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Once-Weekly TransCon CNP in Children with Achondroplasia (ACcomplisH): A Phase 2, Multicentre, Randomised, Double-Blind, Placebo-Controlled, Dose-Escalation Trial

https://doi.org/10.2139/ssrn.4488940
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Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1016/0092-8674(94)90302-6openalex-extracted
Mutations in the transmembrane domain of FGFR3 cause the most common genetic form of dwarfism, achondroplasia
inferredcites →https://doi.org/10.1016/j.ajhg.2012.10.014openalex-extracted
Evaluation of the Therapeutic Potential of a CNP Analog in a Fgfr3 Mouse Model Recapitulating Achondroplasia
inferredcites →https://doi.org/10.1016/j.berh.2007.12.008openalex-extracted
Achondroplasia
inferredcites →https://doi.org/10.1016/j.bone.2021.115872openalex-extracted
Lifetime impact of achondroplasia: Current evidence and perspectives on the natural history
inferredcites →https://doi.org/10.1016/s0092-8674(00)81069-7openalex-extracted
Fibroblast Growth Factor Receptor 3 Is a Negative Regulator of Bone Growth
inferredcites →https://doi.org/10.1016/s0140-6736(07)61090-3openalex-extracted
Achondroplasia
inferredcites →https://doi.org/10.1016/s0140-6736(20)31541-5openalex-extracted
Once-daily, subcutaneous vosoritide therapy in children with achondroplasia: a randomised, double-blind, phase 3, placebo-controlled, multicentre trial
inferredcites →https://doi.org/10.1038/371252a0openalex-extracted
Mutations in the gene encoding fibroblast growth factor receptor-3 in achondroplasia
inferredcites →https://doi.org/10.1038/nm971openalex-extracted
Overexpression of CNP in chondrocytes rescues achondroplasia through a MAPK-dependent pathway
inferredcites →https://doi.org/10.1038/s41574-021-00595-xopenalex-extracted
International Consensus Statement on the diagnosis, multidisciplinary management and lifelong care of individuals with achondroplasia
inferredcites →https://doi.org/10.1056/nejmoa1813446openalex-extracted
C-Type Natriuretic Peptide Analogue Therapy in Children with Achondroplasia
inferredcites →https://doi.org/10.1111/bcp.15369openalex-extracted
Phase 1 safety, tolerability, pharmacokinetics and pharmacodynamics results of a long‐acting C‐type natriuretic peptide prodrug, TransCon CNP
inferredcites →https://doi.org/10.1124/jpet.119.258251openalex-extracted
TransCon CNP, a Sustained-Release C-Type Natriuretic Peptide Prodrug, a Potentially Safe and Efficacious New Therapeutic Modality for the Treatment of Comorbidities Associated with Fibroblast Growth Factor Receptor 3–Related Skeletal Dysplasias
inferredcites →https://doi.org/10.1136/adc.41.219.454openalex-extracted
Standards from birth to maturity for height, weight, height velocity, and weight velocity: British children, 1965. I.
inferredcites →https://doi.org/10.1159/000520812openalex-extracted
Evolution and Future of Growth Plate Therapeutics
inferredcites →https://doi.org/10.1177/1759720x221084848openalex-extracted
Infigratinib in children with achondroplasia: the PROPEL and PROPEL 2 studies
inferredcites →https://doi.org/10.1186/s13023-018-0972-6openalex-extracted
Achondroplasia: a comprehensive clinical review
inferredcites →https://doi.org/10.1186/s13023-021-02141-4openalex-extracted
Growth in achondroplasia including stature, weight, weight-for-height and head circumference from CLARITY: achondroplasia natural history study—a multi-center retrospective cohort study of achondroplasia in the US
inferredcites →https://doi.org/10.1371/journal.pone.0244368openalex-extracted
In vitro and in vivo characterization of Recifercept, a soluble fibroblast growth factor receptor 3, as treatment for achondroplasia
inferredcites →https://doi.org/10.1515/jpem-2020-0298openalex-extracted
Growth in achondroplasia, from birth to adulthood, analysed by the JPA-2 model
inferredcites →https://doi.org/10.21203/rs.3.rs-743762/v1openalex-extracted
inferredcites →https://doi.org/10.3390/ijms23115916openalex-extracted
Molecular Mechanism of Induction of Bone Growth by the C-Type Natriuretic Peptide
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