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Vascular inflammation and atherogenesis are activated via receptors for PAMPs and suppressed by regulatory T cells

https://doi.org/10.1016/j.ddstr.2008.11.003

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inferredcites →https://doi.org/10.1161/hq0302.105360openalex-snapshot

Purinergic Signaling and Vascular Cell Proliferation and Death

inferredcites →https://doi.org/10.1161/hq1001.097780openalex-snapshot

Lipoprotein Size and Atherosclerosis Susceptibility in Apoe −/− and Ldlr −/− Mice

inferredcites →https://doi.org/10.1172/jci18607openalex-snapshot

Disruption of TGF-β signaling in T cells accelerates atherosclerosis

inferredcites →https://doi.org/10.1172/jci200423411openalex-snapshot

An integrated view of suppressor T cell subsets in immunoregulation

inferredcites →https://doi.org/10.1172/jci20479openalex-snapshot

IL-5 links adaptive and natural immunity specific for epitopes of oxidized LDL and protects from atherosclerosis

inferredcites →https://doi.org/10.1172/jci25482openalex-snapshot

Modulation of atherosclerosis in mice by Toll-like receptor 2

inferredcites →https://doi.org/10.1172/jci26390openalex-snapshot

Autophagy in cell death: an innocent convict?

inferredcites →https://doi.org/10.1172/jci27196openalex-snapshot

Platelets in inflammation and atherogenesis

inferredcites →https://doi.org/10.1177/000331970405500308openalex-snapshot

Atherogenic Effect of Interleukin-2 and Antiatherogenic Effect of Interleukin-2 Antibody in Apo-E-Deficient Mice

inferredcites →https://doi.org/10.1177/039463200702000109openalex-snapshot

HLA-A2.1-Restricted T Cells React to Serex-Defined Tumor Antigen CML66L and are Suppressed by CD4+CD25+ Regulatory T Cells

inferredcites →https://doi.org/10.1177/039463200802100401openalex-snapshot

CD25 high T Cells with a Prolonged Survival Inhibit Development of Diabetes

inferredcites →https://doi.org/10.1182/blood-2002-08-2606openalex-snapshot

Hyperhomocysteinemia accelerates atherosclerosis in cystathionine β-synthase and apolipoprotein E double knock-out mice with and without dietary perturbation

inferredcites →https://doi.org/10.1182/blood-2006-03-014126openalex-snapshot

Histone deacetylase inhibitors prevent exocytosis of interleukin-1β-containing secretory lysosomes: role of microtubules

inferredcites →https://doi.org/10.1182/blood-2006-04-019109openalex-snapshot

Transfer of differentiation signal by membrane microvesicles harboring hedgehog morphogens

inferredcites →https://doi.org/10.1182/blood-2007-06-096701openalex-snapshot

Homocysteine inhibits endothelial cell growth via DNA hypomethylation of the cyclin Agene

inferredcites →https://doi.org/10.1182/blood.v89.7.2429openalex-snapshot

Apoptotic Vascular Endothelial Cells Become Procoagulant

inferredcites →https://doi.org/10.1182/blood.v99.3.939openalex-snapshot

Cyclin A transcriptional suppression is the major mechanism mediating homocysteine-induced endothelial cell growth inhibition

inferredcites →https://doi.org/10.1186/1475-2840-5-4openalex-snapshot

Endothelial dysfunction: a comprehensive appraisal.

inferredcites →https://doi.org/10.1186/1475-2859-6-1openalex-snapshot

Polymyxin B as inhibitor of LPS contamination of Schistosoma mansoni recombinant proteins in human cytokine analysis.

inferredcites →https://doi.org/10.1186/ar414openalex-snapshot

The potential of human regulatory T cells generated ex vivo as a treatment for lupus and other chronic inflammatory diseases.

inferredcites →https://doi.org/10.1194/jlr.r400015-jlr200openalex-snapshot

Thematic review series: The Immune System and Atherogenesis. Paying the price for pathogen protection: toll receptors in atherogenesis

inferredcites →https://doi.org/10.1242/jcs.017897openalex-snapshot

The role of adherens junctions and VE-cadherin in the control of vascular permeability

inferredcites →https://doi.org/10.1373/clinchem.2007.097360openalex-snapshot

Inflammation in Atherosclerosis: From Vascular Biology to Biomarker Discovery and Risk Prediction

inferredcites →https://doi.org/10.1530/eje-08-0507openalex-snapshot

Improvement of endothelial function with metformin and rosiglitazone treatment in women with polycystic ovary syndrome

inferredcites →https://doi.org/10.1586/14779072.3.6.1111openalex-snapshot

Inflammation in coronary artery disease: potential role for immunomodulatory therapy

inferredcites →https://doi.org/10.2174/138161208783413248openalex-snapshot

Endothelial Cell Aging and Apoptosis in Prevention and Disease: E-Selectin Expression and Modulation As A Model

inferredcites →https://doi.org/10.2353/ajpath.2007.060857openalex-snapshot

Interleukin-4 Does Not Influence Development of Hypercholesterolemia or Angiotensin II-Induced Atherosclerotic Lesions in Mice

inferredcites →https://doi.org/10.2741/2775openalex-snapshot

Factors regulating apoptosis and homeostasis of CD4+CD25highFOXP3+ regulatory T cells are new therapeutic targets

inferredcites →https://doi.org/10.2741/2986openalex-snapshot

Roles of CD4+CD25high FOXP3+ Tregs in lymphomas and tumors are complex

inferredcites →https://doi.org/10.2741/3217openalex-snapshot

Higher expression of Bax in regulatory T cells increases vascular inflammation

inferredcites →https://doi.org/10.4049/jimmunol.171.9.4604openalex-snapshot

CD4+CD25+ T Cells Lyse Antigen-Presenting B Cells by Fas-Fas Ligand Interaction in an Epitope-Specific Manner

inferredcites →https://doi.org/10.4049/jimmunol.174.12.7711openalex-snapshot

Mouse Endothelial Cells Cross-Present Lymphocyte-Derived Antigen on Class I MHC via a TAP1- and Proteasome-Dependent Pathway

inferredcites →https://doi.org/10.4049/jimmunol.177.10.6991openalex-snapshot

Oligomerization of CXCL10 Is Necessary for Endothelial Cell Presentation and In Vivo Activity

inferredcites →https://doi.org/10.4049/jimmunol.177.10.7155openalex-snapshot

Lipopolysaccharide-Activated CD4+CD25+ T Regulatory Cells Inhibit Neutrophil Function and Promote Their Apoptosis and Death

inferredcites →https://doi.org/10.4049/jimmunol.178.10.6017openalex-snapshot

Immune Regulation by Microvascular Endothelial Cells: Directing Innate and Adaptive Immunity, Coagulation, and Inflammation

inferredcites →https://doi.org/10.4049/jimmunol.179.12.7993openalex-snapshot

COPs and POPs: Modulators of Inflammasome Activity

inferredcites →https://doi.org/10.4049/jimmunol.179.4.2551openalex-snapshot

IL-1 Receptor Accessory Protein and ST2 Comprise the IL-33 Receptor Complex

inferredcites →https://doi.org/10.7326/0003-4819-103-3-486_3openalex-snapshot

Cecil Textbook of Medicine.

inferredcites →https://doi.org/10.7326/0003-4819-114-9-812_1openalex-snapshot

Cecil Textbook of Medicine.

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4639349openalex-extracted

Protein-Rich Foods, Sea Foods, and Gut Microbiota Amplify Immune Responses in Chronic Diseases and Cancers – Targeting Perk as a Novel Therapeutic Strategy for Cancer

inferred← citeshttps://doi.org/10.1002/dni2.70011openalex-snapshot

Novel Organelle‐Based Intracellular Immunity With Mechanistic and Therapeutic Implications

inferred← citeshttps://doi.org/10.1002/inm3.70050openalex-snapshot

A New Paradigm: 46 Structural Cell Types Function as Environment‐Supporting Innate Immune Cells, With Endothelial and Vascular Smooth Muscle Cells as Key Prototypes

inferred← citeshttps://doi.org/10.1002/ptr.6980openalex-snapshot

Poria cocos polysaccharides reduces high‐fat diet‐induced arteriosclerosis in ApoE −/− mice by inhibiting inflammation

inferred← citeshttps://doi.org/10.1007/978-1-4419-1599-3_7openalex-snapshot

The Forkhead Transcription Factors Play Important Roles in Vascular Pathology and Immunology

inferred← citeshttps://doi.org/10.1007/978-3-031-62806-1_2openalex-snapshot

Cardiovascular Disease Risk Factors, Immune Checkpoints and Tregs

inferred← citeshttps://doi.org/10.1007/s00296-011-1840-zopenalex-snapshot

Coexistence of vasculitides with Familial Mediterranean Fever

inferred← citeshttps://doi.org/10.1007/s12265-015-9664-yopenalex-snapshot

Metabolic Diseases Downregulate the Majority of Histone Modification Enzymes, Making a Few Upregulated Enzymes Novel Therapeutic Targets—“Sand Out and Gold Stays”

inferred← citeshttps://doi.org/10.1007/s12265-016-9700-6openalex-snapshot

Lysophospholipid Receptors, as Novel Conditional Danger Receptors and Homeostatic Receptors Modulate Inflammation—Novel Paradigm and Therapeutic Potential

inferred← citeshttps://doi.org/10.1016/j.abb.2018.12.002openalex-snapshot

Proton leak regulates mitochondrial reactive oxygen species generation in endothelial cell activation and inflammation - A novel concept

inferred← citeshttps://doi.org/10.1016/j.cca.2017.11.005openalex-snapshot

Pyroptosis and its relationship to atherosclerosis

inferred← citeshttps://doi.org/10.1016/j.cyto.2017.06.003openalex-snapshot

IL-35, as a newly proposed homeostasis-associated molecular pattern, plays three major functions including anti-inflammatory initiator, effector, and blocker in cardiovascular diseases

inferred← citeshttps://doi.org/10.1016/j.intimp.2020.107170openalex-snapshot

Imperatorin reduces the inflammatory response of atherosclerosis by regulating MAPKs signaling pathway in vivo and in vitro

inferred← citeshttps://doi.org/10.1016/j.jlr.2023.100399openalex-snapshot

Associations between serum cholesterol and immunophenotypical characteristics of circulatory B cells and Tregs

inferred← citeshttps://doi.org/10.1016/j.pharmthera.2020.107715openalex-snapshot

Circular RNAs are a novel type of non-coding RNAs in ROS regulation, cardiovascular metabolic inflammations and cancers

inferred← citeshttps://doi.org/10.1016/j.pharmthera.2024.108604openalex-snapshot

Protein-rich foods, sea foods, and gut microbiota amplify immune responses in chronic diseases and cancers – Targeting PERK as a novel therapeutic strategy for chronic inflammatory diseases, neurodegenerative disorders, and cancer

inferred← citeshttps://doi.org/10.1016/j.preteyeres.2020.100906openalex-snapshot

Incomplete response to Anti-VEGF therapy in neovascular AMD: Exploring disease mechanisms and therapeutic opportunities

inferred← citeshttps://doi.org/10.1016/j.redox.2018.101095openalex-snapshot

HSP22 suppresses diabetes-induced endothelial injury by inhibiting mitochondrial reactive oxygen species formation

inferred← citeshttps://doi.org/10.1016/j.redox.2019.101221openalex-snapshot

Increased acetylation of H3K14 in the genomic regions that encode trained immunity enzymes in lysophosphatidylcholine-activated human aortic endothelial cells – Novel qualification markers for chronic disease risk factors and conditional DAMPs

inferred← citeshttps://doi.org/10.1016/j.redox.2019.101373openalex-snapshot

Anti-inflammatory cytokines IL-35 and IL-10 block atherogenic lysophosphatidylcholine-induced, mitochondrial ROS-mediated innate immune activation, but spare innate immune memory signature in endothelial cells

inferred← citeshttps://doi.org/10.1016/j.redox.2020.101696openalex-snapshot

ROS systems are a new integrated network for sensing homeostasis and alarming stresses in organelle metabolic processes

inferred← citeshttps://doi.org/10.1016/j.redox.2021.102142openalex-snapshot

Procaspase-1 patrolled to the nucleus of proatherogenic lipid LPC-activated human aortic endothelial cells induces ROS promoter CYP1B1 and strong inflammation

inferred← citeshttps://doi.org/10.1016/j.redox.2023.102771openalex-snapshot

Early hyperlipidemia triggers metabolomic reprogramming with increased SAH, increased acetyl-CoA-cholesterol synthesis, and decreased glycolysis

inferred← citeshttps://doi.org/10.1074/jbc.m113.460626openalex-snapshot

Identification of Novel Pretranslational Regulatory Mechanisms for NF-κB Activation

inferred← citeshttps://doi.org/10.1074/jbc.m115.641191openalex-snapshot

Inhibition of Caspase-1 Activation in Endothelial Cells Improves Angiogenesis

inferred← citeshttps://doi.org/10.1074/jbc.m115.663286openalex-snapshot

Interleukin-35 Inhibits Endothelial Cell Activation by Suppressing MAPK-AP-1 Pathway

inferred← citeshttps://doi.org/10.1074/jbc.m115.690081openalex-snapshot

Interleukin-17A Promotes Aortic Endothelial Cell Activation via Transcriptionally and Post-translationally Activating p38 Mitogen-activated Protein Kinase (MAPK) Pathway

inferred← citeshttps://doi.org/10.1089/ars.2017.7069openalex-snapshot

Lysophospholipids and Their Receptors Serve as Conditional DAMPs and DAMP Receptors in Tissue Oxidative and Inflammatory Injury

inferred← citeshttps://doi.org/10.1089/ars.2022.0087openalex-snapshot

Cigarette Smoke Modulates Inflammation and Immunity via Reactive Oxygen Species-Regulated Trained Immunity and Trained Tolerance Mechanisms

inferred← citeshttps://doi.org/10.1089/omi.2015.0128openalex-snapshot

Vasoregression: A Shared Vascular Pathology Underlying Macrovascular And Microvascular Pathologies?

inferred← citeshttps://doi.org/10.1139/cjpp-2016-0515openalex-snapshot

Mitochondrial ROS, uncoupled from ATP synthesis, determine endothelial activation for both physiological recruitment of patrolling cells and pathological recruitment of inflammatory cells

inferred← citeshttps://doi.org/10.1155/2010/459798openalex-snapshot

Inflammatory and Autoimmune Reactions in Atherosclerosis and Vaccine Design Informatics

inferred← citeshttps://doi.org/10.1155/2019/9483647openalex-snapshot

Dynamic Profile of CD4+ T-Cell-Associated Cytokines/Chemokines following Murine Myocardial Infarction/Reperfusion

inferred← citeshttps://doi.org/10.1155/2021/3928323openalex-snapshot

Hyperlipidemia May Synergize with Hypomethylation in Establishing Trained Immunity and Promoting Inflammation in NASH and NAFLD

inferred← citeshttps://doi.org/10.1155/2021/6664453openalex-snapshot

Ultrasound May Suppress Tumor Growth, Inhibit Inflammation, and Establish Tolerogenesis by Remodeling Innatome via Pathways of ROS, Immune Checkpoints, Cytokines, and Trained Immunity/Tolerance

inferred← citeshttps://doi.org/10.1155/2022/1433323openalex-snapshot

29 m6A-RNA Methylation (Epitranscriptomic) Regulators Are Regulated in 41 Diseases including Atherosclerosis and Tumors Potentially via ROS Regulation – 102 Transcriptomic Dataset Analyses

inferred← citeshttps://doi.org/10.1161/atvbaha.115.305282openalex-snapshot

Early Hyperlipidemia Promotes Endothelial Activation via a Caspase-1-Sirtuin 1 Pathway

inferred← citeshttps://doi.org/10.1161/atvbaha.120.314330openalex-snapshot

Vascular Endothelial Cells and Innate Immunity

inferred← citeshttps://doi.org/10.1172/jci.insight.152511openalex-snapshot

IL-35 promotes CD4+Foxp3+ Tregs and inhibits atherosclerosis via maintaining CCR5-amplified Treg-suppressive mechanisms

inferred← citeshttps://doi.org/10.1172/jci.insight.158183openalex-snapshot

Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways

inferred← citeshttps://doi.org/10.1172/jci.insight.177229openalex-snapshot

Caspase-4/11 promotes hyperlipidemia and chronic kidney disease–accelerated vascular inflammation by enhancing trained immunity

inferred← citeshttps://doi.org/10.1177/039463200902200208openalex-snapshot

Inflammasomes are Differentially Expressed in Cardiovascular and other Tissues

inferred← citeshttps://doi.org/10.1177/13872877251329583openalex-snapshot

Alzheimer's disease as an auto-innate immune pathology with potential cell trans-differentiation and enhanced trained immunity in 3xTg-AD mouse model

inferred← citeshttps://doi.org/10.1186/2050-490x-1-9openalex-snapshot

Endothelial progenitor cells in cardiovascular diseases: from biomarker to therapeutic agent

inferred← citeshttps://doi.org/10.1186/s13045-014-0080-6openalex-snapshot

Immunosuppressive/anti-inflammatory cytokines directly and indirectly inhibit endothelial dysfunction- a novel mechanism for maintaining vascular function

inferred← citeshttps://doi.org/10.1186/s13045-017-0440-0openalex-snapshot

Thrombus leukocytes exhibit more endothelial cell-specific angiogenic markers than peripheral blood leukocytes do in acute coronary syndrome patients, suggesting a possibility of trans-differentiation: a comprehensive database mining study

inferred← citeshttps://doi.org/10.1186/s13045-017-0504-1openalex-snapshot

Metabolism-associated danger signal-induced immune response and reverse immune checkpoint-activated CD40+ monocyte differentiation

inferred← citeshttps://doi.org/10.1186/s13045-017-0526-8openalex-snapshot

A comprehensive data mining study shows that most nuclear receptors act as newly proposed homeostasis-associated molecular pattern receptors

inferred← citeshttps://doi.org/10.1186/s41038-015-0001-0openalex-snapshot

Pathological conditions re-shape physiological Tregs into pathological Tregs

inferred← citeshttps://doi.org/10.14797/mdcj-12-3-134openalex-snapshot

Nucleic Acid Delivery for Endothelial Dysfunction in Cardiovascular Diseases

inferred← citeshttps://doi.org/10.17116/oftalma202213801190openalex-snapshot

Current view on the issue of insufficient effectiveness of anti-VEGF therapy for age-related macular degeneration

inferred← citeshttps://doi.org/10.2741/3657openalex-snapshot

Th 17 cells interplay with Foxp3+ Tregs in regulation of inflammation and autoimmunity

inferred← citeshttps://doi.org/10.2741/3901openalex-snapshot

Structural evidence of anti-atherogenic microRNAs

inferred← citeshttps://doi.org/10.2741/4055openalex-snapshot

Endothelial progenitor cells in atherosclerosis

inferred← citeshttps://doi.org/10.2741/4258openalex-snapshot

Amylin in vasodilation, energy expenditure and inflammation

inferred← citeshttps://doi.org/10.2741/4595openalex-snapshot

Uremic toxins are conditional danger- or homeostasis-associated molecular patterns

inferred← citeshttps://doi.org/10.2741/4710openalex-snapshot

Co-signaling receptors regulate T-cell plasticity and immune tolerance

inferred← citeshttps://doi.org/10.2741/e35openalex-snapshot

The FOX transcription factors regulate vascular pathology, diabetes and Tregs

inferred← citeshttps://doi.org/10.2741/e555openalex-snapshot

Defining the pathogenesis of inflammatory and immune diseases through database mining

inferred← citeshttps://doi.org/10.2741/s35openalex-snapshot

The FOX transcription factors regulate vascular pathology diabetes and Tregs

inferred← citeshttps://doi.org/10.3389/fcvm.2021.713170openalex-snapshot

Organelle Crosstalk Regulators Are Regulated in Diseases, Tumors, and Regulatory T Cells: Novel Classification of Organelle Crosstalk Regulators

inferred← citeshttps://doi.org/10.3389/fcvm.2021.773473openalex-snapshot

Novel Knowledge-Based Transcriptomic Profiling of Lipid Lysophosphatidylinositol-Induced Endothelial Cell Activation

inferred← citeshttps://doi.org/10.3389/fcvm.2022.892288openalex-snapshot

Editorial: Highlights for Cardiovascular Therapeutics in 2021 – Trained Immunity, Immunometabolism, Gender Differences of Cardiovascular Diseases, and Novel Targets of Cardiovascular Therapeutics

inferred← citeshttps://doi.org/10.3389/fcvm.2022.981544openalex-snapshot

Editorial: Insights in cardiovascular therapeutics: 2021 – cell death, cardiovascular injuries, and novel targets of cardiovascular therapeutics

inferred← citeshttps://doi.org/10.3389/fcvm.2023.1184030openalex-snapshot

Editorial: Insights in cardiovascular therapeutics 2022—cardiovascular innate immunity

inferred← citeshttps://doi.org/10.3389/fcvm.2024.1500775openalex-snapshot

Perspective: Pathological transdifferentiation—a novel therapeutic target for cardiovascular diseases and chronic inflammation

inferred← citeshttps://doi.org/10.3389/fcvm.2026.1782258openalex-snapshot

Reduced R-loop abundance at proinflammatory loci: a shared epigenetic mechanism in inflammatory and metabolic diseases

inferred← citeshttps://doi.org/10.3389/fimmu.2018.00045openalex-snapshot

GATA3, HDAC6, and BCL6 Regulate FOXP3+ Treg Plasticity and Determine Treg Conversion into Either Novel Antigen-Presenting Cell-Like Treg or Th1-Treg

inferred← citeshttps://doi.org/10.3389/fimmu.2020.554301openalex-snapshot

Approaching Inflammation Paradoxes—Proinflammatory Cytokine Blockages Induce Inflammatory Regulators

inferred← citeshttps://doi.org/10.3389/fimmu.2020.619951openalex-snapshot

A Novel Subset of CD95+ Pro-Inflammatory Macrophages Overcome miR155 Deficiency and May Serve as a Switch From Metabolically Healthy Obesity to Metabolically Unhealthy Obesity

inferred← citeshttps://doi.org/10.3389/fimmu.2020.632239openalex-snapshot

Tissue Treg Secretomes and Transcription Factors Shared With Stem Cells Contribute to a Treg Niche to Maintain Treg-Ness With 80% Innate Immune Pathways, and Functions of Immunosuppression and Tissue Repair

inferred← citeshttps://doi.org/10.3389/fimmu.2021.653110openalex-snapshot

Endothelial Immunity Trained by Coronavirus Infections, DAMP Stimulations and Regulated by Anti-Oxidant NRF2 May Contribute to Inflammations, Myelopoiesis, COVID-19 Cytokine Storms and Thromboembolism

inferred← citeshttps://doi.org/10.3389/fimmu.2021.678201openalex-snapshot

Canonical Secretomes, Innate Immune Caspase-1-, 4/11-Gasdermin D Non-Canonical Secretomes and Exosomes May Contribute to Maintain Treg-Ness for Treg Immunosuppression, Tissue Repair and Modulate Anti-Tumor Immunity via ROS Pathways

inferred← citeshttps://doi.org/10.3389/fimmu.2022.1035497openalex-snapshot

Editorial: Endothelial cells as innate immune cells

inferred← citeshttps://doi.org/10.3389/fimmu.2022.858256openalex-snapshot

Aorta in Pathologies May Function as an Immune Organ by Upregulating Secretomes for Immune and Vascular Cell Activation, Differentiation and Trans-Differentiation—Early Secretomes may Serve as Drivers for Trained Immunity

inferred← citeshttps://doi.org/10.3389/fimmu.2023.1113883openalex-snapshot

Caspase-11 promotes high-fat diet-induced NAFLD by increasing glycolysis, OXPHOS, and pyroptosis in macrophages

inferred← citeshttps://doi.org/10.3389/fimmu.2023.1268916openalex-snapshot

ER stress mediates Angiotensin II-augmented innate immunity memory and facilitates distinct susceptibilities of thoracic from abdominal aorta to aneurysm development

inferred← citeshttps://doi.org/10.3389/fimmu.2023.1348238openalex-snapshot

Innate immunity of vascular smooth muscle cells contributes to two-wave inflammation in atherosclerosis, twin-peak inflammation in aortic aneurysms and trans-differentiation potential into 25 cell types

inferred← citeshttps://doi.org/10.3389/fnagi.2021.827503openalex-snapshot

A Comorbid Rat Model of Neuroendocrine-Immune System Alterations Under the Impact of Risk Factors for Stroke

inferred← citeshttps://doi.org/10.3389/fphys.2017.00818openalex-snapshot

Low-Intensity Ultrasound-Induced Anti-inflammatory Effects Are Mediated by Several New Mechanisms Including Gene Induction, Immunosuppressor Cell Promotion, and Enhancement of Exosome Biogenesis and Docking

inferred← citeshttps://doi.org/10.3389/fphys.2018.00516openalex-snapshot

DNA Checkpoint and Repair Factors Are Nuclear Sensors for Intracellular Organelle Stresses—Inflammations and Cancers Can Have High Genomic Risks

inferred← citeshttps://doi.org/10.3390/cells11182810openalex-snapshot

Cigarette Smoke and Morphine Promote Treg Plasticity to Th17 via Enhancing Trained Immunity

inferred← citeshttps://doi.org/10.3390/cells12111482openalex-snapshot

Chronic Kidney Disease Transdifferentiates Veins into a Specialized Immune–Endocrine Organ with Increased MYCN-AP1 Signaling

inferred← citeshttps://doi.org/10.3390/md18120648openalex-snapshot

The Reducing Effects of Pyrogallol-Phloroglucinol-6,6-Bieckol on High-Fat Diet-Induced Pyroptosis in Endothelial and Vascular Smooth Muscle Cells of Mice Aortas

inferred← citeshttps://doi.org/10.3390/pathogens9110879openalex-snapshot

Liver Ischemia Reperfusion Injury, Enhanced by Trained Immunity, Is Attenuated in Caspase 1/Caspase 11 Double Gene Knockout Mice

inferred← citeshttps://doi.org/10.3748/wjg.v24.i41.4622openalex-snapshot

Damage-associated molecular patterns in inflammatory bowel disease: From biomarkers to therapeutic targets

inferred← citeshttps://doi.org/10.53941/ijddp.2024.100022openalex-snapshot

Immune Checkpoints Are New Therapeutic Targets in Regulating Cardio-, and Cerebro-Vascular Diseases and CD4+Foxp3+ Regulatory T Cell Immunosuppression

inferred← citeshttps://doi.org/10.5772/18673openalex-snapshot

Database Mining: Defining the Pathogenesis of Inflammatory and Immunological Diseases

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.