On the public citation graph

Inhibiting Nrf2 Expression Exacerbates Nlrp3 Inflammasome Activation and Dopamine Neuron Degeneration in Parkinson's Disease Models

https://doi.org/10.2139/ssrn.4743925
0 signed · 50 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.
Claim with ORCID
Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.
See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1007/s00018-016-2223-0openalex-extracted
Role of Nrf2/HO-1 system in development, oxidative stress response and diseases: an evolutionarily conserved mechanism
inferredcites →https://doi.org/10.1007/s00109-021-02071-5openalex-extracted
Nrf2 as a potential target for Parkinson’s disease therapy
inferredcites →https://doi.org/10.1007/s00253-020-10614-yopenalex-extracted
Crosstalk between ER stress, NLRP3 inflammasome, and inflammation
inferredcites →https://doi.org/10.1007/s00281-022-00947-3openalex-extracted
The immunology of Parkinson’s disease
inferredcites →https://doi.org/10.1007/s00702-016-1563-0openalex-extracted
Nrf2: a modulator of Parkinson’s disease?
inferredcites →https://doi.org/10.1007/s12035-020-02198-5openalex-extracted
NLRP3 Inflammasome Inhibition Prevents α-Synuclein Pathology by Relieving Autophagy Dysfunction in Chronic MPTP–Treated NLRP3 Knockout Mice
inferredcites →https://doi.org/10.1007/s12264-017-0159-5openalex-extracted
Premotor Diagnosis of Parkinson’s Disease
inferredcites →https://doi.org/10.1007/s12264-023-01023-yopenalex-extracted
NLRP3 Inflammasome-Mediated Neuroinflammation and Related Mitochondrial Impairment in Parkinson’s Disease
inferredcites →https://doi.org/10.1016/j.arr.2022.101756openalex-extracted
Role of Nrf2 in aging, Alzheimer’s and other neurodegenerative diseases
inferredcites →https://doi.org/10.1016/j.intimp.2018.12.019openalex-extracted
The mechanisms of NLRP3 inflammasome/pyroptosis activation and their role in Parkinson's disease
inferredcites →https://doi.org/10.1016/j.lfs.2019.03.057openalex-extracted
Parkinson's disease: Mechanisms, translational models and management strategies
inferredcites →https://doi.org/10.1016/j.mad.2021.111512openalex-extracted
The old guard: Age-related changes in microglia and their consequences
inferredcites →https://doi.org/10.1016/j.nbd.2019.104700openalex-extracted
Autonomic dysfunction in Parkinson’s disease: Implications for pathophysiology, diagnosis, and treatment
inferredcites →https://doi.org/10.1016/j.nbd.2020.105028openalex-extracted
Microglia and astrocyte dysfunction in parkinson's disease
inferredcites →https://doi.org/10.1016/j.neuron.2022.05.009openalex-extracted
Neuronal NLRP3 is a parkin substrate that drives neurodegeneration in Parkinson’s disease
inferredcites →https://doi.org/10.1016/j.neuroscience.2020.08.004openalex-extracted
NLRP3 Inflammasomes in Parkinson’s disease and their Regulation by Parkin
inferredcites →https://doi.org/10.1016/j.pbb.2004.03.006openalex-extracted
Biochemical, behavioral and immunohistochemical alterations in MPTP-treated mouse model of Parkinson's disease
inferredcites →https://doi.org/10.1016/j.redox.2020.101696openalex-extracted
ROS systems are a new integrated network for sensing homeostasis and alarming stresses in organelle metabolic processes
inferredcites →https://doi.org/10.1016/s0140-6736(21)00218-xopenalex-extracted
Parkinson's disease
inferredcites →https://doi.org/10.1016/s0166-4328(01)00309-6openalex-extracted
Behavioral phenotyping of the MPTP mouse model of Parkinson's disease
inferredcites →https://doi.org/10.1016/s1474-4422(21)00030-2openalex-extracted
Challenges in the diagnosis of Parkinson's disease
inferredcites →https://doi.org/10.1016/s1474-4422(21)00061-2openalex-extracted
Progress towards therapies for disease modification in Parkinson's disease
inferredcites →https://doi.org/10.1038/nature09663openalex-extracted
A role for mitochondria in NLRP3 inflammasome activation
inferredcites →https://doi.org/10.1038/nprot.2006.342openalex-extracted
Protocol for the MPTP mouse model of Parkinson's disease
inferredcites →https://doi.org/10.1038/s41418-018-0124-5openalex-extracted
MPTP-driven NLRP3 inflammasome activation in microglia plays a central role in dopaminergic neurodegeneration
inferredcites →https://doi.org/10.1038/s41531-020-00147-6openalex-extracted
Plasma-borne indicators of inflammasome activity in Parkinson’s disease patients
inferredcites →https://doi.org/10.1038/s41577-019-0165-0openalex-extracted
The NLRP3 inflammasome: molecular activation and regulation to therapeutics
inferredcites →https://doi.org/10.1038/s41582-020-00435-yopenalex-extracted
Neuroinflammation and microglial activation in Alzheimer disease: where do we go from here?
inferredcites →https://doi.org/10.1080/00207454.2021.1922402openalex-extracted
NLRP3 inflammasome activation increases brain oxidative stress after transient global cerebral ischemia in rats
inferredcites →https://doi.org/10.1089/ars.2020.8065openalex-extracted
Heme-Oxygenase-1
inferredcites →https://doi.org/10.1093/brain/awab446openalex-extracted
Single-cell sequencing of human midbrain reveals glial activation and a Parkinson-specific neuronal state
inferredcites →https://doi.org/10.1111/acel.13522openalex-extracted
α‐synuclein suppresses microglial autophagy and promotes neurodegeneration in a mouse model of Parkinson’s disease
inferredcites →https://doi.org/10.1111/jnc.13691openalex-extracted
The clinical symptoms of Parkinson's disease
inferredcites →https://doi.org/10.1146/annurev-immunol-081022-021207openalex-extracted
Structural Mechanisms of NLRP3 Inflammasome Assembly and Activation
inferredcites →https://doi.org/10.1152/physrev.00026.2013openalex-extracted
Mitochondrial Reactive Oxygen Species (ROS) and ROS-Induced ROS Release
inferredcites →https://doi.org/10.1155/2016/2183026openalex-extracted
ROS‐Mediated NLRP3 Inflammasome Activation in Brain, Heart, Kidney, and Testis Ischemia/Reperfusion Injury
inferredcites →https://doi.org/10.1186/s13041-017-0340-9openalex-extracted
Oxidative stress and cellular pathologies in Parkinson’s disease
inferredcites →https://doi.org/10.1186/s40478-021-01209-3openalex-extracted
α-Synuclein pathology in Parkinson disease activates homeostatic NRF2 anti-oxidant response
inferredcites →https://doi.org/10.2147/cia.s158513openalex-extracted
Oxidative stress, aging, and diseases
inferredcites →https://doi.org/10.3233/jpd-223237openalex-extracted
Microglia in Parkinson’s Disease
inferredcites →https://doi.org/10.3389/fphar.2022.919233openalex-extracted
Role of Nrf2 in Parkinson’s Disease: Toward New Perspectives
inferredcites →https://doi.org/10.3390/antiox11010026openalex-extracted
NRF2 Activation Ameliorates Oxidative Stress and Improves Mitochondrial Function and Synaptic Plasticity, and in A53T α-Synuclein Hippocampal Neurons
inferredcites →https://doi.org/10.3390/antiox12030731openalex-extracted
Neuroprotection of NRF2 against Ferroptosis after Traumatic Brain Injury in Mice
inferredcites →https://doi.org/10.3390/antiox9070630openalex-extracted
Antioxidant and Anti-inflammatory Effect of Nrf2 Inducer Dimethyl Fumarate in Neurodegenerative Diseases
inferredcites →https://doi.org/10.3390/cells10030515openalex-extracted
Heme Oxgenase-1, a Cardinal Modulator of Regulated Cell Death and Inflammation
inferredcites →https://doi.org/10.3390/ijms20133328openalex-extracted
The NLRP3 Inflammasome: An Overview of Mechanisms of Activation and Regulation
inferredcites →https://doi.org/10.3390/ijms21134777openalex-extracted
NRF2, a Transcription Factor for Stress Response and Beyond
inferredcites →https://doi.org/10.3390/ijms21197152openalex-extracted
Neuroprotective Effect of Antioxidants in the Brain
inferredcites →https://doi.org/10.3390/ijms23115938openalex-extracted
An Overview of Oxidative Stress, Neuroinflammation, and Neurodegenerative Diseases
inferredcites →https://doi.org/10.3390/ijms23137273openalex-extracted
Oxidative Stress in Ageing and Chronic Degenerative Pathologies: Molecular Mechanisms Involved in Counteracting Oxidative Stress and Chronic Inflammation
Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.