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Modular Activation of Nuclear Factor-κB Transcriptional Programs in Human Diabetic Nephropathy
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Outgoing claims: the papers this work cites, builds on, or argues with.
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Relationship between circulating vascular cell adhesion molecule-1 and microvascular complications in Type 2 diabetes mellitus
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A novel method to develop highly specific models for regulatory units detects a new LTR in GenBank which contains a functional promoter
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Antisense RNA Amplification: A Linear Amplification Method for Analyzing the mRNA Population from Single Living Cells
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Potential role of angiotensin-converting enzyme inhibitors and statins on early podocyte damage in a model of type 2 diabetes mellitus, obesity, and mild hypertension
inferredcites →https://doi.org/10.1016/s0168-8227(99)00011-xopenalex-snapshot
Vascular cell adhesion molecule-l expression in the renal interstitium of diabetic KKAy mice
inferredcites →https://doi.org/10.1016/s0344-0338(11)80780-6openalex-snapshot
The Pathogenesis of Chronic Renal Failure in Diabetic Nephropathy
inferredcites →https://doi.org/10.1038/ni1159openalex-snapshot
Downregulation of lipopolysaccharide response in drosophila by negative crosstalk between the AP1 and NF-κB signaling modules
inferredcites →https://doi.org/10.1038/nri1703openalex-snapshot
NF-κB: linking inflammation and immunity to cancer development and progression
inferredcites →https://doi.org/10.1046/j.1432-1327.1999.00308.xopenalex-snapshot
NFκB activation is required for interferon regulatory factor‐1‐mediated interferon β induction
inferredcites →https://doi.org/10.1046/j.1523-1755.1999.00721.xopenalex-snapshot
The tubulointerstitium in progressive diabetic kidney disease: More than an aftermath of glomerular injury?
inferredcites →https://doi.org/10.1046/j.1523-1755.2002.00113.xopenalex-snapshot
Quantitative gene expression analysis in renal biopsies: A novel protocol for a high-throughput multicenter application
inferredcites →https://doi.org/10.1046/j.1523-1755.2003.00736.xopenalex-snapshot
Mycophenolate mofetil prevents the development of glomerular injury in experimental diabetes
inferredcites →https://doi.org/10.1053/j.ajkd.2004.08.039openalex-snapshot
Involvement of extracellular signal-regulated kinase and p38 in human diabetic nephropathy
inferredcites →https://doi.org/10.1073/pnas.0511257103openalex-snapshot
Comparative promoter analysis allows de novo identification of specialized cell junction-associated proteins
inferredcites →https://doi.org/10.1073/pnas.95.25.14863openalex-snapshot
Cluster analysis and display of genome-wide expression patterns
inferredcites →https://doi.org/10.1093/ndt/gfh207openalex-snapshot
NF- B activation and overexpression of regulated genes in human diabetic nephropathy
inferredcites →https://doi.org/10.1093/ndt/gfi155openalex-snapshot
Role of inflammation in diabetic complications
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Role for Nuclear Factor-κB and Signal Transducer and Activator of Transcription 1/Interferon Regulatory Factor-1 in Cytokine-Induced Endothelin-1 Release in Human Vascular Smooth Muscle Cells
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HLA Class I Heavy-Chain Gene Promoter Elements Mediating Synergy Between Tumor Necrosis Factor and Interferons
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Endothelial Interferon Regulatory Factor 1 Cooperates with NF-κB as a Transcriptional Activator of Vascular Cell Adhesion Molecule 1
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Transcriptional Regulation of Inducible Nitric Oxide Synthase in Cultured Neonatal Rat Cardiac Myocytes
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Cluster analysis and promoter modelling as bioinformatics tools for the identification of target genes from expression array data
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Methotrexate Prevents Renal Injury in Experimental Diabetic Rats via Anti-Inflammatory Actions
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Leukocyte Recruitment and Vascular Injury in Diabetic Nephropathy
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Role of the NF-kB Pathway in the Pathogenesis of Human Disease States
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RANTES Promoter Genotype Is Associated With Diabetic Nephropathy in Type 2 Diabetic Subjects
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The interferon-stimulated response element and a kappa B site mediate synergistic induction of murine IP-10 gene transcription by IFN- gamma and TNF- alpha .
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Regulation of RANTES Chemokine Gene Expression Requires Cooperativity Between NF-κB and IFN-Regulatory Factor Transcription Factors
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inferred← citeshttps://doi.org/10.1038/ki.2008.109openalex-snapshot
Failure to phosphorylate AKT in podocytes from mice with early diabetic nephropathy promotes cell death
inferred← citeshttps://doi.org/10.1038/ki.2009.434openalex-snapshot
Genetic analysis of albuminuria in aging mice and concordance with loci for human diabetic nephropathy found in a genome-wide association scan
inferred← citeshttps://doi.org/10.1038/ki.2009.98openalex-snapshot
Knockdown of Stat3 activity in vivo prevents diabetic glomerulopathy
inferred← citeshttps://doi.org/10.1038/ki.2010.270openalex-snapshot
New paradigms in cell death in human diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/ki.2010.428openalex-snapshot
Rho kinase inhibition protects kidneys from diabetic nephropathy without reducing blood pressure
inferred← citeshttps://doi.org/10.1038/ki.2011.116openalex-snapshot
Proteomic identification of vanin-1 as a marker of kidney damage in a rat model of type 1 diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/ki.2011.28openalex-snapshot
Hematopoietic growth factor inducible neurokinin-1 (Gpnmb/Osteoactivin) is a biomarker of progressive renal injury across species
inferred← citeshttps://doi.org/10.1038/ki.2011.314openalex-snapshot
Systems biology of kidney diseases
inferred← citeshttps://doi.org/10.1038/ki.2011.359openalex-snapshot
A systems view of genetics in chronic kidney disease
inferred← citeshttps://doi.org/10.1038/ki.2013.11openalex-snapshot
The TLR4 antagonist CRX-526 protects against advanced diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/ki.2014.116openalex-snapshot
Enhanced glomerular Toll-like receptor 4 expression and signaling in patients with type 2 diabetic nephropathy and microalbuminuria
inferred← citeshttps://doi.org/10.1038/labinvest.2008.158openalex-snapshot
Hypoxia response and VEGF-A expression in human proximal tubular epithelial cells in stable and progressive renal disease
inferred← citeshttps://doi.org/10.1038/labinvest.2014.163openalex-snapshot
Serum amyloid A and inflammation in diabetic kidney disease and podocytes
inferred← citeshttps://doi.org/10.1038/labinvest.3700599openalex-snapshot
Midkine is involved in tubulointerstitial inflammation associated with diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/nm.3902openalex-snapshot
Epithelial-to-mesenchymal transition induces cell cycle arrest and parenchymal damage in renal fibrosis
inferred← citeshttps://doi.org/10.1038/nrneph.2011.51openalex-snapshot
Inflammatory molecules and pathways in the pathogenesis of diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/s41392-026-02653-0openalex-snapshot
Hypoxia inducible factor network reflects kidney disease progression in diabetes and sodium-glucose co-transporters inhibition
inferred← citeshttps://doi.org/10.1038/s41419-020-03169-3openalex-snapshot
Exosomal circ_DLGAP4 promotes diabetic kidney disease progression by sponging miR-143 and targeting ERBB3/NF-κB/MMP-2 axis
inferred← citeshttps://doi.org/10.1038/s41419-023-06175-3openalex-snapshot
PACS-2 deficiency aggravates tubular injury in diabetic kidney disease by inhibiting ER-phagy
inferred← citeshttps://doi.org/10.1038/s41467-017-02094-yopenalex-snapshot
ORAI channels are critical for receptor-mediated endocytosis of albumin
inferred← citeshttps://doi.org/10.1038/s41467-019-12433-wopenalex-snapshot
Arctigenin attenuates diabetic kidney disease through the activation of PP2A in podocytes
inferred← citeshttps://doi.org/10.1038/s41467-020-15383-wopenalex-snapshot
The genetic architecture of membranous nephropathy and its potential to improve non-invasive diagnosis
inferred← citeshttps://doi.org/10.1038/s41467-022-34963-6openalex-snapshot
Epigenome-wide meta-analysis identifies DNA methylation biomarkers associated with diabetic kidney disease
inferred← citeshttps://doi.org/10.1038/s41598-017-15359-9openalex-snapshot
Urinary peptidomics analysis reveals proteases involved in diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/s41598-020-70540-xopenalex-snapshot
Identification of C3 as a therapeutic target for diabetic nephropathy by bioinformatics analysis
inferred← citeshttps://doi.org/10.1038/s41598-022-08547-9openalex-snapshot
Clr-f expression regulates kidney immune and metabolic homeostasis
inferred← citeshttps://doi.org/10.1038/s41598-025-01628-5openalex-snapshot
Integrating bioinformatics and machine learning to identify glomerular injury genes and predict drug targets in diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/s42003-025-08642-3openalex-snapshot
Multi-omics insights of immune cells in the risk and prognosis of idiopathic membranous nephropathy
inferred← citeshttps://doi.org/10.1038/srep33520openalex-snapshot
Role of C/EBP-α in Adriamycin-induced podocyte injury
inferred← citeshttps://doi.org/10.1038/srep41099openalex-snapshot
HIF-1α induction during reperfusion avoids maladaptive repair after renal ischemia/reperfusion involving miR127-3p
inferred← citeshttps://doi.org/10.1042/bj20110910openalex-snapshot
Connective tissue growth factor antagonizes transforming growth factor-β1/Smad signalling in renal mesangial cells
inferred← citeshttps://doi.org/10.1042/cs20130267openalex-snapshot
Role of Toll-like receptors in diabetic nephropathy
inferred← citeshttps://doi.org/10.1042/cs20160636openalex-snapshot
Diabetic nephropathy – is this an immune disorder?
inferred← citeshttps://doi.org/10.1042/cs20258557openalex-snapshot
Intrarenal renin–angiotensin system inactivation ameliorates tubulopathy via attenuation of mitochondrial oxidative stress, dysfunction and senescence in diabetes
inferred← citeshttps://doi.org/10.1053/j.ackd.2017.10.012openalex-snapshot
The Promise of Systems Biology for Diabetic Kidney Disease
inferred← citeshttps://doi.org/10.1053/j.ajkd.2013.10.047openalex-snapshot
New Insights Into Molecular Mechanisms of Diabetic Kidney Disease
inferred← citeshttps://doi.org/10.1055/a-1069-7290openalex-snapshot
Gene Regulatory Effect of Pyruvate Kinase M2 is Involved in Renal Inflammation in Type 2 Diabetic Nephropathy
inferred← citeshttps://doi.org/10.1073/pnas.2004651117openalex-snapshot
A role for NPY-NPY2R signaling in albuminuric kidney disease
inferred← citeshttps://doi.org/10.1074/jbc.m112.359505openalex-snapshot
Down-regulation of NF-κB Transcriptional Activity in HIV-associated Kidney Disease by BRD4 Inhibition
inferred← citeshttps://doi.org/10.1074/jbc.m116.753822openalex-snapshot
Absence of miR-146a in Podocytes Increases Risk of Diabetic Glomerulopathy via Up-regulation of ErbB4 and Notch-1
inferred← citeshttps://doi.org/10.1080/0886022x.2025.2526688openalex-snapshot
Analysis of potential targets of 20S-protopanaxadiol on diabetic nephropathy based on network pharmacology, molecular docking, and experimental validation
inferred← citeshttps://doi.org/10.1080/13543784.2018.1538352openalex-snapshot
Targeting inflammation in diabetic nephropathy: a tale of hope
inferred← citeshttps://doi.org/10.1080/17460441.2018.1398731openalex-snapshot
Novel avenues for drug discovery in diabetic kidney disease
inferred← citeshttps://doi.org/10.1089/ars.2008.2089openalex-snapshot
Molecular Mechanisms and Clinical Implications of Reversible Protein S -Glutathionylation
inferred← citeshttps://doi.org/10.1089/jmf.2014.3342openalex-snapshot
Gallic Acid Decreases Inflammatory Cytokine Secretion Through Histone Acetyltransferase/Histone Deacetylase Regulation in High Glucose-Induced Human Monocytes
inferred← citeshttps://doi.org/10.1089/omi.2011.0121openalex-snapshot
Molecular Pathways and Crosstalk Characterizing the Cardiorenal Syndrome
inferred← citeshttps://doi.org/10.1093/ckj/sfad233openalex-snapshot
Urine MMP7 as a kidney injury biomarker
inferred← citeshttps://doi.org/10.1093/hmg/ddae203openalex-snapshot
Insights into the molecular underpinning of type 2 diabetes complications
inferred← citeshttps://doi.org/10.1093/jimmun/vkag100openalex-snapshot
Longitudinal multiomic and spatial transcriptomic profiling of lupus nephritis progression in a murine model
inferred← citeshttps://doi.org/10.1093/nar/gkad393openalex-snapshot
Enrichr-KG: bridging enrichment analysis across multiple libraries
inferred← citeshttps://doi.org/10.1093/ndt/gfaa121openalex-snapshot
Novel insights into the disease transcriptome of human diabetic glomeruli and tubulointerstitium
inferred← citeshttps://doi.org/10.1093/ndt/gfn157openalex-snapshot
Pioglitazone attenuates diabetic nephropathy through an anti-inflammatory mechanism in type 2 diabetic rats
inferred← citeshttps://doi.org/10.1093/ndt/gfp730openalex-snapshot
Progressive glomerulosclerosis in type 2 diabetes is associated with renal histone H3K9 and H3K23 acetylation, H3K4 dimethylation and phosphorylation at serine 10
inferred← citeshttps://doi.org/10.1093/ndt/gfs489openalex-snapshot
Systems biology building a useful model from multiple markers and profiles
inferred← citeshttps://doi.org/10.1093/ndt/gfv079openalex-snapshot
RNA expression signatures and posttranscriptional regulation in diabetic nephropathy
inferred← citeshttps://doi.org/10.1093/ndt/gfv267openalex-snapshot
Molecular disease presentation in diabetic nephropathy
inferred← citeshttps://doi.org/10.1093/ndt/gfw443openalex-snapshot
Interstitial fibrosis scored on whole-slide digital imaging of kidney biopsies is a predictor of outcome in proteinuric glomerulopathies
inferred← citeshttps://doi.org/10.1093/ndt/gfz168openalex-snapshot
Urinary chemokine C-X-C motif ligand 16 and endostatin as predictors of tubulointerstitial fibrosis in patients with advanced diabetic kidney disease
inferred← citeshttps://doi.org/10.1096/fj.202101852ropenalex-snapshot
Mannose‐binding lectin activates the nuclear factor‐κB and renal inflammation in the progression of diabetic nephropathy
inferred← citeshttps://doi.org/10.1096/fj.202200740rropenalex-snapshot
Integrative transcriptome analysis reveals TEKT2 and PIAS2 involvement in diabetic nephropathy
inferred← citeshttps://doi.org/10.1097/md.0000000000034992openalex-snapshot
Bioinformatics led discovery of biomarkers related to immune infiltration in diabetes nephropathy
inferred← citeshttps://doi.org/10.1097/mnh.0b013e32835b54b0openalex-snapshot
IHG-1 amplifies TGF-β1 signalling and mitochondrial biogenesis and is increased in diabetic kidney disease
inferred← citeshttps://doi.org/10.1101/2021.09.07.459298openalex-snapshot
Micro-dissection and integration of long and short reads to create a robust catalog of kidney compartment-specific isoforms
inferred← citeshttps://doi.org/10.1101/2021.09.09.21262925openalex-snapshot
Multidimensional Data Integration Identifies Tumor Necrosis Factor Activation in Nephrotic Syndrome: A Model for Precision Nephrology
inferred← citeshttps://doi.org/10.1101/2022.02.14.480447openalex-snapshot
Açai ( Euterpe oleracea Mart ) modulates oxidative stress and inflammation by NF-κB inactivation and Nrf2 up-regulation in experimental diabetes
inferred← citeshttps://doi.org/10.1101/2024.06.22.600183openalex-snapshot
A Novel Role of Hyaluronan and its Membrane Receptors, CD44 and RHAMM in Obesity-Related Glomerulopathy
inferred← citeshttps://doi.org/10.1101/2024.07.09.24309717openalex-snapshot
Kidney ‘pathway-orphan’ genes as a potential overlooked source of novel insights into kidney health and disease
inferred← citeshttps://doi.org/10.1101/2024.11.28.24318055openalex-snapshot
Blood methylation biomarkers are associated with diabetic kidney disease progression in type 1 diabetes
inferred← citeshttps://doi.org/10.1101/2025.02.21.25322641openalex-snapshot
WITHDRAWN: Asengeprast, a Novel G-Protein Coupled Receptor 68 Antagonist, Reverses Clinically Relevant Inflammatory and Fibrotic Pathways in Chronic Kidney Disease
inferred← citeshttps://doi.org/10.1101/374983openalex-snapshot
Genome-wide Marginal Epistatic Association Mapping in Case-Control Studies
inferred← citeshttps://doi.org/10.1101/427880openalex-snapshot
Redefining Nephrotic Syndrome in Molecular Terms: Outcome-associated molecular clusters and patient stratification with noninvasive surrogate biomarkers
inferred← citeshttps://doi.org/10.1101/499616openalex-snapshot
Genome-wide association study of diabetic kidney disease highlights biology involved in renal basement membrane collagen
inferred← citeshttps://doi.org/10.1101/gr.155697.113openalex-snapshot
Defining cell-type specificity at the transcriptional level in human disease
inferred← citeshttps://doi.org/10.1111/1753-0407.12891openalex-snapshot
Effect of liraglutide on the Janus kinase/signal transducer and transcription activator (JAK/STAT) pathway in diabetic kidney disease in db/db mice and in cultured endothelial cells
inferred← citeshttps://doi.org/10.1111/dom.13416openalex-snapshot
An integrative systems biology approach for precision medicine in diabetic kidney disease
inferred← citeshttps://doi.org/10.1111/dom.14018openalex-snapshot
A metabolomics‐based molecular pathway analysis of how the sodium‐glucose co‐transporter‐2 inhibitor dapagliflozin may slow kidney function decline in patients with diabetes
inferred← citeshttps://doi.org/10.1111/jcmm.12953openalex-snapshot
MXRA 5 is a TGF ‐β1‐regulated human protein with anti‐inflammatory and anti‐fibrotic properties
inferred← citeshttps://doi.org/10.1111/jcmm.15250openalex-snapshot
Mesenchymal stem cells alleviate rat diabetic nephropathy by suppressing CD103+ DCs‐mediated CD8+ T cell responses
inferred← citeshttps://doi.org/10.1124/jpet.110.175828openalex-snapshot
Role of Intrarenal Angiotensin System Activation, Oxidative Stress, Inflammation, and Impaired Nuclear Factor-Erythroid-2-Related Factor 2 Activity in the Progression of Focal Glomerulosclerosis
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