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NADH as a cancer medicine
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/(sici)1096-911x(199609)27:3<165::aid-mpo6>3.0.co;2-copenalex-snapshot
Phase I trial of extracellular adenosine 5′-triphosphate in patients with advanced cancer
inferredcites →https://doi.org/10.1002/biof.31openalex-snapshot
The bioenergetics of cancer: Is glycolysis the main ATP supplier in all tumor cells?
inferredcites →https://doi.org/10.1007/bf01271203openalex-snapshot
Parenteral application of NADH in Parkinson's disease: Clinical improvement partially due to stimulation of endogenous levodopa biosynthesis
inferredcites →https://doi.org/10.1007/s10637-007-9076-1openalex-snapshot
Intravenous ATP infusions can be safely administered in the home setting: a study in pre-terminal cancer patients
inferredcites →https://doi.org/10.1007/s10863-005-8642-5openalex-snapshot
ANT2 Isoform Required for Cancer Cell Glycolysis
inferredcites →https://doi.org/10.1016/j.bbabio.2010.08.010openalex-snapshot
The Warburg and Crabtree effects: On the origin of cancer cell energy metabolism and of yeast glucose repression
inferredcites →https://doi.org/10.1016/j.bbrc.2007.08.095openalex-snapshot
P2X7 receptors mediate NADH transport across the plasma membranes of astrocytes
inferredcites →https://doi.org/10.1016/j.biocel.2008.06.013openalex-snapshot
Energetic signalling in the control of mitochondrial F1F0 ATP synthase activity in health and disease
inferredcites →https://doi.org/10.1016/j.biocel.2009.12.024openalex-snapshot
The fourth isoform of the adenine nucleotide translocator inhibits mitochondrial apoptosis in cancer cells
inferredcites →https://doi.org/10.1016/j.ccr.2006.04.023openalex-snapshot
Attenuation of LDH-A expression uncovers a link between glycolysis, mitochondrial physiology, and tumor maintenance
inferredcites →https://doi.org/10.1016/j.ccr.2006.10.020openalex-snapshot
A Mitochondria-K+ Channel Axis Is Suppressed in Cancer and Its Normalization Promotes Apoptosis and Inhibits Cancer Growth
inferredcites →https://doi.org/10.1016/j.tibs.2005.01.005openalex-snapshot
Multifaceted roles of glycolytic enzymes
inferredcites →https://doi.org/10.1016/j.yexmp.2010.08.006openalex-snapshot
Cancer metabolism: The Warburg effect today
inferredcites →https://doi.org/10.1016/s0021-9258(18)31720-4openalex-snapshot
Glucose is essential for proliferation and the glycolytic enzyme induction that provokes a transition to glycolytic energy production.
inferredcites →https://doi.org/10.1016/s1081-1206(10)62595-1openalex-snapshot
Therapeutic effects of oral NADH on the symptoms of patients with chronic fatigue syndrome
inferredcites →https://doi.org/10.1016/s1470-2045(00)00257-6openalex-snapshot
Clinical role of positron emission tomography in oncology
inferredcites →https://doi.org/10.1023/a:1006018610986openalex-snapshot
Phase II study of intravenous adenosine 5'-triphosphate in patients with previously untreated stage IIIB and Stage IV non-small cell lung cancer
inferredcites →https://doi.org/10.1023/a:1022498714522openalex-snapshot
Aerobic Glycolysis by Proliferating Cells: Protection against Oxidative Stress at the Expense of Energy Yield
inferredcites →https://doi.org/10.1034/j.1600-0773.2002.900409.xopenalex-snapshot
Bioavailability of Reduced Nicotinamide‐adenin‐dinucleotide (NADH) in the Central Nervous System of the Anaesthetized Rat Measured by Laser‐Induced Fluorescence Spectroscopy
inferredcites →https://doi.org/10.1038/nature13110openalex-snapshot
Metabolic determinants of cancer cell sensitivity to glucose limitation and biguanides
inferredcites →https://doi.org/10.1038/nrc1478openalex-snapshot
Why do cancers have high aerobic glycolysis?
inferredcites →https://doi.org/10.1038/sj.bjp.0705300openalex-snapshot
NADH supplementation decreases pinacidil‐primed IK(ATP) in ventricular cardiomyocytes by increasing intracellular ATP
inferredcites →https://doi.org/10.1042/bj2280353openalex-snapshot
Glutamine and glucose metabolism during thymocyte proliferation. Pathways of glutamine and glutamate metabolism
inferredcites →https://doi.org/10.1046/j.1432-1327.2000.01576.xopenalex-snapshot
Increased formation of reactive oxygen species due to glucose depletion in primary cultures of rat thymocytes inhibits proliferation
inferredcites →https://doi.org/10.1053/jhep.2002.31318openalex-snapshot
Adenosine triphosphate infusion increases liver energy status in advanced lung cancer patients: An in vivo 31P magnetic resonance spectroscopy study
inferredcites →https://doi.org/10.1074/jbc.m511064200openalex-snapshot
Induction of Oxidative Metabolism by Mitochondrial Frataxin Inhibits Cancer Growth
inferredcites →https://doi.org/10.1093/jnci/92.4.290openalex-snapshot
Is ATP (Adenosine 5'-Triphosphate), like STP(R), a Performance-Enhancing Additive for the Tanks of Cancer Patients?
inferredcites →https://doi.org/10.1093/jnci/92.4.321openalex-snapshot
Randomized Clinical Trial of Adenosine 5'-Triphosphate in Patients With Advanced Non-Small-Cell Lung Cancer
inferredcites →https://doi.org/10.1096/fasebj.11.5.9141507openalex-snapshot
Aerobic glycolysis by proliferating cells: a protective strategy against reactive oxygen species 1
inferredcites →https://doi.org/10.1111/j.1600-0404.1993.tb00018.xopenalex-snapshot
Nicotinamide adenine dinucleotide (NADH) — a new therapeutic approach to Parkinson's disease
inferredcites →https://doi.org/10.1111/j.1755-5922.2008.00065.xopenalex-snapshot
Pharmacological Profile of the Selective Mitochondrial F 1 F 0 ATP Hydrolase Inhibitor BMS‐199264 in Myocardial Ischemia
inferredcites →https://doi.org/10.1126/science.123.3191.309openalex-snapshot
On the Origin of Cancer Cells
inferredcites →https://doi.org/10.1128/jb.00433-13openalex-snapshot
Chlamydia trachomatis Transports NAD via the Npt1 ATP/ADP Translocase
inferredcites →https://doi.org/10.1146/annurev-cellbio-092910-154237openalex-snapshot
Aerobic Glycolysis: Meeting the Metabolic Requirements of Cell Proliferation
inferredcites →https://doi.org/10.1146/annurev.med.53.082901.104028openalex-snapshot
Positron Emission Tomography Scanning: Current and Future Applications
inferredcites →https://doi.org/10.1158/0008-5472.can-05-1925openalex-snapshot
Activation of Glycogen Synthase Kinase 3β Disrupts the Binding of Hexokinase II to Mitochondria by Phosphorylating Voltage-Dependent Anion Channel and Potentiates Chemotherapy-Induced Cytotoxicity
inferredcites →https://doi.org/10.1158/0008-5472.can-05-4407openalex-snapshot
Chemosensitization by Knockdown of Adenine Nucleotide Translocase-2
inferredcites →https://doi.org/10.1158/0008-5472.can-06-1501openalex-snapshot
Cancer's Molecular Sweet Tooth and the Warburg Effect
inferredcites →https://doi.org/10.1172/jci64264openalex-snapshot
Mitochondrial complex I activity and NAD+/NADH balance regulate breast cancer progression
inferredcites →https://doi.org/10.1179/rer.2000.5.1.52openalex-snapshot
Control of thymocyte proliferation via redox-regulated expression of glycolytic genes
inferredcites →https://doi.org/10.1186/1471-2407-8-160openalex-snapshot
Over-expression of Adenine Nucleotide Translocase 1 (ANT1) Induces Apoptosis and Tumor Regression in vivo
inferredcites →https://doi.org/10.1186/1476-4598-12-152openalex-snapshot
Tumor glycolysis as a target for cancer therapy: progress and prospects
inferredcites →https://doi.org/10.1200/jco.20.2.371openalex-snapshot
Beneficial Effects of Adenosine Triphosphate on Nutritional Status in Advanced Lung Cancer Patients: A Randomized Clinical Trial
inferredcites →https://doi.org/10.1200/jco.2002.20.2.362openalex-snapshot
Adenosine Triphosphate: Does It Help Cancer Patients “Get Bigger and Stronger”?
inferredcites →https://doi.org/10.1200/jco.2002.20.2.371openalex-snapshot
Beneficial Effects of Adenosine Triphosphate on Nutritional Status in Advanced Lung Cancer Patients: A Randomized Clinical Trial
inferredcites →https://doi.org/10.1201/9780203506707.ch33openalex-snapshot
NADH in Cancer Prevention and Therapy
inferredcites →https://doi.org/10.14670/hh-22.85openalex-snapshot
Protection from oxidative stress by enhanced glycolysis; a possible mechanism of cellular immortalization.
inferredcites →https://doi.org/10.2174/138161211798764933openalex-snapshot
Targeting Mitochondria in Fighting Cancer
inferredcites →https://doi.org/10.2174/187152008783961932openalex-snapshot
The Warburg Effect: Why and How Do Cancer Cells Activate Glycolysis in the Presence of Oxygen?
inferredcites →https://doi.org/10.3892/or.2013.2735openalex-snapshot
Inhibition of LDH-A by oxamate induces G2/M arrest, apoptosis and increases radiosensitivity in nasopharyngeal carcinoma cells
inferredcites →https://doi.org/10.4103/0973-1482.55134openalex-snapshot
Targeting energy metabolism in brain cancer through calorie restriction and the ketogenic diet
inferredcites →https://doi.org/10.4236/jct.2014.53036openalex-snapshot
Want to Cure Cancer? Then Revisit the Past; “Warburg Was Correct”, Cancer Is a Metabolic Disease
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1101/025197openalex-snapshot
Why cancer cells have a more hyperpolarised mitochondrial membrane potential and emergent prospects for therapy
inferred← citeshttps://doi.org/10.1101/035113openalex-snapshot
Cancer cells have distinct electrical properties that predict a susceptibility to lipophilic anions; a new cancer drug paradigm
inferred← citeshttps://doi.org/10.64898/2026.02.15.704522openalex-snapshot
Conformational Diversity and Interaction Signatures of NADH across protein families
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