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Proteinuria and Hyperglycemia Induce Endoplasmic Reticulum Stress

https://doi.org/10.1681/asn.2007121313

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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A tool coming of age: thapsigargin as an inhibitor of sarco-endoplasmic reticulum Ca2+-ATPases

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Signal integration in the endoplasmic reticulum unfolded protein response

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Mediators of endoplasmic reticulum stress‐induced apoptosis

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Renal albumin absorption in physiology and pathology

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Albumin induces endoplasmic reticulum stress and apoptosis in renal proximal tubular cells

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The tubulointerstitium in progressive diabetic kidney disease: More than an aftermath of glomerular injury?

inferredcites →https://doi.org/10.1046/j.1523-1755.2002.00113.xopenalex-snapshot

Quantitative gene expression analysis in renal biopsies: A novel protocol for a high-throughput multicenter application

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Renal damage associated with proteinuria

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Diabetes and the Kidney

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Complement C5b-9 Membrane Attack Complex Increases Expression of Endoplasmic Reticulum Stress Proteins in Glomerular Epithelial Cells

inferredcites →https://doi.org/10.1074/jbc.m411860200openalex-snapshot

Involvement of Endoplasmic Reticulum Stress in Insulin Resistance and Diabetes

inferredcites →https://doi.org/10.1074/jbc.m500729200openalex-snapshot

Role of the Endoplasmic Reticulum Unfolded Protein Response in Glomerular Epithelial Cell Injury

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Oligosaccharide-based Information in Endoplasmic Reticulum Quality Control and Other Biological Systems

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Endoplasmic Reticulum Stress and Oxidative Stress: A Vicious Cycle or a Double-Edged Sword?

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Paracetamol-Induced Renal Tubular Injury

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CHOP induces death by promoting protein synthesis and oxidation in the stressed endoplasmic reticulum

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The endoplasmic reticulum stress response is stimulated through the continuous activation of transcription factors ATF6 and XBP1 in Ins2+/Akita pancreatic β cells

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THE MAMMALIAN UNFOLDED PROTEIN RESPONSE

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Tubulointerstitium as Predictor of Progression of Glomerular Diseases

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Diabetic Nephropathy: The Proteinuria Hypothesis

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Stress Proteins in Glomerular Epithelial Cell Injury

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Targeted disruption of the Chop gene delays endoplasmic reticulum stress–mediated diabetes

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Endoplasmic reticulum stress: cell life and death decisions

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Impact of Endoplasmic Reticulum Stress Pathway on Pancreatic β-Cells and Diabetes Mellitus

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The unfolded protein response

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Activation Signal of Nuclear Factor-.KAPPA.B in Response to Endoplasmic Reticulum Stress is Transduced via IRE1 and Tumor Necrosis Factor Receptor-Associated Factor 2

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Adaptation to ER Stress Is Mediated by Differential Stabilities of Pro-Survival and Pro-Apoptotic mRNAs and Proteins

inferredcites →https://doi.org/10.1677/joe-06-0148openalex-snapshot

Protection of pancreatic β-cells by exendin-4 may involve the reduction of endoplasmic reticulum stress; in vivo and in vitro studies

inferredcites →https://doi.org/10.1681/asn.2006040377openalex-snapshot

How Does Proteinuria Cause Progressive Renal Damage?

inferredcites →https://doi.org/10.1681/asn.2008090959openalex-snapshot

Is the ER Stressed out in Diabetic Kidney Disease?

inferredcites →https://doi.org/10.2169/internalmedicine.43.9openalex-snapshot

Mechanisms of Tubulointerstitial Injury in the Kidney: Final Common Pathways to End-stage Renal Failure

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Modular Activation of Nuclear Factor-κB Transcriptional Programs in Human Diabetic Nephropathy

inferredcites →https://doi.org/10.2337/diabetes.51.2007.s455openalex-snapshot

Endoplasmic Reticulum Stress and the Development of Diabetes

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Co-administration of “L-Lysine, Vitamin C, and Zinc” increased the antioxidant activity, decreased insulin resistance, and improved lipid profile in streptozotocin-induced diabetic rats

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Modulation of albumin-induced endoplasmic reticulum stress in renal proximal tubule cells by upregulation of mapk phosphatase-1

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Clioquinol induces autophagy by down-regulation of calreticulin in human neurotypic SH-SY5Y cells

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Ursodeoxycholic acid and 4-phenylbutyrate prevent endoplasmic reticulum stress-induced podocyte apoptosis in diabetic nephropathy

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Endoplasmic reticulum stress drives proteinuria-induced kidney lesions via Lipocalin 2

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An endoplasmic reticulum stress-regulated lncRNA hosting a microRNA megacluster induces early features of diabetic nephropathy

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Erythropoietin mitigated thioacetamide-induced renal injury via JAK2/STAT5 and AMPK pathway

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(+)-Catechin ameliorates diabetic nephropathy injury by inhibiting endoplasmic reticulum stress-related NLRP3-mediated inflammation

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Translational science in albuminuria: a new view ofde novoalbuminuria under chronic RAS suppression

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Sodium 4-phenylbutyrate treatment protects against renal injury in NZBWF1 mice

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Endoplasmic Reticulum Stress in UMOD-Related Kidney Disease: A Human Pathologic Study

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Plasma Ascorbic Acid Concentrations in Prevalent Patients With End-Stage Renal Disease on Hemodialysis

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Progressive renal injury from transgenic expression of human carbonic anhydrase IV folding mutants is enhanced by deficiency of p58 IPK

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Blocking CHOP-dependent TXNIP shuttling to mitochondria attenuates albuminuria and mitigates kidney injury in nephrotic syndrome

inferred← citeshttps://doi.org/10.1074/jbc.ra118.006939openalex-snapshot

Cell surface expression of 78-kDa glucose-regulated protein (GRP78) mediates diabetic nephropathy

inferred← citeshttps://doi.org/10.1080/0886022x.2024.2396446openalex-snapshot

Natural products derived from traditional Chinese medicines targeting ER stress for the treatment of kidney diseases

inferred← citeshttps://doi.org/10.1080/13813455.2020.1749084openalex-snapshot

MBTPS2 exacerbates albuminuria in streptozotocin-induced type I diabetic nephropathy by promoting endoplasmic reticulum stress-mediated renal damage

inferred← citeshttps://doi.org/10.1080/13813455.2020.1797106openalex-snapshot

Role of advanced glycation end products and insulin resistance in diabetic nephropathy

inferred← citeshttps://doi.org/10.1080/13813455.2022.2105365openalex-snapshot

Cyproheptadine, a SET7/9 inhibitor, reduces hyperglycaemia-induced ER stress alleviating inflammation and fibrosis in renal tubular epithelial cells

inferred← citeshttps://doi.org/10.1080/15548627.2025.2541385openalex-snapshot

GSTK1 and RETREG1/FAM134B-mediated reticulophagy attenuates tubular injury in diabetic nephropathy through endoplasmic reticulum stress and apoptosis

inferred← citeshttps://doi.org/10.1089/ars.2009.2705openalex-snapshot

Endoplasmic Reticulum Stress and Renal Disease

inferred← citeshttps://doi.org/10.1089/ars.2013.5807openalex-snapshot

ER Stress in Diabetic Peripheral Neuropathy: A New Therapeutic Target

inferred← citeshttps://doi.org/10.1089/jop.2010.0051openalex-snapshot

Response of Rat Retinal Capillary Pericytes and Endothelial Cells to Glucose

inferred← citeshttps://doi.org/10.1089/omi.2009.0073openalex-snapshot

Albumin Overload Induces Apoptosis in Renal Tubular Epithelial Cells through a CHOP-Dependent Pathway

inferred← citeshttps://doi.org/10.1093/advances/nmaa075openalex-snapshot

Resveratrol: Evidence for Its Nephroprotective Effect in Diabetic Nephropathy

inferred← citeshttps://doi.org/10.1093/cvr/cvad190openalex-snapshot

The role of endoplasmic reticulum–mitochondria-associated membranes in diabetic kidney disease

inferred← citeshttps://doi.org/10.1093/ndt/gfv079openalex-snapshot

RNA expression signatures and posttranscriptional regulation in diabetic nephropathy

inferred← citeshttps://doi.org/10.1096/fj.201900893ropenalex-snapshot

Klotho regulation by albuminuria is dependent on ATF3 and endoplasmic reticulum stress

inferred← citeshttps://doi.org/10.1096/fj.201903038ropenalex-snapshot

Sitagliptin ameliorates renal tubular injury in diabetic kidney disease via STAT3‐dependent mitochondrial homeostasis through SDF‐1α/CXCR4 pathway

inferred← citeshttps://doi.org/10.1096/fj.202000078rropenalex-snapshot

Suppressed ER‐associated degradation by intraglomerular cross talk between mesangial cells and podocytes causes podocyte injury in diabetic kidney disease

inferred← citeshttps://doi.org/10.1097/md.0000000000026879openalex-snapshot

Levels of circulating GRP78 and CHOP in endoplasmic reticulum stress pathways in Chinese type 2 diabetic kidney disease patients

inferred← citeshttps://doi.org/10.1101/cshperspect.a004317openalex-snapshot

Endoplasmic-Reticulum Calcium Depletion and Disease

inferred← citeshttps://doi.org/10.1111/1440-1681.13556openalex-snapshot

Renal lipotoxicity: Insights from experimental models

inferred← citeshttps://doi.org/10.1111/boc.201800019openalex-snapshot

Endoplasmic reticulum stress and kidney dysfunction

inferred← citeshttps://doi.org/10.1111/dom.13577openalex-snapshot

Sodium‐glucose cotransporter 2 inhibitors regulate ketone body metabolism via inter‐organ crosstalk

inferred← citeshttps://doi.org/10.1111/febs.14100openalex-snapshot

Podocyte‐specific soluble epoxide hydrolase deficiency in mice attenuates acute kidney injury

inferred← citeshttps://doi.org/10.1111/j.1582-4934.2009.00833.xopenalex-snapshot

Diabetes‐ and angiotensin II‐induced cardiac endoplasmic reticulum stress and cell death: metallothionein protection

inferred← citeshttps://doi.org/10.1152/ajprenal.00021.2011openalex-snapshot

The endoplasmic reticulum stress response and diabetic kidney disease

inferred← citeshttps://doi.org/10.1152/ajprenal.00113.2019openalex-snapshot

Bilateral ureteral obstruction is rapidly accompanied by ER stress and activation of autophagic degradation of IMCD proteins, including AQP2

inferred← citeshttps://doi.org/10.1152/ajprenal.00119.2016openalex-snapshot

Endoplasmic reticulum stress inhibition limits the progression of chronic kidney disease in the Dahl salt-sensitive rat

inferred← citeshttps://doi.org/10.1152/ajprenal.00196.2010openalex-snapshot

Regulation of podocyte survival and endoplasmic reticulum stress by fatty acids

inferred← citeshttps://doi.org/10.1152/ajprenal.00223.2015openalex-snapshot

Intrarenal renin-angiotensin system mediates fatty acid-induced ER stress in the kidney

inferred← citeshttps://doi.org/10.1152/ajprenal.00482.2011openalex-snapshot

ER stress contributes to renal proximal tubule injury by increasing SREBP-2-mediated lipid accumulation and apoptotic cell death

inferred← citeshttps://doi.org/10.1152/ajprenal.00485.2015openalex-snapshot

Knockdown of RTN1A attenuates ER stress and kidney injury in albumin overload-induced nephropathy

inferred← citeshttps://doi.org/10.1152/ajprenal.00697.2012openalex-snapshot

ER stress triggers MCP-1 expression through SET7/9-induced histone methylation in the kidneys of db/db mice

inferred← citeshttps://doi.org/10.1152/ajprenal.00710.2010openalex-snapshot

Albumin-induced epithelial-mesenchymal transition and ER stress are regulated through a common ROS-c-Src kinase-mTOR pathway: effect of imatinib mesylate

inferred← citeshttps://doi.org/10.1152/ajprenal.90652.2008openalex-snapshot

Effect of BSA-induced ER stress on SGLT protein expression levels and α-MG uptake in renal proximal tubule cells

inferred← citeshttps://doi.org/10.1152/ajprenal.90748.2008openalex-snapshot

Regulation of mRNA translation in renal physiology and disease

inferred← citeshttps://doi.org/10.1152/physrev.00045.2011openalex-snapshot

Mechanisms of Diabetic Complications

inferred← citeshttps://doi.org/10.1155/2012/628978openalex-snapshot

Autophagy as a Therapeutic Target in Diabetic Nephropathy

inferred← citeshttps://doi.org/10.1155/2014/315494openalex-snapshot

Role of Nutrient-Sensing Signals in the Pathogenesis of Diabetic Nephropathy

inferred← citeshttps://doi.org/10.1155/2016/7934504openalex-snapshot

A Systems Biology Overview on Human Diabetic Nephropathy: From Genetic Susceptibility to Post-Transcriptional and Post-Translational Modifications

inferred← citeshttps://doi.org/10.1155/2017/5903105openalex-snapshot

Renoprotective Effects of Aldose Reductase Inhibitor Epalrestat against High Glucose-Induced Cellular Injury

inferred← citeshttps://doi.org/10.1155/2018/6909035openalex-snapshot

Apoptosis-Associated Speck-Like Protein Containing a CARD Deletion Ameliorates Unilateral Ureteral Obstruction Induced Renal Fibrosis and Endoplasmic Reticulum Stress in Mice

inferred← citeshttps://doi.org/10.1155/2019/5383010openalex-snapshot

Roles of Identified Long Noncoding RNA in Diabetic Nephropathy

inferred← citeshttps://doi.org/10.1155/2020/6104375openalex-snapshot

AdipoRon Protects against Tubular Injury in Diabetic Nephropathy by Inhibiting Endoplasmic Reticulum Stress

inferred← citeshttps://doi.org/10.1155/2021/6614848openalex-snapshot

ROS-ERK Pathway as Dual Mediators of Cellular Injury and Autophagy-Associated Adaptive Response in Urinary Protein-Irritated Renal Tubular Epithelial Cells

inferred← citeshttps://doi.org/10.1155/2021/7277966openalex-snapshot

Endoplasmic Reticulum Stress in Diabetic Nephrology: Regulation, Pathological Role, and Therapeutic Potential

inferred← citeshttps://doi.org/10.1155/2023/8776878openalex-snapshot

Sestrin2 Signaling Pathway Regulates Podocyte Biology and Protects against Diabetic Nephropathy

inferred← citeshttps://doi.org/10.1155/bmri/5534800openalex-snapshot

The Role of Keratin in the Mechanisms of Kidney Diseases

inferred← citeshttps://doi.org/10.1155/jdr/5130183openalex-snapshot

Diosmetin Restores Endoplasmic Reticulum Homeostasis to Ameliorate Podocyte Injury in Diabetic Kidney Disease by Targeting DNAJC3

inferred← citeshttps://doi.org/10.1159/000210573openalex-snapshot

Endoplasmic Reticulum Stress in the Kidney as a Novel Mediator of Kidney Injury

inferred← citeshttps://doi.org/10.1159/000268954openalex-snapshot

Mechanisms and Control of Protein Translation in the Kidney

inferred← citeshttps://doi.org/10.1159/000362974openalex-snapshot

Down-Regulation of PERK-ATF4-CHOP Pathway by Astragaloside IV is Associated with the Inhibition of Endoplasmic Reticulum Stress-Induced Podocyte Apoptosis in Diabetic Rats

inferred← citeshttps://doi.org/10.1159/000443445openalex-snapshot

Urotensin II Induces ER Stress and EMT and Increase Extracellular Matrix Production in Renal Tubular Epithelial Cell in Early Diabetic Mice

inferred← citeshttps://doi.org/10.1161/circresaha.110.226803openalex-snapshot

Interrelationship Between Cardiac Hypertrophy, Heart Failure, and Chronic Kidney Disease

inferred← citeshttps://doi.org/10.1161/hypertensionaha.116.08560openalex-snapshot

Synergistic Interaction of Hypertension and Diabetes in Promoting Kidney Injury and the Role of Endoplasmic Reticulum Stress

inferred← citeshttps://doi.org/10.1166/jbn.2022.3242openalex-snapshot

Icariin Attenuation of Diabetic Kidney Disease Through Inhibition of Endoplasmic Reticulum Stress via G Protein-Coupled Estrogen Receptors

inferred← citeshttps://doi.org/10.1172/jci39492openalex-snapshot

Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging mice

inferred← citeshttps://doi.org/10.1172/jci44771openalex-snapshot

mTORC1 activation in podocytes is a critical step in the development of diabetic nephropathy in mice

inferred← citeshttps://doi.org/10.1177/1470320313507123openalex-snapshot

Protective effects of aliskiren and valsartan in mice with diabetic nephropathy

inferred← citeshttps://doi.org/10.1177/20420188211049615openalex-snapshot

Circulating microRNA-194 levels in Chinese patients with diabetic kidney disease: a case–control study

inferred← citeshttps://doi.org/10.1186/s10020-023-00732-4openalex-snapshot

Research progress on multiple cell death pathways of podocytes in diabetic kidney disease

inferred← citeshttps://doi.org/10.1186/s12882-015-0031-7openalex-snapshot

Astragaloside IV attenuates proteinuria in streptozotocin-induced diabetic nephropathy via the inhibition of endoplasmic reticulum stress

inferred← citeshttps://doi.org/10.1186/s12882-016-0249-zopenalex-snapshot

Bioinformatics analysis of proteomics profiles in senescent human primary proximal tubule epithelial cells

inferred← citeshttps://doi.org/10.1186/s12882-025-04364-0openalex-snapshot

Kidney transcriptomics signature of prospective rapid diabetic kidney disease progression

inferred← citeshttps://doi.org/10.1186/s12967-023-04069-8openalex-snapshot

Identification of transcription factors related to diabetic tubulointerstitial injury

inferred← citeshttps://doi.org/10.1186/s40885-015-0018-5openalex-snapshot

Angiotensin II induces endoplasmic reticulum stress in podocyte, which would be further augmented by PI3-kinase inhibition

inferred← citeshttps://doi.org/10.1210/clinem/dgaf462openalex-snapshot

Blood Plasma Proteome-Wide Association Study Implicates New Proteins in Type 2 Diabetes Mellitus Pathogenesis

inferred← citeshttps://doi.org/10.1242/dmm.048777openalex-snapshot

Loss of phosphatidylserine flippase β-subunit Tmem30a in podocytes leads to albuminuria and glomerulosclerosis

inferred← citeshttps://doi.org/10.12681/eadd/52426openalex-snapshot

Η επίδραση της ενεργοποίησης της κινάσης GCN2 σε ανθρώπινα σπειραματικά ενδοθηλιακά κύτταρα σε περιβάλλον φυσιολογικής και υψηλής γλυκόζης

inferred← citeshttps://doi.org/10.1369/0022155419851103openalex-snapshot

Thrombospondin 1 and Its Diverse Roles as a Regulator of Extracellular Matrix in Fibrotic Disease

inferred← citeshttps://doi.org/10.1371/journal.pone.0014207openalex-snapshot

Transcriptional Analysis Implicates Endoplasmic Reticulum Stress in Bovine Spongiform Encephalopathy

inferred← citeshttps://doi.org/10.1371/journal.pone.0019321openalex-snapshot

The Involvement of SMILE/TMTC3 in Endoplasmic Reticulum Stress Response

inferred← citeshttps://doi.org/10.1371/journal.pone.0020008openalex-snapshot

Study of Histopathological and Molecular Changes of Rat Kidney under Simulated Weightlessness and Resistance Training Protective Effect

inferred← citeshttps://doi.org/10.1371/journal.pone.0030071openalex-snapshot

HIV-1 Promotes Renal Tubular Epithelial Cell Protein Synthesis: Role of mTOR Pathway

inferred← citeshttps://doi.org/10.1371/journal.pone.0056124openalex-snapshot

Suppression of XBP1S Mediates High Glucose-Induced Oxidative Stress and Extracellular Matrix Synthesis in Renal Mesangial Cell and Kidney of Diabetic Rats

inferred← citeshttps://doi.org/10.1371/journal.pone.0092647openalex-snapshot

Pemt Deficiency Ameliorates Endoplasmic Reticulum Stress in Diabetic Nephropathy

inferred← citeshttps://doi.org/10.1371/journal.pone.0096987openalex-snapshot

Transcriptome-Based Analysis of Kidney Gene Expression Changes Associated with Diabetes in OVE26 Mice, in the Presence and Absence of Losartan Treatment

inferred← citeshttps://doi.org/10.1371/journal.pone.0252761openalex-snapshot

LncRNA TUG1/miR-29c-3p/SIRT1 axis regulates endoplasmic reticulum stress-mediated renal epithelial cells injury in diabetic nephropathy model in vitro

inferred← citeshttps://doi.org/10.1530/joe-13-0517openalex-snapshot

Stress in the kidney is the road to pERdition: is endoplasmic reticulum stress a pathogenic mediator of diabetic nephropathy?

inferred← citeshttps://doi.org/10.1530/joe-14-0437openalex-snapshot

Autophagy in diabetic nephropathy

inferred← citeshttps://doi.org/10.1681/asn.0000000000000357openalex-snapshot

Spatiotemporal Landscape of Kidney Tubular Responses to Glomerular Proteinuria

inferred← citeshttps://doi.org/10.1681/asn.2008090959openalex-snapshot

Is the ER Stressed out in Diabetic Kidney Disease?

inferred← citeshttps://doi.org/10.1681/asn.2008121233openalex-snapshot

Urinary Peptidome May Predict Renal Function Decline in Type 1 Diabetes and Microalbuminuria

inferred← citeshttps://doi.org/10.1681/asn.2009030242openalex-snapshot

Reactive Oxygen Species Promote Caspase-12 Expression and Tubular Apoptosis in Diabetic Nephropathy

inferred← citeshttps://doi.org/10.1681/asn.2009070760openalex-snapshot

PKC-δ Promotes Renal Tubular Cell Apoptosis Associated with Proteinuria

inferred← citeshttps://doi.org/10.1681/asn.2010040394openalex-snapshot

Caspase-12 and Diabetic Nephropathy

inferred← citeshttps://doi.org/10.1681/asn.2011040396openalex-snapshot

Increased Tubular Proliferation as an Adaptive Response to Glomerular Albuminuria

inferred← citeshttps://doi.org/10.1681/asn.2012121217openalex-snapshot

Evidence for Activation of the Unfolded Protein Response in Collagen IV Nephropathies

inferred← citeshttps://doi.org/10.1681/asn.2014100986openalex-snapshot

Mesencephalic Astrocyte–Derived Neurotrophic Factor as a Urine Biomarker for Endoplasmic Reticulum Stress–Related Kidney Diseases

inferred← citeshttps://doi.org/10.1681/asn.2015010032openalex-snapshot

Role of Ragulator in the Regulation of Mechanistic Target of Rapamycin Signaling in Podocytes and Glomerular Function

inferred← citeshttps://doi.org/10.1681/asn.2015020196openalex-snapshot

Angiogenin Mediates Cell-Autonomous Translational Control under Endoplasmic Reticulum Stress and Attenuates Kidney Injury

inferred← citeshttps://doi.org/10.1681/asn.2016091001openalex-snapshot

Inhibition of Reticulon-1A–Mediated Endoplasmic Reticulum Stress in Early AKI Attenuates Renal Fibrosis Development

inferred← citeshttps://doi.org/10.1681/asn.2019090966openalex-snapshot

Hypothesis as to How a Common Missense Mutation in COL4A3 May Confer Protection against Diabetic Kidney Disease

inferred← citeshttps://doi.org/10.1681/asn.2021040431openalex-snapshot

CHOP-ASO Ameliorates Glomerular and Tubular Damage on Top of ACE Inhibition in Diabetic Kidney Disease

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