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Extensive enteric nervous system abnormalities in mice transgenic for artificial chromosomes containing Parkinson disease-associated α-synuclein gene mutations precede central nervous system changes

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Current Protocols in Molecular Biology

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What's Wrong With My Mouse?

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The new mutation, E46K, of α‐synuclein causes parkinson and Lewy body dementia

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A large kindred with autosomal dominant Parkinson's disease

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Unbiased stereological estimation of the total number of neurons in the subdivisions of the rat hippocampus using the optical fractionator

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Glia cell number modulates sensitivity to MPTP in mice

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Bowel movement frequency in late‐life and incidental Lewy bodies

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Update on the genetics of Parkinson's disease

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Lesions outside the CNS in Parkinson's disease

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Constipation in parkinson's disease: Objective assessment and response to psyllium

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Critical appraisal of brain pathology staging related to presymptomatic and symptomatic cases of sporadic Parkinson’s disease

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Parkinson’s disease: premotor clinico-pathological correlations

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Neuroanatomy and Pathology of Sporadic Parkinson's Disease

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Normal variation in anorectal manometry

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Parkinson’s disease: a rethink of rodent models

inferredcites →https://doi.org/10.1007/s00401-008-0346-6openalex-snapshot

Applicability of current staging/categorization of α-synuclein pathology and their clinical relevance

inferredcites →https://doi.org/10.1007/s00401-008-0350-xopenalex-snapshot

Animal models of Parkinson’s disease progression

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Characterization of antibodies that selectively detect α-synuclein in pathological inclusions

inferredcites →https://doi.org/10.1007/s00401-008-0381-3openalex-snapshot

Controversies over the staging of α-synuclein pathology in Parkinson’s disease

inferredcites →https://doi.org/10.1007/s00415-002-1301-4openalex-snapshot

Staging of the intracerebral inclusion body pathology associated with idiopathic Parkinson's disease (preclinical and clinical stages)

inferredcites →https://doi.org/10.1007/s00441-004-0956-9openalex-snapshot

Stages in the development of Parkinson’s disease-related pathology

inferredcites →https://doi.org/10.1016/0003-2697(90)90595-zopenalex-snapshot

Molecular cloning: A laboratory manual

inferredcites →https://doi.org/10.1016/0014-2999(96)00403-7openalex-snapshot

Compounds possessing 5-HT3 receptor antagonistic activity inhibit intestinal propulsion in mice

inferredcites →https://doi.org/10.1016/0016-5085(93)90941-5openalex-snapshot

Fluid loading of the human colon: Effects on segmental transit and stool composition

inferredcites →https://doi.org/10.1016/j.expneurol.2007.05.010openalex-snapshot

Loss of enteric dopaminergic neurons and associated changes in colon motility in an MPTP mouse model of Parkinson's disease

inferredcites →https://doi.org/10.1016/j.expneurol.2007.08.006openalex-snapshot

In vivo alpha-synuclein overexpression in rodents: A useful model of Parkinson's disease?

inferredcites →https://doi.org/10.1016/j.jns.2009.08.020openalex-snapshot

Gastrointestinal dysfunction in Parkinson's Disease

inferredcites →https://doi.org/10.1016/j.neulet.2005.11.012openalex-snapshot

Gastric α-synuclein immunoreactive inclusions in Meissner's and Auerbach's plexuses in cases staged for Parkinson's disease-related brain pathology

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Exacerbated synucleinopathy in mice expressing A53T SNCA on a Snca null background

inferredcites →https://doi.org/10.1016/j.neuroscience.2008.02.074openalex-snapshot

Alpha-synuclein-immunopositive myenteric neurons and vagal preganglionic terminals: Autonomic pathway implicated in Parkinson's disease?

inferredcites →https://doi.org/10.1016/s0140-6736(04)17103-1openalex-snapshot

α-synuclein locus duplication as a cause of familial Parkinson's disease

inferredcites →https://doi.org/10.1016/s0140-6736(95)92707-7openalex-snapshot

Dopaminergic defect of enteric nervous system in Parkinson's disease patients with chronic constipation

inferredcites →https://doi.org/10.1016/s0197-4580(02)00065-9openalex-snapshot

Staging of brain pathology related to sporadic Parkinson’s disease

inferredcites →https://doi.org/10.1016/s1044-7431(03)00198-2openalex-snapshot

Transgenic mice expressing mutant A53T human alpha-synuclein show neuronal dysfunction in the absence of aggregate formation

inferredcites →https://doi.org/10.1016/s1474-4422(03)00307-7openalex-snapshot

Gastrointestinal dysfunction in Parkinson's disease

inferredcites →https://doi.org/10.1016/s1474-4422(15)00007-1openalex-snapshot

Gastrointestinal dysfunction in Parkinson's disease

inferredcites →https://doi.org/10.1038/42166openalex-snapshot

α-Synuclein in Lewy bodies

inferredcites →https://doi.org/10.1038/ng0298-106openalex-snapshot

AlaSOPro mutation in the gene encoding α-synuclein in Parkinson's disease

inferredcites →https://doi.org/10.1038/sj.mp.4000446openalex-snapshot

Alpha synuclein is present in Lewy bodies in sporadic Parkinson's disease

inferredcites →https://doi.org/10.1056/nejm2003ra020003openalex-snapshot

Alzheimer's Disease and Parkinson's Disease

inferredcites →https://doi.org/10.1073/pnas.0509567103openalex-snapshot

Lewy bodies

inferredcites →https://doi.org/10.1073/pnas.0610978104openalex-snapshot

GSTπ expression mediates dopaminergic neuron sensitivity in experimental parkinsonism

inferredcites →https://doi.org/10.1073/pnas.0802437105openalex-snapshot

GATA transcription factors directly regulate the Parkinson's disease-linked gene α-synuclein

inferredcites →https://doi.org/10.1073/pnas.0900096106openalex-snapshot

Highly pathogenic H5N1 influenza virus can enter the central nervous system and induce neuroinflammation and neurodegeneration

inferredcites →https://doi.org/10.1073/pnas.132197599openalex-snapshot

Human α-synuclein-harboring familial Parkinson's disease-linked Ala-53 → Thr mutation causes neurodegenerative disease with α-synuclein aggregation in transgenic mice

inferredcites →https://doi.org/10.1074/jbc.m308041200openalex-snapshot

Ubiquitination of α-Synuclein in Lewy Bodies Is a Pathological Event Not Associated with Impairment of Proteasome Function

inferredcites →https://doi.org/10.1080/00365520310003200openalex-snapshot

Constipation in Idiopathic Parkinson's Disease

inferredcites →https://doi.org/10.1093/brain/122.9.1637openalex-snapshot

Frontal, midbrain and striatal dopaminergic function in early and advanced Parkinson's disease A 3D [18F]dopa-PET study

inferredcites →https://doi.org/10.1093/brain/awh081openalex-snapshot

Association between cardiac denervation and parkinsonism caused by α‐synuclein gene triplication

inferredcites →https://doi.org/10.1093/hmg/10.26.3101openalex-snapshot

Effect of allelic variation at the NACP-Rep1 repeat upstream of the alpha-synuclein gene (SNCA) on transcription in a cell culture luciferase reporter system

inferredcites →https://doi.org/10.1097/wnr.0b013e3282ffda5eopenalex-snapshot

Abnormal colonic motility in mice overexpressing human wild-type α-synuclein

inferredcites →https://doi.org/10.1101/gad.1059603openalex-snapshot

Asynchronous replication timing of imprinted loci is independent of DNA methylation, but consistent with differential subnuclear localization

inferredcites →https://doi.org/10.1101/gr.165801openalex-snapshot

Human and Mouse α-Synuclein Genes: Comparative Genomic Sequence Analysis and Identification of a Novel Gene Regulatory Element

inferredcites →https://doi.org/10.1101/gr.749203openalex-snapshot

A Highly Efficient Recombineering-Based Method for Generating Conditional Knockout Mutations

inferredcites →https://doi.org/10.1111/j.1365-2982.2008.01162.xopenalex-snapshot

Parkinson’s disease and the gut: a well known clinical association in need of an effective cure and explanation

inferredcites →https://doi.org/10.1111/j.1365-2990.2008.00937.xopenalex-snapshot

The dorsal motor nucleus of the vagus is not an obligatory trigger site of Parkinson's disease

inferredcites →https://doi.org/10.1111/j.1742-4658.2008.06302.xopenalex-snapshot

Parkinson’s disease: genetic versus toxin‐induced rodent models

inferredcites →https://doi.org/10.1113/expphysiol.2007.040733openalex-snapshot

Heart rate variability in mice: a theoretical and practical guide

inferredcites →https://doi.org/10.1126/science.1090278openalex-snapshot

α-Synuclein Locus Triplication Causes Parkinson's Disease

inferredcites →https://doi.org/10.1126/science.276.5321.2045openalex-snapshot

Mutation in the α-Synuclein Gene Identified in Families with Parkinson's Disease

inferredcites →https://doi.org/10.1136/gut.2008.162503openalex-snapshot

Pathological lesions in colonic biopsies during Parkinson’s disease

inferredcites →https://doi.org/10.1136/gut.39.1.109openalex-snapshot

How well does stool form reflect colonic transit?

inferredcites →https://doi.org/10.1136/jnnp.55.2.138openalex-snapshot

Bilateral olfactory dysfunction in early stage treated and untreated idiopathic Parkinson's disease.

inferredcites →https://doi.org/10.1136/jnnp.70.3.305openalex-snapshot

Ambulatory ECG and analysis of heart rate variability in Parkinson's disease

inferredcites →https://doi.org/10.1152/ajpgi.1999.276.1.g125openalex-snapshot

Peripheral and central actions of orphanin FQ (nociceptin) on murine colon

inferredcites →https://doi.org/10.1159/000117072openalex-snapshot

Colonic Transit Time in Nonidiopathic Parkinson’s Syndrome

inferredcites →https://doi.org/10.1212/01.wnl.0000312279.49272.9fopenalex-snapshot

Invited Article: Nervous system pathology in sporadic Parkinson disease

inferredcites →https://doi.org/10.1212/wnl.42.4.726openalex-snapshot

Gastrointestinal dysfunction in Parkinson's disease

inferredcites →https://doi.org/10.1212/wnl.56.10.1355openalex-snapshot

Familial parkinsonism with synuclein pathology

inferredcites →https://doi.org/10.1212/wnl.57.3.456openalex-snapshot

Frequency of bowel movements and the future risk of Parkinson’s disease

inferredcites →https://doi.org/10.1523/jneurosci.21-16-06348.2001openalex-snapshot

Maintenance of Serotonin in the Intestinal Mucosa and Ganglia of Mice that Lack the High-Affinity Serotonin Transporter: Abnormal Intestinal Motility and the Expression of Cation Transporters

inferredcites →https://doi.org/10.1523/jneurosci.22-20-08797.2002openalex-snapshot

Synaptic Vesicle Depletion Correlates with Attenuated Synaptic Responses to Prolonged Repetitive Stimulation in Mice Lacking α-Synuclein

inferredcites →https://doi.org/10.1523/jneurosci.3982-03.2004openalex-snapshot

Enteric Dopaminergic Neurons: Definition, Developmental Lineage, and Effects of Extrinsic Denervation

inferredcites →https://doi.org/10.1523/jneurosci.4720-05.2006openalex-snapshot

Physiological Modulation of Intestinal Motility by Enteric Dopaminergic Neurons and the D 2 Receptor: Analysis of Dopamine Receptor Expression, Location, Development, and Function in Wild-Type and Knock-Out Mice

inferredcites →https://doi.org/10.2165/00002512-199710040-00002openalex-snapshot

Gastrointestinal Motility Problems in Patients with Parkinsonʼs Disease

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Role of the Gut Microbiome in Aggregation of Intestinal Alpha-Synuclein in Early Parkinson's Disease

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Transgenic Animal Models of Proteinopathies

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Ghrelin Bridges DMV Neuropathology and GI Dysfunction in the Early Stages of Parkinson's Disease

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Acid β‐glucosidase mutants linked to gaucher disease, parkinson disease, and lewy body dementia alter α‐synuclein processing

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Cerebral dopamine neurotrophic factor is essential for enteric neuronal development, maintenance, and regulation of gastrointestinal transit

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Depletion of enteroendocrine and mucus‐secreting cells is associated with colorectal carcinogenesis severity and impaired intestinal motility in rats

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Progression of intestinal permeability changes and alpha‐synuclein expression in a mouse model of Parkinson's disease

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Neuropathology of autonomic dysfunction in synucleinopathies

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High Glucosylceramides and Low Anandamide Contribute to Sensory Loss and Pain in Parkinson's Disease

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Psychosis‐Like Behavior and Hyperdopaminergic Dysregulation in Human α‐Synuclein BAC Transgenic Rats

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Transgenic Rodent Models to Study Alpha-Synuclein Pathogenesis, with a Focus on Cognitive Deficits

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Alpha Synuclein in Parkinson’s Disease

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Models of Neurodegenerative Diseases

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Adenosine A2A Receptor-Mediated Control of Non-Motor Functions in Parkinson’s Disease

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Dopamine and Gastrointestinal Motility

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Morbus Parkinson

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Parkinson’s disease mouse models in translational research

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Involvement of the peripheral nervous system in synucleinopathies, tauopathies and other neurodegenerative proteinopathies of the brain

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Multiple system atrophy prions retain strain specificity after serial propagation in two different Tg(SNCA*A53T) mouse lines

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Evidence of distinct α-synuclein strains underlying disease heterogeneity

inferred← citeshttps://doi.org/10.1007/s00401-021-02288-1openalex-snapshot

Heterogeneity in α-synuclein fibril activity correlates to disease phenotypes in Lewy body dementia

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Multiple system atrophy prions transmit neurological disease to mice expressing wild-type human α-synuclein

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Gastrointestinal manifestations in Parkinson’s disease: prevalence and occurrence before motor symptoms

inferred← citeshttps://doi.org/10.1007/s00441-018-2856-4openalex-snapshot

The enteric nervous system in PD: gateway, bystander victim, or source of solutions

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Gastric smooth muscle cells manifest an abnormal phenotype in Parkinson’s disease rats with gastric dysmotility

inferred← citeshttps://doi.org/10.1007/s00441-026-04062-9openalex-snapshot

Early intestinal barrier changes in A53T transgenic Parkinson’s disease mice

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α-Synuclein in human cerebrospinal fluid is principally derived from neurons of the central nervous system

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Neuropathobiology of non-motor symptoms in Parkinson disease

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Holocranohistochemistry enables the visualization of α-synuclein expression in the murine olfactory system and discovery of its systemic anti-microbial effects

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The interrelationship between intestinal immune cells and enteric α-synuclein in the progression of Parkinson’s disease

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Autonomic dysfunction in Parkinson disease and animal models

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Enteric Neurodegeneration is Mediated Through Independent Neuritic and Somal Mechanisms in Rotenone and MPP+ Toxicity

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Stability switches mechanism and PID control in reaction-diffusion network-organized neural systems with transmission delays

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The Aging Colon: The Role of Enteric Neurodegeneration in Constipation

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Inflammation and α-Synuclein’s Prion-like Behavior in Parkinson's Disease—Is There a Link?

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Hyposmia: a possible biomarker of Parkinson’s disease

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A Progressive Mouse Model of Parkinson's Disease: The Thy1-aSyn (“Line 61”) Mice

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Genetic analysis of SNCA coding mutation in Chinese Han patients with Parkinson disease

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Rodent Models of Autosomal Dominant Parkinson Disease

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Methods and Models of the Nonmotor Symptoms of Parkinson Disease

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Parkinsonism

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Role of rodent models in advancing precision medicine for Parkinson's disease

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Rodent models for exploring the mechanisms contributing to Parkinson's disease pathophysiology

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Autonomic dysfunction in Parkinson disease

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Modeling synucleinopathies in rodents

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The Gut and Nonmotor Symptoms in Parkinson's Disease

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Autonomic nervous system dysregulation in neurodegenerative diseases: Bridging brain and heart

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Dopaminergic Neurons and Brain Reward Pathways

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Genetic variants and animal models in SNCA and Parkinson disease

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The novel adaptive rotating beam test unmasks sensorimotor impairments in a transgenic mouse model of Parkinson’s disease

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Are rodent models of Parkinson’s disease behaving as they should?

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p53 signalling mediates acupuncture-induced neuroprotection in Parkinson's disease

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MPTP-induced parkinsonism extends to a subclass of TH-positive neurons in the gut

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Morphological changes in the enteric nervous system of aging and APP23 transgenic mice

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Cardiovascular dysfunction associated with neurodegeneration in an experimental model of Parkinson’s disease

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Enteric nervous system manifestations of neurodegenerative disease

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Enteric plexuses of two choline-acetyltransferase transgenic mouse lines: Chemical neuroanatomy of the fluorescent protein-expressing nerve cells

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Cholesterol enhances lysosome-autophagosome fusion for better α-synuclein clearance in GBA L444P-mutated Parkinson disease

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Impaired brain-gut circuit for gut motility dysfunction in mouse models of Parkinson’s disease

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Gut and brain profiles that resemble pre-motor and early-stage Parkinson’s disease in methamphetamine self-administering rats

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Intergenerational neurotoxicity of polystyrene nanoplastics in offspring mice is mediated by dysfunctional microbe-gut-brain axis

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Oral subchronic exposure to the mycotoxin ochratoxin A induces key pathological features of Parkinson's disease in mice six months after the end of the treatment

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Gut microbiome and Parkinson's disease: Perspective on pathogenesis and treatment

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Calcium influx: An essential process by which α-Synuclein regulates morphology of erythrocytes

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Retake the Center Stage – New Development of Rat Genetics

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Treatment approaches of gastrointestinal dysfunction in Parkinson's disease, therapeutical options and future perspectives

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Autonomic involvement in Parkinson's disease: Pathology, pathophysiology, clinical features and possible peripheral biomarkers

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Optogenetic manipulation of ENS - The brain in the gut

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Preclinical models of disease and multimorbidity with focus upon cardiovascular disease and dementia

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Pathological correlates of gastrointestinal dysfunction in Parkinson's disease

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Animal models of the non-motor features of Parkinson's disease

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Alpha-synuclein overexpressing transgenic mice show internal organ pathology and autonomic deficits

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Alpha-synuclein transgenic mice display age-related slowing of gastrointestinal motility associated with transgene expression in the vagal system

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Region-specific deficits in dopamine, but not norepinephrine, signaling in a novel A30P α-synuclein BAC transgenic mouse

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GPER1-mediated immunomodulation and neuroprotection in the myenteric plexus of a mouse model of Parkinson's disease

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Induction of alpha-synuclein pathology in the enteric nervous system of the rat and non-human primate results in gastrointestinal dysmotility and transient CNS pathology

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Development of an aggregate-selective, human-derived α-synuclein antibody BIIB054 that ameliorates disease phenotypes in Parkinson's disease models

inferred← citeshttps://doi.org/10.1016/j.nbd.2019.104700openalex-snapshot

Autonomic dysfunction in Parkinson’s disease: Implications for pathophysiology, diagnosis, and treatment

inferred← citeshttps://doi.org/10.1016/j.nbd.2022.105625openalex-snapshot

Body-first Parkinson’s disease and variant Creutzfeldt–Jakob disease – similar or different?

inferred← citeshttps://doi.org/10.1016/j.nbd.2023.106166openalex-snapshot

Stabilization of 14-3-3 protein-protein interactions with Fusicoccin-A decreases alpha-synuclein dependent cell-autonomous death in neuronal and mouse models

inferred← citeshttps://doi.org/10.1016/j.nbd.2025.106848openalex-snapshot

Leveraging animal models to understand non-motor symptoms of Parkinson's disease

inferred← citeshttps://doi.org/10.1016/j.neulet.2014.01.033openalex-snapshot

Transgenic mice expressing S129 phosphorylation mutations in α-synuclein

inferred← citeshttps://doi.org/10.1016/j.neulet.2023.137555openalex-snapshot

Distribution, connection and function of ALDH1A1+/TH+ neurons in substantia nigra pars reticulata of mouse

inferred← citeshttps://doi.org/10.1016/j.neures.2012.03.010openalex-snapshot

α-Synuclein BAC transgenic mice as a model for Parkinson's disease manifested decreased anxiety-like behavior and hyperlocomotion

inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2012.07.008openalex-snapshot

Dietary energy intake modifies brainstem autonomic dysfunction caused by mutant α-synuclein

inferred← citeshttps://doi.org/10.1016/j.neuron.2013.09.004openalex-snapshot

The Function of α-Synuclein

inferred← citeshttps://doi.org/10.1016/j.neuron.2019.05.035openalex-snapshot

Transneuronal Propagation of Pathologic α-Synuclein from the Gut to the Brain Models Parkinson’s Disease

inferred← citeshttps://doi.org/10.1016/j.neuroscience.2011.08.004openalex-snapshot

Effect of unilateral subthalamic deep brain stimulation on rat digestive motor activity

inferred← citeshttps://doi.org/10.1016/j.neuroscience.2019.10.029openalex-snapshot

α-Synuclein Regulates Development and Function of Cholinergic Enteric Neurons in the Mouse Colon

inferred← citeshttps://doi.org/10.1016/j.neuroscience.2026.04.007openalex-snapshot

Neuronal vulnerability in Parkinson’s disease: insights from murine α-synuclein pathology models

inferred← citeshttps://doi.org/10.1016/j.parkreldis.2015.10.011openalex-snapshot

Gastric motor dysfunctions in Parkinson's disease: Current pre-clinical evidence

inferred← citeshttps://doi.org/10.1016/j.pneurobio.2011.09.002openalex-snapshot

Biomarkers of Parkinson's disease and Dementia with Lewy bodies

inferred← citeshttps://doi.org/10.1016/j.prdoa.2019.06.001openalex-snapshot

Targeting neurons in the gastrointestinal tract to treat Parkinson's disease

inferred← citeshttps://doi.org/10.1016/j.tins.2016.09.003openalex-snapshot

α-Synuclein-Based Animal Models of Parkinson's Disease: Challenges and Opportunities in a New Era

inferred← citeshttps://doi.org/10.1016/j.tins.2016.10.008openalex-snapshot

The Threshold Theory for Parkinson's Disease

inferred← citeshttps://doi.org/10.1016/j.tins.2024.05.004openalex-snapshot

Effect of host and strain factors on α-synuclein prion pathogenesis

inferred← citeshttps://doi.org/10.1016/j.toxrep.2015.02.013openalex-snapshot

Prolongation of chemically-induced methemoglobinemia in mice lacking α-synuclein: A novel pharmacologic and toxicologic phenotype

inferred← citeshttps://doi.org/10.1016/j.ydbio.2016.06.004openalex-snapshot

Regulators of gene expression in Enteric Neural Crest Cells are putative Hirschsprung disease genes

inferred← citeshttps://doi.org/10.1016/s0079-6123(10)84004-xopenalex-snapshot

Genetic mouse models of Parkinson’s disease

inferred← citeshttps://doi.org/10.1016/s1353-8020(13)70007-5openalex-snapshot

Premotor parkinsonism models

inferred← citeshttps://doi.org/10.1016/s1474-4422(10)70213-1openalex-snapshot

Are synucleinopathies prion-like disorders?

inferred← citeshttps://doi.org/10.1016/s1474-4422(11)70227-7openalex-snapshot

Modelling of Parkinson's disease in mice

inferred← citeshttps://doi.org/10.1017/cbo9781107360556.030openalex-snapshot

Neurodegenerative disorders

inferred← citeshttps://doi.org/10.1017/cbo9781107360556.031openalex-snapshot

Parkinson’s disease

inferred← citeshttps://doi.org/10.1021/acschemneuro.6c00069openalex-snapshot

Fluorescence Detection of Alpha-Synuclein Aggregates in the Gut Using a Peptide Probe

inferred← citeshttps://doi.org/10.1021/acsnano.4c13914openalex-snapshot

Polystyrene Nanoplastics Hitch-Hike the Gut–Brain Axis to Exacerbate Parkinson’s Pathology

inferred← citeshttps://doi.org/10.1021/acsptsci.4c00286openalex-snapshot

Chronic DSS-Induced Colitis Exacerbates Parkinson’s Disease Phenotype and Its Pathological Features Following Intragastric Rotenone Exposure

inferred← citeshttps://doi.org/10.1038/ncomms2032openalex-snapshot

Alternative α-synuclein transcript usage as a convergent mechanism in Parkinson's disease pathology

inferred← citeshttps://doi.org/10.1038/ncomms8314openalex-snapshot

Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiation

inferred← citeshttps://doi.org/10.1038/nrgastro.2016.107openalex-snapshot

The bowel and beyond: the enteric nervous system in neurological disorders

inferred← citeshttps://doi.org/10.1038/nrgastro.2016.76openalex-snapshot

Vagal neurocircuitry and its influence on gastric motility

inferred← citeshttps://doi.org/10.1038/nrn.2017.75openalex-snapshot

Animal models of α-synucleinopathy for Parkinson disease drug development

inferred← citeshttps://doi.org/10.1038/s41419-024-06534-8openalex-snapshot

Genetic and pharmacological reduction of CDK14 mitigates synucleinopathy

inferred← citeshttps://doi.org/10.1038/s41531-016-0002-0openalex-snapshot

The gut-brain axis: is intestinal inflammation a silent driver of Parkinson’s disease pathogenesis?

inferred← citeshttps://doi.org/10.1038/s41531-022-00308-9openalex-snapshot

Similarities and differences between nigral and enteric dopaminergic neurons unravel distinctive involvement in Parkinson’s disease

inferred← citeshttps://doi.org/10.1038/s41575-020-0339-zopenalex-snapshot

Parkinson disease and the gut: new insights into pathogenesis and clinical relevance

inferred← citeshttps://doi.org/10.1038/s41583-020-00416-6openalex-snapshot

Reverse engineering Lewy bodies: how far have we come and how far can we go?

inferred← citeshttps://doi.org/10.1038/s41598-020-68148-2openalex-snapshot

Longitudinal profiling of oligomeric Aβ in human nasal discharge reflecting cognitive decline in probable Alzheimer’s disease

inferred← citeshttps://doi.org/10.1038/s41598-020-77409-zopenalex-snapshot

α-Synuclein promotes IAPP fibril formation in vitro and β-cell amyloid formation in vivo in mice

inferred← citeshttps://doi.org/10.1038/s41598-021-86917-5openalex-snapshot

The association of enteric neuropathy with gut phenotypes in acute and progressive models of Parkinson’s disease

inferred← citeshttps://doi.org/10.1038/s41598-023-32594-5openalex-snapshot

Mutant and curli-producing E. coli enhance the disease phenotype in a hSOD1-G93A mouse model of ALS

inferred← citeshttps://doi.org/10.1038/s41598-024-53093-1openalex-snapshot

Dopaminergic neuron loss in mice due to increased levels of wild-type human α-Synuclein only takes place under conditions of accelerated aging

inferred← citeshttps://doi.org/10.1038/s41598-025-10705-8openalex-snapshot

Templating of monomeric alpha-synuclein results in inflammation and SNpc dopamine neuron death in a genetic mouse model of induced synucleinopathy

inferred← citeshttps://doi.org/10.1038/s41598-025-89051-8openalex-snapshot

Longitudinal characterization reveals behavioral impairments in aged APP knock in mouse models

inferred← citeshttps://doi.org/10.1038/s42003-022-03373-1openalex-snapshot

Pathogenic tau recruits wild-type tau into brain inclusions and induces gut degeneration in transgenic SPAM mice

inferred← citeshttps://doi.org/10.1039/bk9781849731843-00113openalex-snapshot

Animal Models and the Pathogenesis of Parkinson's Disease

inferred← citeshttps://doi.org/10.1039/c0cs00181copenalex-snapshot

Stimuli-responsive electrospun fibers and their applications

inferred← citeshttps://doi.org/10.1042/bst0390862openalex-snapshot

A BACwards glance at neurodegeneration: molecular insights into disease from LRRK2, SNCA and MAPT BAC-transgenic mice

inferred← citeshttps://doi.org/10.1053/j.gastro.2011.04.049openalex-snapshot

Transient Receptor Potential Ankyrin 1 Is Expressed by Inhibitory Motoneurons of the Mouse Intestine

inferred← citeshttps://doi.org/10.1070/rcr4435openalex-snapshot

Electrospinning of functional materials for biomedicine and tissue engineering

inferred← citeshttps://doi.org/10.1073/pnas.1514475112openalex-snapshot

Evidence for α-synuclein prions causing multiple system atrophy in humans with parkinsonism

inferred← citeshttps://doi.org/10.1073/pnas.1619406114openalex-snapshot

Adult enteric nervous system in health is maintained by a dynamic balance between neuronal apoptosis and neurogenesis

inferred← citeshttps://doi.org/10.1073/pnas.2406479121openalex-snapshot

Olfactory deficit and gastrointestinal dysfunction precede motor abnormalities in alpha-Synuclein G51D knock-in mice

inferred← citeshttps://doi.org/10.1074/jbc.m116.744029openalex-snapshot

FTY720/Fingolimod Reduces Synucleinopathy and Improves Gut Motility in A53T Mice

inferred← citeshttps://doi.org/10.1080/00207454.2018.1538141openalex-snapshot

Rotenone impairs oxidant/antioxidant balance both in brain and intestines in zebrafish

inferred← citeshttps://doi.org/10.1080/23279095.2024.2338803openalex-snapshot

Does the side of onset influence symptom severity in Parkinson’s disease? A systematic review and meta-analysis

inferred← citeshttps://doi.org/10.1089/dna.2011.1353openalex-snapshot

Identification of 129S6/SvEvTac-Specific Polymorphisms on Mouse Chromosome 11

inferred← citeshttps://doi.org/10.1093/brain/awag046openalex-snapshot

α-Synuclein aggregation and brain atrophy in SNCA -A53T transgenic monkeys are correlated with parkinsonism

inferred← citeshttps://doi.org/10.1093/brain/aws358openalex-snapshot

A progressive dopaminergic phenotype associated with neurotoxic conversion of α-synuclein in BAC-transgenic rats

inferred← citeshttps://doi.org/10.1093/brain/awu291openalex-snapshot

Augmentation of phenotype in a transgenic Parkinson mouse heterozygous for a Gaucher mutation

inferred← citeshttps://doi.org/10.1093/brain/awz172openalex-snapshot

Parkin interacting substrate zinc finger protein 746 is a pathological mediator in Parkinson’s disease

inferred← citeshttps://doi.org/10.1093/brain/awz380openalex-snapshot

α-Synuclein BAC transgenic mice exhibit RBD-like behaviour and hyposmia: a prodromal Parkinson’s disease model

inferred← citeshttps://doi.org/10.1093/glycob/cwx022openalex-snapshot

The N-glycosylation of immunoglobulin G as a novel biomarker of Parkinson's disease

inferred← citeshttps://doi.org/10.1093/hmg/ddz057openalex-snapshot

The A30P α-synuclein mutation decreases subventricular zone proliferation

inferred← citeshttps://doi.org/10.1097/mcg.0000000000000606openalex-snapshot

Gastrointestinal Dysfunction and Neuropathologic Correlations in Parkinson Disease

inferred← citeshttps://doi.org/10.1097/md.0000000000021145openalex-snapshot

A randomized double-blind placebo-controlled multicenter trial of Bushen Yisui and Ziyin Jiangzhuo formula for constipation in Parkinson disease

inferred← citeshttps://doi.org/10.1097/nen.0000000000000137openalex-snapshot

Early and Persistent Expression of Phosphorylated α-Synuclein in the Enteric Nervous System of A53T Mutant Human α-Synuclein Transgenic Mice

inferred← citeshttps://doi.org/10.1101/2020.06.06.136556openalex-snapshot

Functional and molecular early enteric biomarkers for Parkinson’s disease in mice and men

inferred← citeshttps://doi.org/10.1101/2022.05.23.22274942openalex-snapshot

Safety and feasibility of fecal microbiota transplantation for Parkinson’s disease patients: a protocol for a self-controlled interventional donor-FMT pilot study

inferred← citeshttps://doi.org/10.1101/2022.07.13.499896openalex-snapshot

Structural and functional landscape of α-synuclein fibril conformations amplified from cerebrospinal fluid

inferred← citeshttps://doi.org/10.1101/2022.12.22.521569openalex-snapshot

Intragastric administration of low-dose rotenone post-colitis exacerbates damage to the nigrostriatal dopaminergic system in Parkinson’s disease: The pace accelerates even more

inferred← citeshttps://doi.org/10.1101/2023.09.05.556425openalex-snapshot

Robust nuclease-dead S. aureus dCas9-mediated alpha-synuclein knockdown in substantia nigra in a humanized mouse model of Parkinson’s disease

inferred← citeshttps://doi.org/10.1101/2024.05.24.595745openalex-snapshot

APP-KI mice do not display the hallmark age-dependent cognitive decline of amyloid diseases

inferred← citeshttps://doi.org/10.1101/2024.07.29.605647openalex-snapshot

Templating of Monomeric Alpha-Synuclein Induces Inflammation and SNpc Dopamine Neuron Death in a Genetic Mouse Model of Synucleinopathy

inferred← citeshttps://doi.org/10.1101/2024.12.15.628584openalex-snapshot

Optimization of protocols for immunohistochemical assessment of enteric nervous system in formalin fixed human tissue

inferred← citeshttps://doi.org/10.1101/505164openalex-snapshot

Experimental colitis drives enteric alpha-synuclein accumulation and Parkinson-like brain pathology

inferred← citeshttps://doi.org/10.1101/cshperspect.a009399openalex-snapshot

-Synuclein in Parkinson's Disease

inferred← citeshttps://doi.org/10.1101/cshperspect.a041618openalex-snapshot

The Gut–Brain Axis in Parkinson's Disease

inferred← citeshttps://doi.org/10.1101/lm.052282.120openalex-snapshot

Iron-responsive-like elements and neurodegenerative ferroptosis

inferred← citeshttps://doi.org/10.1111/coa.12816openalex-snapshot

New olfactometric findings in Parkinson's disease

inferred← citeshttps://doi.org/10.1111/ejn.13476openalex-snapshot

Modelling idiopathic Parkinson disease as a complex illness can inform incidence rate in healthy adults: the PREDIGT score

inferred← citeshttps://doi.org/10.1111/ejn.14300openalex-snapshot

Genetically engineered animal models of Parkinson's disease: From worm to rodent

inferred← citeshttps://doi.org/10.1111/j.1365-2982.2011.01678.xopenalex-snapshot

Aging and gastrointestinal neuromuscular function: insights from within and outside the gut

inferred← citeshttps://doi.org/10.1111/j.1365-2982.2011.01794.xopenalex-snapshot

Parallel manifestations of neuropathologies in the enteric and central nervous systems

inferred← citeshttps://doi.org/10.1111/j.1365-2982.2012.01974.xopenalex-snapshot

Mice overexpressing wild‐type human alpha‐synuclein display alterations in colonic myenteric ganglia and defecation

inferred← citeshttps://doi.org/10.1111/jnc.13570openalex-snapshot

The contribution of alpha synuclein to neuronal survival and function – Implications for Parkinson's disease

inferred← citeshttps://doi.org/10.1111/jnc.70021openalex-snapshot

Limitations and Applications of Rodent Models in Tauopathy and Synucleinopathy Research

inferred← citeshttps://doi.org/10.1111/nan.12298openalex-snapshot

Review: Sporadic Parkinson's disease: development and distribution ofα‐synuclein pathology

inferred← citeshttps://doi.org/10.1111/nmo.12691openalex-snapshot

Response of colonic motility to dopaminergic stimulation is subverted in rats with nigrostriatal lesion: relevance to gastrointestinal dysfunctions in Parkinson's disease

inferred← citeshttps://doi.org/10.1111/nmo.12873openalex-snapshot

Activity and Me CP 2‐dependent regulation of nNOS levels in enteric neurons

inferred← citeshttps://doi.org/10.1111/nmo.12985openalex-snapshot

Rotenone and elevated extracellular potassium concentration induce cell‐specific fibrillation of α‐synuclein in axons of cholinergic enteric neurons in the guinea‐pig ileum

inferred← citeshttps://doi.org/10.1111/nmo.13755openalex-snapshot

Chronic isolation stress is associated with increased colonic and motor symptoms in the A53T mouse model of Parkinson’s disease

inferred← citeshttps://doi.org/10.1111/nmo.13994openalex-snapshot

A simple automated approach to measure mouse whole gut transit

inferred← citeshttps://doi.org/10.1128/msphere.00379-23openalex-snapshot

Microbiota-mediated effects of Parkinson’s disease medications on Parkinsonian non-motor symptoms in male transgenic mice

inferred← citeshttps://doi.org/10.1136/bmjopen-2023-071766openalex-snapshot

Safety and feasibility of faecal microbiota transplantation for patients with Parkinson’s disease: a protocol for a self-controlled interventional donor-FMT pilot study

inferred← citeshttps://doi.org/10.1136/bmjopen-2025-102851openalex-snapshot

Randomised, double-blind, placebo-controlled, parallel-group study to assess the efficacy and safety of antibiotic faecal microbiota transplantation in patients with Parkinson’s disease (FLORA-PD): a study protocol

inferred← citeshttps://doi.org/10.1136/jmedgenet-2019-106210openalex-snapshot

Open questions on the nature of Parkinson’s disease: from triggers to spreading pathology

inferred← citeshttps://doi.org/10.1152/jn.00548.2020openalex-snapshot

Baroreflex dysfunction in Parkinson’s disease: integration of central and peripheral mechanisms

inferred← citeshttps://doi.org/10.1155/2012/142372openalex-snapshot

The Anticholinesterase Phenserine and Its Enantiomer Posiphen as 5′Untranslated-Region-Directed Translation Blockers of the Parkinson’s Alpha Synuclein Expression

inferred← citeshttps://doi.org/10.1155/2016/6762528openalex-snapshot

Gastrointestinal Dysfunctions in Parkinson’s Disease: Symptoms and Treatments

inferred← citeshttps://doi.org/10.1172/jci.insight.172192openalex-snapshot

Gut mucosal cells transfer α-synuclein to the vagus nerve

inferred← citeshttps://doi.org/10.1172/jci.insight.92295openalex-snapshot

α-Synuclein in gut endocrine cells and its implications for Parkinson’s disease

inferred← citeshttps://doi.org/10.1172/jci143775openalex-snapshot

Neurodegenerative disorders and gut-brain interactions

inferred← citeshttps://doi.org/10.1177/0192623313501893openalex-snapshot

Subsite Awareness in Neuropathology Evaluation of National Toxicology Program (NTP) Studies

inferred← citeshttps://doi.org/10.1177/13872877261489296openalex-snapshot

Role of subcortical iron in Parkinson's disease, prodromal-depression, and dementia: Comparative links to anemias and osteoporosis

inferred← citeshttps://doi.org/10.1177/1759091418812587openalex-snapshot

α -Synuclein Trafficking in Parkinson’s Disease: Insights From Fly and Mouse Models

inferred← citeshttps://doi.org/10.1177/1877718x241289046openalex-snapshot

Chronic urinary tract infections cause persistent microglial changes in a humanized ɑ-synuclein mouse model

inferred← citeshttps://doi.org/10.1177/2042018813479645openalex-snapshot

Ghrelin is neuroprotective in Parkinson’s disease: molecular mechanisms of metabolic neuroprotection

inferred← citeshttps://doi.org/10.1186/s12974-020-01940-zopenalex-snapshot

Immunoregulation of microglial polarization: an unrecognized physiological function of α-synuclein

inferred← citeshttps://doi.org/10.1186/s12974-022-02413-1openalex-snapshot

α-Synuclein-containing erythrocytic extracellular vesicles: essential contributors to hyperactivation of monocytes in Parkinson’s disease

inferred← citeshttps://doi.org/10.1186/s13024-021-00439-2openalex-snapshot

Parkinson mice show functional and molecular changes in the gut long before motoric disease onset

inferred← citeshttps://doi.org/10.1186/s13024-024-00738-4openalex-snapshot

Single molecule array measures of LRRK2 kinase activity in serum link Parkinson’s disease severity to peripheral inflammation

inferred← citeshttps://doi.org/10.1186/s40035-017-0099-zopenalex-snapshot

Current understanding of the molecular mechanisms in Parkinson's disease: Targets for potential treatments

inferred← citeshttps://doi.org/10.1186/s40035-019-0146-zopenalex-snapshot

Constipation, deficit in colon contractions and alpha-synuclein inclusions within the colon precede motor abnormalities and neurodegeneration in the central nervous system in a mouse model of alpha-synucleinopathy

inferred← citeshttps://doi.org/10.1186/s40478-015-0236-9openalex-snapshot

Distribution of α-synuclein in the spinal cord and dorsal root ganglia in an autopsy cohort of elderly persons

inferred← citeshttps://doi.org/10.1186/s40478-015-0254-7openalex-snapshot

Prion-like propagation of human brain-derived alpha-synuclein in transgenic mice expressing human wild-type alpha-synuclein

inferred← citeshttps://doi.org/10.1186/s40478-019-0703-9openalex-snapshot

Replication of multiple system atrophy prions in primary astrocyte cultures from transgenic mice expressing human α-synuclein

inferred← citeshttps://doi.org/10.1186/s40478-019-0733-3openalex-snapshot

Comparative analyses of the in vivo induction and transmission of α-synuclein pathology in transgenic mice by MSA brain lysate and recombinant α-synuclein fibrils

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