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Induction of B7-1 in podocytes is associated with nephrotic syndrome
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1006/excr.1997.3739openalex-snapshot
Rearrangements of the Cytoskeleton and Cell Contacts Induce Process Formation during Differentiation of Conditionally Immortalized Mouse Podocyte Cell Lines
inferredcites →https://doi.org/10.1007/s001340000593openalex-snapshot
Microalbuminuria as an early index of impairment of glomerular permeability in postoperative septic patients
inferredcites →https://doi.org/10.1016/0040-8166(81)90041-0openalex-snapshot
The syncytial nature and phagocytic activity of the branchial podocytes in the grass shrimp, Palaemonetes pugio
inferredcites →https://doi.org/10.1016/0165-2478(96)02526-6openalex-snapshot
Transmembrane signaling through CD80 (B7-1) induces growth arrest and cell spreading of human B lymphocytes accompanied by protein tyrosine phosphorylation
inferredcites →https://doi.org/10.1016/s0002-9440(10)64421-5openalex-snapshot
Co-Localization of Nephrin, Podocin, and the Actin Cytoskeleton
inferredcites →https://doi.org/10.1016/s0002-9440(10)65046-8openalex-snapshot
Expression of Agrin, Dystroglycan, and Utrophin in Normal Renal Tissue and in Experimental Glomerulopathies
inferredcites →https://doi.org/10.1016/s0140-6736(03)14184-0openalex-snapshot
Nephrotic syndrome in childhood
inferredcites →https://doi.org/10.1016/s0167-5699(98)01418-2openalex-snapshot
Structure and evolution of the extended B7 family
inferredcites →https://doi.org/10.1016/s0955-0674(00)00130-7openalex-snapshot
Functions of α3β1 integrin
inferredcites →https://doi.org/10.1016/s0955-0674(99)80027-1openalex-snapshot
Costimulatory regulation of T cell function
inferredcites →https://doi.org/10.1016/s1074-7613(00)80333-7openalex-snapshot
B7-1 and B7-2 Have Overlapping, Critical Roles in Immunoglobulin Class Switching and Germinal Center Formation
inferredcites →https://doi.org/10.1016/s1097-2765(00)80057-xopenalex-snapshot
Positionally Cloned Gene for a Novel Glomerular Protein—Nephrin—Is Mutated in Congenital Nephrotic Syndrome
inferredcites →https://doi.org/10.1038/301527a0openalex-snapshot
A severe combined immunodeficiency mutation in the mouse
inferredcites →https://doi.org/10.1038/70905openalex-snapshot
T-cell stimulation: an abundance of B7s
inferredcites →https://doi.org/10.1038/73456openalex-snapshot
Mutations in ACTN4, encoding α-actinin-4, cause familial focal segmental glomerulosclerosis
inferredcites →https://doi.org/10.1038/74139openalex-snapshot
Getting a foothold in nephrotic syndrome
inferredcites →https://doi.org/10.1038/74166openalex-snapshot
NPHS2, encoding the glomerular protein podocin, is mutated in autosomal recessive steroid-resistant nephrotic syndrome
inferredcites →https://doi.org/10.1038/89712openalex-snapshot
Kissing cousins: immunological and neurological synapses
inferredcites →https://doi.org/10.1038/ni846openalex-snapshot
CTLA-4–Ig regulates tryptophan catabolism in vivo
inferredcites →https://doi.org/10.1038/nri1004openalex-snapshot
Innate immune sensing and its roots: the story of endotoxin
inferredcites →https://doi.org/10.1046/j.1523-1747.2000.00997.xopenalex-snapshot
Allergens and Irritants Transcriptionally Upregulate CD80 Gene Expression in Human Keratinocytes
inferredcites →https://doi.org/10.1046/j.1523-1755.1998.00044.xopenalex-snapshot
Progression of glomerular diseases: Is the podocyte the culprit?
inferredcites →https://doi.org/10.1046/j.1523-1755.2000.00070.xopenalex-snapshot
Regulation of mouse podocyte process dynamics by protein tyrosine phosphatases
inferredcites →https://doi.org/10.1046/j.1523-1755.2003.00726.xopenalex-snapshot
Hypercholesterolemia is a prerequisite for puromycin inducible damage in mouse kidney
inferredcites →https://doi.org/10.1073/pnas.88.12.5096openalex-snapshot
Direct derivation of conditionally immortal cell lines from an H-2Kb-tsA58 transgenic mouse.
inferredcites →https://doi.org/10.1074/jbc.m204806200openalex-snapshot
Determinants of Vascular Permeability in the Kidney Glomerulus
inferredcites →https://doi.org/10.1083/jcb.111.3.1255openalex-snapshot
The tight junction protein ZO-1 is concentrated along slit diaphragms of the glomerular epithelium.
inferredcites →https://doi.org/10.1083/jcb.139.1.193openalex-snapshot
Synaptopodin: An Actin-associated Protein in Telencephalic Dendrites and Renal Podocytes
inferredcites →https://doi.org/10.1093/hmg/10.1.1openalex-snapshot
The murine nephrin gene is specifically expressed in kidney, brain and pancreas: inactivation of the gene leads to massive proteinuria and neonatal death
inferredcites →https://doi.org/10.1093/jimmunol/194.9.4066openalex-snapshot
Pillars Article: The Immunological Synapse: A Molecular Machine Controlling T Cell Activation. Science. 1999. 285: 221–227
inferredcites →https://doi.org/10.1097/01.lab.0000029620.13097.19openalex-snapshot
Glomerular Endothelium Exhibits Enhanced Expression of Costimulatory Adhesion Molecules, CD80 and CD86, by Warm Ischemia/Reperfusion Injury in Rats
inferredcites →https://doi.org/10.1126/science.282.5397.2266openalex-snapshot
A Receptor/Cytoskeletal Movement Triggered by Costimulation During T Cell Activation
inferredcites →https://doi.org/10.1126/science.285.5425.221openalex-snapshot
The Immunological Synapse: A Molecular Machine Controlling T Cell Activation
inferredcites →https://doi.org/10.1126/science.286.5438.312openalex-snapshot
Congenital Nephrotic Syndrome in Mice Lacking CD2-Associated Protein
inferredcites →https://doi.org/10.1128/mcb.21.14.4829-4836.2001openalex-snapshot
Proteinuria and Perinatal Lethality in Mice Lacking NEPH1, a Novel Protein with Homology to NEPHRIN
inferredcites →https://doi.org/10.1146/annurev.immunol.18.1.165openalex-snapshot
T Cell Activation and the Cytoskeleton
inferredcites →https://doi.org/10.1146/annurev.immunol.19.1.375openalex-snapshot
The Immunological Synapse
inferredcites →https://doi.org/10.1146/annurev.immunol.21.120601.141126openalex-snapshot
Toll-Like Receptors
inferredcites →https://doi.org/10.1152/ajprenal.1996.271.3.f770openalex-snapshot
Novel mouse embryonic renal marker gene products differentially expressed during kidney development
inferredcites →https://doi.org/10.1159/000017097openalex-snapshot
Neuroimaging Findings in Twins Discordant for Alzheimer’s Disease
inferredcites →https://doi.org/10.1172/jci9244openalex-snapshot
CTLA4Ig inhibits T cell–dependent B-cell maturation in murine systemic lupus erythematosus
inferredcites →https://doi.org/10.1242/dev.122.11.3537openalex-snapshot
Alpha 3 beta 1 integrin has a crucial role in kidney and lung organogenesis
inferredcites →https://doi.org/10.1242/jcs.112.17.2925openalex-snapshot
α3β1 integrin regulates epithelial cytoskeletal organization
inferredcites →https://doi.org/10.1681/asn.v10151openalex-snapshot
The Dysregulated Podocyte Phenotype
inferredcites →https://doi.org/10.1681/asn.v113403openalex-snapshot
Glomerular Expression of Dystroglycans Is Reduced in Minimal Change Nephrosis But Not in Focal Segmental Glomerulosclerosis
inferredcites →https://doi.org/10.4049/jimmunol.153.11.5038openalex-snapshot
Characterization of the murine B7-1 genomic locus reveals an additional exon encoding an alternative cytoplasmic domain and a chromosomal location of chromosome 16, band B5.
inferredcites →https://doi.org/10.4049/jimmunol.162.3.1367openalex-snapshot
TCR, LFA-1, and CD28 Play Unique and Complementary Roles in Signaling T Cell Cytoskeletal Reorganization
inferredcites →https://doi.org/10.4049/jimmunol.165.3.1243openalex-snapshot
Age-Dependent Alterations in the Assembly of Signal Transduction Complexes at the Site of T Cell/APC Interaction
inferredcites →https://doi.org/10.4049/jimmunol.166.11.6491openalex-snapshot
Mechanism of Melphalan-Induced B7-1 Gene Expression in P815 Tumor Cells
inferredcites →https://doi.org/10.4049/jimmunol.166.9.5688openalex-snapshot
Endotoxin-Induced Maturation of MyD88-Deficient Dendritic Cells
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1002/9781119309642.ch8openalex-snapshot
Diabetic Nephropathy – Research
inferred← citeshttps://doi.org/10.1002/art.38740openalex-snapshot
The Beneficial Effect of Abatacept in Lupus Nephritis May Include Stabilization of β1 Integrin Activation in Podocytes and Treg Cell Repopulation: Comment on the Article by Furie et al
inferred← citeshttps://doi.org/10.1002/art.39499openalex-snapshot
Lupus Nephritis IgG Induction of Calcium/Calmodulin‐Dependent Protein Kinase IV Expression in Podocytes and Alteration of Their Function
inferred← citeshttps://doi.org/10.1002/art.40157openalex-snapshot
Editorial: Podocytes as Active Participants in Lupus Nephritis
inferred← citeshttps://doi.org/10.1002/cbf.3719openalex-snapshot
Comprehensively characterizing cellular changes and the expression of THSD7A and PLA2R1 under multiple in vitro models of podocyte injury
inferred← citeshttps://doi.org/10.1002/cm.21162openalex-snapshot
Visualization of cytoskeletal dynamics in podocytes using adenoviral vectors
inferred← citeshttps://doi.org/10.1002/jcla.22713openalex-snapshot
Efficacy of different hemodialysis methods on dendritic cell marker CD40 and CD80 and platelet activation marker CD62P and P10 in patients with chronic renal failure
inferred← citeshttps://doi.org/10.1002/kjm2.12385openalex-snapshot
Podocentric view of glomerular proteinuria: Focused on cytoskeletal changes and toward promising targeted therapies and challenges
inferred← citeshttps://doi.org/10.1002/mco2.614openalex-snapshot
Membranous nephropathy: pathogenesis and treatments
inferred← citeshttps://doi.org/10.1002/med.22074openalex-snapshot
Canonical effects of cytokines on glomerulonephritis: A new outlook in nephrology
inferred← citeshttps://doi.org/10.1002/path.4235openalex-snapshot
Inhibition of type I interferon signalling prevents TLR ligand‐mediated proteinuria
inferred← citeshttps://doi.org/10.1002/path.5508openalex-snapshot
Podocytes present antigen to activate specific T cell immune responses in inflammatory renal disease
inferred← citeshttps://doi.org/10.1007/978-0-387-84828-0_482openalex-snapshot
Spectrum of Minimal Change Disease to Focal Segmental Glomerulosclerosis
inferred← citeshttps://doi.org/10.1007/978-1-4614-8166-9_2openalex-snapshot
Minimal Change Disease in Adults
inferred← citeshttps://doi.org/10.1007/978-3-031-49931-9_1openalex-snapshot
Nephritic syndrome: An algorithmic approach
inferred← citeshttps://doi.org/10.1007/978-3-319-27334-1_10-1openalex-snapshot
Focal Segmental Glomerulosclerosis, Pediatric
inferred← citeshttps://doi.org/10.1007/978-3-319-27334-1_3-1openalex-snapshot
Diagnostic Testing in Glomerular Disease
inferred← citeshttps://doi.org/10.1007/978-3-319-27334-1_7-1openalex-snapshot
Minimal Change Disease in Adults
inferred← citeshttps://doi.org/10.1007/978-3-319-43359-2_5openalex-snapshot
Immune-Mediated Mechanisms of Proteinuria
inferred← citeshttps://doi.org/10.1007/978-3-319-47385-7_6openalex-snapshot
Immunopathology of the Urinary System
inferred← citeshttps://doi.org/10.1007/978-3-319-49379-4_10openalex-snapshot
Focal Segmental Glomerulosclerosis, Pediatric
inferred← citeshttps://doi.org/10.1007/978-3-319-49379-4_3openalex-snapshot
Diagnostic Testing in Glomerular Disease
inferred← citeshttps://doi.org/10.1007/978-3-319-49379-4_7openalex-snapshot
Minimal Change Disease in Adults
inferred← citeshttps://doi.org/10.1007/978-3-540-76341-3_29openalex-snapshot
Immune-mediated Glomerular Injury
inferred← citeshttps://doi.org/10.1007/978-3-642-27843-3_24-1openalex-snapshot
Idiopathic Nephrotic Syndrome in Children: Clinical Aspects
inferred← citeshttps://doi.org/10.1007/978-3-642-27843-3_24-2openalex-snapshot
Idiopathic Nephrotic Syndrome in Children: Clinical Aspects
inferred← citeshttps://doi.org/10.1007/978-3-642-27843-3_25-1openalex-snapshot
Immune-Mediated Glomerular Injury in Children
inferred← citeshttps://doi.org/10.1007/978-3-642-27843-3_25-2openalex-snapshot
Immune-Mediated Glomerular Injury in Children
inferred← citeshttps://doi.org/10.1007/978-3-662-43596-0_24openalex-snapshot
Idiopathic Nephrotic Syndrome in Children: Clinical Aspects
inferred← citeshttps://doi.org/10.1007/978-3-662-43596-0_25openalex-snapshot
Immune-Mediated Glomerular Injury in Children
inferred← citeshttps://doi.org/10.1007/978-4-431-55270-3_13openalex-snapshot
Podocytes as a Direct Target of Drugs Used in Idiopathic Nephrotic Syndrome
inferred← citeshttps://doi.org/10.1007/978-4-431-55270-3_4openalex-snapshot
Co-stimulatory Molecule CD80 (B7.1) in MCNS
inferred← citeshttps://doi.org/10.1007/978-4-431-55270-3_5openalex-snapshot
Energy and Mammalian Target of Rapamycin Complex 1 (mTORC1) in Minimal Change Nephrotic Syndrome
inferred← citeshttps://doi.org/10.1007/978-4-431-55270-3_7openalex-snapshot
Regulatory T Cells and Oxidative Stress in Minimal Change Nephropathy
inferred← citeshttps://doi.org/10.1007/978-4-431-55270-3_8openalex-snapshot
Cytokines as Active Factors in Minimal Change Nephrotic Syndrome
inferred← citeshttps://doi.org/10.1007/978-94-007-7699-9_4openalex-snapshot
Biomarkers in Focal Segmental Glomerulosclerosis
inferred← citeshttps://doi.org/10.1007/978-94-007-7743-9_4-1openalex-snapshot
Biomarkers in Focal Segmental Glomerulosclerosis
inferred← citeshttps://doi.org/10.1007/978-981-13-8871-2_10openalex-snapshot
Role of Podocyte Injury in Glomerulosclerosis
inferred← citeshttps://doi.org/10.1007/s00011-015-0813-1openalex-snapshot
The experimental model of nephrotic syndrome induced by Doxorubicin in rodents: an update
inferred← citeshttps://doi.org/10.1007/s00011-017-1058-yopenalex-snapshot
Montelukast improves the changes of cytoskeletal and adaptor proteins of human podocytes by interleukin-13
inferred← citeshttps://doi.org/10.1007/s00018-025-06014-yopenalex-snapshot
Podocytes as novel sources of neutrophil serine proteases: expression and regulation by inflammatory molecular patterns
inferred← citeshttps://doi.org/10.1007/s00108-019-0590-yopenalex-snapshot
Minimal-change-Glomerulonephritis und fokal-segmentale Glomerulosklerose
inferred← citeshttps://doi.org/10.1007/s00109-012-0960-6openalex-snapshot
The calcineurin–NFAT pathway allows for urokinase receptor-mediated beta3 integrin signaling to cause podocyte injury
inferred← citeshttps://doi.org/10.1007/s00109-017-1601-xopenalex-snapshot
Inducible ATF3–NFAT axis aggravates podocyte injury
inferred← citeshttps://doi.org/10.1007/s00125-015-3766-6openalex-snapshot
CTLA4-Ig in B7-1-positive diabetic and non-diabetic kidney disease
inferred← citeshttps://doi.org/10.1007/s00134-008-1124-6openalex-snapshot
Polymyxin-B hemoperfusion inactivates circulating proapoptotic factors
inferred← citeshttps://doi.org/10.1007/s00281-007-0094-zopenalex-snapshot
Minimal change nephropathy and focal segmental glomerulosclerosis
inferred← citeshttps://doi.org/10.1007/s00281-013-0415-3openalex-snapshot
Immunopathogenesis of idiopathic nephrotic syndrome with relapse
inferred← citeshttps://doi.org/10.1007/s00424-017-1993-zopenalex-snapshot
Podocytes from the diagnostic and therapeutic point of view
inferred← citeshttps://doi.org/10.1007/s00424-017-2015-xopenalex-snapshot
Podocytes and the quest for precision medicines for kidney diseases
inferred← citeshttps://doi.org/10.1007/s00431-021-04357-9openalex-snapshot
The immunopathogenesis of idiopathic nephrotic syndrome: a narrative review of the literature
inferred← citeshttps://doi.org/10.1007/s00441-019-03147-yopenalex-snapshot
Idiopathic nephrotic syndrome and serum permeability factors: a molecular jigsaw puzzle
inferred← citeshttps://doi.org/10.1007/s00467-006-0409-7openalex-snapshot
Familial steroid-sensitive nephrotic syndrome in Southern Israel: clinical and genetic observations
inferred← citeshttps://doi.org/10.1007/s00467-009-1425-1openalex-snapshot
Enzymatic disease of the podocyte
inferred← citeshttps://doi.org/10.1007/s00467-010-1676-xopenalex-snapshot
Minimal change disease: a “two-hit” podocyte immune disorder?
inferred← citeshttps://doi.org/10.1007/s00467-011-2001-zopenalex-snapshot
Role of circulating fibroblast growth factor-2 in lipopolysaccharide-induced acute kidney injury in mice
inferred← citeshttps://doi.org/10.1007/s00467-012-2376-5openalex-snapshot
Animal models of nephrotic syndrome
inferred← citeshttps://doi.org/10.1007/s00467-013-2498-4openalex-snapshot
Serum from minimal change patients in relapse increases CD80 expression in cultured podocytes
inferred← citeshttps://doi.org/10.1007/s00467-013-2679-1openalex-snapshot
CD80 and suPAR in patients with minimal change disease and focal segmental glomerulosclerosis: diagnostic and pathogenic significance
inferred← citeshttps://doi.org/10.1007/s00467-014-2835-2openalex-snapshot
Increased risk of idiopathic nephrotic syndrome in children with atopic dermatitis
inferred← citeshttps://doi.org/10.1007/s00467-014-2874-8openalex-snapshot
Minimal change disease: a dysregulation of the podocyte CD80–CTLA-4 axis?
inferred← citeshttps://doi.org/10.1007/s00467-014-2915-3openalex-snapshot
Urinary CD80 levels as a diagnostic biomarker of minimal change disease
inferred← citeshttps://doi.org/10.1007/s00467-014-2957-6openalex-snapshot
Case series: CTLA4-IgG1 therapy in minimal change disease and focal segmental glomerulosclerosis
inferred← citeshttps://doi.org/10.1007/s00467-015-3082-xopenalex-snapshot
The glomerular permeability factors in idiopathic nephrotic syndrome
inferred← citeshttps://doi.org/10.1007/s00467-015-3116-4openalex-snapshot
Podocyte directed therapy of nephrotic syndrome—can we bring the inside out?
inferred← citeshttps://doi.org/10.1007/s00467-016-3379-4openalex-snapshot
Pathogenesis of proteinuria in idiopathic minimal change disease: molecular mechanisms
inferred← citeshttps://doi.org/10.1007/s00467-017-3613-8openalex-snapshot
Toll-like receptor 3 (TLR-3), TLR-4 and CD80 expression in peripheral blood mononuclear cells and urinary CD80 levels in children with idiopathic nephrotic syndrome
inferred← citeshttps://doi.org/10.1007/s00467-018-3886-6openalex-snapshot
Urinary CD80: a biomarker for a favorable response to corticosteroids in minimal change disease
inferred← citeshttps://doi.org/10.1007/s00467-019-4203-8openalex-snapshot
Nephrotic syndrome in a dish: recent developments in modeling in vitro
inferred← citeshttps://doi.org/10.1007/s00467-020-04541-3openalex-snapshot
Glomerular endothelial cells and podocytes can express CD80 in patients with minimal change disease during relapse
inferred← citeshttps://doi.org/10.1007/s00467-020-04867-yopenalex-snapshot
Biomarkers in pediatric glomerulonephritis and nephrotic syndrome
inferred← citeshttps://doi.org/10.1007/s00467-021-05196-4openalex-snapshot
How immunosuppressive drugs may directly target podocytes in glomerular diseases
inferred← citeshttps://doi.org/10.1007/s00467-021-05401-4openalex-snapshot
An updated view of the pathogenesis of steroid-sensitive nephrotic syndrome
inferred← citeshttps://doi.org/10.1007/s00467-022-05549-7openalex-snapshot
Benefit of B7-1 staining and abatacept for treatment-resistant post-transplant focal segmental glomerulosclerosis in a predominantly pediatric cohort: time for a reappraisal
inferred← citeshttps://doi.org/10.1007/s00467-024-06643-8openalex-snapshot
Past and future in vitro and in vivo approaches toward circulating factors and biomarkers in idiopathic nephrotic syndrome
inferred← citeshttps://doi.org/10.1007/s10157-017-1489-3openalex-snapshot
Interaction of CD80 with Neph1: a potential mechanism of podocyte injury
inferred← citeshttps://doi.org/10.1007/s10157-022-02218-9openalex-snapshot
Fractalkine deficiency attenuates LPS-induced acute kidney injury and podocyte apoptosis by targeting the PI3K/Akt signal pathway
inferred← citeshttps://doi.org/10.1007/s10238-023-01184-0openalex-snapshot
Mild mesangial proliferative IgA nephropathy with and without minimal change disease
inferred← citeshttps://doi.org/10.1007/s10620-019-05581-yopenalex-snapshot
Gut Leakage of Fungal-Derived Inflammatory Mediators: Part of a Gut-Liver-Kidney Axis in Bacterial Sepsis
inferred← citeshttps://doi.org/10.1007/s11255-023-03592-3openalex-snapshot
The role of toll-like receptors (TLRs) and their therapeutic applications in glomerulonephritis
inferred← citeshttps://doi.org/10.1007/s12079-012-0184-0openalex-snapshot
LPS and PAN-induced podocyte injury in an in vitro model of minimal change disease: changes in TLR profile
inferred← citeshttps://doi.org/10.1007/s12098-018-2734-9openalex-snapshot
Cytotoxic T- Lymphocyte Antigen-4 (CTLA4) Gene Expression and Urinary CTLA4 Levels in Idiopathic Nephrotic Syndrome
inferred← citeshttps://doi.org/10.1007/s12519-015-0003-9openalex-snapshot
Pathogenesis of childhood idiopathic nephrotic syndrome: a paradigm shift from T-cells to podocytes
inferred← citeshttps://doi.org/10.1007/s12519-020-00400-1openalex-snapshot
Immunopathogenesis of idiopathic nephrotic syndrome in children: two sides of the coin
inferred← citeshttps://doi.org/10.1007/s13730-020-00554-xopenalex-snapshot
Spontaneous remission of minimal change nephrotic syndrome in an elderly man
inferred← citeshttps://doi.org/10.1007/s13730-025-01087-xopenalex-snapshot
A case report of spontaneous remission and relapse in minimal change disease: a heterogeneous disease entity
inferred← citeshttps://doi.org/10.1007/s40272-015-0148-yopenalex-snapshot
Emerging Agents for the Management of Nephrotic Syndrome: Progress to Date
inferred← citeshttps://doi.org/10.1007/s40472-017-0149-6openalex-snapshot
Xenotransplantation: Where Are We with Potential Kidney Recipients? Recent Progress and Potential Future Clinical Trials
inferred← citeshttps://doi.org/10.1007/s40620-016-0272-yopenalex-snapshot
Focal segmental glomerular sclerosis: do not overlook the role of immune response
inferred← citeshttps://doi.org/10.1007/s40620-016-0361-yopenalex-snapshot
Combination of cilostazol and probucol protected podocytes from lipopolysaccharide-induced injury by both anti-inflammatory and anti-oxidative mechanisms
inferred← citeshttps://doi.org/10.1007/s40620-017-0402-1openalex-snapshot
Available and incoming therapies for idiopathic focal and segmental glomerulosclerosis in adults
inferred← citeshttps://doi.org/10.1007/s40620-019-00691-2openalex-snapshot
The renal adverse effects of cancer immunotherapy
inferred← citeshttps://doi.org/10.1007/s40746-016-0044-xopenalex-snapshot
Nephrotic Syndrome: Updates and Approaches to Treatment
inferred← citeshttps://doi.org/10.1016/b978-0-12-374994-9.10026-9openalex-snapshot
Pathogenesis of Lupus Nephritis
inferred← citeshttps://doi.org/10.1016/b978-0-12-375672-5.10010-6openalex-snapshot
Biomarkers in Glomerular Disease
inferred← citeshttps://doi.org/10.1016/b978-0-12-800102-8.00029-1openalex-snapshot
Evidence for Renal Progenitors in the Human Kidney
inferred← citeshttps://doi.org/10.1016/b978-0-12-803014-1.00013-3openalex-snapshot
Biomarkers in Glomerular Disease
inferred← citeshttps://doi.org/10.1016/b978-0-323-05876-6.00015-0openalex-snapshot
Introduction to Glomerular Disease
inferred← citeshttps://doi.org/10.1016/b978-0-323-05876-6.00016-2openalex-snapshot
Introduction to Glomerular Disease
inferred← citeshttps://doi.org/10.1016/b978-0-443-33957-8.00038-xopenalex-snapshot
Pathogenesis of lupus nephitis
inferred← citeshttps://doi.org/10.1016/b978-012088488-9.50077-2openalex-snapshot
Renal Filtration, Transport, and Metabolism of Albumin and Albuminuria
inferred← citeshttps://doi.org/10.1016/b978-1-4377-0987-2.00035-2openalex-snapshot
Biological Agents in Kidney Transplantation
inferred← citeshttps://doi.org/10.1016/j.addr.2010.08.012openalex-snapshot
Targeting podocyte-associated diseases☆
inferred← citeshttps://doi.org/10.1016/j.ajpath.2014.05.005openalex-snapshot
Protein Tyrosine Phosphatase 1B Inhibition Protects against Podocyte Injury and Proteinuria
inferred← citeshttps://doi.org/10.1016/j.amjms.2018.01.003openalex-snapshot
Can Focal Segmental Glomerulosclerosis Be Differentiated From Minimal Change Nephrotic Syndrome Using Biomarkers?
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2018.02.036openalex-snapshot
The gut microbiome and elevated cardiovascular risk in obesity and autoimmunity
inferred← citeshttps://doi.org/10.1016/j.bbrc.2005.02.142openalex-snapshot
Expression and regulation of adrenomedullin in renal glomerular podocytes
inferred← citeshttps://doi.org/10.1016/j.bbrc.2022.11.081openalex-snapshot
ANGPTL4 promotes nephrotic syndrome by downregulating podocyte expression of ACTN4 and podocin
inferred← citeshttps://doi.org/10.1016/j.clim.2018.10.001openalex-snapshot
Fra-2 is a novel candidate drug target expressed in the podocytes of lupus nephritis
inferred← citeshttps://doi.org/10.1016/j.clim.2024.110180openalex-snapshot
The role of podocytes in lupus nephritis: Insights and implications
inferred← citeshttps://doi.org/10.1016/j.coi.2004.09.013openalex-snapshot
New insights into disease pathogenesis from mouse lupus genetics
inferred← citeshttps://doi.org/10.1016/j.coph.2017.03.010openalex-snapshot
New biologics in the treatment of rare glomerular diseases of childhood
inferred← citeshttps://doi.org/10.1016/j.ddmod.2010.09.002openalex-snapshot
Models of FSGS and minimal change nephropathy
inferred← citeshttps://doi.org/10.1016/j.drudis.2015.06.003openalex-snapshot
Targeting the podocyte to treat glomerular kidney disease
inferred← citeshttps://doi.org/10.1016/j.ekir.2017.08.013openalex-snapshot
Randomized Clinical Trial Design to Assess Abatacept in Resistant Nephrotic Syndrome
inferred← citeshttps://doi.org/10.1016/j.ekir.2020.08.001openalex-snapshot
Urinary CD80 Discriminates Among Glomerular Disease Types and Reflects Disease Activity
inferred← citeshttps://doi.org/10.1016/j.ekir.2020.09.027openalex-snapshot
Urinary CD 80 in Nephrotic Syndrome: A Biomarker to Distinguish Minimal Change Disease From Other Glomerular Diseases
inferred← citeshttps://doi.org/10.1016/j.gendis.2020.07.005openalex-snapshot
Lipopolysaccharide-induced podocyte injury is regulated by calcineurin/NFAT and TLR4/MyD88/NF-κB signaling pathways through angiopoietin-like protein 4
inferred← citeshttps://doi.org/10.1016/j.humimm.2020.12.001openalex-snapshot
Predicting the functional consequences of genetic variants in co-stimulatory ligand B7-1 using in-silico approaches
inferred← citeshttps://doi.org/10.1016/j.humpath.2006.09.006openalex-snapshot
Upregulated expression of cardiac ankyrin–repeated protein in renal podocytes is associated with proteinuria severity in lupus nephritis
inferred← citeshttps://doi.org/10.1016/j.immuni.2007.05.016openalex-snapshot
Programmed Death-1 Ligand 1 Interacts Specifically with the B7-1 Costimulatory Molecule to Inhibit T Cell Responses
inferred← citeshttps://doi.org/10.1016/j.intimp.2014.08.023openalex-snapshot
Curcumin prevents diabetic nephropathy against inflammatory response via reversing caveolin-1 Tyr14 phosphorylation influenced TLR4 activation
inferred← citeshttps://doi.org/10.1016/j.intimp.2022.108549openalex-snapshot
Podocyte protection by Angptl3 knockout via inhibiting ROS/GRP78 pathway in LPS-induced acute kidney injury
inferred← citeshttps://doi.org/10.1016/j.isci.2022.104694openalex-snapshot
Pulmonary surfactants and the respiratory-renal connection in steroid-sensitive nephrotic syndrome of childhood
inferred← citeshttps://doi.org/10.1016/j.isci.2024.109749openalex-snapshot
Contrasting consequences of podocyte insulin-like growth factor 1 receptor inhibition
inferred← citeshttps://doi.org/10.1016/j.jbiosc.2011.05.001openalex-snapshot
Practical non-targeted gas chromatography/mass spectrometry-based metabolomics platform for metabolic phenotype analysis
inferred← citeshttps://doi.org/10.1016/j.jpeds.2025.114595openalex-snapshot
Unraveling the Immunogenetic Mechanisms of Childhood Idiopathic Nephrotic Syndrome
inferred← citeshttps://doi.org/10.1016/j.jpha.2023.09.003openalex-snapshot
The role of innate immunity in diabetic nephropathy and their therapeutic consequences
inferred← citeshttps://doi.org/10.1016/j.kint.2016.01.012openalex-snapshot
Podocyte injury and its consequences
inferred← citeshttps://doi.org/10.1016/j.kint.2016.06.022openalex-snapshot
The costimulatory receptor B7-1 is not induced in injured podocytes
inferred← citeshttps://doi.org/10.1016/j.kint.2016.09.042openalex-snapshot
Podocyte-specific chemokine (C-C motif) receptor 2 overexpression mediates diabetic renal injury in mice
inferred← citeshttps://doi.org/10.1016/j.kint.2016.10.037openalex-snapshot
Glomerular common gamma chain confers B- and T-cell–independent protection against glomerulonephritis
inferred← citeshttps://doi.org/10.1016/j.kint.2017.12.026openalex-snapshot
Drug repurposing in kidney disease
inferred← citeshttps://doi.org/10.1016/j.kint.2017.12.028openalex-snapshot
Actin dynamics at focal adhesions: a common endpoint and putative therapeutic target for proteinuric kidney diseases
inferred← citeshttps://doi.org/10.1016/j.kint.2025.01.009openalex-snapshot
Melanocortin 5 receptor signaling protects against podocyte injury in proteinuric glomerulopathies
inferred← citeshttps://doi.org/10.1016/j.kint.2026.06.035openalex-snapshot
A vesicular trafficking pathway mediated by N-ethylmaleimide-sensitive factor governs integrin maturation and podocyte integrity
inferred← citeshttps://doi.org/10.1016/j.krcp.2015.03.003openalex-snapshot
Nonimmunologic targets of immunosuppressive agents in podocytes
inferred← citeshttps://doi.org/10.1016/j.lfs.2021.119155openalex-snapshot
Toll-like receptor 4: An attractive therapeutic target for acute kidney injury
inferred← citeshttps://doi.org/10.1016/j.molmed.2018.02.003openalex-snapshot
Mechanisms of Tissue Injury in Lupus Nephritis
inferred← citeshttps://doi.org/10.1016/j.nefro.2018.05.002openalex-snapshot
Inmunoterapia en el cáncer: grandes expectativas en el mundo de la oncología, pero un motivo de preocupación renal
inferred← citeshttps://doi.org/10.1016/j.nefroe.2018.05.005openalex-snapshot
Immunotherapy in cancer: Great expectations in the world of oncology, but a reason for renal concern
inferred← citeshttps://doi.org/10.1016/j.nephro.2011.11.010openalex-snapshot
Syndromes néphrotiques idiopathiques : physiopathologie et prise en charge thérapeutique spécifique chez l’adulte
inferred← citeshttps://doi.org/10.1016/j.nephro.2018.09.004openalex-snapshot
La néphrite de Lyme chez l’homme : bases physiopathologiques et spectre lésionnel rénal
inferred← citeshttps://doi.org/10.1016/j.semarthrit.2016.10.005openalex-snapshot
Unraveling the podocyte injury in lupus nephritis: Clinical and experimental approaches
inferred← citeshttps://doi.org/10.1016/j.semnephrol.2011.06.002openalex-snapshot
Minimal Change Disease: A CD80 podocytopathy?
inferred← citeshttps://doi.org/10.1016/j.semnephrol.2012.07.006openalex-snapshot
Can Existing Drugs Approved for Other Indications Retard Renal Function Decline in Patients With Type 1 Diabetes and Nephropathy?
inferred← citeshttps://doi.org/10.1016/j.semnephrol.2013.11.008openalex-snapshot
Autophagy and Immune Response in Kidneys
inferred← citeshttps://doi.org/10.1016/j.semnephrol.2016.09.008openalex-snapshot
Podocyte-Targeted Treatment for Proteinuric Kidney Disease
inferred← citeshttps://doi.org/10.1016/j.transproceed.2007.02.010openalex-snapshot
Focal Segmental Glomerulosclerosis and Renal Transplantation
inferred← citeshttps://doi.org/10.1016/j.transproceed.2015.07.013openalex-snapshot
Complete Remission of Post-transplantation Recurrence of Focal Segmental Glomerulosclerosis With the Use of Adrenocorticotrophic Hormone Gel: Case Report
inferred← citeshttps://doi.org/10.1016/j.trre.2010.04.001openalex-snapshot
Therapeutic approach to focal and segmental glomerulosclerosis recurrence in kidney transplant recipients
inferred← citeshttps://doi.org/10.1016/j.trsl.2011.06.001openalex-snapshot
Mechanisms of HO-1 mediated attenuation of renal immune injury: a gene profiling study
inferred← citeshttps://doi.org/10.1016/s0001-4079(19)30490-xopenalex-snapshot
La hyalinose segmentaire et focale (HSF)
inferred← citeshttps://doi.org/10.1016/s0031-3939(11)70526-1openalex-snapshot
Idiopatyczny zespół nerczycowy u dzieci – czy jesteśmy świadkami ewolucji obrazu klinicznego i histopatologicznego choroby?
inferred← citeshttps://doi.org/10.1016/s0140-6736(14)60659-0openalex-snapshot
Rare inherited kidney diseases: challenges, opportunities, and perspectives
inferred← citeshttps://doi.org/10.1016/s0140-6736(18)30536-1openalex-snapshot
Idiopathic nephrotic syndrome in children
inferred← citeshttps://doi.org/10.1016/s1762-0945(10)48820-8openalex-snapshot
Syndromes néphrotiques idiopathiques. Physiopathologie et prise en charge thérapeutique spécifique chez l'adulte
inferred← citeshttps://doi.org/10.1016/s1762-0945(19)86766-9openalex-snapshot
10.1016/s1762-0945(19)86766-9
inferred← citeshttps://doi.org/10.1017/erm.2014.3openalex-snapshot
Role of chemokines in proteinuric kidney disorders
inferred← citeshttps://doi.org/10.1038/cddis.2015.125openalex-snapshot
Podocyte apoptosis is prevented by blocking the Toll-like receptor pathway
inferred← citeshttps://doi.org/10.1038/emm.2016.169openalex-snapshot
Interleukin-20 targets podocytes and is upregulated in experimental murine diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/ki.2008.203openalex-snapshot
Triptolide protects podocytes from puromycin aminonucleoside induced injury in vivo and in vitro
inferred← citeshttps://doi.org/10.1038/ki.2009.424openalex-snapshot
Proteinuria: an enzymatic disease of the podocyte?
inferred← citeshttps://doi.org/10.1038/ki.2009.559openalex-snapshot
Toward the development of podocyte-specific drugs
inferred← citeshttps://doi.org/10.1038/ki.2010.111openalex-snapshot
Innate immune receptors and autophagy: implications for autoimmune kidney injury
inferred← citeshttps://doi.org/10.1038/ki.2010.143openalex-snapshot
Urinary CD80 is elevated in minimal change disease but not in focal segmental glomerulosclerosis
inferred← citeshttps://doi.org/10.1038/ki.2010.148openalex-snapshot
Maximal ‘CD80-uria’ with minimal change
inferred← citeshttps://doi.org/10.1038/ki.2010.560openalex-snapshot
ISN Forefronts Symposium 2010 in Sylt, Germany: ‘Induction and Resolution of Renal Inflammation’
inferred← citeshttps://doi.org/10.1038/ki.2013.317openalex-snapshot
The endothelium as part of the integrative glomerular barrier complex
inferred← citeshttps://doi.org/10.1038/ki.2014.116openalex-snapshot
Enhanced glomerular Toll-like receptor 4 expression and signaling in patients with type 2 diabetic nephropathy and microalbuminuria
inferred← citeshttps://doi.org/10.1038/ki.2014.196openalex-snapshot
Albumin-induced podocyte injury and protection are associated with regulation of COX-2
inferred← citeshttps://doi.org/10.1038/ki.2014.428openalex-snapshot
Fine-tuning of NFκB by glycogen synthase kinase 3β directs the fate of glomerular podocytes upon injury
inferred← citeshttps://doi.org/10.1038/ki.2015.140openalex-snapshot
Alternatively spliced isoforms of WT1 control podocyte-specific gene expression
inferred← citeshttps://doi.org/10.1038/ki.2015.5openalex-snapshot
New insights into mechanisms of glomerular injury and repair from the 10th International Podocyte Conference 2014
inferred← citeshttps://doi.org/10.1038/labinvest.2011.198openalex-snapshot
Gα12 activation in podocytes leads to cumulative changes in glomerular collagen expression, proteinuria and glomerulosclerosis
inferred← citeshttps://doi.org/10.1038/labinvest.2012.133openalex-snapshot
Impairment of podocyte function by diphtheria toxin—a new reversible proteinuria model in mice
inferred← citeshttps://doi.org/10.1038/ncomms4296openalex-snapshot
Notch2 activation ameliorates nephrosis
inferred← citeshttps://doi.org/10.1038/ncpneph0300openalex-snapshot
Renal Toll-like receptors: recent advances and implications for disease
inferred← citeshttps://doi.org/10.1038/ng.792openalex-snapshot
Common variation in GPC5 is associated with acquired nephrotic syndrome
inferred← citeshttps://doi.org/10.1038/ng1592openalex-snapshot
TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function
inferred← citeshttps://doi.org/10.1038/nm.1857openalex-snapshot
The actin cytoskeleton of kidney podocytes is a direct target of the antiproteinuric effect of cyclosporine A
inferred← citeshttps://doi.org/10.1038/nm.3313openalex-snapshot
AKT2 is essential to maintain podocyte viability and function during chronic kidney disease
inferred← citeshttps://doi.org/10.1038/nm.4242openalex-snapshot
Bone marrow-derived immature myeloid cells are a main source of circulating suPAR contributing to proteinuric kidney disease
inferred← citeshttps://doi.org/10.1038/nm0111-44openalex-snapshot
Filtering new facts about kidney disease
inferred← citeshttps://doi.org/10.1038/nm0209-133bopenalex-snapshot
Is the LPS-mediated proteinuria mouse model relevant to human kidney disease?
inferred← citeshttps://doi.org/10.1038/nm1696openalex-snapshot
Modification of kidney barrier function by the urokinase receptor
inferred← citeshttps://doi.org/10.1038/nrneph.2009.70openalex-snapshot
Monoclonal antibodies for podocytopathies: rationale and clinical responses
inferred← citeshttps://doi.org/10.1038/nrneph.2013.276openalex-snapshot
Abate or applaud abatacept in proteinuric kidney disease?
inferred← citeshttps://doi.org/10.1038/nrneph.2015.178openalex-snapshot
New data do not support use of abatacept in diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/nrneph.2015.33openalex-snapshot
Genetics and pathogenesis of systemic lupus erythematosus and lupus nephritis
inferred← citeshttps://doi.org/10.1038/nrneph.2016.147openalex-snapshot
Minimal change disease and idiopathic FSGS: manifestations of the same disease
inferred← citeshttps://doi.org/10.1038/nrrheum.2015.159openalex-snapshot
What is damaging the kidney in lupus nephritis?
inferred← citeshttps://doi.org/10.1038/pr.2014.159openalex-snapshot
Effects of gluten-free, dairy-free diet on childhood nephrotic syndrome and gut microbiota
inferred← citeshttps://doi.org/10.1038/pr.2015.80openalex-snapshot
Risk of idiopathic nephrotic syndrome among children with asthma: a nationwide, population-based cohort study
inferred← citeshttps://doi.org/10.1038/pr.2018.11openalex-snapshot
Antiproteinuric effect of an endothelin-1 receptor antagonist in puromycin aminonucleoside-induced nephrosis in rat
inferred← citeshttps://doi.org/10.1038/s41418-019-0349-yopenalex-snapshot
Exosomal miRNA-19b-3p of tubular epithelial cells promotes M1 macrophage activation in kidney injury
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