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β-Amyloid peptides display protective activity against the human Alzheimer’s disease-associated herpes simplex virus-1

https://doi.org/10.1007/s10522-014-9538-8
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/path.1127openalex-snapshot
Herpesviruses in brain and Alzheimer's disease
inferredcites →https://doi.org/10.1002/path.2449openalex-snapshot
Herpes simplex virus type 1 DNA is located within Alzheimer's disease amyloid plaques
inferredcites →https://doi.org/10.1002/rmv.531openalex-snapshot
Understanding HSV‐1 entry glycoproteins
inferredcites →https://doi.org/10.1002/syn.20957openalex-snapshot
Alzheimer's disease: β‐amyloid plaque formation in human brain
inferredcites →https://doi.org/10.1007/0-306-46881-6_287openalex-snapshot
Fibril formation and neurotoxicity by a herpes simplex virus glycoprotein B fragment with homology to Alzheimer’ β amyloid peptide
inferredcites →https://doi.org/10.1007/s13365-013-0189-3openalex-snapshot
Immunological control of herpes simplex virus infections
inferredcites →https://doi.org/10.1016/j.bpj.2012.06.045openalex-snapshot
Two-Step Mechanism of Membrane Disruption by Aβ through Membrane Fragmentation and Pore Formation
inferredcites →https://doi.org/10.1016/j.coi.2014.05.005openalex-snapshot
Human CNS immune senescence and neurodegeneration
inferredcites →https://doi.org/10.1016/j.coviro.2011.05.024openalex-snapshot
Viral attachment strategies: the many faces of adenoviruses
inferredcites →https://doi.org/10.1016/j.coviro.2011.12.008openalex-snapshot
Structure of human adenovirus
inferredcites →https://doi.org/10.1016/j.coviro.2013.01.005openalex-snapshot
Live cell imaging of viral entry
inferredcites →https://doi.org/10.1016/j.disamonth.2010.06.001openalex-snapshot
Alzheimer Disease
inferredcites →https://doi.org/10.1016/j.it.2013.10.012openalex-snapshot
Innate antiviral signalling in the central nervous system
inferredcites →https://doi.org/10.1016/j.jcv.2013.05.010openalex-snapshot
A case of late herpes simplex encephalitis relapse
inferredcites →https://doi.org/10.1016/j.neulet.2007.09.077openalex-snapshot
Herpes simplex virus infection causes cellular β-amyloid accumulation and secretase upregulation
inferredcites →https://doi.org/10.1016/j.neuron.2013.04.014openalex-snapshot
Alzheimer’s Disease Risk Gene CD33 Inhibits Microglial Uptake of Amyloid Beta
inferredcites →https://doi.org/10.1017/s0317167100013408openalex-snapshot
Amyloid and Alzheimer's Disease: Inside and Out
inferredcites →https://doi.org/10.1017/s1462399411002006openalex-snapshot
Emerging roles of pathogens in Alzheimer disease
inferredcites →https://doi.org/10.1021/bi000029fopenalex-snapshot
Fibril Formation and Neurotoxicity by a Herpes Simplex Virus Glycoprotein B Fragment with Homology to the Alzheimer's Aβ Peptide
inferredcites →https://doi.org/10.1021/bi00463a003openalex-snapshot
Molecular determinants of amyloid deposition in Alzheimer's disease: conformational studies of synthetic .beta.-protein fragments
inferredcites →https://doi.org/10.1021/ct300916fopenalex-snapshot
Mechanisms for the Insertion of Toxic, Fibril-like β-Amyloid Oligomers into the Membrane
inferredcites →https://doi.org/10.1038/nn.3028openalex-snapshot
The toxic Aβ oligomer and Alzheimer's disease: an emperor in need of clothes
inferredcites →https://doi.org/10.1038/nrd3505openalex-snapshot
The amyloid cascade hypothesis for Alzheimer's disease: an appraisal for the development of therapeutics
inferredcites →https://doi.org/10.1038/tp.2013.11openalex-snapshot
Soluble amyloid precursor protein β as blood-based biomarker of Alzheimer’s disease
inferredcites →https://doi.org/10.1039/b912533gopenalex-snapshot
The chemistry and biology of LL-37
inferredcites →https://doi.org/10.1056/nejmra0909142openalex-snapshot
Alzheimer's Disease
inferredcites →https://doi.org/10.1073/pnas.0914251107openalex-snapshot
Truncated β-amyloid peptide channels provide an alternative mechanism for Alzheimer’s Disease and Down syndrome
inferredcites →https://doi.org/10.1093/gerona/glu057openalex-snapshot
Chronic Inflammation (Inflammaging) and Its Potential Contribution to Age-Associated Diseases
inferredcites →https://doi.org/10.1099/vir.0.003087-0openalex-snapshot
Adenoviruses: update on structure and function
inferredcites →https://doi.org/10.1101/cshperspect.a006262openalex-snapshot
Biochemistry of Amyloid -Protein and Amyloid Deposits in Alzheimer Disease
inferredcites →https://doi.org/10.1111/j.1574-6976.2011.00320.xopenalex-snapshot
The molecular basis of herpes simplex virus latency
inferredcites →https://doi.org/10.1111/j.1742-4658.2009.07402.xopenalex-snapshot
Viral entry mechanisms: cellular and viral mediators of herpes simplex virus entry
inferredcites →https://doi.org/10.1113/jphysiol.2006.111203openalex-snapshot
Physiological roles for amyloid β peptides
inferredcites →https://doi.org/10.1126/science.1090284openalex-snapshot
Three-Dimensional Structure of Herpes Simplex Virus from Cryo-Electron Tomography
inferredcites →https://doi.org/10.1126/science.1126548openalex-snapshot
Crystal Structure of Glycoprotein B from Herpes Simplex Virus 1
inferredcites →https://doi.org/10.1126/science.2474201openalex-snapshot
Neurotoxicity of a Fragment of the Amyloid Precursor Associated with Alzheimer's Disease
inferredcites →https://doi.org/10.1128/aac.42.9.2206openalex-snapshot
Activities of LL-37, a Cathelin-Associated Antimicrobial Peptide of Human Neutrophils
inferredcites →https://doi.org/10.1177/0891988710383571openalex-snapshot
Review Article: Genetics of Alzheimer Disease
inferredcites →https://doi.org/10.1189/jlb.0704380openalex-snapshot
Impact of LL-37 on anti-infective immunity
inferredcites →https://doi.org/10.1371/journal.pone.0003637openalex-snapshot
Seropositivity to Herpes Simplex Virus Antibodies and Risk of Alzheimer's Disease: A Population-Based Cohort Study
inferredcites →https://doi.org/10.1371/journal.pone.0009505openalex-snapshot
The Alzheimer's Disease-Associated Amyloid β-Protein Is an Antimicrobial Peptide
inferredcites →https://doi.org/10.1371/journal.pone.0013989openalex-snapshot
APP Processing Induced by Herpes Simplex Virus Type 1 (HSV-1) Yields Several APP Fragments in Human and Rat Neuronal Cells
inferredcites →https://doi.org/10.1371/journal.pone.0025152openalex-snapshot
Antivirals Reduce the Formation of Key Alzheimer's Disease Molecules in Cell Cultures Acutely Infected with Herpes Simplex Virus Type 1
inferredcites →https://doi.org/10.1371/journal.pone.0101364openalex-snapshot
Alzheimer's Associated β-Amyloid Protein Inhibits Influenza A Virus and Modulates Viral Interactions with Phagocytes
inferredcites →https://doi.org/10.1373/clinchem.2008.112797openalex-snapshot
The MIQE Guidelines: Minimum Information for Publication of Quantitative Real-Time PCR Experiments
inferredcites →https://doi.org/10.2174/138920112802273155openalex-snapshot
Role of Defensins and Cathelicidin LL37 in Auto-Immune and Auto- Inflammatory Diseases
inferredcites →https://doi.org/10.2217/fmb.12.10openalex-snapshot
Could Antivirals be used to Treat Alzheimer‘s Disease?
inferredcites →https://doi.org/10.3233/jad-2009-1015openalex-snapshot
Dramatic Shifts in Circulating CD4 but not CD8 T Cell Subsets in Mild Alzheimer's Disease
inferredcites →https://doi.org/10.3389/fphar.2014.00097openalex-snapshot
HSV-1 and Alzheimer’s disease: more than a hypothesis
inferredcites →https://doi.org/10.3390/ijms13067303openalex-snapshot
The Toxicity of Amyloid ß Oligomers
inferredcites →https://doi.org/10.3390/v4050800openalex-snapshot
Herpes Virus Fusion and Entry: A Story with Many Characters

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Alzheimerrs Disease-Associated -amyloid Is Rapidly Seeded by herpesviridae to Protect Against Brain Infection
inferred← citeshttps://doi.org/10.2139/ssrn.3158775openalex-extracted
Fermented Coconut As Cost-Effective, Safe, and (More) Elegant Remedy for Anti-Microbial Resistance Epidemics
inferred← citeshttps://doi.org/10.1002/adfm.202102978openalex-snapshot
Design and Engineering of Amyloid Aggregation‐Prone Fragments and Their Antimicrobial Conjugates with Multi‐Target Functionality
inferred← citeshttps://doi.org/10.1002/advs.202503997openalex-snapshot
Structural and Functional Versatility of the Amyloidogenic Non‐Amidated Variant of the Antimicrobial Peptide Citropin 1.3
inferred← citeshttps://doi.org/10.1002/alz.12834openalex-snapshot
Herpes simplex virus alters Alzheimer's disease biomarkers ‐ A hypothesis paper
inferred← citeshttps://doi.org/10.1002/ijch.201600097openalex-snapshot
Flexible N‐Termini of Amyloid β‐Protein Oligomers: A Link between Structure and Activity?
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Herpesviral infections and antimicrobial protection for Alzheimer's disease: Implications for prevention and treatment
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Infectious Agents as Potential Drivers of α‐Synucleinopathies
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What can AlphaFold do for antimicrobial amyloids?
inferred← citeshttps://doi.org/10.1002/psc.3534openalex-snapshot
Antimicrobial peptides for novel antiviral strategies in the current post‐COVID‐19 pandemic
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Neurodegeneration Associated with HIV-1 in the Era of cART
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Immunosenescence and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-031-15080-7_137openalex-snapshot
Neurodegeneration Associated with HIV-1 in the Era of cART
inferred← citeshttps://doi.org/10.1007/978-3-031-68513-2_12openalex-snapshot
Infection, Neuroinflammation and Interventions for Healthy Brain and Longevity
inferred← citeshttps://doi.org/10.1007/978-3-032-05273-5_16openalex-snapshot
The Many Lives of a Single Sequence: Functional Plasticity Through Amyloid Polymorphism
inferred← citeshttps://doi.org/10.1007/978-981-16-6760-2_1openalex-snapshot
Gut–Brain Axis: Role of Gut Microbiota in Neurodegenerative Disease
inferred← citeshttps://doi.org/10.1007/s00018-019-03298-9openalex-snapshot
What is strain in neurodegenerative diseases?
inferred← citeshttps://doi.org/10.1007/s00018-020-03711-8openalex-snapshot
Mechanisms and therapeutic potential of interactions between human amyloids and viruses
inferred← citeshttps://doi.org/10.1007/s00406-020-01157-4openalex-snapshot
Association of herpes zoster with dementia and effect of antiviral therapy on dementia: a population-based cohort study
inferred← citeshttps://doi.org/10.1007/s10495-025-02142-9openalex-snapshot
HSV-1 hijacks mitochondrial dynamics: potential molecular mechanisms linking viral infection to neurodegenerative disorders
inferred← citeshttps://doi.org/10.1007/s10522-015-9615-7openalex-snapshot
From inflamm-aging to immune-paralysis: a slippery slope during aging for immune-adaptation
inferred← citeshttps://doi.org/10.1007/s12031-019-01274-3openalex-snapshot
Insights into the Potential Role of Mercury in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12250-018-0049-7openalex-snapshot
Common Infections May Lead to Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s12539-021-00462-3openalex-snapshot
HD5 and LL-37 Inhibit SARS-CoV and SARS-CoV-2 Binding to Human ACE2 by Molecular Simulation
inferred← citeshttps://doi.org/10.1007/s13365-019-00732-3openalex-snapshot
Does HIV infection contribute to increased beta-amyloid synthesis and plaque formation leading to neurodegeneration and Alzheimer’s disease?
inferred← citeshttps://doi.org/10.1007/s40263-020-00737-1openalex-snapshot
Targeting Infectious Agents as a Therapeutic Strategy in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s40484-020-0222-5openalex-snapshot
Prediction and analysis of human‐herpes simplex virus type 1 protein‐protein interactions by integrating multiple methods
inferred← citeshttps://doi.org/10.1007/s40588-021-00168-6openalex-snapshot
The Role of Chronic Infection in Alzheimer’s Disease: Instigators, Co-conspirators, or Bystanders?
inferred← citeshttps://doi.org/10.1016/b978-0-12-822569-1.00014-7openalex-snapshot
The role of inflammaging in the development of chronic diseases of older people
inferred← citeshttps://doi.org/10.1016/b978-0-12-823967-4.00019-1openalex-snapshot
Droplets of life: role of phase separation in virus replication and compartmentalization
inferred← citeshttps://doi.org/10.1016/b978-0-323-46152-8.00006-8openalex-snapshot
Antimicrobial Nanostructures for Neurodegenerative Infections
inferred← citeshttps://doi.org/10.1016/bs.apcsb.2025.10.016openalex-snapshot
Antimicrobial peptides act as a component of brain immunity against microbes in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/bs.pmbts.2019.05.007openalex-snapshot
Intrinsically disordered proteins in various hypotheses on the pathogenesis of Alzheimer's and Parkinson's diseases
inferred← citeshttps://doi.org/10.1016/j.arr.2018.07.008openalex-snapshot
‘Evolutionary medicine’ perspectives on Alzheimer’s Disease: Review and new directions
inferred← citeshttps://doi.org/10.1016/j.bbi.2023.01.010openalex-snapshot
Blood-brain barrier penetration of non-replicating SARS-CoV-2 and S1 variants of concern induce neuroinflammation which is accentuated in a mouse model of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2024.123017openalex-snapshot
Bacterial clustering biomaterials as anti-infective therapies
inferred← citeshttps://doi.org/10.1016/j.biopha.2021.111575openalex-snapshot
Amyloid-β: A double agent in Alzheimer's disease?
inferred← citeshttps://doi.org/10.1016/j.bpc.2021.106735openalex-snapshot
A mechanistic survey of Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.brainresbull.2016.11.008openalex-snapshot
Platelets are responsible for the accumulation of β-amyloid in blood clots inside and around blood vessels in mouse brain after thrombosis
inferred← citeshttps://doi.org/10.1016/j.brainresbull.2025.111506openalex-snapshot
Neuroprotection effect of HSV-1 LAT-derived miR-H2 and miR-H3 associated with Tau and alpha-synuclein downregulation
inferred← citeshttps://doi.org/10.1016/j.cbpa.2023.102318openalex-snapshot
Designed inhibitors to reduce amyloid virulence and cytotoxicity and combat neurodegenerative and infectious diseases
inferred← citeshttps://doi.org/10.1016/j.cell.2019.09.001openalex-snapshot
Alzheimer Disease: An Update on Pathobiology and Treatment Strategies
inferred← citeshttps://doi.org/10.1016/j.ddmod.2019.10.006openalex-snapshot
Do herpesviruses play a role in Alzheimer’s disease pathogenesis?
inferred← citeshttps://doi.org/10.1016/j.exger.2017.12.019openalex-snapshot
Role of the peripheral innate immune system in the development of Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.jalz.2018.06.3040openalex-snapshot
The antimicrobial protection hypothesis of Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.jmgm.2022.108143openalex-snapshot
Molecular docking and molecular dynamics simulations studies on the protective and pathogenic roles of the amyloid-β peptide between herpesvirus infection and Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.neubiorev.2022.104660openalex-snapshot
Strain-specific pre-existing immunity: A key to understanding the role of chronic Toxoplasma infection in cognition and Alzheimer's diseases?
inferred← citeshttps://doi.org/10.1016/j.neuint.2019.03.007openalex-snapshot
Autism genes and the leukocyte transcriptome in autistic toddlers relate to pathogen interactomes, infection and the immune system. A role for excess neurotrophic sAPPα and reduced antimicrobial Aβ
inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2015.06.014openalex-snapshot
Herpes simplex virus type 2 infection induces AD-like neurodegeneration markers in human neuroblastoma cells
inferred← citeshttps://doi.org/10.1016/j.neuron.2018.06.030openalex-snapshot
Alzheimer’s Disease-Associated β-Amyloid Is Rapidly Seeded by Herpesviridae to Protect against Brain Infection
inferred← citeshttps://doi.org/10.1016/j.neuron.2019.12.031openalex-snapshot
Human Herpesvirus 6 Detection in Alzheimer’s Disease Cases and Controls across Multiple Cohorts
inferred← citeshttps://doi.org/10.1016/j.neuron.2020.06.005openalex-snapshot
Tipping the Scales: Peptide-Dependent Dysregulation of Neural Circuit Dynamics in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1016/j.scitotenv.2022.157114openalex-snapshot
Increased occurrence of Treponema spp. and double-species infections in patients with Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.tim.2020.03.003openalex-snapshot
Herpes Simplex Virus-1 in the Brain: The Dark Side of a Sneaky Infection
inferred← citeshttps://doi.org/10.1016/j.virol.2025.110523openalex-snapshot
Infections, genetics, and Alzheimer's disease: Exploring the pathogenic factors for innovative therapies
inferred← citeshttps://doi.org/10.1021/acs.biomac.2c00582openalex-snapshot
Natural Antimicrobial Peptides Self-assemble as α/β Chameleon Amyloids
inferred← citeshttps://doi.org/10.1021/acs.jpcb.0c07716openalex-snapshot
Cross-Linked Amyloid β-Protein Oligomers: A Missing Link in Alzheimer’s Disease Pathology?
inferred← citeshttps://doi.org/10.1021/acschemneuro.0c00719openalex-snapshot
Viral Involvement in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1021/acschemneuro.1c00584openalex-snapshot
Indication of Neurodegenerative Cascade Initiation by Amyloid-like Aggregate-Forming EBV Proteins and Peptide in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1021/acschemneuro.2c00679openalex-snapshot
Neuroprotective Effects of Lactoferrin in Alzheimer’s and Parkinson’s Diseases: A Narrative Review
inferred← citeshttps://doi.org/10.1021/acschemneuro.3c00293openalex-snapshot
Repurposing Antimicrobial Protegrin-1 as a Dual-Function Amyloid Inhibitor via Cross-seeding
inferred← citeshttps://doi.org/10.1021/acschemneuro.5c00444openalex-snapshot
Modulation of Amyloid-β Aggregation by Surface Proteins from Pathogens Associated with Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1038/s41380-021-01138-6openalex-snapshot
The viral hypothesis: how herpesviruses may contribute to Alzheimer’s disease
inferred← citeshttps://doi.org/10.1038/s41380-022-01790-6openalex-snapshot
Human antimicrobial peptide LL-37 contributes to Alzheimer’s disease progression
inferred← citeshttps://doi.org/10.1038/s41392-023-01484-7openalex-snapshot
Amyloid β-based therapy for Alzheimer’s disease: challenges, successes and future
inferred← citeshttps://doi.org/10.1038/s41467-017-01447-xopenalex-snapshot
Self-assembling dipeptide antibacterial nanostructures with membrane disrupting activity
inferred← citeshttps://doi.org/10.1038/s41467-022-32039-zopenalex-snapshot
The Cryo-EM structures of two amphibian antimicrobial cross-β amyloid fibrils
inferred← citeshttps://doi.org/10.1038/s41467-023-40000-xopenalex-snapshot
HIV-1 promotes ubiquitination of the amyloidogenic C-terminal fragment of APP to support viral replication
inferred← citeshttps://doi.org/10.1038/s41582-020-0323-9openalex-snapshot
Do infections have a role in the pathogenesis of Alzheimer disease?
inferred← citeshttps://doi.org/10.1038/s41593-025-02157-0openalex-snapshot
Phosphorylated tau exhibits antimicrobial activity capable of neutralizing herpes simplex virus 1 infectivity in human neurons
inferred← citeshttps://doi.org/10.1038/s42003-020-0865-9openalex-snapshot
Why does the Aβ peptide of Alzheimer share structural similarity with antimicrobial peptides?
inferred← citeshttps://doi.org/10.1039/d1sc01133bopenalex-snapshot
Antimicrobial α-defensins as multi-target inhibitors against amyloid formation and microbial infection
inferred← citeshttps://doi.org/10.1039/d3cs00878aopenalex-snapshot
Exploring pathological link between antimicrobial and amyloid peptides
inferred← citeshttps://doi.org/10.1051/parasite/2025019openalex-snapshot
No evidence of Alzheimer’s disease pathology in mice infected with Toxocara canis
inferred← citeshttps://doi.org/10.1063/5.0093889openalex-snapshot
Virus-induced Alzheimer’s disease: Potential roles of viral infections in AD neuropathogenesis from two aspects: Aberrant protein accumulations with neuroinflammatory response and virus-induced ablation of adult neurogenesis
inferred← citeshttps://doi.org/10.1073/pnas.2014442118openalex-snapshot
The amphibian antimicrobial peptide uperin 3.5 is a cross-α/cross-β chameleon functional amyloid
inferred← citeshttps://doi.org/10.1080/01480545.2019.1681444openalex-snapshot
Long-term consumption of alcohol exacerbates neural lesions by destroying the functional integrity of the blood–brain barrier
inferred← citeshttps://doi.org/10.1080/1040841x.2019.1660615openalex-snapshot
Roles of HSV-1 infection-induced microglial immune responses in CNS diseases: friends or foes?
inferred← citeshttps://doi.org/10.1080/1040841x.2020.1794789openalex-snapshot
Dysregulation of cofilin-1 activity—the missing link between herpes simplex virus type-1 infection and Alzheimer’s disease
inferred← citeshttps://doi.org/10.1080/1040841x.2021.1876630openalex-snapshot
The role of microbial infection in the pathogenesis of Alzheimer’s disease and the opportunity for protection by anti-microbial peptides
inferred← citeshttps://doi.org/10.1080/14737175.2023.2244162openalex-snapshot
Viruses - a major cause of amyloid deposition in the brain
inferred← citeshttps://doi.org/10.1080/14787210.2017.1264271openalex-snapshot
A protective role for herpes simplex virus type-1-specific humoral immunity in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1093/brain/awz244openalex-snapshot
Time to test antibacterial therapy in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1093/discim/kyac003openalex-snapshot
Expression of antimicrobial host defence peptides in the central nervous system during health and disease
inferred← citeshttps://doi.org/10.1096/fj.201700360openalex-snapshot
Herpes simplex virus type 1 and Alzheimer's disease: possible mechanisms and signposts
inferred← citeshttps://doi.org/10.1098/rsob.170228openalex-snapshot
The role of astrocytes in amyloid production and Alzheimer's disease
inferred← citeshttps://doi.org/10.1101/2022.01.08.475498openalex-snapshot
The Cryo-EM Structures of two Amphibian Antimicrobial Cross-β Amyloid Fibrils
inferred← citeshttps://doi.org/10.1101/2025.01.31.635854openalex-snapshot
Structural and Functional Versatility of the Amyloidogenic Antimicrobial Peptide Citropin 1.3
inferred← citeshttps://doi.org/10.1101/2025.06.04.655278openalex-snapshot
High reelin expression can explain why the entorhinal cortex is a cradle for Alzheimer’s disease
inferred← citeshttps://doi.org/10.1101/2025.06.11.659008openalex-snapshot
Viral Infection Induces Alzheimer’s Disease-Related Pathways and Senescence in iPSC-Derived Neuronal Models
inferred← citeshttps://doi.org/10.1101/2025.09.24.25336349openalex-snapshot
Lactoferrin levels in cerebrospinal fluid exhibits isoform-specific associations with Alzheimer’s disease
inferred← citeshttps://doi.org/10.1111/1751-7915.14462openalex-snapshot
Do microbes play a role in Alzheimer's disease?
inferred← citeshttps://doi.org/10.1111/cei.13493openalex-snapshot
Accumulation of amyloid beta in human glioblastomas
inferred← citeshttps://doi.org/10.1111/jnc.15538openalex-snapshot
Biological and methodological complexities of beta‐amyloid peptide: Implications for Alzheimer’s disease research
inferred← citeshttps://doi.org/10.1111/joim.70060openalex-snapshot
The dysregulation of innate immunity by Porphyromonas gingivalis in the etiology of Alzheimer's disease
inferred← citeshttps://doi.org/10.1126/scitranslmed.aaf1059openalex-snapshot
Amyloid-β peptide protects against microbial infection in mouse and worm models of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1128/cmr.00143-20openalex-snapshot
Human Herpesviruses 6A and 6B in Brain Diseases: Association versus Causation
inferred← citeshttps://doi.org/10.1128/iai.00085-26openalex-snapshot
Pathological or preventative? Amyloid-β as an effector of innate immunity
inferred← citeshttps://doi.org/10.1128/jvi.01874-19openalex-snapshot
Amyloid-β and p-Tau Anti-Threat Response to Herpes Simplex Virus 1 Infection in Primary Adult Murine Hippocampal Neurons
inferred← citeshttps://doi.org/10.1146/annurev-biochem-032620-105157openalex-snapshot
Structural Mimicry in Microbial and Antimicrobial Amyloids
inferred← citeshttps://doi.org/10.1155/2017/3948360openalex-snapshot
AβPeptide Originated from Platelets Promises New Strategy in Anti-Alzheimer’s Drug Development
inferred← citeshttps://doi.org/10.1155/2018/1672568openalex-snapshot
Amyloid-Beta 1-42 Cross-Reactive Antibody Prevalent in Human Sera May Contribute to Intraneuronal Deposition of A-Beta-P-42
inferred← citeshttps://doi.org/10.1155/2019/2302835openalex-snapshot
Nitric Oxide Influences HSV-1-Induced Neuroinflammation
inferred← citeshttps://doi.org/10.1155/2019/6927380openalex-snapshot
Syk and Hrs Regulate TLR3-Mediated Antiviral Response in Murine Astrocytes
inferred← citeshttps://doi.org/10.1177/13872877251317228openalex-snapshot
Increased risk of dementia associated with herpes simplex virus infections: Evidence from a retrospective cohort study using U.S. electronic health records
inferred← citeshttps://doi.org/10.1177/13872877251346044openalex-snapshot
What is frustrating the search for treatment of dementia? Fraud or ideas?
inferred← citeshttps://doi.org/10.1177/13872877251415571openalex-snapshot
Unmasking the hidden catalyst: How infections trigger Alzheimer's disease
inferred← citeshttps://doi.org/10.1186/s12883-025-04500-1openalex-snapshot
Human herpesvirus 6 (HHV-6) infection and risk of Alzheimer’s disease: a systematic review and meta-analysis
inferred← citeshttps://doi.org/10.1186/s12934-021-01520-7openalex-snapshot
Multi-pathogen infections and Alzheimer’s disease
inferred← citeshttps://doi.org/10.1186/s12974-019-1663-5openalex-snapshot
Loss of amyloid precursor protein exacerbates early inflammation in Niemann-Pick disease type C
inferred← citeshttps://doi.org/10.1186/s12974-024-03139-yopenalex-snapshot
Contribution of CNS and extra-CNS infections to neurodegeneration: a narrative review
inferred← citeshttps://doi.org/10.1186/s13062-024-00546-2openalex-snapshot
Multiplex antimicrobial activities of the self-assembled amphiphilic polypeptide β nanofiber KF-5 against vaginal pathogens
inferred← citeshttps://doi.org/10.1186/s13195-019-0552-6openalex-snapshot
HHV-6A infection induces amyloid-beta expression and activation of microglial cells
inferred← citeshttps://doi.org/10.1186/s13195-026-02204-3openalex-snapshot
Herpes simplex virus and kallikrein-related peptidases – a functional connection in Alzheimer‘s disease
inferred← citeshttps://doi.org/10.1186/s40035-018-0139-3openalex-snapshot
Amyloid, tau, pathogen infection and antimicrobial protection in Alzheimer’s disease –conformist, nonconformist, and realistic prospects for AD pathogenesis
inferred← citeshttps://doi.org/10.1212/nxi.0000000000000739openalex-snapshot
Brain amyloid in virally suppressed HIV-associated neurocognitive disorder
inferred← citeshttps://doi.org/10.1242/dmm.026674openalex-snapshot
HSV presence in brains of individuals without dementia: the TASTY brain series
inferred← citeshttps://doi.org/10.1371/journal.pcbi.1012709openalex-snapshot
Intraneuronal binding of amyloid beta with reelin—Implications for the onset of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1371/journal.pcbi.1014532openalex-snapshot
High reelin expression may explain why a subgroup of entorhinal cortex neurons functions as an initial nucleation site of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1371/journal.pone.0194001openalex-snapshot
Critical role of C-terminal residues of the Alzheimer's associated β-amyloid protein in mediating antiviral activity and modulating viral and bacterial interactions with neutrophils
inferred← citeshttps://doi.org/10.1371/journal.ppat.1010929openalex-snapshot
Infectious origin of Alzheimer’s disease: Amyloid beta as a component of brain antimicrobial immunity
inferred← citeshttps://doi.org/10.14283/jpad.2022.68openalex-snapshot
The role of Aβ in Alzheimer's Disease as an Evolutionary Outcome of Optimized Innate Immune Defense
inferred← citeshttps://doi.org/10.14336/ad.2018.1129openalex-snapshot
Characterization of Alzheimer’s Disease Using Ultra-high b-values Apparent Diffusion Coefficient and Diffusion Kurtosis Imaging
inferred← citeshttps://doi.org/10.14670/hh-18-111openalex-snapshot
Platelet-generated amyloid beta peptides in Alzheimer's disease and glaucoma.
inferred← citeshttps://doi.org/10.1515/revneuro-2023-0100openalex-snapshot
Diversity of amyloid beta peptide actions
inferred← citeshttps://doi.org/10.1515/revneuro-2025-0034openalex-snapshot
Antimicrobial peptides and proteins in Alzheimer’s and Parkinson’s diseases: implications for biomarker exploration
inferred← citeshttps://doi.org/10.17816/rmmar83615openalex-snapshot
The role of infectious agents in the development of neurodegenerative diseases
inferred← citeshttps://doi.org/10.18502/jovr.v19i4.15011openalex-snapshot
Serum Oxidative Stress-related Biomarkers in Ocular Hypertension and Glaucoma
inferred← citeshttps://doi.org/10.18632/oncoscience.309openalex-snapshot
Persistent infections, immune-senescence and Alzheimer's disease
inferred← citeshttps://doi.org/10.21203/rs.3.rs-8249669/v1openalex-snapshot
Expanding the amyloid landscape: structural plasticity of antimicrobial peptides
inferred← citeshttps://doi.org/10.21203/rs.3.rs-9678397/v1openalex-snapshot
Lactoferrin Levels in Cerebrospinal Fluid Exhibit Differential Associations with Alzheimer’s Disease
inferred← citeshttps://doi.org/10.2147/jmdh.s514397openalex-snapshot
Bibliometric and Visual Analysis of Alzheimer’s Disease and Herpes Simplex Virus Type 1 Infection Between 1990 and 2024
inferred← citeshttps://doi.org/10.2147/ndt.s264910openalex-snapshot
Targeting Impaired Antimicrobial Immunity in the Brain for the Treatment of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.2174/0115672050295561240327055835openalex-snapshot
Evaluation and Characterization of Modified K114 Method to Localize Plaques in Rodent and Plaques and Tangles in Human Brain Tissue
inferred← citeshttps://doi.org/10.2174/0929867326666190805151654openalex-snapshot
Antiviral Activities of Human Host Defense Peptides
inferred← citeshttps://doi.org/10.2174/1871527315666161122144659openalex-snapshot
How Immune-inflammatory Processes Link CNS and Psychiatric Disorders: Classification and Treatment Implications
inferred← citeshttps://doi.org/10.2174/9781681081472115040003openalex-snapshot
Melatoninergic Pathways in Alzheimer's Disease
inferred← citeshttps://doi.org/10.23937/2378-3001/3/1/1036openalex-snapshot
Inhalable Curcumin is an Efficacious Treatment Strategy for Herpes Simplex Virus Type 1-Induced Neuropathology
inferred← citeshttps://doi.org/10.30629/2658-7947-2022-27-3-64-73openalex-snapshot
Infectious hypothesis of neurodegenerative diseases. What waits us after the COVID-19 pandemic?
inferred← citeshttps://doi.org/10.3233/adr-170017openalex-snapshot
Genetic, Transcriptome, Proteomic, and Epidemiological Evidence for Blood-Brain Barrier Disruption and Polymicrobial Brain Invasion as Determinant Factors in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3233/adr-170037openalex-snapshot
Prion Protein PRNP: A New Player in Innate Immunity? The Aβ Connection
inferred← citeshttps://doi.org/10.3233/adr-220061openalex-snapshot
Interaction Mechanism Between the HSV-1 Glycoprotein B and the Antimicrobial Peptide Amyloid-β
inferred← citeshttps://doi.org/10.3233/jad-142718openalex-snapshot
Variants in Antiviral Genes are Risk Factors for Cognitive Decline and Dementia
inferred← citeshttps://doi.org/10.3233/jad-150652openalex-snapshot
Protective Effect of Amyloid-β Peptides Against Herpes Simplex Virus-1 Infection in a Neuronal Cell Culture Model
inferred← citeshttps://doi.org/10.3233/jad-160152openalex-snapshot
Microbes and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3233/jad-160517openalex-snapshot
Anti-Viral Properties of Amyloid-β Peptides
inferred← citeshttps://doi.org/10.3233/jad-160607openalex-snapshot
Herpes and Alzheimer’s Disease: Subversion in the Central Nervous System and How It Might Be Halted
inferred← citeshttps://doi.org/10.3233/jad-161305openalex-snapshot
Interaction between Cytomegalovirus and Herpes Simplex Virus Type 1 Associated with the Risk of Alzheimer’s Disease Development
inferred← citeshttps://doi.org/10.3233/jad-170223openalex-snapshot
Evidence that the Human Innate Immune Peptide LL-37 may be a Binding Partner of Amyloid-β and Inhibitor of Fibril Assembly
inferred← citeshttps://doi.org/10.3233/jad-170961openalex-snapshot
Reaction of Amyloid-β Peptide Antibody with Different Infectious Agents Involved in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3233/jad-171133openalex-snapshot
Alzheimer’s Amyloid-β is an Antimicrobial Peptide: A Review of the Evidence
inferred← citeshttps://doi.org/10.3233/jad-171162openalex-snapshot
Herpes Simplex Virus, APOE ɛ4, and Cognitive Decline in Old Age: Results from the Betula Cohort Study
inferred← citeshttps://doi.org/10.3233/jad-180319openalex-snapshot
Association between Alzheimer’s Disease and Oral and Gut Microbiota: Are Pore Forming Proteins the Missing Link?
inferred← citeshttps://doi.org/10.3233/jad-200607openalex-snapshot
Independent and Correlated Role of Apolipoprotein E ɛ 4 Genotype and Herpes Simplex Virus Type 1 in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3233/jad-210234openalex-snapshot
Insulin Resistance as a Common Link Between Current Alzheimer’s Disease Hypotheses
inferred← citeshttps://doi.org/10.3233/jad-221279openalex-snapshot
The Adult Neurogenesis Theory of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3233/jad-231202openalex-snapshot
The Catastrophe of Intracerebral Hemorrhage Drives the Capillary-Hemorrhage Dementias, Including Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3389/fcimb.2022.1026457openalex-snapshot
Association between Porphyromonas Gingivalis and systemic diseases: Focus on T cells-mediated adaptive immunity
inferred← citeshttps://doi.org/10.3389/fgene.2018.00362openalex-snapshot
Role of Microbes in the Development of Alzheimer’s Disease: State of the Art – An International Symposium Presented at the 2017 IAGG Congress in San Francisco
inferred← citeshttps://doi.org/10.3389/fimmu.2017.00783openalex-snapshot
Differential Phenotypes of Myeloid-Derived Suppressor and T Regulatory Cells and Cytokine Levels in Amnestic Mild Cognitive Impairment Subjects Compared to Mild Alzheimer Diseased Patients
inferred← citeshttps://doi.org/10.3389/fimmu.2019.02980openalex-snapshot
Amyloids in Site-Specific Autoimmune Reactions and Inflammatory Responses
inferred← citeshttps://doi.org/10.3389/fimmu.2020.571083openalex-snapshot
On the Role of Platelet-Generated Amyloid Beta Peptides in Certain Amyloidosis Health Complications
inferred← citeshttps://doi.org/10.3389/fimmu.2022.831815openalex-snapshot
Activation of NLRP3 Inflammasome by Virus-Like Particles of Human Polyomaviruses in Macrophages
inferred← citeshttps://doi.org/10.3389/fnagi.2017.00408openalex-snapshot
The Porphyromonas gingivalis/Host Interactome Shows Enrichment in GWASdb Genes Related to Alzheimer's Disease, Diabetes and Cardiovascular Diseases
inferred← citeshttps://doi.org/10.3389/fnagi.2018.00048openalex-snapshot
Molecular Mechanisms for Herpes Simplex Virus Type 1 Pathogenesis in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3389/fnagi.2018.00118openalex-snapshot
The Physiological Roles of Amyloid-β Peptide Hint at New Ways to Treat Alzheimer's Disease
inferred← citeshttps://doi.org/10.3389/fnagi.2018.00224openalex-snapshot
Can an Infection Hypothesis Explain the Beta Amyloid Hypothesis of Alzheimer’s Disease?
inferred← citeshttps://doi.org/10.3389/fnagi.2018.00324openalex-snapshot
Corroboration of a Major Role for Herpes Simplex Virus Type 1 in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3389/fnagi.2019.00070openalex-snapshot
Low Evolutionary Selection Pressure in Senescence Does Not Explain the Persistence of Aβ in the Vertebrate Genome
inferred← citeshttps://doi.org/10.3389/fnagi.2019.00143openalex-snapshot
The Post-amyloid Era in Alzheimer's Disease: Trust Your Gut Feeling
inferred← citeshttps://doi.org/10.3389/fnagi.2022.1026260openalex-snapshot
High resolution 16S rRNA gene Next Generation Sequencing study of brain areas associated with Alzheimer’s and Parkinson’s disease
inferred← citeshttps://doi.org/10.3389/fnagi.2022.858429openalex-snapshot
Peripheral Pathways to Neurovascular Unit Dysfunction, Cognitive Impairment, and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3389/fncel.2016.00269openalex-snapshot
Biotic/Abiotic Stress-Driven Alzheimer's Disease
inferred← citeshttps://doi.org/10.3389/fncel.2019.00046openalex-snapshot
Herpes Simplex Virus Type 1 Infection of the Central Nervous System: Insights Into Proposed Interrelationships With Neurodegenerative Disorders
inferred← citeshttps://doi.org/10.3389/fncel.2020.544612openalex-snapshot
Participation of Endosomes in Toll-Like Receptor 3 Transportation Pathway in Murine Astrocytes
inferred← citeshttps://doi.org/10.3389/fncel.2021.638686openalex-snapshot
Inflammation Spreading: Negative Spiral Linking Systemic Inflammatory Disorders and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3389/fncel.2021.691136openalex-snapshot
Microbial Infections Are a Risk Factor for Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.3389/fncel.2022.843790openalex-snapshot
Regulatory Roles of Antimicrobial Peptides in the Nervous System: Implications for Neuronal Aging
inferred← citeshttps://doi.org/10.3389/fneur.2024.1471736openalex-snapshot
Association between herpes zoster and Parkinson’s disease and dementia: a systematic review and meta-analysis
inferred← citeshttps://doi.org/10.3389/fnins.2021.806260openalex-snapshot
Specificity of Adaptive Immune Responses in Central Nervous System Health, Aging and Diseases
inferred← citeshttps://doi.org/10.3389/fnins.2024.1439055openalex-snapshot
A review of the roles of pathogens in Alzheimer’s disease
inferred← citeshttps://doi.org/10.3389/fnins.2024.1513095openalex-snapshot
Alzheimer’s disease and infectious agents: a comprehensive review of pathogenic mechanisms and microRNA roles
inferred← citeshttps://doi.org/10.3389/fnmol.2020.609073openalex-snapshot
Modulation of β-Amyloid Fibril Formation in Alzheimer’s Disease by Microglia and Infection
inferred← citeshttps://doi.org/10.3390/biom9080350openalex-snapshot
Accumulation of Amyloid Beta (Aβ) Peptide on Blood Vessel Walls in the Damaged Brain after Transient Middle Cerebral Artery Occlusion
inferred← citeshttps://doi.org/10.3390/biomedicines11041215openalex-snapshot
Controversial Properties of Amyloidogenic Proteins and Peptides: New Data in the COVID Era
inferred← citeshttps://doi.org/10.3390/biomedicines12091969openalex-snapshot
A Triple Mystery of Insidious Organ Failure: Are the Lung, Kidney and Brain All Damaged by the Ageing Pulse?
inferred← citeshttps://doi.org/10.3390/brainsci12081010openalex-snapshot
New Insights into the Molecular Interplay between Human Herpesviruses and Alzheimer’s Disease—A Narrative Review
inferred← citeshttps://doi.org/10.3390/ijms19061705openalex-snapshot
Amyloid Beta Peptide Is Released during Thrombosis in the Skin
inferred← citeshttps://doi.org/10.3390/ijms21031126openalex-snapshot
Impaired Innate Immunity Mechanisms in the Brain of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3390/ijms22147263openalex-snapshot
Activation of Endogenous Retrovirus, Brain Infections and Environmental Insults in Neurodegeneration and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3390/ijms232113502openalex-snapshot
The Increased Amyloidogenicity of Spike RBD and pH-Dependent Binding to ACE2 May Contribute to the Transmissibility and Pathogenic Properties of SARS-CoV-2 Omicron as Suggested by In Silico Study
inferred← citeshttps://doi.org/10.3390/ijms24032377openalex-snapshot
The Effects of Viruses on Insulin Sensitivity and Blood–Brain Barrier Function
inferred← citeshttps://doi.org/10.3390/ijms25010259openalex-snapshot
Re-Arranging the Puzzle between the Amyloid-Beta and Tau Pathology: An APP-Centric Approach
inferred← citeshttps://doi.org/10.3390/ph9030053openalex-snapshot
The Role of Antimicrobial Peptides in Influenza Virus Infection and Their Potential as Antiviral and Immunomodulatory Therapy
inferred← citeshttps://doi.org/10.3390/v11100966openalex-snapshot
Persistent Infection with Herpes Simplex Virus 1 and Alzheimer’s Disease—A Call to Study How Variability in Both Virus and Host may Impact Disease
inferred← citeshttps://doi.org/10.3390/v18070783openalex-snapshot
Neuroinflammation, Pericyte Dysfunction, and Alzheimer’s Disease-Associated Gene Expression and Pathway Activation in the Brain of SARS-CoV-2-Infected Mice
inferred← citeshttps://doi.org/10.3390/vaccines8030517openalex-snapshot
Immunomodulatory Role of the Antimicrobial LL-37 Peptide in Autoimmune Diseases and Viral Infections
inferred← citeshttps://doi.org/10.3410/f.734259403.793552075openalex-snapshot
Faculty Opinions recommendation of Corroboration of a major role for herpes simplex virus type 1 in alzheimer's disease.
inferred← citeshttps://doi.org/10.4049/jimmunol.2000037openalex-snapshot
Depletion of NK Cells Improves Cognitive Function in the Alzheimer Disease Mouse Model
inferred← citeshttps://doi.org/10.4103/nrr.nrr-d-25-00872openalex-snapshot
Analysis of the dual role of amyloid-beta in Alzheimer’s disease through multi-omics integration
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