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https://doi.org/10.2139/ssrn.3949876

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Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/eji.201040797openalex-extracted
Targeting SIRP‐α protects from type 2‐driven allergic airway inflammation
inferredcites →https://doi.org/10.1007/s00467-014-2915-3openalex-extracted
Urinary CD80 levels as a diagnostic biomarker of minimal change disease
inferredcites →https://doi.org/10.1007/s00467-015-3082-xopenalex-extracted
The glomerular permeability factors in idiopathic nephrotic syndrome
inferredcites →https://doi.org/10.1007/s00467-017-3613-8openalex-extracted
Toll-like receptor 3 (TLR-3), TLR-4 and CD80 expression in peripheral blood mononuclear cells and urinary CD80 levels in children with idiopathic nephrotic syndrome
inferredcites →https://doi.org/10.1007/s00467-017-3677-5openalex-extracted
Immunology of idiopathic nephrotic syndrome
inferredcites →https://doi.org/10.1007/s00467-018-3885-7openalex-extracted
Urinary CD80 excretion is a predictor of good outcome in children with primary nephrotic syndrome
inferredcites →https://doi.org/10.1007/s00467-020-04541-3openalex-extracted
Glomerular endothelial cells and podocytes can express CD80 in patients with minimal change disease during relapse
inferredcites →https://doi.org/10.1016/s0022-3476(86)80876-9openalex-extracted
Role of respiratory viruses in exacerbations of primary nephrotic syndrome
inferredcites →https://doi.org/10.1016/s0140-6736(18)30536-1openalex-extracted
Idiopathic nephrotic syndrome in children
inferredcites →https://doi.org/10.1016/s0140-6736(76)90600-0openalex-extracted
HLA ANTIGENS AND ATOPIC FEATURES IN STEROID-RESPONSIVE NEPHROTIC SYNDROME OF CHILDHOOD
inferredcites →https://doi.org/10.1016/s1521-6616(03)00042-1openalex-extracted
Surfactant protein D (SP-D) serum levels in patients with community-acquired pneumonia
inferredcites →https://doi.org/10.1038/ki.2008.19openalex-extracted
Decreased tyrosine phosphorylation of nephrin in rat and human nephrosis
inferredcites →https://doi.org/10.1038/ki.2010.143openalex-extracted
Urinary CD80 is elevated in minimal change disease but not in focal segmental glomerulosclerosis
inferredcites →https://doi.org/10.1038/nm.2261openalex-extracted
Podocyte-secreted angiopoietin-like-4 mediates proteinuria in glucocorticoid-sensitive nephrotic syndrome
inferredcites →https://doi.org/10.1038/nm.3396openalex-extracted
Circulating angiopoietin-like 4 links proteinuria with hypertriglyceridemia in nephrotic syndrome
inferredcites →https://doi.org/10.1038/s41598-017-00564-3openalex-extracted
Protein tyrosine phosphatase Shp2 deficiency in podocytes attenuates lipopolysaccharide-induced proteinuria
inferredcites →https://doi.org/10.1053/j.ajkd.2009.03.019openalex-extracted
Idiopathic Nephrotic Syndrome and Atopy: Is There a Common Link?
inferredcites →https://doi.org/10.1074/jbc.273.21.13223openalex-extracted
Integrin-mediated Tyrosine Phosphorylation of SHPS-1 and Its Association with SHP-2
inferredcites →https://doi.org/10.1074/jbc.m114.612721openalex-extracted
Increased SHP-1 Protein Expression by High Glucose Levels Reduces Nephrin Phosphorylation in Podocytes
inferredcites →https://doi.org/10.1074/jbc.m701565200openalex-extracted
Dual Regulation of SIRPα Phosphorylation by Integrins and CD47
inferredcites →https://doi.org/10.1093/ndt/18.1.70openalex-extracted
Altered ultrastructural distribution of nephrin in minimal change nephrotic syndrome
inferredcites →https://doi.org/10.1093/ndt/gfr271openalex-extracted
Toll-like receptor 3 ligands induce CD80 expression in human podocytes via an NF- B-dependent pathway
inferredcites →https://doi.org/10.1093/ndt/gfs543openalex-extracted
Toll-like receptor 3 ligand, polyIC, induces proteinuria and glomerular CD80, and increases urinary CD80 in mice
inferredcites →https://doi.org/10.1111/j.1442-200x.2011.03398.xopenalex-extracted
Serum KL‐6 and surfactant protein D in children with 2009 pandemic H1N1 influenza infection
inferredcites →https://doi.org/10.1128/mcb.00956-15openalex-extracted
Shp2 Associates with and Enhances Nephrin Tyrosine Phosphorylation and Is Necessary for Foot Process Spreading in Mouse Models of Podocyte Injury
inferredcites →https://doi.org/10.1152/ajprenal.00571.2009openalex-extracted
SIRP-α-CD47 system functions as an intercellular signal in the renal glomerulus
inferredcites →https://doi.org/10.1155/2011/376543openalex-extracted
Podocyte Protein, Nephrin, Is a Substrate of Protein Tyrosine Phosphatase 1B
inferredcites →https://doi.org/10.1164/rccm.200711-1661ocopenalex-extracted
Surfactant Proteins A and D Suppress Alveolar Macrophage Phagocytosis via Interaction with SIRPα
inferredcites →https://doi.org/10.1172/jci20402openalex-extracted
Induction of B7-1 in podocytes is associated with nephrotic syndrome
inferredcites →https://doi.org/10.1172/jci60070openalex-extracted
Crk1/2-dependent signaling is necessary for podocyte foot process spreading in mouse models of glomerular disease
inferredcites →https://doi.org/10.1183/09031936.00156508openalex-extracted
Serum surfactant protein D is steroid sensitive and associated with exacerbations of COPD
inferredcites →https://doi.org/10.1681/asn.2007080836openalex-extracted
Urinary CD80 Excretion Increases in Idiopathic Minimal-Change Disease
inferredcites →https://doi.org/10.1681/asn.2009090991openalex-extracted
Inhibition of Podocyte FAK Protects against Proteinuria and Foot Process Effacement
inferredcites →https://doi.org/10.1681/asn.v133630openalex-extracted
A Conditionally Immortalized Human Podocyte Cell Line Demonstrating Nephrin and Podocin Expression
inferredcites →https://doi.org/10.2174/138161206775474387openalex-extracted
Pulmonary Surfactant Proteins A and D: Innate Immune Functions and Biomarkers for Lung Diseases
inferredcites →https://doi.org/10.2215/cjn.05000516openalex-extracted
Minimal Change Disease
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