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Persistent Infection with Herpes Simplex Virus 1 and Alzheimer’s Disease—A Call to Study How Variability in Both Virus and Host may Impact Disease
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Association of Serum Antibodies to Herpes Simplex Virus 1 With Cognitive Deficits in Individuals With Schizophrenia
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Correlation Between Elevated Levels of Amyloid β-Peptide in the Brain and Cognitive Decline
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The herpes simplex virus type 1 Fc receptor discriminates between IgG1 allotypes
inferredcites →https://doi.org/10.1002/1522-2683(200102)22:3<445::aid-elps445>3.0.co;2-8openalex-snapshot
Evidence for the relation of herpes simplex virus type 1 to Down syndrome and Alzheimer's disease
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Herpesviruses in brains in Alzheimer's and Parkinson's diseases
inferredcites →https://doi.org/10.1002/dneu.22046openalex-snapshot
The major histocompatibility complex and autism spectrum disorder
inferredcites →https://doi.org/10.1002/jmv.1890250109openalex-snapshot
History of recurrent mucocutaneous herpes correlates with relatively low interferon production by herpes simplex virus‐exposed cultured monocytes
inferredcites →https://doi.org/10.1002/jmv.1890330403openalex-snapshot
Latent herpes simplex virus type 1 in normal and Alzheimer's disease brains
inferredcites →https://doi.org/10.1002/jmv.20271openalex-snapshot
Productive herpes simplex virus in brain of elderly normal subjects and Alzheimer's disease patients
inferredcites →https://doi.org/10.1002/jnr.23003openalex-snapshot
Herpes simplex virus type 1 induces nuclear accumulation of hyperphosphorylated tau in neuronal cells
inferredcites →https://doi.org/10.1002/path.1711670403openalex-snapshot
Herpes simplex virus type 1 DNA is present in specific regions of brain from aged people with and without senile dementia of the Alzheimer type
inferredcites →https://doi.org/10.1002/path.2449openalex-snapshot
Herpes simplex virus type 1 DNA is located within Alzheimer's disease amyloid plaques
inferredcites →https://doi.org/10.1002/stem.3072openalex-snapshot
Herpes Simplex Virus Type-1 Infection Impairs Adult Hippocampal Neurogenesis via Amyloid-β Protein Accumulation
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The Amyloid Precursor Protein Is Developmentally Regulated and Correlated with Synaptogenesis
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Correlation between Herpes Simplex Virus Type 1 Rate of Reactivation from Latent Infection and the Number of Infected Neurons in Trigeminal Ganglia
inferredcites →https://doi.org/10.1007/bf00293315openalex-snapshot
A sequence of cytoskeleton changes related to the formation of neurofibrillary tangles and neuropil threads
inferredcites →https://doi.org/10.1007/s10522-014-9538-8openalex-snapshot
β-Amyloid peptides display protective activity against the human Alzheimer’s disease-associated herpes simplex virus-1
inferredcites →https://doi.org/10.1007/s11481-010-9212-0openalex-snapshot
A Common Neuronal Response to Alphaherpesvirus Infection
inferredcites →https://doi.org/10.1007/s12031-012-9783-8openalex-snapshot
Neuroglial Expression of the MHCI Pathway and PirB Receptor Is Upregulated in the Hippocampus with Advanced Aging
inferredcites →https://doi.org/10.1007/s12035-012-8320-7openalex-snapshot
Infectious Agents and Neurodegeneration
inferredcites →https://doi.org/10.1007/s13311-018-0611-xopenalex-snapshot
Anti-herpetic Medications and Reduced Risk of Dementia in Patients with Herpes Simplex Virus Infections—a Nationwide, Population-Based Cohort Study in Taiwan
inferredcites →https://doi.org/10.1007/s13365-011-0071-0openalex-snapshot
The herpes simplex virus type 1 latency associated transcript locus is required for the maintenance of reactivation competent latent infections
inferredcites →https://doi.org/10.1016/0014-4800(84)90050-9openalex-snapshot
Herpesviridae in the endothelial and smooth muscle cells of the proximal aorta in arteriosclerotic patients
inferredcites →https://doi.org/10.1016/0165-5728(95)00068-dopenalex-snapshot
Immune cell infiltration and persistence in the mouse trigeminal ganglion after infection of the cornea with herpes simplex virus type 1
inferredcites →https://doi.org/10.1016/0278-5846(95)00195-6openalex-snapshot
Cortical mapping of Alzheimer pathology in brains of aged non-demented subjects
inferredcites →https://doi.org/10.1016/0304-3940(92)90736-qopenalex-snapshot
Presence of abnormally phosphorylated Tau proteins in the entorhinal cortex of aged non-demented subjects
inferredcites →https://doi.org/10.1016/0304-3940(93)90740-copenalex-snapshot
Distribution of herpes simplex virus DNA in the brains of human long-term survivors of encephalitis
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Synaptic pathology in alzheimer's disease: Immunological data for markers of synaptic and large dense-core vesicles
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Glutamate neurotoxicity and diseases of the nervous system
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Revisiting the cholinergic hypothesis of behavioral and psychological symptoms in dementia of the Alzheimer's type
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Immune modulation of learning, memory, neural plasticity and neurogenesis
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Major Histocompatibility Complex I in Brain Development and Schizophrenia
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Immune Escape via a Transient Gene Expression Program Enables Productive Replication of a Latent Pathogen
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HSV-1 ICP34.5 Confers Neurovirulence by Targeting the Beclin 1 Autophagy Protein
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Chronic herpesvirus reactivation occurs in aging
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Immunity to latent viral infection: many skirmishes but few fatalities
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Intracerebral propagation of Alzheimer's disease: Strengthening evidence of a herpes simplex virus etiology
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Reactivated herpes simplex infection increases the risk of Alzheimer's disease
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Herpes simplex infection and the risk of Alzheimer's disease: A nested case-control study
inferredcites →https://doi.org/10.1016/j.jalz.2018.06.3040openalex-snapshot
The antimicrobial protection hypothesis of Alzheimer's disease
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Blood-brain barrier alterations in ageing and dementia
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Comparison of Aβ levels in the brain of familial and sporadic Alzheimer's disease
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Herpes simplex virus infection causes cellular β-amyloid accumulation and secretase upregulation
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2003.04.007openalex-snapshot
Clogging of axons by tau, inhibition of axonal traffic and starvation of synapses
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2010.12.010openalex-snapshot
Herpes simplex virus type I induces the accumulation of intracellular β-amyloid in autophagic compartments and the inhibition of the non-amyloidogenic pathway in human neuroblastoma cells
inferredcites →https://doi.org/10.1016/j.neuron.2009.08.039openalex-snapshot
Immune Activation in Brain Aging and Neurodegeneration: Too Much or Too Little?
inferredcites →https://doi.org/10.1016/j.neuron.2009.09.001openalex-snapshot
Immune Proteins in Brain Development and Synaptic Plasticity
inferredcites →https://doi.org/10.1016/j.neuron.2009.09.044openalex-snapshot
MHC Class I: An Unexpected Role in Neuronal Plasticity
inferredcites →https://doi.org/10.1016/j.neuron.2018.05.023openalex-snapshot
Multiscale Analysis of Independent Alzheimer’s Cohorts Finds Disruption of Molecular, Genetic, and Clinical Networks by Human Herpesvirus
inferredcites →https://doi.org/10.1016/j.neuron.2018.06.030openalex-snapshot
Alzheimer’s Disease-Associated β-Amyloid Is Rapidly Seeded by Herpesviridae to Protect against Brain Infection
inferredcites →https://doi.org/10.1016/j.pharmthera.2018.11.006openalex-snapshot
Synaptic dysfunction in Alzheimer's disease: Mechanisms and therapeutic strategies
inferredcites →https://doi.org/10.1016/j.tim.2012.08.005openalex-snapshot
A cultured affair: HSV latency and reactivation in neurons
inferredcites →https://doi.org/10.1016/j.virol.2012.08.043openalex-snapshot
Sequence and comparative analysis of the genome of HSV-1 strain McKrae
inferredcites →https://doi.org/10.1016/j.virol.2016.02.013openalex-snapshot
Viral forensic genomics reveals the relatedness of classic herpes simplex virus strains KOS, KOS63, and KOS79
inferredcites →https://doi.org/10.1016/j.virusres.2013.01.001openalex-snapshot
HSV-1 strain McKrae is more neuroinvasive than HSV-1 KOS after corneal or vaginal inoculation in mice
inferredcites →https://doi.org/10.1016/s0042-6822(95)80080-8openalex-snapshot
In situ DNA PCR and RNA hybridization detection of herpes simplex virus sequences in trigeminal gangliaof latently infected mice
inferredcites →https://doi.org/10.1016/s0140-6736(96)10149-5openalex-snapshot
Herpes simplex virus type 1 in brain and risk of Alzheimer's disease
inferredcites →https://doi.org/10.1016/s0165-0173(00)00019-9openalex-snapshot
Tau protein isoforms, phosphorylation and role in neurodegenerative disorders11These authors contributed equally to this work.
inferredcites →https://doi.org/10.1016/s0197-4580(96)00170-4openalex-snapshot
β-amyloid deposition and other measures of neuropathology predict cognitive status in Alzheimer's disease
inferredcites →https://doi.org/10.1016/s1074-7613(03)00112-2openalex-snapshot
Herpes Simplex Virus-Specific Memory CD8+ T Cells Are Selectively Activated and Retained in Latently Infected Sensory Ganglia
inferredcites →https://doi.org/10.1016/s1357-4310(98)01229-5openalex-snapshot
Molecular pathogenesis of sporadic and familial forms of Alzheimer's disease
inferredcites →https://doi.org/10.1016/s1474-4422(15)70016-5openalex-snapshot
Neuroinflammation in Alzheimer's disease
inferredcites →https://doi.org/10.1016/s1474-4422(18)30403-4openalex-snapshot
Global, regional, and national burden of Alzheimer's disease and other dementias, 1990–2016: a systematic analysis for the Global Burden of Disease Study 2016
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First estimates of the global and regional incidence of neonatal herpes infection
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Virology and Histopathology of the Trigeminal Ganglia of Americans and Japanese
inferredcites →https://doi.org/10.1017/s0317167100044115openalex-snapshot
“Limbic Predilection in Alzheimer Dementia: Is Reactivated Herpesvirus Involved?”
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Familial Alzheimer's disease in kindreds with missense mutations in a gene on chromosome 1 related to the Alzheimer's disease type 3 gene
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Cold sore susceptibility gene-1 genotypes affect the expression of herpes labialis in unrelated human subjects
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MicroRNAs expressed by herpes simplex virus 1 during latent infection regulate viral mRNAs
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Origin and physiological roles of inflammation
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Chromatin control of herpes simplex virus lytic and latent infection
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Genome-wide association study (GWAS) of human host factors influencing viral severity of herpes simplex virus type 2 (HSV-2)
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The viral protein corona directs viral pathogenesis and amyloid aggregation
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Inferred father-to-son transmission of herpes simplex virus results in near-perfect preservation of viral genome identity and in vivo phenotypes
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Herpes Simplex Virus type-1 infection induces synaptic dysfunction in cultured cortical neurons via GSK-3 activation and intraneuronal amyloid-β protein accumulation
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Detection of herpes simplex virus type 1 DNA sequences in normal and Alzheimer's disease brain using polymerase chain reaction
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APP is required during an early phase of memory formation
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Infections with Herpes Simplex Viruses
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Infections with Herpes Simplex Viruses
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Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice
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Viruses in the etiology of atherosclerosis.
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ICP34.5 Protein of Herpes Simplex Virus Facilitates the Initiation of Protein Translation by Bridging Eukaryotic Initiation Factor 2α (eIF2α) and Protein Phosphatase 1
inferredcites →https://doi.org/10.1074/jbc.m111553200openalex-snapshot
An N-terminal Arginine-rich Cluster and a Proline-Alanine-Threonine Repeat Region Determine the Cellular Localization of the Herpes Simplex Virus Type 1 ICP34.5 Protein and Its Ligand, Protein Phosphatase 1
inferredcites →https://doi.org/10.1080/135502801300069773openalex-snapshot
Lack of association between HSV-1 DNA in the brain, Alzheimer's disease and apolipoprotein E4
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CONDITIONS DETERMINING THE TRANSPLANTABILITY OF TISSUES IN THE BRAIN
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The impact of different presenilin 1 andpresenilin 2 mutations on amyloid deposition, neurofibrillary changes and neuronal loss in the familial Alzheimer's disease brain
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Regional Distribution of Neurofibrillary Tangles and Senile Plaques in the Cerebral Cortex of Elderly Patients: A Quantitative Evaluation of a One-Year Autopsy Population from a Geriatric Hospital
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Differential Effects of Aging and Insulin-like Growth Factor-1 on Synapses in CA1 of Rat Hippocampus
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CNS-wide Sexually Dimorphic Induction of the Major Histocompatibility Complex 1 Pathway With Aging
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Estimation of the Genetic Contribution of Presenilin-1 and -2 Mutations in a Population-Based Study of Presenile Alzheimer Disease
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C21orf91 Genotypes Correlate With Herpes Simplex Labialis (Cold Sore) Frequency: Description of a Cold Sore Susceptibility Gene
inferredcites →https://doi.org/10.1093/infdis/jis317openalex-snapshot
Immunoglobulin Genes and Immunity to Herpes Simplex Virus Type 1
inferredcites →https://doi.org/10.1093/infdis/jiy631openalex-snapshot
Viral Genetics Modulate Orolabial Herpes Simplex Virus Type 1 Shedding in Humans
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Altered Proteins in the Aging Brain
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Exposure to Herpes Simplex Virus Type 1 and Cognitive Impairments in Individuals With Schizophrenia
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ApoE4 is more efficient than E3 in brain access by herpes simplex virus type 1
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Aging-associated Changes in Human Brain
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Unique Aspects of Central Nervous System Immunology
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Alzheimer Disease Pathology As a Host Response
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Correlation of Alzheimer Disease Neuropathologic Changes With Cognitive Status: A Review of the Literature
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Cytokine production in the nervous system of mice during acute and latent infection with herpes simplex virus type 1.
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Herpes simplex virus type 1 mutants containing the KOS strain ICP34.5 gene in place of the McKrae ICP34.5 gene have McKrae-like spontaneous reactivation but non-McKrae-like virulence
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Genotyping of herpes simplex virus type 1 by whole-genome sequencing
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Inflammation in Alzheimer Disease--A Brief Review of the Basic Science and Clinical Literature
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A study on the association between infectious burden and A lzheimer's disease
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The aging of the immune system
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AGE‐RELATED SYNAPTIC CHANGES IN THE CA1 STRATUM RADIATUM AND SPATIAL LEARNING IMPAIRMENT IN RATS
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Interleukin‐6: A Cytokine for Gerontolgists
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The molecular basis of herpes simplex virus latency
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Inflamm‐aging: An Evolutionary Perspective on Immunosenescence
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Alzheimer's Disease Is a Synaptic Failure
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Viral Persistence in Neurons Explained by Lack of Major Histocompatibility Class I Expression
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RNA Complementary to a Herpesvirus α Gene mRNA Is Prominent in Latently Infected Neurons
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Human apoE Isoforms Differentially Regulate Brain Amyloid-β Peptide Clearance
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Experimental investigation of herpes simplex virus latency
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Detection of an immediate early herpes simplex virus type 1 polypeptide in trigeminal ganglia from latently infected animals
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Comparative Neurovirulence of Herpes Simplex Virus Type 1 Strains After Peripheral or Intracerebral Inoculation of BALB/c Mice
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Effect of Apolipoprotein E on the Cerebral Load of Latent Herpes Simplex Virus Type 1 DNA
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Sequence Variability in Clinical and Laboratory Isolates of Herpes Simplex Virus 1 Reveals New Mutations
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High-Throughput Analysis of Human Cytomegalovirus Genome Diversity Highlights the Widespread Occurrence of Gene-Disrupting Mutations and Pervasive Recombination
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Two Small RNAs Encoded within the First 1.5 Kilobases of the Herpes Simplex Virus Type 1 Latency-Associated Transcript Can Inhibit Productive Infection and Cooperate To Inhibit Apoptosis
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Molecular Evolution of Herpes Simplex Virus 2 Complete Genomes: Comparison between Primary and Recurrent Infections
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Restarting Lytic Gene Transcription at the Onset of Herpes Simplex Virus Reactivation
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Comparison of Herpes Simplex Virus 1 Strains Circulating in Finland Demonstrates the Uncoupling of Whole-Genome Relatedness and Phenotypic Outcomes of Viral Infection
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Evolution and Diversity in Human Herpes Simplex Virus Genomes
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During latency, herpes simplex virus type 1 DNA is associated with nucleosomes in a chromatin structure
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Herpes simplex virus type 1 encodes two Fc receptors which have different binding characteristics for monomeric immunoglobulin G (IgG) and IgG complexes
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Herpes simplex virus type 1 Fc receptor protects infected cells from antibody-dependent cellular cytotoxicity
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ICP34.5 mutants of herpes simplex virus type 1 strain 17syn+ are attenuated for neurovirulence in mice and for replication in confluent primary mouse embryo cell cultures
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The latency-associated transcript gene of herpes simplex virus type 1 (HSV-1) is required for efficient in vivo spontaneous reactivation of HSV-1 from latency
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Quantification of transcripts from the ICP4 and thymidine kinase genes in mouse ganglia latently infected with herpes simplex virus
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An avirulent ICP34.5 deletion mutant of herpes simplex virus type 1 is capable of in vivo spontaneous reactivation
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The herpes simplex virus type 1 2.0-kilobase latency-associated transcript is a stable intron which branches at a guanosine
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The herpes simplex virus type 1 latency-associated transcript gene regulates the establishment of latency
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A viral function represses accumulation of transcripts from productive-cycle genes in mouse ganglia latently infected with herpes simplex virus
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Herpes Simplex Virus Type 1 Latency-Associated Transcripts Suppress Viral Replication and Reduce Immediate-Early Gene mRNA Levels in a Neuronal Cell Line
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Intrastrain Variants of Herpes Simplex Virus Type 1 Isolated from a Neonate with Fatal Disseminated Infection Differ in the ICP34.5 Gene, Glycoprotein Processing, and Neuroinvasiveness
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Herpes Simplex Virus Type 1 Latency-Associated Transcript Gene Promotes Neuronal Survival
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Strain-Dependent Structural Variants of Herpes Simplex Virus Type 1 ICP34.5 Determine Viral Plaque Size, Efficiency of Glycoprotein Processing, and Viral Release and Neuroinvasive Disease Potential
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Laser-Capture Microdissection: Refining Estimates of the Quantity and Distribution of Latent Herpes Simplex Virus 1 and Varicella-Zoster Virus DNA in Human Trigeminal Ganglia at the Single-Cell Level
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Rapid Genome Assembly and Comparison Decode Intrastrain Variation in Human Alphaherpesviruses
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Genotypic and Phenotypic Diversity of Herpes Simplex Virus 2 within the Infected Neonatal Population
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Detection of HSV1 DNA by in situ hybridisation in human brain after immunosuppression.
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The Complement System: An Unexpected Role in Synaptic Pruning During Development and Disease
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