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Hypoxia/Inflammation-Induced Upregulation of HIF-1α and C/EBPβ, Promoted Nephroblastoma EMT by Improving HOXA11-AS Transcription

https://doi.org/10.2139/ssrn.4186473
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Hypoxia induced EMT: A review on the mechanism of tumor progression and metastasis in OSCC
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Hypoxia, HIF1 and glucose metabolism in the solid tumour
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The genetic changes of Wilms tumour
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Cancer LncRNA Census reveals evidence for deep functional conservation of long noncoding RNAs in tumorigenesis
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The transcription factor HIF-1α plays a critical role in the growth factor-dependent regulation of both aerobic and anaerobic glycolysis
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Silencing of hypoxia inducible factor‐1α by RNA interference inhibits growth of SK ‐ NEP ‐1 Wilms tumour cells in vitro, and suppresses tumourigenesis and angiogenesis in vivo
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Comprehensive Analysis of a Long Noncoding RNA-Associated Competing Endogenous RNA Network in Wilms Tumor
inferredcites →https://doi.org/10.1186/1471-2164-13-251openalex-extracted
Evolution of coding and non-coding genes in HOX clusters of a marsupial
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Overexpression of lncRNA HOXA11-AS promotes cell epithelial–mesenchymal transition by repressing miR-200b in non-small cell lung cancer
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Over-expressed long noncoding RNA HOXA11-AS promotes cell cycle progression and metastasis in gastric cancer
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The lncRNA HOXA11-AS functions as a competing endogenous RNA to regulate PADI2 expression by sponging miR-125a-5p in liver metastasis of colorectal cancer
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Impact of the First Generation of Children’s Oncology Group Clinical Trials on Clinical Practice for Wilms Tumor
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