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Dramatic Shifts in Circulating CD4 but not CD8 T Cell Subsets in Mild Alzheimer's Disease
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/0470871628.ch13openalex-snapshot
Regulatory Cells in Transplantation
inferredcites →https://doi.org/10.1002/ana.410100603openalex-snapshot
Immunological studies in senile dementia of the alzheimer type: Evidence for enhanced suppressor cell activity
inferredcites →https://doi.org/10.1006/clin.1995.1078openalex-snapshot
T Lymphocyte Subpopulations and Activation Markers Correlate with Severity of Alzheimer's Disease
inferredcites →https://doi.org/10.1007/s12017-007-8015-9openalex-snapshot
Increased T-cell Reactivity and Elevated Levels of CD8+ Memory T-cells in Alzheimer’s Disease-patients and T-cell Hyporeactivity in an Alzheimer’s Disease-mouse Model: Implications for Immunotherapy
inferredcites →https://doi.org/10.1016/0022-510x(92)90284-ropenalex-snapshot
The decrease of CD8-positive lymphocytes in Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.exger.2006.11.016openalex-snapshot
Aging and T-cell diversity☆
inferredcites →https://doi.org/10.1016/j.jneuroim.2007.05.011openalex-snapshot
Higher frequency of regulatory T cells in the elderly and increased suppressive activity in neurodegeneration
inferredcites →https://doi.org/10.1016/j.jpsychires.2006.01.010openalex-snapshot
Altered lymphocyte distribution in Alzheimer’s disease
inferredcites →https://doi.org/10.1016/j.nbd.2006.03.008openalex-snapshot
Aβ-specific T-cells reverse cognitive decline and synaptic loss in Alzheimer's mice
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2006.05.020openalex-snapshot
Lymphocyte subset patterns and cytokine production in Alzheimer's disease patients
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2006.07.007openalex-snapshot
Genetic risk profiles for Alzheimer's disease: Integration of APOE genotype and variants that up-regulate inflammation
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2006.08.004openalex-snapshot
Transcriptional profiling of Alzheimer blood mononuclear cells by microarray
inferredcites →https://doi.org/10.1016/s0047-6374(00)00240-2openalex-snapshot
The SENIEUR protocol after 16 years
inferredcites →https://doi.org/10.1016/s0047-6374(00)00242-6openalex-snapshot
The SENIEUR protocol after 16 years: the next step is to study the interaction of ageing and disease
inferredcites →https://doi.org/10.1016/s0074-7742(07)82011-3openalex-snapshot
Systemic and Acquired Immune Responses in Alzheimer's Disease
inferredcites →https://doi.org/10.1016/s0140-6736(08)61075-2openalex-snapshot
Long-term effects of Aβ42 immunisation in Alzheimer's disease: follow-up of a randomised, placebo-controlled phase I trial
inferredcites →https://doi.org/10.1016/s0165-5728(99)00046-6openalex-snapshot
Characterization of cytokine production, screening of lymphocyte subset patterns and in vitro apoptosis in healthy and Alzheimer's Disease (AD) individuals
inferredcites →https://doi.org/10.1016/s0197-4580(02)00043-xopenalex-snapshot
Telomere shortening in T cells correlates with Alzheimer's disease status
inferredcites →https://doi.org/10.1038/44385openalex-snapshot
Two subsets of memory T lymphocytes with distinct homing potentials and effector functions
inferredcites →https://doi.org/10.1038/nri1027openalex-snapshot
Regulatory T cells in transplantation tolerance
inferredcites →https://doi.org/10.1055/s-2006-954591openalex-snapshot
Apoptosis of CD4+ T and Natural Killer Cells in Alzheimer's Disease
inferredcites →https://doi.org/10.1093/gerona/60.5.556openalex-snapshot
An Immune Risk Phenotype, Cognitive Impairment, and Survival in Very Late Life: Impact of Allostatic Load in Swedish Octogenarian and Nonagenarian Humans
inferredcites →https://doi.org/10.1097/nen.0b013e318177eaf4openalex-snapshot
Alzheimer Disease Pathology As a Host Response
inferredcites →https://doi.org/10.1176/ajp.151.8.1105openalex-snapshot
Inflammatory mechanisms in Alzheimer's disease: implications for therapy
inferredcites →https://doi.org/10.1182/blood-2004-04-1596openalex-snapshot
The CD28 family: a T-cell rheostat for therapeutic control of T-cell activation
inferredcites →https://doi.org/10.1186/1742-2094-5-25openalex-snapshot
Interactions between APP secretases and inflammatory mediators
inferredcites →https://doi.org/10.1212/wnl.34.7.939openalex-snapshot
Clinical diagnosis of Alzheimer's disease
inferredcites →https://doi.org/10.1212/wnl.51.4.1014openalex-snapshot
Oxidative damage to DNA in lymphocytes from AD patients
inferredcites →https://doi.org/10.2147/ciia.2007.2.1.33openalex-snapshot
Immunosupportive therapies in aging
inferredcites →https://doi.org/10.2174/156720508784533330openalex-snapshot
Role of the Region 23-28 in Aβ Fibril Formation: Insights from Simulations of the Monomers and Dimers of Alzheimers Peptides Aβ40 and Aβ42
inferredcites →https://doi.org/10.2174/157488906775245354openalex-snapshot
Therapeutic Perspectives in Alzheimers Disease
inferredcites →https://doi.org/10.3233/jad-2008-13402openalex-snapshot
Inflammatory Aspects of Alzheimer Disease and Other Neurodegenerative Disorders
inferredcites →https://doi.org/10.4049/jimmunol.175.3.1566openalex-snapshot
CD25-Expressing CD8+ T Cells Are Potent Memory Cells in Old Age
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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T Cells—Protective or Pathogenic in Alzheimer’s Disease?
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Targeting Infectious Agents as a Therapeutic Strategy in Alzheimer’s Disease
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inferred← citeshttps://doi.org/10.1016/j.addr.2022.114398openalex-snapshot
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inferred← citeshttps://doi.org/10.1016/j.bbi.2010.12.004openalex-snapshot
Increased activity of Th-17 and Th-9 lymphocytes and a skewing of the post-thymic differentiation pathway are seen in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1016/j.bbi.2019.01.004openalex-snapshot
CREB signals as PBMC-based biomarkers of cognitive dysfunction: A novel perspective of the brain-immune axis
inferred← citeshttps://doi.org/10.1016/j.biopha.2019.108843openalex-snapshot
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inferred← citeshttps://doi.org/10.1016/j.cell.2025.11.035openalex-snapshot
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inferred← citeshttps://doi.org/10.1016/j.cellimm.2023.104778openalex-snapshot
Differential roles of regulatory T cells in Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.celrep.2023.113313openalex-snapshot
Stress regulates Alzheimer’s disease progression via selective enrichment of CD8+ T cells
inferred← citeshttps://doi.org/10.1016/j.coi.2022.102233openalex-snapshot
Spontaneous and induced adaptive immune responses in Alzheimer’s disease: new insights into old observations
inferred← citeshttps://doi.org/10.1016/j.conb.2022.102603openalex-snapshot
The role of antiviral CD8+ T cells in cognitive impairment
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Linking T cells to Alzheimer's disease: from neurodegeneration to neurorepair
inferred← citeshttps://doi.org/10.1016/j.csbj.2023.10.058openalex-snapshot
A glance through the effects of CD4 + T cells, CD8 + T cells, and cytokines on Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.exger.2015.03.002openalex-snapshot
Migration of blood cells to β-amyloid plaques in Alzheimer's disease
inferred← citeshttps://doi.org/10.1016/j.exger.2017.12.019openalex-snapshot
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inferred← citeshttps://doi.org/10.1016/j.intimp.2024.111807openalex-snapshot
CD28null T cells in aging and diseases: From biology to assessment and intervention
inferred← citeshttps://doi.org/10.1016/j.intimp.2025.116039openalex-snapshot
IL-24 ameliorates cognitive dysfunction via the inhibition PERK-eIF2α Signaling pathway in Alzheimer's disease
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Microglia crosstalk with T cells in neurodegenerative diseases: pathogenesis and treatment targets
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Plasma ceramide mediates the association of peripheral T cells with Alzheimer’s disease
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Tetravalent Aβ1–15 vaccine reduces TCR-positive cell infiltration and up-regulates p75 in Tg2576 brains compared to Aβ42 vaccine
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Immune profiling of Alzheimer patients
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inferred← citeshttps://doi.org/10.1016/j.jneuroim.2013.09.020openalex-snapshot
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Granulocyte-macrophage colony-stimulating factor neuroprotective activities in Alzheimer’s disease mice
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Long-term immunomodulatory effect of amniotic stem cells in an Alzheimer's disease model
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Time for the systems-level integration of aging: Resilience enhancing strategies to prevent Alzheimer’s disease
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inferred← citeshttps://doi.org/10.1038/s41467-024-54779-wopenalex-snapshot
Homeostatic microglia initially seed and activated microglia later reshape amyloid plaques in Alzheimer’s Disease
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T cell exhaustion is associated with cognitive status and amyloid accumulation in Alzheimer’s disease
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inferred← citeshttps://doi.org/10.1093/gerona/glt120openalex-snapshot
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inferred← citeshttps://doi.org/10.1111/bpa.12656openalex-snapshot
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inferred← citeshttps://doi.org/10.1111/ene.14360openalex-snapshot
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inferred← citeshttps://doi.org/10.1136/jnnp-2020-323603openalex-snapshot
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inferred← citeshttps://doi.org/10.1155/2022/1315248openalex-snapshot
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inferred← citeshttps://doi.org/10.1186/1743-422x-10-172openalex-snapshot
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inferred← citeshttps://doi.org/10.1186/s12974-017-0910-xopenalex-snapshot
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inferred← citeshttps://doi.org/10.1186/s12974-018-1380-5openalex-snapshot
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inferred← citeshttps://doi.org/10.1186/s12974-019-1554-9openalex-snapshot
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inferred← citeshttps://doi.org/10.1186/s12974-021-02308-7openalex-snapshot
CD4+ effector T cells accelerate Alzheimer’s disease in mice
inferred← citeshttps://doi.org/10.1186/s12974-022-02605-9openalex-snapshot
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Hyperactivation of monocytes and macrophages in MCI patients contributes to the progression of Alzheimer's disease
inferred← citeshttps://doi.org/10.1186/s12979-024-00443-2openalex-snapshot
Terminally differentiated effector memory T cells associate with cognitive and AD-related biomarkers in an aging-based community cohort
inferred← citeshttps://doi.org/10.1186/s12979-024-00445-0openalex-snapshot
Neuroinflammation in Alzheimer’s disease: insights from peripheral immune cells
inferred← citeshttps://doi.org/10.1186/s12979-025-00529-5openalex-snapshot
Systemic inflammation as a central player in the initiation and development of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1186/s13024-020-00375-7openalex-snapshot
Harnessing regulatory T cell neuroprotective activities for treatment of neurodegenerative disorders
inferred← citeshttps://doi.org/10.1186/s13024-023-00692-7openalex-snapshot
Amyloid-β specific regulatory T cells attenuate Alzheimer’s disease pathobiology in APP/PS1 mice
inferred← citeshttps://doi.org/10.1186/s13195-016-0226-6openalex-snapshot
LW-AFC, a new formula derived from Liuwei Dihuang decoction, ameliorates behavioral and pathological deterioration via modulating the neuroendocrine-immune system in PrP-hAβPPswe/PS1ΔE9 transgenic mice
inferred← citeshttps://doi.org/10.1186/s13195-017-0241-2openalex-snapshot
Role of neuroinflammation in neurodegeneration: new insights
inferred← citeshttps://doi.org/10.1186/s13195-026-02144-yopenalex-snapshot
Blood-based immunophenotyping of T cell profiles in patients with neurodegenerative disorders
inferred← citeshttps://doi.org/10.1189/jlb.3ri0716-335ropenalex-snapshot
Human T cell immunosenescence and inflammation in aging
inferred← citeshttps://doi.org/10.1371/journal.pone.0033276openalex-snapshot
Beta-Amyloid Peptides Enhance the Proliferative Response of Activated CD4+CD28+ Lymphocytes from Alzheimer Disease Patients and from Healthy Elderly
inferred← citeshttps://doi.org/10.1371/journal.pone.0066664openalex-snapshot
Enhanced Chemokine Receptor Expression on Leukocytes of Patients with Alzheimer's Disease
inferred← citeshttps://doi.org/10.1371/journal.pone.0077921openalex-snapshot
Decreased Proportion of Cytomegalovirus Specific CD8 T-Cells but No Signs of General Immunosenescence in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1371/journal.pone.0144052openalex-snapshot
Network Topology Analysis of Post-Mortem Brain Microarrays Identifies More Alzheimer’s Related Genes and MicroRNAs and Points to Novel Routes for Fighting with the Disease
inferred← citeshttps://doi.org/10.15226/2374-6858/2/1/00112openalex-snapshot
Alzheimer's Disease and the Immune System
inferred← citeshttps://doi.org/10.15789/2220-7619-iov-17834openalex-snapshot
Impact of viruses and retrotransposons on genetic instability and aging
inferred← citeshttps://doi.org/10.1586/ern.09.118openalex-snapshot
Can peripheral leukocytes be used as Alzheimer’s disease biomarkers?
inferred← citeshttps://doi.org/10.1615/critrevimmunol.2023050145openalex-snapshot
The Role of T Cells in Alzheimer's Disease Pathogenesis
inferred← citeshttps://doi.org/10.18632/aging.101990openalex-snapshot
CA-30, an oligosaccharide fraction derived from Liuwei Dihuang decoction, ameliorates cognitive deterioration via the intestinal microbiome in the senescence-accelerated mouse prone 8 strain
inferred← citeshttps://doi.org/10.18632/aging.203090openalex-snapshot
Cistanche deserticola polysaccharides alleviate cognitive decline in aging model mice by restoring the gut microbiota-brain axis
inferred← citeshttps://doi.org/10.18632/aging.203803openalex-snapshot
Retinal drusen in patients with chronic myeloproliferative blood cancers are associated with an increased proportion of senescent T cells and signs of an aging immune system
inferred← citeshttps://doi.org/10.18632/oncotarget.10777openalex-snapshot
Immunomodulation by memantine in therapy of Alzheimer's disease is mediated through inhibition of Kv1.3 channels and T cell responsiveness
inferred← citeshttps://doi.org/10.18632/oncotarget.4930openalex-snapshot
Splenocytes derived from young WT mice prevent AD progression in APPswe/PSENldE9 transgenic mice
inferred← citeshttps://doi.org/10.21203/rs.3.rs-2153516/v2openalex-snapshot
Alpha-synuclein-specific regulatory T cells ameliorate Parkinson’s disease progression in mice
inferred← citeshttps://doi.org/10.2147/ndt.s264910openalex-snapshot
Targeting Impaired Antimicrobial Immunity in the Brain for the Treatment of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.2147/opth.s82116openalex-snapshot
The association between neovascular age-related macular degeneration and regulatory T cells in peripheral blood
inferred← citeshttps://doi.org/10.2174/138161210790883769openalex-snapshot
Immune-Inflammatory Responses and Oxidative Stress in Alzheimers Disease: Therapeutic Implications
inferred← citeshttps://doi.org/10.2174/2212707003666160810143854openalex-snapshot
Effectiveness of the Treatment with Botulinum Toxin Type A (BTX-A) in the Management of the Spasticity in Patients with Amyotrophic Lateral Sclerosis (ALS)
inferred← citeshttps://doi.org/10.2217/ahe.12.78openalex-snapshot
The Immune System in the Elderly: A Fair Fight Against Diseases?
inferred← citeshttps://doi.org/10.31083/jin26393openalex-snapshot
Frequencies of Circulating Immune Cells in Patients with Parkinson’s Disease: Correlation with MDS-UPDRS Scores
inferred← citeshttps://doi.org/10.31887/dcns.2019.21.1/vharoutunianopenalex-snapshot
Is Alzheimer disease a failure of mobilizing immune defense? Lessons from cognitively fit oldest-old
inferred← citeshttps://doi.org/10.3233/jad-121984openalex-snapshot
Alzheimer's Disease Modeling: Ups, Downs, and Perspectives for Human Induced Pluripotent Stem Cells
inferred← citeshttps://doi.org/10.3233/jad-140861openalex-snapshot
Function and Redox State of Peritoneal Leukocytes as Preclinical and Prodromic Markers in a Longitudinal Study of Triple-Transgenic Mice for Alzheimer's Disease
inferred← citeshttps://doi.org/10.3233/jad-142412openalex-snapshot
Double Negative (IgG + IgD − CD27 − ) B Cells are Increased in a Cohort of Moderate-Severe Alzheimer's Disease Patients and Show a Pro-Inflammatory Trafficking Receptor Phenotype
inferred← citeshttps://doi.org/10.3233/jad-150652openalex-snapshot
Protective Effect of Amyloid-β Peptides Against Herpes Simplex Virus-1 Infection in a Neuronal Cell Culture Model
inferred← citeshttps://doi.org/10.3233/jad-160517openalex-snapshot
Anti-Viral Properties of Amyloid-β Peptides
inferred← citeshttps://doi.org/10.3233/jad-160677openalex-snapshot
Amylin Treatment Reduces Neuroinflammation and Ameliorates Abnormal Patterns of Gene Expression in the Cerebral Cortex of an Alzheimer’s Disease Mouse Model
inferred← citeshttps://doi.org/10.3233/jad-161304openalex-snapshot
Alterations in the Peripheral Immune System in Dementia
inferred← citeshttps://doi.org/10.3233/jad-170124openalex-snapshot
Polymorphonuclear Neutrophil Functions are Differentially Altered in Amnestic Mild Cognitive Impairment and Mild Alzheimer’s Disease Patients
inferred← citeshttps://doi.org/10.3233/jad-2010-091696openalex-snapshot
PD1 Negative and PD1 Positive CD4+ T Regulatory Cells in Mild Cognitive Impairment and Alzheimer's Disease
inferred← citeshttps://doi.org/10.3233/jad-2010-091714openalex-snapshot
Systemic Immune Responses in Alzheimer's Disease: In Vitro Mononuclear Cell Activation and Cytokine Production
inferred← citeshttps://doi.org/10.3233/jad-220091openalex-snapshot
Association of Peripheral Lymphocyte Subsets with Cognitive Decline and Dementia: The Cardiovascular Health Study
inferred← citeshttps://doi.org/10.3233/jad-220261openalex-snapshot
B Lymphocytes in Alzheimer’s Disease—A Comprehensive Review
inferred← citeshttps://doi.org/10.3233/jpd-223228openalex-snapshot
Age-Related Adaptive Immune Changes in Parkinson’s Disease
inferred← citeshttps://doi.org/10.3389/fcimb.2020.00098openalex-snapshot
Fecal Microbiota Transplantation in Neurological Disorders
inferred← citeshttps://doi.org/10.3389/fimmu.2013.00271openalex-snapshot
Human T Cell Aging and the Impact of Persistent Viral Infections
inferred← citeshttps://doi.org/10.3389/fimmu.2017.00783openalex-snapshot
Differential Phenotypes of Myeloid-Derived Suppressor and T Regulatory Cells and Cytokine Levels in Amnestic Mild Cognitive Impairment Subjects Compared to Mild Alzheimer Diseased Patients
inferred← citeshttps://doi.org/10.3389/fimmu.2018.01213openalex-snapshot
Imbalance of Circulating Th17 and Regulatory T Cells in Alzheimer’s Disease: A Case Control Study
inferred← citeshttps://doi.org/10.3389/fimmu.2019.02171openalex-snapshot
Protective and Regenerative Roles of T Cells in Central Nervous System Disorders
inferred← citeshttps://doi.org/10.3389/fimmu.2023.1325530openalex-snapshot
The immunomodulatory effects of mesenchymal stem cell-derived extracellular vesicles in Alzheimer's disease
inferred← citeshttps://doi.org/10.3389/fnagi.2015.00061openalex-snapshot
miR-155 is involved in Alzheimer’s disease by regulating T lymphocyte function
inferred← citeshttps://doi.org/10.3389/fnagi.2020.624304openalex-snapshot
Mild Cognitive Impairment Patients Have Higher Regulatory T-Cell Proportions Compared With Alzheimer's Disease-Related Dementia Patients
inferred← citeshttps://doi.org/10.3389/fnagi.2021.650047openalex-snapshot
Roles and Mechanisms of Gut Microbiota in Patients With Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3389/fnagi.2022.888946openalex-snapshot
Association of Peripheral Blood Cell Profile With Alzheimer's Disease: A Meta-Analysis
inferred← citeshttps://doi.org/10.3389/fnagi.2026.1838306openalex-snapshot
Treg–microglia crosstalk in Alzheimer’s disease: stage-dependent dynamics, molecular mechanisms, and translational challenge
inferred← citeshttps://doi.org/10.3389/fncel.2023.1281763openalex-snapshot
CD8+ T cells in brain injury and neurodegeneration
inferred← citeshttps://doi.org/10.3389/fnins.2020.614828openalex-snapshot
Acylated Ghrelin as a Multi-Targeted Therapy for Alzheimer's and Parkinson's Disease
inferred← citeshttps://doi.org/10.3389/fnins.2021.806260openalex-snapshot
Specificity of Adaptive Immune Responses in Central Nervous System Health, Aging and Diseases
inferred← citeshttps://doi.org/10.3389/fnins.2024.1488382openalex-snapshot
Myeloid antigen-presenting cells in neurodegenerative diseases: a focus on classical and non-classical MHC molecules
inferred← citeshttps://doi.org/10.3390/cells13090749openalex-snapshot
CD4+ T-Cell Senescence in Neurodegenerative Disease: Pathogenesis and Potential Therapeutic Targets
inferred← citeshttps://doi.org/10.3390/diseases9020029openalex-snapshot
Cholinergic Modulation of the Immune System in Neuroinflammatory Diseases
inferred← citeshttps://doi.org/10.3390/ijms24065925openalex-snapshot
Immune Regulatory Functions of Macrophages and Microglia in Central Nervous System Diseases
inferred← citeshttps://doi.org/10.3390/ijms242015237openalex-snapshot
Alpha-Synuclein-Specific Regulatory T Cells Ameliorate Parkinson’s Disease Progression in Mice
inferred← citeshttps://doi.org/10.3390/ijms25020783openalex-snapshot
Therapeutic Effects of Aβ-Specific Regulatory T Cells in Alzheimer’s Disease: A Study in 5xFAD Mice
inferred← citeshttps://doi.org/10.3390/md16090313openalex-snapshot
Cognitive Impairment in Chronic Obstructive Pulmonary Disease (COPD): Possible Utility of Marine Bioactive Compounds
inferred← citeshttps://doi.org/10.3390/microorganisms10020424openalex-snapshot
Evaluation of BCG Vaccination and Plasma Amyloid: A Prospective, Pilot Study with Implications for Alzheimer’s Disease
inferred← citeshttps://doi.org/10.3724/cbls.2026102openalex-snapshot
Research progress on the mechanism and intervention strategies of immunosenescence
inferred← citeshttps://doi.org/10.4081/ar.2010.e5openalex-snapshot
The central role of T-cell memory in Alzheimer’s disease vaccination
inferred← citeshttps://doi.org/10.4082/kjfm.2010.31.10.747openalex-snapshot
Integrative Approach to Elderly Frailty
inferred← citeshttps://doi.org/10.4103/1673-5374.357903openalex-snapshot
Neuro faces of beneficial T cells: essential in brain, impaired in aging and neurological diseases, and activated functionally by neurotransmitters and neuropeptides
inferred← citeshttps://doi.org/10.4103/nrr.nrr-d-24-00793openalex-snapshot
Alzheimer’s disease and the immune system: the emerging role of TEMRA cells
inferred← citeshttps://doi.org/10.56499/jppres23.1742_12.1.154openalex-snapshot
Evaluation of the beneficial effects of ETAS® on normal aging or mild cognitive impairment subjects: A pilot randomized controlled trial
inferred← citeshttps://doi.org/10.6084/m9.figshare.14821079openalex-snapshot
Additional file 1 of Hyperactivation of monocytes and macrophages in MCI patients contributes to the progression of Alzheimer's disease
inferred← citeshttps://doi.org/10.6084/m9.figshare.14821082openalex-snapshot
Additional file 2 of Hyperactivation of monocytes and macrophages in MCI patients contributes to the progression of Alzheimer's disease
inferred← citeshttps://doi.org/10.6084/m9.figshare.14821085openalex-snapshot
Additional file 3 of Hyperactivation of monocytes and macrophages in MCI patients contributes to the progression of Alzheimer's disease
inferred← citeshttps://doi.org/10.6084/m9.figshare.14821091openalex-snapshot
Additional file 4 of Hyperactivation of monocytes and macrophages in MCI patients contributes to the progression of Alzheimer's disease
inferred← citeshttps://doi.org/10.6084/m9.figshare.24864141openalex-snapshot
Additional file 1 of Amyloid-β specific regulatory T cells attenuate Alzheimer’s disease pathobiology in APP/PS1 mice
inferred← citeshttps://doi.org/10.70675/0542df04zd02ez4a58za844z605f237e8f0bopenalex-snapshot
Implication des polynucléaires neutrophiles au cours de la maladie d’Alzheimer et des tauopathies
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