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Galectin-3, A Novel Endogenous Trem2 Ligand, Regulates Inflammatory Response and Aβ Fibrilation in Alzheimer’s Disease

https://doi.org/10.1101/477927
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Candidate Single-Nucleotide Polymorphisms From a Genomewide Association Study of Alzheimer Disease
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Galectin‐3‐Binding Glycomimetics that Strongly Reduce Bleomycin‐Induced Lung Fibrosis and Modulate Intracellular Glycan Recognition
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Preparation of Primary Microglia Cultures from Postnatal Mouse and Rat Brains
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The HERC1 E3 Ubiquitin Ligase is essential for normal development and for neurotransmission at the mouse neuromuscular junction
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Clearance of cerebral Aβ in Alzheimer’s disease: reassessing the role of microglia and monocytes
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Alzheimer’s disease is not “brain aging”: neuropathological, genetic, and epidemiological human studies
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Abnormal accumulation of autophagic vesicles correlates with axonal and synaptic pathology in young Alzheimer’s mice hippocampus
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Microglia actions in Alzheimer’s disease
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Nitric Oxide Decreases the Enzymatic Activity of Insulin Degrading Enzyme in APP/PS1 Mice
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Inflammatory Cytokines and Alzheimer’s Disease: A Review from the Perspective of Genetic Polymorphisms
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Fluorometric determination of amyloid fibrils in vitro using the fluorescent dye, thioflavine T
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Fluorescence polarization as an analytical tool to evaluate galectin–ligand interactions
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TREM2 Lipid Sensing Sustains the Microglial Response in an Alzheimer’s Disease Model
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A Unique Microglia Type Associated with Restricting Development of Alzheimer’s Disease
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Microglia-Secreted Galectin-3 Acts as a Toll-like Receptor 4 Ligand and Contributes to Microglial Activation
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Temporal Tracking of Microglia Activation in Neurodegeneration at Single-Cell Resolution
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The TREM2-APOE Pathway Drives the Transcriptional Phenotype of Dysfunctional Microglia in Neurodegenerative Diseases
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Ways toward an early diagnosis in Alzheimer's disease: The Alzheimer's Disease Neuroimaging Initiative (ADNI)
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Galectin-3 contributes to neonatal hypoxic–ischemic brain injury
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Immune hyperreactivity of Aβ plaque-associated microglia in Alzheimer's disease
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Nitration of Tyrosine 10 Critically Enhances Amyloid β Aggregation and Plaque Formation
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Il10 Deficiency Rebalances Innate Immunity to Mitigate Alzheimer-Like Pathology
inferredcites →https://doi.org/10.1016/j.neuron.2016.05.003openalex-snapshot
TREM2 Haplodeficiency in Mice and Humans Impairs the Microglia Barrier Function Leading to Decreased Amyloid Compaction and Severe Axonal Dystrophy
inferredcites →https://doi.org/10.1016/j.neuron.2018.02.002openalex-snapshot
Elevated TREM2 Gene Dosage Reprograms Microglia Responsivity and Ameliorates Pathological Phenotypes in Alzheimer’s Disease Models
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Caspases orchestrate microglia instrumental functions
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Alzheimer's disease
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Genetic insights in Alzheimer's disease
inferredcites →https://doi.org/10.1016/s1474-4422(15)70016-5openalex-snapshot
Neuroinflammation in Alzheimer's disease
inferredcites →https://doi.org/10.1021/acs.jmedchem.7b01626openalex-snapshot
Systematic Tuning of Fluoro-galectin-3 Interactions Provides Thiodigalactoside Derivatives with Single-Digit nM Affinity and High Selectivity
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Super Resolution Imaging of Nanoparticles Cellular Uptake and Trafficking
inferredcites →https://doi.org/10.1021/bi1009584openalex-snapshot
Monovalent Interactions of Galectin-1
inferredcites →https://doi.org/10.1038/mp.2013.86openalex-snapshot
ATP5H/KCTD2 locus is associated with Alzheimer's disease risk
inferredcites →https://doi.org/10.1038/nature11729openalex-snapshot
NLRP3 is activated in Alzheimer’s disease and contributes to pathology in APP/PS1 mice
inferredcites →https://doi.org/10.1038/nature25158openalex-snapshot
Microglia-derived ASC specks cross-seed amyloid-β in Alzheimer’s disease
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NetworkAnalyst for statistical, visual and network-based meta-analysis of gene expression data
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Innate immune activation in neurodegenerative disease
inferredcites →https://doi.org/10.1038/nrn.2016.7openalex-snapshot
TREM2 variants: new keys to decipher Alzheimer disease pathogenesis
inferredcites →https://doi.org/10.1038/s41583-018-0057-5openalex-snapshot
Microglial signatures and their role in health and disease
inferredcites →https://doi.org/10.1038/srep41689openalex-snapshot
Galectin-3 released in response to traumatic brain injury acts as an alarmin orchestrating brain immune response and promoting neurodegeneration
inferredcites →https://doi.org/10.1056/nejmoa1211851openalex-snapshot
TREM2 Variants in Alzheimer's Disease
inferredcites →https://doi.org/10.1073/pnas.1604032113openalex-snapshot
IL-33 ameliorates Alzheimer’s disease-like pathology and cognitive decline
inferredcites →https://doi.org/10.1073/pnas.1710311114openalex-snapshot
TREM2 deficiency attenuates neuroinflammation and protects against neurodegeneration in a mouse model of tauopathy
inferredcites →https://doi.org/10.1074/jbc.273.49.32730openalex-snapshot
Insulin-degrading Enzyme Regulates Extracellular Levels of Amyloid β-Protein by Degradation
inferredcites →https://doi.org/10.1074/jbc.m109.098160openalex-snapshot
Mutational Tuning of Galectin-3 Specificity and Biological Function
inferredcites →https://doi.org/10.1074/jbc.m111.267583openalex-snapshot
Neuronal Death Induced by Nanomolar Amyloid β Is Mediated by Primary Phagocytosis of Neurons by Microglia
inferredcites →https://doi.org/10.1074/jbc.m113.487793openalex-snapshot
Galectin-3 Guides Intracellular Trafficking of Some Human Serotransferrin Glycoforms
inferredcites →https://doi.org/10.1084/jem.20142322openalex-snapshot
TREM2 deficiency eliminates TREM2+ inflammatory macrophages and ameliorates pathology in Alzheimer’s disease mouse models
inferredcites →https://doi.org/10.1084/jem.20160844openalex-snapshot
Soluble TREM2 induces inflammatory responses and enhances microglial survival
inferredcites →https://doi.org/10.1086/519795openalex-snapshot
PLINK: A Tool Set for Whole-Genome Association and Population-Based Linkage Analyses
inferredcites →https://doi.org/10.1093/bioinformatics/bth457openalex-snapshot
Haploview: analysis and visualization of LD and haplotype maps
inferredcites →https://doi.org/10.1093/brain/awx243openalex-snapshot
Tau pathology and neurodegeneration contribute to cognitive impairment in Alzheimer’s disease
inferredcites →https://doi.org/10.1093/glycob/cwm026openalex-snapshot
Affinity of galectin-8 and its carbohydrate recognition domains for ligands in solution and at the cell surface
inferredcites →https://doi.org/10.1093/nar/gkh103openalex-snapshot
The UCSC Table Browser data retrieval tool
inferredcites →https://doi.org/10.1093/nar/gkj144openalex-snapshot
The UCSC Genome Browser Database: update 2006
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A role for TREM2 ligands in the phagocytosis of apoptotic neuronal cells by microglia
inferredcites →https://doi.org/10.1126/science.1194516openalex-snapshot
Peripherally Applied Aβ-Containing Inoculates Induce Cerebral β-Amyloidosis
inferredcites →https://doi.org/10.1186/1742-2094-5-23openalex-snapshot
Toll-like receptor 4-dependent upregulation of cytokines in a transgenic mouse model of Alzheimer's disease
inferredcites →https://doi.org/10.1186/gm249openalex-snapshot
The membrane-spanning 4-domains, subfamily A (MS4A) gene cluster contains a common variant associated with Alzheimer's disease
inferredcites →https://doi.org/10.1186/s40478-015-0203-5openalex-snapshot
Induction of a common microglia gene expression signature by aging and neurodegenerative conditions: a co-expression meta-analysis
inferredcites →https://doi.org/10.1371/journal.pgen.1001308openalex-snapshot
Genome-Wide Association of Familial Late-Onset Alzheimer's Disease Replicates BIN1 and CLU and Nominates CUGBP2 in Interaction with APOE
inferredcites →https://doi.org/10.1371/journal.pone.0026560openalex-snapshot
Galectin-1-Binding Glycoforms of Haptoglobin with Altered Intracellular Trafficking, and Increase in Metastatic Breast Cancer Patients
inferredcites →https://doi.org/10.1371/journal.pone.0094661openalex-snapshot
Gene-Wide Analysis Detects Two New Susceptibility Genes for Alzheimer's Disease
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In VivoDetection of Age- and Disease-Related Increases in Neuroinflammation by18F-GE180 TSPO MicroPET Imaging in Wild-Type and Alzheimer's Transgenic Mice
inferredcites →https://doi.org/10.1523/jneurosci.1498-12.2012openalex-snapshot
Galectin-3 Is Required for Resident Microglia Activation and Proliferation in Response to Ischemic Injury
inferredcites →https://doi.org/10.15252/embr.201743922openalex-snapshot
TREM2 deficiency impairs chemotaxis and microglial responses to neuronal injury
inferredcites →https://doi.org/10.15252/emmm.201606370openalex-snapshot
TREM2 deficiency reduces the efficacy of immunotherapeutic amyloid clearance
inferredcites →https://doi.org/10.2174/13816128113199990068openalex-snapshot
Inhibition of Amyloid-β Aggregation in Alzheimer's Disease
inferredcites →https://doi.org/10.3233/jad-122257openalex-snapshot
Genetic Study of Neurexin and Neuroligin Genes in Alzheimer's Disease
inferredcites →https://doi.org/10.3233/jad-140495openalex-snapshot
Early Neuronal Loss and Axonal/Presynaptic Damage is Associated with Accelerated Amyloid-β Accumulation in AβPP/PS1 Alzheimer's Disease Mice Subiculum
inferredcites →https://doi.org/10.3791/53308openalex-snapshot
Intracerebroventricular Injection of Amyloid-β Peptides in Normal Mice to Acutely Induce Alzheimer-like Cognitive Deficits
inferredcites →https://doi.org/10.4049/jimmunol.1101121openalex-snapshot
TLR2 Is a Primary Receptor for Alzheimer’s Amyloid β Peptide To Trigger Neuroinflammatory Activation
inferredcites →https://doi.org/10.4049/jimmunol.1502532openalex-snapshot
Activated Microglia Desialylate and Phagocytose Cells via Neuraminidase, Galectin-3, and Mer Tyrosine Kinase
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