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α‐synuclein suppresses microglial autophagy and promotes neurodegeneration in a mouse model of Parkinson’s disease
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/glia.22437openalex-snapshot
Toll‐like receptor 4 is required for α‐synuclein dependent activation of microglia and astroglia
inferredcites →https://doi.org/10.1007/978-1-4939-6786-5_23openalex-snapshot
Isolation of Microglia and Immune Infiltrates from Mouse and Primate Central Nervous System
inferredcites →https://doi.org/10.1007/s00401-018-1907-yopenalex-snapshot
Picomolar concentrations of oligomeric alpha-synuclein sensitizes TLR4 to play an initiating role in Parkinson’s disease pathogenesis
inferredcites →https://doi.org/10.1007/s00401-018-1919-7openalex-snapshot
Correction to: Picomolar concentrations of oligomeric alpha-synuclein sensitizes TLR4 to play an initiating role in Parkinson’s disease pathogenesis
inferredcites →https://doi.org/10.1007/s00401-020-02227-6openalex-snapshot
Neuronal activity modulates alpha-synuclein aggregation and spreading in organotypic brain slice cultures and in vivo
inferredcites →https://doi.org/10.1016/j.bbi.2017.07.159openalex-snapshot
Impaired CBS-H2S signaling axis contributes to MPTP-induced neurodegeneration in a mouse model of Parkinson’s disease
inferredcites →https://doi.org/10.1016/j.bbi.2020.10.010openalex-snapshot
Impaired autophagy in microglia aggravates dopaminergic neurodegeneration by regulating NLRP3 inflammasome activation in experimental models of Parkinson’s disease
inferredcites →https://doi.org/10.1016/j.immuni.2007.05.022openalex-snapshot
Toll-like Receptor 4 Is a Sensor for Autophagy Associated with Innate Immunity
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2006.11.013openalex-snapshot
α-Synuclein activates stress signaling protein kinases in THP-1 cells and microglia
inferredcites →https://doi.org/10.1016/j.neuron.2013.06.046openalex-snapshot
Microglial Beclin 1 Regulates Retromer Trafficking and Phagocytosis and Is Impaired in Alzheimer’s Disease
inferredcites →https://doi.org/10.1016/j.neuron.2018.09.014openalex-snapshot
Abrogating Native α-Synuclein Tetramers in Mice Causes a L-DOPA-Responsive Motor Syndrome Closely Resembling Parkinson’s Disease
inferredcites →https://doi.org/10.1016/j.tox.2017.06.011openalex-snapshot
Mitochondrial dysfunction in glial cells: Implications for neuronal homeostasis and survival
inferredcites →https://doi.org/10.1038/mp.2016.103openalex-snapshot
Deficient autophagy in microglia impairs synaptic pruning and causes social behavioral defects
inferredcites →https://doi.org/10.1038/nature04723openalex-snapshot
Loss of autophagy in the central nervous system causes neurodegeneration in mice
inferredcites →https://doi.org/10.1038/nature04724openalex-snapshot
Suppression of basal autophagy in neural cells causes neurodegenerative disease in mice
inferredcites →https://doi.org/10.1038/nature10324openalex-snapshot
α-Synuclein occurs physiologically as a helically folded tetramer that resists aggregation
inferredcites →https://doi.org/10.1038/ncomms2534openalex-snapshot
Neuron-released oligomeric α-synuclein is an endogenous agonist of TLR2 for paracrine activation of microglia
inferredcites →https://doi.org/10.1038/s41467-020-15119-wopenalex-snapshot
Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration
inferredcites →https://doi.org/10.1038/s41593-018-0332-9openalex-snapshot
Mitophagy inhibits amyloid-β and tau pathology and reverses cognitive deficits in models of Alzheimer’s disease
inferredcites →https://doi.org/10.1038/s41593-019-0566-1openalex-snapshot
Lipid-droplet-accumulating microglia represent a dysfunctional and proinflammatory state in the aging brain
inferredcites →https://doi.org/10.1073/pnas.2023418118openalex-snapshot
Autophagy deficiency modulates microglial lipid homeostasis and aggravates tau pathology and spreading
inferredcites →https://doi.org/10.1074/jbc.m115.705095openalex-snapshot
Effects of Serine 129 Phosphorylation on α-Synuclein Aggregation, Membrane Association, and Internalization
inferredcites →https://doi.org/10.1080/15548627.2015.1100930openalex-snapshot
A pivotal role of FOS-mediated BECN1/Beclin 1 upregulation in dopamine D2 and D3 receptor agonist-induced autophagy activation
inferredcites →https://doi.org/10.1080/15548627.2017.1402992openalex-snapshot
Autophagy balances inflammation in innate immunity
inferredcites →https://doi.org/10.1080/15548627.2018.1556946openalex-snapshot
TLR4 (toll-like receptor 4) activation suppresses autophagy through inhibition of FOXO3 and impairs phagocytic capacity of microglia
inferredcites →https://doi.org/10.1080/15548627.2019.1635384openalex-snapshot
New insights into the interplay between autophagy, gut microbiota and inflammatory responses in IBD
inferredcites →https://doi.org/10.1080/15548627.2020.1719723openalex-snapshot
Microglial autophagy defect causes parkinson disease-like symptoms by accelerating inflammasome activation in mice
inferredcites →https://doi.org/10.1083/jcb.201701049openalex-snapshot
p38 MAPK inhibits autophagy and promotes microglial inflammatory responses by phosphorylating ULK1
inferredcites →https://doi.org/10.1093/brain/awaa090openalex-snapshot
Microglial exosomes facilitate α-synuclein transmission in Parkinson’s disease
inferredcites →https://doi.org/10.1093/brain/awv346openalex-snapshot
Induction of α-synuclein aggregate formation by CSF exosomes from patients with Parkinson’s disease and dementia with Lewy bodies
inferredcites →https://doi.org/10.1093/ijnp/pyu103openalex-snapshot
Toll-Like Receptor Expression in the Blood and Brain of Patients and a Mouse Model of Parkinson's Disease
inferredcites →https://doi.org/10.1111/acel.13522openalex-snapshot
α‐synuclein suppresses microglial autophagy and promotes neurodegeneration in a mouse model of Parkinson’s disease
inferredcites →https://doi.org/10.1111/j.1471-4159.2010.06695.xopenalex-snapshot
Non‐classical exocytosis of α‐synuclein is sensitive to folding states and promoted under stress conditions
inferredcites →https://doi.org/10.1111/jnc.13266openalex-snapshot
Genes associated with Parkinson's disease: regulation of autophagy and beyond
inferredcites →https://doi.org/10.1111/jnc.14222openalex-snapshot
DJ ‐1 deficiency impairs autophagy and reduces alpha‐synuclein phagocytosis by microglia
inferredcites →https://doi.org/10.1186/s13024-018-0241-0openalex-snapshot
Extracellular α-synuclein levels are regulated by neuronal activity
inferredcites →https://doi.org/10.1186/s13024-019-0335-3openalex-snapshot
Microglia affect α-synuclein cell-to-cell transfer in a mouse model of Parkinson’s disease
inferredcites →https://doi.org/10.1186/s40478-019-0865-5openalex-snapshot
Organotypic slice culture model demonstrates inter-neuronal spreading of alpha-synuclein aggregates
inferredcites →https://doi.org/10.1523/jneurosci.0209-12.2012openalex-snapshot
Development and Characterization of a New Parkinson's Disease Model Resulting from Impaired Autophagy
inferredcites →https://doi.org/10.1523/jneurosci.0692-05.2005openalex-snapshot
Intravesicular Localization and Exocytosis of α-Synuclein and its Aggregates
inferredcites →https://doi.org/10.1523/jneurosci.1292-12.2012openalex-snapshot
Antibody-Aided Clearance of Extracellular α-Synuclein Prevents Cell-to-Cell Aggregate Transmission
inferredcites →https://doi.org/10.15252/embj.201899430openalex-snapshot
Beclin1‐driven autophagy modulates the inflammatory response of microglia via NLRP3
inferredcites →https://doi.org/10.3389/fnagi.2018.00378openalex-snapshot
A Critical Role of Autophagy in Regulating Microglia Polarization in Neurodegeneration
inferredcites →https://doi.org/10.3791/57648openalex-snapshot
Quantifying Microglia Morphology from Photomicrographs of Immunohistochemistry Prepared Tissue Using ImageJ
inferredcites →https://doi.org/10.3791/57648-vopenalex-snapshot
Quantifying Microglia Morphology from Photomicrographs of Immunohistochemistry Prepared Tissue Using ImageJ
inferredcites →https://doi.org/10.4161/auto.29647openalex-snapshot
Autophagy in microglia degrades extracellular β-amyloid fibrils and regulates the NLRP3 inflammasome
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Inhibiting Nrf2 Expression Exacerbates Nlrp3 Inflammasome Activation and Dopamine Neuron Degeneration in Parkinson's Disease Models
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Taurochenodeoxycholic acid activates autophagy and suppresses inflammatory responses in microglia of MPTP-induced Parkinson’s disease mice via AMPK/mTOR, AKT/NFκB and Pink1/Parkin signaling pathways mediated by Takeda G protein-coupled receptor 5
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Splice Variant of Retinal G-Protein-Coupled Receptor Deletion-Mediated Dysregulation of Autophagy Increases the Susceptibility to Age-Related Macular Degeneration-Like Defects
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Osmotin-derived 9-amino-acid peptide alleviates α-synuclein and MPTP-induced glial cell activation mediated neuroinflammation, protecting dopaminergic neurons in Parkinson’s disease mice brain
inferred← citeshttps://doi.org/10.1186/s12974-024-03268-4openalex-snapshot
α-Synuclein disrupts microglial autophagy through STAT1-dependent suppression of Ulk1 transcription
inferred← citeshttps://doi.org/10.1186/s13024-024-00780-2openalex-snapshot
NOTCH2NLC GGC intermediate repeat with serine induces hypermyelination and early Parkinson’s disease-like phenotypes in mice
inferred← citeshttps://doi.org/10.1186/s13024-025-00827-yopenalex-snapshot
Immune cell metabolic dysfunction in Parkinson’s disease
inferred← citeshttps://doi.org/10.1186/s40035-023-00392-8openalex-snapshot
Parkinson’s disease and gut microbiota: from clinical to mechanistic and therapeutic studies
inferred← citeshttps://doi.org/10.1371/journal.pone.0281957openalex-snapshot
Ethyl pyruvate protects SHSY5Y cells against 6-hydroxydopamine-induced neurotoxicity by upregulating autophagy
inferred← citeshttps://doi.org/10.1631/jzus.b2400186openalex-snapshot
Rescuing lysosomal/autophagic defects via nanoapproach: implications for lysosomal/autophagic defect-related diseases
inferred← citeshttps://doi.org/10.21037/atm-22-5159openalex-snapshot
BAG3 promotes autophagy and suppresses NLRP3 inflammasome activation in Parkinson’s disease
inferred← citeshttps://doi.org/10.21203/rs.3.rs-9843195/v1openalex-snapshot
RUNX3 negatively regulates autophagic degradation of aggregated α-synuclein
inferred← citeshttps://doi.org/10.2147/ijn.s570452openalex-snapshot
Nanotherapeutic Interventions for Parkinson’s Disease: Modulating Pathogenic Mechanisms and Overcoming Therapeutic Obstacles
inferred← citeshttps://doi.org/10.2147/jir.s478683openalex-snapshot
Nrf2 Deficiency Exacerbates Parkinson’s Disease by Aggravating NLRP3 Inflammasome Activation in MPTP-Induced Mouse Models and LPS-Induced BV2 Cells
inferred← citeshttps://doi.org/10.2147/ndt.s476969openalex-snapshot
Shisandra Decoction Alleviates Parkinson’s Disease Symptoms in a Mouse Model Through PI3K/AKT/mTOR Signalling Pathway
inferred← citeshttps://doi.org/10.2174/0115672050336499240903113255openalex-snapshot
Microglial Circadian Rhythms and Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.31083/jin50932openalex-snapshot
Extracellular Vesicle-Mediated Delivery of VEGF and NGF Protects Dopaminergic Neurons in 6-OHDA-Induced Parkinson’s Disease Models
inferred← citeshttps://doi.org/10.3389/fcell.2024.1421360openalex-snapshot
Alpha-synuclein shapes monocyte and macrophage cell biology and functions by bridging alterations of autophagy and inflammatory pathways
inferred← citeshttps://doi.org/10.3389/fcell.2024.1510897openalex-snapshot
Glial cells improve Parkinson’s disease by modulating neuronal function and regulating neuronal ferroptosis
inferred← citeshttps://doi.org/10.3389/fcell.2025.1518991openalex-snapshot
Canonical and noncanonical autophagy: involvement in Parkinson’s disease
inferred← citeshttps://doi.org/10.3389/fcell.2026.1906331openalex-snapshot
Breaking the proteasome balance: α-synuclein and the ubiquitin-proteasome system
inferred← citeshttps://doi.org/10.3389/fimmu.2022.1077335openalex-snapshot
The roles of fungus in CNS autoimmune and neurodegeneration disorders
inferred← citeshttps://doi.org/10.3389/fimmu.2023.1305933openalex-snapshot
Immunological dimensions of neuroinflammation and microglial activation: exploring innovative immunomodulatory approaches to mitigate neuroinflammatory progression
inferred← citeshttps://doi.org/10.3389/fimmu.2025.1615001openalex-snapshot
Protein regulatory network mediated by palmitoylation modifications in the pathological progression of Parkinson’s disease: a narrative review
inferred← citeshttps://doi.org/10.3389/fnagi.2022.1018848openalex-snapshot
Interaction between autophagy and the NLRP3 inflammasome in Alzheimer’s and Parkinson’s disease
inferred← citeshttps://doi.org/10.3389/fnagi.2022.1039780openalex-snapshot
The role of microglial autophagy in Parkinson’s disease
inferred← citeshttps://doi.org/10.3389/fnagi.2022.1065183openalex-snapshot
Microglial autophagy in Alzheimer’s disease and Parkinson’s disease
inferred← citeshttps://doi.org/10.3389/fnagi.2022.919343openalex-snapshot
Hypoxia and Alpha-Synuclein: Inextricable Link Underlying the Pathologic Progression of Parkinson's Disease
inferred← citeshttps://doi.org/10.3389/fnagi.2024.1411104openalex-snapshot
Emerging role of microglia in inter-cellular transmission of α-synuclein in Parkinson’s disease
inferred← citeshttps://doi.org/10.3389/fncel.2022.903469openalex-snapshot
The Mechanism and Function of Glia in Parkinson's Disease
inferred← citeshttps://doi.org/10.3389/fnins.2024.1506358openalex-snapshot
Microglia: roles and genetic risk in Parkinson’s disease
inferred← citeshttps://doi.org/10.3389/fnmol.2026.1742864openalex-snapshot
Mesenchymal stem cells and the central nervous system: historical perspectives and future directions
inferred← citeshttps://doi.org/10.3389/fnut.2024.1496616openalex-snapshot
Microbiome-based therapies for Parkinson’s disease
inferred← citeshttps://doi.org/10.3389/fphar.2024.1408152openalex-snapshot
The role of autophagy in Parkinson’s disease: a gender difference overview
inferred← citeshttps://doi.org/10.3390/biom13101435openalex-snapshot
Autophagy in Parkinson’s Disease
inferred← citeshttps://doi.org/10.3390/biomedicines10102649openalex-snapshot
Cellular Models of Alpha-Synuclein Aggregation: What Have We Learned and Implications for Future Study
inferred← citeshttps://doi.org/10.3390/biomedicines12051074openalex-snapshot
Insights into Calpain Activation and Rho-ROCK Signaling in Parkinson’s Disease and Aging
inferred← citeshttps://doi.org/10.3390/brainsci15121260openalex-snapshot
Alpha-Synuclein Neurobiology in Parkinson’s Disease: A Comprehensive Review of Its Role, Mechanisms, and Therapeutic Perspectives
inferred← citeshttps://doi.org/10.3390/cells11182908openalex-snapshot
Neuroinflammation and Parkinson’s Disease—From Neurodegeneration to Therapeutic Opportunities
inferred← citeshttps://doi.org/10.3390/cells12040621openalex-snapshot
Pathogenic Aspects and Therapeutic Avenues of Autophagy in Parkinson’s Disease
inferred← citeshttps://doi.org/10.3390/cimb47050344openalex-snapshot
Balancing Microglial Density and Activation in Central Nervous System Development and Disease
inferred← citeshttps://doi.org/10.3390/ijms24108593openalex-snapshot
Cellular Protein Aggregates: Formation, Biological Effects, and Ways of Elimination
inferred← citeshttps://doi.org/10.3390/ijms25020986openalex-snapshot
From Synaptic Physiology to Synaptic Pathology: The Enigma of α-Synuclein
inferred← citeshttps://doi.org/10.3390/ijms25084330openalex-snapshot
Experimental Models to Study Immune Dysfunction in the Pathogenesis of Parkinson’s Disease
inferred← citeshttps://doi.org/10.3390/ijms26041615openalex-snapshot
α-Synuclein Degradation in Brain Pericytes Is Mediated via Akt, ERK, and p38 MAPK Signaling Pathways
inferred← citeshttps://doi.org/10.3390/nu14194108openalex-snapshot
Dietary Restriction against Parkinson’s Disease: What We Know So Far
inferred← citeshttps://doi.org/10.3892/ijmm.2023.5253openalex-snapshot
Autophagy‑regulating miRNAs: Novel therapeutic targets for Parkinson's disease (Review)
inferred← citeshttps://doi.org/10.4018/979-8-3693-5908-2.ch001openalex-snapshot
Autophagy and Its Role in Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.4103/1673-5374.391334openalex-snapshot
Endoplasmic reticulum stress, autophagy, neuroinflammation, and sigma 1 receptors as contributors to depression and its treatment
inferred← citeshttps://doi.org/10.4103/regenmed.regenmed-d-24-00005openalex-snapshot
Role of non-neuronal cells in neurorepair: a focus on proteinopathy and neurodegeneration
inferred← citeshttps://doi.org/10.52711/0974-360x.2024.00791openalex-snapshot
Molecular Mechanisms Involved in Pathogenesis of Parkinson's Disease
inferred← citeshttps://doi.org/10.7150/thno.83082openalex-snapshot
Deletion of Calhm2 alleviates MPTP-induced Parkinson's disease pathology by inhibiting EFHD2-STAT3 signaling in microglia
inferred← citeshttps://doi.org/10.7717/peerj.21361openalex-snapshot
The role of YKL-40 in Alzheimer’s disease pathology and drug targeting
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