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Matrix cross-linking lysyl oxidases are induced in response to myocardial infarction and promote cardiac dysfunction

https://doi.org/10.1093/cvr/cvv214
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1 of the 55 references listed on this page is in the Retraction Watch database as retracted (export 2026-10-21): 10.1038/nature04695 (notice 2020-03-18).

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/cmdc.200900247openalex-snapshot
Bioreductively Activated Lysyl Oxidase Inhibitors against Hypoxic Tumours
inferredcites →https://doi.org/10.1002/jcb.20605openalex-snapshot
Diverse biological functions of extracellular collagen processing enzymes
inferredcites →https://doi.org/10.1002/path.1700730126openalex-snapshot
The aortic and skeletal lesions of lathyrism in rats on a diet of sweet pea
inferredcites →https://doi.org/10.1006/cbir.1997.0130openalex-snapshot
STIMULATION OF COLLAGEN SYNTHESIS IN RAT CARDIAC FIBROBLASTS BY EXPOSURE TO HYPOXIC CULTURE CONDITIONS AND SUPPRESSION OF THE EFFECT BY NATRIURETIC PEPTIDES
inferredcites →https://doi.org/10.1007/978-1-59745-019-5_9openalex-snapshot
Analysis of Cardiac Myocyte Biology in Transgenic Mice: A Protocol for Preparation of Neonatal Mouse Cardiac Myocyte Cultures
inferredcites →https://doi.org/10.1007/bf01906668openalex-snapshot
Connective tissue content and myocardial stiffness in pressure overload hypertrophy A combined study of morphologic, morphometric, biochemical, and mechanical parameters
inferredcites →https://doi.org/10.1007/s00018-013-1349-6openalex-snapshot
The pathogenesis of cardiac fibrosis
inferredcites →https://doi.org/10.1007/s00395-008-0739-7openalex-snapshot
Transforming growth factor beta inhibition increases mortality and left ventricular dilatation after myocardial infarction
inferredcites →https://doi.org/10.1016/j.ajpath.2014.11.009openalex-snapshot
Disruption of Collagen Homeostasis Can Reverse Established Age-Related Myocardial Fibrosis
inferredcites →https://doi.org/10.1016/j.bbamcr.2012.08.023openalex-snapshot
Fibroblasts in post-infarction inflammation and cardiac repair
inferredcites →https://doi.org/10.1016/j.ccr.2008.11.012openalex-snapshot
Hypoxia-Induced Lysyl Oxidase Is a Critical Mediator of Bone Marrow Cell Recruitment to Form the Premetastatic Niche
inferredcites →https://doi.org/10.1016/j.cell.2009.10.027openalex-snapshot
Matrix Crosslinking Forces Tumor Progression by Enhancing Integrin Signaling
inferredcites →https://doi.org/10.1016/j.cyto.2011.03.024openalex-snapshot
Induction of cardiac fibroblast lysyl oxidase by TGF-β1 requires PI3K/Akt, Smad3, and MAPK signaling
inferredcites →https://doi.org/10.1016/j.jacc.2005.08.045openalex-snapshot
Hypoxia-Inducible Factor 1-Alpha Reduces Infarction and Attenuates Progression of Cardiac Dysfunction After Myocardial Infarction in the Mouse
inferredcites →https://doi.org/10.1016/j.yjmcc.2010.10.033openalex-snapshot
Transforming growth factor (TGF)-β signaling in cardiac remodeling
inferredcites →https://doi.org/10.1016/j.yjmcc.2014.08.008openalex-snapshot
Attenuated development of cardiac fibrosis in left ventricular pressure overload by SM16, an orally active inhibitor of ALK5
inferredcites →https://doi.org/10.1016/s0006-291x(72)80203-1openalex-snapshot
On the inhibition of lysyl oxidase by β-aminopropionitrile
inferredcites →https://doi.org/10.1016/s0008-6363(02)00733-2openalex-snapshot
Cross-linking influences the impact of quantitative changes in myocardial collagen on cardiac stiffness and remodelling in hypertension in rats
inferredcites →https://doi.org/10.1017/s143192761101227xopenalex-snapshot
Myofibroblasts in the Infarct Area: Concepts and Challenges
inferredcites →https://doi.org/10.1038/hr.2012.92openalex-snapshot
Cardiomyocyte-specific transgenic expression of lysyl oxidase-like protein-1 induces cardiac hypertrophy in mice
inferredcites →https://doi.org/10.1038/nature04695openalex-snapshot
Lysyl oxidase is essential for hypoxia-induced metastasis
inferredcites →https://doi.org/10.1038/nm1613openalex-snapshot
Endothelial-to-mesenchymal transition contributes to cardiac fibrosis
inferredcites →https://doi.org/10.1038/nrcardio.2009.199openalex-snapshot
Myocardial remodeling after infarction: the role of myofibroblasts
inferredcites →https://doi.org/10.1038/nrcardio.2012.158openalex-snapshot
Myofibroblast-mediated mechanisms of pathological remodelling of the heart
inferredcites →https://doi.org/10.1056/nejm200003023420904openalex-snapshot
Early Expression of Angiogenesis Factors in Acute Myocardial Ischemia and Infarction
inferredcites →https://doi.org/10.1073/pnas.1113483108openalex-snapshot
Hypoxia-inducible factor 1 is a master regulator of breast cancer metastatic niche formation
inferredcites →https://doi.org/10.1093/cvr/cvn315openalex-snapshot
A synthetic peptide from transforming growth factor- 1 type III receptor prevents myocardial fibrosis in spontaneously hypertensive rats
inferredcites →https://doi.org/10.1093/cvr/cvt100openalex-snapshot
Osteopontin-mediated myocardial fibrosis in heart failure: a role for lysyl oxidase?
inferredcites →https://doi.org/10.1093/cvr/cvu068openalex-snapshot
Induction of the calcineurin variant CnAβ1 after myocardial infarction reduces post-infarction ventricular remodelling by promoting infarct vascularization
inferredcites →https://doi.org/10.1093/hmg/ddt614openalex-snapshot
Hypoxia-induced epigenetic modifications are associated with cardiac tissue fibrosis and the development of a myofibroblast-like phenotype
inferredcites →https://doi.org/10.1111/j.1749-6632.1985.tb51167.xopenalex-snapshot
Post‐Translational Processing of Procollagens
inferredcites →https://doi.org/10.1111/j.1755-5922.2010.00207.xopenalex-snapshot
Temporal and Spatial Expression of Matrix Metalloproteinases and Tissue Inhibitors of Metalloproteinases Following Myocardial Infarction
inferredcites →https://doi.org/10.1111/j.1755-5922.2010.00228.xopenalex-snapshot
Targeting Fibrosis for the Treatment of Heart Failure: A Role for Transforming Growth Factor‐β
inferredcites →https://doi.org/10.1128/mcb.00036-13openalex-snapshot
LOXL4 Is Induced by Transforming Growth Factor β1 through Smad and JunB/Fra2 and Contributes to Vascular Matrix Remodeling
inferredcites →https://doi.org/10.1152/ajpheart.00335.2010openalex-snapshot
Role of lysyl oxidase in myocardial fibrosis: from basic science to clinical aspects
inferredcites →https://doi.org/10.1152/ajpheart.00398.2009openalex-snapshot
Myocardial lysyl oxidase regulation of cardiac remodeling in a murine model of diet-induced metabolic syndrome
inferredcites →https://doi.org/10.1152/ajpheart.00529.2009openalex-snapshot
Inhibition of transforming growth factor-β signaling induces left ventricular dilation and dysfunction in the pressure-overloaded heart
inferredcites →https://doi.org/10.1152/ajpheart.01048.2009openalex-snapshot
Targeted inhibition of activin receptor-like kinase 5 signaling attenuates cardiac dysfunction following myocardial infarction
inferredcites →https://doi.org/10.1152/ajpheart.1994.266.1.h354openalex-snapshot
Regional differences in LV collagen accumulation and mature cross-linking after myocardial infarction in rats
inferredcites →https://doi.org/10.1152/ajpheart.1995.269.3.h863openalex-snapshot
Inhibition of collagen cross-linking: effects on fibrillar collagen and ventricular diastolic function
inferredcites →https://doi.org/10.1161/01.cir.96.6.1991openalex-snapshot
Myocardial Stiffness Is Attributed to Alterations in Cross-Linked Collagen Rather Than Total Collagen or Phenotypes in Spontaneously Hypertensive Rats
inferredcites →https://doi.org/10.1161/01.res.43.4.632openalex-snapshot
The effect of lathyrogen beta-amino proprionitrile (BAPN) on the mechanical properties of experimentally hypertrophied rat cardiac muscle.
inferredcites →https://doi.org/10.1161/01.res.53.3.378openalex-snapshot
Myocardial healing and repair after experimental infarction in the rabbit.
inferredcites →https://doi.org/10.1161/01.res.62.4.757openalex-snapshot
Collagen remodeling of the pressure-overloaded, hypertrophied nonhuman primate myocardium.
inferredcites →https://doi.org/10.1161/circresaha.115.303794openalex-snapshot
Developmental Heterogeneity of Cardiac Fibroblasts Does Not Predict Pathological Proliferation and Activation
inferredcites →https://doi.org/10.1161/circulationaha.110.012211openalex-snapshot
Calcineurin Splicing Variant Calcineurin Aβ1 Improves Cardiac Function After Myocardial Infarction Without Inducing Hypertrophy
inferredcites →https://doi.org/10.1161/jaha.112.000034openalex-snapshot
Impaired Collagen Biosynthesis and Cross‐linking in Aorta of Patients With Bicuspid Aortic Valve
inferredcites →https://doi.org/10.1167/iovs.11-7287openalex-snapshot
Transforming Growth Factor–β Induces Extracellular Matrix Protein Cross-Linking Lysyl Oxidase ( LOX ) Genes in Human Trabecular Meshwork Cells
inferredcites →https://doi.org/10.1172/jci74783openalex-snapshot
Resident fibroblast lineages mediate pressure overload–induced cardiac fibrosis
inferredcites →https://doi.org/10.1210/en.2008-0165openalex-snapshot
Transforming Growth Factor-β Blockade Down-Regulates the Renin-Angiotensin System and Modifies Cardiac Remodeling after Myocardial Infarction
inferredcites →https://doi.org/10.1258/ebm.2012.012070openalex-snapshot
Immunohistochemical detection of differentially localized up-regulation of lysyl oxidase and down-regulation of matrix metalloproteinase-1 in rhesus monkey model of chronic myocardial infarction
inferredcites →https://doi.org/10.1371/journal.pone.0064986openalex-snapshot
Left and Right Ventricle Late Remodeling Following Myocardial Infarction in Rats
inferredcites →https://doi.org/10.14670/hh-24.651openalex-snapshot
Lysyl oxidases in mammalian development and certain pathological conditions.
inferredcites →https://doi.org/10.1681/asn.2013070799openalex-snapshot
A Pathogenetic Role for Endothelin-1 in Peritoneal Dialysis-Associated Fibrosis
inferredcites →https://doi.org/10.2147/ott.s38110openalex-snapshot
The potential for targeting extracellular LOX proteins in human malignancy

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4654347openalex-extracted
Inhibition of Lysyl Oxidase Family of Enzymes Has No Effect on Healthy Fibroblast – Extracellular Matrix Cross-Talk in vitro
inferred← citeshttps://doi.org/10.1002/adhm.202001856openalex-snapshot
The Plasticity of Nanofibrous Matrix Regulates Fibroblast Activation in Fibrosis
inferred← citeshttps://doi.org/10.1002/cbf.4066openalex-snapshot
Periostin regulates lysyl oxidase through ERK1/2 MAPK‐dependent serum response factor in activated cardiac fibroblasts
inferred← citeshttps://doi.org/10.1002/cphy.c160046openalex-snapshot
Cardiac Fibrosis and Arrhythmogenesis
inferred← citeshttps://doi.org/10.1002/j.2040-4603.2017.tb00771.xopenalex-snapshot
Cardiac Fibrosis and Arrhythmogenesis
inferred← citeshttps://doi.org/10.1002/jex2.125openalex-snapshot
Cardiomyocyte intercellular signalling increases oxidative stress and reprograms the global‐ and phospho‐proteome of cardiac fibroblasts
inferred← citeshttps://doi.org/10.1002/stem.3009openalex-snapshot
Concise Review: Reduction of Adverse Cardiac Scarring Facilitates Pluripotent Stem Cell-Based Therapy for Myocardial Infarction
inferred← citeshttps://doi.org/10.1002/term.3118openalex-snapshot
Fibrin hydrogels promote scar formation and prevent therapeutic angiogenesis in the heart
inferred← citeshttps://doi.org/10.1007/978-3-031-23965-6_10openalex-snapshot
Cardiac Microvascular Endothelial Cells and Pressure Overload-Induced Cardiac Fibrosis
inferred← citeshttps://doi.org/10.1007/s00018-022-04133-4openalex-snapshot
Zyxin protects from hypertension-induced cardiac dysfunction
inferred← citeshttps://doi.org/10.1007/s00395-021-00846-yopenalex-snapshot
AMPKα1 deletion in myofibroblasts exacerbates post-myocardial infarction fibrosis by a connexin 43 mechanism
inferred← citeshttps://doi.org/10.1007/s00441-016-2431-9openalex-snapshot
Cardiac fibrosis in myocardial infarction—from repair and remodeling to regeneration
inferred← citeshttps://doi.org/10.1007/s10554-026-03682-0openalex-snapshot
Next-generation molecular imaging of cardiac fibrosis
inferred← citeshttps://doi.org/10.1007/s10741-016-9536-9openalex-snapshot
The fibrosis-cell death axis in heart failure
inferred← citeshttps://doi.org/10.1007/s11748-018-0969-zopenalex-snapshot
Sustained release of basic fibroblast growth factor using gelatin hydrogel improved left ventricular function through the alteration of collagen subtype in a rat chronic myocardial infarction model
inferred← citeshttps://doi.org/10.1007/s11897-023-00630-0openalex-snapshot
Hydrogels for Cardiac Restorative Support: Relevance of Gelation Mechanisms for Prospective Clinical Use
inferred← citeshttps://doi.org/10.1007/s11926-018-0710-zopenalex-snapshot
Matrix Stiffness: the Conductor of Organ Fibrosis
inferred← citeshttps://doi.org/10.1007/s12012-026-10167-yopenalex-snapshot
Dual-Metal Regulation of Cardiac Remodeling: Targeting the LOXL2-Ferroptosis Axis in Metal-Induced Cardiac Dysfunction
inferred← citeshttps://doi.org/10.1016/b978-0-12-801238-3.99828-3openalex-snapshot
OBSOLETE: Cardiac Fibroblast
inferred← citeshttps://doi.org/10.1016/b978-0-12-809657-4.99828-4openalex-snapshot
Cardiac Fibroblast
inferred← citeshttps://doi.org/10.1016/b978-0-443-45056-3.00015-7openalex-snapshot
Mesenchymal stem cells and extracellular vesicles in cardiac regeneration
inferred← citeshttps://doi.org/10.1016/bs.ctm.2021.07.004openalex-snapshot
Mechanotransduction in fibrosis: Mechanisms and treatment targets
inferred← citeshttps://doi.org/10.1016/j.addr.2021.03.014openalex-snapshot
Biologics and their delivery systems: Trends in myocardial infarction
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2019.06.001openalex-snapshot
The complex dynamics of myocardial interstitial fibrosis in heart failure. Focus on collagen cross-linking
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2019.07.016openalex-snapshot
Reprint of “The complex dynamics of myocardial interstitial fibrosis in heart failure. Focus on collagen cross-linking”
inferred← citeshttps://doi.org/10.1016/j.bbrc.2020.05.115openalex-snapshot
Targeting JMJD3 histone demethylase mediates cardiac fibrosis and cardiac function following myocardial infarction
inferred← citeshttps://doi.org/10.1016/j.bbrc.2023.149360openalex-snapshot
Integrin αV mediated activation of myofibroblast via mechanoparacrine of transforming growth factor β1 in promoting fibrous scar formation after myocardial infarction
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2017.01.030openalex-snapshot
Biomechanically primed liver microtumor array as a high-throughput mechanopharmacological screening platform for stroma-reprogrammed combinatorial therapy
inferred← citeshttps://doi.org/10.1016/j.carpath.2017.04.001openalex-snapshot
Association of altered collagen content and lysyl oxidase expression in degenerative mitral valve disease
inferred← citeshttps://doi.org/10.1016/j.carpath.2024.107711openalex-snapshot
Tocilizumab does not ameliorate inflammation-induced left ventricular dysfunction in a collagen-induced arthritis rat model
inferred← citeshttps://doi.org/10.1016/j.cbpa.2024.111769openalex-snapshot
The influence of triiodothyronine on the immune response and extracellular matrix remodeling during zebrafish heart regeneration
inferred← citeshttps://doi.org/10.1016/j.clinthera.2020.09.006openalex-snapshot
The Cardiac Injury Immune Response as a Target for Regenerative and Cellular Therapies
inferred← citeshttps://doi.org/10.1016/j.exer.2016.11.011openalex-snapshot
Major review: Exfoliation syndrome; advances in disease genetics, molecular biology, and epidemiology
inferred← citeshttps://doi.org/10.1016/j.exer.2019.04.019openalex-snapshot
Decreased lysyl oxidase level protects against development of retinal vascular lesions in diabetic retinopathy
inferred← citeshttps://doi.org/10.1016/j.ijcard.2019.01.078openalex-snapshot
Loss of Sox9 in cardiomyocytes delays the onset of cardiac hypertrophy and fibrosis
inferred← citeshttps://doi.org/10.1016/j.ijcard.2022.01.050openalex-snapshot
Exploring the potential relationship between collagen cross-linking and impaired myocardial relaxation in Marfan syndrome: An observational study using serum biomarkers
inferred← citeshttps://doi.org/10.1016/j.jacbts.2022.11.006openalex-snapshot
PDGF-AB Reduces Myofibroblast Differentiation Without Increasing Proliferation After Myocardial Infarction
inferred← citeshttps://doi.org/10.1016/j.jaccao.2022.04.002openalex-snapshot
Cardiovascular Disease in Myeloproliferative Neoplasms
inferred← citeshttps://doi.org/10.1016/j.jare.2023.01.018openalex-snapshot
Single-cell analysis reveals lysyl oxidase (Lox)+ fibroblast subset involved in cardiac fibrosis of diabetic mice
inferred← citeshttps://doi.org/10.1016/j.jff.2020.103895openalex-snapshot
Stevioside improved hyperglycemia-induced cardiac dysfunction by attenuating the development of fibrosis and promoting the degradation of established fibrosis
inferred← citeshttps://doi.org/10.1016/j.jnutbio.2022.109180openalex-snapshot
Copper promotes cardiac functional recovery via suppressing the transformation of fibroblasts to myofibroblasts in ischemia-infarcted monkey hearts
inferred← citeshttps://doi.org/10.1016/j.jsxm.2018.01.011openalex-snapshot
Amelioration of Cavernosal Fibrosis and Erectile Function by Lysyl Oxidase Inhibition in a Rat Model of Cavernous Nerve Injury
inferred← citeshttps://doi.org/10.1016/j.lfs.2023.121598openalex-snapshot
Collagen matricryptin promotes cardiac function by mediating scar formation
inferred← citeshttps://doi.org/10.1016/j.matbio.2017.12.001openalex-snapshot
Understanding cardiac extracellular matrix remodeling to develop biomarkers of myocardial infarction outcomes
inferred← citeshttps://doi.org/10.1016/j.matbio.2021.05.005openalex-snapshot
Collagen denaturation in the infarcted myocardium involves temporally distinct effects of MT1-MMP-dependent proteolysis and mechanical tension
inferred← citeshttps://doi.org/10.1016/j.matbio.2021.12.002openalex-snapshot
ADAM15 is required for optimal collagen cross-linking and scar formation following myocardial infarction
inferred← citeshttps://doi.org/10.1016/j.matbio.2024.06.005openalex-snapshot
MiR-214–3p regulates Piezo1, lysyl oxidases and mitochondrial function in human cardiac fibroblasts
inferred← citeshttps://doi.org/10.1016/j.matbio.2026.102056openalex-snapshot
Fibromodulin: From Matrisome Architecture to Master Regulator of Cellular Fate
inferred← citeshttps://doi.org/10.1016/j.molmed.2015.12.006openalex-snapshot
The Living Scar – Cardiac Fibroblasts and the Injured Heart
inferred← citeshttps://doi.org/10.1016/j.mtbio.2024.100978openalex-snapshot
Hydrogel-based cardiac repair and regeneration function in the treatment of myocardial infarction
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2020.107575openalex-snapshot
Mechano-therapeutics: Targeting Mechanical Signaling in Fibrosis and Tumor Stroma
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2017.06.013openalex-snapshot
Detrimental role of lysyl oxidase in cardiac remodeling
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2019.04.007openalex-snapshot
Matrix metalloproteinase-12 produced by Ly6Clow macrophages prolongs the survival after myocardial infarction by preventing neutrophil influx
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2019.06.015openalex-snapshot
Inhibition of Rho-associated kinases suppresses cardiac myofibroblast function in engineered connective and heart muscle tissues
inferred← citeshttps://doi.org/10.1038/s41419-020-03211-4openalex-snapshot
Legumain is a predictor of all-cause mortality and potential therapeutic target in acute myocardial infarction
inferred← citeshttps://doi.org/10.1038/s41467-019-14263-2openalex-snapshot
Macrophages directly contribute collagen to scar formation during zebrafish heart regeneration and mouse heart repair
inferred← citeshttps://doi.org/10.1038/s41536-017-0027-yopenalex-snapshot
Heart regeneration in the salamander relies on macrophage-mediated control of fibroblast activation and the extracellular landscape
inferred← citeshttps://doi.org/10.1038/s41569-022-00799-2openalex-snapshot
Cardiac fibroblasts and mechanosensation in heart development, health and disease
inferred← citeshttps://doi.org/10.1038/s41569-026-01322-7openalex-snapshot
The cardiac 4D nucleome: nuclear and chromatin dynamics across development, disease and ageing
inferred← citeshttps://doi.org/10.1038/s41598-017-16255-yopenalex-snapshot
Pyridoxamine improves survival and limits cardiac dysfunction after MI
inferred← citeshttps://doi.org/10.1038/s41598-018-23881-7openalex-snapshot
Cardiac (myo)fibroblasts modulate the migration of monocyte subsets
inferred← citeshttps://doi.org/10.1038/s41598-018-35314-6openalex-snapshot
Loss of miR-144 signaling interrupts extracellular matrix remodeling after myocardial infarction leading to worsened cardiac function
inferred← citeshttps://doi.org/10.1038/s41598-020-69254-xopenalex-snapshot
Identification of potential biomarkers for predicting the early onset of diabetic cardiomyopathy in a mouse model
inferred← citeshttps://doi.org/10.1038/s41598-024-56816-6openalex-snapshot
Proteomic study of left ventricle and cortex in rats after myocardial infarction
inferred← citeshttps://doi.org/10.1038/s42003-021-02354-0openalex-snapshot
An in situ activity assay for lysyl oxidases
inferred← citeshttps://doi.org/10.1038/s42003-025-08083-yopenalex-snapshot
A secretome atlas of cardiac fibroblasts from healthy and infarcted mouse hearts
inferred← citeshttps://doi.org/10.1038/s43856-025-01349-wopenalex-snapshot
Molecular insights in HIV-associated cardiac dysfunction
inferred← citeshttps://doi.org/10.1039/9781788015783-00145openalex-snapshot
Inhibition of LOXL2 and Other Lysyl Oxidase (Like) Enzymes: Intervention at the Core of Fibrotic Pathology
inferred← citeshttps://doi.org/10.1042/cs20191014openalex-snapshot
Neuron-derived orphan receptor-1 modulates cardiac gene expression and exacerbates angiotensin II-induced cardiac hypertrophy
inferred← citeshttps://doi.org/10.1042/ebc20180051openalex-snapshot
Collagen cross-linking mediated by lysyl hydroxylase 2: an enzymatic battlefield to combat fibrosis
inferred← citeshttps://doi.org/10.1053/j.jvca.2025.08.045openalex-snapshot
Evolving Management of Post-myocardial Infarction Ventricular Septal or Free Wall Rupture
inferred← citeshttps://doi.org/10.1073/pnas.1910650117openalex-snapshot
Matrix-transmitted paratensile signaling enables myofibroblast – fibroblast cross talk in fibrosis expansion
inferred← citeshttps://doi.org/10.1074/jbc.ra119.007674openalex-snapshot
A hierarchical network of hypoxia-inducible factor and SMAD proteins governs procollagen lysyl hydroxylase 2 induction by hypoxia and transforming growth factor β1
inferred← citeshttps://doi.org/10.1080/02713683.2017.1390770openalex-snapshot
Crosslinking Enzyme Lysyl Oxidase Modulates Scleral Remodeling in Form-Deprivation Myopia
inferred← citeshttps://doi.org/10.1093/cvr/cvaa137openalex-snapshot
Stimulating pro-reparative immune responses to prevent adverse cardiac remodelling: consensus document from the joint 2019 meeting of the ESC Working Groups of cellular biology of the heart and myocardial function
inferred← citeshttps://doi.org/10.1093/cvr/cvag001openalex-snapshot
Dual roles of ADAM17: from fibroblast foe to myofibroblast friend
inferred← citeshttps://doi.org/10.1093/cvr/cvx053openalex-snapshot
Tissue transglutaminase induction in the pressure-overloaded myocardium regulates matrix remodelling
inferred← citeshttps://doi.org/10.1093/cvr/cvx105openalex-snapshot
Myocardial fibrosis in response to pressure overload: elucidating the contribution of tissue transglutaminase
inferred← citeshttps://doi.org/10.1093/cvr/cvy265openalex-snapshot
New heights and hot topics inCardiovascular Research
inferred← citeshttps://doi.org/10.1093/cvr/cvz176openalex-snapshot
Role of the lysyl oxidase enzyme family in cardiac function and disease
inferred← citeshttps://doi.org/10.1097/ijg.0000000000000910openalex-snapshot
Lysyl Oxidases: Functions and Disorders
inferred← citeshttps://doi.org/10.1101/2024.10.13.618070openalex-snapshot
From Transcriptomic Signatures to Active-Site Docking and ADMET Properties Prediction: Bioinformatics-Guided Exploration of Binding Mechanisms of Cardioprotective Phytoconstituents Against Dysregulated Targets in Hypertrophic Cardiomyopathy
inferred← citeshttps://doi.org/10.1101/2024.12.02.626322openalex-snapshot
Pharmacological inhibition of 11ßhydroxysteroid dehydrogenase type 1 after myocardial infarction targets extracellular matrix processing and preserves cardiac function in a translational mini-pig model
inferred← citeshttps://doi.org/10.1111/bph.70297openalex-snapshot
Pharmacological inhibition of hydroxysteroid 11‐beta dehydrogenase type 1 (11‐βHSD1) after myocardial infarction preserves cardiac function in a translational mini‐pig model
inferred← citeshttps://doi.org/10.1111/febs.14718openalex-snapshot
Cardiomyocyte calcineurin is required for the onset and progression of cardiac hypertrophy and fibrosis in adult mice
inferred← citeshttps://doi.org/10.1111/febs.70212openalex-snapshot
Collagen XV preserves heart function and protects from pathological remodelling after myocardial infarction
inferred← citeshttps://doi.org/10.1111/febs.70418openalex-snapshot
Decoding ECM remodeling: proteases as gatekeepers of Lysyl oxidase family‐mediated crosslinking
inferred← citeshttps://doi.org/10.1111/jcmm.14074openalex-snapshot
The lysyl oxidase like 2/3 enzymatic inhibitor, PXS‐5153A, reduces crosslinks and ameliorates fibrosis
inferred← citeshttps://doi.org/10.1111/jcmm.14313openalex-snapshot
The role and mechanism of transforming growth factor beta 3 in human myocardial infarction‐induced myocardial fibrosis
inferred← citeshttps://doi.org/10.1146/annurev-physiol-043024-115512openalex-snapshot
Scarred by Fibrosis: The Heart–Kidney Disease Connection
inferred← citeshttps://doi.org/10.1152/ajpcell.00176.2023openalex-snapshot
Lysyl oxidase like-2 in fibrosis and cardiovascular disease
inferred← citeshttps://doi.org/10.1152/ajpcell.01043.2024openalex-snapshot
The theatrics of collagens in the myocardium: the supreme architect of the fibrotic heart
inferred← citeshttps://doi.org/10.1152/ajpheart.00086.2018openalex-snapshot
Inhibitor of lysyl oxidase improves cardiac function and the collagen/MMP profile in response to volume overload
inferred← citeshttps://doi.org/10.1152/ajpheart.00110.2024openalex-snapshot
Sex differences and role of lysyl oxidase-like 2 in angiotensin II-induced hypertension in mice
inferred← citeshttps://doi.org/10.1152/ajpheart.00112.2019openalex-snapshot
CD8+T-cells negatively regulate inflammation post-myocardial infarction
inferred← citeshttps://doi.org/10.1152/ajpheart.00138.2022openalex-snapshot
Ectonucleoside triphosphate diphosphohydrolase-1 (CD39) impacts TGF-β1 responses: insights into cardiac fibrosis and function following myocardial infarction
inferred← citeshttps://doi.org/10.1152/ajpheart.00315.2018openalex-snapshot
LOXury of inhibiting fibrosis in volume overload cardiomyopathy
inferred← citeshttps://doi.org/10.1152/ajpheart.00453.2017openalex-snapshot
Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction
inferred← citeshttps://doi.org/10.1152/ajpheart.00594.2022openalex-snapshot
Combining three independent pathological stressors induces a heart failure with preserved ejection fraction phenotype
inferred← citeshttps://doi.org/10.1152/ajpheart.00688.2020openalex-snapshot
Gαi-biased apelin analog protects against isoproterenol-induced myocardial dysfunction in rats
inferred← citeshttps://doi.org/10.1155/2020/4375127openalex-snapshot
Macrophage Activities in Myocardial Infarction and Heart Failure
inferred← citeshttps://doi.org/10.1161/circresaha.116.309685openalex-snapshot
Titin-Based Cardiac Myocyte Stiffening Contributes to Early Adaptive Ventricular Remodeling After Myocardial Infarction
inferred← citeshttps://doi.org/10.1161/circresaha.121.319482openalex-snapshot
Matricellular Protein Cilp1 Promotes Myocardial Fibrosis in Response to Myocardial Infarction
inferred← citeshttps://doi.org/10.1161/circresaha.122.321123openalex-snapshot
Tropoelastin Improves Post-Infarct Cardiac Function
inferred← citeshttps://doi.org/10.1161/hypertensionaha.120.16352openalex-snapshot
Transient ACE (Angiotensin-Converting Enzyme) Inhibition Suppresses Future Fibrogenic Capacity and Heterogeneity of Cardiac Fibroblast Subpopulations
inferred← citeshttps://doi.org/10.1165/rcmb.2017-0252ocopenalex-snapshot
Lysyl Oxidase–Like 1 Protein Deficiency Protects Mice from Adenoviral Transforming Growth Factor-β1–induced Pulmonary Fibrosis
inferred← citeshttps://doi.org/10.1167/iovs.17-22963openalex-snapshot
Posttranscriptional Regulation of LOXL1 Expression Via Alternative Splicing and Nonsense-Mediated mRNA Decay as an Adaptive Stress Response
inferred← citeshttps://doi.org/10.1172/jci98215openalex-snapshot
Specialized fibroblast differentiated states underlie scar formation in the infarcted mouse heart
inferred← citeshttps://doi.org/10.1177/15353702211042020openalex-snapshot
Extracellular matrix remodeling is associated with the survival of cardiomyocytes in the subendocardial region of the ischemic myocardium
inferred← citeshttps://doi.org/10.1186/s43556-026-00504-6openalex-snapshot
Cardiac regeneration and repair: the emerging mechanisms and therapeutic approaches
inferred← citeshttps://doi.org/10.1186/s43556-026-00587-1openalex-snapshot
Cardiac fibrosis: mechanistic insights and translational advances
inferred← citeshttps://doi.org/10.1517/14728222.2016.1151003openalex-snapshot
Lysyl Oxidase Isoforms and Potential Therapeutic Opportunities for Fibrosis and Cancer
inferred← citeshttps://doi.org/10.17776/csj.1246590openalex-snapshot
Metabolic Infrastructure of Pregnant Women with Fetuses Having Nervous System Abnormalities; Metabolomic Analysis
inferred← citeshttps://doi.org/10.2147/dddt.s588634openalex-snapshot
Astragaloside IV in Combination with Tanshinone IIA Ameliorates Cardiac Fibrosis After Myocardial Infarction by Inhibiting CTR1/ATOX1/LOX Axis-Mediated Collagen Cross-Linking in Myofibroblasts
inferred← citeshttps://doi.org/10.2174/0109298673306240240612055116openalex-snapshot
Lysyl Oxidase-like 2 Dysregulation Increases the Risk of Post- Operative Fibrotic Scars Formation in the Female Reproductive Tract: A Novel Therapeutic Target to Reduce Fibrogenesis
inferred← citeshttps://doi.org/10.2174/1386207325666220517110651openalex-snapshot
Identification of Hypoxia-related Genes in Acute Myocardial Infarction using Bioinformatics Analysis
inferred← citeshttps://doi.org/10.23866/brnrev:2020-0012openalex-snapshot
Mechanotherapeutics for the Treatment of Idiopathic Pulmonary Fibrosis
inferred← citeshttps://doi.org/10.31083/rcm27152openalex-snapshot
The Role of Signalling Pathways in Myocardial Fibrosis in Hypertrophic Cardiomyopathy
inferred← citeshttps://doi.org/10.33846/hn50407openalex-snapshot
Percutaneous Coronary Intervention in Post-Infarction Ventricular Septal Rupture
inferred← citeshttps://doi.org/10.3389/fcell.2024.1496884openalex-snapshot
Molecular and metabolomic characterization of hiPSC-derived cardiac fibroblasts transitioning to myofibroblasts
inferred← citeshttps://doi.org/10.3389/fcvm.2019.00101openalex-snapshot
The Role of Macrophages in the Infarcted Myocardium: Orchestrators of ECM Remodeling
inferred← citeshttps://doi.org/10.3389/fcvm.2022.1015473openalex-snapshot
Transcriptome and proteome profiling of activated cardiac fibroblasts supports target prioritization in cardiac fibrosis
inferred← citeshttps://doi.org/10.3389/fcvm.2025.1501397openalex-snapshot
Subepicardial adipose genes contribute to the deterioration of heart failure preserved ejection fraction
inferred← citeshttps://doi.org/10.3389/fcvm.2026.1948993openalex-snapshot
Atrial fibroblast metabolic reprogramming in atrial fibrillation: a candidate mechanism within multicellular atrial cardiomyopathy
inferred← citeshttps://doi.org/10.3389/fmolb.2022.1030226openalex-snapshot
Exploring the cardiac ECM during fibrosis: A new era with next-gen proteomics
inferred← citeshttps://doi.org/10.3389/fonc.2025.1601261openalex-snapshot
Bibliometric analysis combined with visualization on universal trends and hot topics of LOX family in human diseases: 1995 to 2025
inferred← citeshttps://doi.org/10.3390/antiox11010075openalex-snapshot
Human Lysyl Oxidase Over-Expression Enhances Baseline Cardiac Oxidative Stress but Does Not Aggravate ROS Generation or Infarct Size Following Myocardial Ischemia-Reperfusion
inferred← citeshttps://doi.org/10.3390/biom9100610openalex-snapshot
Emerging Roles of Lysyl Oxidases in the Cardiovascular System: New Concepts and Therapeutic Challenges
inferred← citeshttps://doi.org/10.3390/biomedicines13051170openalex-snapshot
Emerging Epigenetic Therapies for the Treatment of Cardiac Fibrosis
inferred← citeshttps://doi.org/10.3390/cells10040770openalex-snapshot
Cardiac Fibrosis: Key Role of Integrins in Cardiac Homeostasis and Remodeling
inferred← citeshttps://doi.org/10.3390/cells10071824openalex-snapshot
Fibrosis, the Bad Actor in Cardiorenal Syndromes: Mechanisms Involved
inferred← citeshttps://doi.org/10.3390/cells8121483openalex-snapshot
The Role of Lysyl Oxidase Enzymes in Cardiac Function and Remodeling
inferred← citeshttps://doi.org/10.3390/ijms222111903openalex-snapshot
Increased Collagen Crosslinking in Stiff Clubfoot Tissue: Implications for the Improvement of Therapeutic Strategies
inferred← citeshttps://doi.org/10.3390/ijms26083846openalex-snapshot
Proteome Alterations in Cardiac Fibroblasts: Insights from Experimental Myocardial Infarction and Clinical Ischaemic Cardiomyopathy
inferred← citeshttps://doi.org/10.3390/jfb16050175openalex-snapshot
Wharton’s Jelly Bioscaffolds Improve Cardiac Repair with Bone Marrow Mononuclear Stem Cells in Rats
inferred← citeshttps://doi.org/10.3390/pharmaceutics14081599openalex-snapshot
Targeting Myocardial Fibrosis—A Magic Pill in Cardiovascular Medicine?
inferred← citeshttps://doi.org/10.3410/f.733068856.793545339openalex-snapshot
Faculty Opinions recommendation of Specialized fibroblast differentiated states underlie scar formation in the infarcted mouse heart.
inferred← citeshttps://doi.org/10.3410/f.733068856.793547256openalex-snapshot
Faculty Opinions recommendation of Specialized fibroblast differentiated states underlie scar formation in the infarcted mouse heart.
inferred← citeshttps://doi.org/10.70675/09e4e522z15b7z4673zb589z9a4fe2a72bc5openalex-snapshot
Étude du blocage pharmacologique de l'intégrine alpha v (CD51) dans le développement du remodelage cardiaque induit par surcharge de pression
inferred← citeshttps://doi.org/10.70675/7976cae4z9a2ez4880z87d3z064337f81368openalex-snapshot
Biomarqueurs de la fonction ventriculaire après un infarctus du myocarde : différences hommes-femmes
inferred← citeshttps://doi.org/10.7150/thno.51648openalex-snapshot
Group 2 innate lymphoid cells contribute to IL-33-mediated alleviation of cardiac fibrosis
inferred← citeshttps://doi.org/10.7150/thno.82543openalex-snapshot
Therapeutic silencing of lncRNA RMST alleviates cardiac fibrosis and improves heart function after myocardial infarction in mice and swine
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