On the public citation graph

Selective and mechanistic sources of recurrent rearrangements across the cancer genome

0 signed · 93 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.

Claim with ORCID

Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.

See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1016/j.cell.2010.11.055openalex-snapshot

Massive Genomic Rearrangement Acquired in a Single Catastrophic Event during Cancer Development

inferredcites →https://doi.org/10.1016/j.cell.2013.01.006openalex-snapshot

Identification of Early Replicating Fragile Sites that Contribute to Genome Instability

inferredcites →https://doi.org/10.1016/j.cell.2013.03.021openalex-snapshot

Punctuated Evolution of Prostate Cancer Genomes

inferredcites →https://doi.org/10.1016/j.cell.2013.04.010openalex-snapshot

Diverse Mechanisms of Somatic Structural Variations in Human Cancer Genomes

inferredcites →https://doi.org/10.1016/j.cell.2016.10.024openalex-snapshot

Insulated Neighborhoods: Structural and Functional Units of Mammalian Gene Control

inferredcites →https://doi.org/10.1016/j.cell.2016.12.025openalex-snapshot

Insertions and Deletions Target Lineage-Defining Genes in Human Cancers

inferredcites →https://doi.org/10.1016/j.gde.2012.02.012openalex-snapshot

Mechanisms for recurrent and complex human genomic rearrangements

inferredcites →https://doi.org/10.1016/j.gde.2016.03.008openalex-snapshot

TAD disruption as oncogenic driver

inferredcites →https://doi.org/10.1016/j.tig.2014.01.001openalex-snapshot

The role of microhomology in genomic structural variation

inferredcites →https://doi.org/10.1038/nature08768openalex-snapshot

Signatures of mutation and selection in the cancer genome

inferredcites →https://doi.org/10.1038/nature08822openalex-snapshot

The landscape of somatic copy-number alteration across human cancers

inferredcites →https://doi.org/10.1038/nature09504openalex-snapshot

Selective inhibition of BET bromodomains

inferredcites →https://doi.org/10.1038/nature11082openalex-snapshot

Topological domains in mammalian genomes identified by analysis of chromatin interactions

inferredcites →https://doi.org/10.1038/nature12753openalex-snapshot

Topology of mammalian developmental enhancers and their regulatory landscapes

inferredcites →https://doi.org/10.1038/nature12912openalex-snapshot

Discovery and saturation analysis of cancer genes across 21 tumour types

inferredcites →https://doi.org/10.1038/nature13379openalex-snapshot

Enhancer hijacking activates GFI1 family oncogenes in medulloblastoma

inferredcites →https://doi.org/10.1038/nature14248openalex-snapshot

Integrative analysis of 111 reference human epigenomes

inferredcites →https://doi.org/10.1038/nature16508openalex-snapshot

Response and resistance to BET bromodomain inhibitors in triple-negative breast cancer

inferredcites →https://doi.org/10.1038/nature21063openalex-snapshot

Whole-genome landscape of pancreatic neuroendocrine tumours

inferredcites →https://doi.org/10.1038/nbt.2049openalex-snapshot

High order chromatin architecture shapes the landscape of chromosomal alterations in cancer

inferredcites →https://doi.org/10.1038/ncomms5846openalex-snapshot

The landscape of kinase fusions in cancer

inferredcites →https://doi.org/10.1038/ng.2682openalex-snapshot

Recurrent somatic alterations of FGFR1 and NTRK2 in pilocytic astrocytoma

inferredcites →https://doi.org/10.1038/ng.2760openalex-snapshot

Pan-cancer patterns of somatic copy number alteration

inferredcites →https://doi.org/10.1038/ng.3500openalex-snapshot

MYB-QKI rearrangements in angiocentric glioma drive tumorigenicity through a tripartite mechanism

inferredcites →https://doi.org/10.1038/ng.3722openalex-snapshot

Pan-cancer analysis of somatic copy-number alterations implicates IRS4 and IGF2 in enhancer hijacking

inferredcites →https://doi.org/10.1038/ng.3781openalex-snapshot

Systematic analysis of telomere length and somatic alterations in 31 cancer types

inferredcites →https://doi.org/10.1038/nrc.2017.5openalex-snapshot

Tissue-specific tumorigenesis: context matters

inferredcites →https://doi.org/10.1038/nrc3947openalex-snapshot

The emerging complexity of gene fusions in cancer

inferredcites →https://doi.org/10.1038/nrg2593openalex-snapshot

Mechanisms of change in gene copy number

inferredcites →https://doi.org/10.1038/nrg2883openalex-snapshot

Triggers for genomic rearrangements: insights into genomic, cellular and environmental influences

inferredcites →https://doi.org/10.1038/onc.2008.292openalex-snapshot

Klotho: a tumor suppressor and a modulator of the IGF-1 and FGF pathways in human breast cancer

inferredcites →https://doi.org/10.1038/sj.onc.1209802openalex-snapshot

DSG3 is overexpressed in head neck cancer and is a potential molecular target for inhibition of oncogenesis

inferredcites →https://doi.org/10.1038/sj.onc.1210406openalex-snapshot

Identification of a PP2A-interacting protein that functions as a negative regulator of phosphatase activity in the ATM/ATR signaling pathway

inferredcites →https://doi.org/10.1056/nejmra1112635openalex-snapshot

Targeting Epigenetic Readers in Cancer

inferredcites →https://doi.org/10.1073/pnas.0504501102openalex-snapshot

Deleted in liver cancer 2 ( DLC2 ) suppresses cell transformation by means of inhibition of RhoA activity

inferredcites →https://doi.org/10.1073/pnas.1418577112openalex-snapshot

Single-molecule analysis reveals widespread structural variation in multiple myeloma

inferredcites →https://doi.org/10.1073/pnas.1422165112openalex-snapshot

An in-tumor genetic screen reveals that the BET bromodomain protein, BRD4, is a potential therapeutic target in ovarian carcinoma

inferredcites →https://doi.org/10.1073/pnas.1614265114openalex-snapshot

THADA fusion is a mechanism of IGF2BP3 activation and IGF1R signaling in thyroid cancer

inferredcites →https://doi.org/10.1093/bioinformatics/btu356openalex-snapshot

Toward better understanding of artifacts in variant calling from high-coverage samples

inferredcites →https://doi.org/10.1093/carcin/bgh240openalex-snapshot

Oxidative damage-related genes AKR1C3 and OGG1 modulate risks for lung cancer due to exposure to PAH-rich coal combustion emissions

inferredcites →https://doi.org/10.1093/carcin/bgp195openalex-snapshot

Induction of neoplastic transformation by ectopic expression of human aldo-keto reductase 1C isoforms in NIH3T3 cells

inferredcites →https://doi.org/10.1093/emboj/19.14.3822openalex-snapshot

Inverted Alu repeats unstable in yeast are excluded from the human genome

inferredcites →https://doi.org/10.1093/nar/gkh103openalex-snapshot

The UCSC Table Browser data retrieval tool

inferredcites →https://doi.org/10.1097/jto.0b013e31815e8b60openalex-snapshot

EML4-ALK Fusion Is Linked to Histological Characteristics in a Subset of Lung Cancers

inferredcites →https://doi.org/10.1101/gr.080531.108openalex-snapshot

The consensus coding sequence (CCDS) project: Identifying a common protein-coding gene set for the human and mouse genomes

inferredcites →https://doi.org/10.1101/gr.158801openalex-snapshot

Biased Distribution of Inverted and Direct Alus in the Human Genome: Implications for Insertion, Exclusion, and Genome Stability

inferredcites →https://doi.org/10.1101/gr.163519.113openalex-snapshot

Functional and topological characteristics of mammalian regulatory domains

inferredcites →https://doi.org/10.1101/gr.191189.115openalex-snapshot

Read clouds uncover variation in complex regions of the human genome

inferredcites →https://doi.org/10.1101/gr.214007.116openalex-snapshot

Discovery and genotyping of structural variation from long-read haploid genome sequence data

inferredcites →https://doi.org/10.1111/j.2517-6161.1995.tb02031.xopenalex-snapshot

Controlling the False Discovery Rate: A Practical and Powerful Approach to Multiple Testing

inferredcites →https://doi.org/10.1126/science.1181369openalex-snapshot

Comprehensive Mapping of Long-Range Interactions Reveals Folding Principles of the Human Genome

inferredcites →https://doi.org/10.1128/mcb.22.18.6509-6520.2002openalex-snapshot

A Mammalian Bromodomain Protein, Brd4, Interacts with Replication Factor C and Inhibits Progression to S Phase

inferredcites →https://doi.org/10.1128/mcb.5.10.2599openalex-snapshot

Mechanisms of Nonhomologous Recombination in Mammalian Cells

inferredcites →https://doi.org/10.1158/0008-5472.can-13-2066openalex-snapshot

Notch3 Pathway Alterations in Ovarian Cancer

inferredcites →https://doi.org/10.1158/1541-7786.mcr-16-0003openalex-snapshot

Understanding TERT Promoter Mutations: A Common Path to Immortality

inferredcites →https://doi.org/10.1186/gb-2011-12-4-r41openalex-snapshot

GISTIC2.0 facilitates sensitive and confident localization of the targets of focal somatic copy-number alteration in human cancers

inferredcites →https://doi.org/10.1371/journal.pgen.1003026openalex-snapshot

Microhomology Directs Diverse DNA Break Repair Pathways and Chromosomal Translocations

inferredcites →https://doi.org/10.1371/journal.pone.0044196openalex-snapshot

Three-Dimensional Genome Architecture Influences Partner Selection for Chromosomal Translocations in Human Disease

inferredcites →https://doi.org/10.1371/journal.pone.0064088openalex-snapshot

DSG3 Facilitates Cancer Cell Growth and Invasion through the DSG3-Plakoglobin-TCF/LEF-Myc/Cyclin D1/MMP Signaling Pathway

inferredcites →https://doi.org/10.17615/hvp3-wg08openalex-snapshot

Integrated genomic analyses of ovarian carcinoma

inferredcites →https://doi.org/10.2353/ajpath.2010.100316openalex-snapshot

Notch3 Overexpression Is Related to the Recurrence of Ovarian Cancer and Confers Resistance to Carboplatin

inferredcites →https://doi.org/10.4172/1948-5956.1000119openalex-snapshot

TMPRSS2-ERG Fusion Gene Expression in Prostate Tumor Cells and Its Clinical and Biological Significance in Prostate Cancer Progression

inferredcites →https://doi.org/10.7554/elife.04449openalex-snapshot

LINKIN, a new transmembrane protein necessary for cell adhesion

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.1016/j.lungcan.2021.02.016openalex-snapshot

Revealing the BRD4-NOTCH3 fusion: A novel hill in the cancer landscape

inferred← citeshttps://doi.org/10.1016/j.tcb.2018.10.006openalex-snapshot

Homologous Recombination and the Formation of Complex Genomic Rearrangements

inferred← citeshttps://doi.org/10.1038/s12276-018-0112-3openalex-snapshot

Patterns and mechanisms of structural variations in human cancer

inferred← citeshttps://doi.org/10.1038/s41408-020-00367-2openalex-snapshot

An enhanced genetic model of relapsed IGH-translocated multiple myeloma evolutionary dynamics

inferred← citeshttps://doi.org/10.1038/s41467-018-07552-9openalex-snapshot

Cyclin A2/E1 activation defines a hepatocellular carcinoma subclass with a rearrangement signature of replication stress

inferred← citeshttps://doi.org/10.1038/s41467-019-11107-xopenalex-snapshot

Whole-genome landscape of mucosal melanoma reveals diverse drivers and therapeutic targets

inferred← citeshttps://doi.org/10.1038/s41467-019-13885-wopenalex-snapshot

High-coverage whole-genome analysis of 1220 cancers reveals hundreds of genes deregulated by rearrangement-mediated cis-regulatory alterations

inferred← citeshttps://doi.org/10.1038/s41586-024-07747-9openalex-snapshot

The genomic landscape of 2,023 colorectal cancers

inferred← citeshttps://doi.org/10.1038/s41588-018-0331-5openalex-snapshot

The landscape of selection in 551 esophageal adenocarcinomas defines genomic biomarkers for the clinic

inferred← citeshttps://doi.org/10.1038/s41588-021-00920-0openalex-snapshot

Genomic and evolutionary classification of lung cancer in never smokers

inferred← citeshttps://doi.org/10.1038/s42003-022-03238-7openalex-snapshot

Rearrangement processes and structural variations show evidence of selection in oesophageal adenocarcinomas

inferred← citeshttps://doi.org/10.1073/pnas.1808631116openalex-snapshot

Chromatin features constrain structural variation across evolutionary timescales

inferred← citeshttps://doi.org/10.1093/bib/bbad520openalex-snapshot

Accurate identification of structural variations from cancer samples

inferred← citeshttps://doi.org/10.1101/179705openalex-snapshot

Pan-cancer analysis of whole genomes reveals driver rearrangements promoted by LINE-1 retrotransposition in human tumours

inferred← citeshttps://doi.org/10.1101/190330openalex-snapshot

The whole-genome panorama of cancer drivers

inferred← citeshttps://doi.org/10.1101/2019.12.18.881086openalex-snapshot

Revealing the impact of recurrent and rare structural variants in multiple myeloma

inferred← citeshttps://doi.org/10.1101/214494openalex-snapshot

A deep learning system can accurately classify primary and metastatic cancers based on patterns of passenger mutations

inferred← citeshttps://doi.org/10.1101/522524openalex-snapshot

R-loops and regulatory changes in chronologically ageing fission yeast cells drive non-random patterns of genome rearrangements

inferred← citeshttps://doi.org/10.1101/739474openalex-snapshot

SVFX: a machine-learning framework to quantify the pathogenicity of structural variants

inferred← citeshttps://doi.org/10.1101/761510openalex-snapshot

Improving personalized prediction of cancer prognoses with clonal evolution models

inferred← citeshttps://doi.org/10.1146/annurev-genom-120121-101149openalex-snapshot

Structural Variation in Cancer: Role, Prevalence, and Mechanisms

inferred← citeshttps://doi.org/10.1186/s13059-019-1818-9openalex-snapshot

Global impact of somatic structural variation on the DNA methylome of human cancers

inferred← citeshttps://doi.org/10.1186/s13059-020-02178-xopenalex-snapshot

SVFX: a machine learning framework to quantify the pathogenicity of structural variants

inferred← citeshttps://doi.org/10.1371/journal.pcbi.1008777openalex-snapshot

Assessing the contribution of tumor mutational phenotypes to cancer progression risk

inferred← citeshttps://doi.org/10.1371/journal.pgen.1009784openalex-snapshot

R-loops and regulatory changes in chronologically ageing fission yeast cells drive non-random patterns of genome rearrangements

inferred← citeshttps://doi.org/10.21203/rs.3.rs-136297/v1openalex-snapshot

Annotations of Recurrent Structural Variant Events in Pan-cancer Whole Genome Data for Precision Medicine

inferred← citeshttps://doi.org/10.3390/cells11010075openalex-snapshot

Three-Dimensional Genome Organization in Breast and Gynecological Cancers: How Chromatin Folding Influences Tumorigenic Transcriptional Programs

inferred← citeshttps://doi.org/10.70675/d5e7c2e4zaff4z46eaza0fcza52f9c3d46f1openalex-snapshot

Analyse de signatures transcriptomiques et épigénétiques des carcinomes hépatocellulaires

inferred← citeshttps://doi.org/10.70675/e88d9a08za929z4c0fzb8d9z9f01cd69ca39openalex-snapshot

Adeno-associated virus in the liver : natural history of the infection and consequences in tumor development

inferred← citeshttps://doi.org/10.7554/elife.37294openalex-snapshot

Tumor copy number alteration burden is a pan-cancer prognostic factor associated with recurrence and death

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.