On the public citation graph

Activation of AMPK-SIRT3 signaling is chondroprotective by preserving mitochondrial DNA integrity and function

https://doi.org/10.1016/j.joca.2018.07.004

0 signed · 174 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.

Claim with ORCID

Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.

See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/art.10837openalex-snapshot

Mitochondrial respiratory activity is altered in osteoarthritic human articular chondrocytes

inferredcites →https://doi.org/10.1002/art.30333openalex-snapshot

Chondrocyte AMP-activated protein kinase activity suppresses matrix degradation responses to proinflammatory cytokines interleukin-1β and tumor necrosis factor α

inferredcites →https://doi.org/10.1002/art.34453openalex-snapshot

Osteoarthritis: A disease of the joint as an organ

inferredcites →https://doi.org/10.1002/art.34508openalex-snapshot

Mitochondrial dysfunction increases inflammatory responsiveness to cytokines in normal human chondrocytes

inferredcites →https://doi.org/10.1002/art.37782openalex-snapshot

Mitochondrial dysfunction in osteoarthritis is associated with down‐regulation of superoxide dismutase 2

inferredcites →https://doi.org/10.1002/art.38791openalex-snapshot

Peroxisome Proliferator–Activated Receptor γ Coactivator 1α and FoxO3A Mediate Chondroprotection by AMP‐Activated Protein Kinase

inferredcites →https://doi.org/10.1002/art.39182openalex-snapshot

Mitochondrial Biogenesis Is Impaired in Osteoarthritis Chondrocytes but Reversible via Peroxisome Proliferator–Activated Receptor γ Coactivator 1α

inferredcites →https://doi.org/10.1002/art.39618openalex-snapshot

Aging Promotes Sirtuin 3–Dependent Cartilage Superoxide Dismutase 2 Acetylation and Osteoarthritis

inferredcites →https://doi.org/10.1007/s11926-016-0591-yopenalex-snapshot

The Role of Sirtuins in Cartilage Homeostasis and Osteoarthritis

inferredcites →https://doi.org/10.1016/j.bbadis.2016.10.021openalex-snapshot

Sirt3 deficiency exacerbates diabetic cardiac dysfunction: Role of Foxo3A-Parkin-mediated mitophagy

inferredcites →https://doi.org/10.1016/j.cell.2013.05.039openalex-snapshot

The Hallmarks of Aging

inferredcites →https://doi.org/10.1016/j.dnarep.2008.01.003openalex-snapshot

OGG1 is degraded by calpain following oxidative stress and cisplatin exposure

inferredcites →https://doi.org/10.1016/j.freeradbiomed.2009.06.010openalex-snapshot

Role of mitochondrial hOGG1 and aconitase in oxidant-induced lung epithelial cell apoptosis

inferredcites →https://doi.org/10.1016/j.freeradbiomed.2010.10.692openalex-snapshot

The DNA glycosylase Ogg1 defends against oxidant-induced mtDNA damage and apoptosis in pulmonary artery endothelial cells

inferredcites →https://doi.org/10.1016/j.joca.2005.06.011openalex-snapshot

Accumulation of mitochondrial DNA with 4977-bp deletion in knee cartilage – an association with idiopathic osteoarthritis

inferredcites →https://doi.org/10.1016/j.joca.2008.05.009openalex-snapshot

Diminished mitochondrial DNA integrity and repair capacity in OA chondrocytes

inferredcites →https://doi.org/10.1016/j.joca.2012.05.009openalex-snapshot

DNA damage, discoordinated gene expression and cellular senescence in osteoarthritic chondrocytes

inferredcites →https://doi.org/10.1016/j.joca.2014.12.016openalex-snapshot

Inflammation and intracellular metabolism: new targets in OA

inferredcites →https://doi.org/10.1016/j.joca.2017.07.020openalex-snapshot

Parkin clearance of dysfunctional mitochondria regulates ROS levels and increases survival of human chondrocytes

inferredcites →https://doi.org/10.1016/j.tibs.2015.12.006openalex-snapshot

Mechanisms and Dynamics of Protein Acetylation in Mitochondria

inferredcites →https://doi.org/10.1016/s0378-1119(01)00813-7openalex-snapshot

Mitochondrial DNA repair of oxidative damage in mammalian cells

inferredcites →https://doi.org/10.1038/290457a0openalex-snapshot

Sequence and organization of the human mitochondrial genome

inferredcites →https://doi.org/10.1038/cddis.2013.254openalex-snapshot

Interaction of Sirt3 with OGG1 contributes to repair of mitochondrial DNA and protects from apoptotic cell death under oxidative stress

inferredcites →https://doi.org/10.1038/nrrheum.2011.109openalex-snapshot

Autophagy and cartilage homeostasis mechanisms in joint health, aging and OA

inferredcites →https://doi.org/10.1038/srep11722openalex-snapshot

Mechanical overloading causes mitochondrial superoxide and SOD2 imbalance in chondrocytes resulting in cartilage degeneration

inferredcites →https://doi.org/10.1038/srep43245openalex-snapshot

AMPK deficiency in chondrocytes accelerated the progression of instability-induced and ageing-associated osteoarthritis in adult mice

inferredcites →https://doi.org/10.1073/pnas.0705070104openalex-snapshot

AMP-activated protein kinase (AMPK) action in skeletal muscle via direct phosphorylation of PGC-1α

inferredcites →https://doi.org/10.1073/pnas.1302961110openalex-snapshot

Label-free quantitative proteomics of the lysine acetylome in mitochondria identifies substrates of SIRT3 in metabolic pathways

inferredcites →https://doi.org/10.1073/pnas.94.2.514openalex-snapshot

Mitochondrial DNA damage is more extensive and persists longer than nuclear DNA damage in human cells following oxidative stress

inferredcites →https://doi.org/10.1074/jbc.m112.355206openalex-snapshot

SIRT3 Protein Deacetylates Isocitrate Dehydrogenase 2 (IDH2) and Regulates Mitochondrial Redox Status

inferredcites →https://doi.org/10.1074/jbc.m115.693523openalex-snapshot

Oxidative Stress Promotes Peroxiredoxin Hyperoxidation and Attenuates Pro-survival Signaling in Aging Chondrocytes

inferredcites →https://doi.org/10.1083/jcb.201010024openalex-snapshot

Mitochondrial DNA mutations in disease and aging

inferredcites →https://doi.org/10.1089/ars.2016.6682openalex-snapshot

Mitochondrial Dynamics Is Linked to Longevity and Protects from End-Organ Injury: The Emerging Role of Sirtuin 3

inferredcites →https://doi.org/10.1093/carcin/20.9.1863openalex-snapshot

Overexpression of Ogg1 in mammalian cells: effects on induced and spontaneous oxidative DNA damage and mutagenesis

inferredcites →https://doi.org/10.1093/carcin/22.9.1459openalex-snapshot

Age-related and tissue-specific accumulation of oxidative DNA base damage in 7,8-dihydro-8-oxoguanine-DNA glycosylase (Ogg1) deficient mice

inferredcites →https://doi.org/10.1111/acel.12538openalex-snapshot

Function of the SIRT 3 mitochondrial deacetylase in cellular physiology, cancer, and neurodegenerative disease

inferredcites →https://doi.org/10.1128/mcb.01483-13openalex-snapshot

SIRT3 Deacetylates and Activates OPA1 To Regulate Mitochondrial Dynamics during Stress

inferredcites →https://doi.org/10.1152/ajpcell.00140.2013openalex-snapshot

Mitochondrial 8-oxoguanine glycosylase decreases mitochondrial fragmentation and improves mitochondrial function in H9C2 cells under oxidative stress conditions

inferredcites →https://doi.org/10.1152/ajpheart.00157.2011openalex-snapshot

Cardiac overexpression of 8-oxoguanine DNA glycosylase 1 protects mitochondrial DNA and reduces cardiac fibrosis following transaortic constriction

inferredcites →https://doi.org/10.1152/ajpheart.01162.2006openalex-snapshot

Thiol-based mechanisms of the thioredoxin and glutaredoxin systems: implications for diseases in the cardiovascular system

inferredcites →https://doi.org/10.1161/jaha.117.006114openalex-snapshot

Sirtuin 3 Deficiency Accelerates Hypertensive Cardiac Remodeling by Impairing Angiogenesis

inferredcites →https://doi.org/10.1186/ar4254openalex-snapshot

Linked decreases in liver kinase B1 and AMP-activated protein kinase activity modulate matrix catabolic responses to biomechanical injury in chondrocytes

inferredcites →https://doi.org/10.18632/oncotarget.20807openalex-snapshot

Metformin and berberine, two versatile drugs in treatment of common metabolic diseases

inferredcites →https://doi.org/10.18632/oncotarget.9717openalex-snapshot

Sirt3-mediated mitophagy protects tumor cells against apoptosis under hypoxia

inferredcites →https://doi.org/10.3389/fnagi.2013.00048openalex-snapshot

Forever young: SIRT3 a shield against mitochondrial meltdown, aging, and neurodegeneration

inferredcites →https://doi.org/10.3390/ijms160921486openalex-snapshot

The Role of Mitochondrial DNA in Mediating Alveolar Epithelial Cell Apoptosis and Pulmonary Fibrosis

inferredcites →https://doi.org/10.3390/ijms161125943openalex-snapshot

Chondrocyte Apoptosis in the Pathogenesis of Osteoarthritis

inferredcites →https://doi.org/10.4049/jimmunol.180.12.8378openalex-snapshot

Transamidation by Transglutaminase 2 Transforms S100A11 Calgranulin into a Procatabolic Cytokine for Chondrocytes

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4652901openalex-extracted

inferred← citeshttps://doi.org/10.1002/advs.202206144openalex-snapshot

Reprogramming of Mitochondrial Respiratory Chain Complex by Targeting SIRT3‐COX4I2 Axis Attenuates Osteoarthritis Progression

inferred← citeshttps://doi.org/10.1002/btm2.10187openalex-snapshot

Cartilage‐targeting ultrasmall lipid‐polymer hybrid nanoparticles for the prevention of cartilage degradation

inferred← citeshttps://doi.org/10.1002/jbmr.4721openalex-snapshot

Sirt3 Promotes Chondrogenesis, Chondrocyte Mitochondrial Respiration and the Development of High-Fat Diet-Induced Osteoarthritis in Mice

inferred← citeshttps://doi.org/10.1002/jcp.30319openalex-snapshot

SIRT3 mitigates intervertebral disc degeneration by delaying oxidative stress‐induced senescence of nucleus pulposus cells

inferred← citeshttps://doi.org/10.1002/med.22068openalex-snapshot

OGG1: An emerging multifunctional therapeutic target for the treatment of diseases caused by oxidative DNA damage

inferred← citeshttps://doi.org/10.1007/978-1-0716-1119-7_14openalex-snapshot

Mitochondrial Biogenesis, Activity, and DNA Isolation in Chondrocytes

inferred← citeshttps://doi.org/10.1007/978-3-030-04624-8_24openalex-snapshot

Nutraceuticals in Arthritis

inferred← citeshttps://doi.org/10.1007/s00018-020-03497-9openalex-snapshot

The role of SIRT3-mediated mitochondrial homeostasis in osteoarthritis

inferred← citeshttps://doi.org/10.1007/s00415-023-12062-wopenalex-snapshot

AMPK role in epilepsy: a promising therapeutic target?

inferred← citeshttps://doi.org/10.1007/s10067-019-04890-xopenalex-snapshot

Mitochondrial DNA haplogroups participate in osteoarthritis: current evidence based on a meta-analysis

inferred← citeshttps://doi.org/10.1007/s10735-026-10965-2openalex-snapshot

SIRT3-mediated DRP1 acetylation changes are associated with the restoration of mitochondrial function and alleviation of anterior cruciate ligament injury-induced osteoarthritis

inferred← citeshttps://doi.org/10.1007/s11010-023-04818-9openalex-snapshot

The role and intervention of mitochondrial metabolism in osteoarthritis

inferred← citeshttps://doi.org/10.1007/s11033-020-05900-xopenalex-snapshot

The role of AMPK in metabolism and its influence on DNA damage repair

inferred← citeshttps://doi.org/10.1007/s11033-026-12929-xopenalex-snapshot

Prolonged tadalafil exposure promotes skeletal muscle myogenesis and regulates early myogenic markers activation in C2C12 cell lines

inferred← citeshttps://doi.org/10.1007/s11914-019-00506-0openalex-snapshot

The Emerging Role of Glucose Metabolism in Cartilage Development

inferred← citeshttps://doi.org/10.1007/s12035-025-05576-zopenalex-snapshot

SIRT3/AMPK-Driven Mitophagy Failure Underlies Methylmercury-Induced Neurotoxicity: Evidence from Humans to Cells

inferred← citeshttps://doi.org/10.1016/j.archger.2022.104626openalex-snapshot

Downregulation of miR-3680–3p inhibits the progression of osteoarthritis via targeting OGG1

inferred← citeshttps://doi.org/10.1016/j.arr.2020.101249openalex-snapshot

The role of metabolism in chondrocyte dysfunction and the progression of osteoarthritis

inferred← citeshttps://doi.org/10.1016/j.arr.2021.101315openalex-snapshot

Mechanisms linking mitochondrial mechanotransduction and chondrocyte biology in the pathogenesis of osteoarthritis

inferred← citeshttps://doi.org/10.1016/j.arres.2025.100130openalex-snapshot

Global hotspots and prospective trends for chondrocyte metabolic changes and oxidative stress in osteoarthritis: A bibliometric analysis

inferred← citeshttps://doi.org/10.1016/j.bcp.2024.116723openalex-snapshot

7-Hydroxy-3-(4′-methoxyphenyl) coumarin (C12) attenuates bleomycin-induced acute lung injury and fibrosis through activation of SIRT3

inferred← citeshttps://doi.org/10.1016/j.biochi.2025.07.014openalex-snapshot

The role of mitochondrial DNA in bone/cartilage diseases

inferred← citeshttps://doi.org/10.1016/j.biopha.2023.115819openalex-snapshot

Harnessing the multifunctionality of lipid-based drug delivery systems for the local treatment of osteoarthritis

inferred← citeshttps://doi.org/10.1016/j.bone.2019.01.018openalex-snapshot

Sirtuins and FoxOs in osteoporosis and osteoarthritis

inferred← citeshttps://doi.org/10.1016/j.cellsig.2022.110309openalex-snapshot

Sirt3 ameliorates mitochondrial dysfunction and oxidative stress through regulating mitochondrial biogenesis and dynamics in cardiomyoblast.

inferred← citeshttps://doi.org/10.1016/j.cellsig.2025.112063openalex-snapshot

SIRT3 mitigates osteoarthritis by suppressing ferroptosis through activating AMPK signaling pathway

inferred← citeshttps://doi.org/10.1016/j.drudis.2023.103583openalex-snapshot

Role of SIRT3 in mitochondrial biology and its therapeutic implications in neurodegenerative disorders

inferred← citeshttps://doi.org/10.1016/j.ecoenv.2024.116360openalex-snapshot

Molecular crosstalk and putative mechanisms underlying mitochondrial quality control: The hidden link with methylmercury-induced cognitive impairment

inferred← citeshttps://doi.org/10.1016/j.fct.2026.116161openalex-snapshot

The TFAM-OGG1 axis mediates T-2 toxin-induced chondrocyte mitochondrial dysfunction and cartilage degeneration

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2022.08.038openalex-snapshot

LONP1 downregulation with ageing contributes to osteoarthritis via mitochondrial dysfunction

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2023.06.023openalex-snapshot

DMT1 differentially regulates mitochondrial complex activities to reduce glutathione loss and mitigate ferroptosis

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2024.10.299openalex-snapshot

IKKα affects the susceptibility of primary human osteoarthritis chondrocytes to oxidative stress-induced DNA damage by tuning autophagy

inferred← citeshttps://doi.org/10.1016/j.ijbiomac.2023.128270openalex-snapshot

Sirtuins in macrophage immune metabolism: A novel target for cardiovascular disorders

inferred← citeshttps://doi.org/10.1016/j.intimp.2022.108600openalex-snapshot

SIRT3-AMPK signaling pathway as a protective target in endothelial dysfunction of early sepsis

inferred← citeshttps://doi.org/10.1016/j.intimp.2024.111755openalex-snapshot

SIRT3 alleviates high glucose-induced chondrocyte injury through the promotion of autophagy and suppression of apoptosis in osteoarthritis progression

inferred← citeshttps://doi.org/10.1016/j.intimp.2026.117176openalex-snapshot

The potential mechanism investigation of Lipocalin-2 in ferroptosis via the AMPK/SIRT3/FOXO3a axis in sepsis-associated acute kidney injury

inferred← citeshttps://doi.org/10.1016/j.jep.2024.119294openalex-snapshot

Yanghe decoction alleviates osteoarthritis by AMPK-SIRT3 positive feedback loop-mediated mitochondrial autophagy

inferred← citeshttps://doi.org/10.1016/j.joca.2020.01.011openalex-snapshot

Role of TLR2 and TLR4 in regulation of articular chondrocyte homeostasis

inferred← citeshttps://doi.org/10.1016/j.joca.2021.10.004openalex-snapshot

Oral administration of berberine limits post-traumatic osteoarthritis development and associated pain via AMP-activated protein kinase (AMPK) in mice

inferred← citeshttps://doi.org/10.1016/j.joca.2021.10.009openalex-snapshot

Mitochondrial quality control in cartilage damage and osteoarthritis: new insights and potential therapeutic targets

inferred← citeshttps://doi.org/10.1016/j.joca.2022.05.005openalex-snapshot

Metformin as a potential disease-modifying drug in osteoarthritis: a systematic review of pre-clinical and human studies

inferred← citeshttps://doi.org/10.1016/j.joca.2023.11.002openalex-snapshot

Osteoarthritis year in review 2023: Biology

inferred← citeshttps://doi.org/10.1016/j.jot.2026.101100openalex-snapshot

Targeting mitochondrial dysfunction: Mitochondria transplantation as a regenerative strategy for musculoskeletal repair

inferred← citeshttps://doi.org/10.1016/j.lfs.2020.118602openalex-snapshot

Role of mitochondria in mediating chondrocyte response to mechanical stimuli

inferred← citeshttps://doi.org/10.1016/j.mad.2022.111708openalex-snapshot

Metformin mitigates stress-induced premature senescence by upregulating AMPKα at Ser485 phosphorylation induced SIRT3 expression and inactivating mitochondrial oxidants

inferred← citeshttps://doi.org/10.1016/j.phrs.2021.105497openalex-snapshot

Mechanistic insights into AMPK-SIRT3 positive feedback loop-mediated chondrocyte mitochondrial quality control in osteoarthritis pathogenesis

inferred← citeshttps://doi.org/10.1016/j.phymed.2024.155353openalex-snapshot

Isolation of Calenduloside E from Achyranthes bidentata Blume and its effects on LPS/D-GalN-induced acute liver injury in mice by regulating the AMPK-SIRT3 signaling pathway

inferred← citeshttps://doi.org/10.1016/j.phymed.2026.157959openalex-snapshot

Homoplantaginin ameliorates osteoarthritis by activating Sirt3/PINK1/Parkin signaling to promote mitophagy and attenuate inflammation in chondrocytes

inferred← citeshttps://doi.org/10.1016/j.prmcm.2025.100689openalex-snapshot

Targeting AMPK signaling: The therapeutic potential of berberine in diabetes and its complications

inferred← citeshttps://doi.org/10.1016/j.semarthrit.2019.03.008openalex-snapshot

The CNS theory of osteoarthritis: Opportunities beyond the joint

inferred← citeshttps://doi.org/10.1016/j.taap.2022.116179openalex-snapshot

Ononin alleviates endoplasmic reticulum stress in doxorubicin-induced cardiotoxicity by activating SIRT3

inferred← citeshttps://doi.org/10.1016/j.tcb.2025.08.006openalex-snapshot

The role of nutrient stress in DNA damage

inferred← citeshttps://doi.org/10.1021/acs.bioconjchem.5c00418openalex-snapshot

Bioorthogonal Probe BTD-Az Enables Sensitive and Rapid In Vivo Profiling of Protein Cysteine Sulfenylation

inferred← citeshttps://doi.org/10.1038/s41392-022-01257-8openalex-snapshot

The sirtuin family in health and disease

inferred← citeshttps://doi.org/10.1038/s41413-025-00460-xopenalex-snapshot

The impact of mitochondrial dysfunction on osteoarthritis cartilage: current insights and emerging mitochondria-targeted therapies

inferred← citeshttps://doi.org/10.1038/s41413-026-00548-yopenalex-snapshot

Exploring mitochondrial functions and dysfunctions in chondrocytes: toward the identification of novel therapies for osteoarthritis

inferred← citeshttps://doi.org/10.1038/s41467-024-55335-2openalex-snapshot

GATD3A-deficiency-induced mitochondrial dysfunction facilitates senescence of fibroblast-like synoviocytes and osteoarthritis progression

inferred← citeshttps://doi.org/10.1038/s41598-020-65423-0openalex-snapshot

Manipulating Sirtuin 3 pathway ameliorates renal damage in experimental diabetes

inferred← citeshttps://doi.org/10.1039/d4md00673aopenalex-snapshot

Therapeutic upregulation of DNA repair pathways: strategies and small molecule activators

inferred← citeshttps://doi.org/10.1080/14728222.2026.2737637openalex-snapshot

Sirtuin signaling as therapeutic targets of immunosenescence

inferred← citeshttps://doi.org/10.1080/15384101.2022.2044136openalex-snapshot

POLG mutations lead to abnormal mitochondrial remodeling during neural differentiation of human pluripotent stem cells via SIRT3/AMPK pathway inhibition

inferred← citeshttps://doi.org/10.1088/1748-605x/abee62openalex-snapshot

Polymer particles for the intra-articular delivery of drugs to treat osteoarthritis

inferred← citeshttps://doi.org/10.1089/wound.2024.0270openalex-snapshot

Chondrocyte Mitochondrial Quality Control: A Novel Insight into Osteoarthritis and Cartilage Regeneration

inferred← citeshttps://doi.org/10.1093/ijnp/pyaa008openalex-snapshot

Induced Ketosis as a Treatment for Neuroprogressive Disorders: Food for Thought?

inferred← citeshttps://doi.org/10.1093/jbmrpl/ziag004openalex-snapshot

USP34 attenuates cartilage degradation in temporomandibular joint osteoarthritis by ANT1-mediated mitophagy

inferred← citeshttps://doi.org/10.1093/lifemedi/lnaf019openalex-snapshot

Mitochondrial sirtuins, key regulators of aging

inferred← citeshttps://doi.org/10.1096/fj.201901900ropenalex-snapshot

Activation of AMP‐activated protein kinase during sepsis/inflammation improves survival by preserving cellular metabolic fitness

inferred← citeshttps://doi.org/10.1111/1750-3841.15625openalex-snapshot

Major royal jelly proteins prevents NAFLD by improving mitochondrial function and lipid accumulation through activating the AMPK / SIRT3 pathway in vitro

inferred← citeshttps://doi.org/10.1111/jcmm.14805openalex-snapshot

Celastrol exerts anti‐inflammatory effect in liver fibrosis via activation of AMPK‐SIRT3 signalling

inferred← citeshttps://doi.org/10.1126/sciadv.adp7872openalex-snapshot

Bioenergetic-active exosomes for cartilage regeneration and homeostasis maintenance

inferred← citeshttps://doi.org/10.1134/s1990750820020043openalex-snapshot

Type 2 Diabetes Mellitus in Osteoarthritic Patients: Does Association Between Metabolic Impairments, Joint Destruction, and Pain Exist?

inferred← citeshttps://doi.org/10.1155/2019/4616034openalex-snapshot

Stimulation of Na+/K+-ATPase with an Antibody against Its 4thExtracellular Region Attenuates Angiotensin II-Induced H9c2 Cardiomyocyte Hypertrophy via an AMPK/SIRT3/PPARγSignaling Pathway

inferred← citeshttps://doi.org/10.1155/2020/4143802openalex-snapshot

Insulin Resistance in Osteoarthritis: Similar Mechanisms to Type 2 Diabetes Mellitus

inferred← citeshttps://doi.org/10.1155/2020/7517219openalex-snapshot

Oleanolic Acid Decreases IL-1β-Induced Activation of Fibroblast-Like Synoviocytes via the SIRT3-NF-κB Axis in Osteoarthritis

inferred← citeshttps://doi.org/10.1155/2021/5712280openalex-snapshot

circFAM160A2 Promotes Mitochondrial Stabilization and Apoptosis Reduction in Osteoarthritis Chondrocytes by Targeting miR‐505‐3p and SIRT3

inferred← citeshttps://doi.org/10.1155/2022/5774641openalex-snapshot

OGG1 in the Kidney: Beyond Base Excision Repair

inferred← citeshttps://doi.org/10.1155/2023/1708251openalex-snapshot

Curcumin Ameliorates Age-Induced Tight Junction Impaired in Porcine Sertoli Cells by Inactivating the NLRP3 Inflammasome through the AMPK/SIRT3/SOD2/mtROS Signaling Pathway

inferred← citeshttps://doi.org/10.1159/000521720openalex-snapshot

Advanced Glycation End Products Induced Mitochondrial Dysfunction of Chondrocytes through Repression of AMPKα-SIRT1-PGC-1α Pathway

inferred← citeshttps://doi.org/10.1186/s12891-022-05911-8openalex-snapshot

Effects of mitochondrial dysfunction on bone metabolism and related diseases: a scientometric study from 2003 to 2022

inferred← citeshttps://doi.org/10.1186/s12964-022-00943-yopenalex-snapshot

HK2: a potential regulator of osteoarthritis via glycolytic and non-glycolytic pathways

inferred← citeshttps://doi.org/10.1186/s12964-026-02827-xopenalex-snapshot

The SDF-1α/CXCR4 axis regulates chondrocyte mitochondrial dynamics via the ERK/AMPKα pathway

inferred← citeshttps://doi.org/10.1186/s13020-025-01215-wopenalex-snapshot

Detumescence Analgesic Plaster mitigates knee osteoarthritis via active ingredients targeting mitochondrial complex 1/AMPK/MYL3-regulated cartilage homeostasis

inferred← citeshttps://doi.org/10.1186/s13075-022-02983-8openalex-snapshot

The role of the sirtuin family in cartilage and osteoarthritis: molecular mechanisms and therapeutic targets

inferred← citeshttps://doi.org/10.1186/s13075-025-03509-8openalex-snapshot

SDF-1 alleviates osteoarthritis by resolving mitochondrial dysfunction through the activation of the Sirt3/PGC-1α signalling pathway

inferred← citeshttps://doi.org/10.12182/20211160206openalex-snapshot

[Research Progress in Glucose Metabolism of Chondrocytes].

inferred← citeshttps://doi.org/10.14336/ad.2020.0702openalex-snapshot

Metformin: A Potential Candidate for Targeting Aging Mechanisms

inferred← citeshttps://doi.org/10.14336/ad.2021.1228openalex-snapshot

The Metabolic Landscape in Osteoarthritis

inferred← citeshttps://doi.org/10.15406/mseij.2023.07.00220openalex-snapshot

Nanotechnology-based drug delivery systems for treatment of knee injuries and Alzheimer’s disease – a review

inferred← citeshttps://doi.org/10.15406/mseij.7.4openalex-snapshot

inferred← citeshttps://doi.org/10.18097/pbmc20196506441openalex-snapshot

there any association of metabolic disturbances with joint destruction and pain?

inferred← citeshttps://doi.org/10.21203/rs.3.rs-1930373/v1openalex-snapshot

Effects of mitochondrial dysfunction on bone metabolism and related diseases: a scientometric study from 2003 to 2022

inferred← citeshttps://doi.org/10.2139/ssrn.5263791openalex-snapshot

In Vivo Profiling of Cysteine Sulfenylation Dynamics Links Redox Stress to Cartilage Degeneration in Aging Osteoarthritis

inferred← citeshttps://doi.org/10.2147/ccid.s420385openalex-snapshot

Erzhiwan Ameliorates Restraint Stress- and Monobenzone-Induced Depigmentation in Mice by Inhibiting Macrophage Migration Inhibitory Factor and 8-Hydroxy-2-Deoxyguanosine

inferred← citeshttps://doi.org/10.2147/jep.s237479openalex-snapshot

Experimental Therapeutics for the Treatment of Osteoarthritis

inferred← citeshttps://doi.org/10.32948/ajsep.2024.11.22openalex-snapshot

Molecular basis of senescence in osteoarthritis

inferred← citeshttps://doi.org/10.3389/fimmu.2023.1140653openalex-snapshot

Sirtuins in osteoarthritis: current understanding

inferred← citeshttps://doi.org/10.3389/fimmu.2026.1741730openalex-snapshot

The emerging role of histone acetylation in rheumatic diseases: unraveling mechanisms and therapeutic prospects

inferred← citeshttps://doi.org/10.3389/fimmu.2026.1823905openalex-snapshot

Signaling pathways and ion channels in osteoarthritis: a review of recent advances

inferred← citeshttps://doi.org/10.3389/fmed.2020.00506openalex-snapshot

Purinergic System Signaling in Metainflammation-Associated Osteoarthritis

inferred← citeshttps://doi.org/10.3389/fmed.2020.581402openalex-snapshot

Mitochondria: Potential Targets for Osteoarthritis

inferred← citeshttps://doi.org/10.3389/fonc.2025.1605085openalex-snapshot

Senescence-associated secretory phenotype in lung cancer: remodeling the tumor microenvironment for metastasis and immune suppression

inferred← citeshttps://doi.org/10.3389/fphar.2022.952560openalex-snapshot

The effects of metformin in the treatment of osteoarthritis: Current perspectives

inferred← citeshttps://doi.org/10.3389/fphar.2023.1169019openalex-snapshot

PGC-1α in osteoarthritic chondrocytes: From mechanism to target of action

inferred← citeshttps://doi.org/10.3389/fphar.2023.1248507openalex-snapshot

Role of SIRT3 in bone homeostasis and its application in preventing and treating bone diseases

inferred← citeshttps://doi.org/10.3389/fphar.2024.1488036openalex-snapshot

Antioxidant hydrogels for the treatment of osteoarthritis: mechanisms and recent advances

inferred← citeshttps://doi.org/10.3389/fphar.2025.1538662openalex-snapshot

Recent development of mitochondrial metabolism and dysfunction in osteoarthritis

inferred← citeshttps://doi.org/10.3389/fphar.2025.1681610openalex-snapshot

AMPK signaling in osteoarthritis: from mechanisms to targeted therapeutics

inferred← citeshttps://doi.org/10.3390/antiox10121972openalex-snapshot

Ginsenoside Rg3 Attenuates TNF-α-Induced Damage in Chondrocytes through Regulating SIRT1-Mediated Anti-Apoptotic and Anti-Inflammatory Mechanisms

inferred← citeshttps://doi.org/10.3390/antiox9090810openalex-snapshot

Irisin Mitigates Oxidative Stress, Chondrocyte Dysfunction and Osteoarthritis Development through Regulating Mitochondrial Integrity and Autophagy

inferred← citeshttps://doi.org/10.3390/biology11070996openalex-snapshot

Potential Methods of Targeting Cellular Aging Hallmarks to Reverse Osteoarthritic Phenotype of Chondrocytes

inferred← citeshttps://doi.org/10.3390/biomedicines10071477openalex-snapshot

The Role of Mitochondrial Metabolism, AMPK-SIRT Mediated Pathway, LncRNA and MicroRNA in Osteoarthritis

inferred← citeshttps://doi.org/10.3390/bios11020031openalex-snapshot

Biosensors for Detection of Biochemical Markers Relevant to Osteoarthritis

inferred← citeshttps://doi.org/10.3390/gucdd3030017openalex-snapshot

Follow the Molecule from Crystal Arthropathy to Comorbidities: The 2024 G-CAN Gold Medal Award Awardee Lecture

inferred← citeshttps://doi.org/10.3390/ijms25147740openalex-snapshot

Exploring Sirtuins: New Frontiers in Managing Heart Failure with Preserved Ejection Fraction

inferred← citeshttps://doi.org/10.34133/bmr.0402openalex-snapshot

A Comprehensive Reactive-Oxygen-Species-Scavenging Metal–Organic Framework Orchestrates the AMPK–DOT1L–H3K79me3 Cascade to Alleviate Osteoarthritis

inferred← citeshttps://doi.org/10.5582/bst.2018.01263openalex-snapshot

Protective effects of metformin against osteoarthritis through upregulation of SIRT3-mediated PINK1/Parkin-dependent mitophagy in primary chondrocytes

inferred← citeshttps://doi.org/10.6084/m9.figshare.21629346openalex-snapshot

Additional file 1 of Effects of mitochondrial dysfunction on bone metabolism and related diseases: a scientometric study from 2003 to 2022

inferred← citeshttps://doi.org/10.6084/m9.figshare.21629349openalex-snapshot

Additional file 2 of Effects of mitochondrial dysfunction on bone metabolism and related diseases: a scientometric study from 2003 to 2022

inferred← citeshttps://doi.org/10.6084/m9.figshare.21629352openalex-snapshot

Additional file 3 of Effects of mitochondrial dysfunction on bone metabolism and related diseases: a scientometric study from 2003 to 2022

inferred← citeshttps://doi.org/10.6084/m9.figshare.21629355openalex-snapshot

Additional file 4 of Effects of mitochondrial dysfunction on bone metabolism and related diseases: a scientometric study from 2003 to 2022

inferred← citeshttps://doi.org/10.6084/m9.figshare.21629361openalex-snapshot

Additional file 5 of Effects of mitochondrial dysfunction on bone metabolism and related diseases: a scientometric study from 2003 to 2022

inferred← citeshttps://doi.org/10.6084/m9.figshare.21629364openalex-snapshot

Additional file 6 of Effects of mitochondrial dysfunction on bone metabolism and related diseases: a scientometric study from 2003 to 2022

inferred← citeshttps://doi.org/10.7507/1002-1892.202109091openalex-snapshot

[The role of chondrocyte mitochondrial biogenesis in the pathogenesis of osteoarthritis].

inferred← citeshttps://doi.org/10.7507/1002-1892.202302056openalex-snapshot

[Research progress of optic atrophy 1-mediated mitochondrial dynamics in skeletal system diseases].

inferred← citeshttps://doi.org/10.7507/1002-1892.202303006openalex-snapshot

[Research progress on the role of chondrocyte mitochondrial homeostasis imbalance in the pathogenesis of osteoarthritis].

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.