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Tumor Establishment Requires Tumor Autonomous and Non-Autonomous Decoupling of EGF Signaling from Apoptosis

https://doi.org/10.2139/ssrn.3329888
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Outgoing claims: the papers this work cites, builds on, or argues with.

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c‐Src drives intestinal regeneration and transformation
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Nonchordate Classic Cadherins Have a Structurally and Functionally Unique Domain That Is Absent from Chordate Classic Cadherins
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RHBDD2: a 5-fluorouracil responsive gene overexpressed in the advanced stages of colorectal cancer
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Molecular interactions in cancer cell metastasis
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E-cadherin's dark side: Possible role in tumor progression
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Epidermal growth factor receptor targeting in cancer: A review of trends and strategies
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A Two-Step Model for Colon Adenoma Initiation and Progression Caused by APC Loss
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Cytokine/Jak/Stat Signaling Mediates Regeneration and Homeostasis in the Drosophila Midgut
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Altered Modes of Stem Cell Division Drive Adaptive Intestinal Growth
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Morphological and Molecular Characterization of Adult Midgut Compartmentalization in Drosophila
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Enteroendocrine Cells Support Intestinal Stem-Cell-Mediated Homeostasis in Drosophila
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Cell Competition Drives the Growth of Intestinal Adenomas in Drosophila
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Tumors as Organs: Complex Tissues that Interface with the Entire Organism
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The ErbB/HER family of protein-tyrosine kinases and cancer
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EGFR/Ras/MAPK Signaling Mediates Adult Midgut Epithelial Homeostasis and Regeneration in Drosophila
inferredcites →https://doi.org/10.1016/j.ydbio.2013.03.020openalex-extracted
APC loss-induced intestinal tumorigenesis in Drosophila: Roles of Ras in Wnt signaling activation and tumor progression
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Regulation of Armadillo by a Drosophila APC Inhibits Neuronal Apoptosis during Retinal Development
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Mosaic Analysis with a Repressible Cell Marker for Studies of Gene Function in Neuronal Morphogenesis
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Src42A modulates tumor invasion and cell death via Ben/dUev1a-mediated JNK activation in Drosophila
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Correction of aberrant growth preserves tissue homeostasis
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Feedback regulation of steady-state epithelial turnover and organ size
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Niche appropriation by Drosophila intestinal stem cell tumours
inferredcites →https://doi.org/10.1038/ncomms10219openalex-extracted
Accumulation of differentiating intestinal stem cell progenies drives tumorigenesis
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Rhomboid domain containing 1 promotes colorectal cancer growth through activation of the EGFR signalling pathway
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Microenvironmental regulation of tumor progression and metastasis
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Fiji: an open-source platform for biological-image analysis
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Control of cancer formation by intrinsic genetic noise and microenvironmental cues
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Beyond β-catenin: prospects for a larger catenin network in the nucleus
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Cadherins and cancer: how does cadherin dysfunction promote tumor progression?
inferredcites →https://doi.org/10.1073/pnas.0900641106openalex-extracted
Directed, efficient, and versatile modifications of the Drosophila genome by genomic engineering
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P{Switch}, a system for spatial and temporal control of gene expression in Drosophila melanogaster
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Regulatory mechanisms required for DE-cadherin function in cell migration and other types of adhesion
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Beyond the Niche: Tissue-Level Coordination of Stem Cell Dynamics
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Molecular Genetics of Colorectal Cancer
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Cell Turnover and Adult Tissue Homeostasis: From Humans to Planarians
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RHBDL2 Is a Critical Membrane Protease for Anoikis Resistance in Human Malignant Epithelial Cells
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Correction to: RHBDD1 promotes colorectal cancer metastasis through the Wnt signaling pathway and its downstream target ZEB1
inferredcites →https://doi.org/10.1242/dev.02398openalex-extracted
Testing hypotheses for the functions of APC family proteins using null and truncation alleles inDrosophila
inferredcites →https://doi.org/10.1242/dev.035196openalex-extracted
Adenomatous polyposis coli regulates Drosophila intestinal stem cell proliferation
inferredcites →https://doi.org/10.1242/dev.078261openalex-extracted
Non-autonomous crosstalk between the Jak/Stat and Egfr pathways mediates Apc1-driven intestinal stem cell hyperplasia in the Drosophila adult midgut
inferredcites →https://doi.org/10.1371/journal.pgen.1006870openalex-extracted
Intestinal stem cell overproliferation resulting from inactivation of the APC tumor suppressor requires the transcription cofactors Earthbound and Erect wing
inferredcites →https://doi.org/10.15252/embj.201591739openalex-extracted
E‐cadherin can limit the transforming properties of activating β‐catenin mutations
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Homeostatic Signaling by Cell–Cell Junctions and Its Dysregulation during Cancer Progression
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Wingless/Wnt Signaling in Intestinal Development, Homeostasis, Regeneration and Tumorigenesis: A Drosophila Perspective
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