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Oxidative DNA Lesions Destabilize Centromeres and Drive Chromosome Instability

https://doi.org/10.2139/ssrn.6754412
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Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/mc.2940050406openalex-snapshot
Efficient protection against oxidative DNA damage in chromatin
inferredcites →https://doi.org/10.1006/jmbi.2001.4679openalex-snapshot
Structure of a C-rich strand fragment of the human centromeric satellite III: a pH-dependent intercalation topology
inferredcites →https://doi.org/10.1007/978-1-62703-236-0_4openalex-snapshot
p53 and Cell Cycle Effects After DNA Damage
inferredcites →https://doi.org/10.1007/s10577-025-09778-yopenalex-snapshot
The centromere: the punching bag of the chromosome
inferredcites →https://doi.org/10.1016/bs.mie.2017.03.004openalex-snapshot
Sequencing DNA for the Oxidatively Modified Base 8-Oxo-7,8-Dihydroguanine
inferredcites →https://doi.org/10.1016/j.canlet.2012.04.005openalex-snapshot
Oxidatively generated complex DNA damage: Tandem and clustered lesions
inferredcites →https://doi.org/10.1016/j.cell.2009.06.021openalex-snapshot
Mammalian Telomeres Resemble Fragile Sites and Require TRF1 for Efficient Replication
inferredcites →https://doi.org/10.1016/j.cell.2011.02.002openalex-snapshot
Epigenetic Centromere Propagation and the Nature of CENP-A Nucleosomes
inferredcites →https://doi.org/10.1016/j.cell.2013.08.062openalex-snapshot
The Proliferation-Quiescence Decision Is Controlled by a Bifurcation in CDK2 Activity at Mitotic Exit
inferredcites →https://doi.org/10.1016/j.cell.2017.01.002openalex-snapshot
Endogenous DNA Damage as a Source of Genomic Instability in Cancer
inferredcites →https://doi.org/10.1016/j.celrep.2018.08.092openalex-snapshot
Interplay of Histone Marks with Serine ADP-Ribosylation
inferredcites →https://doi.org/10.1016/j.cels.2017.09.015openalex-snapshot
Orchestration of DNA Damage Checkpoint Dynamics across the Human Cell Cycle
inferredcites →https://doi.org/10.1016/j.cotox.2017.10.009openalex-snapshot
DNA repair after oxidative stress: Current challenges
inferredcites →https://doi.org/10.1016/j.dnarep.2017.09.007openalex-snapshot
Not breathing is not an option: How to deal with oxidative DNA damage
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2012.08.593openalex-snapshot
The cross talk between pathways in the repair of 8-oxo-7,8-dihydroguanine in mouse and human cells
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2016.12.026openalex-snapshot
Chromatin associated mechanisms in base excision repair - nucleosome remodeling and DNA transcription, two key players
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2016.12.049openalex-snapshot
Formation and repair of oxidatively generated damage in cellular DNA
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2017.03.030openalex-snapshot
Oxidative DNA damage & repair: An introduction
inferredcites →https://doi.org/10.1016/j.mad.2018.03.013openalex-snapshot
The impact of oxidative DNA damage and stress on telomere homeostasis
inferredcites →https://doi.org/10.1016/j.molcel.2016.06.002openalex-snapshot
Temporal and Spatial Uncoupling of DNA Double Strand Break Repair Pathways within Mammalian Heterochromatin
inferredcites →https://doi.org/10.1016/j.molcel.2019.04.024openalex-snapshot
Targeted and Persistent 8-Oxoguanine Base Damage at Telomeres Promotes Telomere Loss and Crisis
inferredcites →https://doi.org/10.1016/j.molcel.2022.05.004openalex-snapshot
Hallmarks of DNA replication stress
inferredcites →https://doi.org/10.1016/j.molcel.2023.01.004openalex-snapshot
Centromeres as universal hotspots of DNA breakage, driving RAD51-mediated recombination during quiescence
inferredcites →https://doi.org/10.1016/j.molcel.2024.01.018openalex-snapshot
Specialized replication mechanisms maintain genome stability at human centromeres
inferredcites →https://doi.org/10.1016/j.molcel.2024.12.018openalex-snapshot
Centromere inactivation during aging can be rescued in human cells
inferredcites →https://doi.org/10.1016/j.tibs.2023.11.001openalex-snapshot
Causes and consequences of DNA single-strand breaks
inferredcites →https://doi.org/10.1016/j.xgen.2025.100819openalex-snapshot
Conservation of dichromatin organization along regional centromeres
inferredcites →https://doi.org/10.1016/s0021-9258(19)62715-8openalex-snapshot
Modulation of chromatin superstructure induced by poly(ADP-ribose) synthesis and degradation.
inferredcites →https://doi.org/10.1038/ncb2897openalex-snapshot
Causes and consequences of replication stress
inferredcites →https://doi.org/10.1038/ncb3344openalex-snapshot
Centromeric DNA replication reconstitution reveals DNA loops and ATR checkpoint suppression
inferredcites →https://doi.org/10.1038/nchembio.2180openalex-snapshot
Serine is a new target residue for endogenous ADP-ribosylation on histones
inferredcites →https://doi.org/10.1038/nmeth.3735openalex-snapshot
A genetically targetable near-infrared photosensitizer
inferredcites →https://doi.org/10.1038/nrc3344openalex-snapshot
Crosstalk between chromatin state and DNA damage response in cellular senescence and cancer
inferredcites →https://doi.org/10.1038/nrg.2016.157openalex-snapshot
Chromatin dynamics during the cell cycle at centromeres
inferredcites →https://doi.org/10.1038/nrm.2015.5openalex-snapshot
The molecular basis for centromere identity and function
inferredcites →https://doi.org/10.1038/nrm1963openalex-snapshot
Poly(ADP-ribose): novel functions for an old molecule
inferredcites →https://doi.org/10.1038/s41467-018-06545-yopenalex-snapshot
The dark side of centromeres: types, causes and consequences of structural abnormalities implicating centromeric DNA
inferredcites →https://doi.org/10.1038/s41467-022-30212-yopenalex-snapshot
High-throughput analysis of single human cells reveals the complex nature of DNA replication timing control
inferredcites →https://doi.org/10.1038/s41467-023-37341-yopenalex-snapshot
Excessive reactive oxygen species induce transcription-dependent replication stress
inferredcites →https://doi.org/10.1038/s41467-024-47222-7openalex-snapshot
PARP2 promotes Break Induced Replication-mediated telomere fragility in response to replication stress
inferredcites →https://doi.org/10.1038/s41467-025-56090-8openalex-snapshot
Mitotic chromatin marking governs the segregation of DNA damage
inferredcites →https://doi.org/10.1038/s41467-025-60391-3openalex-snapshot
Direct detection of 8-oxo-dG using nanopore sequencing
inferredcites →https://doi.org/10.1038/s41594-022-00790-yopenalex-snapshot
Telomeric 8-oxo-guanine drives rapid premature senescence in the absence of telomere shortening
inferredcites →https://doi.org/10.1038/s41598-023-41770-6openalex-snapshot
CRISPR imaging reveals chromatin fluctuation at the centromere region related to cellular senescence
inferredcites →https://doi.org/10.1073/pnas.2015634118openalex-snapshot
CENP-A chromatin prevents replication stress at centromeres to avoid structural aneuploidy
inferredcites →https://doi.org/10.1073/pnas.2505385122openalex-snapshot
Total whole-arm chromosome losses predict malignancy in human cancer
inferredcites →https://doi.org/10.1073/pnas.79.11.3423openalex-snapshot
Poly(ADP-ribosyl)ation of polynucleosomes causes relaxation of chromatin structure.
inferredcites →https://doi.org/10.1074/jbc.m116.726067openalex-snapshot
Preferential Phosphorylation on Old Histones during Early Mitosis in Human Cells
inferredcites →https://doi.org/10.1074/jbc.m200620200openalex-snapshot
Centromere Proteins Cenpa, Cenpb, and Bub3 Interact with Poly(ADP-ribose) Polymerase-1 Protein and Are Poly(ADP-ribosyl)ated
inferredcites →https://doi.org/10.1083/jcb.201306073openalex-snapshot
Higher-order unfolding of satellite heterochromatin is a consistent and early event in cell senescence
inferredcites →https://doi.org/10.1089/ars.2010.3466openalex-snapshot
Base Excision Repair and Lesion-Dependent Subpathways for Repair of Oxidative DNA Damage
inferredcites →https://doi.org/10.1093/carcin/bgh296openalex-snapshot
Senescence and immortalization: role of telomeres and telomerase
inferredcites →https://doi.org/10.1093/hmg/11.19.2319openalex-snapshot
Poly(ADP-ribose) polymerase 2 localizes to mammalian active centromeres and interacts with PARP-1, Cenpa, Cenpb and Bub3, but not Cenpc
inferredcites →https://doi.org/10.1093/nar/gkm1059openalex-snapshot
DNA replication stress, genome instability and aging
inferredcites →https://doi.org/10.1093/nar/gkn640openalex-snapshot
Hairpin structures formed by alpha satellite DNA of human centromeres are cleaved by human topoisomerase II
inferredcites →https://doi.org/10.1093/nar/gkp1247openalex-snapshot
Oxidative stress triggers the preferential assembly of base excision repair complexes on open chromatin regions
inferredcites →https://doi.org/10.1093/nar/gkt076openalex-snapshot
Mammalian Base Excision Repair: the Forgotten Archangel
inferredcites →https://doi.org/10.1093/nar/gkt1233openalex-snapshot
Novel method for site-specific induction of oxidative DNA damage reveals differences in recruitment of repair proteins to heterochromatin and euchromatin
inferredcites →https://doi.org/10.1093/narcan/zcad019openalex-snapshot
Molecular mechanisms protecting centromeres from self-sabotage and implications for cancer therapy
inferredcites →https://doi.org/10.1098/rsbl.2017.0463openalex-snapshot
Does oxidative stress shorten telomeres in vivo ? A review
inferredcites →https://doi.org/10.1139/o82-139openalex-snapshot
Hyper(ADP-ribosyl)ation of histone H1
inferredcites →https://doi.org/10.1158/0008-5472.can-09-3848openalex-snapshot
Three-Dimensional Cell Growth Confers Radioresistance by Chromatin Density Modification
inferredcites →https://doi.org/10.1371/journal.pgen.1000266openalex-snapshot
A Role for Oxidized DNA Precursors in Huntington's Disease–Like Striatal Neurodegeneration
inferredcites →https://doi.org/10.1371/journal.pgen.1000951openalex-snapshot
Characterization of Oxidative Guanine Damage and Repair in Mammalian Telomeres
inferredcites →https://doi.org/10.1371/journal.pgen.1003639openalex-snapshot
Defective Repair of Oxidative Base Lesions by the DNA Glycosylase Nth1 Associates with Multiple Telomere Defects
inferredcites →https://doi.org/10.1371/journal.pgen.1010306openalex-snapshot
Enrichment of centromeric DNA from human cells
inferredcites →https://doi.org/10.15252/msb.202211087openalex-snapshot
The molecular architecture of cell cycle arrest
inferredcites →https://doi.org/10.2174/156652412800792651openalex-snapshot
Oxidatively Generated DNA Lesions as Potential Biomarkers of In Vivo Oxidative Stress
inferredcites →https://doi.org/10.3390/genes11070810openalex-snapshot
Guarding the Genome: CENP-A-Chromatin in Health and Cancer
inferredcites →https://doi.org/10.3390/ijms20174258openalex-snapshot
Oxidative Stress: Role and Response of Short Guanine Tracts at Genomic Locations
inferredcites →https://doi.org/10.7554/elife.34334openalex-snapshot
Serine is the major residue for ADP-ribosylation upon DNA damage
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