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Interaction between Cytomegalovirus and Herpes Simplex Virus Type 1 Associated with the Risk of Alzheimer’s Disease Development

https://doi.org/10.3233/jad-161305
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/jmv.23397openalex-snapshot
Reactivation of herpes simplex virus type 1 is associated with cytomegalovirus and age
inferredcites →https://doi.org/10.1007/s10522-014-9538-8openalex-snapshot
β-Amyloid peptides display protective activity against the human Alzheimer’s disease-associated herpes simplex virus-1
inferredcites →https://doi.org/10.1007/s10552-011-9799-3openalex-snapshot
Human immunoglobulin G levels of viruses and associated glioma risk
inferredcites →https://doi.org/10.1007/s10654-014-9977-1openalex-snapshot
The Consortium on Health and Ageing: Network of Cohorts in Europe and the United States (CHANCES) project—design, population and data harmonization of a large-scale, international study
inferredcites →https://doi.org/10.1016/j.jalz.2014.04.522openalex-snapshot
Reactivated herpes simplex infection increases the risk of Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.jalz.2014.07.157openalex-snapshot
Herpes simplex infection and the risk of Alzheimer's disease: A nested case-control study
inferredcites →https://doi.org/10.1016/j.mad.2016.06.005openalex-snapshot
Cytomegalovirus driven immunosenescence—An immune phenotype with or without clinical impact?
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2005.01.018openalex-snapshot
Beta-amyloid deposition and Alzheimer's type changes induced by Borrelia spirochetes
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2010.12.010openalex-snapshot
Herpes simplex virus type I induces the accumulation of intracellular β-amyloid in autophagic compartments and the inhibition of the non-amyloidogenic pathway in human neuroblastoma cells
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2013.06.024openalex-snapshot
Herpes virus in Alzheimer's disease: relation to progression of the disease
inferredcites →https://doi.org/10.1080/14034950310001432openalex-snapshot
Cardiovascular disease and diabetes in the Northern Sweden Health and Disease Study Cohort- evaluation of risk factors and their interactions
inferredcites →https://doi.org/10.1086/524144openalex-snapshot
C‐Reactive Protein Mediates the Effect of Apolipoprotein E on Cytomegalovirus Infection
inferredcites →https://doi.org/10.1093/infdis/jit210openalex-snapshot
Virological and Immunological Characteristics of Human Cytomegalovirus Infection Associated With Alzheimer Disease
inferredcites →https://doi.org/10.1093/infdis/jit665openalex-snapshot
Cytomegalovirus: An Improbable Cause of Alzheimer Disease
inferredcites →https://doi.org/10.1093/infdis/jiu437openalex-snapshot
Cytomegalovirus Infection and Risk of Alzheimer Disease in Older Black and White Individuals
inferredcites →https://doi.org/10.1097/wad.0000000000000133openalex-snapshot
Temporal Cognitive Decline Associated With Exposure to Infectious Agents in a Population-based, Aging Cohort
inferredcites →https://doi.org/10.1111/ene.12477openalex-snapshot
A study on the association between infectious burden and A lzheimer's disease
inferredcites →https://doi.org/10.1111/j.1474-9726.2007.00332.xopenalex-snapshot
Apolipoprotein E genotype influences vertical transmission of herpes simplex virus type 1 in a gender specific manner
inferredcites →https://doi.org/10.1111/j.1532-5415.2006.00796.xopenalex-snapshot
The Influence of Latent Viral Infection on Rate of Cognitive Decline over 4 Years
inferredcites →https://doi.org/10.1126/scitranslmed.aaf1059openalex-snapshot
Amyloid-β peptide protects against microbial infection in mouse and worm models of Alzheimer’s disease
inferredcites →https://doi.org/10.1161/01.str.0000086754.32238.daopenalex-snapshot
Impact of Viral and Bacterial Burden on Cognitive Impairment in Elderly Persons With Cardiovascular Diseases
inferredcites →https://doi.org/10.1176/appi.books.9780890423349openalex-snapshot
Diagnostic and Statistical Manual of Mental Disorders, Fourth Edition, Text Revision (DSM-IV-TR)
inferredcites →https://doi.org/10.1371/journal.pone.0003637openalex-snapshot
Seropositivity to Herpes Simplex Virus Antibodies and Risk of Alzheimer's Disease: A Population-Based Cohort Study
inferredcites →https://doi.org/10.1371/journal.pone.0009505openalex-snapshot
The Alzheimer's Disease-Associated Amyloid β-Protein Is an Antimicrobial Peptide
inferredcites →https://doi.org/10.1371/journal.pone.0077921openalex-snapshot
Decreased Proportion of Cytomegalovirus Specific CD8 T-Cells but No Signs of General Immunosenescence in Alzheimer’s Disease
inferredcites →https://doi.org/10.1371/journal.pone.0101364openalex-snapshot
Alzheimer's Associated β-Amyloid Protein Inhibits Influenza A Virus and Modulates Viral Interactions with Phagocytes
inferredcites →https://doi.org/10.3233/jad-140822openalex-snapshot
Herpes Viruses Increase the Risk of Alzheimer’s Disease: A Meta-Analysis
inferredcites →https://doi.org/10.3233/jad-160152openalex-snapshot
Microbes and Alzheimer’s Disease
inferredcites →https://doi.org/10.3389/fnagi.2014.00304openalex-snapshot
Detection of bacterial antigens and Alzheimer’s disease-like pathology in the central nervous system of BALB/c mice following intranasal infection with a laboratory isolate of Chlamydia pneumoniae

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Early Β-Amyloid Accumulation in the Brain Is Associated With Blood T and B Cell Alterations
inferred← citeshttps://doi.org/10.1002/alz.12008openalex-snapshot
Interaction between APOE4 and herpes simplex virus type 1 in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.12834openalex-snapshot
Herpes simplex virus alters Alzheimer's disease biomarkers ‐ A hypothesis paper
inferred← citeshttps://doi.org/10.1002/alz.13070openalex-snapshot
Association of common infections with cognitive performance in the Baltimore Epidemiologic Catchment Area study follow‐up
inferred← citeshttps://doi.org/10.1002/alz.13136openalex-snapshot
Early β‐amyloid accumulation in the brain is associated with peripheral T cell alterations
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Clinically apparent Helicobacter pylori infection and the risk of incident Alzheimer's disease: A population‐based nested case‐control study
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The association of common infectious exposures with cognitive performance in community‐dwelling older adults
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Herpesviral infections and antimicrobial protection for Alzheimer's disease: Implications for prevention and treatment
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Alzheimer's disease and herpes viruses: Current events and perspectives
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Herpesvirus infections, antiviral treatment, and the risk of dementia—a registry‐based cohort study in Sweden
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VALZ‐Pilot: High‐dose valacyclovir treatment in patients with early‐stage Alzheimer's disease
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Effects of Cytomegalovirus on Pericytes
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Association between herpesviruses and alzheimer’s disease: a meta-analysis based on case–control studies
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Role of viruses, prions and miRNA in neurodegenerative disorders and dementia
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Association of microbiota-derived propionic acid and Alzheimer’s disease; bioinformatics analysis
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Targeting Infectious Agents as a Therapeutic Strategy in Alzheimer’s Disease
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Alzheimer’s disease as a viral disease: Revisiting the infectious hypothesis
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Intermittent cytomegalovirus infection alters neurobiological metabolism and induces cognitive deficits in mice
inferred← citeshttps://doi.org/10.1016/j.ddmod.2019.10.006openalex-snapshot
Do herpesviruses play a role in Alzheimer’s disease pathogenesis?
inferred← citeshttps://doi.org/10.1016/j.jinf.2021.12.004openalex-snapshot
MAMDC2, a gene highly expressed in microglia in experimental models of Alzheimers Disease, positively regulates the innate antiviral response during neurotropic virus infection
inferred← citeshttps://doi.org/10.1016/j.jocn.2020.10.044openalex-snapshot
The association between herpes simplex virus type 1 infection and Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.neuint.2020.104852openalex-snapshot
Environmental toxins and Alzheimer's disease progression
inferred← citeshttps://doi.org/10.1016/j.neuron.2019.12.031openalex-snapshot
Human Herpesvirus 6 Detection in Alzheimer’s Disease Cases and Controls across Multiple Cohorts
inferred← citeshttps://doi.org/10.1016/j.nrl.2020.12.007openalex-snapshot
Association between psoriasis and dementia: A systematic review
inferred← citeshttps://doi.org/10.1016/j.nrleng.2023.12.005openalex-snapshot
Association between psoriasis and dementia: A systematic review
inferred← citeshttps://doi.org/10.1016/j.scitotenv.2022.157114openalex-snapshot
Increased occurrence of Treponema spp. and double-species infections in patients with Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.tins.2025.05.009openalex-snapshot
What would it take to prove that a chronic infection is a causal agent in Alzheimer’s disease?
inferred← citeshttps://doi.org/10.1016/j.trci.2019.09.014openalex-snapshot
A genetic signature including apolipoprotein Eε4 potentiates the risk of herpes simplex–associated Alzheimer's disease
inferred← citeshttps://doi.org/10.1021/acschemneuro.1c00584openalex-snapshot
Indication of Neurodegenerative Cascade Initiation by Amyloid-like Aggregate-Forming EBV Proteins and Peptide in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1021/acschemneuro.3c00293openalex-snapshot
Repurposing Antimicrobial Protegrin-1 as a Dual-Function Amyloid Inhibitor via Cross-seeding
inferred← citeshttps://doi.org/10.1038/s41598-019-45129-8openalex-snapshot
Shared Molecular Signatures Across Neurodegenerative Diseases and Herpes Virus Infections Highlights Potential Mechanisms for Maladaptive Innate Immune Responses
inferred← citeshttps://doi.org/10.1080/10408398.2023.2222404openalex-snapshot
Targeting gut-brain axis by dietary flavonoids ameliorate aging-related cognition decline: Evidences and mechanisms
inferred← citeshttps://doi.org/10.1080/1040841x.2020.1794789openalex-snapshot
Dysregulation of cofilin-1 activity—the missing link between herpes simplex virus type-1 infection and Alzheimer’s disease
inferred← citeshttps://doi.org/10.1093/aje/kwaa238openalex-snapshot
Association Between Immune Response to Cytomegalovirus and Cognition in the Health and Retirement Study
inferred← citeshttps://doi.org/10.1093/discim/kyad015openalex-snapshot
The association of microbial infection and adaptive immune cell activation in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1093/infdis/jiac484openalex-snapshot
Common Vaccines and the Risk of Incident Dementia: A Population-based Cohort Study
inferred← citeshttps://doi.org/10.1111/1346-8138.15277openalex-snapshot
Increased risk of dementia in patients with genital warts: A nationwide cohort study in Taiwan
inferred← citeshttps://doi.org/10.1155/2018/3128758openalex-snapshot
Immunosenescence of Natural Killer Cells, Inflammation, and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1159/000548365openalex-snapshot
Herpes Simplex Virus Infection and Risk of Alzheimer’s Disease: A Systematic Review and Meta-Analysis
inferred← citeshttps://doi.org/10.1177/13872877261484863openalex-snapshot
Revisiting the role of amyloid-β: A molecular and immunological perspective
inferred← citeshttps://doi.org/10.1186/s12879-024-09155-wopenalex-snapshot
Time trends in herpesvirus seroepidemiology among Swedish adults
inferred← citeshttps://doi.org/10.1186/s12883-025-04279-1openalex-snapshot
Relationship between cytomegalovirus antibody levels and cognitive performance is dependent on age and genetic risk
inferred← citeshttps://doi.org/10.1186/s12934-021-01520-7openalex-snapshot
Multi-pathogen infections and Alzheimer’s disease
inferred← citeshttps://doi.org/10.1186/s12974-019-1494-4openalex-snapshot
Antibiotics, gut microbiota, and Alzheimer’s disease
inferred← citeshttps://doi.org/10.1186/s12979-024-00443-2openalex-snapshot
Terminally differentiated effector memory T cells associate with cognitive and AD-related biomarkers in an aging-based community cohort
inferred← citeshttps://doi.org/10.1186/s12985-025-02786-8openalex-snapshot
Oxidative stress, inflammation, and apoptosis in Alzheimer’s disease associated with HSV-1 and CMV coinfection
inferred← citeshttps://doi.org/10.1186/s13024-021-00514-8openalex-snapshot
Murine roseolovirus does not accelerate amyloid-β pathology and human roseoloviruses are not over-represented in Alzheimer disease brains
inferred← citeshttps://doi.org/10.1186/s13024-022-00559-3openalex-snapshot
When the infectious environment meets the AD brain
inferred← citeshttps://doi.org/10.1371/journal.pone.0227670openalex-snapshot
Torque teno virus viral load is related to age, CMV infection and HLA type but not to Alzheimer's disease
inferred← citeshttps://doi.org/10.1371/journal.ppat.1010929openalex-snapshot
Infectious origin of Alzheimer’s disease: Amyloid beta as a component of brain antimicrobial immunity
inferred← citeshttps://doi.org/10.1371/journal.ppat.1013599openalex-snapshot
Microbial links to Alzheimer’s disease
inferred← citeshttps://doi.org/10.14283/jpad.2023.126openalex-snapshot
Cytomegalovirus Infection and Alzheimer's Disease: A Meta-Analysis
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The elusive role of herpesviruses in Alzheimer’s disease: current evidence and future directions
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Gut microbiota and pro/prebiotics in Alzheimer’s disease
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Multi-pathogen Infections and Alzheimer’s Disease
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Bibliometric and Visual Analysis of Alzheimer’s Disease and Herpes Simplex Virus Type 1 Infection Between 1990 and 2024
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On the Possible Relevance of Bottom-up Pathways in the Pathogenesis of Alzheimer’s Disease
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Altered Synaptic Plasticity: Plausible Mechanisms Associated With Viral Infections
inferred← citeshttps://doi.org/10.3233/adr-210008openalex-snapshot
Plasma Amyloid-β in Relation to Antibodies Against Herpes Simplex Virus, Cytomegalovirus, and Chlamydophila pneumoniae
inferred← citeshttps://doi.org/10.3233/jad-171162openalex-snapshot
Herpes Simplex Virus, APOE ɛ4, and Cognitive Decline in Old Age: Results from the Betula Cohort Study
inferred← citeshttps://doi.org/10.3233/jad-190765openalex-snapshot
Reevaluating the Microbial Infection Link to Alzheimer’s Disease
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Potential Role for Herpesviruses in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3233/jad-200957openalex-snapshot
Herpes Labialis, Chlamydophila pneumoniae, Helicobacter pylori, and Cytomegalovirus Infections and Risk of Dementia: The Framingham Heart Study
inferred← citeshttps://doi.org/10.3233/jad-201534openalex-snapshot
Infectious Disease Burden and the Risk of Alzheimer’s Disease: A Population-Based Study
inferred← citeshttps://doi.org/10.3233/jad-221116openalex-snapshot
Longitudinal Effects of Herpesviruses on Multiple Cognitive Outcomes in Healthy Elderly Adults
inferred← citeshttps://doi.org/10.3233/jad-230220openalex-snapshot
Anti-CMV IgG Seropositivity is Associated with Plasma Biomarker Evidence of Amyloid-β Accumulation
inferred← citeshttps://doi.org/10.3233/jad-230718openalex-snapshot
Herpes Simplex Viral Infection Doubles the Risk of Dementia in a Contemporary Cohort of Older Adults: A Prospective Study
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Amyloid Beta Revisited: A Versatile Immune Polypeptide
inferred← citeshttps://doi.org/10.32388/2esvyp.2openalex-snapshot
Amyloid Beta Revisited: A Versatile Immune Polypeptide
inferred← citeshttps://doi.org/10.3389/fimmu.2021.796867openalex-snapshot
The Impact of Systemic Inflammation on Alzheimer’s Disease Pathology
inferred← citeshttps://doi.org/10.3389/fnagi.2018.00042openalex-snapshot
Lipopolysaccharide Associates with Amyloid Plaques, Neurons and Oligodendrocytes in Alzheimer’s Disease Brain: A Review
inferred← citeshttps://doi.org/10.3389/fnagi.2018.00224openalex-snapshot
Can an Infection Hypothesis Explain the Beta Amyloid Hypothesis of Alzheimer’s Disease?
inferred← citeshttps://doi.org/10.3389/fncel.2021.695738openalex-snapshot
The Hippocampal Vulnerability to Herpes Simplex Virus Type I Infection: Relevance to Alzheimer’s Disease and Memory Impairment
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Infection and Immunometabolism in the Central Nervous System: A Possible Mechanistic Link Between Metabolic Imbalance and Dementia
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The interplay between the gut-brain axis and the microbiome: A perspective on psychiatric and neurodegenerative disorders
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A review of the roles of pathogens in Alzheimer’s disease
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Alzheimer’s disease and infectious agents: a comprehensive review of pathogenic mechanisms and microRNA roles
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Herpes simplex virus 2 and dementia risk: a systematic review and meta-analysis
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Implications of Microorganisms in Alzheimer’s Disease
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The Use of Antimicrobial and Antiviral Drugs in Alzheimer’s Disease
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Evaluation of BCG Vaccination and Plasma Amyloid: A Prospective, Pilot Study with Implications for Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3390/nu13020361openalex-snapshot
The Immunopathogenesis of Alzheimer’s Disease Is Related to the Composition of Gut Microbiota
inferred← citeshttps://doi.org/10.3390/pathogens13030240openalex-snapshot
Viral Infections, Are They a Trigger and Risk Factor of Alzheimer’s Disease?
inferred← citeshttps://doi.org/10.3390/pathogens13050417openalex-snapshot
Human Cytomegalovirus Infection and Neurocognitive and Neuropsychiatric Health
inferred← citeshttps://doi.org/10.3390/pathogens13110974openalex-snapshot
Infectious Disease as a Modifiable Risk Factor for Dementia: A Narrative Review
inferred← citeshttps://doi.org/10.3390/ph14050458openalex-snapshot
Some Candidate Drugs for Pharmacotherapy of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3390/vaccines13060572openalex-snapshot
Human Alpha Herpesviruses Infections (HSV1, HSV2, and VZV), Alzheimer’s Disease, and the Potential Benefits of Targeted Treatment or Vaccination—A Virological Perspective
inferred← citeshttps://doi.org/10.36233/0507-4088-32openalex-snapshot
Herpesvirus infections and immunological disturbances in patients with different stages of Alzheimer’s disease
inferred← citeshttps://doi.org/10.4049/jimmunol.2000351openalex-snapshot
An Ig γ Marker Genotype Is a Strong Risk Factor for Alzheimer Disease, Independent of Apolipoprotein E ε4 Genotype
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Preventive Measures against the Development of Dementia in Old Age
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Alzheimer’s disease risk after COVID-19: a view from the perspective of the infectious hypothesis of neurodegeneration
inferred← citeshttps://doi.org/10.4103/ant.ant_113_0046openalex-snapshot
Gut Microbiota and Alzheimer Disease
inferred← citeshttps://doi.org/10.52547/mlj.15.4.39openalex-snapshot
Evaluation of Role of Herpes Simplex Virus Types 1 and 2 and Cytomegalovirus in Alzheimer's Disease
inferred← citeshttps://doi.org/10.6084/m9.figshare.13663188openalex-snapshot
Additional file 1 of Multi-pathogen infections and Alzheimer’s disease
inferred← citeshttps://doi.org/10.6084/m9.figshare.18482527openalex-snapshot
Additional file 1 of Murine roseolovirus does not accelerate amyloid-β pathology and human roseoloviruses are not over-represented in Alzheimer disease brains
inferred← citeshttps://doi.org/10.6084/m9.figshare.18482530openalex-snapshot
Additional file 2 of Murine roseolovirus does not accelerate amyloid-β pathology and human roseoloviruses are not over-represented in Alzheimer disease brains
inferred← citeshttps://doi.org/10.6084/m9.figshare.18482533openalex-snapshot
Additional file 3 of Murine roseolovirus does not accelerate amyloid-β pathology and human roseoloviruses are not over-represented in Alzheimer disease brains
inferred← citeshttps://doi.org/10.64336/001c.89250openalex-snapshot
Treatment options for Alzheimer’s Disease: An overview of the amyloid, tau, and alternate hypotheses
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Associations of antibodies against several infections with Alzheimer disease neuropathology: a prospective cohort study analysis
inferred← citeshttps://doi.org/10.64898/2026.08.03.742616openalex-snapshot
Peripheral T-cell co-signalling states mark vulnerability and resilience to cerebral Aβ pathology
inferred← citeshttps://doi.org/10.69860/nobel.9786053359166.13openalex-snapshot
Association of Infections with Alzheimer’s Disease
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Herpes Simplex Virus type 1 : a potential target for the prevention of Alzheimer's disease?
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