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eIF2α Phosphorylation-Mediated Transcriptional Reprogramming Suppresses Impairment of Mitochondrial and GSH Homeostasis and Thereby Ferroptosis During ER Stress

https://doi.org/10.2139/ssrn.4648361
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Mitochondrial Fission Triggered by Hyperglycemia Is Mediated by ROCK1 Activation in Podocytes and Endothelial Cells
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Coincident Phosphatidic Acid Interaction Restrains Drp1 in Mitochondrial Division
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ER and Nutrient Stress Promote Assembly of Respiratory Chain Supercomplexes through the PERK-eIF2α Axis
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Nrf2 mediates redox adaptations to exercise
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NRF2 plays a critical role in mitigating lipid peroxidation and ferroptosis
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Real-Time Monitoring of Glutathione in Living Cells Reveals that High Glutathione Levels Are Required to Maintain Stem Cell Function
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Transsulfuration, minor player or crucial for cysteine homeostasis in cancer
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Transcriptional integration of mitochondrial biogenesis
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Stress-responsive regulation of mitochondria through the ER unfolded protein response
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Mitochondrial Dynamics and Metabolic Regulation
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The Nrf2 regulatory network provides an interface between redox and intermediary metabolism
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Glutathione dynamics is a potential predictive and therapeutic trait for neoadjuvant chemotherapy response in bladder cancer
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Mechanisms Controlling Mitochondrial Biogenesis and Respiration through the Thermogenic Coactivator PGC-1
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Endoplasmic reticulum stress induces ligand-independent TNFR1-mediated necroptosis in L929 cells
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SLP‐2 is required for stress‐induced mitochondrial hyperfusion
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AMPK regulates energy expenditure by modulating NAD+ metabolism and SIRT1 activity
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TFEB controls cellular lipid metabolism through a starvation-induced autoregulatory loop
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ER-stress-induced transcriptional regulation increases protein synthesis leading to cell death
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MSFragger: ultrafast and comprehensive peptide identification in mass spectrometry–based proteomics
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Transcription factors in ferroptotic cell death
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Increased mitochondrial respiration promotes survival from endoplasmic reticulum stress
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Carbon monoxide-induced TFEB nuclear translocation enhances mitophagy/mitochondrial biogenesis in hepatocytes and ameliorates inflammatory liver injury
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Endoplasmic reticulum stress-mediated cell death in liver injury
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Synergistic mechanism between the endoplasmic reticulum and mitochondria and their crosstalk with other organelles
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Endoplasmic reticulum stress signals in the tumour and its microenvironment
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Ferroptosis: mechanisms, biology and role in disease
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AMP-activated protein kinase (AMPK) action in skeletal muscle via direct phosphorylation of PGC-1α
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Nuclear Respiratory Factor 2 Induces the Expression of Many but Not All Human Proteins Acting in Mitochondrial DNA Transcription and Replication
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Integrated Stress Response Modulates Cellular Redox State via Induction of Cystathionine γ-Lyase
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Loss of Mitofusin 2 Promotes Endoplasmic Reticulum Stress
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Basal Levels of eIF2α Phosphorylation Determine Cellular Antioxidant Status by Regulating ATF4 and xCT Expression
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Upstream Open Reading Frames Differentially Regulate Gene-specific Translation in the Integrated Stress Response
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Phosphorylation of EIF2S1 (eukaryotic translation initiation factor 2 subunit alpha) is indispensable for nuclear translocation of TFEB and TFE3 during ER stress
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CaM kinase Iα–induced phosphorylation of Drp1 regulates mitochondrial morphology
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Multi-omics analysis identifies ATF4 as a key regulator of the mitochondrial stress response in mammals
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Endoplasmic Reticulum Stress and Oxidative Stress in Cell Fate Decision and Human Disease
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The eIF2 kinase PERK and the integrated stress response facilitate activation of ATF6 during endoplasmic reticulum stress
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IRE1–RACK1 axis orchestrates ER stress preconditioning-elicited cytoprotection from ischemia/reperfusion injury in liver
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Transcriptional cross talk between orphan nuclear receptor ERR and transmembrane transcription factor ATF6 coordinates endoplasmic reticulum stress response
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Transcriptional regulatory circuits controlling mitochondrial biogenesis and function
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Physiological/pathological ramifications of transcription factors in the unfolded protein response
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The PERKs of mitochondria protection during stress: insights for PERK modulation in neurodegenerative and metabolic diseases
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Cell death induced by endoplasmic reticulum stress
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Mitochondrial oxidative stress causes mitochondrial fragmentation via differential modulation of mitochondrial fission–fusion proteins
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Melatonin prevents D rp1‐mediated mitochondrial fission in diabetic hearts through SIRT 1‐ PGC 1α pathway
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The Unfolded Protein Response: From Stress Pathway to Homeostatic Regulation
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Mitochondrial Fission, Fusion, and Stress
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IRF2 transcriptionally induces GSDMD expression for pyroptosis
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Nrf2- and ATF4-Dependent Upregulation of xCT Modulates the Sensitivity of T24 Bladder Carcinoma Cells to Proteasome Inhibition
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Nrf2 Is a Direct PERK Substrate and Effector of PERK-Dependent Cell Survival
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Control of Mitochondrial Transcription Specificity Factors (TFB1M and TFB2M) by Nuclear Respiratory Factors (NRF-1 and NRF-2) and PGC-1 Family Coactivators
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The Nrf2/PGC1α Pathway Regulates Antioxidant and Proteasomal Activity to Alter Cisplatin Sensitivity in Ovarian Cancer
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Endoplasmic reticulum stress is sufficient for the induction of IL-1β production via activation of the NF-κB and inflammasome pathways
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Mitochondrial fusion is required for regulation of mitochondrial DNA replication
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TFEB and TFE3 are novel components of the integrated stress response
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PERK signaling promotes mitochondrial elongation by remodeling membrane phosphatidic acid
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ATF4-Dependent NRF2 Transcriptional Regulation Promotes Antioxidant Protection during Endoplasmic Reticulum Stress
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IRF2 contributes to myocardial infarction via regulation of GSDMD induced pyroptosis
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Deficiency in the mitochondrial apoptotic pathway reveals the toxic potential of autophagy under ER stress conditions
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The mTORC1-mediated activation of ATF4 promotes protein and glutathione synthesis downstream of growth signals
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