On the public citation graph

Transcriptional analysis defines TCR and cytokine-stimulated MAIT cells as rapid polyfunctional effector T cells that can coordinate the immune response

0 signed · 73 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.

Claim with ORCID

Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.

See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/0471142735.im0723s111openalex-snapshot

Isolation and Functional Analysis of Human Neutrophils

inferredcites →https://doi.org/10.1002/art.39117openalex-snapshot

CCR2 Expression in Neutrophils Plays a Critical Role in Their Migration Into the Joints in Rheumatoid Arthritis

inferredcites →https://doi.org/10.1002/eji.200737800openalex-snapshot

CD137 ligand reverse signaling has multiple functions in human dendritic cells during an adaptive immune response

inferredcites →https://doi.org/10.1002/eji.201343509openalex-snapshot

CD 161 ++ CD 8 + T cells, including the MAIT cell subset, are specifically activated by IL ‐12+ IL ‐18 in a TCR ‐independent manner

inferredcites →https://doi.org/10.1002/eji.201445313openalex-snapshot

IL‐15 dependent induction of IL‐18 secretion as a feedback mechanism controlling human MAIT‐cell effector functions

inferredcites →https://doi.org/10.1002/eji.201545969openalex-snapshot

TLR signaling in human antigen‐presenting cells regulates MR1‐dependent activation of MAIT cells

inferredcites →https://doi.org/10.1006/bbrc.2001.4363openalex-snapshot

Neutrophils and B Cells Express XCR1 Receptor and Chemotactically Respond to Lymphotactin

inferredcites →https://doi.org/10.1007/s00134-013-3163-xopenalex-snapshot

Specific MAIT cell behaviour among innate-like T lymphocytes in critically ill patients with severe infections

inferredcites →https://doi.org/10.1007/s10969-009-9070-2openalex-snapshot

The E. coli single protein production system for production and structural analysis of membrane proteins

inferredcites →https://doi.org/10.1016/b978-0-12-407708-9.00002-9openalex-snapshot

Epigenetic Control of Cytokine Gene Expression

inferredcites →https://doi.org/10.1016/j.cell.2017.12.033openalex-snapshot

Non-classical Immunity Controls Microbiota Impact on Skin Immunity and Tissue Repair

inferredcites →https://doi.org/10.1016/j.celrep.2019.08.050openalex-snapshot

TCR and Inflammatory Signals Tune Human MAIT Cells to Exert Specific Tissue Repair and Effector Functions

inferredcites →https://doi.org/10.1016/j.cyto.2014.11.010openalex-snapshot

Structural and agonist properties of XCL2, the other member of the C-chemokine subfamily

inferredcites →https://doi.org/10.1016/j.cyto.2017.07.007openalex-snapshot

TNFα and IL-1β in the synovial fluid facilitate mucosal-associated invariant T (MAIT) cell migration

inferredcites →https://doi.org/10.1016/j.immuni.2010.04.016openalex-snapshot

Epigenetic Instability of Cytokine and Transcription Factor Gene Loci Underlies Plasticity of the T Helper 17 Cell Lineage

inferredcites →https://doi.org/10.1016/j.immuni.2015.12.005openalex-snapshot

Diversity of T Cells Restricted by the MHC Class I-Related Molecule MR1 Facilitates Differential Antigen Recognition

inferredcites →https://doi.org/10.1016/j.jaci.2017.08.009openalex-snapshot

IL-12 and IL-7 synergize to control mucosal-associated invariant T-cell cytotoxic responses to bacterial infection

inferredcites →https://doi.org/10.1016/j.molimm.2010.12.002openalex-snapshot

The non-conventional MHC class I MR1 molecule controls infection by Klebsiella pneumoniae in mice

inferredcites →https://doi.org/10.1016/j.stem.2013.03.001openalex-snapshot

Expansion of Functional Human Mucosal-Associated Invariant T Cells via Reprogramming to Pluripotency and Redifferentiation

inferredcites →https://doi.org/10.1038/mi.2014.81openalex-snapshot

MAIT cells are licensed through granzyme exchange to kill bacterially sensitized targets

inferredcites →https://doi.org/10.1038/mi.2016.39openalex-snapshot

Mucosal-associated invariant T-cell activation and accumulation after in vivo infection depends on microbial riboflavin synthesis and co-stimulatory signals

inferredcites →https://doi.org/10.1038/nature01433openalex-snapshot

Selection of evolutionarily conserved mucosal-associated invariant T cells by MR1

inferredcites →https://doi.org/10.1038/nature11605openalex-snapshot

MR1 presents microbial vitamin B metabolites to MAIT cells

inferredcites →https://doi.org/10.1038/nature13160openalex-snapshot

T-cell activation by transitory neo-antigens derived from distinct microbial pathways

inferredcites →https://doi.org/10.1038/ncomms11653openalex-snapshot

MAIT cells are activated during human viral infections

inferredcites →https://doi.org/10.1038/ncomms4143openalex-snapshot

Acquisition of innate-like microbial reactivity in mucosal tissues during human fetal MAIT-cell development

inferredcites →https://doi.org/10.1038/ncomms4866openalex-snapshot

Parallel T-cell cloning and deep sequencing of human MAIT cells reveal stable oligoclonal TCRβ repertoire

inferredcites →https://doi.org/10.1038/ng1180openalex-snapshot

PGC-1α-responsive genes involved in oxidative phosphorylation are coordinately downregulated in human diabetes

inferredcites →https://doi.org/10.1038/ni.1890openalex-snapshot

Antimicrobial activity of mucosal-associated invariant T cells

inferredcites →https://doi.org/10.1038/ni.3211openalex-snapshot

TH17 cells promote microbial killing and innate immune sensing of DNA via interleukin 26

inferredcites →https://doi.org/10.1038/s41467-018-05202-8openalex-snapshot

MAIT cells protect against pulmonary Legionella longbeachae infection

inferredcites →https://doi.org/10.1038/s41467-018-07207-9openalex-snapshot

MAIT cells contribute to protection against lethal influenza infection in vivo

inferredcites →https://doi.org/10.1073/pnas.0506580102openalex-snapshot

Gene set enrichment analysis: A knowledge-based approach for interpreting genome-wide expression profiles

inferredcites →https://doi.org/10.1073/pnas.0914839107openalex-snapshot

Analysis of CD161 expression on human CD8 + T cells defines a distinct functional subset with tissue-homing properties

inferredcites →https://doi.org/10.1073/pnas.101329598openalex-snapshot

Granzymes A and B directly cleave lamins and disrupt the nuclear lamina during granule-mediated cytolysis

inferredcites →https://doi.org/10.1073/pnas.1302799110openalex-snapshot

MAIT cells are critical for optimal mucosal immune responses during in vivo pulmonary bacterial infection

inferredcites →https://doi.org/10.1073/pnas.1401750111openalex-snapshot

Loss of the death receptor CD95 (Fas) expression by dendritic cells protects from a chronic viral infection

inferredcites →https://doi.org/10.1073/pnas.1416922112openalex-snapshot

IL-4 abrogates T H 17 cell-mediated inflammation by selective silencing of IL-23 in antigen-presenting cells

inferredcites →https://doi.org/10.1073/pnas.1610750113openalex-snapshot

Human mucosal-associated invariant T cells contribute to antiviral influenza immunity via IL-18–dependent activation

inferredcites →https://doi.org/10.1073/pnas.1705759114openalex-snapshot

Multiple layers of heterogeneity and subset diversity in human MAIT cell responses to distinct microorganisms and to innate cytokines

inferredcites →https://doi.org/10.1074/jbc.m501087200openalex-snapshot

Evidence for MR1 Antigen Presentation to Mucosal-associated Invariant T Cells

inferredcites →https://doi.org/10.1084/jem.189.12.1907openalex-snapshot

An Invariant T Cell Receptor α Chain Defines a Novel TAP-independent Major Histocompatibility Complex Class Ib–restricted α/β T Cell Subpopulation in Mammals

inferredcites →https://doi.org/10.1093/intimm/dxs069openalex-snapshot

Analysis of stem-cell-like properties of human CD161++IL-18Rα+ memory CD8+ T cells

inferredcites →https://doi.org/10.1093/nar/gkw419openalex-snapshot

Heatmapper: web-enabled heat mapping for all

inferredcites →https://doi.org/10.1101/490649openalex-snapshot

Activation and in vivo evolution of the MAIT cell transcriptome in mice and humans reveals diverse functionality

inferredcites →https://doi.org/10.1111/imm.12925openalex-snapshot

Innate‐like CD 8+ T‐cells and NK cells: converging functions and phenotypes

inferredcites →https://doi.org/10.1128/iai.00279-12openalex-snapshot

Polyclonal Mucosa-Associated Invariant T Cells Have Unique Innate Functions in Bacterial Infection

inferredcites →https://doi.org/10.1128/iai.00455-10openalex-snapshot

Involvement of Inflammatory Chemokines in Survival of Human Monocytes Fed with Malarial Pigment

inferredcites →https://doi.org/10.1128/mbio.00334-16openalex-snapshot

CXCL10 Acts as a Bifunctional Antimicrobial Molecule against Bacillus anthracis

inferredcites →https://doi.org/10.1172/jci.insight.86292openalex-snapshot

Distinct activation thresholds of human conventional and innate-like memory T cells

inferredcites →https://doi.org/10.1182/blood-2010-08-303339openalex-snapshot

Human MAIT cells are xenobiotic-resistant, tissue-targeted, CD161hi IL-17–secreting T cells

inferredcites →https://doi.org/10.1182/blood-2011-02-334698openalex-snapshot

Innate signals overcome acquired TCR signaling pathway regulation and govern the fate of human CD161hi CD8α+ semi-invariant T cells

inferredcites →https://doi.org/10.1186/1471-2199-11-90openalex-snapshot

Selection of reliable reference genes during THP-1 monocyte differentiation into macrophages

inferredcites →https://doi.org/10.1186/s13059-014-0550-8openalex-snapshot

Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2

inferredcites →https://doi.org/10.1186/s13075-017-1257-5openalex-snapshot

Activation status of mucosal-associated invariant T cells reflects disease activity and pathology of systemic lupus erythematosus

inferredcites →https://doi.org/10.1189/jlb.4a0317-116ropenalex-snapshot

Human mucosal-associated invariant T (MAIT) cells possess capacity for B cell help

inferredcites →https://doi.org/10.1371/journal.pbio.1000054openalex-snapshot

Stepwise Development of MAIT Cells in Mouse and Human

inferredcites →https://doi.org/10.1371/journal.pbio.1000407openalex-snapshot

Human Mucosal Associated Invariant T Cells Detect Bacterially Infected Cells

inferredcites →https://doi.org/10.1371/journal.pone.0018553openalex-snapshot

Antigen-Independent IFN-γ Production by Human Naïve CD4+ T Cells Activated by IL-12 Plus IL-18

inferredcites →https://doi.org/10.1371/journal.pone.0058937openalex-snapshot

The E. coli Effector Protein NleF Is a Caspase Inhibitor

inferredcites →https://doi.org/10.1371/journal.pone.0109891openalex-snapshot

Reduced Mucosal Associated Invariant T-Cells Are Associated with Increased Disease Severity and Pseudomonas aeruginosa Infection in Cystic Fibrosis

inferredcites →https://doi.org/10.1371/journal.ppat.1003681openalex-snapshot

MAIT Cells Detect and Efficiently Lyse Bacterially-Infected Epithelial Cells

inferredcites →https://doi.org/10.1371/journal.ppat.1004210openalex-snapshot

Toll-Like Receptor 8 Agonist and Bacteria Trigger Potent Activation of Innate Immune Cells in Human Liver

inferredcites →https://doi.org/10.1371/journal.ppat.1004648openalex-snapshot

CXCL9 Contributes to Antimicrobial Protection of the Gut during Citrobacter rodentium Infection Independent of Chemokine-Receptor Signaling

inferredcites →https://doi.org/10.1371/journal.ppat.1005072openalex-snapshot

Arming of MAIT Cell Cytolytic Antimicrobial Activity Is Induced by IL-7 and Defective in HIV-1 Infection

inferredcites →https://doi.org/10.3389/fimmu.2017.01031openalex-snapshot

Shared and Distinct Phenotypes and Functions of Human CD161++ Vα7.2+ T Cell Subsets

inferredcites →https://doi.org/10.4049/jimmunol.1203218openalex-snapshot

IL-7 Licenses Activation of Human Liver Intrasinusoidal Mucosal-Associated Invariant T Cells

inferredcites →https://doi.org/10.4049/jimmunol.1401018openalex-snapshot

Microbe-Specific Unconventional T Cells Induce Human Neutrophil Differentiation into Antigen Cross-Presenting Cells

inferredcites →https://doi.org/10.4049/jimmunol.1600990openalex-snapshot

Unconventional Human T Cells Accumulate at the Site of Infection in Response to Microbial Ligands and Induce Local Tissue Remodeling

inferredcites →https://doi.org/10.4049/jimmunol.1700615openalex-snapshot

Activation of Human Mucosal-Associated Invariant T Cells Induces CD40L-Dependent Maturation of Monocyte-Derived and Primary Dendritic Cells

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.1002/eji.201948279openalex-snapshot

Type I interferons are important co‐stimulatory signals during T cell receptor mediated human MAIT cell activation

inferred← citeshttps://doi.org/10.1101/745414openalex-snapshot

Neutrophils Suppress Mucosal-Associated Invariant T Cells

inferred← citeshttps://doi.org/10.3389/fimmu.2024.1369236openalex-snapshot

Mucosal-associated invariant T cells in cancer: dual roles, complex interactions and therapeutic potential

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.