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Neuronal BIN1 Regulates Presynaptic Neurotransmitter Release and Memory Consolidation

https://doi.org/10.1016/j.celrep.2020.02.026

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What this work says

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Incoming claims: where this work appears in other papers’ reference lists and arguments.

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inferred← citeshttps://doi.org/10.1515/hsz-2025-0250openalex-snapshot

Endocytic scaffolds at the crossroads of Alzheimer’s disease and neuronal aging

inferred← citeshttps://doi.org/10.1523/eneuro.0304-25.2026openalex-snapshot

Parvalbumin Neuron–Targeted Loss of Alzheimer’s Disease Risk Gene BIN1 Is Insufficient to Drive Cognitive or Network Excitability Changes

inferred← citeshttps://doi.org/10.1523/jneurosci.0619-24.2024openalex-snapshot

Trans-synaptic Association of Vesicular Zinc Transporter 3 and Shank3 Supports Synapse-Specific Dendritic Spine Structure and Function in the Mouse Auditory Cortex

inferred← citeshttps://doi.org/10.15252/embr.202153877openalex-snapshot

FTLD-TDP assemblies seed neoaggregates with subtype-specific features via a prion-like cascade

inferred← citeshttps://doi.org/10.15252/emmm.202114745openalex-snapshot

LAG3 is not expressed in human and murine neurons and does not modulate α‐synucleinopathies

inferred← citeshttps://doi.org/10.17116/jnevro202612603145openalex-snapshot

BIN1 (amphiphysin 2) in the pathogenesis of cognitive impairment

inferred← citeshttps://doi.org/10.17116/patol20258706161openalex-snapshot

Amyloidogenesis and neurotrophic dysfunction in age-related macular degeneration in correlation with Alzheimer’s disease

inferred← citeshttps://doi.org/10.21203/rs.3.rs-7262443/v1openalex-snapshot

Microglial BIN1 deficiency elicits enhanced microglial inflammatory responses that mimic early AD pathology

inferred← citeshttps://doi.org/10.21248/gups.85791openalex-snapshot

Translational study on the role of genetic and prenatal risk factors in neurodevelopmental psychiatric disorders

inferred← citeshttps://doi.org/10.2174/0118715273264879231027070642openalex-snapshot

Role of Imaging Genetics in Alzheimer’s Disease: A Systematic Review and Current Update

inferred← citeshttps://doi.org/10.3389/fncel.2022.956029openalex-snapshot

Conditional deletion of MAD2B in forebrain neurons enhances hippocampus-dependent learning and memory in mice

inferred← citeshttps://doi.org/10.3389/fnins.2022.1023665openalex-snapshot

Integration of structural MRI and epigenetic analyses hint at linked cellular defects of the subventricular zone and insular cortex in autism: Findings from a case study

inferred← citeshttps://doi.org/10.3389/fnins.2022.858524openalex-snapshot

Secreted Amyloid Precursor Protein Alpha, a Neuroprotective Protein in the Brain Has Widespread Effects on the Transcriptome and Proteome of Human Inducible Pluripotent Stem Cell-Derived Glutamatergic Neurons Related to Memory Mechanisms

inferred← citeshttps://doi.org/10.3389/fnins.2023.1219299openalex-snapshot

Implication of tau propagation on neurodegeneration in Alzheimer’s disease

inferred← citeshttps://doi.org/10.3389/fpsyt.2021.753909openalex-snapshot

Association of Gene Polymorphisms in APOE and BIN1 With Dementia of Alzheimer's Type Susceptibility in Chinese Han Population

inferred← citeshttps://doi.org/10.3390/cells11203201openalex-snapshot

Amyloidogenesis and Neurotrophic Dysfunction in Alzheimer’s Disease: Do They have a Common Regulating Pathway?

inferred← citeshttps://doi.org/10.3390/ijms222212470openalex-snapshot

Digging Deeper: Advancements in Visualization of Inhibitory Synapses in Neurodegenerative Disorders

inferred← citeshttps://doi.org/10.3390/ijms24119683openalex-snapshot

The Cleavage-Specific Tau 12A12mAb Exerts an Anti-Amyloidogenic Action by Modulating the Endocytic and Bioenergetic Pathways in Alzheimer’s Disease Mouse Model

inferred← citeshttps://doi.org/10.3390/ijms26136460openalex-snapshot

Cognitive and Affective Dysregulation in Neuropathic Pain: Associated Hippocampal Remodeling and Microglial Activation

inferred← citeshttps://doi.org/10.3390/ijms262311314openalex-snapshot

Post-Operative Delirium in Elderly Patients: A Narrative Review

inferred← citeshttps://doi.org/10.3390/pathogens11090997openalex-snapshot

Amphiphysin AoRvs167-Mediated Membrane Curvature Facilitates Trap Formation, Endocytosis, and Stress Resistance in Arthrobotrys oligospora

inferred← citeshttps://doi.org/10.3788/cjl241349openalex-snapshot

单分子定位超分辨显微技术在神经生物学中的应用（特邀）

inferred← citeshttps://doi.org/10.4103/1673-5374.393106openalex-snapshot

Unveiling DNA methylation in Alzheimer’s disease: a review of array-based human brain studies

inferred← citeshttps://doi.org/10.4103/nrr.nrr-d-25-00243openalex-snapshot

Bridging autophagy and endolysosomal dysfunction: Role of bridging integrator 1 in Alzheimer’s disease

inferred← citeshttps://doi.org/10.6084/m9.figshare.16599510openalex-snapshot

Additional file 1 of Synaptic tau: A pathological or physiological phenomenon?

inferred← citeshttps://doi.org/10.6084/m9.figshare.18093090openalex-snapshot

Additional file 11 of The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

inferred← citeshttps://doi.org/10.6084/m9.figshare.18093093openalex-snapshot

Additional file 1 of The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

inferred← citeshttps://doi.org/10.6084/m9.figshare.18093096openalex-snapshot

Additional file 2 of The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

inferred← citeshttps://doi.org/10.6084/m9.figshare.18093099openalex-snapshot

Additional file 3 of The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

inferred← citeshttps://doi.org/10.6084/m9.figshare.18093102openalex-snapshot

Additional file 4 of The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

inferred← citeshttps://doi.org/10.6084/m9.figshare.18093105openalex-snapshot

Additional file 5 of The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

inferred← citeshttps://doi.org/10.6084/m9.figshare.18093108openalex-snapshot

Additional file 6 of The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

inferred← citeshttps://doi.org/10.6084/m9.figshare.18093111openalex-snapshot

Additional file 7 of The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

inferred← citeshttps://doi.org/10.6084/m9.figshare.18093117openalex-snapshot

Additional file 8 of The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

inferred← citeshttps://doi.org/10.64898/2026.02.03.703406openalex-snapshot

Synergistic retinal UCHL1 dysregulation and synaptic vulnerability reflect Alzheimer’s disease severity

inferred← citeshttps://doi.org/10.70675/5a5d3bacz6b6ez4a35z9159z6459b40efcdfopenalex-snapshot

Huntingtin functions in the regulation of axonal transport : consequences on neuronal network homeostasis and behavior, in health and disease

inferred← citeshttps://doi.org/10.7554/elife.57354openalex-snapshot

Alzheimer’s disease risk gene BIN1 induces Tau-dependent network hyperexcitability

inferred← citeshttps://doi.org/10.7554/elife.81011openalex-snapshot

Huntingtin recruits KIF1A to transport synaptic vesicle precursors along the mouse axon to support synaptic transmission and motor skill learning

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