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Systemic LPS-Induced RACK1 Downregulation Contributes to Neuronal Necroptosis in the Limbic System in Mice: A Mechanism of Sepsis-Associated Encephalopathy and a Potential Target of Tanshinone IIA

https://doi.org/10.2139/ssrn.4662337
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Long-term Cognitive Impairment and Functional Disability Among Survivors of Severe Sepsis
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The Protective Effects and the Involved Mechanisms of Tanshinone IIA on Sepsis-Induced Brain Damage in Mice
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Carbon Monoxide Modulation of Microglia-Neuron Communication: Anti-Neuroinflammatory and Neurotrophic Role
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The novel and potent anti-depressive action of triptolide and its influences on hippocampal neuroinflammation in a rat model of depression comorbidity of chronic pain
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Rh-IFN-α attenuates neuroinflammation and improves neurological function by inhibiting NF-κB through JAK1-STAT1/TRAF3 pathway in an experimental GMH rat model
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Caspase-1 inhibitor exerts brain-protective effects against sepsis-associated encephalopathy and cognitive impairments in a mouse model of sepsis
inferredcites →https://doi.org/10.1016/j.bbi.2021.02.033openalex-extracted
Tumor necrosis factor α negatively regulates the retrieval and reconsolidation of hippocampus-dependent memory
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Brief postpartum separation from offspring promotes resilience to lipopolysaccharide challenge-induced anxiety and depressive-like behaviors and inhibits neuroinflammation in C57BL/6J dams
inferredcites →https://doi.org/10.1016/j.biopha.2023.114286openalex-extracted
Pectolinarigenin alleviated septic acute kidney injury via inhibiting Jak2/Stat3 signaling and mitochondria dysfunction
inferredcites →https://doi.org/10.1016/j.celrep.2021.109639openalex-extracted
Rack1 is essential for corticogenesis by preventing p21-dependent senescence in neural stem cells
inferredcites →https://doi.org/10.1016/j.celrep.2022.111369openalex-extracted
Microglial control of neuronal development via somatic purinergic junctions
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2022.05.023openalex-extracted
Irisin protects against sepsis-associated encephalopathy by suppressing ferroptosis via activation of the Nrf2/GPX4 signal axis
inferredcites →https://doi.org/10.1016/j.immuni.2011.09.020openalex-extracted
RIP Kinase-Dependent Necrosis Drives Lethal Systemic Inflammatory Response Syndrome
inferredcites →https://doi.org/10.1016/j.intimp.2022.109608openalex-extracted
rmMANF prevents sepsis-associated lung injury via inhibiting endoplasmic reticulum stress-induced ferroptosis in mice
inferredcites →https://doi.org/10.1016/j.lfs.2016.02.014openalex-extracted
Deficit of RACK1 contributes to the spatial memory impairment via upregulating BECLIN1 to induce autophagy
inferredcites →https://doi.org/10.1016/j.neuint.2007.08.006openalex-extracted
TNF is a key mediator of septic encephalopathy acting through its receptor, TNF receptor-1
inferredcites →https://doi.org/10.1016/j.neuroscience.2022.04.011openalex-extracted
Protective Effects of Cannabinoid Type 2 Receptor Activation Against Microglia Overactivation and Neuronal Pyroptosis in Sepsis-Associated Encephalopathy
inferredcites →https://doi.org/10.1016/j.phrs.2014.01.002openalex-extracted
Corticosteroids modulate the expression of the PKC-anchoring protein RACK-1 and cytokine release in THP-1 cells
inferredcites →https://doi.org/10.1016/j.phrs.2021.105661openalex-extracted
Tanshinone IIA: A phytochemical as a promising drug candidate for neurodegenerative diseases
inferredcites →https://doi.org/10.1038/cddis.2017.204openalex-extracted
RACK1 depletion in the ribosome induces selective translation for non-canonical autophagy
inferredcites →https://doi.org/10.1038/cr.2013.162openalex-extracted
RACK1 modulates NF-κB activation by interfering with the interaction between TRAF2 and the IKK complex
inferredcites →https://doi.org/10.1038/nature14445openalex-extracted
Hippocampal–prefrontal input supports spatial encoding in working memory
inferredcites →https://doi.org/10.1038/ncb2883openalex-extracted
Plasma membrane translocation of trimerized MLKL protein is required for TNF-induced necroptosis
inferredcites →https://doi.org/10.1038/nchembio.83openalex-extracted
Identification of RIP1 kinase as a specific cellular target of necrostatins
inferredcites →https://doi.org/10.1038/ncomms14329openalex-extracted
RIP1 autophosphorylation is promoted by mitochondrial ROS and is essential for RIP3 recruitment into necrosome
inferredcites →https://doi.org/10.1038/nn.4608openalex-extracted
Necroptosis activation in Alzheimer's disease
inferredcites →https://doi.org/10.1038/nrd.2015.6openalex-extracted
Regulated necrosis: disease relevance and therapeutic opportunities
inferredcites →https://doi.org/10.1038/nrneurol.2012.183openalex-extracted
Sepsis-associated encephalopathy
inferredcites →https://doi.org/10.1038/s41418-018-0195-3openalex-extracted
Crashing the computer: apoptosis vs. necroptosis in neuroinflammation
inferredcites →https://doi.org/10.1038/s41422-019-0164-5openalex-extracted
The molecular machinery of regulated cell death
inferredcites →https://doi.org/10.1038/srep20183openalex-extracted
RACK1 promotes maintenance of morphine-associated memory via activation of an ERK-CREB dependent pathway in hippocampus
inferredcites →https://doi.org/10.1097/cm9.0000000000002010openalex-extracted
A new cell death program regulated by toll-like receptor 9 through p38 mitogen-activated protein kinase signaling pathway in a neonatal rat model with sepsis associated encephalopathy
inferredcites →https://doi.org/10.1111/bph.14922openalex-extracted
Morphine‐induced RACK1‐dependent autophagy in immortalized neuronal cell lines
inferredcites →https://doi.org/10.1111/cns.14013openalex-extracted
Hippocampus‐prefrontal cortex inputs modulate spatial learning and memory in a mouse model of sepsis induced by cecal ligation puncture
inferredcites →https://doi.org/10.1111/cns.14095openalex-extracted
PERK‐STING‐RIPK3 pathway facilitates cognitive impairment by inducing neuronal necroptosis in sepsis‐associated encephalopathy
inferredcites →https://doi.org/10.1111/jcmm.15061openalex-extracted
Dual role of RACK1 in airway epithelial mesenchymal transition and apoptosis
inferredcites →https://doi.org/10.1111/jnc.15491openalex-extracted
Systemic inflammation induced the delayed reduction of excitatory synapses in the CA3 during ageing
inferredcites →https://doi.org/10.1111/jphp.12650openalex-extracted
Tanshinone IIA attenuates Aβ25–35-induced spatial memory impairment via upregulating receptors for activated C kinase1 and inhibiting autophagy in hippocampus
inferredcites →https://doi.org/10.1111/jphp.13087openalex-extracted
Tanshinone IIA protects against lipopolysaccharide-induced lung injury through targeting Sirt1
inferredcites →https://doi.org/10.1172/jci.insight.99364openalex-extracted
Targeting inflammatory monocytes in sepsis-associated encephalopathy and long-term cognitive impairment
inferredcites →https://doi.org/10.1186/s12974-018-1235-0openalex-extracted
Necroptosis: a regulated inflammatory mode of cell death
inferredcites →https://doi.org/10.1186/s12974-022-02517-8openalex-extracted
FKBP51 mediates resilience to inflammation-induced anxiety through regulation of glutamic acid decarboxylase 65 expression in mouse hippocampus
inferredcites →https://doi.org/10.1186/s13054-017-1643-zopenalex-extracted
Neuroanatomy of sepsis-associated encephalopathy
inferredcites →https://doi.org/10.1371/journal.pgen.1009558openalex-extracted
RACK1 modulates polyglutamine-induced neurodegeneration by promoting ERK degradation in Drosophila
inferredcites →https://doi.org/10.21203/rs.3.rs-995338/v1openalex-extracted
inferredcites →https://doi.org/10.2139/ssrn.3753810openalex-extracted
inferredcites →https://doi.org/10.3389/fphar.2022.1002920openalex-extracted
Xenon attenuated neonatal lipopolysaccharide exposure induced neuronal necroptosis and subsequently improved cognition in juvenile rats
inferredcites →https://doi.org/10.3389/fphar.2022.884821openalex-extracted
Neuronal MD2 induces long-term mental impairments in septic mice by facilitating necroptosis and apoptosis
inferredcites →https://doi.org/10.3390/ijerph17165852openalex-extracted
Sepsis-Associated Brain Dysfunction: A Review of Current Literature
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