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Minimally Invasive Transverse Aortic Constriction in Mice
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/978-1-60761-247-6_7openalex-snapshot
Mouse Surgical Models in Cardiovascular Research
inferredcites →https://doi.org/10.1016/j.echo.2008.07.001openalex-snapshot
Echocardiography in Translational Research: Of Mice and Men
inferredcites →https://doi.org/10.1016/j.jtcvs.2010.03.029openalex-snapshot
Induction of heart failure by minimally invasive aortic constriction in mice: Reduced peroxisome proliferator-activated receptor γ coactivator levels and mitochondrial dysfunction
inferredcites →https://doi.org/10.1016/j.ultrasmedbio.2007.11.019openalex-snapshot
Doppler Estimation of Reduced Coronary Flow Reserve in Mice with Pressure Overload Cardiac Hypertrophy
inferredcites →https://doi.org/10.1073/pnas.88.18.8277openalex-snapshot
RETRACTED: Segregation of atrial-specific and inducible expression of an atrial natriuretic factor transgene in an in vivo murine model of cardiac hypertrophy.
inferredcites →https://doi.org/10.1152/ajpheart.00108.2003openalex-snapshot
Minimally invasive aortic banding in mice: effects of altered cardiomyocyte insulin signaling during pressure overload
inferredcites →https://doi.org/10.1172/jci40096openalex-snapshot
Excessive cardiac insulin signaling exacerbates systolic dysfunction induced by pressure overload in rodents
inferredcites →https://doi.org/10.3791/1729-vopenalex-snapshot
Transverse Aortic Constriction in Mice
inferredcites →https://doi.org/10.3791/54101openalex-snapshot
Assessment of Cardiac Morphological and Functional Changes in Mouse Model of Transverse Aortic Constriction by Echocardiographic Imaging
inferredcites →https://doi.org/10.3791/54101-vopenalex-snapshot
Assessment of Cardiac Morphological and Functional Changes in Mouse Model of Transverse Aortic Constriction by Echocardiographic Imaging
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4766307openalex-extracted
Sweroside Alleviates Pressure Overload-Induced Heart Failure Through Targeting Camkiiδ to Inhibit Ros-Mediated Nf-Κb/Nlrp3 in Cardiomyocytes
inferred← citeshttps://doi.org/10.1002/nbm.4641openalex-snapshot
Quantification of myocardial fibrosis using noninvasive T2‐mapping magnetic resonance imaging: Preclinical models of aging and pressure overload
inferred← citeshttps://doi.org/10.1007/978-1-4939-8597-5_14openalex-snapshot
Conventional Method of Transverse Aortic Constriction in Mice
inferred← citeshttps://doi.org/10.1007/s12265-021-10170-4openalex-snapshot
A New Minimally Invasive Method of Transverse Aortic Constriction in Mice
inferred← citeshttps://doi.org/10.1007/s12265-023-10376-8openalex-snapshot
RyR2 Stabilizer Attenuates Cardiac Hypertrophy by Downregulating TNF-α/NF-κB/NLRP3 Signaling Pathway through Inhibiting Calcineurin
inferred← citeshttps://doi.org/10.1016/j.bbadis.2024.167650openalex-snapshot
Serinc2 antagonizes pressure overload-induced cardiac hypertrophy via regulating the amino acid/mTORC1 signaling pathway
inferred← citeshttps://doi.org/10.1016/j.bbrc.2020.01.062openalex-snapshot
Fendrr involves in the pathogenesis of cardiac fibrosis via regulating miR-106b/SMAD3 axis
inferred← citeshttps://doi.org/10.1016/j.biopha.2021.111885openalex-snapshot
Hirsutella sinensis fungus improves cardiac function in mouse model of heart failure
inferred← citeshttps://doi.org/10.1016/j.lfs.2025.123873openalex-snapshot
Electrophysiological changes induced by G1, the selective agonist of the G-protein coupled estrogen receptor (GPER), in the clip-TAC mice model. A new potential cardioprotective mechanism
inferred← citeshttps://doi.org/10.1016/j.phymed.2025.157371openalex-snapshot
Baicalin and ginsenoside Rb1 suppress the activation of cardiac fibroblasts via regulating the GRK2/AT1R/MasR network
inferred← citeshttps://doi.org/10.1016/j.phymed.2025.157625openalex-snapshot
Chlorogenic acid targets STING to alleviate pressure overload-induced heart failure and myocardial fibrosis
inferred← citeshttps://doi.org/10.1016/j.redox.2024.103223openalex-snapshot
Sweroside alleviates pressure overload-induced heart failure through targeting CaMKⅡδ to inhibit ROS-mediated NF-κB/NLRP3 in cardiomyocytes
inferred← citeshttps://doi.org/10.1016/j.redox.2025.103745openalex-snapshot
USP10 protects against pressure overload-induced mitochondrial morphofunctional defects and pathological cardiac hypertrophy through stabilizing cytoplasmic Mfn2
inferred← citeshttps://doi.org/10.1016/j.xjon.2022.02.027openalex-snapshot
Cardio-omentopexy requires a cardioprotective innate immune response to promote myocardial angiogenesis in mice
inferred← citeshttps://doi.org/10.1016/j.ymthe.2024.08.030openalex-snapshot
Integrative proteomic and metabolomic elucidation of cardiomyopathy with in vivo and in vitro models and clinical samples
inferred← citeshttps://doi.org/10.1038/s41401-021-00743-zopenalex-snapshot
Cardiomyocyte peroxisome proliferator-activated receptor α is essential for energy metabolism and extracellular matrix homeostasis during pressure overload-induced cardiac remodeling.
inferred← citeshttps://doi.org/10.1038/s41420-022-00967-wopenalex-snapshot
ATP6AP2 knockdown in cardiomyocyte deteriorates heart function via compromising autophagic flux and NLRP3 inflammasome activation
inferred← citeshttps://doi.org/10.1038/s41467-022-31996-9openalex-snapshot
CDC-like kinase 4 deficiency contributes to pathological cardiac hypertrophy by modulating NEXN phosphorylation
inferred← citeshttps://doi.org/10.1073/pnas.1902165116openalex-snapshot
Endothelial cell Piezo1 mediates pressure-induced lung vascular hyperpermeability via disruption of adherens junctions
inferred← citeshttps://doi.org/10.1093/cvr/cvab234openalex-snapshot
Resistin deletion protects against heart failure injury by targeting DNA damage response
inferred← citeshttps://doi.org/10.1097/fjc.0000000000001301openalex-snapshot
Transient Receptor Potential Vanilloid Type 1 Protects Against Pressure Overload–Induced Cardiac Hypertrophy by Promoting Mitochondria-Associated Endoplasmic Reticulum Membranes
inferred← citeshttps://doi.org/10.1101/2024.07.07.602358openalex-snapshot
Unveiling reversibility and plasticity in cardiac hypertrophy: insights from a transverse aortic constriction-release model
inferred← citeshttps://doi.org/10.1111/1440-1681.13672openalex-snapshot
Angiotensin receptor‐neprilysin inhibitor attenuates cardiac hypertrophy and improves diastolic dysfunction in a mouse model of heart failure with preserved ejection fraction
inferred← citeshttps://doi.org/10.1155/2021/1257283openalex-snapshot
Ivabradine Ameliorates Cardiac Function in Heart Failure with Preserved and Reduced Ejection Fraction via Upregulation of miR‐133a
inferred← citeshttps://doi.org/10.1161/circulationaha.124.072661openalex-snapshot
FMO2 Prevents Pathological Cardiac Hypertrophy by Maintaining the ER-Mitochondria Association Through Interaction With IP3R2-Grp75-VDAC1
inferred← citeshttps://doi.org/10.18632/aging.204901openalex-snapshot
Identification of TGF-β-related genes in cardiac hypertrophy and heart failure based on single cell RNA sequencing
inferred← citeshttps://doi.org/10.20517/jca.2023.18openalex-snapshot
Characterization of atrial and ventricular remodeling in an improved minimally invasive mouse model of transverse aortic constriction
inferred← citeshttps://doi.org/10.3389/fcvm.2020.581362openalex-snapshot
Identification of Transcriptional Variation in Aortic Remodeling Using a Murine Transverse Aortic Constriction (TAC) Model
inferred← citeshttps://doi.org/10.3389/fcvm.2021.641272openalex-snapshot
Distinct Phenotypes Induced by Different Degrees of Transverse Aortic Constriction in C57BL/6N Mice
inferred← citeshttps://doi.org/10.3389/fcvm.2023.1110032openalex-snapshot
A time-series minimally invasive transverse aortic constriction mouse model for pressure overload-induced cardiac remodeling and heart failure
inferred← citeshttps://doi.org/10.3389/fcvm.2023.953514openalex-snapshot
Murine model of elastase-induced proximal thoracic aortic aneurysm through a midline incision in the anterior neck
inferred← citeshttps://doi.org/10.3389/fcvm.2025.1603082openalex-snapshot
First-phase ejection fraction is associated with myocardial fibrosis in the pressure overloaded heart
inferred← citeshttps://doi.org/10.3389/fphar.2022.858118openalex-snapshot
Recombinant EGFL7 Mitigated Pressure Overload-Induced Cardiac Remodeling by Blocking PI3Kγ/AKT/NFκB Signaling in Macrophages
inferred← citeshttps://doi.org/10.3389/fphys.2021.696286openalex-snapshot
Whole-Mount Kidney Clearing and Visualization Reveal Renal Sympathetic Hyperinnervation in Heart Failure Mice
inferred← citeshttps://doi.org/10.3389/fphys.2022.1026884openalex-snapshot
Evaluation of the transverse aortic constriction model in ICR and C57BL/6J mice
inferred← citeshttps://doi.org/10.3389/fphys.2022.777284openalex-snapshot
Differential mRNA Expression and Circular RNA-Based Competitive Endogenous RNA Networks in the Three Stages of Heart Failure in Transverse Aortic Constriction Mice
inferred← citeshttps://doi.org/10.35537/10915/156967openalex-snapshot
Rol fisiopatológico del receptor de estrógenos acoplado a proteína G (GPER) en la electrofisiología cardíaca
inferred← citeshttps://doi.org/10.3791/57397openalex-snapshot
A Closed-chest Model to Induce Transverse Aortic Constriction in Mice
inferred← citeshttps://doi.org/10.3892/ijmm.2021.5066openalex-snapshot
EGCG prevents pressure overload‑induced myocardial remodeling by downregulating overexpression of HDAC5 in mice
inferred← citeshttps://doi.org/10.4067/s0718-85602018000300194openalex-snapshot
En el modelo preclínico de insuficiencia cardíaca por disfunción sistólica en ratón (coartación de aorta torácica) la velocidad de flujo carotídeo determinada precozmente se asocia a función ventricular izquierda tardía
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