On the public citation graph
Tau PET patterns mirror clinical and neuroanatomical variability in Alzheimer’s disease
0 signed · 1263 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/archneurol.2007.34openalex-snapshot
Increased Frequency of Learning Disability in Patients With Primary Progressive Aphasia and Their First-Degree Relatives
inferredcites →https://doi.org/10.1001/archneurol.2011.3354openalex-snapshot
Amyloid-β–Associated Clinical Decline Occurs Only in the Presence of Elevated P-tau
inferredcites →https://doi.org/10.1001/jama.2010.2008openalex-snapshot
Use of Florbetapir-PET for Imaging β-Amyloid Pathology
inferredcites →https://doi.org/10.1001/jama.2015.4668openalex-snapshot
Prevalence of Cerebral Amyloid Pathology in Persons Without Dementia
inferredcites →https://doi.org/10.1001/jama.2015.4669openalex-snapshot
Prevalence of Amyloid PET Positivity in Dementia Syndromes
inferredcites →https://doi.org/10.1002/ana.20009openalex-snapshot
Imaging brain amyloid in Alzheimer's disease with Pittsburgh Compound‐B
inferredcites →https://doi.org/10.1002/ana.21451openalex-snapshot
Aβ amyloid and glucose metabolism in three variants of primary progressive aphasia
inferredcites →https://doi.org/10.1002/ana.22068openalex-snapshot
18 F‐flutemetamol amyloid imaging in Alzheimer disease and mild cognitive impairment: A phase 2 trial
inferredcites →https://doi.org/10.1002/ana.22424openalex-snapshot
Clinicopathological correlations in corticobasal degeneration
inferredcites →https://doi.org/10.1002/ana.24321openalex-snapshot
Tau, amyloid, and hypometabolism in a patient with posterior cortical atrophy
inferredcites →https://doi.org/10.1002/ana.24517openalex-snapshot
Validating novel tau positron emission tomography tracer [F‐18]‐AV‐1451 (T807) on postmortem brain tissue
inferredcites →https://doi.org/10.1002/ana.24546openalex-snapshot
Tau positron emission tomographic imaging in aging and early A lzheimer disease
inferredcites →https://doi.org/10.1002/ana.410380219openalex-snapshot
Apolipoprotein E ϵ4 and cerebral hemorrhage associated with amyloid angiopathy
inferredcites →https://doi.org/10.1002/ana.410410106openalex-snapshot
Neuronal loss correlates with but exceeds neurofibrillary tangles in Alzheimer's disease
inferredcites →https://doi.org/10.1002/hbm.22927openalex-snapshot
Atrophy patterns in early clinical stages across distinct phenotypes of A lzheimer's disease
inferredcites →https://doi.org/10.1007/bf00308809openalex-snapshot
Neuropathological stageing of Alzheimer-related changes
inferredcites →https://doi.org/10.1007/s11940-013-0282-1openalex-snapshot
Diagnosis and Treatment of Corticobasal Degeneration
inferredcites →https://doi.org/10.1007/s12264-013-1414-zopenalex-snapshot
Tau-induced neurodegeneration: mechanisms and targets
inferredcites →https://doi.org/10.1016/j.bandl.2012.12.008openalex-snapshot
Patterns of longitudinal brain atrophy in the logopenic variant of primary progressive aphasia
inferredcites →https://doi.org/10.1016/j.jalz.2011.03.003openalex-snapshot
Toward defining the preclinical stages of Alzheimer's disease: Recommendations from the National Institute on Aging‐Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.jalz.2011.03.005openalex-snapshot
The diagnosis of dementia due to Alzheimer's disease: Recommendations from the National Institute on Aging‐Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.jalz.2011.03.008openalex-snapshot
The diagnosis of mild cognitive impairment due to Alzheimer's disease: Recommendations from the National Institute on Aging‐Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.jalz.2011.07.003openalex-snapshot
Amyloid imaging in dementias with atypical presentation
inferredcites →https://doi.org/10.1016/j.jalz.2012.07.003openalex-snapshot
Impact of molecular imaging on the diagnostic process in a memory clinic
inferredcites →https://doi.org/10.1016/j.jalz.2012.11.008openalex-snapshot
[ 18 F]T807, a novel tau positron emission tomography imaging agent for Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2013.02.013openalex-snapshot
Different patterns of gray matter atrophy in early- and late-onset Alzheimer’s disease
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2014.03.009openalex-snapshot
Medial temporal atrophy in early and late-onset Alzheimer's disease
inferredcites →https://doi.org/10.1016/j.neuroimage.2009.01.009openalex-snapshot
Mapping the effect of APOE ε4 on gray matter loss in Alzheimer's disease in vivo
inferredcites →https://doi.org/10.1016/j.neuroimage.2011.07.098openalex-snapshot
Not quite PIB-positive, not quite PIB-negative: Slight PIB elevations in elderly normal control subjects are biologically relevant
inferredcites →https://doi.org/10.1016/j.neuron.2011.11.033openalex-snapshot
Propagation of Tau Pathology in a Model of Early Alzheimer's Disease
inferredcites →https://doi.org/10.1016/j.neuron.2011.12.040openalex-snapshot
A Network Diffusion Model of Disease Progression in Dementia
inferredcites →https://doi.org/10.1016/j.neuron.2012.03.004openalex-snapshot
Predicting Regional Neurodegeneration from the Healthy Brain Functional Connectome
inferredcites →https://doi.org/10.1016/j.neuron.2013.12.003openalex-snapshot
Biomarker Modeling of Alzheimer’s Disease
inferredcites →https://doi.org/10.1016/j.neuron.2014.05.004openalex-snapshot
The Intersection of Amyloid Beta and Tau at Synapses in Alzheimer’s Disease
inferredcites →https://doi.org/10.1016/s0896-6273(02)00569-xopenalex-snapshot
Whole Brain Segmentation
inferredcites →https://doi.org/10.1016/s1474-4422(10)70306-9openalex-snapshot
Early-onset versus late-onset Alzheimer's disease: the case of the missing APOE ɛ4 allele
inferredcites →https://doi.org/10.1016/s1474-4422(11)70077-1openalex-snapshot
Cerebral amyloid-β PET with florbetaben (18F) in patients with Alzheimer's disease and healthy controls: a multicentre phase 2 diagnostic study
inferredcites →https://doi.org/10.1016/s1474-4422(11)70156-9openalex-snapshot
Neuropathologically defined subtypes of Alzheimer's disease with distinct clinical characteristics: a retrospective study
inferredcites →https://doi.org/10.1016/s1474-4422(11)70289-7openalex-snapshot
Posterior cortical atrophy
inferredcites →https://doi.org/10.1016/s1474-4422(14)70090-0openalex-snapshot
Advancing research diagnostic criteria for Alzheimer's disease: the IWG-2 criteria
inferredcites →https://doi.org/10.1016/s1474-4422(14)70194-2openalex-snapshot
Age-specific population frequencies of cerebral β-amyloidosis and neurodegeneration among people with normal cognitive function aged 50–89 years: a cross-sectional study
inferredcites →https://doi.org/10.1016/s1474-4422(14)70252-2openalex-snapshot
Tau imaging: early progress and future directions
inferredcites →https://doi.org/10.1038/ncb1901openalex-snapshot
Transmission and spreading of tauopathy in transgenic mouse brain
inferredcites →https://doi.org/10.1046/j.1365-2990.2002.00385.xopenalex-snapshot
Synapse loss is greater in presenile than senile onset Alzheimer disease: implications for the cognitive reserve hypothesis
inferredcites →https://doi.org/10.1056/nejmoa1304839openalex-snapshot
Two Phase 3 Trials of Bapineuzumab in Mild-to-Moderate Alzheimer's Disease
inferredcites →https://doi.org/10.1073/pnas.1434398100openalex-snapshot
Carboxyl-terminal-truncated apolipoprotein E4 causes Alzheimer's disease-like neurodegeneration and behavioral deficits in transgenic mice
inferredcites →https://doi.org/10.1073/pnas.72.5.1858openalex-snapshot
A protein factor essential for microtubule assembly.
inferredcites →https://doi.org/10.1073/pnas.90.20.9649openalex-snapshot
Increased amyloid beta-peptide deposition in cerebral cortex as a consequence of apolipoprotein E genotype in late-onset Alzheimer disease.
inferredcites →https://doi.org/10.1093/brain/awp326openalex-snapshot
Increased metabolic vulnerability in early-onset Alzheimer’s disease is not related to amyloid burden
inferredcites →https://doi.org/10.1093/brain/aws076openalex-snapshot
Clinically concordant variations of Alzheimer pathology in aphasic versus amnestic dementia
inferredcites →https://doi.org/10.1093/brain/aws113openalex-snapshot
Amyloid burden and metabolic function in early-onset Alzheimer's disease: parietal lobe involvement
inferredcites →https://doi.org/10.1093/brain/aws327openalex-snapshot
Diverging patterns of amyloid deposition and hypometabolism in clinical variants of probable Alzheimer’s disease
inferredcites →https://doi.org/10.1093/brain/awt242openalex-snapshot
Handedness and language learning disability differentially distribute in progressive aphasia variants
inferredcites →https://doi.org/10.1093/brain/awt266openalex-snapshot
Deciphering logopenic primary progressive aphasia: a clinical, imaging and biomarker investigation
inferredcites →https://doi.org/10.1093/brain/awv191openalex-snapshot
The behavioural/dysexecutive variant of Alzheimer’s disease: clinical, neuroimaging and pathological features
inferredcites →https://doi.org/10.1093/brain/awv229openalex-snapshot
Asymmetries of amyloid-β burden and neuronal dysfunction are positively correlated in Alzheimer’s disease
inferredcites →https://doi.org/10.1093/cercor/bht076openalex-snapshot
Is Verbal Episodic Memory in Elderly with Amyloid Deposits Preserved Through Altered Neuronal Function?
inferredcites →https://doi.org/10.1093/hmg/dds161openalex-snapshot
Evidence for a role of the rare p.A152T variant in MAPT in increasing the risk for FTD-spectrum and Alzheimer's diseases
inferredcites →https://doi.org/10.1093/jnen/61.11.935openalex-snapshot
Office of Rare Diseases Neuropathologic Criteria for Corticobasal Degeneration
inferredcites →https://doi.org/10.1097/00004647-199609000-00008openalex-snapshot
Distribution Volume Ratios without Blood Sampling from Graphical Analysis of PET Data
inferredcites →https://doi.org/10.1097/00004728-199507000-00006openalex-snapshot
Preserved Pontine Glucose Metabolism in Alzheimer Disease
inferredcites →https://doi.org/10.1097/00146965-200312000-00002openalex-snapshot
Distinctive Neuropsychological Patterns in Frontotemporal Dementia, Semantic Dementia, And Alzheimer Disease
inferredcites →https://doi.org/10.1097/nen.0b013e31825018f7openalex-snapshot
Correlation of Alzheimer Disease Neuropathologic Changes With Cognitive Status: A Review of the Literature
inferredcites →https://doi.org/10.1097/wad.0b013e318279d02aopenalex-snapshot
Potential Impact of Amyloid Imaging on Diagnosis and Intended Management in Patients With Progressive Cognitive Decline
inferredcites →https://doi.org/10.1111/ene.12761openalex-snapshot
Regional β‐amyloid burden does not correlate with cognitive or language deficits in Alzheimer's disease presenting as aphasia
inferredcites →https://doi.org/10.1111/j.1471-4159.2009.06181.xopenalex-snapshot
The amyloid hypothesis for Alzheimer’s disease: a critical reappraisal
inferredcites →https://doi.org/10.1111/j.1749-6632.1996.tb34415.xopenalex-snapshot
Apolipoprotein E, Plaques, Tangles and Cholinergic Dysfunction in Alzheimer's Diseasea
inferredcites →https://doi.org/10.1136/jnnp-2013-305858openalex-snapshot
Greater medial temporal hypometabolism and lower cortical amyloid burden in ApoE4-positive AD patients
inferredcites →https://doi.org/10.1159/000058331openalex-snapshot
Posterior Cortical Atrophy: Clinical Characteristics and Differences Compared to Alzheimer’s Disease
inferredcites →https://doi.org/10.1159/000339367openalex-snapshot
Impact of Beta-Amyloid-Specific Florbetaben PET Imaging on Confidence in Early Diagnosis of Alzheimer’s Disease
inferredcites →https://doi.org/10.1159/000477538openalex-snapshot
Qualitative Assessment of Verbal Fluency Performance in Frontotemporal Dementia
inferredcites →https://doi.org/10.1176/appi.focus.11.1.96openalex-snapshot
The Diagnosis of Mild Cognitive Impairment due to Alzheimer’s Disease: Recommendations from the National Institute on Aging-Alzheimer’s Association Workgroups on Diagnostic Guidelines for Alzheimer’s Disease
inferredcites →https://doi.org/10.1177/0891988706297086openalex-snapshot
Early-Onset Alzheimer’s Disease Is Associated With Greater Pathologic Burden
inferredcites →https://doi.org/10.1177/1533317512454711openalex-snapshot
Nonamnestic Presentations of Early-Onset Alzheimer’s Disease
inferredcites →https://doi.org/10.1186/s13195-014-0093-yopenalex-snapshot
Predicting amyloid status in corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography
inferredcites →https://doi.org/10.1212/01.wnl.0000128091.92139.0fopenalex-snapshot
Impact of APOE genotype on neuropathologic and neurochemical markers of Alzheimer disease
inferredcites →https://doi.org/10.1212/wnl.0000000000000032openalex-snapshot
Practical utility of amyloid and FDG-PET in an academic dementia center
inferredcites →https://doi.org/10.1212/wnl.0000000000000550openalex-snapshot
Vascular risk and Aβ interact to reduce cortical thickness in AD vulnerable brain regions
inferredcites →https://doi.org/10.1212/wnl.0000000000000824openalex-snapshot
Asymmetry of cortical decline in subtypes of primary progressive aphasia
inferredcites →https://doi.org/10.1212/wnl.0000000000001877openalex-snapshot
Amyloid-β 11 C-PiB-PET imaging results from 2 randomized bapineuzumab phase 3 AD trials
inferredcites →https://doi.org/10.1212/wnl.0b013e31821103e6openalex-snapshot
Classification of primary progressive aphasia and its variants
inferredcites →https://doi.org/10.1212/wnl.0b013e31825dce28openalex-snapshot
Early-onset Alzheimer disease clinical variants
inferredcites →https://doi.org/10.1212/wnl.0b013e31827f0889openalex-snapshot
Differential effect of APOE genotype on amyloid load and glucose metabolism in AD dementia
inferredcites →https://doi.org/10.1212/wnl.42.3.631openalex-snapshot
Neurofibrillary tangles but not senile plaques parallel duration and severity of Alzheimer's disease
inferredcites →https://doi.org/10.1371/journal.pone.0062471openalex-snapshot
FDG PET and MRI in Logopenic Primary Progressive Aphasia versus Dementia of the Alzheimer’s Type
inferredcites →https://doi.org/10.3233/jad-121960openalex-snapshot
Cerebrospinal Fluid Biomarkers Mirror Rate of Cognitive Decline
inferredcites →https://doi.org/10.3233/jad-122059openalex-snapshot
Early Clinical PET Imaging Results with the Novel PHF-Tau Radioligand [F-18]-T807
inferredcites →https://doi.org/10.3233/jad-130098openalex-snapshot
Early Clinical PET Imaging Results with the Novel PHF-Tau Radioligand [F18]-T808
inferredcites →https://doi.org/10.3233/jad-2011-111694openalex-snapshot
Injury Markers but not Amyloid Markers are Associated with Rapid Progression from Mild Cognitive Impairment to Dementia in Alzheimer's Disease
inferredcites →https://doi.org/10.3233/jad-2012-111934openalex-snapshot
Early Onset Alzheimer's Disease is Associated with a Distinct Neuropsychological Profile
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.3716976openalex-extracted
Neurotoxic Astrocytes Secreted Glypican-4 Drives Alzheimer's Tau Pathology
inferred← citeshttps://doi.org/10.1001/jama.2018.12917openalex-snapshot
Discriminative Accuracy of [ 18 F]flortaucipir Positron Emission Tomography for Alzheimer Disease vs Other Neurodegenerative Disorders
inferred← citeshttps://doi.org/10.1001/jamanetworkopen.2019.13909openalex-snapshot
Association of Apolipoprotein E ɛ4, Educational Level, and Sex With Tau Deposition and Tau-Mediated Metabolic Dysfunction in Older Adults
inferred← citeshttps://doi.org/10.1001/jamaneurol.2016.2481openalex-snapshot
Tau and β-Amyloid—The Malignant Duo
inferred← citeshttps://doi.org/10.1001/jamaneurol.2016.3338openalex-snapshot
Tau Positron Emission Tomographic Imaging in the Lewy Body Diseases
inferred← citeshttps://doi.org/10.1001/jamaneurol.2016.5349openalex-snapshot
Alzheimer Disease Signature Neurodegeneration and APOE Genotype in Mild Cognitive Impairment With Suspected Non–Alzheimer Disease Pathophysiology
inferred← citeshttps://doi.org/10.1001/jamaneurol.2016.5701openalex-snapshot
Tau and Cortical Thickness in Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2016.5755openalex-snapshot
Association of In Vivo [ 18 F]AV-1451 Tau PET Imaging Results With Cortical Atrophy and Symptoms in Typical and Atypical Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2017.0323openalex-snapshot
Linking Amyloid-β and Tau Deposition in Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2017.2216openalex-snapshot
Region-Specific Association of Subjective Cognitive Decline With Tauopathy Independent of Global β-Amyloid Burden
inferred← citeshttps://doi.org/10.1001/jamaneurol.2017.3713openalex-snapshot
Associations Between Tau, β-Amyloid, and Cognition in Parkinson Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2017.4026openalex-snapshot
Tau Positron Emission Tomography in Autosomal Dominant Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2018.3692openalex-snapshot
Correlation of In Vivo [18F]Flortaucipir With Postmortem Alzheimer Disease Tau Pathology
inferred← citeshttps://doi.org/10.1001/jamaneurol.2019.1424openalex-snapshot
Association of Amyloid and Tau With Cognition in Preclinical Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2019.1971openalex-snapshot
Prevalence of Biologically vs Clinically Defined Alzheimer Spectrum Entities Using the National Institute on Aging–Alzheimer’s Association Research Framework
inferred← citeshttps://doi.org/10.1001/jamaneurol.2019.3606openalex-snapshot
Selective Vulnerability of the Nucleus Basalis of Meynert Among Neuropathologic Subtypes of Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2019.4421openalex-snapshot
Association of Apolipoprotein E ε4 With Medial Temporal Tau Independent of Amyloid-β
inferred← citeshttps://doi.org/10.1001/jamaneurol.2019.4509openalex-snapshot
Tau Positron Emission Tomographic Findings in a Former US Football Player With Pathologically Confirmed Chronic Traumatic Encephalopathy
inferred← citeshttps://doi.org/10.1001/jamaneurol.2019.5154openalex-snapshot
Assessment of Demographic, Genetic, and Imaging Variables Associated With Brain Resilience and Cognitive Resilience to Pathological Tau in Patients With Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2020.0989openalex-snapshot
Diagnostic Performance of RO948 F 18 Tau Positron Emission Tomography in the Differentiation of Alzheimer Disease From Other Neurodegenerative Disorders
inferred← citeshttps://doi.org/10.1001/jamaneurol.2020.1741openalex-snapshot
Prevalence and Clinical Phenotype of Quadruple Misfolded Proteins in Older Adults
inferred← citeshttps://doi.org/10.1001/jamaneurol.2020.3921openalex-snapshot
Association of Initial β-Amyloid Levels With Subsequent Flortaucipir Positron Emission Tomography Changes in Persons Without Cognitive Impairment
inferred← citeshttps://doi.org/10.1001/jamaneurol.2020.5505openalex-snapshot
Aggregated Tau Measured by Visual Interpretation of Flortaucipir Positron Emission Tomography and the Associated Risk of Clinical Progression of Mild Cognitive Impairment and Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2021.1858openalex-snapshot
Accuracy of Tau Positron Emission Tomography as a Prognostic Marker in Preclinical and Prodromal Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2021.4417openalex-snapshot
Research Criteria for the Behavioral Variant of Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2021.4654openalex-snapshot
Biomarker-Based Prediction of Longitudinal Tau Positron Emission Tomography in Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2022.0676openalex-snapshot
Divergent Cortical Tau Positron Emission Tomography Patterns Among Patients With Preclinical Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2023.1067openalex-snapshot
Comparison of Group-Level and Individualized Brain Regions for Measuring Change in Longitudinal Tau Positron Emission Tomography in Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2024.0784openalex-snapshot
Clinicopathologic Heterogeneity and Glial Activation Patterns in Alzheimer Disease
inferred← citeshttps://doi.org/10.1001/jamaneurol.2025.0013openalex-snapshot
Sex Differences in Longitudinal Tau-PET in Preclinical Alzheimer Disease
inferred← citeshttps://doi.org/10.1002/9783527845583.oth1openalex-snapshot
Declaration of Competing Interest
inferred← citeshttps://doi.org/10.1002/acn3.51038openalex-snapshot
MRI and flortaucipir relationships in Alzheimer's phenotypes are heterogeneous
inferred← citeshttps://doi.org/10.1002/acn3.51237openalex-snapshot
Ante‐ and postmortem tau in autosomal dominant and late‐onset Alzheimer’s disease
inferred← citeshttps://doi.org/10.1002/acn3.51297openalex-snapshot
Flortaucipir (tau) PET in LGI1 antibody encephalitis
inferred← citeshttps://doi.org/10.1002/acn3.51457openalex-snapshot
Amyloid‐dependent and amyloid‐independent effects of Tau in individuals without dementia
inferred← citeshttps://doi.org/10.1002/acn3.631openalex-snapshot
[ 18 F]AV‐1451 binding is increased in frontotemporal dementia due to C9orf72 expansion
inferred← citeshttps://doi.org/10.1002/adtp.202400008openalex-snapshot
Effect of APOE ɛ4 Status on Brain Amyloid‐β and Cognitive Function in Amnestic and Nonamnestic Mild Cognitive Impairment: A [18F] Florbetapir PET‐CT Study
inferred← citeshttps://doi.org/10.1002/alz.12124openalex-snapshot
An agnostic reevaluation of the amyloid cascade hypothesis of Alzheimer's disease pathogenesis: The role of APP homeostasis
inferred← citeshttps://doi.org/10.1002/alz.12173openalex-snapshot
APOE interacts with tau PET to influence memory independently of amyloid PET in older adults without dementia
inferred← citeshttps://doi.org/10.1002/alz.12245openalex-snapshot
Cerebral amyloid‐β load is associated with neurodegeneration and gliosis: Mediation by p‐tau and interactions with risk factors early in the Alzheimer's continuum
inferred← citeshttps://doi.org/10.1002/alz.12249openalex-snapshot
Distinct effects of beta‐amyloid and tau on cortical thickness in cognitively healthy older adults
inferred← citeshttps://doi.org/10.1002/alz.12349openalex-snapshot
Reduced synchrony in alpha oscillations during life predicts post mortem neurofibrillary tangle density in early‐onset and atypical Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.12356openalex-snapshot
Towards clinical application of tau PET tracers for diagnosing dementia due to Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.12445openalex-snapshot
Relationships among tau burden, atrophy, age, and naming in the aphasic variant of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.12487openalex-snapshot
The protective gene dose effect of the APOEε2 allele on gray matter volume in cognitively unimpaired individuals
inferred← citeshttps://doi.org/10.1002/alz.12518openalex-snapshot
Differential expression of tau species and the association with cognitive decline and synaptic loss in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.12749openalex-snapshot
Phosphorylated tau sites that are elevated in Alzheimer's disease fluid biomarkers are visualized in early neurofibrillary tangle maturity levels in the post mortem brain
inferred← citeshttps://doi.org/10.1002/alz.12867openalex-snapshot
Functional network segregation is associated with attenuated tau spreading in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.12875openalex-snapshot
Tau‐PET is superior to phospho‐tau when predicting cognitive decline in symptomatic AD patients
inferred← citeshttps://doi.org/10.1002/alz.12922openalex-snapshot
Initial non‐amnestic symptoms relate to faster rate of functional and cognitive decline compared to amnestic symptoms in neuropathologically confirmed dementias
inferred← citeshttps://doi.org/10.1002/alz.12943openalex-snapshot
Does Alzheimer's disease with mesial temporal lobe epilepsy represent a distinct disease subtype?
inferred← citeshttps://doi.org/10.1002/alz.12963openalex-snapshot
Alzheimer's disease phenotypes show different sleep architecture
inferred← citeshttps://doi.org/10.1002/alz.12978openalex-snapshot
Protein disulfide isomerases as CSF biomarkers for the neuronal response to tau pathology
inferred← citeshttps://doi.org/10.1002/alz.13011openalex-snapshot
Cortical tau is associated with microstructural imaging biomarkers of neurite density and dendritic complexity in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.13355openalex-snapshot
The impact of tau deposition and hypometabolism on cognitive impairment and longitudinal cognitive decline
inferred← citeshttps://doi.org/10.1002/alz.13438openalex-snapshot
Successful cognitive aging is associated with thicker anterior cingulate cortex and lower tau deposition compared to typical aging
inferred← citeshttps://doi.org/10.1002/alz.13444openalex-snapshot
Characterizing the emergence of amyloid and tau burden in Down syndrome
inferred← citeshttps://doi.org/10.1002/alz.13769openalex-snapshot
Data‐driven decomposition and staging of flortaucipir uptake in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.13859openalex-snapshot
Revised criteria for diagnosis and staging of Alzheimer's disease: Alzheimer's Association Workgroup
inferred← citeshttps://doi.org/10.1002/alz.13908openalex-snapshot
Harmonizing tau positron emission tomography in Alzheimer's disease: The CenTauR scale and the joint propagation model
inferred← citeshttps://doi.org/10.1002/alz.13914openalex-snapshot
Relationships between PET and blood plasma biomarkers in corticobasal syndrome
inferred← citeshttps://doi.org/10.1002/alz.14060openalex-snapshot
Associations of cerebral amyloid beta and tau with cognition from midlife
inferred← citeshttps://doi.org/10.1002/alz.14338openalex-snapshot
Updated appropriate use criteria for amyloid and tau PET: A report from the Alzheimer's Association and Society for Nuclear Medicine and Molecular Imaging Workgroup
inferred← citeshttps://doi.org/10.1002/alz.14411openalex-snapshot
Plasma pTau217 ratio predicts continuous regional brain tau accumulation in amyloid‐positive early Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.14423openalex-snapshot
Characterizing visual read tau‐PET‐negative participants with Alzheimer's disease dementia
inferred← citeshttps://doi.org/10.1002/alz.14489openalex-snapshot
Dissociable spatial topography of cortical atrophy in early‐onset and late‐onset Alzheimer's disease: A head‐to‐head comparison of the LEADS and ADNI cohorts
inferred← citeshttps://doi.org/10.1002/alz.14515openalex-snapshot
Cognitive aging outcomes are related to both tau pathology and maintenance of cingulate cortex structure
inferred← citeshttps://doi.org/10.1002/alz.14600openalex-snapshot
Machine learning prediction of tau‐PET in Alzheimer's disease using plasma, MRI, and clinical data
inferred← citeshttps://doi.org/10.1002/alz.14625openalex-snapshot
Tau‐related reduction of glucose metabolism in mild cognitive impairment occurs independently of APOE ε4 genotype and is influenced by Aβ
inferred← citeshttps://doi.org/10.1002/alz.70003openalex-snapshot
Impact of the polygenic risk scores for attention‐deficit/hyperactivity disorder in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.70061openalex-snapshot
Connectome‐based predictive modeling of brain pathology and cognition in autosomal dominant Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.70075openalex-snapshot
Amyloid PET predicts longitudinal functional and cognitive trajectories in a heterogeneous cohort
inferred← citeshttps://doi.org/10.1002/alz.70153openalex-snapshot
Heterogeneous tau deposition patterns in the preclinical stage link to domain‐specific cognitive deficits
inferred← citeshttps://doi.org/10.1002/alz.70346openalex-snapshot
Tau burden is best captured by magnitude and extent: Tau‐MaX as a measure of global tau
inferred← citeshttps://doi.org/10.1002/alz.70352openalex-snapshot
Integrating visual assessments and quantification methods for tau PET staging
inferred← citeshttps://doi.org/10.1002/alz.70511openalex-snapshot
Tau, atrophy, and domain‐specific cognitive impairment in typical Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.70520openalex-snapshot
Comparison of visual and analytic methods of Alzheimer's disease pathologic staging
inferred← citeshttps://doi.org/10.1002/alz.70689openalex-snapshot
The relationship of soluble tau species with Alzheimer's disease amyloid plaque removal and tau pathology
inferred← citeshttps://doi.org/10.1002/alz.70725openalex-snapshot
Expanding the horizon of sex‐specific proteomic insights in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.70749openalex-snapshot
Catecholaminergic nucleus integrity and Alzheimer's pathology, symptoms, and progression
inferred← citeshttps://doi.org/10.1002/alz.70881openalex-snapshot
Combining CSF MTBR‐tau243 and plasma pTau217 ratio enhances the prediction of continuous regional tau PET burden in early Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.71017openalex-snapshot
Characterizing amyloid and tau positron emission tomography‐based stages across the clinical continuum
inferred← citeshttps://doi.org/10.1002/alz.71319openalex-snapshot
Florzolotau (18F) retention is linked to neuropsychological performance in tauopathy
inferred← citeshttps://doi.org/10.1002/alz.71427openalex-snapshot
Locus coeruleus degeneration is associated with cortical tau deposition and cognitive decline in older adults at familial risk of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.71431openalex-snapshot
Emerging directions in tauopathy research
inferred← citeshttps://doi.org/10.1002/alz.71483openalex-snapshot
Evaluation of an in vivo biomarker of arteriolosclerosis (ARTS) and its associations with cognition and multimodal ATN(V) biomarkers in a cardiometabolic‐risk enriched community cohort
inferred← citeshttps://doi.org/10.1002/alz.71491openalex-snapshot
Sex differences in brain glucose metabolism and Alzheimer's disease risk and progression
inferred← citeshttps://doi.org/10.1002/alz.71571openalex-snapshot
Vulnerability of anterior medial temporal lobe subregions to early tau‐related neurodegeneration in Alzheimer's disease: Converging evidence from tau‐PET and plasma p‐tau217
inferred← citeshttps://doi.org/10.1002/alz.71617openalex-snapshot
Modular brain networks shape amyloid‐driven tau spread and cognitive decline
inferred← citeshttps://doi.org/10.1002/alz.71718openalex-snapshot
Group‐level versus individualized approaches for capturing tau PET changes and clinical progression in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/alz.71829openalex-snapshot
Operationalizing Alzheimer's disease trials in the era of targeted therapies
inferred← citeshttps://doi.org/10.1002/alz.71898openalex-snapshot
Plasma eMTBR‐tau243 as a disease biomarker across Alzheimer's disease clinical phenotypes
inferred← citeshttps://doi.org/10.1002/alz.71899openalex-snapshot
Brain microstructure mediates the relationship between Alzheimer's proteinopathies and memory
inferred← citeshttps://doi.org/10.1002/alz.71914openalex-snapshot
Network‐dependent remote effects and local tau burden jointly shape neuronal dysfunction in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/ana.24711openalex-snapshot
In vivo cortical spreading pattern of tau and amyloid in the Alzheimer disease spectrum
inferred← citeshttps://doi.org/10.1002/ana.24825openalex-snapshot
AV‐1451 tau and β‐amyloid positron emission tomography imaging in dementia with Lewy bodies
inferred← citeshttps://doi.org/10.1002/ana.24910openalex-snapshot
Fluorodeoxyglucose metabolism associated with tau‐amyloid interaction predicts memory decline
inferred← citeshttps://doi.org/10.1002/ana.25060openalex-snapshot
18F‐flortaucipir tau positron emission tomography distinguishes established progressive supranuclear palsy from controls and Parkinson disease: A multicenter study
inferred← citeshttps://doi.org/10.1002/ana.25183openalex-snapshot
[18F]AV‐1451 tau‐PET and primary progressive aphasia
inferred← citeshttps://doi.org/10.1002/ana.25406openalex-snapshot
Longitudinal tau accumulation and atrophy in aging and alzheimer disease
inferred← citeshttps://doi.org/10.1002/ana.25968openalex-snapshot
Diagnostic Accuracy of Amyloid versus18F‐Fluorodeoxyglucose Positron Emission Tomography in Autopsy‐Confirmed Dementia
inferred← citeshttps://doi.org/10.1002/ana.26620openalex-snapshot
Change in Cerebrospinal Fluid Tau Microtubule Binding Region Detects Symptom Onset, Cognitive Decline, Tangles, and Atrophy in Dominantly Inherited Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/ana.78300openalex-snapshot
Distribution of Big Tau Isoforms in the Human Central and Peripheral Nervous System
inferred← citeshttps://doi.org/10.1002/brb3.1548openalex-snapshot
Frontal variant of Alzheimer's disease with asymmetric presentation mimicking frontotemporal dementia: Case report and literature review
inferred← citeshttps://doi.org/10.1002/brh2.70008openalex-snapshot
Transforming Brain Health With Neurotechnological Convergence (Part I): Precision Diagnostics and Targeted Interventions
inferred← citeshttps://doi.org/10.1002/cns.71125openalex-snapshot
GWAS of Tau‐Neurodegeneration Mismatch Identifies New Risk Loci for Susceptibility to Tau
inferred← citeshttps://doi.org/10.1002/dad2.12125openalex-snapshot
Association between neurite metrics and tau/inflammatory pathology in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/dad2.12200openalex-snapshot
Spatio‐spectral relationships between pathological neural dynamics and cognitive impairment along the Alzheimer's disease spectrum
inferred← citeshttps://doi.org/10.1002/dad2.12217openalex-snapshot
Current advances in digital cognitive assessment for preclinical Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/dad2.12218openalex-snapshot
Hippocampal subfield imaging and fractional anisotropy show parallel changes in Alzheimer's disease tau progression using simultaneous tau‐PET/MRI at 3T
inferred← citeshttps://doi.org/10.1002/dad2.12284openalex-snapshot
The neuroinflammatory marker sTNFR2 relates to worse cognition and tau in women across the Alzheimer's disease spectrum
inferred← citeshttps://doi.org/10.1002/dad2.12454openalex-snapshot
CenTauR: Toward a universal scale and masks for standardizing tau imaging studies
inferred← citeshttps://doi.org/10.1002/dad2.12550openalex-snapshot
APOE ε4 allele status modulates the spatial patterns of progressive atrophy in the temporal lobes after mild traumatic brain injury
inferred← citeshttps://doi.org/10.1002/dad2.70033openalex-snapshot
Survey among experts on the future role of tau‐PET in clinical practice and trials
inferred← citeshttps://doi.org/10.1002/dad2.70264openalex-snapshot
Hallucinations and delusions are associated with elevated tau PET signal independent of age, clinical severity, and amyloid burden
inferred← citeshttps://doi.org/10.1002/dad2.70365openalex-snapshot
From clinical trial to clinical experience: Lecanemab therapy in a real‐world case series
inferred← citeshttps://doi.org/10.1002/dad2.70442openalex-snapshot
Decreased cerebral metabolic rate of oxygen (CMRO 2 ) is associated with increased tau deposition in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/dad2.70475openalex-snapshot
Plasma GFAP and language‐network tau mediate the effect of amyloid burden on semantic fluency: A mediation analysis
inferred← citeshttps://doi.org/10.1002/eji.70250openalex-snapshot
Active Vaccination Strategies for Alzheimer's Disease: An Expanded Review
inferred← citeshttps://doi.org/10.1002/gps.4926openalex-snapshot
Chronic depressive symptomatology and CSF amyloid beta and tau levels in mild cognitive impairment
inferred← citeshttps://doi.org/10.1002/gps.5310openalex-snapshot
Joint trajectories of cognition and gait speed in Mexican American and European American older adults: The San Antonio longitudinal study of aging
inferred← citeshttps://doi.org/10.1002/hbm.23772openalex-snapshot
Gray matter asymmetries in aging and neurodegeneration: A review and meta‐analysis
inferred← citeshttps://doi.org/10.1002/hbm.23874openalex-snapshot
Tau PET imaging predicts cognition in atypical variants of Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/hbm.23897openalex-snapshot
Regionally specific changes in the hippocampal circuitry accompany progression of cerebrospinal fluid biomarkers in preclinical Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/hbm.24056openalex-snapshot
Cortical microstructure in young onset Alzheimer's disease using neurite orientation dispersion and density imaging
inferred← citeshttps://doi.org/10.1002/hbm.24401openalex-snapshot
Data‐driven approaches for tau‐PET imaging biomarkers in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/hbm.24473openalex-snapshot
Regional multimodal relationships between tau, hypometabolism, atrophy, and fractional anisotropy in atypical Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/hbm.24782openalex-snapshot
Applications of amyloid, tau, and neuroinflammation PET imaging to Alzheimer's disease and mild cognitive impairment
inferred← citeshttps://doi.org/10.1002/hbm.25105openalex-snapshot
Finding specificity in structural brain alterations through Bayesian reverse inference
inferred← citeshttps://doi.org/10.1002/hbm.26043openalex-snapshot
Tau as a biomarker of cognitive impairment and neuropsychiatric symptom in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/hbm.26388openalex-snapshot
Network anatomy in logopenic variant of primary progressive aphasia
inferred← citeshttps://doi.org/10.1002/hbm.26689openalex-snapshot
Characterization of tau propagation pattern and cascading hypometabolism from functional connectivity in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/mds.27252openalex-snapshot
Distinct patterns of amyloid‐dependent tau accumulation in Lewy body diseases
inferred← citeshttps://doi.org/10.1002/mds.27356openalex-snapshot
[18F]AV‐1451 binding and postmortem pathology of CBD
inferred← citeshttps://doi.org/10.1002/mds.27603openalex-snapshot
18F‐AV‐1451 uptake differs between dementia with lewy bodies and posterior cortical atrophy
inferred← citeshttps://doi.org/10.1002/mds.27677openalex-snapshot
Reply to letter: Basis of cingulate island sign may differ in dementia with lewy bodies and posterior cortical atrophy
inferred← citeshttps://doi.org/10.1002/mp.17902openalex-snapshot
Physical phantom validation of clustering‐initiated factorization in dynamic PET
inferred← citeshttps://doi.org/10.1002/nbm.70383openalex-snapshot
Quantifying the Diamagnetic and Paramagnetic Components of Beta‐Amyloid Plaques Using Decomposed Quantitative Susceptibility Mapping in Mouse Models of Alzheimer's Disease
inferred← citeshttps://doi.org/10.1002/npr2.12281openalex-snapshot
Findings of 18F‐PI ‐2620 tau PET imaging in patients with Alzheimer’s disease and healthy controls in relation to the plasma P‐tau181 levels in a Japanese sample
inferred← citeshttps://doi.org/10.1002/ptr.7387openalex-snapshot
A novel DPP ‐4 inhibitor Gramcyclin A attenuates cognitive deficits in APP / PS1 /tau triple transgenic mice via enhancing brain GLP ‐1‐dependent glucose uptake
inferred← citeshttps://doi.org/10.1002/trc2.12096openalex-snapshot
Relationship of amyloid beta and neurofibrillary tau deposition in Neurodegeneration in Aging Down Syndrome (NiAD) study at baseline
inferred← citeshttps://doi.org/10.1002/trc2.12154openalex-snapshot
Association between regional tau pathology and neuropsychiatric symptoms in aging and dementia due to Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/trc2.12356openalex-snapshot
Free water derived by multi‐shell diffusion MRI reflects tau/neuroinflammatory pathology in Alzheimer's disease
inferred← citeshttps://doi.org/10.1002/trc2.12372openalex-snapshot
Development, initial validation, and application of a visual read method for [ 18 F]MK‐6240 tau PET
inferred← citeshttps://doi.org/10.1002/trc2.12490openalex-snapshot
Relationships between plasma biomarkers, tau PET, FDG PET, and volumetric MRI in mild to moderate Alzheimer's disease patients
inferred← citeshttps://doi.org/10.1002/trc2.70101openalex-snapshot
Creatine monohydrate pilot in Alzheimer's: Feasibility, brain creatine, and cognition
inferred← citeshttps://doi.org/10.1002/wnan.1946openalex-snapshot
Advances in nanoprobes for molecular MRI of Alzheimer's disease
inferred← citeshttps://doi.org/10.1007/7854_2021_267openalex-snapshot
Psychedelics as Novel Therapeutics in Alzheimer’s Disease: Rationale and Potential Mechanisms
inferred← citeshttps://doi.org/10.1007/978-1-0716-3195-9_25openalex-snapshot
Machine Learning for Alzheimer’s Disease and Related Dementias
inferred← citeshttps://doi.org/10.1007/978-1-4939-7704-8_15openalex-snapshot
Quantification of Tau Load in Alzheimer’s Disease Clinical Trials Using Positron Emission Tomography
inferred← citeshttps://doi.org/10.1007/978-1-4939-7704-8_3openalex-snapshot
Systems Biology Methods for Alzheimer’s Disease Research Toward Molecular Signatures, Subtypes, and Stages and Precision Medicine: Application in Cohort Studies and Trials
inferred← citeshttps://doi.org/10.1007/978-3-030-01523-7_5openalex-snapshot
18F-FDG-PET/CT (FDG-PET) in Neurodegenerative Disease
inferred← citeshttps://doi.org/10.1007/978-3-030-20351-1_29openalex-snapshot
A Longitudinal Model for Tau Aggregation in Alzheimer’s Disease Based on Structural Connectivity
inferred← citeshttps://doi.org/10.1007/978-3-030-48419-4_20openalex-snapshot
Positron Emission Tomography
inferred← citeshttps://doi.org/10.1007/978-3-030-51140-1_11openalex-snapshot
Progressive Supranuclear Palsy and Corticobasal Degeneration
inferred← citeshttps://doi.org/10.1007/978-3-030-53168-3_12openalex-snapshot
Neuroimaging Findings in Mild Cognitive Impairment
inferred← citeshttps://doi.org/10.1007/978-3-030-53168-3_9openalex-snapshot
PET Neuroimaging in Dementia Conditions
inferred← citeshttps://doi.org/10.1007/978-3-030-82367-2_12openalex-snapshot
Tau Imaging in Neurodegenerative Dementia
inferred← citeshttps://doi.org/10.1007/978-3-031-10909-6_30openalex-snapshot
Functional MRI Studies of Memory in Aging, Mild Cognitive Impairment, and Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-031-35098-6_6openalex-snapshot
Traits and Trammels of Tau Tracer Imaging
inferred← citeshttps://doi.org/10.1007/978-3-031-43356-6_11openalex-snapshot
Neuroimaging Biomarkers in Alzheimer’s Disease and Related Disorders
inferred← citeshttps://doi.org/10.1007/978-3-031-52641-1_69-1openalex-snapshot
APOE Polymorphism and Brain Imaging
inferred← citeshttps://doi.org/10.1007/978-3-031-68237-7_51openalex-snapshot
Immunotherapy of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-3-031-80849-4_20openalex-snapshot
Imaging Markers of Amyloid and Tau in Alzheimer Disease
inferred← citeshttps://doi.org/10.1007/978-3-032-38470-6_59openalex-snapshot
Stage-Aware Adaptive Reaction–Diffusion Framework for Modeling Alzheimer’s Disease Progression with Tau and Amyloid-$$\beta $$ PET Scans
inferred← citeshttps://doi.org/10.1007/978-981-16-7977-3_19openalex-snapshot
PET Imaging of Amyloid and Tau in Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-981-32-9358-8_15openalex-snapshot
Associations Between APOE Variants, Tau and α-Synuclein
inferred← citeshttps://doi.org/10.1007/978-981-32-9628-2_2openalex-snapshot
Therapeutic Vaccines Targeting Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/978-981-95-7121-5_1openalex-snapshot
Introduction: Stages and Progression of Alzheimer’s Disease
inferred← citeshttps://doi.org/10.1007/s00115-019-0697-3openalex-snapshot
Posteriore kortikale Atrophie – ein heterogenes Syndrom
inferred← citeshttps://doi.org/10.1007/s00115-019-0773-8openalex-snapshot
Innovation in der Diagnostik – mobile Technologien
inferred← citeshttps://doi.org/10.1007/s00234-025-03781-0openalex-snapshot
Spatiotemporal patterns of amyloid deposition as prognostic markers of Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00259-017-3790-5openalex-snapshot
18F–FDG PET diagnostic and prognostic patterns do not overlap in Alzheimer’s disease (AD) patients at the mild cognitive impairment (MCI) stage
inferred← citeshttps://doi.org/10.1007/s00259-017-3840-zopenalex-snapshot
Multimodal correlation of dynamic [18F]-AV-1451 perfusion PET and neuronal hypometabolism in [18F]-FDG PET
inferred← citeshttps://doi.org/10.1007/s00259-018-4012-5openalex-snapshot
Dual tracer tau PET imaging reveals different molecular targets for 11C-THK5351 and 11C-PBB3 in the Alzheimer brain
inferred← citeshttps://doi.org/10.1007/s00259-018-4075-3openalex-snapshot
Distinct [18F]THK5351 binding patterns in primary progressive aphasia variants
inferred← citeshttps://doi.org/10.1007/s00259-018-4113-1openalex-snapshot
The relationship between CSF biomarkers and cerebral metabolism in early-onset Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00259-018-4125-xopenalex-snapshot
Differences in gray and white matter 18F-THK5351 uptake between behavioral-variant frontotemporal dementia and other dementias
inferred← citeshttps://doi.org/10.1007/s00259-019-04342-3openalex-snapshot
Level of education mitigates the impact of tau pathology on neuronal function
inferred← citeshttps://doi.org/10.1007/s00259-019-04488-0openalex-snapshot
Advances in CNS PET: the state-of-the-art for new imaging targets for pathophysiology and drug development
inferred← citeshttps://doi.org/10.1007/s00259-019-04496-0openalex-snapshot
Head-to-head comparison of tau positron emission tomography tracers [18F]flortaucipir and [18F]RO948
inferred← citeshttps://doi.org/10.1007/s00259-019-04519-wopenalex-snapshot
Three-year changes of cortical 18F-FDG in amnestic vs. non-amnestic sporadic early-onset Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00259-019-04640-wopenalex-snapshot
A future for PET imaging in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00259-019-4273-7openalex-snapshot
Imaging of tau deposits in adults with Niemann-Pick type C disease: a case-control study
inferred← citeshttps://doi.org/10.1007/s00259-020-04758-2openalex-snapshot
Regional [18F]flortaucipir PET is more closely associated with disease severity than CSF p-tau in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00259-020-04831-wopenalex-snapshot
Tau pathology and relative cerebral blood flow are independently associated with cognition in Alzheimer’s disease
inferred← citeshttps://doi.org/10.1007/s00259-020-04923-7openalex-snapshot
Tau PET imaging with 18F-PI-2620 in aging and neurodegenerative diseases
inferred← citeshttps://doi.org/10.1007/s00259-020-05099-wopenalex-snapshot
The impact of demographic, clinical, genetic, and imaging variables on tau PET status
inferred← citeshttps://doi.org/10.1007/s00259-020-05118-wopenalex-snapshot
Clinical validity of increased cortical uptake of [18F]flortaucipir on PET as a biomarker for Alzheimer’s disease in the context of a structured 5-phase biomarker development framework
inferred← citeshttps://doi.org/10.1007/s00259-020-05152-8openalex-snapshot
Comparing ATN-T designation by tau PET visual reads, tau PET quantification, and CSF PTau181 across three cohorts
inferred← citeshttps://doi.org/10.1007/s00259-020-05183-1openalex-snapshot
Indication of retrograde tau spreading along Braak stages and functional connectivity pathways
inferred← citeshttps://doi.org/10.1007/s00259-021-05192-8openalex-snapshot
Differential associations of APOE-ε2 and APOE-ε4 alleles with PET-measured amyloid-β and tau deposition in older individuals without dementia
inferred← citeshttps://doi.org/10.1007/s00259-021-05401-4openalex-snapshot
A multicenter comparison of [18F]flortaucipir, [18F]RO948, and [18F]MK6240 tau PET tracers to detect a common target ROI for differential diagnosis
inferred← citeshttps://doi.org/10.1007/s00259-021-05662-zopenalex-snapshot
Image-level trajectory inference of tau pathology using variational autoencoder for Flortaucipir PET
inferred← citeshttps://doi.org/10.1007/s00259-021-05669-6openalex-snapshot
Differential associations between neocortical tau pathology and blood flow with cognitive deficits in early-onset vs late-onset Alzheimer’s disease
showing the first 200 of 1173 · page through all 1173
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.