On the public citation graph

NR1H4 rs35724 G>C Variant Modulates Liver Damage in Nonalcoholic Fatty Liver Disease

https://doi.org/10.2139/ssrn.3736702
0 signed · 19 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.
Claim with ORCID
Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.
See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/hep.20701openalex-extracted
Design and validation of a histological scoring system for nonalcoholic fatty liver disease†
inferredcites →https://doi.org/10.1016/0092-8674(95)90530-8openalex-extracted
Identification of a nuclear receptor that is activated by farnesol metabolites
inferredcites →https://doi.org/10.1016/j.bbagrm.2016.03.002openalex-extracted
Nuclear receptors and nonalcoholic fatty liver disease
inferredcites →https://doi.org/10.1016/j.febslet.2008.02.038openalex-extracted
Activation of the farnesoid X receptor represses PCSK9 expression in human hepatocytes
inferredcites →https://doi.org/10.1016/j.jhep.2015.10.016openalex-extracted
MERTK rs4374383 polymorphism affects the severity of fibrosis in non-alcoholic fatty liver disease
inferredcites →https://doi.org/10.1016/j.jhep.2020.02.020openalex-extracted
Update on NAFLD genetics: From new variants to the clinic
inferredcites →https://doi.org/10.1016/s0168-8278(03)00191-0openalex-extracted
Impact of liver biopsy size on histological evaluation of chronic viral hepatitis: the smaller the sample, the milder the disease
inferredcites →https://doi.org/10.1016/s0168-8278(15)30169-0openalex-extracted
LP15 : Separate and combined effects of obeticholic acid and weight loss in nonalcoholic steatohepatitis (NASH)
inferredcites →https://doi.org/10.1016/s0168-8278(17)31537-4openalex-extracted
Farnesoid-X Receptor single nucleotide polymorphism rs35724 is potentially associated with patients’ outcome in decompensated liver cirrhosis
inferredcites →https://doi.org/10.1074/jbc.m209525200openalex-extracted
The Farnesoid X-receptor Is an Essential Regulator of Cholesterol Homeostasis
inferredcites →https://doi.org/10.1074/jbc.m510258200openalex-extracted
The Farnesoid X Receptor Modulates Adiposity and Peripheral Insulin Sensitivity in Mice
inferredcites →https://doi.org/10.1111/apt.14492openalex-extracted
Clinical and metabolic effects associated with weight changes and obeticholic acid in non‐alcoholic steatohepatitis
inferredcites →https://doi.org/10.1111/jgh.14700openalex-extracted
Impact of farnesoid X receptor single nucleotide polymorphisms on hepatic decompensation and mortality in cirrhotic patients with portal hypertension
inferredcites →https://doi.org/10.1111/joim.12719openalex-extracted
Causal relationship of hepatic fat with liver damage and insulin resistance in nonalcoholic fatty liver
inferredcites →https://doi.org/10.1172/jci21025openalex-extracted
Bile acids lower triglyceride levels via a pathway involving FXR, SHP, and SREBP-1c
inferredcites →https://doi.org/10.1194/jlr.m081935openalex-extracted
FXR activation by obeticholic acid or nonsteroidal agonists induces a human-like lipoprotein cholesterol change in mice with humanized chimeric liver
inferredcites →https://doi.org/10.1210/me.2002-0120openalex-extracted
Bile Acids Induce the Expression of the Human Peroxisome Proliferator-Activated Receptor α Gene via Activation of the Farnesoid X Receptor
inferredcites →https://doi.org/10.2174/0929867324666170124151940openalex-extracted
Farnesoid-X Receptor (FXR) as a Promising Pharmaceutical Target in Atherosclerosis
inferredcites →https://doi.org/10.2337/diacare.21.1.s5openalex-extracted
Report of the Expert Committee on the Diagnosis and Classification of Diabetes Mellitus
Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.