediated Anti-Apoptotic and Anti-Inflammatory Effects in Spinal Cord Injury

inferred← citeshttps://doi.org/10.3389/fcell.2025.1659986openalex-snapshot

FRA1 (FOSL1) suppresses neoplastic transformation and modulates radiation responses via transcriptional control of mitogenic and stress-responsive networks

inferred← citeshttps://doi.org/10.3389/fimmu.2022.954744openalex-snapshot

The Fra-1: Novel role in regulating extensive immune cell states and affecting inflammatory diseases

inferred← citeshttps://doi.org/10.3389/fmolb.2023.1164220openalex-snapshot

Bioinformatic and systems biology approach revealing the shared genes and molecular mechanisms between COVID-19 and non-alcoholic hepatitis

inferred← citeshttps://doi.org/10.3390/ijms23031521openalex-snapshot

Role of the Transcription Factor FOSL1 in Organ Development and Tumorigenesis

inferred← citeshttps://doi.org/10.3390/ijms25105362openalex-snapshot

FOSL1’s Oncogene Roles in Glioma/Glioma Stem Cells and Tumorigenesis: A Comprehensive Review

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.