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Chronic kidney disease alters vascular smooth muscle cell phenotype
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/jama.1992.03480130164050openalex-snapshot
Journal of the American Society of Nephrology
inferredcites →https://doi.org/10.1002/bjs.1800810904openalex-snapshot
Pathobiology of intimal hyperplasia
inferredcites →https://doi.org/10.1002/jcp.22132openalex-snapshot
The chromatin remodeling factor SRCAP modulates expression of prostate specific antigen and cellular proliferation in prostate cancer cells
inferredcites →https://doi.org/10.1016/j.bbrc.2011.11.144openalex-snapshot
Upregulation of BAD, a pro-apoptotic protein of the BCL2 family, in vascular smooth muscle cells exposed to uremic conditions
inferredcites →https://doi.org/10.1016/j.cell.2007.02.005openalex-snapshot
Chromatin Modifications and Their Function
inferredcites →https://doi.org/10.1016/j.devcel.2005.05.017openalex-snapshot
Phenotypic Modulation of Smooth Muscle Cells through Interaction of Foxo4 and Myocardin
inferredcites →https://doi.org/10.1016/j.jvs.2004.10.020openalex-snapshot
Chronic kidney disease and the risks of death, cardiovascular events, and hospitalization
inferredcites →https://doi.org/10.1016/j.jvs.2007.10.041openalex-snapshot
The natural history of autologous fistulas as first-time dialysis access in the KDOQI era
inferredcites →https://doi.org/10.1016/s0966-3274(97)80005-6openalex-snapshot
Smooth muscle migration in vascular development and pathogenesis
inferredcites →https://doi.org/10.1038/ki.2010.353openalex-snapshot
Chronic kidney disease aggravates arteriovenous fistula damage in rats
inferredcites →https://doi.org/10.1038/nm0410-396openalex-snapshot
Endoplasmic reticulum stress and atherosclerosis
inferredcites →https://doi.org/10.1038/nrneph.2013.76openalex-snapshot
Vascular access in haemodialysis: strengthening the Achilles' heel
inferredcites →https://doi.org/10.1053/ajkd.2002.34924openalex-snapshot
Cost analysis of ongoing care of patients with end-stage renal disease: The impact of dialysis modality and dialysis access
inferredcites →https://doi.org/10.1053/j.ackd.2009.06.009openalex-snapshot
Advances and New Frontiers in the Pathophysiology of Venous Neointimal Hyperplasia and Dialysis Access Stenosis
inferredcites →https://doi.org/10.1053/j.ajkd.2011.04.004openalex-snapshot
Cardiovascular Mortality in the General Population Versus Dialysis: A Glass Half Full or Empty?
inferredcites →https://doi.org/10.1056/nejmoa041031openalex-snapshot
Chronic Kidney Disease and the Risks of Death, Cardiovascular Events, and Hospitalization
inferredcites →https://doi.org/10.1093/ndt/gfg161openalex-snapshot
Uraemic medium accelerates proliferation but does not induce apoptosis of endothelial cells in culture
inferredcites →https://doi.org/10.1093/ndt/gfi214openalex-snapshot
Ultrasonographic measurement of intima-media thickness of radial artery in pre-dialysis uraemic patients: comparison with histological examination
inferredcites →https://doi.org/10.1093/ndt/gfq733openalex-snapshot
Severe venous neointimal hyperplasia prior to dialysis access surgery
inferredcites →https://doi.org/10.1152/ajpcell.00439.2010openalex-snapshot
Anti-inflammatory cytokine interleukin-19 inhibits smooth muscle cell migration and activation of cytoskeletal regulators of VSMC motility
inferredcites →https://doi.org/10.1152/physiolgenomics.00160.2011openalex-snapshot
Interleukin-1β modulates smooth muscle cell phenotype to a distinct inflammatory state relative to PDGF-DD via NF-κB-dependent mechanisms
inferredcites →https://doi.org/10.1152/physrev.00041.2003openalex-snapshot
Molecular Regulation of Vascular Smooth Muscle Cell Differentiation in Development and Disease
inferredcites →https://doi.org/10.1152/physrev.1995.75.3.487openalex-snapshot
Regulation of differentiation of vascular smooth muscle cells
inferredcites →https://doi.org/10.1159/000327131openalex-snapshot
Does Uremia Cause Vascular Dysfunction
inferredcites →https://doi.org/10.1159/000479439openalex-snapshot
Lanthanum Chloride Inhibits LPS Mediated Expressions of Pro-Inflammatory Cytokines and Adhesion Molecules in HUVECs: Involvement of NF-κB-Jmjd3 Signaling
inferredcites →https://doi.org/10.1161/01.cir.94.6.1283openalex-snapshot
Enhanced cellular proliferation in intact stenotic lesions derived from human arteriovenous fistulas and peripheral bypass grafts. Does it correlate with flow parameters?
inferredcites →https://doi.org/10.1172/jci10522openalex-snapshot
Molecular mechanisms of decreased smooth muscle differentiation marker expression after vascular injury
inferredcites →https://doi.org/10.1540/jsmr.39.95openalex-snapshot
Intimal Changes in the Cephalic Vein of Renal Failure Patients before Arterio-Venous Fistula (AVF) Construction
inferredcites →https://doi.org/10.1681/asn.2007121312openalex-snapshot
CKD Accelerates Development of Neointimal Hyperplasia in Arteriovenous Fistulas
inferredcites →https://doi.org/10.1681/asn.v74523openalex-snapshot
Hemodialysis vascular access morbidity.
inferredcites →https://doi.org/10.7326/0003-4819-141-2-200407200-00007openalex-snapshot
The Incidence of End-Stage Renal Disease Is Increasing Faster than the Prevalence of Chronic Renal Insufficiency
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Understanding Intimal Hyperplasia Biology in Hemodialysis Access
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Regenerative potential of platelets in patients with chronic kidney disease
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Caspase-1 Plays a Critical Role in Accelerating Chronic Kidney Disease-Promoted Neointimal Hyperplasia in the Carotid Artery
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Immuno-regenerative biomaterials for in situ cardiovascular tissue engineering – Do patient characteristics warrant precision engineering?
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Uremia modulates the phenotype of aortic smooth muscle cells
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Uremic Toxins and Cardiovascular System
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ApoB and apoM – New aspects of lipoprotein biology in uremia-induced atherosclerosis
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Significance of Kidney Disease in Cardiovascular Disease Patients
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Nitric Oxide-releasing Nanofibers Prevent Restenosis After Arterial Injury in a Renal Failure Model
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Emerging role of myostatin and its inhibition in the setting of chronic kidney disease
inferred← citeshttps://doi.org/10.1016/j.lfs.2020.117862openalex-snapshot
Biochemical analysis of ectonucleotidases on primary rat vascular smooth muscle cells and in silico investigation of their role in vascular diseases
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MicroRNAs in vascular smooth muscle cells: Mechanisms, therapeutic potential, and advances in delivery systems
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2020.107715openalex-snapshot
Circular RNAs are a novel type of non-coding RNAs in ROS regulation, cardiovascular metabolic inflammations and cancers
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2024.108604openalex-snapshot
Protein-rich foods, sea foods, and gut microbiota amplify immune responses in chronic diseases and cancers – Targeting PERK as a novel therapeutic strategy for chronic inflammatory diseases, neurodegenerative disorders, and cancer
inferred← citeshttps://doi.org/10.1016/j.redox.2019.101373openalex-snapshot
Anti-inflammatory cytokines IL-35 and IL-10 block atherogenic lysophosphatidylcholine-induced, mitochondrial ROS-mediated innate immune activation, but spare innate immune memory signature in endothelial cells
inferred← citeshttps://doi.org/10.1016/j.redox.2020.101460openalex-snapshot
End-stage renal disease is different from chronic kidney disease in upregulating ROS-modulated proinflammatory secretome in PBMCs - A novel multiple-hit model for disease progression
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Procaspase-1 patrolled to the nucleus of proatherogenic lipid LPC-activated human aortic endothelial cells induces ROS promoter CYP1B1 and strong inflammation
inferred← citeshttps://doi.org/10.1016/j.tice.2023.102286openalex-snapshot
Zic family member 3 attenuates oxidative stress-induced vascular smooth muscle cell apoptosis in patients with chronic kidney disease
inferred← citeshttps://doi.org/10.1038/s41598-017-06816-6openalex-snapshot
Uremia does not affect neointima formation in mice
inferred← citeshttps://doi.org/10.1053/j.ajkd.2016.02.044openalex-snapshot
Pre-existing and Postoperative Intimal Hyperplasia and Arteriovenous Fistula Outcomes
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Lysophospholipids induce innate immune transdifferentiation of endothelial cells, resulting in prolonged endothelial activation
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Hyperlipidemia and endothelial dysfunction: effects of eNOS phosphorylation and vascular ultrastructure in normoglycemic Psammomys obesus
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Histological Analysis of Arterial and Venous Grafts Used in Coronary Bypass for Patients With Renal Insufficiency: A Prospective Multicentre Observational Study
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Single-cell mapper (scMappR): using scRNA-seq to infer the cell-type specificities of differentially expressed genes
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Non‐coding RNAs to treat vascular smooth muscle cell dysfunction
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The Effect of Mesenchymal Stem Cells on the Reactivity of Smooth Muscle Cells of Pial Arteries in Nephrectomized Rats
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Cellular and molecular mechanisms underlying hemodialysis arteriovenous fistula dysfunction and approaches to promote maturation: a vascular perspective
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Uremic serum exposure leads to differential phenotypic switch in porcine arterial and venous smooth muscle cells
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29 m6A-RNA Methylation (Epitranscriptomic) Regulators Are Regulated in 41 Diseases including Atherosclerosis and Tumors Potentially via ROS Regulation – 102 Transcriptomic Dataset Analyses
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IL-35 (Interleukin-35) Suppresses Endothelial Cell Activation by Inhibiting Mitochondrial Reactive Oxygen Species-Mediated Site-Specific Acetylation of H3K14 (Histone 3 Lysine 14)
inferred← citeshttps://doi.org/10.1161/atvbaha.120.314330openalex-snapshot
Vascular Endothelial Cells and Innate Immunity
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IL-35 promotes CD4+Foxp3+ Tregs and inhibits atherosclerosis via maintaining CCR5-amplified Treg-suppressive mechanisms
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Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways
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Caspase-4/11 promotes hyperlipidemia and chronic kidney disease–accelerated vascular inflammation by enhancing trained immunity
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Factors affecting arteriovenous fistula dysfunction: A narrative review
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Alzheimer's disease as an auto-innate immune pathology with potential cell trans-differentiation and enhanced trained immunity in 3xTg-AD mouse model
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An overview of AVF maturation and endothelial dysfunction in an advanced renal failure
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Liraglutide Reduces Both Atherosclerosis and Kidney Inflammation in Moderately Uremic LDLr-/- Mice
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Uremic toxins are conditional danger- or homeostasis-associated molecular patterns
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Co-signaling receptors regulate T-cell plasticity and immune tolerance
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Mécanismes impliqués dans le remodelage artériel associé à la maladie rénale chronique
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