On the public citation graph
Plasma insulin is required for the increase in plasma angiopoietin‐like protein 8 in response to nutrient ingestion
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/dmrr.2919openalex-snapshot
ANGPTL8 (betatrophin) role in diabetes and metabolic diseases
inferredcites →https://doi.org/10.1002/dmrr.3367openalex-snapshot
High TG to HDL ratio plays a significant role on atherosclerosis extension in prediabetes and newly diagnosed type 2 diabetes subjects
inferredcites →https://doi.org/10.1016/j.atherosclerosissup.2010.03.003openalex-snapshot
The mechanism of the formation and secretion of chylomicrons
inferredcites →https://doi.org/10.1016/j.cyto.2018.05.030openalex-snapshot
Serum Angiopoietin-like peptide 4 levels in patients with hepatic steatosis
inferredcites →https://doi.org/10.1016/j.jlr.2021.100068openalex-snapshot
ApoA5 lowers triglyceride levels via suppression of ANGPTL3/8-mediated LPL inhibition
inferredcites →https://doi.org/10.1016/j.jlr.2022.100198openalex-snapshot
An anti-ANGPTL3/8 antibody decreases circulating triglycerides by binding to a LPL-inhibitory leucine zipper-like motif
inferredcites →https://doi.org/10.1016/j.molmet.2017.06.014openalex-snapshot
ANGPTL8 promotes the ability of ANGPTL3 to bind and inhibit lipoprotein lipase
inferredcites →https://doi.org/10.1016/j.plipres.2020.101067openalex-snapshot
The multi-faces of Angptl8 in health and disease: Novel functions beyond lipoprotein lipase modulation
inferredcites →https://doi.org/10.1016/j.plipres.2021.101140openalex-snapshot
An updated ANGPTL3-4-8 model as a mechanism of triglyceride partitioning between fat and oxidative tissues
inferredcites →https://doi.org/10.1016/s0022-2828(03)00205-0openalex-snapshot
Evidence for rapid “metabolic switching” through lipoprotein lipase occupation of endothelial-binding sites
inferredcites →https://doi.org/10.1038/35080071openalex-snapshot
How proteins move lipids and lipids move proteins
inferredcites →https://doi.org/10.1038/ncpendmet1066openalex-snapshot
Dyslipidemia in type 2 diabetes mellitus
inferredcites →https://doi.org/10.1038/nrendo.2017.119openalex-snapshot
Angiopoietin-like 3 in lipoprotein metabolism
inferredcites →https://doi.org/10.1038/nrg2481openalex-snapshot
Plasma lipoproteins: genetic influences and clinical implications
inferredcites →https://doi.org/10.1038/srep10949openalex-snapshot
Higher plasma betatrophin/ANGPTL8 level in Type 2 Diabetes subjects does not correlate with blood glucose or insulin resistance
inferredcites →https://doi.org/10.1073/pnas.1217552109openalex-snapshot
Atypical angiopoietin-like protein that regulates ANGPTL3
inferredcites →https://doi.org/10.1073/pnas.1315292110openalex-snapshot
Mice lacking ANGPTL8 (Betatrophin) manifest disrupted triglyceride metabolism without impaired glucose homeostasis
inferredcites →https://doi.org/10.1080/21645515.2021.1888633openalex-snapshot
Exploring perception and hesitancy toward COVID-19 vaccine: A study from Jordan
inferredcites →https://doi.org/10.1098/rsob.150272openalex-snapshot
The ANGPTL3-4-8 model, a molecular mechanism for triglyceride trafficking
inferredcites →https://doi.org/10.1152/ajpendo.00084.2012openalex-snapshot
Identification of RIFL, a novel adipocyte-enriched insulin target gene with a role in lipid metabolism
inferredcites →https://doi.org/10.1152/ajpendo.00339.2019openalex-snapshot
Regulation of ANGPTL8 in liver and adipose tissue by nutritional and hormonal signals and its effect on glucose homeostasis in mice
inferredcites →https://doi.org/10.1161/circulationaha.113.002529openalex-snapshot
Association Between Plasma Triglycerides and High-Density Lipoprotein Cholesterol and Microvascular Kidney Disease and Retinopathy in Type 2 Diabetes Mellitus
inferredcites →https://doi.org/10.1186/1475-2840-12-17openalex-snapshot
Lipoprotein lipase links vitamin D, insulin resistance, and type 2 diabetes: a cross-sectional epidemiological study
inferredcites →https://doi.org/10.1186/s12933-016-0346-0openalex-snapshot
Circulating angiopoietin-like protein 8 (betatrophin) association with HsCRP and metabolic syndrome
inferredcites →https://doi.org/10.1194/jlr.m075689openalex-snapshot
ANGPTL8 requires ANGPTL3 to inhibit lipoprotein lipase and plasma triglyceride clearance
inferredcites →https://doi.org/10.1194/jlr.m088807openalex-snapshot
On the mechanism of angiopoietin-like protein 8 for control of lipoprotein lipase activity
inferredcites →https://doi.org/10.1194/jlr.ra120000781openalex-snapshot
Angiopoietin-like protein 8 differentially regulates ANGPTL3 and ANGPTL4 during postprandial partitioning of fatty acids
inferredcites →https://doi.org/10.1210/en.2005-0476openalex-snapshot
Transgenic Angiopoietin-Like (Angptl)4 Overexpression and Targeted Disruption of Angptl4 and Angptl3: Regulation of Triglyceride Metabolism
inferredcites →https://doi.org/10.1210/jc.2015-1254openalex-snapshot
Regulation of Angiopoietin-Like Proteins (ANGPTLs) 3 and 8 by Insulin
inferredcites →https://doi.org/10.1371/journal.pone.0170758openalex-snapshot
Relationship of Serum Betatrophin with Nonalcoholic Fatty Liver in a Chinese Population
inferredcites →https://doi.org/10.1515/cclm-2013-0358openalex-snapshot
Recent advances in physiological lipoprotein metabolism
inferredcites →https://doi.org/10.2337/db05-1200openalex-snapshot
Insulin Secretion and Action in Subjects With Impaired Fasting Glucose and Impaired Glucose Tolerance
inferredcites →https://doi.org/10.2337/dc05-2179openalex-snapshot
Contributions of β-Cell Dysfunction and Insulin Resistance to the Pathogenesis of Impaired Glucose Tolerance and Impaired Fasting Glucose
inferredcites →https://doi.org/10.2337/dc14-0602openalex-snapshot
Increased Circulating Levels of Betatrophin in Newly Diagnosed Type 2 Diabetic Patients
inferredcites →https://doi.org/10.3390/biom11071016openalex-snapshot
Lipoprotein Lipase and Its Delivery of Fatty Acids to the Heart
inferredcites →https://doi.org/10.3390/metabo11070425openalex-snapshot
Betatrophin Levels Are Related to the Early Histological Findings in Nonalcoholic Fatty Liver Disease
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4795161openalex-extracted
Adipose Angptl8 Controls Glucose and Energy Metabolism and its Suppression in Mice Improves Diet Induced Obesity and Inflammation
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2024.117545openalex-snapshot
Modification of lipoprotein metabolism and function driving atherogenesis in diabetes
inferred← citeshttps://doi.org/10.1016/j.bbalip.2024.159461openalex-snapshot
Silencing ANGPTL8 reduces mouse preadipocyte differentiation and insulin signaling
inferred← citeshttps://doi.org/10.1016/j.isci.2024.111292openalex-snapshot
Adipocyte Angptl8 deletion improves glucose and energy metabolism and obesity associated inflammation in mice
inferred← citeshttps://doi.org/10.1152/ajpendo.00162.2025openalex-snapshot
Obesity versus endurance exercise training: plasma triacylglycerol and muscle lipoprotein lipase activity at the crossroads of lipid accumulation in muscle
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