On the public citation graph

Neutrophils Suppress Mucosal-Associated Invariant T Cells

https://doi.org/10.1101/745414
0 signed · 47 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.
Claim with ORCID
Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.
See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/eji.200636542openalex-snapshot
Arginase activity mediates reversible T cell hyporesponsiveness in human pregnancy
inferredcites →https://doi.org/10.1002/eji.200939897openalex-snapshot
The A2aR adenosine receptor controls cytokine production in iNKT cells
inferredcites →https://doi.org/10.1002/eji.201343509openalex-snapshot
CD 161 ++ CD 8 + T cells, including the MAIT cell subset, are specifically activated by IL ‐12+ IL ‐18 in a TCR ‐independent manner
inferredcites →https://doi.org/10.1002/eji.201343664openalex-snapshot
Neutrophils suppress γδ T ‐cell function
inferredcites →https://doi.org/10.1002/eji.201545969openalex-snapshot
TLR signaling in human antigen‐presenting cells regulates MR1‐dependent activation of MAIT cells
inferredcites →https://doi.org/10.1002/jlb.4ru0418-161ropenalex-snapshot
Inducible nitric oxide synthase: An asset to neutrophils
inferredcites →https://doi.org/10.1007/s00018-013-1495-xopenalex-snapshot
Neutrophil uptake of nitrogen-bisphosphonates leads to the suppression of human peripheral blood γδ T cells
inferredcites →https://doi.org/10.1016/s1470-2045(03)01218-xopenalex-snapshot
Infections in patients with cancer undergoing chemotherapy: aetiology, prevention, and treatment
inferredcites →https://doi.org/10.1038/mi.2014.81openalex-snapshot
MAIT cells are licensed through granzyme exchange to kill bacterially sensitized targets
inferredcites →https://doi.org/10.1038/nature01433openalex-snapshot
Selection of evolutionarily conserved mucosal-associated invariant T cells by MR1
inferredcites →https://doi.org/10.1038/nature11605openalex-snapshot
MR1 presents microbial vitamin B metabolites to MAIT cells
inferredcites →https://doi.org/10.1038/nature13160openalex-snapshot
T-cell activation by transitory neo-antigens derived from distinct microbial pathways
inferredcites →https://doi.org/10.1073/pnas.0914839107openalex-snapshot
Analysis of CD161 expression on human CD8 + T cells defines a distinct functional subset with tissue-homing properties
inferredcites →https://doi.org/10.1073/pnas.1302799110openalex-snapshot
MAIT cells are critical for optimal mucosal immune responses during in vivo pulmonary bacterial infection
inferredcites →https://doi.org/10.1074/jbc.m110675200openalex-snapshot
Regulation of T Cell Receptor CD3ζ Chain Expression byl-Arginine
inferredcites →https://doi.org/10.1074/jbc.ra118.004789openalex-snapshot
Heterogeneity of hypochlorous acid production in individual neutrophil phagosomes revealed by a rhodamine-based probe
inferredcites →https://doi.org/10.1084/jem.20160637openalex-snapshot
MAIT cells promote inflammatory monocyte differentiation into dendritic cells during pulmonary intracellular infection
inferredcites →https://doi.org/10.1093/intimm/dxh320openalex-snapshot
Differential expression of the granzymes A, K and M and perforin in human peripheral blood lymphocytes
inferredcites →https://doi.org/10.1101/495556openalex-snapshot
Role of Innate-like Lymphocytes in the Pathogenesis of Community Acquired Pneumonia
inferredcites →https://doi.org/10.1101/600189openalex-snapshot
Transcriptional analysis defines TCR and cytokine-stimulated MAIT cells as rapid polyfunctional effector T cells that can coordinate the immune response
inferredcites →https://doi.org/10.1136/ard.49.9.665openalex-snapshot
Tumour necrosis factor in serum and synovial fluid of patients with active and severe rheumatoid arthritis.
inferredcites →https://doi.org/10.1158/1078-0432.ccr-11-1107openalex-snapshot
Neutrophil Degranulation and Immunosuppression in Patients with GBM: Restoration of Cellular Immune Function by Targeting Arginase I
inferredcites →https://doi.org/10.1161/01.res.0000250174.31269.70openalex-snapshot
ATP Release From Activated Neutrophils Occurs via Connexin 43 and Modulates Adenosine-Dependent Endothelial Cell Function
inferredcites →https://doi.org/10.1161/atvbaha.111.226845openalex-snapshot
Regulation of Neutrophil Function by Adenosine
inferredcites →https://doi.org/10.1164/rccm.201401-0106ocopenalex-snapshot
Mucosal-associated Invariant T-Cell Function Is Modulated by Programmed Death-1 Signaling in Patients with Active Tuberculosis
inferredcites →https://doi.org/10.1172/jci57990openalex-snapshot
A subset of neutrophils in human systemic inflammation inhibits T cell responses through Mac-1
inferredcites →https://doi.org/10.1182/blood-2006-11-010389openalex-snapshot
Suppression of T-cell functions by human granulocyte arginase
inferredcites →https://doi.org/10.1182/blood-2010-08-303339openalex-snapshot
Human MAIT cells are xenobiotic-resistant, tissue-targeted, CD161hi IL-17–secreting T cells
inferredcites →https://doi.org/10.1182/blood-2010-11-322206openalex-snapshot
A novel TNFR1-triggered apoptosis pathway mediated by class IA PI3Ks in neutrophils
inferredcites →https://doi.org/10.1182/blood.v87.10.4411.bloodjournal87104411openalex-snapshot
Release of oxygen metabolites from chemoattractant-stimulated neutrophils is inhibited by resting platelets: role of extracellular adenosine and actin polymerization
inferredcites →https://doi.org/10.1186/cc14003openalex-snapshot
Neutrophils with myeloid derived suppressor function deplete arginine and constrain T cell function in septic shock patients
inferredcites →https://doi.org/10.1189/jlb.1109737openalex-snapshot
Exocytosis of azurophil and arginase 1-containing granules by activated polymorphonuclear neutrophils is required to inhibit T lymphocyte proliferation
inferredcites →https://doi.org/10.1189/jlb.4a0317-116ropenalex-snapshot
Human mucosal-associated invariant T (MAIT) cells possess capacity for B cell help
inferredcites →https://doi.org/10.1378/chest.108.5.1303openalex-snapshot
Inflammatory Cytokines in the BAL of Patients With ARDS
inferredcites →https://doi.org/10.3389/fimmu.2014.00450openalex-snapshot
Mucosal-Associated Invariant T-Cells: New Players in Anti-Bacterial Immunity
inferredcites →https://doi.org/10.3389/fimmu.2015.00471openalex-snapshot
How Neutrophils Shape Adaptive Immune Responses
inferredcites →https://doi.org/10.3389/fimmu.2017.00093openalex-snapshot
Arginine Metabolism in Myeloid Cells Shapes Innate and Adaptive Immunity
inferredcites →https://doi.org/10.3389/fimmu.2018.00756openalex-snapshot
Unique and Common Features of Innate-Like Human Vδ2+ γδT Cells and Mucosal-Associated Invariant T Cells
inferredcites →https://doi.org/10.3389/fphys.2018.00113openalex-snapshot
Neutrophil: A Cell with Many Roles in Inflammation or Several Cell Types?
inferredcites →https://doi.org/10.4049/jimmunol.1101273openalex-snapshot
Neutrophilic Granulocytes Modulate Invariant NKT Cell Function in Mice and Humans
inferredcites →https://doi.org/10.4049/jimmunol.1401018openalex-snapshot
Microbe-Specific Unconventional T Cells Induce Human Neutrophil Differentiation into Antigen Cross-Presenting Cells
inferredcites →https://doi.org/10.4049/jimmunol.162.4.2347openalex-snapshot
Neutrophil Recruitment by Human IL-17 Via C-X-C Chemokine Release in the Airways
inferredcites →https://doi.org/10.4049/jimmunol.1700615openalex-snapshot
Activation of Human Mucosal-Associated Invariant T Cells Induces CD40L-Dependent Maturation of Monocyte-Derived and Primary Dendritic Cells
inferredcites →https://doi.org/10.4049/jimmunol.172.5.2731openalex-snapshot
Of Mice and Not Men: Differences between Mouse and Human Immunology

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.1146/annurev-immunol-080719-015428openalex-snapshot
MAIT Cells in Health and Disease
inferred← citeshttps://doi.org/10.3389/fimmu.2026.1931069openalex-snapshot
Multifaceted mechanisms of unconventional T cells in ischemia-reperfusion injury
inferred← citeshttps://doi.org/10.37349/ei.2022.00039openalex-snapshot
Novel insights based on the plasticity of γδ T cells in the tumor microenvironment
Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.