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Re-Examining the Role of TNF in MS Pathogenesis and Therapy
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Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/archneur.60.8.1082openalex-snapshot
Excitatory Amino Acids and Multiple Sclerosis
inferredcites →https://doi.org/10.1002/(sici)1521-4141(199902)29:02<626::aid-immu626>3.0.co;2-aopenalex-snapshot
Severity of symptoms and demyelination in MOG-induced EAE depends on TNFR1
inferredcites →https://doi.org/10.1002/(sici)1521-4141(199903)29:03<774::aid-immu774>3.0.co;2-topenalex-snapshot
TNF accelerates the onset but does not alter the incidence and severity of myelin basic protein-induced experimental autoimmune encephalomyelitis
inferredcites →https://doi.org/10.1002/1097-0029(20000801)50:3<184::aid-jemt2>3.0.co;2-hopenalex-snapshot
TNF? and the TNF receptor superfamily: Structure-function relationship(s)
inferredcites →https://doi.org/10.1002/1529-0131(200112)44:12<2862::aid-art474>3.0.co;2-wopenalex-snapshot
Demyelination occurring during anti-tumor necrosis factor ? therapy for inflammatory arthritides
inferredcites →https://doi.org/10.1002/ana.1077openalex-snapshot
Multiple sclerosis: Altered glutamate homeostasis in lesions correlates with oligodendrocyte and axonal damage
inferredcites →https://doi.org/10.1002/ana.20913openalex-snapshot
How to successfully apply animal studies in experimental allergic encephalomyelitis to research on multiple sclerosis
inferredcites →https://doi.org/10.1002/ana.24408openalex-snapshot
Cerebrospinal fluid markers reveal intrathecal inflammation in progressive multiple sclerosis
inferredcites →https://doi.org/10.1002/ana.25197openalex-snapshot
Inflammatory intrathecal profiles and cortical damage in multiple sclerosis
inferredcites →https://doi.org/10.1002/ana.25786openalex-snapshot
The CSF Profile Linked to Cortical Damage Predicts Multiple Sclerosis Activity
inferredcites →https://doi.org/10.1002/ana.410370404openalex-snapshot
The adhesion molecule and cytokine profile of multiple sclerosis lesions
inferredcites →https://doi.org/10.1002/eji.1830180824openalex-snapshot
Both interleukin 1 and tumor necrosis factor enhance thymocyte proliferation
inferredcites →https://doi.org/10.1002/eji.1830270424openalex-snapshot
Differential generation of class I H‐2D‐ versus H‐2K‐restricted cytotoxicity against a demyelinating virus following central nervous system infection
inferredcites →https://doi.org/10.1002/eji.1830270822openalex-snapshot
Tumor necrosis factor blockade in actively induced experimental autoimmune encephalomyelitis prevents clinical disease despite activated T cell infiltration to the central nervous system
inferredcites →https://doi.org/10.1002/eji.200635921openalex-snapshot
Transmembrane TNF protects mutant mice against intracellular bacterial infections, chronic inflammation and autoimmunity
inferredcites →https://doi.org/10.1002/eji.201040639openalex-snapshot
TNFR1 delivers pro‐survival signals that are required for limiting TNFR2‐dependent activation‐induced cell death (AICD) in CD8+ T cells
inferredcites →https://doi.org/10.1002/eji.201041205openalex-snapshot
TNF optimally activatives regulatory T cells by inducing TNF receptor superfamily members TNFR2, 4‐1BB and OX40
inferredcites →https://doi.org/10.1002/eji.201141659openalex-snapshot
TNFR2 on non‐haematopoietic cells is required for Foxp3+ Treg‐cell function and disease suppression in EAE
inferredcites →https://doi.org/10.1002/glia.20089openalex-snapshot
Astroglial‐derived lymphotoxin‐α exacerbates inflammation and demyelination, but not remyelination
inferredcites →https://doi.org/10.1002/glia.20297openalex-snapshot
Upregulation of the stress‐associated gene p8 in mouse models of demyelination and in multiple sclerosis tissues
inferredcites →https://doi.org/10.1006/cimm.2000.1706openalex-snapshot
Divergent Roles for p55 and p75 Tumor Necrosis Factor Receptors in the Pathogenesis of MOG35-55-Induced Experimental Autoimmune Encephalomyelitis
inferredcites →https://doi.org/10.1006/cyto.2002.1984openalex-snapshot
CEREBROSPINAL FLUID AND SERUM PROTEIN LEVELS OF TUMOUR NECROSIS FACTOR-ALPHA (TNF-α), INTERLEUKIN-6 (IL-6) AND SOLUBLE INTERLEUKIN-6 RECEPTOR (sIL-6R gp80) IN MULTIPLE SCLEROSIS PATIENTS
inferredcites →https://doi.org/10.1006/exnr.2002.7871openalex-snapshot
TNFα Transport across the Blood–Brain Barrier Is Abolished in Receptor Knockout Mice
inferredcites →https://doi.org/10.1006/nbdi.2001.0414openalex-snapshot
Tumor Necrosis Factor-α Induces cFOS and Strongly Potentiates Glutamate-Mediated Cell Death in the Rat Spinal Cord
inferredcites →https://doi.org/10.1007/978-1-4615-4685-6_20openalex-snapshot
Transgenic Models of Tnf Induced Demyelination
inferredcites →https://doi.org/10.1007/978-3-7091-6301-6_19openalex-snapshot
Changes in cytokines and neurotrophins in Parkinson’s disease
inferredcites →https://doi.org/10.1007/s00011-019-01286-0openalex-snapshot
Tumor necrosis factor alpha (TNF-α) and its soluble receptors are associated with disability, disability progression and clinical forms of multiple sclerosis
inferredcites →https://doi.org/10.1007/s001150050636openalex-snapshot
Multiple Sklerose Aktuelle Übersicht zu fehlgeschlagenen und abgebrochenen Therapiestudien
inferredcites →https://doi.org/10.1007/s00415-005-5002-7openalex-snapshot
The pathology of multiple sclerosis is the result of focal inflammatory demyelination with axonal damage
inferredcites →https://doi.org/10.1007/s00415-006-0090-6openalex-snapshot
TNF–α converting enzyme (TACE) protein expression in different clinical subtypes of multiple sclerosis
inferredcites →https://doi.org/10.1007/s00702-009-0342-6openalex-snapshot
Mild experimental autoimmune encephalitis as a tool to induce blood–brain barrier dysfunction
inferredcites →https://doi.org/10.1007/s00702-014-1188-0openalex-snapshot
The role of glutamate and its receptors in multiple sclerosis
inferredcites →https://doi.org/10.1007/s11910-017-0742-1openalex-snapshot
CNS Demyelination with TNF-α Blockers
inferredcites →https://doi.org/10.1007/s12035-016-9846-xopenalex-snapshot
Cytokine Profile in Patients with Progressive Multiple Sclerosis and Its Association with Disease Progression and Disability
inferredcites →https://doi.org/10.1016/0090-1229(92)90036-nopenalex-snapshot
The role of tumor necrosis factor in sepsis
inferredcites →https://doi.org/10.1016/0092-8674(86)90780-4openalex-snapshot
Induction of β2-interferon by tumor necrosis factor: A homeostatic mechanism in the control of cell proliferation
inferredcites →https://doi.org/10.1016/0092-8674(93)90134-copenalex-snapshot
Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection
inferredcites →https://doi.org/10.1016/0092-8674(95)90192-2openalex-snapshot
The transmembrane form of tumor necrosis factor is the prime activating ligand of the 80 kDa tumor necrosis factor receptor
inferredcites →https://doi.org/10.1016/0165-5728(91)90182-7openalex-snapshot
Soluble CD4 and CD8 in the peripheral blood of patients with multiple sclerosis and HTLV-1-associated myelopathy
inferredcites →https://doi.org/10.1016/0165-5728(96)00032-xopenalex-snapshot
Multiple sclerosis: the proinflammatory cytokines lymphotoxin-α and tumour necrosis factor-α are upregulated in cerebrospinal fluid mononuclear cells
inferredcites →https://doi.org/10.1016/0304-3940(91)90490-kopenalex-snapshot
Elevated circulating tumor necrosis factor levels in Alzheimer's disease
inferredcites →https://doi.org/10.1016/0304-3940(94)90684-xopenalex-snapshot
Immunocytochemical analysis of tumor necrosis factor and its receptors in Parkinson's disease
inferredcites →https://doi.org/10.1016/0896-6273(88)90162-6openalex-snapshot
Glutamate neurotoxicity and diseases of the nervous system
inferredcites →https://doi.org/10.1016/1074-7613(95)90001-2openalex-snapshot
Tumor necrosis factor-α is required in the protective immune response against mycobacterium tuberculosis in mice
inferredcites →https://doi.org/10.1016/j.bbi.2019.06.021openalex-snapshot
Exogenous activation of tumor necrosis factor receptor 2 promotes recovery from sensory and motor disease in a model of multiple sclerosis
inferredcites →https://doi.org/10.1016/j.bbi.2019.11.017openalex-snapshot
Oligodendrocytes modulate the immune-inflammatory response in EAE via TNFR2 signaling
inferredcites →https://doi.org/10.1016/j.brainres.2004.11.014openalex-snapshot
TNFα potentiates glutamate neurotoxicity by inhibiting glutamate uptake in organotypic brain slice cultures: neuroprotection by NFκB inhibition
inferredcites →https://doi.org/10.1016/j.cell.2006.03.040openalex-snapshot
Miniature Neurotransmission Stabilizes Synaptic Function via Tonic Suppression of Local Dendritic Protein Synthesis
inferredcites →https://doi.org/10.1016/j.cell.2015.11.023openalex-snapshot
Neuroinflammatory TNFα Impairs Memory via Astrocyte Signaling
inferredcites →https://doi.org/10.1016/j.celrep.2015.02.051openalex-snapshot
Activation of Necroptosis in Multiple Sclerosis
inferredcites →https://doi.org/10.1016/j.celrep.2016.11.083openalex-snapshot
Opposing Functions of Microglial and Macrophagic TNFR2 in the Pathogenesis of Experimental Autoimmune Encephalomyelitis
inferredcites →https://doi.org/10.1016/j.cyto.2015.08.260openalex-snapshot
The role of cytokines in the pathogenesis of rheumatoid arthritis – Practical and potential application of cytokines as biomarkers and targets of personalized therapy
inferredcites →https://doi.org/10.1016/j.cyto.2018.05.036openalex-snapshot
Activated human Foxp3+ regulatory T cells produce membrane-bound TNF
inferredcites →https://doi.org/10.1016/j.cytogfr.2014.07.016openalex-snapshot
TNF and TNF-receptors: From mediators of cell death and inflammation to therapeutic giants – past, present and future
inferredcites →https://doi.org/10.1016/j.expneurol.2012.07.010openalex-snapshot
TNF-α-mediated anxiety in a mouse model of multiple sclerosis
inferredcites →https://doi.org/10.1016/j.febslet.2011.04.025openalex-snapshot
Contrasting effects of TNF and anti‐TNF on the activation of effector T cells and regulatory T cells in autoimmunity
inferredcites →https://doi.org/10.1016/j.iac.2009.02.001openalex-snapshot
The Blood–Brain Barrier in Psychoneuroimmunology
inferredcites →https://doi.org/10.1016/j.jaut.2016.06.015openalex-snapshot
Meningeal mast cell-T cell crosstalk regulates T cell encephalitogenicity
inferredcites →https://doi.org/10.1016/j.jcm.2016.04.016openalex-snapshot
The Physiological Role of Tumor Necrosis Factor in Human Immunity and Its Potential Implications in Spinal Manipulative Therapy: A Narrative Literature Review
inferredcites →https://doi.org/10.1016/j.jconrel.2009.12.015openalex-snapshot
Therapeutic effect of PEGylated TNFR1-selective antagonistic mutant TNF in experimental autoimmune encephalomyelitis mice
inferredcites →https://doi.org/10.1016/j.jneuroim.2011.04.006openalex-snapshot
The prospects of minocycline in multiple sclerosis
inferredcites →https://doi.org/10.1016/j.ncl.2006.03.009openalex-snapshot
The Blood–Brain Barrier in Psychoneuroimmunology
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2006.02.012openalex-snapshot
Serum TNF-alpha levels are increased and correlate negatively with free IGF-I in Alzheimer disease
inferredcites →https://doi.org/10.1016/j.neuron.2008.02.031openalex-snapshot
Rapid Synaptic Scaling Induced by Changes in Postsynaptic Firing
inferredcites →https://doi.org/10.1016/j.neuron.2009.09.001openalex-snapshot
Immune Proteins in Brain Development and Synaptic Plasticity
inferredcites →https://doi.org/10.1016/j.neuron.2011.02.003openalex-snapshot
TNFα Controls Glutamatergic Gliotransmission in the Hippocampal Dentate Gyrus
inferredcites →https://doi.org/10.1016/j.neuroscience.2015.06.038openalex-snapshot
TNF and its receptors in the CNS: The essential, the desirable and the deleterious effects
inferredcites →https://doi.org/10.1016/j.pharmthera.2007.10.001openalex-snapshot
Tumor necrosis factor antagonist mechanisms of action: A comprehensive review
inferredcites →https://doi.org/10.1016/j.smim.2014.05.004openalex-snapshot
Regulation of NF-κB by TNF family cytokines
inferredcites →https://doi.org/10.1016/j.tins.2009.05.001openalex-snapshot
Tripartite synapses: astrocytes process and control synaptic information
inferredcites →https://doi.org/10.1016/j.tins.2012.06.001openalex-snapshot
TNFα in synaptic function: switching gears
inferredcites →https://doi.org/10.1016/j.vaccine.2010.08.103openalex-snapshot
Experimental autoimmune encephalomyelitis as a testing paradigm for adjuvants and vaccines
inferredcites →https://doi.org/10.1016/j.virol.2010.02.031openalex-snapshot
A critical role for virus-specific CD8+ CTLs in protection from Theiler's virus-induced demyelination in disease-susceptible SJL mice
inferredcites →https://doi.org/10.1016/s0002-9440(10)65622-2openalex-snapshot
Oligodendrocyte Apoptosis and Primary Demyelination Induced by Local TNF/p55TNF Receptor Signaling in the Central Nervous System of Transgenic Mice
inferredcites →https://doi.org/10.1016/s0006-2952(99)00296-8openalex-snapshot
Oxidative stress and nuclear factor-κB activation
inferredcites →https://doi.org/10.1016/s0022-510x(97)00142-1openalex-snapshot
Tumor necrosis factor α and its receptors in relapsing-remitting multiple sclerosis
inferredcites →https://doi.org/10.1016/s0140-6736(94)90628-9openalex-snapshot
Randomised double-blind comparison of chimeric monoclonal antibody to tumour necrosis factor α (cA2) versus placebo in rheumatoid arthritis
inferredcites →https://doi.org/10.1016/s0165-5728(00)00396-9openalex-snapshot
Monocytes in multiple sclerosis: phenotype and cytokine profile
inferredcites →https://doi.org/10.1016/s0165-5728(96)00183-xopenalex-snapshot
TNF-α receptor fusion protein prevents experimental auto-immune encephalomyelitis and demyelination in Lewis rats: an overview
inferredcites →https://doi.org/10.1016/s0165-5728(97)00009-xopenalex-snapshot
Differential expression, cytokine modulation, and specific functions of type-1 and type-2 tumor necrosis factor receptors in rat glia
inferredcites →https://doi.org/10.1016/s0896-6273(00)80086-0openalex-snapshot
Knockout of Glutamate Transporters Reveals a Major Role for Astroglial Transport in Excitotoxicity and Clearance of Glutamate
inferredcites →https://doi.org/10.1016/s1043-4666(05)80008-0openalex-snapshot
Cytokine mRNA expression in inflammatory multiple sclerosis lesions: Detection by non-radioactive in situ hybridization
inferredcites →https://doi.org/10.1016/s1074-7613(01)00215-1openalex-snapshot
Membrane-Bound TNF Supports Secondary Lymphoid Organ Structure but Is Subservient to Secreted TNF in Driving Autoimmune Inflammation
inferredcites →https://doi.org/10.1016/s1474-4422(14)70101-2openalex-snapshot
Pathogenesis of multiple sclerosis: insights from molecular and metabolic imaging
inferredcites →https://doi.org/10.1016/s1474-4422(18)30460-5openalex-snapshot
Spinal cord involvement in multiple sclerosis and neuromyelitis optica spectrum disorders
inferredcites →https://doi.org/10.1017/s1740925x05000608openalex-snapshot
TNFα-induced AMPA-receptor trafficking in CNS neurons; relevance to excitotoxicity?
inferredcites →https://doi.org/10.1023/a:1021608724117openalex-snapshot
Expression of the p75 TNF Receptor Is Linked to TNF-Induced NFκB Translocation and Oxyradical Neutralization in Glial Cells
inferredcites →https://doi.org/10.1038/32588openalex-snapshot
Dendritic cells and the control of immunity
inferredcites →https://doi.org/10.1038/cdd.2009.179openalex-snapshot
The link between inflammation, synaptic transmission and neurodegeneration in multiple sclerosis
inferredcites →https://doi.org/10.1038/nature04671openalex-snapshot
Synaptic scaling mediated by glial TNF-α
inferredcites →https://doi.org/10.1038/nature11307openalex-snapshot
TNF receptor 1 genetic risk mirrors outcome of anti-TNF therapy in multiple sclerosis
inferredcites →https://doi.org/10.1038/ni.1818openalex-snapshot
Regulatory T cells exert checks and balances on self tolerance and autoimmunity
inferredcites →https://doi.org/10.1038/ni1178openalex-snapshot
Naturally arising Foxp3-expressing CD25+CD4+ regulatory T cells in immunological tolerance to self and non-self
inferredcites →https://doi.org/10.1038/nm0198-078openalex-snapshot
TNF is a potent anti-inflammatory cytokine in autoimmune-mediated demyelination
inferredcites →https://doi.org/10.1038/nn738openalex-snapshot
TNFα promotes proliferation of oligodendrocyte progenitors and remyelination
inferredcites →https://doi.org/10.1038/nri2526openalex-snapshot
The role of TNF superfamily members in T-cell function and diseases
inferredcites →https://doi.org/10.1038/nri3871openalex-snapshot
Immunopathology of multiple sclerosis
inferredcites →https://doi.org/10.1038/nrn3900openalex-snapshot
Exploring the origins of grey matter damage in multiple sclerosis
inferredcites →https://doi.org/10.1038/nrneurol.2015.222openalex-snapshot
Synaptopathy connects inflammation and neurodegeneration in multiple sclerosis
inferredcites →https://doi.org/10.1038/nrrheum.2015.169openalex-snapshot
TNF biology, pathogenic mechanisms and emerging therapeutic strategies
inferredcites →https://doi.org/10.1038/s41598-017-13984-yopenalex-snapshot
TNFR1 inhibition with a Nanobody protects against EAE development in mice
inferredcites →https://doi.org/10.1038/s41598-018-31957-7openalex-snapshot
Anti-TNFR1 targeting in humanized mice ameliorates disease in a model of multiple sclerosis
inferredcites →https://doi.org/10.1038/sj.cdd.4401189openalex-snapshot
Tumor necrosis factor signaling
inferredcites →https://doi.org/10.1046/j.1365-2249.2003.02053.xopenalex-snapshot
Monocyte-derived cytokines in multiple sclerosis
inferredcites →https://doi.org/10.1046/j.1365-2362.1997.2250774.xopenalex-snapshot
Neurotransmitters in cerebrospinal fluid reflect pathological activity
inferredcites →https://doi.org/10.1046/j.1471-4159.1998.70051876.xopenalex-snapshot
The Transcription Factor NF‐κB Mediates Increases in Calcium Currents and Decreases in NMDA‐ and AMPA/Kainate‐Induced Currents Induced by Tumor Necrosis Factor‐α in Hippocampal Neurons
inferredcites →https://doi.org/10.1056/nejm199108153250704openalex-snapshot
Association between Tumor Necrosis Factor-α and Disease Progression in Patients with Multiple Sclerosis
inferredcites →https://doi.org/10.1073/pnas.0701252104openalex-snapshot
Loss of blood–brain barrier integrity in the spinal cord is common to experimental allergic encephalomyelitis in knockout mouse models
inferredcites →https://doi.org/10.1073/pnas.1807499115openalex-snapshot
Intrinsic TNFR2 signaling in T regulatory cells provides protection in CNS autoimmunity
inferredcites →https://doi.org/10.1073/pnas.88.20.9292openalex-snapshot
The two different receptors for tumor necrosis factor mediate distinct cellular responses.
inferredcites →https://doi.org/10.1073/pnas.92.24.11294openalex-snapshot
Spontaneous inflammatory demyelinating disease in transgenic mice showing central nervous system-specific expression of tumor necrosis factor alpha.
inferredcites →https://doi.org/10.1074/jbc.m311766200openalex-snapshot
Tumor Necrosis Factor (TNF)-mediated Neuroprotection against Glutamate-induced Excitotoxicity Is Enhanced by N-Methyl-D-aspartate Receptor Activation
inferredcites →https://doi.org/10.1076/apab.109.4.350.4238openalex-snapshot
Upregulation of the Transport System for TNFa at the Blood-Brain Barrier
inferredcites →https://doi.org/10.1080/01616412.2017.1420522openalex-snapshot
Circulating levels of interleukin-17A, tumor necrosis factor-alpha, interleukin-18, interleukin-10, and cognitive performance of patients with relapsing-remitting multiple sclerosis
inferredcites →https://doi.org/10.1080/08916930400001859openalex-snapshot
Treatment with Soluble Tumor Necrosis Factor Receptor (sTNFR):Fc/p80 Fusion Protein Ameliorates Relapsing-remitting Experimental Autoimmune Encephalomyelitis and Decreases Chemokine Expression
inferredcites →https://doi.org/10.1084/jem.170.2.607openalex-snapshot
Tumor necrosis factor identified in multiple sclerosis brain.
inferredcites →https://doi.org/10.1084/jem.184.4.1397openalex-snapshot
Immune and inflammatory responses in TNF alpha-deficient mice: a critical requirement for TNF alpha in the formation of primary B cell follicles, follicular dendritic cell networks and germinal centers, and in the maturation of the humoral immune response.
inferredcites →https://doi.org/10.1084/jem.187.9.1517openalex-snapshot
Challenging Cytokine Redundancy: Inflammatory Cell Movement and Clinical Course of Experimental Autoimmune Encephalomyelitis Are Normal in Lymphotoxin-deficient, but Not Tumor Necrosis Factor–deficient, Mice
inferredcites →https://doi.org/10.1084/jem.191.5.813openalex-snapshot
Mast Cells Are Essential for Early Onset and Severe Disease in a Murine Model of Multiple Sclerosis
inferredcites →https://doi.org/10.1084/jem.193.4.427openalex-snapshot
Uncoupling the Proinflammatory from the Immunosuppressive Properties of Tumor Necrosis Factor (Tnf) at the P55 TNF Receptor Level
inferredcites →https://doi.org/10.1084/jem.20020110openalex-snapshot
Phenotype, Localization, and Mechanism of Suppression of CD4+CD25+ Human Thymocytes
inferredcites →https://doi.org/10.1084/jem.20191390openalex-snapshot
Pro-inflammatory activation following demyelination is required for myelin clearance and oligodendrogenesis
inferredcites →https://doi.org/10.1089/neu.2009.1016openalex-snapshot
Tumor Necrosis Factor Alpha Enhances Glutamatergic Transmission onto Spinal Motoneurons
inferredcites →https://doi.org/10.1093/brain/119.1.213openalex-snapshot
Augmented expression of tumour necrosis factor-α and lymphotoxin in mononuclear cells in multiple sclerosis and optic neuritis
inferredcites →https://doi.org/10.1093/brain/awl075openalex-snapshot
Understanding pathogenesis and therapy of multiple sclerosis via animal models: 70 years of merits and culprits in experimental autoimmune encephalomyelitis research
inferredcites →https://doi.org/10.1093/brain/awr199openalex-snapshot
Inhibition of soluble tumour necrosis factor is therapeutic in experimental autoimmune encephalomyelitis and promotes axon preservation and remyelination
inferredcites →https://doi.org/10.1093/brain/awr203openalex-snapshot
Transmembrane tumour necrosis factor is neuroprotective and regulates experimental autoimmune encephalomyelitis via neuronal nuclear factor-κB
inferredcites →https://doi.org/10.1093/cercor/bhg108openalex-snapshot
Involvement of Tumor Necrosis Factor Alpha in Hippocampal Development and Function
inferredcites →https://doi.org/10.1093/intimm/2.6.539openalex-snapshot
Lymphotoxin and tumor necrosis factor-alpha production by myelin basic protein-specific T cell clones correlates with encephalitogenicity
inferredcites →https://doi.org/10.1093/intimm/8.7.1001openalex-snapshot
The level of tumor necrosis factor-α producing cells in the spinal cord correlates with the degree of Theiler's murine encephalomyelitis virus-induced demyelinating disease
inferredcites →https://doi.org/10.1093/intimm/dxu102openalex-snapshot
Anti-TNF therapy: past, present and future
inferredcites →https://doi.org/10.1097/wco.0000000000000622openalex-snapshot
Multiple sclerosis: clinical aspects
inferredcites →https://doi.org/10.1111/cei.12209openalex-snapshot
Blockade of tumour necrosis factor-α in experimental autoimmune encephalomyelitis reveals differential effects on the antigen-specific immune response and central nervous system histopathology
inferredcites →https://doi.org/10.1111/j.1365-2249.2011.04484.xopenalex-snapshot
Role of tumour necrosis factor (TNF)-α and TNFRSF1A R92Q mutation in the pathogenesis of TNF receptor-associated periodic syndrome and multiple sclerosis
inferredcites →https://doi.org/10.1111/j.1476-5381.2011.01302.xopenalex-snapshot
Experimental autoimmune encephalomyelitis (EAE) as a model for multiple sclerosis (MS)
inferredcites →https://doi.org/10.1111/j.1600-065x.1999.tb01315.xopenalex-snapshot
On the role of tumor necrosis factor and receptors in models of multiorgan failure, rheumatoid arthritis, multiple sclerosis and inflammatory bowel disease
inferredcites →https://doi.org/10.1111/nan.12569openalex-snapshot
Peripheral T cells from multiple sclerosis patients trigger synaptotoxic alterations in central neurons
inferredcites →https://doi.org/10.1126/science.1067859openalex-snapshot
Control of Synaptic Strength by Glial TNFα
inferredcites →https://doi.org/10.1126/scitranslmed.aaa9223openalex-snapshot
Genetic variants associated with autoimmunity drive NFκB signaling and responses to inflammatory stimuli
inferredcites →https://doi.org/10.1128/jvi.65.12.6612-6620.1991openalex-snapshot
Cytotoxic T cells isolated from the central nervous systems of mice infected with Theiler's virus
inferredcites →https://doi.org/10.1128/microbiolspec.mchd-0022-2015openalex-snapshot
Anti-TNF Therapy
inferredcites →https://doi.org/10.1152/physrev.00045.2017openalex-snapshot
The TNF Family of Ligands and Receptors: Communication Modules in the Immune System and Beyond
inferredcites →https://doi.org/10.1155/2014/861231openalex-snapshot
Tumor Necrosis Factor Alpha: A Link between Neuroinflammation and Excitotoxicity
inferredcites →https://doi.org/10.1172/jci.insight.132527openalex-snapshot
TNFR2 limits proinflammatory astrocyte functions during EAE induced by pathogenic DR2b-restricted T cells
inferredcites →https://doi.org/10.1172/jci.insight.87455openalex-snapshot
Therapeutic inhibition of soluble brain TNF promotes remyelination by increasing myelin phagocytosis by microglia
inferredcites →https://doi.org/10.1172/jci115102openalex-snapshot
Identification of lymphotoxin and tumor necrosis factor in multiple sclerosis lesions.
inferredcites →https://doi.org/10.1172/jci42945openalex-snapshot
Pathogenic T cells have a paradoxical protective effect in murine autoimmune diabetes by boosting Tregs
inferredcites →https://doi.org/10.1172/jci66168openalex-snapshot
iRHOM2 is a critical pathogenic mediator of inflammatory arthritis
inferredcites →https://doi.org/10.1177/135245850100700302openalex-snapshot
Intracellular cytokine profile in T-cell subsets of multiple sclerosis patients: different features in primary progressive disease
inferredcites →https://doi.org/10.1177/135245850100700303openalex-snapshot
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inferredcites →https://doi.org/10.1177/1352458508096871openalex-snapshot
Increase of CD4+TNFα+IL-2−T cells in cerebrospinal fluid of multiple sclerosis patients
inferredcites →https://doi.org/10.1177/1352458510379612openalex-snapshot
Improving the translational hit of experimental treatments in multiple sclerosis
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PPMS onset upon adalimumab treatment extends the spectrum of anti-TNF-α therapy-associated demyelinating disorders
inferredcites →https://doi.org/10.1182/blood-2005-11-4567openalex-snapshot
TNF downmodulates the function of human CD4+CD25hi T-regulatory cells
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Meningeal inflammation changes the balance of TNF signalling in cortical grey matter in multiple sclerosis
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TNFRSF1A polymorphisms rs1800693 and rs4149584 in patients with multiple sclerosis
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Onset of multiple sclerosis associated with anti-TNF therapy
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Cytokine Mechanisms of Central Sensitization: Distinct and Overlapping Role of Interleukin-1β, Interleukin-6, and Tumor Necrosis Factor-α in Regulating Synaptic and Neuronal Activity in the Superficial Spinal Cord
inferredcites →https://doi.org/10.1523/jneurosci.5804-08.2009openalex-snapshot
Inflammation Triggers Synaptic Alteration and Degeneration in Experimental Autoimmune Encephalomyelitis
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The cerebrospinal fluid in multiple sclerosis: far beyond the bands
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Pathogenic and Protective Functions of TNF in Neuroinflammation Are Defined by Its Expression in T Lymphocytes and Myeloid Cells
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TNFR2 Is Critical for the Stabilization of the CD4+Foxp3+ Regulatory T Cell Phenotype in the Inflammatory Environment
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Effector T Cells Boost Regulatory T Cell Expansion by IL-2, TNF, OX40, and Plasmacytoid Dendritic Cells Depending on the Immune Context
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Tumor necrosis factor (TNF) receptor expression in T lymphocytes. Differential regulation of the type I TNF receptor during activation of resting and effector T cells
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Stimulation of human T-cell proliferation by specific activation of the 75-kDa tumor necrosis factor receptor.
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Differential Expression of Inflammatory Cytokines Parallels Progression of Central Nervous System Pathology in Two Clinically Distinct Models of Multiple Sclerosis
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inferredcites →https://doi.org/10.4049/jimmunol.179.1.154openalex-snapshot
Interaction of TNF with TNF Receptor Type 2 Promotes Expansion and Function of Mouse CD4+CD25+ T Regulatory Cells
inferredcites →https://doi.org/10.4161/21688370.2014.978720openalex-snapshot
Blood cells and endothelial barrier function
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inferred← citeshttps://doi.org/10.3390/cells14221749openalex-snapshot
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inferred← citeshttps://doi.org/10.3390/ijms231810262openalex-snapshot
Cytokine Secretion Dynamics of Isolated PBMC after Cladribine Exposure in RRMS Patients
inferred← citeshttps://doi.org/10.3390/ijms24076717openalex-snapshot
1,25(OH)2D3 Differently Modulates the Secretory Activity of IFN-DC and IL4-DC: A Study in Cells from Healthy Donors and MS Patients
inferred← citeshttps://doi.org/10.3390/ijms25179588openalex-snapshot
Glial Cells as Key Regulators in Neuroinflammatory Mechanisms Associated with Multiple Sclerosis
inferred← citeshttps://doi.org/10.3390/ijms252111499openalex-snapshot
Dairy: Friend or Foe? Bovine Milk-Derived Extracellular Vesicles and Autoimmune Diseases
inferred← citeshttps://doi.org/10.3390/ijms27020884openalex-snapshot
Glial Cells as Key Mediators in the Pathophysiology of Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.3390/jcm12103545openalex-snapshot
Brodalumab for the Treatment of Moderate-to-Severe Psoriasis: An Expert Delphi Consensus Statement
inferred← citeshttps://doi.org/10.3390/jcm13133693openalex-snapshot
TNF-alfa Gene Polymorphism Associations with Multiple Sclerosis
inferred← citeshttps://doi.org/10.3390/life11070620openalex-snapshot
Comparison of the Intestinal Microbiome of Italian Patients with Multiple Sclerosis and Their Household Relatives
inferred← citeshttps://doi.org/10.3390/nu16172996openalex-snapshot
Potential Application of Plant-Derived Compounds in Multiple Sclerosis Management
inferred← citeshttps://doi.org/10.3390/nu16183100openalex-snapshot
Impact of Lifestyle Interventions on Multiple Sclerosis: Focus on Adipose Tissue
inferred← citeshttps://doi.org/10.3390/ph18101478openalex-snapshot
Curcumin–Piperine Self-Nanoemulsifying Delivery in Zanthoxylum rhetsa Seed Oil Attenuates Cuprizone-Induced Frontal Cortex Toxicity
inferred← citeshttps://doi.org/10.3390/ph19071004openalex-snapshot
Therapeutic Effects of Combined 6-Shogaol and Ibudilast on Neuroinflammation and Behavioral Deficits in a Cuprizone Mouse Model of Multiple Sclerosis
inferred← citeshttps://doi.org/10.4081/btvb.2023.66openalex-snapshot
Tumor necrosis factor superfamily in multiple sclerosis: from pathology to therapeutic implications
inferred← citeshttps://doi.org/10.4103/nrr.nrr-d-25-00825openalex-snapshot
ALDH2 deficiency exacerbates acrolein-mediated neuropathology in multiple sclerosis
inferred← citeshttps://doi.org/10.5114/aoms/201446openalex-snapshot
The Importance of TNF-α Signaling – Potential Risk Factor in Neurodegenerative and Cardiovascular Diseases
inferred← citeshttps://doi.org/10.52711/0974-360x.2023.00248openalex-snapshot
Are 5HT7 Receptors Possible Target for Multiple Sclerosis?
inferred← citeshttps://doi.org/10.5812/semj-138014openalex-snapshot
Effects of Levothyroxine on Behavior and Cognitive Decline in a Rat Model of Multiple Sclerosis: A Biochemical Study
inferred← citeshttps://doi.org/10.6084/m9.figshare.17696298openalex-snapshot
Additional file 1 of Citrullinated myelin induces microglial TNFα and inhibits endogenous repair in the cuprizone model of demyelination
inferred← citeshttps://doi.org/10.6084/m9.figshare.21366140openalex-snapshot
Additional file 1 of ISGylation is induced in neurons by demyelination driving ISG15-dependent microglial activation
inferred← citeshttps://doi.org/10.64898/2026.01.14.26343929openalex-snapshot
Exploring Sleep, Inflammation and Myocardial Infarction Recurrence in Aging
inferred← citeshttps://doi.org/10.70675/defcbd86z76d7z43bfz9103zb8a743a479f5openalex-snapshot
Analyse protéomique et interactomique de l'activation des lymphocytes T humains primaires
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