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Coexpression of the Type 2 Diabetes Susceptibility Gene Variants KCNJ11 E23K and ABCC8 S1369A Alter the ATP and Sulfonylurea Sensitivities of the ATP-Sensitive K+ Channel
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/bf02658512openalex-snapshot
Sequence variations in the human Kir6.2 gene, a subunit of the beta-cell ATP-sensitive K-channel: no association with NIDDM in white caucasian subjects or evidence of abnormal function when expressed in vitro
inferredcites →https://doi.org/10.1007/s00125-006-0176-9openalex-snapshot
Risk of mortality and adverse cardiovascular outcomes in type 2 diabetes: a comparison of patients treated with sulfonylureas and metformin
inferredcites →https://doi.org/10.1007/s00424-006-0116-zopenalex-snapshot
ABCC8 and ABCC9: ABC transporters that regulate K+ channels
inferredcites →https://doi.org/10.1016/j.ejheart.2008.05.013openalex-snapshot
The Risk of Heart Failure in Patients with Type 2 Diabetes Treated with Oral Agent Monotherapy
inferredcites →https://doi.org/10.1016/j.tig.2008.09.004openalex-snapshot
Type 2 diabetes: new genes, new understanding
inferredcites →https://doi.org/10.1056/nejmoa032922openalex-snapshot
Activating Mutations in the Gene Encoding the ATP-Sensitive Potassium-Channel Subunit Kir6.2 and Permanent Neonatal Diabetes
inferredcites →https://doi.org/10.1056/nejmoa055068openalex-snapshot
Activating Mutations in theABCC8Gene in Neonatal Diabetes Mellitus
inferredcites →https://doi.org/10.1073/pnas.0707428104openalex-snapshot
Increased ATPase activity produced by mutations at arginine-1380 in nucleotide-binding domain 2 of ABCC8 causes neonatal diabetes
inferredcites →https://doi.org/10.1074/jbc.m804318200openalex-snapshot
Functional Clustering of Mutations in the Dimer Interface of the Nucleotide Binding Folds of the Sulfonylurea Receptor
inferredcites →https://doi.org/10.1093/hmg/ddi086openalex-snapshot
Relapsing diabetes can result from moderately activating mutations in KCNJ11
inferredcites →https://doi.org/10.1096/fj.06-6730hypopenalex-snapshot
Defining a binding pocket for sulfonylureas in ATP‐sensitive potassium channels
inferredcites →https://doi.org/10.1124/jpet.107.123224openalex-snapshot
Testing the Bipartite Model of the Sulfonylurea Receptor Binding Site: Binding of A-, B-, and A + B-Site Ligands
inferredcites →https://doi.org/10.1126/science.272.5269.1785openalex-snapshot
Adenosine Diphosphate as an Intracellular Regulator of Insulin Secretion
inferredcites →https://doi.org/10.2174/1381612054546879openalex-snapshot
Insulin Secretagogues, Sulfonylurea Receptors and KATP Channels
inferredcites →https://doi.org/10.2337/dc07-2248openalex-snapshot
Ser1369Ala Variant in Sulfonylurea Receptor Gene ABCC8 Is Associated With Antidiabetic Efficacy of Gliclazide in Chinese Type 2 Diabetic Patients
inferredcites →https://doi.org/10.2337/diabetes.48.6.1341openalex-snapshot
Identification of the high-affinity tolbutamide site on the SUR1 subunit of the K(ATP) channel.
inferredcites →https://doi.org/10.2337/diabetes.51.2007.s363openalex-snapshot
The Common Single Nucleotide Polymorphism E23K in KIR6.2 Sensitizes Pancreatic β-Cell ATP-Sensitive Potassium Channels Toward Activation Through Nucleoside Diphosphates
inferredcites →https://doi.org/10.2337/diabetes.51.3.875openalex-snapshot
KIR6.2 Polymorphism Predisposes to Type 2 Diabetes by Inducing Overactivity of Pancreatic β-Cell ATP-Sensitive K+ Channels
inferredcites →https://doi.org/10.2337/diabetes.52.10.2630openalex-snapshot
Kir6.2 Polymorphisms Sensitize β-Cell ATP-Sensitive Potassium Channels to Activation by Acyl CoAs
inferredcites →https://doi.org/10.2337/diabetes.52.2.568openalex-snapshot
Large-Scale Association Studies of Variants in Genes Encoding the Pancreatic β-Cell KATP Channel Subunits Kir6.2 (KCNJ11) and SUR1 (ABCC8) Confirm That the KCNJ11 E23K Variant Is Associated With Type 2 Diabetes
inferredcites →https://doi.org/10.2337/diabetes.52.2.573openalex-snapshot
The E23K Variant of Kir6.2 Associates With Impaired Post-OGTT Serum Insulin Response and Increased Risk of Type 2 Diabetes
inferredcites →https://doi.org/10.2337/diabetes.53.5.1360openalex-snapshot
Haplotype Structure and Genotype-Phenotype Correlations of the Sulfonylurea Receptor and the Islet ATP-Sensitive Potassium Channel Gene Region
inferredcites →https://doi.org/10.2337/diabetes.53.suppl_3.s176openalex-snapshot
Roles of ATP-Sensitive K+ Channels as Metabolic Sensors
inferredcites →https://doi.org/10.2337/diabetes.54.7.2070openalex-snapshot
Saturated and cis/trans Unsaturated Acyl CoA Esters Differentially Regulate Wild-Type and Polymorphic β-Cell ATP-Sensitive K+ Channels
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