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Chronic inflammation induces telomere dysfunction and accelerates ageing in mice
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
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Tumor Necrosis Factor Receptor 1 Gain-of-Function Mutation Aggravates Nonalcoholic Fatty Liver Disease but Does Not Cause Insulin Resistance in a Murine Model
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Preferential Accumulation of Single-Stranded Regions in Telomeres of Human Fibroblasts
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Anti-Inflammatory Drugs in the 21st Century
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Molecular inflammation: Underpinnings of aging and age-related diseases
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Cellular senescence involves an intracrine prostaglandin E2 pathway in human fibroblasts
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The effect of non-steroidal anti-inflammatory agents on behavioural changes and cytokine production following systemic inflammation: Implications for a role of COX-1
inferredcites →https://doi.org/10.1016/j.cell.2008.03.038openalex-snapshot
Chemokine Signaling via the CXCR2 Receptor Reinforces Senescence
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inferredcites →https://doi.org/10.1016/j.cell.2008.09.034openalex-snapshot
Telomerase Reverse Transcriptase Delays Aging in Cancer-Resistant Mice
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Factors that influence telomeric oxidative base damage and repair by DNA glycosylase OGG1
inferredcites →https://doi.org/10.1016/j.exger.2010.01.018openalex-snapshot
Quantitative assessment of markers for cell senescence
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Growth Hormone Stimulates Proliferation of Old–Aged Regenerating Liver Through Forkhead Box M1b
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The NF-κB p50:p50:HDAC-1 repressor complex orchestrates transcriptional inhibition of multiple pro-inflammatory genes
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Inflammaging and anti-inflammaging: A systemic perspective on aging and longevity emerged from studies in humans
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Gross energy metabolism in mice under late onset, short term caloric restriction
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Telomere dysfunction induces metabolic and mitochondrial compromise
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Hypothalamic programming of systemic ageing involving IKK-β, NF-κB and GnRH
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Oxygen sensitivity severely limits the replicative lifespan of murine fibroblasts
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Telomeric DNA damage is irreparable and causes persistent DNA-damage-response activation
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Telomerase directly regulates NF-κB-dependent transcription
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Cdkn1a deletion improves stem cell function and lifespan of mice with dysfunctional telomeres without accelerating cancer formation
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Group III secreted phospholipase A2 transgenic mice spontaneously develop inflammation
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Intestinal Transport During the Life Span of the Mouse
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Nuclear lamina defects cause ATM-dependent NF-κB activation and link accelerated aging to a systemic inflammatory response
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inferredcites →https://doi.org/10.1111/acel.12068openalex-snapshot
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inferredcites →https://doi.org/10.1111/j.1474-9726.2008.00414.xopenalex-snapshot
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inferredcites →https://doi.org/10.1111/j.1474-9726.2009.00481.xopenalex-snapshot
DNA damage response and cellular senescence in tissues of aging mice
inferredcites →https://doi.org/10.1111/j.1474-9726.2011.00702.xopenalex-snapshot
Fat maintenance is a predictor of the murine lifespan response to dietary restriction
inferredcites →https://doi.org/10.1111/j.1474-9726.2012.00795.xopenalex-snapshot
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inferredcites →https://doi.org/10.1111/j.1474-9726.2012.00870.xopenalex-snapshot
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Cellular Senescence in Aging Primates
inferredcites →https://doi.org/10.1159/000447068openalex-snapshot
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inferredcites →https://doi.org/10.1172/jci45785openalex-snapshot
NF-κB inhibition delays DNA damage–induced senescence and aging in mice
inferredcites →https://doi.org/10.1172/jci64098openalex-snapshot
Cellular senescence and the senescent secretory phenotype: therapeutic opportunities
inferredcites →https://doi.org/10.1186/2046-2395-1-3openalex-snapshot
Male mice retain a metabolic memory of improved glucose tolerance induced during adult onset, short-term dietary restriction
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A guide to defining and implementing protocols for the welfare assessment of laboratory animals: eleventh report of the BVAAWF/FRAME/RSPCA/UFAW Joint Working Group on Refinement
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Mitochondrial Dysfunction Accounts for the Stochastic Heterogeneity in Telomere-Dependent Senescence
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Genetic Determinants of Human Health Span and Life Span: Progress and New Opportunities
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DNA damage foci in mitosis are devoid of 53BP1
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Age-related biochemical profiling reveals multi-system physiological decline in the annual fish Nothobranchius guentheri
inferred← citeshttps://doi.org/10.1007/s11010-017-3200-0openalex-snapshot
Apoptosis and expression of apoptosis-related genes in mouse intestinal tissue after whole-body proton exposure
inferred← citeshttps://doi.org/10.1007/s11010-023-04878-xopenalex-snapshot
NRF2 signaling pathway and telomere length in aging and age-related diseases
inferred← citeshttps://doi.org/10.1007/s11357-021-00343-zopenalex-snapshot
IL-6 can singlehandedly drive many features of frailty in mice
inferred← citeshttps://doi.org/10.1007/s11357-024-01121-3openalex-snapshot
RelA-mediated signaling connects adaptation to chronic cardiomyocyte stress with myocardial and systemic inflammation in the ADCY8 model of accelerated aging
inferred← citeshttps://doi.org/10.1007/s11357-025-01818-zopenalex-snapshot
Chronic inflammation mediates the relationship between physical activity and telomere length
inferred← citeshttps://doi.org/10.1007/s12602-019-09576-zopenalex-snapshot
Cell-Free Culture Supernatant of Probiotic Lactobacillus fermentum Protects Against H2O2-Induced Premature Senescence by Suppressing ROS-Akt-mTOR Axis in Murine Preadipocytes
inferred← citeshttps://doi.org/10.1007/s12672-024-01655-0openalex-snapshot
Identification and construction of prognostic clusters and risk-prognosis model based on aging-immune related genes in bladder cancer
inferred← citeshttps://doi.org/10.1007/s13770-017-0097-3openalex-snapshot
Endogenous Stem Cells in Homeostasis and Aging
inferred← citeshttps://doi.org/10.1007/s40139-015-0071-5openalex-snapshot
Stem Cell Senescence as the Memory of Past Injuries
inferred← citeshttps://doi.org/10.1007/s40256-023-00602-8openalex-snapshot
SGLT2 Inhibitors in Aging-Related Cardiovascular Disease: A Review of Potential Mechanisms
inferred← citeshttps://doi.org/10.1007/s40501-015-0046-5openalex-snapshot
Staging Models and Functional Outcomes in Bipolar Disorder: Clinical Implications
inferred← citeshttps://doi.org/10.1007/s40520-025-03046-zopenalex-snapshot
Causal effect between telomere length and thirteen types of cancer in Asian population: a bidirectional Mendelian randomization study
inferred← citeshttps://doi.org/10.1007/s40520-025-03116-2openalex-snapshot
Association between daily sitting time and accelerated aging in women: double mediation effects of systemic immune-inflammation index and creatinine
inferred← citeshttps://doi.org/10.1016/b978-0-12-801816-3.00035-2openalex-snapshot
Mechanisms of Action of Curcumin on Aging
inferred← citeshttps://doi.org/10.1016/b978-0-12-801816-3.00043-1openalex-snapshot
Vitamin C, Antioxidant Status, and Cardiovascular Aging
inferred← citeshttps://doi.org/10.1016/b978-0-12-809479-2.00011-1openalex-snapshot
Big Mechanisms of Aging via System Identification and Big Database Mining
inferred← citeshttps://doi.org/10.1016/b978-0-12-815962-0.00003-2openalex-snapshot
Mechanisms of cell senescence in aging
inferred← citeshttps://doi.org/10.1016/b978-0-12-818649-7.00032-1openalex-snapshot
Mediterranean diet, inflammation, and telomere length maintenance
inferred← citeshttps://doi.org/10.1016/b978-0-12-819182-8.00028-4openalex-snapshot
Bipolar disorder and accelerated aging: Shared mechanisms and implications
inferred← citeshttps://doi.org/10.1016/b978-0-12-820071-1.00018-9openalex-snapshot
Therapeutic approaches for the treatment of aging-induced stem cell dysfunction
inferred← citeshttps://doi.org/10.1016/b978-0-12-822514-1.00005-5openalex-snapshot
Cellular senescence during aging and chronic liver diseases
inferred← citeshttps://doi.org/10.1016/b978-0-12-822514-1.00013-4openalex-snapshot
Cell senescence is a cause of frailty
inferred← citeshttps://doi.org/10.1016/b978-0-12-823761-8.00017-3openalex-snapshot
Cellular senescence during aging
inferred← citeshttps://doi.org/10.1016/b978-0-12-823761-8.00029-xopenalex-snapshot
Telomeres and cell homeostasis in aging
inferred← citeshttps://doi.org/10.1016/bs.acr.2021.02.001openalex-snapshot
Autophagy and senescence, converging roles in pathophysiology as seen through mouse models
inferred← citeshttps://doi.org/10.1016/bs.ircmb.2016.04.003openalex-snapshot
NF-κB signaling as a driver of ageing
inferred← citeshttps://doi.org/10.1016/bs.ircmb.2018.05.006openalex-snapshot
Mitochondria and Reactive Oxygen Species in Aging and Age-Related Diseases
inferred← citeshttps://doi.org/10.1016/bs.irn.2020.03.019openalex-snapshot
Cellular senescence in aging and age-related diseases: Implications for neurodegenerative diseases
inferred← citeshttps://doi.org/10.1016/bs.pmbts.2015.08.002openalex-snapshot
Understanding the Roles of the NF-κB Pathway in Regulatory T Cell Development, Differentiation and Function
inferred← citeshttps://doi.org/10.1016/j.actbio.2024.01.044openalex-snapshot
Mesoporous bioactive glass scaffolds for the delivery of bone marrow stem cell-derived osteoinductive extracellular vesicles lncRNA promote senescent bone defect repair by targeting the miR-1843a-5p/Mob3a/YAP axis
inferred← citeshttps://doi.org/10.1016/j.ajpath.2020.10.013openalex-snapshot
Modulation of Oxidative Stress and Inflammation in the Aged Lacrimal Gland
inferred← citeshttps://doi.org/10.1016/j.arr.2015.07.005openalex-snapshot
DAMPs and influenza virus infection in ageing
inferred← citeshttps://doi.org/10.1016/j.arr.2015.11.006openalex-snapshot
Ageing and the telomere connection: An intimate relationship with inflammation
inferred← citeshttps://doi.org/10.1016/j.arr.2016.12.004openalex-snapshot
HIV-associated cellular senescence: A contributor to accelerated aging
inferred← citeshttps://doi.org/10.1016/j.arr.2017.05.006openalex-snapshot
Vascular aging: Molecular mechanisms and potential treatments for vascular rejuvenation
inferred← citeshttps://doi.org/10.1016/j.arr.2018.09.002openalex-snapshot
Telomere Length and All-Cause Mortality: A Meta-analysis
inferred← citeshttps://doi.org/10.1016/j.arr.2020.101027openalex-snapshot
Exploiting the telomere machinery to put the brakes on inflamm-aging
inferred← citeshttps://doi.org/10.1016/j.arr.2020.101231openalex-snapshot
Thymus involution sets the clock of the aging T-cell landscape: Implications for declined immunity and tissue repair
inferred← citeshttps://doi.org/10.1016/j.arr.2021.101374openalex-snapshot
miR-21 and miR-146a: The microRNAs of inflammaging and age-related diseases
inferred← citeshttps://doi.org/10.1016/j.arr.2021.101456openalex-snapshot
How good is the evidence that cellular senescence causes skin ageing?
inferred← citeshttps://doi.org/10.1016/j.arr.2022.101659openalex-snapshot
Frailty in rodents: Models, underlying mechanisms, and management
inferred← citeshttps://doi.org/10.1016/j.arr.2022.101687openalex-snapshot
SARS-CoV-2 and the central nervous system: Emerging insights into hemorrhage-associated neurological consequences and therapeutic considerations
inferred← citeshttps://doi.org/10.1016/j.arr.2023.101943openalex-snapshot
Exploring cellular senescence in the musculoskeletal system: Any insights for biomarkers discovery?
inferred← citeshttps://doi.org/10.1016/j.arr.2023.102083openalex-snapshot
A new face of old cells: An overview about the role of senescence and telomeres in inflammatory bowel diseases
inferred← citeshttps://doi.org/10.1016/j.arr.2024.102558openalex-snapshot
Oxidative stress and cell senescence as drivers of ageing: Chicken and egg
inferred← citeshttps://doi.org/10.1016/j.arr.2024.102653openalex-snapshot
The immunosenescence clock: A new method for evaluating biological age and predicting mortality risk
inferred← citeshttps://doi.org/10.1016/j.arr.2025.102760openalex-snapshot
The context-dependent effect of cellular senescence: From embryogenesis and wound healing to aging
inferred← citeshttps://doi.org/10.1016/j.arr.2026.103108openalex-snapshot
S100A8/A9 as a central hub in inflammaging: Cross-system mechanisms
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2017.03.023openalex-snapshot
Accelerated cellular senescence as underlying mechanism for functionally impaired bone marrow-derived progenitor cells in ischemic heart disease
inferred← citeshttps://doi.org/10.1016/j.bbadis.2018.01.018openalex-snapshot
Metformin regulates mitochondrial biogenesis and senescence through AMPK mediated H3K79 methylation: Relevance in age-associated vascular dysfunction
inferred← citeshttps://doi.org/10.1016/j.bbadis.2018.06.010openalex-snapshot
Telomere length alterations in microsatellite stable colorectal cancer and association with the immune response
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2020.118643openalex-snapshot
Oxidative stress induces transcription of telomeric repeat-containing RNA (TERRA) by engaging PKA signaling and cytoskeleton dynamics
inferred← citeshttps://doi.org/10.1016/j.bbi.2015.08.024openalex-snapshot
Partial sleep deprivation activates the DNA damage response (DDR) and the senescence-associated secretory phenotype (SASP) in aged adult humans
inferred← citeshttps://doi.org/10.1016/j.bbi.2019.04.021openalex-snapshot
Maternal pro-inflammatory state during pregnancy and newborn leukocyte telomere length: A prospective investigation
inferred← citeshttps://doi.org/10.1016/j.bbrc.2025.151677openalex-snapshot
Long-term aerobic exercise enhances liver health: miRNA regulation and oxidative stress alleviation
inferred← citeshttps://doi.org/10.1016/j.bbrc.2025.152881openalex-snapshot
Deciphering the emerging interrelationship between cellular senescence and extracellular vesicles: Implications for therapy and diagnosis
inferred← citeshttps://doi.org/10.1016/j.bioadv.2026.215196openalex-snapshot
The application of engineered exosome-mimetic lipid nanoparticles in the hepatic rejuvenation of aged donor livers
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2020.119997openalex-snapshot
Catcher in the rel: Nanoparticles-antibody conjugate as NF-κB nuclear translocation blocker
inferred← citeshttps://doi.org/10.1016/j.biopha.2024.116415openalex-snapshot
Tetramethylpyrazine Nitrone alleviates D-galactose-induced murine skeletal muscle aging and motor deficits by activating the AMPK signaling pathway
inferred← citeshttps://doi.org/10.1016/j.bone.2019.01.015openalex-snapshot
Cellular senescence in bone
inferred← citeshttps://doi.org/10.1016/j.canlet.2023.216459openalex-snapshot
Telomerase and hallmarks of cancer: An intricate interplay governing cancer cell evolution
inferred← citeshttps://doi.org/10.1016/j.cardfail.2015.06.013openalex-snapshot
Ethanol-Associated Cardiomyocyte Apoptosis and Left Ventricular Dilation Are Unrelated to Changes in Myocardial Telomere Length in Rats
inferred← citeshttps://doi.org/10.1016/j.ccell.2016.09.003openalex-snapshot
Distinct Functions of Senescence-Associated Immune Responses in Liver Tumor Surveillance and Tumor Progression
inferred← citeshttps://doi.org/10.1016/j.ccell.2018.06.007openalex-snapshot
PTBP1-Mediated Alternative Splicing Regulates the Inflammatory Secretome and the Pro-tumorigenic Effects of Senescent Cells
inferred← citeshttps://doi.org/10.1016/j.cell.2019.10.005openalex-snapshot
Cellular Senescence: Defining a Path Forward
inferred← citeshttps://doi.org/10.1016/j.cell.2024.05.059openalex-snapshot
Guidelines for minimal information on cellular senescence experimentation in vivo
inferred← citeshttps://doi.org/10.1016/j.cell.2024.08.021openalex-snapshot
Metformin decelerates aging clock in male monkeys
inferred← citeshttps://doi.org/10.1016/j.celrep.2015.03.069openalex-snapshot
DNA-Damage-Induced Type I Interferon Promotes Senescence and Inhibits Stem Cell Function
inferred← citeshttps://doi.org/10.1016/j.celrep.2017.02.056openalex-snapshot
Canonical Wnt Signaling Ameliorates Aging of Intestinal Stem Cells
inferred← citeshttps://doi.org/10.1016/j.celrep.2021.108779openalex-snapshot
Inflammation-driven senescence-associated secretory phenotype in cancer-associated fibroblasts enhances peritoneal dissemination
inferred← citeshttps://doi.org/10.1016/j.celrep.2021.109098openalex-snapshot
Persistent NF-κB activation in muscle stem cells induces proliferation-independent telomere shortening
inferred← citeshttps://doi.org/10.1016/j.celrep.2023.113656openalex-snapshot
Oxidative guanine base damage plays a dual role in regulating productive ALT-associated homology-directed repair
inferred← citeshttps://doi.org/10.1016/j.celrep.2024.114496openalex-snapshot
Citrate metabolism controls the senescent microenvironment via the remodeling of pro-inflammatory enhancers
inferred← citeshttps://doi.org/10.1016/j.chemosphere.2022.134633openalex-snapshot
Long-term respiratory exposure to amorphous silica nanoparticles promoted systemic inflammation and progression of fibrosis in a susceptible mouse model
inferred← citeshttps://doi.org/10.1016/j.clim.2025.110486openalex-snapshot
T cell aging and exhaustion: Mechanisms and clinical implications
inferred← citeshttps://doi.org/10.1016/j.clinre.2019.07.011openalex-snapshot
The aged liver: Beyond cellular senescence
inferred← citeshttps://doi.org/10.1016/j.clnves.2026.100093openalex-snapshot
The emerging role of aging in tumorigenesis
inferred← citeshttps://doi.org/10.1016/j.cmet.2018.03.016openalex-snapshot
A Potent and Specific CD38 Inhibitor Ameliorates Age-Related Metabolic Dysfunction by Reversing Tissue NAD+ Decline
inferred← citeshttps://doi.org/10.1016/j.cmet.2018.12.008openalex-snapshot
Obesity-Induced Cellular Senescence Drives Anxiety and Impairs Neurogenesis
inferred← citeshttps://doi.org/10.1016/j.cmet.2020.06.004openalex-snapshot
Small Extracellular Vesicles Have GST Activity and Ameliorate Senescence-Related Tissue Damage
inferred← citeshttps://doi.org/10.1016/j.dci.2021.104124openalex-snapshot
Senescence-associated secretory phenotype and activation of NF-κB in splenocytes of old mice exposed to irradiation at a young age
inferred← citeshttps://doi.org/10.1016/j.ddmod.2017.07.005openalex-snapshot
Modelling ageing and age-related disease
inferred← citeshttps://doi.org/10.1016/j.ddmod.2019.01.002openalex-snapshot
A primer on ageing studies in mice: Considerations, opportunities and limitations
inferred← citeshttps://doi.org/10.1016/j.dnarep.2015.01.001openalex-snapshot
Roles of mutagenic translesion synthesis in mammalian genome stability, health and disease
inferred← citeshttps://doi.org/10.1016/j.dnarep.2016.05.022openalex-snapshot
DNA damage processing at telomeres: The ends justify the means
inferred← citeshttps://doi.org/10.1016/j.dnarep.2023.103591openalex-snapshot
Telomeres as hotspots for innate immunity and inflammation
inferred← citeshttps://doi.org/10.1016/j.dsx.2024.103050openalex-snapshot
Joint association of diabetes mellitus and inflammation status with biological ageing acceleration and premature mortality
inferred← citeshttps://doi.org/10.1016/j.ebiom.2015.07.029openalex-snapshot
Inflammation, But Not Telomere Length, Predicts Successful Ageing at Extreme Old Age: A Longitudinal Study of Semi-supercentenarians
inferred← citeshttps://doi.org/10.1016/j.ebiom.2016.11.020openalex-snapshot
Therapeutic Manipulation of Ageing: Repurposing Old Dogs and Discovering New Tricks
inferred← citeshttps://doi.org/10.1016/j.ebiom.2017.03.020openalex-snapshot
Mitochondria in Cell Senescence: Is Mitophagy the Weakest Link?
inferred← citeshttps://doi.org/10.1016/j.ebiom.2017.03.027openalex-snapshot
Telomeres and Cell Senescence - Size Matters Not
inferred← citeshttps://doi.org/10.1016/j.ebiom.2017.04.013openalex-snapshot
Cellular Senescence: A Translational Perspective
inferred← citeshttps://doi.org/10.1016/j.ebiom.2018.09.015openalex-snapshot
Fisetin is a senotherapeutic that extends health and lifespan
inferred← citeshttps://doi.org/10.1016/j.ebiom.2019.01.056openalex-snapshot
Senolytics and senostatics as adjuvant tumour therapy
inferred← citeshttps://doi.org/10.1016/j.ebiom.2023.104905openalex-snapshot
Implications of cellular senescence in paediatric pituitary tumours
inferred← citeshttps://doi.org/10.1016/j.ecoenv.2022.114266openalex-snapshot
Selenomethionine mitigate PM2.5-induced cellular senescence in the lung via attenuating inflammatory response mediated by cGAS/STING/NF-κB pathway
inferred← citeshttps://doi.org/10.1016/j.ecoenv.2024.116438openalex-snapshot
Associations of phthalates with accelerated aging and the mitigating role of physical activity
inferred← citeshttps://doi.org/10.1016/j.ecoenv.2025.118431openalex-snapshot
Molecular regulatory networks of microplastics and cadmium mediated hepatotoxicity from NAFLD to tumorigenesis via integrated approaches
inferred← citeshttps://doi.org/10.1016/j.ejcb.2020.151108openalex-snapshot
Aging of the cells: Insight into cellular senescence and detection Methods
inferred← citeshttps://doi.org/10.1016/j.ejmech.2020.112891openalex-snapshot
Oxidative stress mitigation by antioxidants - An overview on their chemistry and influences on health status
inferred← citeshttps://doi.org/10.1016/j.ejphar.2026.178718openalex-snapshot
Mesenchymal stem cell exosomes and platelet-rich fibrin repair endometrial injury in rats: Insights into histological regeneration, inflammation modulation, and molecular signaling crosstalk
inferred← citeshttps://doi.org/10.1016/j.envint.2021.106906openalex-snapshot
FLEXiGUT: Rationale for exposomics associations with chronic low-grade gut inflammation
inferred← citeshttps://doi.org/10.1016/j.envint.2022.107125openalex-snapshot
Association between maternal per- and polyfluoroalkyl substance exposure and newborn telomere length: Effect modification by birth seasons
inferred← citeshttps://doi.org/10.1016/j.envpol.2023.121855openalex-snapshot
Chain-mediating effect of interaction between telomeres and mitochondria under oxidative stress in coke oven workers
inferred← citeshttps://doi.org/10.1016/j.etap.2021.103611openalex-snapshot
An update in toxicology of ageing
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