On the public citation graph
Mitochondrial Biogenesis Is Impaired in Osteoarthritis Chondrocytes but Reversible via Peroxisome Proliferator–Activated Receptor γ Coactivator 1α
0 signed · 344 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/art.10837openalex-snapshot
Mitochondrial respiratory activity is altered in osteoarthritic human articular chondrocytes
inferredcites →https://doi.org/10.1002/art.20149openalex-snapshot
Mediation of spontaneous knee osteoarthritis by progressive chondrocyte ATP depletion in Hartley guinea pigs
inferredcites →https://doi.org/10.1002/art.23799openalex-snapshot
The antioxidant resveratrol protects against chondrocyte apoptosis via effects on mitochondrial polarization and ATP production
inferredcites →https://doi.org/10.1002/art.27305openalex-snapshot
Autophagy is a protective mechanism in normal cartilage, and its aging‐related loss is linked with cell death and osteoarthritis
inferredcites →https://doi.org/10.1002/art.30279openalex-snapshot
Tumor necrosis factor α–mediated cleavage and inactivation of SirT1 in human osteoarthritic chondrocytes
inferredcites →https://doi.org/10.1002/art.30333openalex-snapshot
Chondrocyte AMP-activated protein kinase activity suppresses matrix degradation responses to proinflammatory cytokines interleukin-1β and tumor necrosis factor α
inferredcites →https://doi.org/10.1002/art.33407openalex-snapshot
75‐kd sirtuin 1 blocks tumor necrosis factor α–mediated apoptosis in human osteoarthritic chondrocytes
inferredcites →https://doi.org/10.1002/art.34508openalex-snapshot
Mitochondrial dysfunction increases inflammatory responsiveness to cytokines in normal human chondrocytes
inferredcites →https://doi.org/10.1002/art.37782openalex-snapshot
Mitochondrial dysfunction in osteoarthritis is associated with down‐regulation of superoxide dismutase 2
inferredcites →https://doi.org/10.1002/art.37977openalex-snapshot
Glucosamine Activates Autophagy In Vitro and In Vivo
inferredcites →https://doi.org/10.1002/art.38791openalex-snapshot
Peroxisome Proliferator–Activated Receptor γ Coactivator 1α and FoxO3A Mediate Chondroprotection by AMP‐Activated Protein Kinase
inferredcites →https://doi.org/10.1002/jor.22268openalex-snapshot
The overexpression of SIRT1 inhibited osteoarthritic gene expression changes induced by interleukin‐1β in human chondrocytes
inferredcites →https://doi.org/10.1007/s00467-009-1310-yopenalex-snapshot
Chondrocyte autophagy is stimulated by HIF-1 dependent AMPK activation and mTOR suppression
inferredcites →https://doi.org/10.1016/j.arr.2011.12.005openalex-snapshot
AMP-activated protein kinase (AMPK) controls the aging process via an integrated signaling network
inferredcites →https://doi.org/10.1016/j.bone.2012.03.023openalex-snapshot
Effects of aging on articular cartilage homeostasis
inferredcites →https://doi.org/10.1016/j.cellsig.2009.02.014openalex-snapshot
SIRT1: Regulation of longevity via autophagy
inferredcites →https://doi.org/10.1016/j.joca.2005.04.002openalex-snapshot
Oxygen and reactive oxygen species in cartilage degradation: friends or foes?
inferredcites →https://doi.org/10.1016/j.joca.2006.03.008openalex-snapshot
Mitochondrial activity is modulated by TNFα and IL-1β in normal human chondrocyte cells
inferredcites →https://doi.org/10.1016/j.joca.2008.05.009openalex-snapshot
Diminished mitochondrial DNA integrity and repair capacity in OA chondrocytes
inferredcites →https://doi.org/10.1016/j.joca.2012.01.003openalex-snapshot
Mitochondrial electron transport and glycolysis are coupled in articular cartilage
inferredcites →https://doi.org/10.1016/j.joca.2012.11.017openalex-snapshot
SIRT1, a class III histone deacetylase, regulates TNF-α-induced inflammation in human chondrocytes
inferredcites →https://doi.org/10.1016/j.mito.2004.07.022openalex-snapshot
Mitochondrial dysfunction in osteoarthritis
inferredcites →https://doi.org/10.1016/j.yjmcc.2014.10.003openalex-snapshot
A time to reap, a time to sow: Mitophagy and biogenesis in cardiac pathophysiology
inferredcites →https://doi.org/10.1016/s1063-4584(03)00150-xopenalex-snapshot
The role of reactive oxygen species in homeostasis and degradation of cartilage
inferredcites →https://doi.org/10.1016/s1567-7249(01)00037-xopenalex-snapshot
Invited review: the mitochondrion in osteoarthritis
inferredcites →https://doi.org/10.1038/nrd4023openalex-snapshot
Pharmacological approaches to restore mitochondrial function
inferredcites →https://doi.org/10.1038/nrrheum.2010.213openalex-snapshot
The role of mitochondria in osteoarthritis
inferredcites →https://doi.org/10.1038/nrrheum.2011.109openalex-snapshot
Autophagy and cartilage homeostasis mechanisms in joint health, aging and OA
inferredcites →https://doi.org/10.1053/joca.2002.0525openalex-snapshot
Subchondral bone of the human knee joint in aging and osteoarthritis
inferredcites →https://doi.org/10.1073/pnas.0712145105openalex-snapshot
A role for the NAD-dependent deacetylase Sirt1 in the regulation of autophagy
inferredcites →https://doi.org/10.1074/jbc.m110.102574openalex-snapshot
DYRK1A and DYRK3 Promote Cell Survival through Phosphorylation and Activation of SIRT1
inferredcites →https://doi.org/10.1074/jbc.m803196200openalex-snapshot
Regulation of Cartilage-specific Gene Expression in Human Chondrocytes by SirT1 and Nicotinamide Phosphoribosyltransferase
inferredcites →https://doi.org/10.1111/j.1749-6632.2012.06738.xopenalex-snapshot
Role of PGC‐1α signaling in skeletal muscle health and disease
inferredcites →https://doi.org/10.1136/annrheumdis-2011-200557openalex-snapshot
Autophagy activation by rapamycin reduces severity of experimental osteoarthritis
inferredcites →https://doi.org/10.1136/annrheumdis-2012-202620openalex-snapshot
Disruption of Sirt1 in chondrocytes causes accelerated progression of osteoarthritis under mechanical stress and during ageing in mice
inferredcites →https://doi.org/10.1136/ard.2004.022152openalex-snapshot
Effect of nitric oxide on mitochondrial respiratory activity of human articular chondrocytes
inferredcites →https://doi.org/10.1152/advan.00052.2006openalex-snapshot
PGC-1α: a key regulator of energy metabolism
inferredcites →https://doi.org/10.1152/ajpendo.00745.2009openalex-snapshot
AMPK and SIRT1: a long-standing partnership?
inferredcites →https://doi.org/10.1159/000151428openalex-snapshot
Autophagy: A New Phase in the Maturation of Growth Plate Chondrocytes Is Regulated by HIF, mTOR and AMP Kinase
inferredcites →https://doi.org/10.1186/ar4254openalex-snapshot
Linked decreases in liver kinase B1 and AMP-activated protein kinase activity modulate matrix catabolic responses to biomechanical injury in chondrocytes
inferredcites →https://doi.org/10.1371/journal.pone.0004020openalex-snapshot
Phosphorylation Regulates SIRT1 Function
inferredcites →https://doi.org/10.1371/journal.pone.0006611openalex-snapshot
CK2 Is the Regulator of SIRT1 Substrate-Binding Affinity, Deacetylase Activity and Cellular Response to DNA-Damage
inferredcites →https://doi.org/10.1371/journal.pone.0008414openalex-snapshot
JNK1 Phosphorylates SIRT1 and Promotes Its Enzymatic Activity
inferredcites →https://doi.org/10.2741/4105openalex-snapshot
Towards elucidating the role of SirT1 in osteoarthritis
inferredcites →https://doi.org/10.3892/mmr.2012.863openalex-snapshot
Mitochondrial genome sequencing of chondrocytes in osteoarthritis by Human Mitochondria RT2 Profiler™ PCR Array
inferredcites →https://doi.org/10.5435/jaaos-20-04-261openalex-snapshot
Autophagy: A New Therapeutic Target in Cartilage Injury and Osteoarthritis
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4100420openalex-extracted
Based on Network Pharmacology and Molecular Docking, it Reveals the Potential Molecular Mechanism of Sabia Intervention Knee Osteoarthritis
inferred← citeshttps://doi.org/10.1002/advs.202206144openalex-snapshot
Reprogramming of Mitochondrial Respiratory Chain Complex by Targeting SIRT3‐COX4I2 Axis Attenuates Osteoarthritis Progression
inferred← citeshttps://doi.org/10.1002/art.39842openalex-snapshot
Review: Metabolic Regulation of Inflammation in Osteoarthritis
inferred← citeshttps://doi.org/10.1002/art.40104openalex-snapshot
Regulated in Development and DNA Damage Response 1 Deficiency Impairs Autophagy and Mitochondrial Biogenesis in Articular Cartilage and Increases the Severity of Experimental Osteoarthritis
inferred← citeshttps://doi.org/10.1002/art.40456openalex-snapshot
Impaired Proteasomal Function in Human Osteoarthritic Chondrocytes Can Contribute to Decreased Levels of SOX 9 and Aggrecan
inferred← citeshttps://doi.org/10.1002/art.40517openalex-snapshot
Review: Unmet Needs and the Path Forward in Joint Disease Associated With Calcium Pyrophosphate Crystal Deposition
inferred← citeshttps://doi.org/10.1002/btm2.10187openalex-snapshot
Cartilage‐targeting ultrasmall lipid‐polymer hybrid nanoparticles for the prevention of cartilage degradation
inferred← citeshttps://doi.org/10.1002/ctd2.161openalex-snapshot
Curcumin relieves corticosterone‐induced behaviour deficits via PGC‐1α‐mediated mitochondrial biogenesis and mitophagy
inferred← citeshttps://doi.org/10.1002/eji.202451381openalex-snapshot
Metabolic Reprogramming in Stromal and Immune Cells in Rheumatoid Arthritis and Osteoarthritis: Therapeutic Possibilities
inferred← citeshttps://doi.org/10.1002/jcp.25644openalex-snapshot
Low Shear Stress Attenuates COX‐2 Expression Induced by Resistin in Human Osteoarthritic Chondrocytes
inferred← citeshttps://doi.org/10.1002/jor.23420openalex-snapshot
Emerging role of metabolic signaling in synovial joint remodeling and osteoarthritis
inferred← citeshttps://doi.org/10.1002/jor.23882openalex-snapshot
Mitoprotective therapy preserves chondrocyte viability and prevents cartilage degeneration in an ex vivo model of posttraumatic osteoarthritis
inferred← citeshttps://doi.org/10.1002/ptr.7935openalex-snapshot
Natural anti‐inflammatory products for osteoarthritis: From molecular mechanism to drug delivery systems and clinical trials
inferred← citeshttps://doi.org/10.1002/smll.202405049openalex-snapshot
Injectable Regenerated Silk Fibroin Micro/Nanosphere with Enhanced Permeability and Stability for Osteoarthritis Therapy
inferred← citeshttps://doi.org/10.1002/tox.23414openalex-snapshot
Galectin‐3 facilitates inflammation and apoptosis in chondrocytes through upregulation of the TLR ‐4‐mediated oxidative stress pathway in TC28a2 human chondrocyte cells
inferred← citeshttps://doi.org/10.1002/tox.23885openalex-snapshot
Cisplatin triggers oxidative stress, apoptosis and pro‐inflammatory responses by inhibiting the SIRT1 ‐mediated Nrf2 pathway in chondrocytes
inferred← citeshttps://doi.org/10.1007/978-1-0716-1119-7_14openalex-snapshot
Mitochondrial Biogenesis, Activity, and DNA Isolation in Chondrocytes
inferred← citeshttps://doi.org/10.1007/978-981-99-2452-3_7openalex-snapshot
Role and Application of Biomolecules for Regeneration of Cartilage Tissue
inferred← citeshttps://doi.org/10.1007/s00018-020-03497-9openalex-snapshot
The role of SIRT3-mediated mitochondrial homeostasis in osteoarthritis
inferred← citeshttps://doi.org/10.1007/s00109-016-1422-3openalex-snapshot
Chaperonin 60 regulation of SOX9 ubiquitination mitigates the development of knee osteoarthritis
inferred← citeshttps://doi.org/10.1007/s00109-016-1445-9openalex-snapshot
Fis1 depletion in osteoarthritis impairs chondrocyte survival and peroxisomal and lysosomal function
inferred← citeshttps://doi.org/10.1007/s00296-018-4103-4openalex-snapshot
Regulation of energy metabolism in the growth plate and osteoarthritic chondrocytes
inferred← citeshttps://doi.org/10.1007/s00393-022-01188-3openalex-snapshot
Das DRFZ – ein Vorreiter bei der Erforschung des Zusammenspiels von Immun- und Stromazellen bei der De- und Regeneration des Bewegungsapparats
inferred← citeshttps://doi.org/10.1007/s00438-022-01878-2openalex-snapshot
PGC-1α affects skeletal muscle and adipose tissue development by regulating mitochondrial biogenesis
inferred← citeshttps://doi.org/10.1007/s00438-026-02417-zopenalex-snapshot
Targeting WWP1 ameliorates osteoarthritis by suppressing KLF15 ubiquitination to restore mtDNA and mitochondrial function
inferred← citeshttps://doi.org/10.1007/s10067-020-05406-8openalex-snapshot
Mitochondrial DNA in osteoarthritis disease
inferred← citeshttps://doi.org/10.1007/s10522-021-09910-5openalex-snapshot
Aging and age‐related diseases: from mechanisms to therapeutic strategies
inferred← citeshttps://doi.org/10.1007/s10863-022-09932-9openalex-snapshot
Circ-LRP1B functions as a competing endogenous RNA to regulate proliferation, apoptosis and oxidative stress of LPS-induced human C28/I2 chondrocytes
inferred← citeshttps://doi.org/10.1007/s11010-023-04818-9openalex-snapshot
The role and intervention of mitochondrial metabolism in osteoarthritis
inferred← citeshttps://doi.org/10.1007/s11064-023-03934-8openalex-snapshot
The Role of PGC-1α-Mediated Mitochondrial Biogenesis in Neurons
inferred← citeshttps://doi.org/10.1007/s11427-023-2593-4openalex-snapshot
Activation of the PGC-1α-mediated mitochondrial glutamine metabolism pathway attenuates female offspring osteoarthritis induced by prenatal excessive prednisone
inferred← citeshttps://doi.org/10.1007/s11596-026-00166-zopenalex-snapshot
Lactate and Acidification: Emerging Metabolic Regulators of Osteoarthritis
inferred← citeshttps://doi.org/10.1007/s11926-016-0591-yopenalex-snapshot
The Role of Sirtuins in Cartilage Homeostasis and Osteoarthritis
inferred← citeshttps://doi.org/10.1007/s11926-021-00994-zopenalex-snapshot
Intersections Between Mitochondrial Metabolism and Redox Biology Mediate Posttraumatic Osteoarthritis
inferred← citeshttps://doi.org/10.1007/s11926-026-01229-9openalex-snapshot
Adipose–Joint Crosstalk in Obesity–Induced Osteoarthritis: Mechanisms, Biomarkers, and Translational Therapeutic Opportunities
inferred← citeshttps://doi.org/10.1007/s12011-026-05139-wopenalex-snapshot
Lycopene Attenuates Hexavalent Chromium–Induced Kidney Injury By Modulating PINK1/Parkin-Mediated Mitophagy
inferred← citeshttps://doi.org/10.1007/s12015-022-10348-6openalex-snapshot
Metabolic Regulation: A Potential Strategy for Rescuing Stem Cell Senescence
inferred← citeshttps://doi.org/10.1007/s12015-022-10357-5openalex-snapshot
Artificial Mitochondrial Transfer (AMT) for the Management of Age-related Musculoskeletal Degenerative Disorders: An Emerging Avenue for Bone and Cartilage Metabolism Regulation
inferred← citeshttps://doi.org/10.1007/s12015-022-10436-7openalex-snapshot
BMSCs-derived Mitochondria Improve Osteoarthritis by Ameliorating Mitochondrial Dysfunction and Promoting Mitochondrial Biogenesis in Chondrocytes
inferred← citeshttps://doi.org/10.1007/s12035-025-05611-zopenalex-snapshot
Deciphering the PGC-1α-TFAM Axis in Parkinson’s Disease (PD) — A Mechanism Approach Targeting Therapeutics for PD
inferred← citeshttps://doi.org/10.1007/s13346-022-01229-zopenalex-snapshot
Exploration of novel drug delivery systems in topical management of osteoarthritis
inferred← citeshttps://doi.org/10.1016/b978-0-443-15500-0.00014-1openalex-snapshot
Age-related disease: Joints
inferred← citeshttps://doi.org/10.1016/j.abb.2020.108416openalex-snapshot
Mechanisms involved in the unbalanced redox homeostasis in osteoblastic cellular model of Alkaptonuria
inferred← citeshttps://doi.org/10.1016/j.abb.2024.109960openalex-snapshot
Exosomes derived from diabetic serum accelerate the progression of osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.abb.2024.110083openalex-snapshot
Impaired mitochondrial quality control in fibromyalgia: Mechanisms involved in skeletal muscle alteration
inferred← citeshttps://doi.org/10.1016/j.ajhg.2023.02.011openalex-snapshot
Dynamic chromatin accessibility tuning by the long noncoding RNA ELDR accelerates chondrocyte senescence and osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.archger.2024.105522openalex-snapshot
Mitochondrial quality control dysfunction in osteoarthritis: Mechanisms, therapeutic strategies & future prospects
inferred← citeshttps://doi.org/10.1016/j.arr.2020.101249openalex-snapshot
The role of metabolism in chondrocyte dysfunction and the progression of osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.arr.2021.101315openalex-snapshot
Mechanisms linking mitochondrial mechanotransduction and chondrocyte biology in the pathogenesis of osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.arr.2025.102695openalex-snapshot
Adenosine metabolism and receptors in aging of the skin, musculoskeletal, immune and cardiovascular systems
inferred← citeshttps://doi.org/10.1016/j.arr.2026.103291openalex-snapshot
Metabolic reprogramming-driven neuroimmunoregulation: Key mechanisms and therapeutic opportunities and challenges in central nervous system disorders
inferred← citeshttps://doi.org/10.1016/j.bbadis.2015.12.017openalex-snapshot
Mitochondrial DNA haplogroups modify the risk of osteoarthritis by altering mitochondrial function and intracellular mitochondrial signals
inferred← citeshttps://doi.org/10.1016/j.bbadis.2019.165575openalex-snapshot
Icariin protects human nucleus pulposus cells from hydrogen peroxide-induced mitochondria-mediated apoptosis by activating nuclear factor erythroid 2-related factor 2
inferred← citeshttps://doi.org/10.1016/j.bbadis.2024.167301openalex-snapshot
Role of mitochondrial dysfunction and biogenesis in fibromyalgia syndrome: Molecular mechanism in central nervous system
inferred← citeshttps://doi.org/10.1016/j.bbi.2022.02.015openalex-snapshot
Sestrin2 overexpression attenuates osteoarthritis pain via induction of AMPK/PGC-1α-mediated mitochondrial biogenesis and suppression of neuroinflammation
inferred← citeshttps://doi.org/10.1016/j.bcp.2018.02.002openalex-snapshot
Adenosine metabolism, immunity and joint health
inferred← citeshttps://doi.org/10.1016/j.bcp.2019.03.008openalex-snapshot
Role of mitochondrial dysfunction on rheumatic diseases
inferred← citeshttps://doi.org/10.1016/j.bcp.2020.114005openalex-snapshot
Maresin conjugates in tissue regeneration 1 prevents lipopolysaccharide-induced cardiac dysfunction through improvement of mitochondrial biogenesis and function
inferred← citeshttps://doi.org/10.1016/j.bioactmat.2026.06.015openalex-snapshot
Restoring cartilage-subchondral bone metabolism with biphasic magnesium microspheres ameliorate early osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.biopha.2018.05.003openalex-snapshot
Quercetin attenuates mitochondrial dysfunction and biogenesis via upregulated AMPK/SIRT1 signaling pathway in OA rats
inferred← citeshttps://doi.org/10.1016/j.biopha.2023.115552openalex-snapshot
Mechanical stress abnormalities promote chondrocyte senescence - The pathogenesis of knee osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.biopha.2024.116673openalex-snapshot
The interplay between mitochondrial dysfunction and NLRP3 inflammasome in multiple sclerosis: Therapeutic implications and animal model studies
inferred← citeshttps://doi.org/10.1016/j.bone.2019.01.018openalex-snapshot
Sirtuins and FoxOs in osteoporosis and osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.bone.2020.115827openalex-snapshot
Deletion of SIRT3 inhibits osteoclastogenesis and alleviates aging or estrogen deficiency-induced bone loss in female mice
inferred← citeshttps://doi.org/10.1016/j.bone.2024.117313openalex-snapshot
Carbonic anhydrase 2 is important for articular chondrocyte function and metabolic homeostasis
inferred← citeshttps://doi.org/10.1016/j.bonr.2023.101658openalex-snapshot
Post-traumatic osteoarthritis: A review of pathogenic mechanisms and novel targets for mitigation
inferred← citeshttps://doi.org/10.1016/j.cbi.2024.111238openalex-snapshot
Telomerase reverse transcriptase protects against diabetic kidney disease by promoting AMPK/PGC-1a-regulated mitochondrial energy homeostasis
inferred← citeshttps://doi.org/10.1016/j.cej.2024.155662openalex-snapshot
Lesion-localized hydrogels functionalized with engineered extracellular matrix restore cellular homeostasis by enhancing mitochondrial energy metabolism for osteoarthritis therapy
inferred← citeshttps://doi.org/10.1016/j.cej.2025.163554openalex-snapshot
Bioactive multifunctional hydrogel to ameliorate adverse joint microenvironment and enhance cartilage regeneration
inferred← citeshttps://doi.org/10.1016/j.celrep.2020.108207openalex-snapshot
Reducing NADPH Synthesis Counteracts Diabetic Nephropathy through Restoration of AMPK Activity in Type 1 Diabetic Rats
inferred← citeshttps://doi.org/10.1016/j.clnu.2024.06.001openalex-snapshot
SIRT3 facilitates mitochondrial structural repair and functional recovery in rats after ischemic stroke by promoting OPA1 expression and activity
inferred← citeshttps://doi.org/10.1016/j.compositesb.2022.110257openalex-snapshot
Dynamic protein hydrogel with supramolecularly enveloped kartogenin promotes cartilage regeneration through mitochondrial activation
inferred← citeshttps://doi.org/10.1016/j.ecoenv.2026.120822openalex-snapshot
SIRT1/PGC-1α-regulated mitochondrial biogenesis is involved in T-2 toxin-induced chondrocyte injury
inferred← citeshttps://doi.org/10.1016/j.exger.2024.112379openalex-snapshot
The role of mitochondrial quality control mechanisms in chondrocyte senescence
inferred← citeshttps://doi.org/10.1016/j.fbio.2024.103793openalex-snapshot
Effect of Yamadazyma triangularis derived peptide XHY69AP on muscle fatigue via regulation of AMPK/PGC-1α and Nrf2/Keap1 pathways
inferred← citeshttps://doi.org/10.1016/j.fbio.2024.105260openalex-snapshot
Icariin promotes skeletal muscle mitochondrial biogenesis by the AMPK-PPARδ-PGC-1α signaling pathway: A potential mechanism for beneficial muscle function homeostasis
inferred← citeshttps://doi.org/10.1016/j.fct.2026.116161openalex-snapshot
The TFAM-OGG1 axis mediates T-2 toxin-induced chondrocyte mitochondrial dysfunction and cartilage degeneration
inferred← citeshttps://doi.org/10.1016/j.foodchem.2025.147604openalex-snapshot
Food-derived antioxidant peptides mediated multifunctional bioactivities and bioavailability-enhancing delivery strategies via the Nrf2/Keap1 signaling pathway
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2018.08.038openalex-snapshot
Reactive oxygen species, aging and articular cartilage homeostasis
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2019.12.007openalex-snapshot
Relevance of Nrf2 and heme oxygenase-1 in articular diseases
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2022.08.038openalex-snapshot
LONP1 downregulation with ageing contributes to osteoarthritis via mitochondrial dysfunction
inferred← citeshttps://doi.org/10.1016/j.gendis.2023.101180openalex-snapshot
NRF2 in age-related musculoskeletal diseases: Role and treatment prospects
inferred← citeshttps://doi.org/10.1016/j.ijbiomac.2025.144482openalex-snapshot
Intra-articular metformin-curcumin cationic PLGA nanoparticles rejuvenate articular structure in MIA induced osteoarthritis model via modulating the crosstalk between miR93, TNFAIP3/TLR/NF-κB and AMPK/SIRT1 trajectories
inferred← citeshttps://doi.org/10.1016/j.intimp.2024.111755openalex-snapshot
SIRT3 alleviates high glucose-induced chondrocyte injury through the promotion of autophagy and suppression of apoptosis in osteoarthritis progression
inferred← citeshttps://doi.org/10.1016/j.intimp.2024.112858openalex-snapshot
ASA VI controls osteoarthritis in mice by maintaining mitochondrial homeostasis through Sirtuin 3
inferred← citeshttps://doi.org/10.1016/j.jbiomech.2022.111336openalex-snapshot
Impacts of aging on murine cartilage biomechanics and chondrocyte in situ calcium signaling
inferred← citeshttps://doi.org/10.1016/j.jbspin.2023.105642openalex-snapshot
The role of autophagy in mitigating osteoarthritis progression via regulation of chondrocyte apoptosis: A review
inferred← citeshttps://doi.org/10.1016/j.jcyt.2020.08.010openalex-snapshot
Mitochondrial DNA from osteoarthritic patients drives functional impairment of mitochondrial activity: a study on transmitochondrial cybrids
inferred← citeshttps://doi.org/10.1016/j.joca.2017.10.008openalex-snapshot
Osteoarthritis year in review 2017: biology
inferred← citeshttps://doi.org/10.1016/j.joca.2018.05.018openalex-snapshot
Mitochondria and mitophagy: biosensors for cartilage degradation and osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.joca.2018.07.004openalex-snapshot
Activation of AMPK-SIRT3 signaling is chondroprotective by preserving mitochondrial DNA integrity and function
inferred← citeshttps://doi.org/10.1016/j.joca.2019.10.010openalex-snapshot
Osteoarthritis-associated basic calcium phosphate crystals alter immune cell metabolism and promote M1 macrophage polarization
inferred← citeshttps://doi.org/10.1016/j.joca.2021.04.006openalex-snapshot
Alpha defensin-1 attenuates surgically induced osteoarthritis in association with promoting M1 to M2 macrophage polarization
inferred← citeshttps://doi.org/10.1016/j.joca.2021.04.016openalex-snapshot
Cellular carbon stress is a mediator of obesity-associated osteoarthritis development
inferred← citeshttps://doi.org/10.1016/j.joca.2021.06.013openalex-snapshot
Fundamentals of OA. An initiative of Osteoarthritis and Cartilage. Obesity and metabolic factors in OA
inferred← citeshttps://doi.org/10.1016/j.joca.2021.10.004openalex-snapshot
Oral administration of berberine limits post-traumatic osteoarthritis development and associated pain via AMP-activated protein kinase (AMPK) in mice
inferred← citeshttps://doi.org/10.1016/j.joca.2021.10.009openalex-snapshot
Mitochondrial quality control in cartilage damage and osteoarthritis: new insights and potential therapeutic targets
inferred← citeshttps://doi.org/10.1016/j.joca.2023.03.013openalex-snapshot
Synovial fluid mitochondrial DNA concentration reflects the degree of cartilage damage after naturally occurring articular injury
inferred← citeshttps://doi.org/10.1016/j.joca.2023.04.003openalex-snapshot
Cross-talk of inflammation and chondrocyte intracellular metabolism in osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.joca.2025.10.002openalex-snapshot
Fibroblast and myeloid cells with high mitochondrial RNA content represent biologically significant populations within the OA synovium
inferred← citeshttps://doi.org/10.1016/j.lfs.2020.118602openalex-snapshot
Role of mitochondria in mediating chondrocyte response to mechanical stimuli
inferred← citeshttps://doi.org/10.1016/j.mad.2016.02.003openalex-snapshot
Regulation of SIRT1 in aging: Roles in mitochondrial function and biogenesis
inferred← citeshttps://doi.org/10.1016/j.mad.2020.111215openalex-snapshot
SIRT1 and aging related signaling pathways
inferred← citeshttps://doi.org/10.1016/j.mad.2024.111993openalex-snapshot
The role of mitochondria in cytokine and chemokine signalling during ageing
inferred← citeshttps://doi.org/10.1016/j.mam.2025.101361openalex-snapshot
Implications of obesity-mediated cellular dysfunction and adipocytokine signaling pathways in the pathogenesis of osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.matbio.2020.12.002openalex-snapshot
Role of autophagy in intervertebral disc and cartilage function: implications in health and disease
inferred← citeshttps://doi.org/10.1016/j.matdes.2026.116383openalex-snapshot
Biomimetic hydrogel delivers engineered exosomes to attenuate cell senescence by AKG-mediated mitochondrial function restoration for osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.mbm.2025.100164openalex-snapshot
Mechanical cues enhance chondrocyte function: Insights from mechanoreception, regulation, and biological responses
inferred← citeshttps://doi.org/10.1016/j.mito.2024.101923openalex-snapshot
Role of mitochondrial homeostasis in D-galactose-induced cardiovascular ageing from bench to bedside
inferred← citeshttps://doi.org/10.1016/j.mtbio.2022.100512openalex-snapshot
Platelet-derived extracellular vesicles ameliorate intervertebral disc degeneration by alleviating mitochondrial dysfunction
inferred← citeshttps://doi.org/10.1016/j.mtbio.2025.102056openalex-snapshot
Bioadhesive and antioxidant hydrogel by MoS2-TA dual-catalytic system initiated free radical polymerization combined with phototherapy for cartilage regeneration
inferred← citeshttps://doi.org/10.1016/j.mtbio.2026.103300openalex-snapshot
Microfluidic methacrylated hyaluronic acid microspheres incorporating MnO2 and exosomes for antioxidant defense and inflammation regulation in osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.npep.2026.102637openalex-snapshot
PACAP promotes mitochondrial biogenesis in rat primary chondrocytes via the SIRT1/PGC-1α pathway
inferred← citeshttps://doi.org/10.1016/j.omtn.2025.102552openalex-snapshot
Antibody and aptamer-based therapies for osteoarthritis: Application of antibodies and promise of aptamers
inferred← citeshttps://doi.org/10.1016/j.phrs.2020.105369openalex-snapshot
Common risk factors and therapeutic targets in obstructive sleep apnea and osteoarthritis: An unexpectable link?
inferred← citeshttps://doi.org/10.1016/j.phrs.2021.105497openalex-snapshot
Mechanistic insights into AMPK-SIRT3 positive feedback loop-mediated chondrocyte mitochondrial quality control in osteoarthritis pathogenesis
inferred← citeshttps://doi.org/10.1016/j.phrs.2024.107141openalex-snapshot
Sirtuin 1 in osteoarthritis: Perspectives on regulating glucose metabolism
inferred← citeshttps://doi.org/10.1016/j.phymed.2024.156115openalex-snapshot
Vinpocetine protects against osteoarthritis by inhibiting ferroptosis and extracellular matrix degradation via activation of the Nrf2/GPX4 pathway
inferred← citeshttps://doi.org/10.1016/j.redox.2018.01.010openalex-snapshot
Homocysteine causes dysfunction of chondrocytes and oxidative stress through repression of SIRT1/AMPK pathway: A possible link between hyperhomocysteinemia and osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.rvsc.2025.105941openalex-snapshot
Hypoxia promotes mitochondrial biogenesis in myocardium of broiler with pulmonary hypertension syndrome through AMPK/PGC-1 alpha pathway
inferred← citeshttps://doi.org/10.1016/j.semcdb.2016.11.008openalex-snapshot
MSC exosome as a cell-free MSC therapy for cartilage regeneration: Implications for osteoarthritis treatment
inferred← citeshttps://doi.org/10.1016/j.trsl.2026.07.003openalex-snapshot
Multidimensional regulatory mechanisms and targeted intervention strategies of the gut-joint axis in metabolic osteoarthritis
inferred← citeshttps://doi.org/10.1016/j.yexcr.2018.06.012openalex-snapshot
Autophagy attenuates compression-induced apoptosis of human nucleus pulposus cells via MEK/ERK/NRF1/Atg7 signaling pathways during intervertebral disc degeneration
inferred← citeshttps://doi.org/10.1021/acsptsci.4c00706openalex-snapshot
A Molecular Chemical Perspective: Mitochondrial Dynamics Is Not a Bystander of Cartilage Diseases
inferred← citeshttps://doi.org/10.1038/nrrheum.2016.65openalex-snapshot
Ageing and the pathogenesis of osteoarthritis
inferred← citeshttps://doi.org/10.1038/nrrheum.2017.50openalex-snapshot
The role of metabolism in the pathogenesis of osteoarthritis
inferred← citeshttps://doi.org/10.1038/s12276-020-0408-yopenalex-snapshot
The role of selenium metabolism and selenoproteins in cartilage homeostasis and arthropathies
inferred← citeshttps://doi.org/10.1038/s12276-026-01719-xopenalex-snapshot
Vertebral BMSC-EVs under estrogen deficiency drive senescence-related mitochondrial dysfunction in endplate chondrocytes via MRPL1 mRNA delivery
inferred← citeshttps://doi.org/10.1038/s41392-022-01257-8openalex-snapshot
The sirtuin family in health and disease
inferred← citeshttps://doi.org/10.1038/s41392-023-01330-wopenalex-snapshot
Osteoarthritis: pathogenic signaling pathways and therapeutic targets
inferred← citeshttps://doi.org/10.1038/s41392-024-01756-wopenalex-snapshot
Peroxisome proliferator-activated receptor gamma coactivator-1 (PGC-1) family in physiological and pathophysiological process and diseases
inferred← citeshttps://doi.org/10.1038/s41392-025-02371-zopenalex-snapshot
Immunosenescence: signaling pathways, diseases and therapeutic targets
inferred← citeshttps://doi.org/10.1038/s41392-025-02556-6openalex-snapshot
Osteoarthritis: molecular pathogenesis and potential therapeutic options
inferred← citeshttps://doi.org/10.1038/s41413-025-00460-xopenalex-snapshot
The impact of mitochondrial dysfunction on osteoarthritis cartilage: current insights and emerging mitochondria-targeted therapies
inferred← citeshttps://doi.org/10.1038/s41413-026-00540-6openalex-snapshot
Novel therapeutic strategies for osteoarthritis: from mechanistic insights to precision medicine
inferred← citeshttps://doi.org/10.1038/s41413-026-00548-yopenalex-snapshot
Exploring mitochondrial functions and dysfunctions in chondrocytes: toward the identification of novel therapies for osteoarthritis
inferred← citeshttps://doi.org/10.1038/s41418-022-01053-5openalex-snapshot
Sirt3 mediates the benefits of exercise on bone in aged mice
inferred← citeshttps://doi.org/10.1038/s41467-024-55335-2openalex-snapshot
GATD3A-deficiency-induced mitochondrial dysfunction facilitates senescence of fibroblast-like synoviocytes and osteoarthritis progression
inferred← citeshttps://doi.org/10.1038/s41584-018-0001-0openalex-snapshot
Mitochondrial DNA variation and the pathogenesis of osteoarthritis phenotypes
inferred← citeshttps://doi.org/10.1038/s41584-020-0407-3openalex-snapshot
Interplay between genetics and epigenetics in osteoarthritis
inferred← citeshttps://doi.org/10.1038/s41598-018-25348-1openalex-snapshot
Apple procyanidins promote mitochondrial biogenesis and proteoglycan biosynthesis in chondrocytes
inferred← citeshttps://doi.org/10.1038/s41598-021-88083-0openalex-snapshot
Mitochondrial DNA impact on joint damaged process in a conplastic mouse model after being surgically induced with osteoarthritis
inferred← citeshttps://doi.org/10.1038/s41598-025-90069-1openalex-snapshot
Dexmedetomidine ameliorates hepatic ischemia reperfusion injury via modulating SIRT3 mediated mitochondrial quality control
inferred← citeshttps://doi.org/10.1038/s41598-025-92328-7openalex-snapshot
α-Lipoic acid alleviate myocardial infarction by suppressing age-independent macrophage senescence
inferred← citeshttps://doi.org/10.1042/bsr20190189openalex-snapshot
The role of sirtuin 1 and its activator, resveratrol in osteoarthritis
inferred← citeshttps://doi.org/10.1073/pnas.2207993120openalex-snapshot
NOS inhibition reverses TLR2-induced chondrocyte dysfunction and attenuates age-related osteoarthritis
inferred← citeshttps://doi.org/10.1080/00325481.2020.1730669openalex-snapshot
Osteoarthritis and inflammation: a serious disease with overlapping phenotypic patterns
inferred← citeshttps://doi.org/10.1080/10408398.2022.2145264openalex-snapshot
5’AMP-activated protein kinase: an emerging target of phytochemicals to treat chronic inflammatory diseases
inferred← citeshttps://doi.org/10.1080/13813455.2020.1727927openalex-snapshot
Catalpol promotes mitochondrial biogenesis in chondrocytes
inferred← citeshttps://doi.org/10.1080/13813455.2020.1819337openalex-snapshot
Omentin-1 promotes mitochondrial biogenesis via PGC1α-AMPK pathway in chondrocytes
inferred← citeshttps://doi.org/10.1080/14728222.2026.2690139openalex-snapshot
The protective role of SIRT1 in cardiovascular disease: from molecular mechanisms to potential therapeutic strategies
inferred← citeshttps://doi.org/10.1080/21691401.2019.1576710openalex-snapshot
The use of stromal vascular fraction (SVF), platelet-rich plasma (PRP) and stem cells in the treatment of osteoarthritis: an overview of clinical trials
inferred← citeshttps://doi.org/10.1089/jir.2019.0139openalex-snapshot
Magnolol Alleviates IL-1β-Induced Dysfunction of Chondrocytes Through Repression of SIRT1/AMPK/PGC-1α Signaling Pathway
inferred← citeshttps://doi.org/10.1089/wound.2024.0270openalex-snapshot
Chondrocyte Mitochondrial Quality Control: A Novel Insight into Osteoarthritis and Cartilage Regeneration
inferred← citeshttps://doi.org/10.1093/milmed/usac093openalex-snapshot
Anterior Cruciate Ligament Reconstruction Surgery: Creating a Permissive Healing Phenotype in Military Personnel and Civilians for Faster Recovery
inferred← citeshttps://doi.org/10.1093/rheumatology/keaa565openalex-snapshot
Expression of mitochondrial TSPO and FAM173B is associated with inflammation and symptoms in patients with painful knee osteoarthritis
inferred← citeshttps://doi.org/10.1093/rheumatology/kew394openalex-snapshot
A replication study and meta-analysis of mitochondrial DNA variants in the radiographic progression of knee osteoarthritis
inferred← citeshttps://doi.org/10.1096/fj.202201228ropenalex-snapshot
In vitro study to identify ligand‐independent function of estrogen receptor‐α in suppressing DNA damage‐induced chondrocyte senescence
inferred← citeshttps://doi.org/10.1097/bor.0000000000000352openalex-snapshot
Genetic association studies in osteoarthritis: is it fairytale?
inferred← citeshttps://doi.org/10.1097/bor.0000000000000373openalex-snapshot
Metabolic syndrome-associated osteoarthritis
inferred← citeshttps://doi.org/10.1097/bor.0000000000000456openalex-snapshot
Targeting aging for disease modification in osteoarthritis
inferred← citeshttps://doi.org/10.1097/bor.0000000000000855openalex-snapshot
Is osteoarthritis a mitochondrial disease? What is the evidence
inferred← citeshttps://doi.org/10.1097/md.0000000000016355openalex-snapshot
Relationship between oxidative balance score and quality of life in patients with osteoarthritis
inferred← citeshttps://doi.org/10.1101/2025.10.28.684789openalex-snapshot
Multimodal analysis of osteoarthritic chondrocytes reveals mitochondrial alterations and patient-specific OxPhos response to bezafibrate
inferred← citeshttps://doi.org/10.1111/acel.13662openalex-snapshot
Urolithin A improves mitochondrial health, reduces cartilage degeneration, and alleviates pain in osteoarthritis
inferred← citeshttps://doi.org/10.1111/acel.14294openalex-snapshot
β‐Hydroxybutyrate enhances chondrocyte mitophagy and reduces cartilage degeneration in osteoarthritis via the HCAR2 / AMPK / PINK1 /Parkin pathway
inferred← citeshttps://doi.org/10.1111/cns.70530openalex-snapshot
Osteocalcin Ameliorates CUMS ‐Induced Depressive‐Like Behaviors by Reducing Mitochondrial Damage in Hippocampal Neurons
inferred← citeshttps://doi.org/10.1111/febs.70247openalex-snapshot
Propionibacterium acnes evades microbicidal phagocytosis by inhibiting the mitochondrial biogenesis of nucleus pulposus cells
inferred← citeshttps://doi.org/10.1111/jcmm.15194openalex-snapshot
Mitochondrial biogenesis: An update
inferred← citeshttps://doi.org/10.1111/jcmm.70236openalex-snapshot
Hydrogen Protects Mitochondrial Function by Increasing the Expression of PGC ‐1α and Ameliorating Myocardial Ischaemia–Reperfusion Injury
inferred← citeshttps://doi.org/10.1111/jpi.12815openalex-snapshot
Recharge of chondrocyte mitochondria by sustained release of melatonin protects cartilage matrix homeostasis in osteoarthritis
inferred← citeshttps://doi.org/10.1111/nyas.13930openalex-snapshot
Phenotypic instability of chondrocytes in osteoarthritis: on a path to hypertrophy
inferred← citeshttps://doi.org/10.1111/nyas.14025openalex-snapshot
CITED2 mediates the mechanical loading–induced suppression of adipokines in the infrapatellar fat pad
inferred← citeshttps://doi.org/10.1126/sciadv.aec1031openalex-snapshot
Aptamer-functionalized apoptotic vesicles ameliorate osteoarthritis via resuming mitochondria OXPHOS of chondrocytes
inferred← citeshttps://doi.org/10.1134/s1990750820020043openalex-snapshot
Type 2 Diabetes Mellitus in Osteoarthritic Patients: Does Association Between Metabolic Impairments, Joint Destruction, and Pain Exist?
inferred← citeshttps://doi.org/10.1136/annrheumdis-2016-210131openalex-snapshot
Mitochondrial DNA haplogroups influence the risk of incident knee osteoarthritis in OAI and CHECK cohorts. A meta-analysis and functional study
inferred← citeshttps://doi.org/10.1136/ard-2022-223570openalex-snapshot
A meta-analysis and a functional study support the influence of mtDNA variant m.16519C on the risk of rapid progression of knee osteoarthritis
inferred← citeshttps://doi.org/10.1155/2016/6432867openalex-snapshot
Deferoxamine Suppresses Collagen Cleavage and Protease, Cytokine, and COL10A1 Expression and Upregulates AMPK and Krebs Cycle Genes in Human Osteoarthritic Cartilage
inferred← citeshttps://doi.org/10.1155/2018/1368142openalex-snapshot
Upregulation of SIRT1 by Kartogenin Enhances Antioxidant Functions and Promotes Osteogenesis in Human Mesenchymal Stem Cells
inferred← citeshttps://doi.org/10.1155/2018/3075293openalex-snapshot
Emerging Players at the Intersection of Chondrocyte Loss of Maturational Arrest, Oxidative Stress, Senescence and Low‐Grade Inflammation in Osteoarthritis
inferred← citeshttps://doi.org/10.1155/2020/4143802openalex-snapshot
Insulin Resistance in Osteoarthritis: Similar Mechanisms to Type 2 Diabetes Mellitus
inferred← citeshttps://doi.org/10.1155/2020/9423593openalex-snapshot
Mitochondrial ROS-Modulated mtDNA: A Potential Target for Cardiac Aging
inferred← citeshttps://doi.org/10.1155/2023/1708251openalex-snapshot
Curcumin Ameliorates Age-Induced Tight Junction Impaired in Porcine Sertoli Cells by Inactivating the NLRP3 Inflammasome through the AMPK/SIRT3/SOD2/mtROS Signaling Pathway
inferred← citeshttps://doi.org/10.1155/2024/6822664openalex-snapshot
Identification and Verification of Hub Mitochondrial Dysfunction Genes in Osteoarthritis Based on Bioinformatics Analysis
inferred← citeshttps://doi.org/10.1159/000521720openalex-snapshot
Advanced Glycation End Products Induced Mitochondrial Dysfunction of Chondrocytes through Repression of AMPKα-SIRT1-PGC-1α Pathway
inferred← citeshttps://doi.org/10.1161/jaha.117.005945openalex-snapshot
Alogliptin, a Dipeptidyl Peptidase‐4 Inhibitor, Alleviates Atrial Remodeling and Improves Mitochondrial Function and Biogenesis in Diabetic Rabbits
inferred← citeshttps://doi.org/10.1172/jci.insight.170513openalex-snapshot
Mocetinostat activates Krüppel-like factor 4 and protects against tissue destruction and inflammation in osteoarthritis
inferred← citeshttps://doi.org/10.1177/03000605211021752openalex-snapshot
The Rho kinase signaling pathway participates in tubular mitochondrial oxidative injury and apoptosis in uric acid nephropathy
inferred← citeshttps://doi.org/10.1177/17448069261444495openalex-snapshot
Redox-associated gene and microRNA signatures in degenerative intervertebral disc disease
inferred← citeshttps://doi.org/10.1177/19476035211063858openalex-snapshot
Role of Mitochondria in Physiology of Chondrocytes and Diseases of Osteoarthritis and Rheumatoid Arthritis
inferred← citeshttps://doi.org/10.1177/1947603521993209openalex-snapshot
Sirt5 Deficiency Causes Posttranslational Protein Malonylation and Dysregulated Cellular Metabolism in Chondrocytes Under Obesity Conditions
inferred← citeshttps://doi.org/10.1186/s11658-023-00489-yopenalex-snapshot
Oxidative stress as a key modulator of cell fate decision in osteoarthritis and osteoporosis: a narrative review
inferred← citeshttps://doi.org/10.1186/s12891-017-1621-2openalex-snapshot
The mitochondrial inhibitor oligomycin induces an inflammatory response in the rat knee joint
inferred← citeshttps://doi.org/10.1186/s12891-022-05911-8openalex-snapshot
Effects of mitochondrial dysfunction on bone metabolism and related diseases: a scientometric study from 2003 to 2022
inferred← citeshttps://doi.org/10.1186/s12906-019-2673-7openalex-snapshot
RETRACTED ARTICLE: Acteoside inhibits inflammatory response via JAK/STAT signaling pathway in osteoarthritic rats
inferred← citeshttps://doi.org/10.1186/s12933-019-0964-4openalex-snapshot
Empagliflozin, a sodium glucose co-transporter-2 inhibitor, alleviates atrial remodeling and improves mitochondrial function in high-fat diet/streptozotocin-induced diabetic rats
inferred← citeshttps://doi.org/10.1186/s12964-022-00943-yopenalex-snapshot
HK2: a potential regulator of osteoarthritis via glycolytic and non-glycolytic pathways
inferred← citeshttps://doi.org/10.1186/s12964-023-01069-5openalex-snapshot
FGF19 increases mitochondrial biogenesis and fusion in chondrocytes via the AMPKα-p38/MAPK pathway
showing the first 200 of 296 · page through all 296
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.