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Uremic toxins are conditional danger- or homeostasis-associated molecular patterns
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/bdd.1834openalex-snapshot
Albumin is the main plasma binding protein for indoxyl sulfate and p‐cresyl sulfate
inferredcites →https://doi.org/10.1002/mas.20323openalex-snapshot
Update of uremic toxin research by mass spectrometry
inferredcites →https://doi.org/10.1007/s12265-015-9664-yopenalex-snapshot
Metabolic Diseases Downregulate the Majority of Histone Modification Enzymes, Making a Few Upregulated Enzymes Novel Therapeutic Targets—“Sand Out and Gold Stays”
inferredcites →https://doi.org/10.1007/s12265-016-9683-3openalex-snapshot
Caspase-1 Plays a Critical Role in Accelerating Chronic Kidney Disease-Promoted Neointimal Hyperplasia in the Carotid Artery
inferredcites →https://doi.org/10.1007/s12265-016-9700-6openalex-snapshot
Lysophospholipid Receptors, as Novel Conditional Danger Receptors and Homeostatic Receptors Modulate Inflammation—Novel Paradigm and Therapeutic Potential
inferredcites →https://doi.org/10.1016/j.atherosclerosis.2008.07.041openalex-snapshot
Expression of TCTP antisense in CD25high regulatory T cells aggravates cuff-injured vascular inflammation
inferredcites →https://doi.org/10.1016/j.bcp.2012.07.033openalex-snapshot
A uremic toxin, 3-carboxy-4-methyl-5-propyl-2-furanpropionate induces cell damage to proximal tubular cells via the generation of a radical intermediate
inferredcites →https://doi.org/10.1016/j.cardfail.2006.11.009openalex-snapshot
High Serum Level of Pentosidine, an Advanced Glycation End Product (AGE), is a Risk Factor of Patients with Heart Failure
inferredcites →https://doi.org/10.1016/j.cell.2015.11.055openalex-snapshot
Non-lethal Inhibition of Gut Microbial Trimethylamine Production for the Treatment of Atherosclerosis
inferredcites →https://doi.org/10.1016/j.cmet.2012.12.011openalex-snapshot
Trimethylamine-N-Oxide, a Metabolite Associated with Atherosclerosis, Exhibits Complex Genetic and Dietary Regulation
inferredcites →https://doi.org/10.1016/j.ddstr.2008.11.003openalex-snapshot
Vascular inflammation and atherogenesis are activated via receptors for PAMPs and suppressed by regulatory T cells
inferredcites →https://doi.org/10.1016/j.jbiosc.2014.03.001openalex-snapshot
Dietary trimethylamine N-oxide exacerbates impaired glucose tolerance in mice fed a high fat diet
inferredcites →https://doi.org/10.1016/j.mce.2010.06.018openalex-snapshot
What are nuclear receptor ligands?
inferredcites →https://doi.org/10.1016/j.tcm.2008.11.004openalex-snapshot
Monocyte-Endothelial Cell Interactions in the Development of Atherosclerosis
inferredcites →https://doi.org/10.1016/s0026-895x(25)10691-3openalex-snapshot
The Characterization of Two Specific Drug Binding Sites on Human Serum Albumin
inferredcites →https://doi.org/10.1016/s0145-4145(08)01030-7openalex-snapshot
High Serum Level of Pentosidine, an Advanced Glycation End Product (AGE), is a Risk Factor of Patients with Heart Failure
inferredcites →https://doi.org/10.1038/ki.1992.244openalex-snapshot
Extraction of protein bound ligands from azotemic sera: Comparison of 12 deproteinization methods
inferredcites →https://doi.org/10.1038/ki.2010.278openalex-snapshot
Exosomes/microvesicles as a mechanism of cell-to-cell communication
inferredcites →https://doi.org/10.1038/nature09922openalex-snapshot
Gut flora metabolism of phosphatidylcholine promotes cardiovascular disease
inferredcites →https://doi.org/10.1038/nm.3145openalex-snapshot
Intestinal microbiota metabolism of l-carnitine, a nutrient in red meat, promotes atherosclerosis
inferredcites →https://doi.org/10.1038/nrcardio.2013.15openalex-snapshot
Management of cardiovascular disease in patients with kidney disease
inferredcites →https://doi.org/10.1038/nrneph.2014.145openalex-snapshot
Economic evaluation of dialysis therapies
inferredcites →https://doi.org/10.1038/nrneph.2015.191openalex-snapshot
The gut microbiome, diet, and links to cardiometabolic and chronic disorders
inferredcites →https://doi.org/10.1046/j.1523-1755.1998.00944.xopenalex-snapshot
RNA metabolism in uremic patients: Accumulation of modified ribonucleosides in uremic serum
inferredcites →https://doi.org/10.1046/j.1523-1755.63.s84.43.xopenalex-snapshot
New insights in uremic toxins
inferredcites →https://doi.org/10.1053/j.jrn.2011.10.023openalex-snapshot
Carbamylated Low-Density Lipoprotein: Nontraditional Risk Factor for Cardiovascular Events in Patients With Chronic Kidney Disease
inferredcites →https://doi.org/10.1073/pnas.0812874106openalex-snapshot
Metabolomics analysis reveals large effects of gut microflora on mammalian blood metabolites
inferredcites →https://doi.org/10.1073/pnas.1219002110openalex-snapshot
Depletion of cellular polyamines, spermidine and spermine, causes a total arrest in translation and growth in mammalian cells
inferredcites →https://doi.org/10.1074/jbc.m113.460626openalex-snapshot
Identification of Novel Pretranslational Regulatory Mechanisms for NF-κB Activation
inferredcites →https://doi.org/10.1074/jbc.m115.641191openalex-snapshot
Inhibition of Caspase-1 Activation in Endothelial Cells Improves Angiogenesis
inferredcites →https://doi.org/10.1074/jbc.m115.663286openalex-snapshot
Interleukin-35 Inhibits Endothelial Cell Activation by Suppressing MAPK-AP-1 Pathway
inferredcites →https://doi.org/10.1093/ckj/sfv034openalex-snapshot
Uraemic toxins and new methods to control their accumulation: game changers for the concept of dialysis adequacy
inferredcites →https://doi.org/10.1093/clinchem/43.12.2274openalex-snapshot
Effects of uremic toxins and fatty acids on serum protein binding of furosemide: possible mechanism of the binding defect in uremia
inferredcites →https://doi.org/10.1093/eurheartj/ehu111openalex-snapshot
Carbamylated low-density lipoprotein induces endothelial dysfunction
inferredcites →https://doi.org/10.1093/eurheartj/ehu122openalex-snapshot
Interaction of carbamylated LDL with LOX-1 in the induction of endothelial dysfunction and atherosclerosis: Figure 1
inferredcites →https://doi.org/10.1093/ndt/14.12.2813openalex-snapshot
p-Cresol: a toxin revealing many neglected but relevant aspects of uraemic toxicity
inferredcites →https://doi.org/10.1096/fj.14-258780openalex-snapshot
Decreased nuclear receptor activity and epigenetic modulation associates with down‐regulation of hepatic drug‐metabolizing enzymes in chronic kidney disease
inferredcites →https://doi.org/10.1096/fj.14-267989openalex-snapshot
Hyperhomocysteinemia suppresses bone marrow CD34 + /VEGF receptor 2 + cells and inhibits progenitor cell mobilization and homing to injured vasculature—a role of β1‐integrin in progenitor cell migration and adhesion
inferredcites →https://doi.org/10.1111/j.1525-139x.2009.00576.xopenalex-snapshot
PROGRESS IN UREMIC TOXIN RESEARCH: Protein‐Bound Toxins—Update 2009
inferredcites →https://doi.org/10.1111/j.1525-139x.2011.00925.xopenalex-snapshot
Vascular Incompetence in Dialysis Patients—Protein‐Bound Uremic Toxins and Endothelial Dysfunction
inferredcites →https://doi.org/10.1124/dmd.116.070615openalex-snapshot
Trimethylamine and Trimethylamine N-Oxide, a Flavin-Containing Monooxygenase 3 (FMO3)-Mediated Host-Microbiome Metabolic Axis Implicated in Health and Disease
inferredcites →https://doi.org/10.1159/000094446openalex-snapshot
Reduced Capacity of Mononuclear Cells to Synthesize Cytokines against an Inflammatory Stimulus in Uremic Patients
inferredcites →https://doi.org/10.1159/000351758openalex-snapshot
Cardiovascular Burden Associated with Uremic Toxins in Patients with Chronic Kidney Disease
inferredcites →https://doi.org/10.1159/000369817openalex-snapshot
Uremic Toxins and Their Effects on Multiple Organ Systems
inferredcites →https://doi.org/10.1161/01.res.0000141775.67189.98openalex-snapshot
Pathophysiology of Vascular Calcification in Chronic Kidney Disease
inferredcites →https://doi.org/10.1161/atvbaha.115.305282openalex-snapshot
Early Hyperlipidemia Promotes Endothelial Activation via a Caspase-1-Sirtuin 1 Pathway
inferredcites →https://doi.org/10.1161/atvbaha.115.306964openalex-snapshot
Mitochondrial Reactive Oxygen Species Mediate Lysophosphatidylcholine-Induced Endothelial Cell Activation
inferredcites →https://doi.org/10.1161/circresaha.112.269472openalex-snapshot
Severe Hyperhomocysteinemia Promotes Bone Marrow–Derived and Resident Inflammatory Monocyte Differentiation and Atherosclerosis in LDLr/CBS-Deficient Mice
inferredcites →https://doi.org/10.1161/circresaha.116.305360openalex-snapshot
Gut Microbiota-Dependent Trimethylamine N -Oxide (TMAO) Pathway Contributes to Both Development of Renal Insufficiency and Mortality Risk in Chronic Kidney Disease
inferredcites →https://doi.org/10.1161/circresaha.116.308501openalex-snapshot
Caspase-1 Inflammasome Activation Mediates Homocysteine-Induced Pyrop-Apoptosis in Endothelial Cells
inferredcites →https://doi.org/10.1161/circulationaha.106.678342openalex-snapshot
Chronic Kidney Disease
inferredcites →https://doi.org/10.1177/039463200902200208openalex-snapshot
Inflammasomes are Differentially Expressed in Cardiovascular and other Tissues
inferredcites →https://doi.org/10.1186/1479-5876-7-17openalex-snapshot
RAGE (Receptor for Advanced Glycation Endproducts), RAGE Ligands, and their role in Cancer and Inflammation
inferredcites →https://doi.org/10.1186/1756-8722-5-66openalex-snapshot
MicroRNAs and Toll-like Receptor/Interleukin-1 Receptor Signaling
inferredcites →https://doi.org/10.1186/s13045-014-0080-6openalex-snapshot
Immunosuppressive/anti-inflammatory cytokines directly and indirectly inhibit endothelial dysfunction- a novel mechanism for maintaining vascular function
inferredcites →https://doi.org/10.1186/s13045-016-0351-5openalex-snapshot
Novel extracellular and nuclear caspase-1 and inflammasomes propagate inflammation and regulate gene expression: a comprehensive database mining study
inferredcites →https://doi.org/10.1186/s40591-016-0048-8openalex-snapshot
Albumin-based drug delivery: harnessing nature to cure disease
inferredcites →https://doi.org/10.1186/s41038-015-0001-0openalex-snapshot
Pathological conditions re-shape physiological Tregs into pathological Tregs
inferredcites →https://doi.org/10.1371/journal.pone.0016957openalex-snapshot
The Human Serum Metabolome
inferredcites →https://doi.org/10.1371/journal.pone.0135657openalex-snapshot
An Enlarged Profile of Uremic Solutes
inferredcites →https://doi.org/10.1681/asn.2014101047openalex-snapshot
Uremic Toxicity of Advanced Glycation End Products in CKD
inferredcites →https://doi.org/10.2215/cjn.12931212openalex-snapshot
Comparison of Regulatory T Cells in Hemodialysis Patients and Healthy Controls
inferredcites →https://doi.org/10.2337/db14-0809openalex-snapshot
Hyperhomocysteinemia Potentiates Hyperglycemia-Induced Inflammatory Monocyte Differentiation and Atherosclerosis
inferredcites →https://doi.org/10.2353/ajpath.2009.081036openalex-snapshot
Hyperhomocysteinemia Increases β-Amyloid by Enhancing Expression of γ-Secretase and Phosphorylation of Amyloid Precursor Protein in Rat Brain
inferredcites →https://doi.org/10.2741/3217openalex-snapshot
Higher expression of Bax in regulatory T cells increases vascular inflammation
inferredcites →https://doi.org/10.2741/4127openalex-snapshot
Inflammasomes: sensors of metabolic stresses for vascular inflammation
inferredcites →https://doi.org/10.2741/4337openalex-snapshot
Chronic kidney disease alters vascular smooth muscle cell phenotype
inferredcites →https://doi.org/10.2741/4383openalex-snapshot
Caspase-1 mediates hyperlipidemia-weakened progenitor cell vessel repair
inferredcites →https://doi.org/10.3389/fimmu.2014.00465openalex-snapshot
Exosomes: A Potential Key Target in Cardio-Renal Syndrome
inferredcites →https://doi.org/10.3389/fimmu.2015.00422openalex-snapshot
DAMPs from Cell Death to New Life
inferredcites →https://doi.org/10.3389/fphys.2014.00299openalex-snapshot
Unraveling the mysteries of serum albumin—more than just a serum protein
inferredcites →https://doi.org/10.3390/toxins6020665openalex-snapshot
Protein-Bound Uremic Toxins: New Culprits of Cardiovascular Events in Chronic Kidney Disease Patients
inferredcites →https://doi.org/10.3390/toxins6030934openalex-snapshot
The Aryl Hydrocarbon Receptor-Activating Effect of Uremic Toxins from Tryptophan Metabolism: A New Concept to Understand Cardiovascular Complications of Chronic Kidney Disease
inferredcites →https://doi.org/10.3810/pgm.2000.07.1152openalex-snapshot
Trends in end-stage renal disease
inferredcites →https://doi.org/10.3945/ajcn.115.121269openalex-snapshot
Plasma trimethylamine N-oxide concentration is associated with choline, phospholipids, and methyl metabolism
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4639349openalex-extracted
Protein-Rich Foods, Sea Foods, and Gut Microbiota Amplify Immune Responses in Chronic Diseases and Cancers – Targeting Perk as a Novel Therapeutic Strategy for Cancer
inferred← citeshttps://doi.org/10.1002/inm3.70050openalex-snapshot
A New Paradigm: 46 Structural Cell Types Function as Environment‐Supporting Innate Immune Cells, With Endothelial and Vascular Smooth Muscle Cells as Key Prototypes
inferred← citeshttps://doi.org/10.1002/inm3.70088openalex-snapshot
Challenging the Leukocyte‐Centric Model of Innate Immunity: The Emerging Role of Structural Cells in Immune Defense
inferred← citeshttps://doi.org/10.1007/978-3-031-41192-2_18openalex-snapshot
Uric Acid in Inflammation and the Pathogenesis of Atherosclerosis: Lessons for Cholesterol from the Land of Gout
inferred← citeshttps://doi.org/10.1007/s12012-026-10189-6openalex-snapshot
Trained Immunity as a Mechanistic Bridge Between Uremic Toxins, Atherosclerosis, and Vascular Calcification in Chronic Kidney Disease
inferred← citeshttps://doi.org/10.1007/s13668-024-00535-xopenalex-snapshot
Production of Toxins by the Gut Microbiota: The Role of Dietary Protein
inferred← citeshttps://doi.org/10.1016/j.cca.2019.08.030openalex-snapshot
Chronic kidney disease: Biomarker diagnosis to therapeutic targets
inferred← citeshttps://doi.org/10.1016/j.cyto.2017.06.003openalex-snapshot
IL-35, as a newly proposed homeostasis-associated molecular pattern, plays three major functions including anti-inflammatory initiator, effector, and blocker in cardiovascular diseases
inferred← citeshttps://doi.org/10.1016/j.intimp.2025.115236openalex-snapshot
NEK7 mediates ESRD-associated endothelial injury by activating the NLRP3 inflammasome
inferred← citeshttps://doi.org/10.1016/j.jsps.2023.101818openalex-snapshot
Prunetin in a GPR30-dependent manner mitigates renal ischemia/reperfusion injury in rats via interrupting indoxyl sulfate/TLR4/TRIF, RIPK1/RIPK3/MLKL, and RIPK3/PGAM5/DRP-1 crosstalk
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2020.107715openalex-snapshot
Circular RNAs are a novel type of non-coding RNAs in ROS regulation, cardiovascular metabolic inflammations and cancers
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2024.108604openalex-snapshot
Protein-rich foods, sea foods, and gut microbiota amplify immune responses in chronic diseases and cancers – Targeting PERK as a novel therapeutic strategy for chronic inflammatory diseases, neurodegenerative disorders, and cancer
inferred← citeshttps://doi.org/10.1016/j.redox.2019.101373openalex-snapshot
Anti-inflammatory cytokines IL-35 and IL-10 block atherogenic lysophosphatidylcholine-induced, mitochondrial ROS-mediated innate immune activation, but spare innate immune memory signature in endothelial cells
inferred← citeshttps://doi.org/10.1016/j.redox.2020.101460openalex-snapshot
End-stage renal disease is different from chronic kidney disease in upregulating ROS-modulated proinflammatory secretome in PBMCs - A novel multiple-hit model for disease progression
inferred← citeshttps://doi.org/10.1016/j.redox.2020.101696openalex-snapshot
ROS systems are a new integrated network for sensing homeostasis and alarming stresses in organelle metabolic processes
inferred← citeshttps://doi.org/10.1016/j.redox.2021.102142openalex-snapshot
Procaspase-1 patrolled to the nucleus of proatherogenic lipid LPC-activated human aortic endothelial cells induces ROS promoter CYP1B1 and strong inflammation
inferred← citeshttps://doi.org/10.1016/j.redox.2023.102771openalex-snapshot
Early hyperlipidemia triggers metabolomic reprogramming with increased SAH, increased acetyl-CoA-cholesterol synthesis, and decreased glycolysis
inferred← citeshttps://doi.org/10.1016/j.redox.2024.103331openalex-snapshot
A new paradigm in intracellular immunology: Mitochondria emerging as leading immune organelles
inferred← citeshttps://doi.org/10.1016/j.redox.2025.103724openalex-snapshot
Uremic toxin receptor NR1H3 contributes to hyperlipidemia- and chronic kidney disease-accelerated vascular inflammation, which is partially suppressed by novel YBX2 anti-ROS pathway
inferred← citeshttps://doi.org/10.1084/jem.20211463openalex-snapshot
Inhibitory pattern recognition receptors
inferred← citeshttps://doi.org/10.1097/shk.0000000000002004openalex-snapshot
BACTERIAL PATHOGEN–ASSOCIATED MOLECULAR PATTERNS UPREGULATE HUMAN GLUCOCORTICOID RECEPTOR EXPRESSION IN PERIPHERAL BLOOD MONONUCLEAR CELLS
inferred← citeshttps://doi.org/10.1155/2022/1433323openalex-snapshot
29 m6A-RNA Methylation (Epitranscriptomic) Regulators Are Regulated in 41 Diseases including Atherosclerosis and Tumors Potentially via ROS Regulation – 102 Transcriptomic Dataset Analyses
inferred← citeshttps://doi.org/10.1172/jci.insight.152511openalex-snapshot
IL-35 promotes CD4+Foxp3+ Tregs and inhibits atherosclerosis via maintaining CCR5-amplified Treg-suppressive mechanisms
inferred← citeshttps://doi.org/10.1172/jci.insight.158183openalex-snapshot
Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways
inferred← citeshttps://doi.org/10.1172/jci.insight.177229openalex-snapshot
Caspase-4/11 promotes hyperlipidemia and chronic kidney disease–accelerated vascular inflammation by enhancing trained immunity
inferred← citeshttps://doi.org/10.1186/s13045-017-0526-8openalex-snapshot
A comprehensive data mining study shows that most nuclear receptors act as newly proposed homeostasis-associated molecular pattern receptors
inferred← citeshttps://doi.org/10.22259/2639-3573.0201002openalex-snapshot
Accelerated Atherosclerosis in the Setting of Chronic Kidney Disease
inferred← citeshttps://doi.org/10.3389/fcvm.2021.713170openalex-snapshot
Organelle Crosstalk Regulators Are Regulated in Diseases, Tumors, and Regulatory T Cells: Novel Classification of Organelle Crosstalk Regulators
inferred← citeshttps://doi.org/10.3389/fcvm.2022.892288openalex-snapshot
Editorial: Highlights for Cardiovascular Therapeutics in 2021 – Trained Immunity, Immunometabolism, Gender Differences of Cardiovascular Diseases, and Novel Targets of Cardiovascular Therapeutics
inferred← citeshttps://doi.org/10.3389/fcvm.2023.1184030openalex-snapshot
Editorial: Insights in cardiovascular therapeutics 2022—cardiovascular innate immunity
inferred← citeshttps://doi.org/10.3389/fcvm.2026.1782258openalex-snapshot
Reduced R-loop abundance at proinflammatory loci: a shared epigenetic mechanism in inflammatory and metabolic diseases
inferred← citeshttps://doi.org/10.3389/fimmu.2020.554301openalex-snapshot
Approaching Inflammation Paradoxes—Proinflammatory Cytokine Blockages Induce Inflammatory Regulators
inferred← citeshttps://doi.org/10.3389/fimmu.2020.632239openalex-snapshot
Tissue Treg Secretomes and Transcription Factors Shared With Stem Cells Contribute to a Treg Niche to Maintain Treg-Ness With 80% Innate Immune Pathways, and Functions of Immunosuppression and Tissue Repair
inferred← citeshttps://doi.org/10.3389/fimmu.2021.678201openalex-snapshot
Canonical Secretomes, Innate Immune Caspase-1-, 4/11-Gasdermin D Non-Canonical Secretomes and Exosomes May Contribute to Maintain Treg-Ness for Treg Immunosuppression, Tissue Repair and Modulate Anti-Tumor Immunity via ROS Pathways
inferred← citeshttps://doi.org/10.3389/fimmu.2022.1035497openalex-snapshot
Editorial: Endothelial cells as innate immune cells
inferred← citeshttps://doi.org/10.3389/fimmu.2022.858256openalex-snapshot
Aorta in Pathologies May Function as an Immune Organ by Upregulating Secretomes for Immune and Vascular Cell Activation, Differentiation and Trans-Differentiation—Early Secretomes may Serve as Drivers for Trained Immunity
inferred← citeshttps://doi.org/10.3389/fimmu.2023.1348238openalex-snapshot
Innate immunity of vascular smooth muscle cells contributes to two-wave inflammation in atherosclerosis, twin-peak inflammation in aortic aneurysms and trans-differentiation potential into 25 cell types
inferred← citeshttps://doi.org/10.3389/fnagi.2026.1871569openalex-snapshot
Atherosclerotic-dose TMAO accentuates redox imbalances and motor dysfunctions in a MPTP mouse model of Parkinson’s disease
inferred← citeshttps://doi.org/10.3389/fnmol.2025.1605012openalex-snapshot
Organelle stresses and energetic metabolisms promote endothelial–to–mesenchymal transition and fibrosis via upregulating FOSB and MEOX1 in Alzheimer’s disease
inferred← citeshttps://doi.org/10.3389/fphys.2018.00516openalex-snapshot
DNA Checkpoint and Repair Factors Are Nuclear Sensors for Intracellular Organelle Stresses—Inflammations and Cancers Can Have High Genomic Risks
inferred← citeshttps://doi.org/10.3389/fphys.2021.686249openalex-snapshot
Uremic Toxins: An Alarming Danger Concerning the Cardiovascular System
inferred← citeshttps://doi.org/10.3390/antiox11020246openalex-snapshot
Involvement of Inflammasome Components in Kidney Disease
inferred← citeshttps://doi.org/10.3390/cells12111482openalex-snapshot
Chronic Kidney Disease Transdifferentiates Veins into a Specialized Immune–Endocrine Organ with Increased MYCN-AP1 Signaling
inferred← citeshttps://doi.org/10.3390/jcm11133759openalex-snapshot
Immune System Dysfunction and Inflammation in Hemodialysis Patients: Two Sides of the Same Coin
inferred← citeshttps://doi.org/10.3390/life12050769openalex-snapshot
Translational Medicine in Uremic Vascular Calcification: Scavenging ROS Attenuates p-Cresyl Sulfate-Activated Caspase-1, NLRP3 Inflammasome and Eicosanoid Inflammation in Human Arterial Smooth Muscle Cells
inferred← citeshttps://doi.org/10.3390/medsci12020031openalex-snapshot
Interleukin-6 as a Director of Immunological Events and Tissue Regenerative Capacity in Hemodialyzed Diabetes Patients
inferred← citeshttps://doi.org/10.3390/nu11081821openalex-snapshot
The Microbial Metabolite Trimethylamine N-Oxide Links Vascular Dysfunctions and the Autoimmune Disease Rheumatoid Arthritis
inferred← citeshttps://doi.org/10.3390/toxins17070345openalex-snapshot
Increased Cardiovascular Mortality in Hemodialysis: The Role of Chronic Inflammation, Complement Activation, and Non-Biocompatibility
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