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The DPC4/SMAD4 genetic status determines recurrence patterns and treatment outcomes in resected pancreatic ductal adenocarcinoma: A prospective cohort study
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/1097-0215(20001120)89:6<469::aid-ijc1>3.0.co;2-lopenalex-snapshot
K-ras oncogene subtype mutations are associated with survival but not expression of p53, p16INK4A, p21WAF-1, cyclin D1, erbB-2 and erbB-3 in resected pancreatic ductal adenocarcinoma
inferredcites →https://doi.org/10.1002/anie.201307387openalex-snapshot
Therapeutic Targeting of Oncogenic K‐Ras by a Covalent Catalytic Site Inhibitor
inferredcites →https://doi.org/10.1002/anie.201400233openalex-snapshot
Selective Inhibition of Mutant Ras Protein through Covalent Binding
inferredcites →https://doi.org/10.1002/bjs.5397openalex-snapshot
Redefining the R1 resection in pancreatic cancer
inferredcites →https://doi.org/10.1002/jso.23606openalex-snapshot
Loss of SMAD4 staining in pre-operative cell blocks is associated with distant metastases following pancreaticoduodenectomy with venous resection for pancreatic cancer
inferredcites →https://doi.org/10.1016/j.jvir.2012.01.080openalex-snapshot
Radiofrequency Ablation of Liver Metastasis in Patients with Locally Controlled Pancreatic Ductal Adenocarcinoma
inferredcites →https://doi.org/10.1016/j.surg.2013.05.004openalex-snapshot
Epithelial-to-mesenchymal transition predicts prognosis of pancreatic cancer
inferredcites →https://doi.org/10.1016/j.surg.2013.06.050openalex-snapshot
Repeat pancreatectomy for pancreatic ductal cancer recurrence in the remnant pancreas after initial pancreatectomy: Is it worthwhile?
inferredcites →https://doi.org/10.1038/sj.onc.1208927openalex-snapshot
TGF-β and epithelial-to-mesenchymal transitions
inferredcites →https://doi.org/10.1038/sj.onc.1210741openalex-snapshot
Sustained TGFβ exposure suppresses Smad and non-Smad signalling in mammary epithelial cells, leading to EMT and inhibition of growth arrest and apoptosis
inferredcites →https://doi.org/10.1056/nejmoa0804385openalex-snapshot
K-ras Mutations and Benefit from Cetuximab in Advanced Colorectal Cancer
inferredcites →https://doi.org/10.1097/01.sla.0000245845.06772.7dopenalex-snapshot
Surgery for Recurrent Pancreatic Ductal Adenocarcinoma
inferredcites →https://doi.org/10.1097/mpa.0000000000000315openalex-snapshot
SMAD4 Expression Predicts Local Spread and Treatment Failure in Resected Pancreatic Cancer
inferredcites →https://doi.org/10.1097/mpa.0b013e31825b6ab0openalex-snapshot
Genetic Alterations of K-ras, p53, c-erbB-2, and DPC4 in Pancreatic Ductal Adenocarcinoma and Their Correlation With Patient Survival
inferredcites →https://doi.org/10.1097/sla.0b013e31827fe9ceopenalex-snapshot
Failure Patterns in Resected Pancreas Adenocarcinoma
inferredcites →https://doi.org/10.1097/sla.0b013e3182827a65openalex-snapshot
Immunohistochemically Detected Expression of 3 Major Genes (CDKN2A/p16, TP53, and SMAD4/DPC4) Strongly Predicts Survival in Patients With Resectable Pancreatic Cancer
inferredcites →https://doi.org/10.1101/gad.1478706openalex-snapshot
Smad4 is dispensable for normal pancreas development yet critical in progression and tumor biology of pancreas cancer
inferredcites →https://doi.org/10.1111/pin.12114openalex-snapshot
Molecular pathology of pancreatic cancer
inferredcites →https://doi.org/10.1126/science.271.5247.350openalex-snapshot
DPC4 , A Candidate Tumor Suppressor Gene at Human Chromosome 18q21.1
inferredcites →https://doi.org/10.1158/0008-5472.can-05-3560openalex-snapshot
The Tumor Suppressor Smad4 Is Required for Transforming Growth Factor β–Induced Epithelial to Mesenchymal Transition and Bone Metastasis of Breast Cancer Cells
inferredcites →https://doi.org/10.1158/1078-0432.ccr-09-0227openalex-snapshot
SMAD4 Gene Mutations Are Associated with Poor Prognosis in Pancreatic Cancer
inferredcites →https://doi.org/10.1158/1078-0432.ccr-10-2214openalex-snapshot
Synergistic Effect between Erlotinib and MEK Inhibitors in KRAS Wild-Type Human Pancreatic Cancer Cells
inferredcites →https://doi.org/10.1200/jco.2002.12.063openalex-snapshot
DPC4/Smad4 Expression and Outcome in Pancreatic Ductal Adenocarcinoma
inferredcites →https://doi.org/10.1200/jco.2008.17.7188openalex-snapshot
DPC4 Gene Status of the Primary Carcinoma Correlates With Patterns of Failure in Patients With Pancreatic Cancer
inferredcites →https://doi.org/10.1245/s10434-008-9839-8openalex-snapshot
Most Pancreatic Cancer Resections are R1 Resections
inferredcites →https://doi.org/10.2214/ajr.168.6.9168704openalex-snapshot
Local staging of pancreatic cancer: criteria for unresectability of major vessels as revealed by pancreatic-phase, thin-section helical CT.
inferredcites →https://doi.org/10.3748/wjg.v17.i9.1126openalex-snapshot
CT diagnosis of recurrence after pancreatic cancer: Is there a pattern?
inferredcites →https://doi.org/10.3748/wjg.v20.i24.7864openalex-snapshot
Imaging diagnosis of pancreatic cancer: A state-of-the-art review
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4631952openalex-extracted
Huachansu Improves the Tumor Microenvironment In A Mouse Model Of Pancreatic Cancer By Regulating the TGF-Β/Smad Signaling Pathway
inferred← citeshttps://doi.org/10.1002/bjs.11295openalex-snapshot
Meta-analysis of recurrence pattern after resection for pancreatic cancer
inferred← citeshttps://doi.org/10.1002/jso.26279openalex-snapshot
Poorly differentiated histologic grade correlates with worse survival in SMAD4 negative pancreatic adenocarcinoma patients
inferred← citeshttps://doi.org/10.1007/s00423-017-1627-0openalex-snapshot
Radiofrequency ablation for locally advanced pancreatic cancer: SMAD4 analysis segregates a responsive subgroup of patients
inferred← citeshttps://doi.org/10.1007/s12029-018-0103-6openalex-snapshot
Intratumoral Heterogeneity of SMAD4 Immunohistochemical Expression and Its Role in Prediction of Recurrence Pattern in Patients with Resectable Pancreatic Cancer
inferred← citeshttps://doi.org/10.1016/j.bbcan.2024.189220openalex-snapshot
New insight into the role of SMAD4 mutation/deficiency in the prognosis and therapeutic resistance of pancreatic ductal adenocarcinomas
inferred← citeshttps://doi.org/10.1016/j.ctrv.2019.03.003openalex-snapshot
Genomic profiling in pancreatic ductal adenocarcinoma and a pathway towards therapy individualization: A scoping review
inferred← citeshttps://doi.org/10.1016/j.isci.2020.101551openalex-snapshot
Circadian Dysregulation of the TGFβ/SMAD4 Pathway Modulates Metastatic Properties and Cell Fate Decisions in Pancreatic Cancer Cells
inferred← citeshttps://doi.org/10.1016/j.jep.2024.117872openalex-snapshot
Modulating pancreatic cancer microenvironment: The efficacy of Huachansu in mouse models via TGF-β/Smad pathway
inferred← citeshttps://doi.org/10.1016/j.lpm.2019.02.031openalex-snapshot
Pancreatic cancer – What's next?
inferred← citeshttps://doi.org/10.1016/j.prp.2023.154406openalex-snapshot
Necrosis as a strong independent prognostic factor required in the implementation of pathological reporting for pancreatic adenocarcinoma resection specimens
inferred← citeshttps://doi.org/10.1016/j.radonc.2021.02.037openalex-snapshot
High expression of krüppel-like factor 10 or Smad4 predicts clinical benefit of adjuvant chemoradiotherapy in curatively resected pancreatic adenocarcinoma: From a randomized phase III trial
inferred← citeshttps://doi.org/10.1016/j.suc.2017.09.008openalex-snapshot
The Evolving Role of Radiation in Pancreatic Cancer
inferred← citeshttps://doi.org/10.1016/j.surg.2018.04.017openalex-snapshot
Chronologic changes in clinical and survival features of pancreatic ductal adenocarcinoma since 2000: A single-center experience with 2,029 patients
inferred← citeshttps://doi.org/10.1038/s41598-017-12670-3openalex-snapshot
Synergistic effect of a drug loaded electrospun patch and systemic chemotherapy in pancreatic cancer xenograft
inferred← citeshttps://doi.org/10.1038/s42003-022-03994-6openalex-snapshot
SMAD2/3 mediate oncogenic effects of TGF-β in the absence of SMAD4
inferred← citeshttps://doi.org/10.1053/j.gastro.2023.10.026openalex-snapshot
An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies
inferred← citeshttps://doi.org/10.1080/15548627.2019.1628540openalex-snapshot
TGFB1-induced autophagy affects the pattern of pancreatic cancer progression in distinct ways depending on SMAD4 status
inferred← citeshttps://doi.org/10.1097/md.0000000000034312openalex-snapshot
Clinicopathological and prognostic significance of SMAD4 in non-small cell lung cancer: A meta-analysis and database validation
inferred← citeshttps://doi.org/10.1101/195610openalex-snapshot
EGLN Inhibition Reduces Gastrointestinal Radiation Toxicity and Improves Survival in a Murine Model of Locally Advanced Pancreatic Cancer
inferred← citeshttps://doi.org/10.1111/ajco.12996openalex-snapshot
Chemoradiotherapy versus chemotherapy for locally advanced unresectable pancreatic cancer: A systematic review and meta‐analysis
inferred← citeshttps://doi.org/10.1111/his.14880openalex-snapshot
Loss of function SMAD4 nonstop mutations in human cancer
inferred← citeshttps://doi.org/10.1117/1.jbo.24.11.118001openalex-snapshot
Verteporfin- and sodium porfimer-mediated photodynamic therapy enhances pancreatic cancer cell death without activating stromal cells in the microenvironment
inferred← citeshttps://doi.org/10.1158/2767-9764.22761816openalex-snapshot
Supplementary Figure 6 from Genome-wide CRISPR Screen Reveals RAB10 as a Synthetic Lethal Gene in Colorectal and Pancreatic Cancers Carrying SMAD4 Loss
inferred← citeshttps://doi.org/10.1158/2767-9764.c.6597302.v1openalex-snapshot
Data from Genome-wide CRISPR screen reveals RAB10 as a synthetic lethal gene in colorectal and pancreatic cancers carrying SMAD4 loss.
inferred← citeshttps://doi.org/10.1158/2767-9764.c.6597302.v2openalex-snapshot
Data from Genome-wide CRISPR Screen Reveals RAB10 as a Synthetic Lethal Gene in Colorectal and Pancreatic Cancers Carrying SMAD4 Loss
inferred← citeshttps://doi.org/10.1158/2767-9764.crc-22-0301openalex-snapshot
Genome-wide CRISPR Screen Reveals RAB10 as a Synthetic Lethal Gene in Colorectal and Pancreatic Cancers Carrying SMAD4 Loss
inferred← citeshttps://doi.org/10.1177/10732748231173212openalex-snapshot
Current Treatment of Potentially Resectable Pancreatic Ductal Adenocarcinoma: A Medical Oncologist’s Perspective
inferred← citeshttps://doi.org/10.1186/s12885-019-6251-7openalex-snapshot
SMAD4 Y353C promotes the progression of PDAC
inferred← citeshttps://doi.org/10.1186/s12935-017-0416-8openalex-snapshot
Establishment and characterization of 6 novel patient-derived primary pancreatic ductal adenocarcinoma cell lines from Korean pancreatic cancer patients
inferred← citeshttps://doi.org/10.1186/s13014-023-02328-yopenalex-snapshot
Selection of patients with pancreatic adenocarcinoma who may benefit from radiotherapy
inferred← citeshttps://doi.org/10.1200/cci-26-00200openalex-snapshot
Toward Clinically Actionable Prediction of Recurrence and Metastatic Progression in Pancreatic Ductal Adenocarcinoma: A Systematic Review
inferred← citeshttps://doi.org/10.1371/journal.pone.0250634openalex-snapshot
Association of SMAD4 loss with drug resistance in clinical cancer patients: A systematic meta-analysis
inferred← citeshttps://doi.org/10.14293/s2199-1006.1.sor-.pp0oqrj.v1openalex-snapshot
Comparative Transcriptomic Response Of Pancreatic And Breast Cancer Cells To Anacardic Acid Ans Olaparib
inferred← citeshttps://doi.org/10.14701/ahbps.23-149openalex-snapshot
Prognostication for recurrence patterns after curative resection for pancreatic ductal adenocarcinoma
inferred← citeshttps://doi.org/10.2478/ahem-2022-0036openalex-snapshot
Prognostic value of SMAD4 in resectable pancreatic cancer
inferred← citeshttps://doi.org/10.31557/apjcp.2023.24.8.2895openalex-snapshot
The Mutation Portraits of Oncogenes and Tumor Supressor Genes in Predicting the Overall Survival in Pancreatic Cancer: A Bayesian Network Meta-Analysis
inferred← citeshttps://doi.org/10.3389/fonc.2022.833231openalex-snapshot
Finite Element-Based Personalized Simulation of Duodenal Hydrogel Spacer: Spacer Location Dependent Duodenal Sparing and a Decision Support System for Spacer-Enabled Pancreatic Cancer Radiation Therapy
inferred← citeshttps://doi.org/10.3389/fonc.2025.1651566openalex-snapshot
Editorial: Molecular markers for pancreatic cancers: new technologies and applications in the clinical practice
inferred← citeshttps://doi.org/10.3390/biomedicines11113058openalex-snapshot
Association between SMAD4 Mutations and GATA6 Expression in Paired Pancreatic Ductal Adenocarcinoma Tumor Specimens: Data from Two Independent Molecularly-Characterized Cohorts
inferred← citeshttps://doi.org/10.3390/biomedicines5040065openalex-snapshot
Pancreatic Cancer: Molecular Characterization, Clonal Evolution and Cancer Stem Cells
inferred← citeshttps://doi.org/10.3390/cancers10010017openalex-snapshot
Advances in Molecular Profiling and Categorisation of Pancreatic Adenocarcinoma and the Implications for Therapy
inferred← citeshttps://doi.org/10.3390/cancers14040973openalex-snapshot
The Role of SMAD4 Inactivation in Epithelial–Mesenchymal Plasticity of Pancreatic Ductal Adenocarcinoma: The Missing Link?
inferred← citeshttps://doi.org/10.3390/cancers14081866openalex-snapshot
Clinical Applications of Classical and Novel Biological Markers of Pancreatic Cancer
inferred← citeshttps://doi.org/10.3390/cancers15113061openalex-snapshot
deepPERFECT: Novel Deep Learning CT Synthesis Method for Expeditious Pancreatic Cancer Radiotherapy
inferred← citeshttps://doi.org/10.3390/cancers15153765openalex-snapshot
SMAD4 Positive Pancreatic Ductal Adenocarcinomas Are Associated with Better Outcomes in Patients Receiving FOLFIRINOX-Based Neoadjuvant Therapy
inferred← citeshttps://doi.org/10.3390/cancers15174332openalex-snapshot
Inter-Breath-Hold Geometric and Dosimetric Variations in Organs at Risk during Pancreatic Stereotactic Body Radiotherapy: Implications for Adaptive Radiation Therapy
inferred← citeshttps://doi.org/10.3390/cancers15215212openalex-snapshot
Krüppel-like Factor 10 as a Prognostic and Predictive Biomarker of Radiotherapy in Pancreatic Adenocarcinoma
inferred← citeshttps://doi.org/10.3390/cimb46040177openalex-snapshot
Significance of TP53, CDKN2A, SMAD4 and KRAS in Pancreatic Cancer
inferred← citeshttps://doi.org/10.3390/curroncol31060250openalex-snapshot
Radiochemotherapy in Pancreatic Cancer
inferred← citeshttps://doi.org/10.3390/ijms18051094openalex-snapshot
The Impact of SMAD4 Loss on Outcome in Patients with Advanced Pancreatic Cancer Treated with Systemic Chemotherapy
inferred← citeshttps://doi.org/10.3390/ma16072906openalex-snapshot
Mitoxantrone and Mitoxantrone-Loaded Iron Oxide Nanoparticles Induce Cell Death in Human Pancreatic Ductal Adenocarcinoma Cell Spheroids
inferred← citeshttps://doi.org/10.3390/pharmaceutics13010103openalex-snapshot
Therapeutic Approaches for Metastases from Colorectal Cancer and Pancreatic Ductal Carcinoma
inferred← citeshttps://doi.org/10.3390/reports9030285openalex-snapshot
Unexpected Long-Term Survival in Resected Pancreatic Ductal Adenocarcinoma Harboring KRAS G12R with an Ultra-Low-VAF SMAD4 Mutation: A Case Report and Mini-Review
inferred← citeshttps://doi.org/10.3889/oamjms.2019.104openalex-snapshot
A Review of Pancreatic Cancer: Epidemiology, Genetics, Screening, and Management
inferred← citeshttps://doi.org/10.3892/ijo.2018.4344openalex-snapshot
Mutational burden of resectable pancreatic cancer, as determined by whole transcriptome and whole exome sequencing, predicts a poor prognosis
inferred← citeshttps://doi.org/10.70675/6f1078b7z94d3z4096z85a9z40b435cd6a59openalex-snapshot
Implications oncogéniques des protéines SMAD2 & SMAD3 dans l’adénocarcinome canalaire pancréatique SMAD4-négatif
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