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Enhanced chemo-immunotherapy against melanoma by inhibition of cholesterol esterification in CD8+ T cells
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/s10549-009-0594-8openalex-snapshot
High ACAT1 expression in estrogen receptor negative basal-like breast cancer cells is associated with LDL-induced proliferation
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Potential Role of Acyl-Coenzyme A:Cholesterol Transferase (ACAT) Inhibitors as Hypolipidemic and Antiatherosclerosis Drugs
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A pH-responsive α-helical cell penetrating peptide-mediated liposomal delivery system
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Cholesteryl Ester Accumulation Induced by PTEN Loss and PI3K/AKT Activation Underlies Human Prostate Cancer Aggressiveness
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Chemotherapy and immunotherapy: mapping the road ahead
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Intravital Imaging Reveals Distinct Dynamics for Natural Killer and CD8+ T Cells during Tumor Regression
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T cell metabolic fitness in antitumor immunity
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The SREBP Pathway: Regulation of Cholesterol Metabolism by Proteolysis of a Membrane-Bound Transcription Factor
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Intranasal Vaccination against HIV-1 with Adenoviral Vector-Based Nanocomplex Using Synthetic TLR-4 Agonist Peptide as Adjuvant
inferredcites →https://doi.org/10.1038/emboj.2009.6openalex-snapshot
Accumulation of raft lipids in T‐cell plasma membrane domains engaged in TCR signalling
inferredcites →https://doi.org/10.1038/nature04399openalex-snapshot
Role of cholesterol and lipid organization in disease
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Ca2+ regulates T-cell receptor activation by modulating the charge property of lipids
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Potentiating the antitumour response of CD8+ T cells by modulating cholesterol metabolism
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Sterol regulatory element–binding proteins are essential for the metabolic programming of effector T cells and adaptive immunity
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Advances in targeting cell surface signalling molecules for immune modulation
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Cellular cholesterol trafficking and compartmentalization
inferredcites →https://doi.org/10.1038/onc.2016.168openalex-snapshot
Abrogating cholesterol esterification suppresses growth and metastasis of pancreatic cancer
inferredcites →https://doi.org/10.1038/sj.bjc.6604243openalex-snapshot
Combined expression of caveolin-1 and an activated AKT/mTOR pathway predicts reduced disease-free survival in clinically confined renal cell carcinoma
inferredcites →https://doi.org/10.1074/jbc.m112.386045openalex-snapshot
Cholesterol and Sphingomyelin Drive Ligand-independent T-cell Antigen Receptor Nanoclustering
inferredcites →https://doi.org/10.1074/jbc.m407941200openalex-snapshot
KLF11-mediated Repression Antagonizes Sp1/Sterol-responsive Element-binding protein-induced Transcriptional Activation of Caveolin-1 in Response to Cholesterol Signaling
inferredcites →https://doi.org/10.1084/jem.20042155openalex-snapshot
Coexistence of multivalent and monovalent TCRs explains high sensitivity and wide range of response
inferredcites →https://doi.org/10.1111/j.1365-2184.2011.00758.xopenalex-snapshot
Cholesterol esters as growth regulators of lymphocytic leukaemia cells
inferredcites →https://doi.org/10.1126/scitranslmed.aag2942openalex-snapshot
Anti–PD-1 antitumor immunity is enhanced by local and abrogated by systemic chemotherapy in GBM
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Immune Modulation in Cancer with Antibodies
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ACAT as a Drug Target for Alzheimer’s Disease
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ACAT Inhibitors: The Search for Novel Cholesterol Lowering Agents
inferredcites →https://doi.org/10.2353/ajpath.2006.050959openalex-snapshot
Elevated Levels of Cholesterol-Rich Lipid Rafts in Cancer Cells Are Correlated with Apoptosis Sensitivity Induced by Cholesterol-Depleting Agents
inferredcites →https://doi.org/10.7150/thno.16184openalex-snapshot
A New Concept of Enhancing Immuno-Chemotherapeutic Effects Against B16F10 Tumor via Systemic Administration by Taking Advantages of the Limitation of EPR Effect
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Cholesterol Metabolism-Mediated Immune Regulation and Cancer Immunotherapy
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Pharmacologic and genetic inhibition of cholesterol esterification enzymes reduces tumour burden: A systematic review and meta-analysis of preclinical models
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Avasimibe inhibits the proliferation, migration and invasion of glioma cells by suppressing linc00339
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Lipid metabolism in cancer: Exploring phospholipids as potential biomarkers
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Cholesterol metabolism in immune cells: From mechanisms to therapeutic opportunities
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Advances in traditional herbal formulation based nano-vaccine for cancer immunotherapy: Unraveling the enigma of complex tumor environment and multidrug resistance
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Peptide-functionalized liposomes as therapeutic and diagnostic tools for cancer treatment
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Progress in the polymer-paclitaxel conjugate
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A novel polyphyllin I-based liposome delivery system sensitizes hepatic carcinoma to doxorubicin via cholesterol modulation
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Cholesterol acyltransferase-1: A novel diagnostic biomarker and potential therapeutic target for cancer
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Charge-based immunoreceptor signalling in health and disease
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Cholesterol metabolism in cancer: mechanisms and therapeutic opportunities
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Cholesterol Metabolism in Cancer and Cell Death
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Lipid metabolism in tumor-infiltrating T cells: mechanisms and applications
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High-affinity SOAT1 ligands remodeled cholesterol metabolism program to inhibit tumor growth
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Nanoparticle-mediated synergistic chemoimmunotherapy for tailoring cancer therapy: recent advances and perspectives
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ACAT-1-Regulated Cholesteryl Ester Accumulation Modulates Gemcitabine Resistance in Biliary Tract Cancer
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Atorvastatin Improves Cisplatin Sensitivity Through Modulation of Cholesteryl Ester Homeostasis in Breast Cancer Cells
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Atorvastatin Improves Cisplatin Sensitivity Through Modulation of Cholesteryl Ester Homeostasis in Breast Cancer Cells
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Regulating Cholesterol in Tumorigenesis: A Novel Paradigm for Tumor Nanotherapeutics
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Cholesterol Metabolism as a Potential Therapeutic Target and a Prognostic Biomarker for Cancer Immunotherapy
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Development and Validation of a Cholesterol-related Gene Signature for Prognostic Assessment in Head and Neck Squamous Cell Carcinoma
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Metabolic Traits in Cutaneous Melanoma
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Cholesteryl Ester Promotes Mammary Tumor Growth in MMTV-PyMT Mice and Activates Akt-mTOR Pathway in Tumor Cells
inferred← citeshttps://doi.org/10.3390/cancers12071733openalex-snapshot
RORα Regulates Cholesterol Metabolism of CD8+ T Cells for Anticancer Immunity
inferred← citeshttps://doi.org/10.3390/cancers15051397openalex-snapshot
PCSK9 Inhibitors in Cancer Patients Treated with Immune-Checkpoint Inhibitors to Reduce Cardiovascular Events: New Frontiers in Cardioncology
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Reprogramming T-Cell Metabolism for Better Anti-Tumor Immunity
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Modulating Cholesterol Metabolism via ACAT1 Knockdown Enhances Anti-B-Cell Lymphoma Activities of CD19-Specific Chimeric Antigen Receptor T Cells by Improving the Cell Activation and Proliferation
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Additional file 1 of High-affinity SOAT1 ligands remodeled cholesterol metabolism program to inhibit tumor growth
inferred← citeshttps://doi.org/10.6084/m9.figshare.20454544openalex-snapshot
Additional file 2 of High-affinity SOAT1 ligands remodeled cholesterol metabolism program to inhibit tumor growth
inferred← citeshttps://doi.org/10.6084/m9.figshare.20454547openalex-snapshot
Additional file 3 of High-affinity SOAT1 ligands remodeled cholesterol metabolism program to inhibit tumor growth
inferred← citeshttps://doi.org/10.6084/m9.figshare.20454550openalex-snapshot
Additional file 4 of High-affinity SOAT1 ligands remodeled cholesterol metabolism program to inhibit tumor growth
inferred← citeshttps://doi.org/10.6084/m9.figshare.20454553openalex-snapshot
Additional file 5 of High-affinity SOAT1 ligands remodeled cholesterol metabolism program to inhibit tumor growth
inferred← citeshttps://doi.org/10.6084/m9.figshare.20454556openalex-snapshot
Additional file 6 of High-affinity SOAT1 ligands remodeled cholesterol metabolism program to inhibit tumor growth
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Perturbing plasma membrane lipid: a new paradigm for tumor nanotherapeutics
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