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The antioxidant drug N-acetylcysteine abolishes SOS-mediated mutagenesis produced by fluoroquinolones in bacteria

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/anie.201509340openalex-snapshot

Reactive Oxygen Species Play an Important Role in the Bactericidal Activity of Quinolone Antibiotics

inferredcites →https://doi.org/10.1007/s12275-017-7089-9openalex-snapshot

N-acetylcysteine prevents the development of gastritis induced by Helicobacter pylori infection

inferredcites →https://doi.org/10.1016/j.bbagen.2013.04.016openalex-snapshot

The chemistry and biological activities of N-acetylcysteine

inferredcites →https://doi.org/10.1016/j.cbi.2008.05.003openalex-snapshot

Antioxidant and radical scavenging properties of curcumin

inferredcites →https://doi.org/10.1016/j.cell.2007.06.049openalex-snapshot

A Common Mechanism of Cellular Death Induced by Bactericidal Antibiotics

inferredcites →https://doi.org/10.1016/j.chembiol.2016.02.010openalex-snapshot

RecA Inhibitors Potentiate Antibiotic Activity and Block Evolution of Antibiotic Resistance

inferredcites →https://doi.org/10.1016/j.lfs.2016.03.003openalex-snapshot

A minireview on N -acetylcysteine: An old drug with new approaches

inferredcites →https://doi.org/10.1016/j.mib.2012.07.007openalex-snapshot

Antimicrobials as promoters of genetic variation

inferredcites →https://doi.org/10.1016/j.micinf.2010.04.009openalex-snapshot

SOS regulatory elements are essential for UPEC pathogenesis

inferredcites →https://doi.org/10.1016/j.molcel.2010.01.003openalex-snapshot

Sublethal Antibiotic Treatment Leads to Multidrug Resistance via Radical-Induced Mutagenesis

inferredcites →https://doi.org/10.1016/j.pupt.2004.01.007openalex-snapshot

Effect of inhaled N-acetylcysteine on hydrogen peroxide exhalation in healthy subjects

inferredcites →https://doi.org/10.1016/s0921-8777(99)00073-7openalex-snapshot

Efficient repair of hydrogen peroxide-induced DNA damage by Escherichia coli requires SOS induction of RecA and RuvA proteins

inferredcites →https://doi.org/10.1021/acs.biochem.5b00109openalex-snapshot

Targets for Combating the Evolution of Acquired Antibiotic Resistance

inferredcites →https://doi.org/10.1021/acsinfecdis.7b00122openalex-snapshot

Inhibitors of LexA Autoproteolysis and the Bacterial SOS Response Discovered by an Academic–Industry Partnership

inferredcites →https://doi.org/10.1021/bi00215a030openalex-snapshot

Mutagenic spectrum resulting from DNA damage by oxygen radicals

inferredcites →https://doi.org/10.1038/msb4100135openalex-snapshot

Gyrase inhibitors induce an oxidative damage cellular death pathway in Escherichia coli

inferredcites →https://doi.org/10.1046/j.1365-2958.2000.01826.xopenalex-snapshot

Identification of additional genes belonging to the LexA regulon in Escherichia coli

inferredcites →https://doi.org/10.1073/pnas.1401876111openalex-snapshot

Antibiotics induce redox-related physiological alterations as part of their lethality

inferredcites →https://doi.org/10.1073/pnas.152046799openalex-snapshot

Assigning numbers to the arrows: Parameterizing a gene regulation network by using accurate expression kinetics

inferredcites →https://doi.org/10.1073/pnas.81.14.4490openalex-snapshot

Cell-division control in Escherichia coli: specific induction of the SOS function SfiA protein is sufficient to block septation.

inferredcites →https://doi.org/10.1073/pnas.93.24.13635openalex-snapshot

Superoxide accelerates DNA damage by elevating free-iron levels

inferredcites →https://doi.org/10.1080/10409230701597741openalex-snapshot

Controlling Mutation: Intervening in Evolution as a Therapeutic Strategy

inferredcites →https://doi.org/10.1089/adt.2011.0409openalex-snapshot

High-Throughput Screening for RecA Inhibitors Using a Transcreener Adenosine 5′- O -Diphosphate Assay

inferredcites →https://doi.org/10.1093/jac/dkq496openalex-snapshot

Effect of recA inactivation on mutagenesis of Escherichia coli exposed to sublethal concentrations of antimicrobials

inferredcites →https://doi.org/10.1093/jac/dku080openalex-snapshot

Suramin is a potent and selective inhibitor of Mycobacterium tuberculosis RecA protein and the SOS response: RecA as a potential target for antibacterial drug discovery

inferredcites →https://doi.org/10.1111/j.1365-2958.2007.05642.xopenalex-snapshot

Antibiotic‐mediated recombination: ciprofloxacin stimulates SOS‐independent recombination of divergent sequences in Escherichia coli

inferredcites →https://doi.org/10.1126/science.1219192openalex-snapshot

Oxidation of the Guanine Nucleotide Pool Underlies Cell Death by Bactericidal Antibiotics

inferredcites →https://doi.org/10.1126/science.1232688openalex-snapshot

Killing by Bactericidal Antibiotics Does Not Depend on Reactive Oxygen Species

inferredcites →https://doi.org/10.1126/science.1232751openalex-snapshot

Cell Death from Antibiotics Without the Involvement of Reactive Oxygen Species

inferredcites →https://doi.org/10.1128/9781555816704openalex-snapshot

DNA Repair and Mutagenesis

inferredcites →https://doi.org/10.1128/aac.00013-11openalex-snapshot

Ecology and Evolution as Targets: the Need for Novel Eco-Evo Drugs and Strategies To Fight Antibiotic Resistance

inferredcites →https://doi.org/10.1128/aac.00017-15openalex-snapshot

N-Acetylcysteine Selectively Antagonizes the Activity of Imipenem in Pseudomonas aeruginosa by an OprD-Mediated Mechanism

inferredcites →https://doi.org/10.1128/aac.01334-16openalex-snapshot

Effect of High N -Acetylcysteine Concentrations on Antibiotic Activity against a Large Collection of Respiratory Pathogens

inferredcites →https://doi.org/10.1128/aac.01415-16openalex-snapshot

Mutational Consequences of Ciprofloxacin in Escherichia coli

inferredcites →https://doi.org/10.1128/aac.01617-06openalex-snapshot

Quinolone-Mediated Bacterial Death

inferredcites →https://doi.org/10.1128/jb.00393-13openalex-snapshot

Role of Autofluorescence in Flow Cytometric Analysis of Escherichia coli Treated with Bactericidal Antibiotics

inferredcites →https://doi.org/10.1128/jb.142.1.319-321.1980openalex-snapshot

The recA+ gene product is more important than catalase and superoxide dismutase in protecting Escherichia coli against hydrogen peroxide toxicity

inferredcites →https://doi.org/10.1128/jb.171.11.6141-6147.1989openalex-snapshot

Induction of the SOS response by hydrogen peroxide in various Escherichia coli mutants with altered protection against oxidative DNA damage

inferredcites →https://doi.org/10.1128/jb.181.3.916-922.1999openalex-snapshot

Recovery of DNA Replication in UV-Irradiated Escherichia coli Requires both Excision Repair and RecF Protein Function

inferredcites →https://doi.org/10.1128/mbio.00971-17openalex-snapshot

Quinolone Resistance Reversion by Targeting the SOS Response

inferredcites →https://doi.org/10.1128/mmbr.61.3.377-392.1997openalex-snapshot

DNA gyrase, topoisomerase IV, and the 4-quinolones

inferredcites →https://doi.org/10.1128/msphere.00163-16openalex-snapshot

Systematically Altering Bacterial SOS Activity under Stress Reveals Therapeutic Strategies for Potentiating Antibiotics

inferredcites →https://doi.org/10.1146/annurev-micro-090817-062139openalex-snapshot

Antibiotic-Induced Genetic Variation: How It Arises and How It Can Be Prevented

inferredcites →https://doi.org/10.1146/annurev-pharmtox-010814-124712openalex-snapshot

Unraveling the Physiological Complexities of Antibiotic Lethality

inferredcites →https://doi.org/10.1146/annurev.biochem.71.083101.124707openalex-snapshot

Error-Prone Repair DNA Polymerases in Prokaryotes and Eukaryotes

inferredcites →https://doi.org/10.1146/annurev.biochem.77.061606.161055openalex-snapshot

Cellular Defenses against Superoxide and Hydrogen Peroxide

inferredcites →https://doi.org/10.1159/000335791openalex-snapshot

Baicalein Suppresses the SOS Response System of Staphylococcus Aureus Induced by Ciprofloxacin

inferredcites →https://doi.org/10.1186/1471-2180-10-140openalex-snapshot

N-acetylcysteine inhibit biofilms produced by Pseudomonas aeruginosa

inferredcites →https://doi.org/10.1242/jcs.1984.supplement_6.19openalex-snapshot

Toxicity, Mutagenesis and Stress Responses Induced in Escherichia Coli by Hydrogen Peroxide

inferredcites →https://doi.org/10.1371/journal.pbio.0030176openalex-snapshot

Inhibition of Mutation and Combating the Evolution of Antibiotic Resistance

inferredcites →https://doi.org/10.1371/journal.pbio.2005056openalex-snapshot

Death and population dynamics affect mutation rate estimates and evolvability under stress in bacteria

inferredcites →https://doi.org/10.1371/journal.pgen.1000760openalex-snapshot

SOS Response Induces Persistence to Fluoroquinolones in Escherichia coli

inferredcites →https://doi.org/10.1371/journal.pgen.1003144openalex-snapshot

Starvation, Together with the SOS Response, Mediates High Biofilm-Specific Tolerance to the Fluoroquinolone Ofloxacin

inferredcites →https://doi.org/10.1371/journal.pone.0203941openalex-snapshot

In vitro activity of N-acetylcysteine against Stenotrophomonas maltophilia and Burkholderia cepacia complex grown in planktonic phase and biofilm

inferredcites →https://doi.org/10.1534/g3.117.300120openalex-snapshot

rSalvador: An R Package for the Fluctuation Experiment

inferredcites →https://doi.org/10.1534/genetics.107.072405openalex-snapshot

Involvement ofEscherichia coliDNA Polymerase IV in Tolerance of Cytotoxic Alkylating DNA Lesionsin Vivo

inferredcites →https://doi.org/10.3389/fmicb.2016.01930openalex-snapshot

Identification and Characterization of Novel Compounds Blocking Shiga Toxin Expression in Escherichia coli O157:H7

inferredcites →https://doi.org/10.4161/cc.25691openalex-snapshot

DNA polymerases are error-prone at RecA-mediated recombination intermediates

inferredcites →https://doi.org/10.7150/ijbs.4.338openalex-snapshot

Inducible SOS Response System of DNA Repair and Mutagenesis in Escherichia coli

inferredcites →https://doi.org/10.7554/elife.01222openalex-snapshot

Engineered proteins detect spontaneous DNA breakage in human and bacterial cells

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.1016/j.lfs.2020.118562openalex-snapshot

The central role of the SOS DNA repair system in antibiotics resistance: A new target for a new infectious treatment strategy

inferred← citeshttps://doi.org/10.1101/846378openalex-snapshot

Unveiling the physiological mechanisms that drive the emergence of antibiotic resisters from antibiotic persister population of mycobacteria

inferred← citeshttps://doi.org/10.1371/journal.pgen.1008654openalex-snapshot

Global mistranslation increases cell survival under stress in Escherichia coli

inferred← citeshttps://doi.org/10.3390/pharmaceutics13081289openalex-snapshot

Structural Characterization of the Millennial Antibacterial (Fluoro)Quinolones—Shaping the Fifth Generation

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