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Increased ER–mitochondrial coupling promotes mitochondrial respiration and bioenergetics during early phases of ER stress
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/9780470725207.ch8openalex-snapshot
Calcium Signalling and Mitochondrial Motility
inferredcites →https://doi.org/10.1007/s10495-009-0341-yopenalex-snapshot
Cellular responses to endoplasmic reticulum stress and apoptosis
inferredcites →https://doi.org/10.1007/s11010-005-2996-1openalex-snapshot
Effect of GRP75/mthsp70/PBP74/mortalin overexpression on intracellular ATP level, mitochondrial membrane potential and ROS accumulation following glucose deprivation in PC12 cells
inferredcites →https://doi.org/10.1016/j.bbabio.2009.05.011openalex-snapshot
The role of Ca2+ signaling in the coordination of mitochondrial ATP production with cardiac work
inferredcites →https://doi.org/10.1016/j.bbamcr.2006.02.014openalex-snapshot
Interactions of mitochondria with the actin cytoskeleton
inferredcites →https://doi.org/10.1016/j.biocel.2009.02.017openalex-snapshot
Interactions between the endoplasmic reticulum, mitochondria, plasma membrane and other subcellular organelles
inferredcites →https://doi.org/10.1016/j.ceca.2006.08.016openalex-snapshot
Mitochondrial calcium signalling and cell death: Approaches for assessing the role of mitochondrial Ca2+ uptake in apoptosis
inferredcites →https://doi.org/10.1016/j.cell.2006.07.019openalex-snapshot
Rough Sheets and Smooth Tubules
inferredcites →https://doi.org/10.1016/j.cell.2010.07.007openalex-snapshot
Calcium and Energy: Making the Cake and Eating It too?
inferredcites →https://doi.org/10.1016/j.febslet.2004.04.059openalex-snapshot
Participation of endoplasmic reticulum and mitochondrial calcium handling in apoptosis: more than just neighborhood?
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2007.12.017openalex-snapshot
Ceramide-induced formation of ROS and ATP depletion trigger necrosis in lymphoid cells
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2009.11.017openalex-snapshot
Iron induces protection and necrosis in cultured cardiomyocytes: Role of reactive oxygen species and nitric oxide
inferredcites →https://doi.org/10.1016/j.mam.2004.03.001openalex-snapshot
Mitochondria in health and disease: perspectives on a new mitochondrial biology
inferredcites →https://doi.org/10.1016/j.molcel.2008.11.014openalex-snapshot
Role of SERCA1 Truncated Isoform in the Proapoptotic Calcium Transfer from ER to Mitochondria during ER Stress
inferredcites →https://doi.org/10.1016/j.molcel.2009.08.021openalex-snapshot
Fine-Tuning of the Unfolded Protein Response: Assembling the IRE1α Interactome
inferredcites →https://doi.org/10.1016/j.tcb.2008.12.002openalex-snapshot
MAM: more than just a housekeeper
inferredcites →https://doi.org/10.1016/s0021-9258(19)86107-0openalex-snapshot
The effects of cytochalasins on actin polymerization and actin ATPase provide insights into the mechanism of polymerization.
inferredcites →https://doi.org/10.1016/s0022-3565(25)31589-2openalex-snapshot
Nocodazole action on tubulin assembly, axonal ultrastructure and fast axoplasmic transport.
inferredcites →https://doi.org/10.1016/s0143416002001823openalex-snapshot
The endoplasmic reticulum: a multifunctional signaling organelle
inferredcites →https://doi.org/10.1016/s0167-4889(97)00009-8openalex-snapshot
Induction of the stress protein Grp75 by amino acid deprivation in CHO cells does not involve an increase in Grp75 mRNA levels
inferredcites →https://doi.org/10.1038/293302a0openalex-snapshot
Cytochalasin D inhibits actin polymerization and induces depolymerization of actin filaments formed during platelet shape change
inferredcites →https://doi.org/10.1038/36285openalex-snapshot
Mutation of the mouse klotho gene leads to a syndrome resembling ageing.
inferredcites →https://doi.org/10.1038/47513openalex-snapshot
Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-β
inferredcites →https://doi.org/10.1038/embor.2010.92openalex-snapshot
Further assembly required: construction and dynamics of the endoplasmic reticulum network
inferredcites →https://doi.org/10.1038/ja.2007.62openalex-snapshot
Biosynthesis of the Tunicamycins: A Review
inferredcites →https://doi.org/10.1038/nature07534openalex-snapshot
Mitofusin 2 tethers endoplasmic reticulum to mitochondria
inferredcites →https://doi.org/10.1038/nrm2199openalex-snapshot
Signal integration in the endoplasmic reticulum unfolded protein response
inferredcites →https://doi.org/10.1038/onc.2008.63openalex-snapshot
Hierarchical involvement of Bak, VDAC1 and Bax in cisplatin-induced cell death
inferredcites →https://doi.org/10.1038/sj.emboj.7600559openalex-snapshot
PACS‐2 controls endoplasmic reticulum–mitochondria communication and Bid‐mediated apoptosis
inferredcites →https://doi.org/10.1042/bj2840001openalex-snapshot
Control of respiration and ATP synthesis in mammalian mitochondria and cells
inferredcites →https://doi.org/10.1042/bj3060445openalex-snapshot
Control of inositol 1,4,5-trisphosphate-induced Ca2+ release by cytosolic Ca2+
inferredcites →https://doi.org/10.1073/pnas.0709747104openalex-snapshot
Beyond aerobic glycolysis: Transformed cells can engage in glutamine metabolism that exceeds the requirement for protein and nucleotide synthesis
inferredcites →https://doi.org/10.1073/pnas.0802076105openalex-snapshot
Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress
inferredcites →https://doi.org/10.1074/jbc.m110.101584openalex-snapshot
Rab32 Modulates Apoptosis Onset and Mitochondria-associated Membrane (MAM) Properties
inferredcites →https://doi.org/10.1074/jbc.m212754200openalex-snapshot
Mitofusin-2 Determines Mitochondrial Network Architecture and Mitochondrial Metabolism
inferredcites →https://doi.org/10.1074/jbc.m311698200openalex-snapshot
Effects of Secretagogues and Bile Acids on Mitochondrial Membrane Potential of Pancreatic Acinar Cells
inferredcites →https://doi.org/10.1074/jbc.m703812200openalex-snapshot
Mitofusin 2 Protects Cerebellar Granule Neurons against Injury-induced Cell Death
inferredcites →https://doi.org/10.1074/jbc.m800986200openalex-snapshot
The Reticulon and Dp1/Yop1p Proteins Form Immobile Oligomers in the Tubular Endoplasmic Reticulum
inferredcites →https://doi.org/10.1080/02713680600966785openalex-snapshot
Induction of Endoplasmic Reticulum Stress in Retinal Pericytes by Glucose Deprivation
inferredcites →https://doi.org/10.1083/jcb.200406038openalex-snapshot
Control of mitochondrial motility and distribution by the calcium signal
inferredcites →https://doi.org/10.1083/jcb.200604016openalex-snapshot
Structural and functional features and significance of the physical linkage between ER and mitochondria
inferredcites →https://doi.org/10.1083/jcb.200608073openalex-snapshot
Chaperone-mediated coupling of endoplasmic reticulum and mitochondrial Ca2+ channels
inferredcites →https://doi.org/10.1083/jcb.200911024openalex-snapshot
ER sliding dynamics and ER–mitochondrial contacts occur on acetylated microtubules
inferredcites →https://doi.org/10.1089/ars.2006.8.1391openalex-snapshot
The Endoplasmic Reticulum: Folding, Calcium Homeostasis, Signaling, and Redox Control
inferredcites →https://doi.org/10.1093/cvr/cvm029openalex-snapshot
Changes in mitochondrial dynamics during ceramide-induced cardiomyocyte early apoptosis
inferredcites →https://doi.org/10.1093/jb/mvp194openalex-snapshot
Mechanism and components of endoplasmic reticulum-associated degradation
inferredcites →https://doi.org/10.1111/j.1349-7006.2010.01525.xopenalex-snapshot
Mitochondria regulate the unfolded protein response leading to cancer cell survival under glucose deprivation conditions
inferredcites →https://doi.org/10.1111/j.1365-2818.1993.tb03313.xopenalex-snapshot
Measurement of co‐localization of objects in dual‐colour confocal images
inferredcites →https://doi.org/10.1111/j.1600-0854.2007.00644.xopenalex-snapshot
Moving Mitochondria: Establishing Distribution of an Essential Organelle
inferredcites →https://doi.org/10.1111/j.1745-7270.2008.00409.xopenalex-snapshot
Glucose-regulated protein 75 suppresses apoptosis induced by glucose deprivation in PC12 cells through inhibition of Bax conformational change
inferredcites →https://doi.org/10.1126/science.280.5370.1763openalex-snapshot
Close Contacts with the Endoplasmic Reticulum as Determinants of Mitochondrial Ca 2+ Responses
inferredcites →https://doi.org/10.1152/ajpcell.00402.2010openalex-snapshot
Fragmented mitochondria are sensitized to Bax insertion and activation during apoptosis
inferredcites →https://doi.org/10.1152/physiol.00046.2007openalex-snapshot
Mitochondria: The Hub of Cellular Ca2+Signaling
inferredcites →https://doi.org/10.1242/jcs.02745openalex-snapshot
The axonal transport of mitochondria
inferredcites →https://doi.org/10.1242/jcs.03486openalex-snapshot
Reversible interactions between smooth domains of the endoplasmic reticulum and mitochondria are regulated by physiological cytosolic Ca2+ levels
inferredcites →https://doi.org/10.1242/jcs.053850openalex-snapshot
The axonal transport of mitochondria
inferredcites →https://doi.org/10.1523/jneurosci.0060-05.2005openalex-snapshot
Neurotoxic Calcium Transfer from Endoplasmic Reticulum to Mitochondria Is Regulated by Cyclin-Dependent Kinase 5-Dependent Phosphorylation of Tau
inferredcites →https://doi.org/10.1529/biophysj.103.038422openalex-snapshot
Automatic and Quantitative Measurement of Protein-Protein Colocalization in Live Cells
inferredcites →https://doi.org/10.2353/ajpath.2009.080650openalex-snapshot
The Krebs Cycle and Mitochondrial Mass Are Early Victims of Endothelial Dysfunction
inferredcites →https://doi.org/10.5507/bp.2005.028openalex-snapshot
MICROTUBULE DISRUPTORS AND THEIR INTERACTION WITH BIOTRANSFORMATION ENZYMES
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4648361openalex-extracted
eIF2α Phosphorylation-Mediated Transcriptional Reprogramming Suppresses Impairment of Mitochondrial and GSH Homeostasis and Thereby Ferroptosis During ER Stress
inferred← citeshttps://doi.org/10.1001/jamaneurol.2013.3242openalex-snapshot
COX10Mutations Resulting in Complex Multisystem Mitochondrial Disease That Remains Stable Into Adulthood
inferred← citeshttps://doi.org/10.1002/9781119432463.ch14openalex-snapshot
Molecular Mechanisms Underlying Oxytosis
inferred← citeshttps://doi.org/10.1002/adfm.202424108openalex-snapshot
An Endoplasmic Reticulum‐Targeting and Calcium Metabolism‐Disrupting Nanodrug Enhances Tumor Photodynamic Immunotherapy
inferred← citeshttps://doi.org/10.1002/alz.14417openalex-snapshot
Stabilization of mitochondria‐associated endoplasmic reticulum membranes regulates Aβ generation in a three‐dimensional neural model of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/biof.1673openalex-snapshot
Mitochondrial dysfunction and endoplasmic reticulum stress in the promotion of fibrosis in obstructive nephropathy induced by unilateral ureteral obstruction
inferred← citeshttps://doi.org/10.1002/btpr.1730openalex-snapshot
Chinese hamster ovary K1 host cell enables stable cell line development for antibody molecules which are difficult to express in DUXB11‐derived dihydrofolate reductase deficient host cell
inferred← citeshttps://doi.org/10.1002/cbin.11103openalex-snapshot
ER stress differentially affects pro‐inflammatory changes induced by mitochondrial dysfunction in the human monocytic leukemia cell line, THP‐1
inferred← citeshttps://doi.org/10.1002/cphy.c160013openalex-snapshot
Calcium Transport and Signaling in Mitochondria
inferred← citeshttps://doi.org/10.1002/cphy.c230009openalex-snapshot
Mitochondrial Function and Dysfunction in White Adipocytes and Therapeutic Implications
inferred← citeshttps://doi.org/10.1002/fsn3.70637openalex-snapshot
Piperine Enhances Mitochondrial Biogenesis to Mitigate Stress in SH ‐ SY5Y Neuroblastoma Cells
inferred← citeshttps://doi.org/10.1002/iub.2306openalex-snapshot
Let‐7b‐5p is involved in the response of endoplasmic reticulum stress in acute pulmonary embolism through upregulating the expression of stress‐associated endoplasmic reticulum protein 1
inferred← citeshttps://doi.org/10.1002/j.2040-4603.2017.tb00748.xopenalex-snapshot
Calcium Transport and Signaling in Mitochondria
inferred← citeshttps://doi.org/10.1002/j.2040-4603.2024.tb00302.xopenalex-snapshot
Mitochondrial Function and Dysfunction in White Adipocytes and Therapeutic Implications
inferred← citeshttps://doi.org/10.1002/jcb.26031openalex-snapshot
Endoplasmic Reticulum Stress in Mice Increases Hepatic Expression of Genes Carrying a Premature Termination Codon via a Nutritional Status‐Independent GRP78‐Dependent Mechanism
inferred← citeshttps://doi.org/10.1002/jcp.24360openalex-snapshot
Functional and morphological impact of ER stress on mitochondria
inferred← citeshttps://doi.org/10.1002/jcp.25292openalex-snapshot
Effects of a Sublethal and Transient Stress of the Endoplasmic Reticulum on the Mitochondrial Population
inferred← citeshttps://doi.org/10.1002/jcp.29623openalex-snapshot
A mutation in the Na‐K‐2Cl cotransporter‐1 leads to changes in cellular metabolism
inferred← citeshttps://doi.org/10.1002/jcp.31058openalex-snapshot
Epigenetic regulation of mitochondrial‐endoplasmic reticulum dynamics in kidney diseases
inferred← citeshttps://doi.org/10.1002/jcp.31204openalex-snapshot
ATF4‐dependent increase in mitochondrial‐endoplasmic reticulum tethering following OPA1 deletion in skeletal muscle
inferred← citeshttps://doi.org/10.1002/jnr.23541openalex-snapshot
Adaptive endoplasmic reticulum stress alters cellular responses to the extracellular milieu
inferred← citeshttps://doi.org/10.1002/mco2.70259openalex-snapshot
Mitochondria‐Associated Endoplasmic Reticulum Membranes in Human Health and Diseases
inferred← citeshttps://doi.org/10.1002/mco2.70379openalex-snapshot
Mitochondria‐Associated Endoplasmic Reticulum Membranes in Health and Diseases
inferred← citeshttps://doi.org/10.1002/mds.29525openalex-snapshot
Mitochondria–Endoplasmic Reticulum Contact Sites Dynamics and Calcium Homeostasis Are Differentially Disrupted in PINK1 ‐PD or PRKN ‐PD Neurons
inferred← citeshttps://doi.org/10.1002/mnfr.201900942openalex-snapshot
Mitochondrial Dysfunction is a Key Pathway that Links Saturated Fat Intake to the Development and Progression of NAFLD
inferred← citeshttps://doi.org/10.1002/mnfr.202001076openalex-snapshot
Sulforaphane Balances Ca 2+ Homeostasis Injured by Excessive Fat via Mitochondria‐Associated Membrane (MAM)
inferred← citeshttps://doi.org/10.1002/path.3977openalex-snapshot
Endoplasmic reticulum stress: a new playER in tauopathies
inferred← citeshttps://doi.org/10.1002/prca.201400030openalex-snapshot
Proteomics in neurodegenerative diseases: Methods for obtaining a closer look at the neuronal proteome
inferred← citeshttps://doi.org/10.1007/82_2017_2openalex-snapshot
Mitochondria-Associated Membranes and ER Stress
inferred← citeshttps://doi.org/10.1007/978-1-4939-6824-4_5openalex-snapshot
Isolation of Mitochondria-Associated Membranes (MAM) from Mouse Brain Tissue
inferred← citeshttps://doi.org/10.1007/978-1-62703-770-9_8openalex-snapshot
Endoplasmic Reticulum Stress and Adipose Tissue Function
inferred← citeshttps://doi.org/10.1007/978-3-030-12457-1_14openalex-snapshot
Sarco-Endoplasmic Reticulum Calcium Release Model Based on Changes in the Luminal Calcium Content
inferred← citeshttps://doi.org/10.1007/978-3-031-14740-1_9openalex-snapshot
Co-chaperones of the Human Endoplasmic Reticulum: An Update
inferred← citeshttps://doi.org/10.1007/978-3-319-15539-5_5openalex-snapshot
Oxidative Stress and the Unfolded Protein Response in the Liver
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Mitochondrial Ca2+ Handling and Behind: The Importance of Being in Contact with Other Organelles
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The Role of Mitochondria in the Activation/Maintenance of SOCE: Membrane Contact Sites as Signaling Hubs Sustaining Store-Operated Ca2+ Entry
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Endoplasmic Reticulum-mitochondria connections, calcium cross-talk and cell fate: a closer inspection
inferred← citeshttps://doi.org/10.1007/978-981-10-4567-7_13openalex-snapshot
Role of Endoplasmic Reticulum-Mitochondria Communication in Type 2 Diabetes
inferred← citeshttps://doi.org/10.1007/978-981-10-4567-7_2openalex-snapshot
Over Six Decades of Discovery and Characterization of the Architecture at Mitochondria-Associated Membranes (MAMs)
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Endoplasmic Reticulum-Mitochondria Communication Through Ca2+ Signaling: The Importance of Mitochondria-Associated Membranes (MAMs)
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Endoplasmic Reticulum Stress: Importance in Pathogenesis of Mycobacterium tuberculosis
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Intracellular organelles in the saga of Ca2+ homeostasis: different molecules for different purposes?
inferred← citeshttps://doi.org/10.1007/s00018-022-04189-2openalex-snapshot
PACS2–TRPV1 axis is required for ER–mitochondrial tethering during ER stress and lung fibrosis
inferred← citeshttps://doi.org/10.1007/s00018-022-04211-7openalex-snapshot
ER-mitochondria communication is involved in NLRP3 inflammasome activation under stress conditions in the innate immune system
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Curcumin and NCLX inhibitors share anti-tumoral mechanisms in microsatellite-instability-driven colorectal cancer
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Redox regulation of UPR signalling and mitochondrial ER contact sites
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Disruption of calcium transfer from ER to mitochondria links alterations of mitochondria-associated ER membrane integrity to hepatic insulin resistance
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Endoplasmic reticulum–mitochondria contact sites are signalling hubs connecting nutrient sensing and GLP-1 secretion in L cells of the mouse gut: from physiology to obesity and type 2 diabetes
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PolyGA targets the ER stress-adaptive response by impairing GRP75 function at the MAM in C9ORF72-ALS/FTD
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ER stress–related protein, CHOP, may serve as a biomarker of mechanical asphyxia: a primary study
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Sarcopenia in pulmonary diseases is associated with elevated sarcoplasmic reticulum stress and myonuclear disorganization
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Superresolution imaging of viral protein trafficking
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Leishmania donovani chaperonin 10 regulates parasite internalization and intracellular survival in human macrophages
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Impact of Calcium Influx on Endoplasmic Reticulum in Excitotoxic Neurons: Role of Chemical Chaperone 4-PBA
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Mitochondrial cholesterol: mechanisms of import and effects on mitochondrial function
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The non-apoptotic action of Bcl-xL: regulating Ca2+ signaling and bioenergetics at the ER-mitochondrion interface
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Endoplasmic reticulum stress induces mitochondrial dysfunction but not mitochondrial unfolded protein response in SH-SY5Y cells
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Trimethylamine N-oxide activates cardiac fibroblasts transformation through NLRP3 inflammasome activation with changes in mitochondrial dynamics and ER-mitochondrial contact under ER stress
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Mitochondria, Myocardial Remodeling, and Cardiovascular Disease
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Endoplasmic Reticulum Stress in Hypertension and Salt Sensitivity of Blood Pressure
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Phosphatase 2A Inhibition Affects Endoplasmic Reticulum and Mitochondria Homeostasis Via Cytoskeletal Alterations in Brain Endothelial Cells
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Mitochondria-Associated Membranes (MAMs): Overview and Its Role in Parkinson’s Disease
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The Endoplasmic Reticulum Stress Response in Neuroprogressive Diseases: Emerging Pathophysiological Role and Translational Implications
inferred← citeshttps://doi.org/10.1007/s12035-020-01929-yopenalex-snapshot
Molecular Mechanisms of ER Stress and UPR in the Pathogenesis of Alzheimer’s Disease
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Exploring mitochondrial dynamics and localization under cellular stress: A comprehensive insight
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Getting the better of ER stress
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Role of Endoplasmic Reticulum Stress and Unfolded Protein Responses in Health and Diseases
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Homocysteine activates T cells by enhancing endoplasmic reticulum-mitochondria coupling and increasing mitochondrial respiration
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ER Stress and Autophagy in Obesity and Nonalcoholic Fatty Liver Disease
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Comprehending the Unfolded Protein Response as a Conduit for Improved Mesenchymal Stem Cell-Based Therapeutics
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Mitochondrial Network Enhancing (MiNE) Nanoparticles for the Treatment of Neurodegenerative Disease and the Aging Brain
inferred← citeshttps://doi.org/10.1016/b978-0-12-386033-0.00002-5openalex-snapshot
New Insights into the Role of Mitochondria-Associated Endoplasmic Reticulum Membrane
inferred← citeshttps://doi.org/10.1016/b978-0-12-407704-1.00005-1openalex-snapshot
Endoplasmic Reticulum and the Unfolded Protein Response
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Membrane organization | Topography and Functions of Membrane Contact Sites
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Mitochondrial metabolism and carcinogenesis
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Mitochondria and allergo-inflammation
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Mitochondria in immune cells: their role in immunity, inflammation, and metabolism
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Pore-forming toxins from sea anemones: from protein-membrane interaction to its implications for developing biomedical applications
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Mitochondria-associated membranes (MAMs) in age-related heart diseases, role of endoplasmic reticulum stress
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Sarcoplasmic reticulum and calcium signaling in muscle cells: Homeostasis and disease
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The ER-mitochondria Ca2+ signaling in cancer progression: Fueling the monster
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Role of mitochondria-associated endoplasmic reticulum membrane (MAMs) interactions and calcium exchange in the development of type 2 diabetes
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Targeting Mitochondria-Derived Reactive Oxygen Species to Reduce Epithelial Barrier Dysfunction and Colitis
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Control of mitochondria-associated endoplasmic reticulum membranes by protein S-palmitoylation: Novel therapeutic targets for neurodegenerative diseases
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Dysfunctional mitochondria as critical players in the inflammation of autoimmune diseases: Potential role in Sjögren’s syndrome
inferred← citeshttps://doi.org/10.1016/j.bbabio.2016.03.018openalex-snapshot
Mitochondrial reactive oxygen species: Do they extend or shorten animal lifespan?
inferred← citeshttps://doi.org/10.1016/j.bbadis.2013.04.009openalex-snapshot
Calcium and mitochondrial metabolism in ceramide-induced cardiomyocyte death
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ER-to-mitochondria miscommunication and metabolic diseases
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Biological insights in the pathogenesis of hypermobile Ehlers-Danlos syndrome from proteome profiling of patients' dermal myofibroblasts
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Mitoregulin controls mitochondrial function and stress-adaptation response during early phase of endoplasmic reticulum stress in breast cancer cells
inferred← citeshttps://doi.org/10.1016/j.bbadis.2024.167531openalex-snapshot
Upregulation of GRP78 is accompanied by decreased antioxidant response and mitophagy promotion in streptozotocin-induced type 1 diabetes in rats
inferred← citeshttps://doi.org/10.1016/j.bbalip.2016.08.012openalex-snapshot
Mitochondrial cholesterol import
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2011.06.011openalex-snapshot
Urban planning of the endoplasmic reticulum (ER): How diverse mechanisms segregate the many functions of the ER
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2012.04.013openalex-snapshot
Where the endoplasmic reticulum and the mitochondrion tie the knot: The mitochondria-associated membrane (MAM)
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2013.01.026openalex-snapshot
Regulation of inositol 1,4,5-trisphosphate receptors during endoplasmic reticulum stress
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2013.05.018openalex-snapshot
The role of phospholipids in the biological activity and structure of the endoplasmic reticulum
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2013.07.024openalex-snapshot
When ER stress reaches a dead end
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2013.09.006openalex-snapshot
Herp depletion protects from protein aggregation by up-regulating autophagy
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2014.03.009openalex-snapshot
New functions of mitochondria associated membranes in cellular signaling
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2015.01.022openalex-snapshot
Calcium signaling at ER membrane contact sites
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2017.01.001openalex-snapshot
Endoplasmic reticulum-mitochondria calcium signaling in hepatic metabolic diseases
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2017.01.009openalex-snapshot
The UPR reduces glucose metabolism via IRE1 signaling
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2018.10.016openalex-snapshot
Calcium, mitochondria and cell metabolism: A functional triangle in bioenergetics
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2021.118989openalex-snapshot
Characterization of mitochondrial calpain-5
inferred← citeshttps://doi.org/10.1016/j.bbiosy.2021.100027openalex-snapshot
Bioabsorbable metal zinc differentially affects mitochondria in vascular endothelial and smooth muscle cells
inferred← citeshttps://doi.org/10.1016/j.bbrc.2014.03.004openalex-snapshot
GLP-1 promotes mitochondrial metabolism in vascular smooth muscle cells by enhancing endoplasmic reticulum–mitochondria coupling
inferred← citeshttps://doi.org/10.1016/j.bbrc.2016.07.053openalex-snapshot
GILZ overexpression attenuates endoplasmic reticulum stress-mediated cell death via the activation of mitochondrial oxidative phosphorylation
inferred← citeshttps://doi.org/10.1016/j.bbrc.2016.07.056openalex-snapshot
(Neuro)degenerated Mitochondria-ER contacts
inferred← citeshttps://doi.org/10.1016/j.bbrc.2017.06.097openalex-snapshot
Mitochondria-associated membranes (MAMs): An emerging platform connecting energy and immune sensing to metabolic flexibility
inferred← citeshttps://doi.org/10.1016/j.bbrc.2018.07.053openalex-snapshot
A new model to study cell-to-cell transfer of αSynuclein in vivo
inferred← citeshttps://doi.org/10.1016/j.bcp.2022.115068openalex-snapshot
Molecular dissection of cellular response of pancreatic islet cells to Bisphenol-A (BPA): A comprehensive review
inferred← citeshttps://doi.org/10.1016/j.biocel.2011.10.012openalex-snapshot
Endoplasmic reticulum: ER stress regulates mitochondrial bioenergetics
inferred← citeshttps://doi.org/10.1016/j.biocel.2012.06.008openalex-snapshot
Targeting mitochondria in the infection strategy of the hepatitis C virus
inferred← citeshttps://doi.org/10.1016/j.biocel.2014.01.019openalex-snapshot
Organelle communication: Signaling crossroads between homeostasis and disease
inferred← citeshttps://doi.org/10.1016/j.biochi.2018.02.006openalex-snapshot
Sticholysin II-mediated cytotoxicity involves the activation of regulated intracellular responses that anticipates cell death
inferred← citeshttps://doi.org/10.1016/j.bja.2024.01.007openalex-snapshot
Endoplasmic reticular stress as an emerging therapeutic target for chronic pain: a narrative review
inferred← citeshttps://doi.org/10.1016/j.bpj.2014.03.025openalex-snapshot
Automated Identification of Subcellular Organelles by Coherent Anti-Stokes Raman Scattering
inferred← citeshttps://doi.org/10.1016/j.ceb.2013.03.006openalex-snapshot
Kαλóς και Aγαθóς: how mitochondrial beauty translates into biological virtue
inferred← citeshttps://doi.org/10.1016/j.ceb.2018.04.014openalex-snapshot
Plastic mitochondria-endoplasmic reticulum (ER) contacts use chaperones and tethers to mould their structure and signaling
inferred← citeshttps://doi.org/10.1016/j.ceca.2016.05.001openalex-snapshot
Subcellular ROS imaging methods: Relevance for the study of calcium signaling
inferred← citeshttps://doi.org/10.1016/j.ceca.2017.01.003openalex-snapshot
The endoplasmic reticulum-mitochondria coupling in health and disease: Molecules, functions and significance
inferred← citeshttps://doi.org/10.1016/j.ceca.2017.05.003openalex-snapshot
Mitochondrial and endoplasmic reticulum calcium homeostasis and cell death
inferred← citeshttps://doi.org/10.1016/j.ceca.2017.05.015openalex-snapshot
Endoplasmic reticulum chaperones tweak the mitochondrial calcium rheostat to control metabolism and cell death
inferred← citeshttps://doi.org/10.1016/j.ceca.2017.08.004openalex-snapshot
Calcium signaling at the endoplasmic reticulum: fine-tuning stress responses
inferred← citeshttps://doi.org/10.1016/j.ceca.2017.08.005openalex-snapshot
The regulation of autophagy by calcium signals: Do we have a consensus?
inferred← citeshttps://doi.org/10.1016/j.ceca.2022.102688openalex-snapshot
Heterogeneity of the endoplasmic reticulum Ca2+ store determines colocalization with mitochondria
inferred← citeshttps://doi.org/10.1016/j.ceca.2023.102780openalex-snapshot
Calcium signalling in hepatic metabolism: Health and diseases
inferred← citeshttps://doi.org/10.1016/j.ceca.2024.102961openalex-snapshot
ER stress as a sentinel mechanism for ER Ca2+ homeostasis
inferred← citeshttps://doi.org/10.1016/j.cellsig.2013.10.004openalex-snapshot
Yessotoxin induces ER-stress followed by autophagic cell death in glioma cells mediated by mTOR and BNIP3
inferred← citeshttps://doi.org/10.1016/j.cellsig.2015.10.013openalex-snapshot
CCN1/CYR61 overexpression in hepatic stellate cells induces ER stress-related apoptosis
inferred← citeshttps://doi.org/10.1016/j.cellsig.2018.07.005openalex-snapshot
Portal myofibroblasts are sensitive to CCN-mediated endoplasmic reticulum stress-related apoptosis with potential to attenuate biliary fibrogenesis
inferred← citeshttps://doi.org/10.1016/j.celrep.2014.03.051openalex-snapshot
SPG7 Variant Escapes Phosphorylation-Regulated Processing by AFG3L2, Elevates Mitochondrial ROS, and Is Associated with Multiple Clinical Phenotypes
inferred← citeshttps://doi.org/10.1016/j.celrep.2016.05.013openalex-snapshot
Presenilin 2 Modulates Endoplasmic Reticulum-Mitochondria Coupling by Tuning the Antagonistic Effect of Mitofusin 2
inferred← citeshttps://doi.org/10.1016/j.celrep.2020.108125openalex-snapshot
Atf-6 Regulates Lifespan through ER-Mitochondrial Calcium Homeostasis
inferred← citeshttps://doi.org/10.1016/j.celrep.2022.111899openalex-snapshot
The endoplasmic reticulum kinase PERK interacts with the oxidoreductase ERO1 to metabolically adapt mitochondria
inferred← citeshttps://doi.org/10.1016/j.celrep.2023.112209openalex-snapshot
VMP1 affects endoplasmic reticulum stress sensitivity via differential modulation of the three unfolded protein response arms
inferred← citeshttps://doi.org/10.1016/j.celrep.2025.116486openalex-snapshot
The ER thioredoxin-related transmembrane protein TMX2 controls redox-mediated tethering of ER-mitochondria contacts
inferred← citeshttps://doi.org/10.1016/j.celrep.2026.117021openalex-snapshot
Targeting UGCG sensitizes AML cells to venetoclax through RAB32-mediated endoplasmic reticulum-mitochondria communication
inferred← citeshttps://doi.org/10.1016/j.cmet.2015.04.010openalex-snapshot
snoRNA U17 Regulates Cellular Cholesterol Trafficking
inferred← citeshttps://doi.org/10.1016/j.cmet.2015.06.010openalex-snapshot
Calcium Homeostasis and Organelle Function in the Pathogenesis of Obesity and Diabetes
inferred← citeshttps://doi.org/10.1016/j.cophys.2020.08.010openalex-snapshot
Regulation of Ca2+ exchanges and signaling in mitochondria
inferred← citeshttps://doi.org/10.1016/j.cophys.2020.08.012openalex-snapshot
Pathophysiological significance of calcium signaling at Mitochondria-Associated Endoplasmic Reticulum Membranes (MAMs)
inferred← citeshttps://doi.org/10.1016/j.cophys.2020.08.013openalex-snapshot
The ER-mitochondria tether at the hub of Ca2+ signaling
inferred← citeshttps://doi.org/10.1016/j.critrevonc.2025.104899openalex-snapshot
Mitochondrial homeostasis in cancer: Functions and targeted therapies
inferred← citeshttps://doi.org/10.1016/j.cub.2017.11.040openalex-snapshot
Organelles: The Emerging Signalling Chart of Mitochondrial Dynamics
inferred← citeshttps://doi.org/10.1016/j.diabet.2015.02.006openalex-snapshot
Contribution of mitochondria and endoplasmic reticulum dysfunction in insulin resistance: Distinct or interrelated roles?
inferred← citeshttps://doi.org/10.1016/j.drudis.2021.07.011openalex-snapshot
Hypoxia and the integrated stress response promote pulmonary hypertension and preeclampsia: Implications in drug development
inferred← citeshttps://doi.org/10.1016/j.ebiom.2019.04.044openalex-snapshot
DsbA-L ameliorates high glucose induced tubular damage through maintaining MAM integrity
inferred← citeshttps://doi.org/10.1016/j.ecoenv.2023.114517openalex-snapshot
Endoplasmic reticulum stress mediated by ROS participates in cadmium exposure-induced MC3T3-E1 cell apoptosis
inferred← citeshttps://doi.org/10.1016/j.ecoenv.2024.116679openalex-snapshot
Impact of DEHP on mitochondria-associated endoplasmic reticulum membranes and reproductive toxicity in ovary
inferred← citeshttps://doi.org/10.1016/j.ecoenv.2025.118396openalex-snapshot
Ca2+ transfer via enhancing ER-Mito coupling contributed to BDE-47- induced hippocampal neuronal necroptosis and cognitive dysfunction
inferred← citeshttps://doi.org/10.1016/j.ejcb.2021.151180openalex-snapshot
Specific subdomain localization of ER resident proteins and membrane contact sites resolved by electron microscopy
inferred← citeshttps://doi.org/10.1016/j.ejmech.2025.118122openalex-snapshot
Targeting calreticulin (CALR) in tumors: Cellular mechanisms, structural insights and ligand development advances
inferred← citeshttps://doi.org/10.1016/j.ejphar.2013.11.012openalex-snapshot
Calcium channelopathies and Alzheimer's disease: Insight into therapeutic success and failures
inferred← citeshttps://doi.org/10.1016/j.envpol.2022.119756openalex-snapshot
Glycine ameliorates MBP-induced meiotic abnormalities and apoptosis by regulating mitochondrial-endoplasmic reticulum interactions in porcine oocytes
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2014.01.004openalex-snapshot
Oxidative stress induces mitochondrial dysfunction and a protective unfolded protein response in RPE cells
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2016.04.012openalex-snapshot
Mitochondrial control of cell bioenergetics in Parkinson’s disease
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2023.11.022openalex-snapshot
HERPUD1 governs tumor cell mitochondrial function via inositol 1,4,5-trisphosphate receptor-mediated calcium signaling
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2026.10.014openalex-snapshot
Endoplasmic reticulum stress, redox signaling, and neutrophil effector functions in chronic lung diseases: Evidence and research priorities
inferred← citeshttps://doi.org/10.1016/j.ibneur.2023.02.005openalex-snapshot
Neuroprotective properties of anti-apoptotic BCL-2 proteins in 5xFAD mouse model of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.immuni.2018.02.012openalex-snapshot
Mitochondria-Endoplasmic Reticulum Contact Sites Function as Immunometabolic Hubs that Orchestrate the Rapid Recall Response of Memory CD8+ T Cells
inferred← citeshttps://doi.org/10.1016/j.intimp.2024.112930openalex-snapshot
Unraveling the complex interplay between Mitochondria-Associated Membranes (MAMs) and cardiovascular Inflammation: Molecular mechanisms and therapeutic implications
inferred← citeshttps://doi.org/10.1016/j.isci.2018.07.015openalex-snapshot
Local Oxidative Damage in the Soma and Dendrites Quarantines Neuronal Mitochondria at the Site of Insult
inferred← citeshttps://doi.org/10.1016/j.jad.2023.12.051openalex-snapshot
TOM40 mediates the effect of TSPO on postpartum depression partially through regulating calcium homeostasis in microglia
inferred← citeshttps://doi.org/10.1016/j.jare.2022.10.015openalex-snapshot
Dose-effect of polystyrene microplastics on digestive toxicity in chickens (Gallus gallus): Multi-omics reveals critical role of gut-liver axis
inferred← citeshttps://doi.org/10.1016/j.jbc.2022.101607openalex-snapshot
The mammalian trafficking chaperone protein UNC93B1 maintains the ER calcium sensor STIM1 in a dimeric state primed for translocation to the ER cortex
inferred← citeshttps://doi.org/10.1016/j.jbc.2022.102863openalex-snapshot
Endoplasmic reticulum protein BIK binds to and inhibits mitochondria-localized antiapoptotic proteins
inferred← citeshttps://doi.org/10.1016/j.jbc.2026.111255openalex-snapshot
HuR-driven reversible mitochondrial shuttling buffers cytosolic miRNA levels in hepatic cells
inferred← citeshttps://doi.org/10.1016/j.jconrel.2023.09.023openalex-snapshot
Combination Organelle Mitochondrial Endoplasmic Reticulum Therapy (COMET) for Multidrug Resistant Breast Cancer
inferred← citeshttps://doi.org/10.1016/j.jlr.2026.101146openalex-snapshot
Mitochondria-endoplasmic reticulum-lipid droplet crosstalk in lipid metabolic reprogramming in type 2 diabetes mellitus
inferred← citeshttps://doi.org/10.1016/j.jpha.2026.101736openalex-snapshot
Disturbing mitochondrial functionalities on the utilization of Lenvatinib-conjugated Gallium (III) nanocomposites as therapeutic avenue to potentiate hepatocellular carcinoma inhibition
inferred← citeshttps://doi.org/10.1016/j.jsps.2017.04.029openalex-snapshot
TSA protects H9c2 cells against thapsigargin-induced apoptosis related to endoplasmic reticulum stress-mediated mitochondrial injury
inferred← citeshttps://doi.org/10.1016/j.jtbi.2021.110684openalex-snapshot
Calcium-dependent insulin resistance in hepatocytes: mathematical model
inferred← citeshttps://doi.org/10.1016/j.lfs.2018.04.048openalex-snapshot
Apigenin-7- O -β- d -(-6″- p -coumaroyl)-glucopyranoside pretreatment attenuates myocardial ischemia/reperfusion injury via activating AMPK signaling
inferred← citeshttps://doi.org/10.1016/j.lfs.2019.116587openalex-snapshot
The roles of mitochondria-associated membranes in mitochondrial quality control under endoplasmic reticulum stress
inferred← citeshttps://doi.org/10.1016/j.lfs.2021.119511openalex-snapshot
Mitochondria-associated membrane-modulated Ca2+ transfer: A potential treatment target in cardiac ischemia reperfusion injury and heart failure
inferred← citeshttps://doi.org/10.1016/j.lfs.2022.121112openalex-snapshot
TRPV4 interacts with MFN2 and facilitates endoplasmic reticulum-mitochondrial contact points for Ca2+-buffering
inferred← citeshttps://doi.org/10.1016/j.mad.2026.112179openalex-snapshot
IPMK-1 governs C. elegans physiology through ceTOR but lifespan via a distinct IP3-dependent pathway
inferred← citeshttps://doi.org/10.1016/j.mam.2026.101458openalex-snapshot
Insulin resistance: The central node of convergence between unfolded protein response, diabetes, and cancer
inferred← citeshttps://doi.org/10.1016/j.mce.2017.09.028openalex-snapshot
Mifepristone enhances insulin-stimulated Akt phosphorylation and glucose uptake in skeletal muscle cells
inferred← citeshttps://doi.org/10.1016/j.mehy.2016.01.022openalex-snapshot
Alzheimer’s disease via enhanced calcium signaling caused by the decrease of endoplasmic reticulum–mitochondrial distance
inferred← citeshttps://doi.org/10.1016/j.metabol.2016.05.002openalex-snapshot
Endoplasmic reticulum stress in obesity and obesity-related disorders: An expanded view
inferred← citeshttps://doi.org/10.1016/j.mito.2020.05.005openalex-snapshot
Modulation of host mitochondrial dynamics during bacterial infection
inferred← citeshttps://doi.org/10.1016/j.mito.2021.03.008openalex-snapshot
Endoplasmic reticulum & mitochondrial calcium homeostasis: The interplay with viruses
inferred← citeshttps://doi.org/10.1016/j.mito.2023.07.005openalex-snapshot
Molecular understanding of ER-MT communication dysfunction during neurodegeneration
inferred← citeshttps://doi.org/10.1016/j.mito.2025.102004openalex-snapshot
Sex-dependent adaptations in heart mitochondria from transgenic mice overexpressing cytochrome b5 reductase-3
inferred← citeshttps://doi.org/10.1016/j.mocell.2024.100176openalex-snapshot
eIF2α phosphorylation-ATF4 axis-mediated transcriptional reprogramming mitigates mitochondrial impairment during ER stress
inferred← citeshttps://doi.org/10.1016/j.molcel.2017.08.005openalex-snapshot
The ER-Localized Transmembrane Protein EPG-3/VMP1 Regulates SERCA Activity to Control ER-Isolation Membrane Contacts for Autophagosome Formation
inferred← citeshttps://doi.org/10.1016/j.molcel.2025.03.003openalex-snapshot
Mitochondria-organelle crosstalk in establishing compartmentalized metabolic homeostasis
inferred← citeshttps://doi.org/10.1016/j.molcel.2026.01.012openalex-snapshot
BRD4-mediated ER membrane contact creates functionally distinct mitochondrial subtypes
inferred← citeshttps://doi.org/10.1016/j.nbd.2013.03.010openalex-snapshot
Silencing of the Charcot–Marie–Tooth disease-associated gene GDAP1 induces abnormal mitochondrial distribution and affects Ca2+ homeostasis by reducing store-operated Ca2+ entry
inferred← citeshttps://doi.org/10.1016/j.nbd.2015.08.004openalex-snapshot
Mitochondria and endoplasmic reticulum crosstalk in amyotrophic lateral sclerosis
inferred← citeshttps://doi.org/10.1016/j.neuint.2014.02.001openalex-snapshot
Endoplasmic reticulum stress in cerebral ischemia
inferred← citeshttps://doi.org/10.1016/j.neuint.2017.03.021openalex-snapshot
From dysfunctional endoplasmic reticulum-mitochondria coupling to neurodegeneration
inferred← citeshttps://doi.org/10.1016/j.neuint.2017.04.009openalex-snapshot
Connecting mitochondrial dynamics and life-or-death events via Bcl-2 family proteins
inferred← citeshttps://doi.org/10.1016/j.neuron.2022.04.020openalex-snapshot
Dysregulation of organelle membrane contact sites in neurological diseases
inferred← citeshttps://doi.org/10.1016/j.neuropharm.2025.110440openalex-snapshot
Mitochondrial damage and ER stress in CB1 receptor antagonist-induced apoptosis in human neuroblastoma SH-SY5Y cells
inferred← citeshttps://doi.org/10.1016/j.pestbp.2024.106175openalex-snapshot
Resmethrin disrupts mitochondria-associated membranes and activates endoplasmic reticulum stress, leading to proliferation inhibition in cultured mouse Leydig and Sertoli cells
inferred← citeshttps://doi.org/10.1016/j.phrs.2020.104758openalex-snapshot
ER membranes associated with mitochondria: Possible therapeutic targets in heart-associated diseases
inferred← citeshttps://doi.org/10.1016/j.phrs.2026.108428openalex-snapshot
Crosstalk between UPR and mitochondria: The triad of ER–mitochondria contacts, Ca²⁺, and ROS
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