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The Role of the TGF-β Superfamily in Myocardial Infarction
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Overlapping expression domains of bone morphogenetic protein family members potentially account for limited tissue defects in BMP7 deficient embryos
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Myostatin, a transforming growth factor-? superfamily member, is expressed in heart muscle and is upregulated in cardiomyocytes after infarct
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Myostatin, a transforming growth factor‐β superfamily member, is expressed in heart muscle and is upregulated in cardiomyocytes after infarct
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Pathophysiology of Myocardial Infarction
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Bone morphogenetic protein 4 inhibits liposaccharide‐induced inflammation in the airway
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Development of thymic Foxp3+ regulatory T cells: TGF‐β matters
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TGF‐β signaling: A tale of two responses
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Bone Morphogenetic Protein-10 induces cardiomyocyte proliferation and improves cardiac function after myocardial infarction
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Bone Morphogenetic Protein‐2 Antagonizes Bone Morphogenetic Protein‐4 Induced Cardiomyocyte Hypertrophy and Apoptosis
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Bmp6 and Bmp7 Are Required for Cushion Formation and Septation in the Developing Mouse Heart
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Elevation of Expression of Smads 2, 3, and 4, Decorin and TGF-βin the Chronic Phase of Myocardial Infarct Scar Healing
inferredcites →https://doi.org/10.1006/jmcc.2001.1383openalex-snapshot
Changes in Extracellular Matrix and in Transforming Growth Factor Beta Isoforms After Coronary Artery Ligation in Rats
inferredcites →https://doi.org/10.1007/s00109-006-0090-0openalex-snapshot
Angiotensin II stimulates apoptosis via TGF-β1 signaling in ventricular cardiomyocytes of rat
inferredcites →https://doi.org/10.1007/s00395-008-0739-7openalex-snapshot
Transforming growth factor beta inhibition increases mortality and left ventricular dilatation after myocardial infarction
inferredcites →https://doi.org/10.1007/s00395-010-0108-1openalex-snapshot
TGF-β1 improves cardiac performance via up-regulation of laminin receptor 37/67 in adult ventricular cardiomyocytes
inferredcites →https://doi.org/10.1007/s00395-016-0593-yopenalex-snapshot
Targeted myocardial delivery of GDF11 gene rejuvenates the aged mouse heart and enhances myocardial regeneration after ischemia–reperfusion injury
inferredcites →https://doi.org/10.1007/s00395-019-0728-zopenalex-snapshot
Exogenous GDF11 attenuates non-canonical TGF-β signaling to protect the heart from acute myocardial ischemia–reperfusion injury
inferredcites →https://doi.org/10.1007/s00441-015-2139-2openalex-snapshot
Myostatin induces interstitial fibrosis in the heart via TAK1 and p38
inferredcites →https://doi.org/10.1007/s10753-017-0627-0openalex-snapshot
Endothelial BMP4 Regulates Leukocyte Diapedesis and Promotes Inflammation
inferredcites →https://doi.org/10.1007/s12265-012-9400-9openalex-snapshot
Follistatin-Like 3 Mediates Paracrine Fibroblast Activation by Cardiomyocytes
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Mechanisms of Fibroblast Activation in the Remodeling Myocardium
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In situ localization of transforming growth factor β1 in porcine heart: Enhanced expression after chronic coronary artery constriction
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The role of α-smooth muscle actin in fibroblast-mediated matrix contraction and remodeling
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Cardiac myostatin upregulation occurs immediately after myocardial ischemia and is involved in skeletal muscle activation of atrophy
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Matricellular proteins in the heart: possible role during stress and remodeling
inferredcites →https://doi.org/10.1016/j.cardiores.2004.07.017openalex-snapshot
Inhibition of TGF-? signaling exacerbates early cardiac dysfunction but prevents late remodeling after infarction
inferredcites →https://doi.org/10.1016/j.cardiores.2006.10.002openalex-snapshot
The role of TGF-β signaling in myocardial infarction and cardiac remodeling
inferredcites →https://doi.org/10.1016/j.cell.2013.04.015openalex-snapshot
Growth Differentiation Factor 11 Is a Circulating Factor that Reverses Age-Related Cardiac Hypertrophy
inferredcites →https://doi.org/10.1016/j.cell.2019.07.033openalex-snapshot
GDF15 Is an Inflammation-Induced Central Mediator of Tissue Tolerance
inferredcites →https://doi.org/10.1016/j.cyto.2014.06.017openalex-snapshot
Regulation and actions of activin A and follistatin in myocardial ischaemia–reperfusion injury
inferredcites →https://doi.org/10.1016/j.ejphar.2016.07.053openalex-snapshot
Activin A stimulates the proliferation and differentiation of cardiac fibroblasts via the ERK1/2 and p38-MAPK pathways
inferredcites →https://doi.org/10.1016/j.febslet.2012.02.043openalex-snapshot
Promiscuity and specificity in BMP receptor activation
inferredcites →https://doi.org/10.1016/j.febslet.2012.03.023openalex-snapshot
TGF‐β control of stem cell differentiation genes
inferredcites →https://doi.org/10.1016/j.hlc.2017.05.138openalex-snapshot
Absence of Myostatin Improves Cardiac Function Following Myocardial Infarction
inferredcites →https://doi.org/10.1016/j.jacbts.2018.10.003openalex-snapshot
Cardiomyocyte SMAD4-Dependent TGF-β Signaling is Essential to Maintain Adult Heart Homeostasis
inferredcites →https://doi.org/10.1016/j.jacbts.2019.02.006openalex-snapshot
Fibroblasts in the Infarcted, Remodeling, and Failing Heart
inferredcites →https://doi.org/10.1016/j.jacc.2006.07.060openalex-snapshot
The Role of Platelet-Derived Growth Factor Signaling in Healing Myocardial Infarcts
inferredcites →https://doi.org/10.1016/j.jacc.2015.03.520openalex-snapshot
Interacting Resident Epicardium-Derived Fibroblasts and Recruited Bone Marrow Cells Form Myocardial Infarction Scar
inferredcites →https://doi.org/10.1016/j.lfs.2013.12.030openalex-snapshot
Expression of bone morphogenetic protein 4 and its receptors in the remodeling heart
inferredcites →https://doi.org/10.1016/j.mce.2011.06.030openalex-snapshot
New insights into the mechanisms of activin action and inhibition
inferredcites →https://doi.org/10.1016/j.stem.2012.04.025openalex-snapshot
Small Molecule-Mediated TGF-β Type II Receptor Degradation Promotes Cardiomyogenesis in Embryonic Stem Cells
inferredcites →https://doi.org/10.1016/j.yjmcc.2004.01.004openalex-snapshot
Early degradation and serum appearance of type I collagen fragments after myocardial infarction
inferredcites →https://doi.org/10.1016/j.yjmcc.2010.01.010openalex-snapshot
Bone morphogenetic protein 4 mediates myocardial ischemic injury through JNK-dependent signaling pathway
inferredcites →https://doi.org/10.1016/j.yjmcc.2010.12.021openalex-snapshot
Molecular and cellular mechanisms involved in cardiac remodeling after acute myocardial infarction
inferredcites →https://doi.org/10.1016/j.yjmcc.2013.11.015openalex-snapshot
Fibroblasts in myocardial infarction: A role in inflammation and repair
inferredcites →https://doi.org/10.1016/j.yjmcc.2014.09.001openalex-snapshot
Targeting the transforming growth factor (TGF)-β cascade in the remodeling heart: Benefits and perils
inferredcites →https://doi.org/10.1016/j.yjmcc.2019.05.006openalex-snapshot
Distinct roles of myofibroblast-specific Smad2 and Smad3 signaling in repair and remodeling of the infarcted heart
inferredcites →https://doi.org/10.1016/s0002-9440(10)63598-5openalex-snapshot
Myofibroblast and Endothelial Cell Proliferation during Murine Myocardial Infarct Repair
inferredcites →https://doi.org/10.1016/s0008-6363(00)00158-9openalex-snapshot
Myofibroblasts in reperfused myocardial infarcts express the embryonic form of smooth muscle myosin heavy chain (SMemb)
inferredcites →https://doi.org/10.1016/s0014-5793(03)00507-6openalex-snapshot
BMP‐2 regulates cardiomyocyte contractility in a phosphatidylinositol 3 kinase‐dependent manner
inferredcites →https://doi.org/10.1016/s1074-7613(00)80170-3openalex-snapshot
Abrogation of TGFβ Signaling in T Cells Leads to Spontaneous T Cell Differentiation and Autoimmune Disease
inferredcites →https://doi.org/10.1016/s1359-6101(96)00048-2openalex-snapshot
Transforming growth factor-beta: Vasculogenesis, angiogenesis, and vessel wall integrity
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Activation of latent TGF-β by thrombospondin-1: mechanisms and physiology
inferredcites →https://doi.org/10.1021/bi00481a014openalex-snapshot
The pro domain of pre-pro-transforming growth factor .beta.1 when independently expressed is a functional binding protein for the mature growth factor
inferredcites →https://doi.org/10.1038/nature02006openalex-snapshot
Smad-dependent and Smad-independent pathways in TGF-β family signalling
inferredcites →https://doi.org/10.1038/ncomms12260openalex-snapshot
Genetic lineage tracing defines myofibroblast origin and function in the injured heart
inferredcites →https://doi.org/10.1038/ng.320openalex-snapshot
Lack of the bone morphogenetic protein BMP6 induces massive iron overload
inferredcites →https://doi.org/10.1038/nm.2354openalex-snapshot
GDF-15 is an inhibitor of leukocyte integrin activation required for survival after myocardial infarction in mice
inferredcites →https://doi.org/10.1038/nm1613openalex-snapshot
Endothelial-to-mesenchymal transition contributes to cardiac fibrosis
inferredcites →https://doi.org/10.1038/nrcardio.2014.28openalex-snapshot
The inflammatory response in myocardial injury, repair, and remodelling
inferredcites →https://doi.org/10.1038/nrcardio.2015.156openalex-snapshot
Targeting BMP signalling in cardiovascular disease and anaemia
inferredcites →https://doi.org/10.1038/s41569-018-0002-3openalex-snapshot
Cysteine protease cathepsins in cardiovascular disease: from basic research to clinical trials
inferredcites →https://doi.org/10.1073/pnas.84.16.5788openalex-snapshot
Transforming growth factor type beta induces monocyte chemotaxis and growth factor production.
inferredcites →https://doi.org/10.1073/pnas.90.2.770openalex-snapshot
Transforming growth factor beta 1 null mutation in mice causes excessive inflammatory response and early death.
inferredcites →https://doi.org/10.1073/pnas.90.3.1018openalex-snapshot
Mechanism of the cardioprotective effect of transforming growth factor beta 1 in feline myocardial ischemia and reperfusion.
inferredcites →https://doi.org/10.1074/jbc.274.52.37413openalex-snapshot
Interdependent SMAD and JNK Signaling in Transforming Growth Factor-β-mediated Transcription
inferredcites →https://doi.org/10.1074/jbc.m004536200openalex-snapshot
Transforming Growth Factor-β1 Inhibition of Macrophage Activation Is Mediated via Smad3
inferredcites →https://doi.org/10.1074/jbc.m101463200openalex-snapshot
Bone Morphogenetic Protein-2 Inhibits Serum Deprivation-induced Apoptosis of Neonatal Cardiac Myocytes through Activation of the Smad1 Pathway
inferredcites →https://doi.org/10.1074/jbc.m110.197079openalex-snapshot
Cardiac Myocyte-specific Ablation of Follistatin-like 3 Attenuates Stress-induced Myocardial Hypertrophy
inferredcites →https://doi.org/10.1074/jbc.m111.233577openalex-snapshot
Furin Is the Major Processing Enzyme of the Cardiac-specific Growth Factor Bone Morphogenetic Protein 10
inferredcites →https://doi.org/10.1074/jbc.m117.778506openalex-snapshot
Bone morphogenetic protein 9 (BMP9) and BMP10 enhance tumor necrosis factor-α-induced monocyte recruitment to the vascular endothelium mainly via activin receptor-like kinase 2
inferredcites →https://doi.org/10.1074/jbc.m204597200openalex-snapshot
Modulation of Smad2-mediated Signaling by Extracellular Signal-regulated Kinase
inferredcites →https://doi.org/10.1074/jbc.m802585200openalex-snapshot
Myostatin Directly Regulates Skeletal Muscle Fibrosis
inferredcites →https://doi.org/10.1080/00365513.2017.1318447openalex-snapshot
Connective tissue growth factor and bone morphogenetic protein 2 are induced following myocardial ischemia in mice and humans
inferredcites →https://doi.org/10.1083/jcb.105.6.2861openalex-snapshot
Role of transforming growth factor-beta in the development of the mouse embryo.
inferredcites →https://doi.org/10.1083/jcb.108.2.661openalex-snapshot
Expression of transforming growth factor-beta 1 in specific cells and tissues of adult and neonatal mice.
inferredcites →https://doi.org/10.1084/jem.173.5.1121openalex-snapshot
Transforming growth factor beta 1 (TGF-beta 1) induced neutrophil recruitment to synovial tissues: implications for TGF-beta-driven synovial inflammation and hyperplasia.
inferredcites →https://doi.org/10.1093/cvr/cvu053openalex-snapshot
Integrins αvβ5 and αvβ3 promote latent TGF-β1 activation by human cardiac fibroblast contraction
inferredcites →https://doi.org/10.1093/cvr/cvu229openalex-snapshot
Pharmacological inhibition of TGFβ receptor improves Nkx2.5 cardiomyoblast-mediated regeneration
inferredcites →https://doi.org/10.1093/cvr/cvv273openalex-snapshot
Myeloid cell-derived LRG attenuates adverse cardiac remodelling after myocardial infarction
inferredcites →https://doi.org/10.1093/cvr/cvw076openalex-snapshot
BMP-7 attenuates left ventricular remodelling under pressure overload and facilitates reverse remodelling and functional recovery
inferredcites →https://doi.org/10.1093/emboj/21.7.1743openalex-snapshot
Balancing the activation state of the endothelium via two distinct TGF‐β type I receptors
inferredcites →https://doi.org/10.1093/emboj/cdf366openalex-snapshot
TGF‐β receptor‐activated p38 MAP kinase mediates Smad‐independent TGF‐β responses
inferredcites →https://doi.org/10.1093/eurheartj/ehm465openalex-snapshot
Growth-differentiation factor-15 improves risk stratification in ST-segment elevation myocardial infarction
inferredcites →https://doi.org/10.1096/fj.00-0745fjeopenalex-snapshot
Induction and suppression of interferon‐inducible protein (IP)‐10 in reperfused myocardial infarcts may regulate angiogenesis
inferredcites →https://doi.org/10.1096/fj.02-0072comopenalex-snapshot
Stem cell differentiation requires a paracrine pathway in the heart
inferredcites →https://doi.org/10.1096/fj.201700193rropenalex-snapshot
Role of bone morphogenetic proteins in sprouting angiogenesis: differential BMP receptor‐dependent signaling pathways balance stalk vs . tip cell competence
inferredcites →https://doi.org/10.1096/fj.201701455rropenalex-snapshot
Cardiac fibroblast‐specific p38α MAP kinase promotes cardiac hypertrophy via a putative paracrine interleukin‐6 signaling mechanism
inferredcites →https://doi.org/10.1097/00005344-200112000-00015openalex-snapshot
Cardioprotective Effects of Transforming Growth Factor-β1 During Early Reoxygenation or Reperfusion Are Mediated by p42/p44 MAPK
inferredcites →https://doi.org/10.1097/fjc.0000000000000003openalex-snapshot
The Immune System and the Remodeling Infarcted Heart
inferredcites →https://doi.org/10.1097/shk.0b013e318289728aopenalex-snapshot
Treatment With Bone Morphogenetic Protein 2 Limits Infarct Size After Myocardial Infarction in Mice
inferredcites →https://doi.org/10.1101/cshperspect.a021907openalex-snapshot
Regulation of the Bioavailability of TGF-β and TGF-β-Related Proteins
inferredcites →https://doi.org/10.1101/cshperspect.a022053openalex-snapshot
Signaling Receptors for TGF-β Family Members
inferredcites →https://doi.org/10.1101/gad.1124803openalex-snapshot
An essential role of Bmp4 in the atrioventricular septation of the mouse heart
inferredcites →https://doi.org/10.1101/gad.13.7.804openalex-snapshot
A mechanism of repression of TGFbeta / Smad signaling by oncogenic Ras
inferredcites →https://doi.org/10.1111/1440-1681.12665openalex-snapshot
Bone morphogenetic protein 6—a possible new player in pathophysiology of heart failure
inferredcites →https://doi.org/10.1111/j.1582-4934.2011.01513.xopenalex-snapshot
Small molecule regulators of postnatal Nkx 2.5 cardiomyoblast proliferation and differentiation
inferredcites →https://doi.org/10.1126/science.2164258openalex-snapshot
Mediation of Cardioprotection by Transforming Growth Factor-β
inferredcites →https://doi.org/10.1128/mcb.19.10.7096openalex-snapshot
Bone Morphogenetic Proteins Induce Cardiomyocyte Differentiation through the Mitogen-Activated Protein Kinase Kinase Kinase TAK1 and Cardiac Transcription Factors Csx/Nkx-2.5 and GATA-4
inferredcites →https://doi.org/10.1146/annurev-immunol-032713-120257openalex-snapshot
TGF-β Activation and Function in Immunity
inferredcites →https://doi.org/10.1146/annurev.cellbio.21.022404.142018openalex-snapshot
SPECIFICITY AND VERSATILITY IN TGF-β SIGNALING THROUGH SMADS
inferredcites →https://doi.org/10.1146/annurev.immunol.16.1.137openalex-snapshot
REGULATION OF IMMUNE RESPONSES BY TGF-β
inferredcites →https://doi.org/10.1152/ajpheart.00186.2005openalex-snapshot
Activation of TGF-β1-TAK1-p38 MAPK pathway in spared cardiomyocytes is involved in left ventricular remodeling after myocardial infarction in rats
inferredcites →https://doi.org/10.1152/ajpheart.00311.2007openalex-snapshot
Interaction of BMP10 with Tcap may modulate the course of hypertensive cardiac hypertrophy
inferredcites →https://doi.org/10.1152/ajpheart.00328.2014openalex-snapshot
Regulatory T cells are recruited in the infarcted mouse myocardium and may modulate fibroblast phenotype and function
inferredcites →https://doi.org/10.1152/ajpheart.00367.2014openalex-snapshot
BMP-7 attenuates adverse cardiac remodeling mediated through M2 macrophages in prediabetic cardiomyopathy
inferredcites →https://doi.org/10.1152/ajpheart.01048.2009openalex-snapshot
Targeted inhibition of activin receptor-like kinase 5 signaling attenuates cardiac dysfunction following myocardial infarction
inferredcites →https://doi.org/10.1152/ajpheart.01288.2008openalex-snapshot
Bone morphogenetic protein-4 promotes induction of cardiomyocytes from human embryonic stem cells in serum-based embryoid body development
inferredcites →https://doi.org/10.1152/physrev.00002.2018openalex-snapshot
Activin A in Mammalian Physiology
inferredcites →https://doi.org/10.1152/physrev.00008.2011openalex-snapshot
Matricellular Proteins in Cardiac Adaptation and Disease
inferredcites →https://doi.org/10.1152/physrev.00012.2007openalex-snapshot
Myocardial Matrix Remodeling and the Matrix Metalloproteinases: Influence on Cardiac Form and Function
inferredcites →https://doi.org/10.1160/th07-01-0022openalex-snapshot
Chemokines in ischemia and reperfusion
inferredcites →https://doi.org/10.1161/01.cir.0000120704.97934.41openalex-snapshot
Elevated Levels of Activin A in Heart Failure
inferredcites →https://doi.org/10.1161/01.cir.0000165066.71481.8eopenalex-snapshot
Postinfarction Gene Therapy Against Transforming Growth Factor-β Signal Modulates Infarct Tissue Dynamics and Attenuates Left Ventricular Remodeling and Heart Failure
inferredcites →https://doi.org/10.1161/01.cir.0000167553.49133.81openalex-snapshot
Regeneration of Infarcted Myocardium by Intramyocardial Implantation of Ex Vivo Transforming Growth Factor-β–Preprogrammed Bone Marrow Stem Cells
inferredcites →https://doi.org/10.1161/01.cir.103.22.2745openalex-snapshot
Double-Outlet Right Ventricle and Overriding Tricuspid Valve Reflect Disturbances of Looping, Myocardialization, Endocardial Cushion Differentiation, and Apoptosis in TGF-β 2 –Knockout Mice
inferredcites →https://doi.org/10.1161/01.cir.56.5.786openalex-snapshot
The wavefront phenomenon of ischemic cell death. 1. Myocardial infarct size vs duration of coronary occlusion in dogs.
inferredcites →https://doi.org/10.1161/01.cir.95.3.684openalex-snapshot
Complement C5a, TGF-β1, and MCP-1, in Sequence, Induce Migration of Monocytes Into Ischemic Canine Myocardium Within the First One to Five Hours After Reperfusion
inferredcites →https://doi.org/10.1161/01.cir.95.3.693openalex-snapshot
Induction of Monocyte Chemoattractant Protein-1 in the Small Veins of the Ischemic and Reperfused Canine Myocardium
inferredcites →https://doi.org/10.1161/01.cir.98.7.687openalex-snapshot
Stem Cell Factor Induction Is Associated With Mast Cell Accumulation After Canine Myocardial Ischemia and Reperfusion
inferredcites →https://doi.org/10.1161/01.res.0000119170.70818.4fopenalex-snapshot
Essential Role for Smad3 in Regulating MCP-1 Expression and Vascular Inflammation
inferredcites →https://doi.org/10.1161/01.res.0000163017.13772.3aopenalex-snapshot
CCL2/Monocyte Chemoattractant Protein-1 Regulates Inflammatory Responses Critical to Healing Myocardial Infarcts
inferredcites →https://doi.org/10.1161/01.res.0000187457.24338.3dopenalex-snapshot
NAD(P)H Oxidase 4 Mediates Transforming Growth Factor-β1–Induced Differentiation of Cardiac Fibroblasts Into Myofibroblasts
inferredcites →https://doi.org/10.1161/01.res.0000202805.73038.48openalex-snapshot
The Transforming Growth Factor-β Superfamily Member Growth-Differentiation Factor-15 Protects the Heart From Ischemia/Reperfusion Injury
inferredcites →https://doi.org/10.1161/01.res.69.2.483openalex-snapshot
Differential effects of transforming growth factor-beta 1 and phorbol myristate acetate on cardiac fibroblasts. Regulation of fibrillar collagen mRNAs and expression of early transcription factors.
inferredcites →https://doi.org/10.1161/atvbaha.114.304652openalex-snapshot
Monocytes in Myocardial Infarction
inferredcites →https://doi.org/10.1161/circheartfailure.114.001963openalex-snapshot
Smad3 Signaling Promotes Fibrosis While Preserving Cardiac and Aortic Geometry in Obese Diabetic Mice
inferredcites →https://doi.org/10.1161/circresaha.108.178434openalex-snapshot
BMPER Is an Endothelial Cell Regulator and Controls Bone Morphogenetic Protein-4–Dependent Angiogenesis
inferredcites →https://doi.org/10.1161/circresaha.109.216101openalex-snapshot
Smad3 Signaling Critically Regulates Fibroblast Phenotype and Function in Healing Myocardial Infarction
inferredcites →https://doi.org/10.1161/circresaha.110.217737openalex-snapshot
Potential Therapeutic Targets for Cardiac Fibrosis
inferredcites →https://doi.org/10.1161/circresaha.111.243162openalex-snapshot
Regulation of the Inflammatory Response in Cardiac Repair
inferredcites →https://doi.org/10.1161/circresaha.111.248161openalex-snapshot
Exome Sequencing Identifies SMAD3 Mutations as a Cause of Familial Thoracic Aortic Aneurysm and Dissection With Intracranial and Other Arterial Aneurysms
inferredcites →https://doi.org/10.1161/circresaha.114.302653openalex-snapshot
Cardiomyocyte-Specific Transforming Growth Factor β Suppression Blocks Neutrophil Infiltration, Augments Multiple Cytoprotective Cascades, and Reduces Early Mortality After Myocardial Infarction
inferredcites →https://doi.org/10.1161/circresaha.115.303895openalex-snapshot
Foxp3 + CD4 + T Cells Improve Healing After Myocardial Infarction by Modulating Monocyte/Macrophage Differentiation
inferredcites →https://doi.org/10.1161/circresaha.115.304185openalex-snapshot
Myostatin Regulates Energy Homeostasis in the Heart and Prevents Heart Failure
inferredcites →https://doi.org/10.1161/circresaha.115.307527openalex-snapshot
GDF11 Does Not Rescue Aging-Related Pathological Hypertrophy
inferredcites →https://doi.org/10.1161/circresaha.115.307778openalex-snapshot
Revisiting Cardiac Cellular Composition
inferredcites →https://doi.org/10.1161/circresaha.116.308391openalex-snapshot
Biochemistry and Biology of GDF11 and Myostatin
inferredcites →https://doi.org/10.1161/circresaha.116.308700openalex-snapshot
BMP Antagonist Gremlin 2 Limits Inflammation After Myocardial Infarction
inferredcites →https://doi.org/10.1161/circresaha.118.312955openalex-snapshot
GDF11 Decreases Pressure Overload–Induced Hypertrophy, but Can Cause Severe Cachexia and Premature Death
inferredcites →https://doi.org/10.1161/circresaha.118.314438openalex-snapshot
Protective Effects of Activated Myofibroblasts in the Pressure-Overloaded Myocardium Are Mediated Through Smad-Dependent Activation of a Matrix-Preserving Program
inferredcites →https://doi.org/10.1161/circresaha.118.314569openalex-snapshot
Interleukin-35 Promotes Macrophage Survival and Improves Wound Healing After Myocardial Infarction in Mice
inferredcites →https://doi.org/10.1161/circresaha.119.315069openalex-snapshot
Macrophage Smad3 Protects the Infarcted Heart, Stimulating Phagocytosis and Regulating Inflammation
inferredcites →https://doi.org/10.1161/circulationaha.104.490946openalex-snapshot
Smad1 Protects Cardiomyocytes From Ischemia-Reperfusion Injury
inferredcites →https://doi.org/10.1161/circulationaha.104.510354openalex-snapshot
Critical Role of Endogenous Thrombospondin-1 in Preventing Expansion of Healing Myocardial Infarcts
inferredcites →https://doi.org/10.1161/circulationaha.106.650846openalex-snapshot
Prognostic Value of Growth-Differentiation Factor-15 in Patients With Non–ST-Elevation Acute Coronary Syndrome
inferredcites →https://doi.org/10.1161/circulationaha.107.697714openalex-snapshot
Growth Differentiation Factor 15 for Risk Stratification and Selection of an Invasive Treatment Strategy in Non–ST-Elevation Acute Coronary Syndrome
inferredcites →https://doi.org/10.1161/circulationaha.107.704197openalex-snapshot
Essential Role of Smad3 in Infarct Healing and in the Pathogenesis of Cardiac Remodeling
inferredcites →https://doi.org/10.1161/circulationaha.109.872200openalex-snapshot
Activin A and Follistatin-Like 3 Determine the Susceptibility of Heart to Ischemic Injury
inferredcites →https://doi.org/10.1161/circulationaha.111.044164openalex-snapshot
Activation of CD4 + T Lymphocytes Improves Wound Healing and Survival After Experimental Myocardial Infarction in Mice
inferredcites →https://doi.org/10.1161/circulationaha.112.115089openalex-snapshot
Therapeutic Impact of Follistatin-Like 1 on Myocardial Ischemic Injury in Preclinical Models
inferredcites →https://doi.org/10.1161/circulationaha.116.026238openalex-snapshot
Fibroblast-Specific Genetic Manipulation of p38 Mitogen-Activated Protein Kinase In Vivo Reveals Its Central Regulatory Role in Fibrosis
inferredcites →https://doi.org/10.1161/circulationaha.117.029622openalex-snapshot
Opposing Actions of Fibroblast and Cardiomyocyte Smad3 Signaling in the Infarcted Myocardium
inferredcites →https://doi.org/10.1161/hypertensionaha.111.00562openalex-snapshot
Bone Morphogenetic Protein-4 Mediates Cardiac Hypertrophy, Apoptosis, and Fibrosis in Experimentally Pathological Cardiac Hypertrophy
inferredcites →https://doi.org/10.1161/jaha.113.000269openalex-snapshot
SMAD3 Deficiency Promotes Inflammatory Aortic Aneurysms in Angiotensin II–Infused Mice Via Activation of iNOS
inferredcites →https://doi.org/10.1164/rccm.201201-0084ocopenalex-snapshot
Lactic Acid Is Elevated in Idiopathic Pulmonary Fibrosis and Induces Myofibroblast Differentiation via pH-Dependent Activation of Transforming Growth Factor-β
inferredcites →https://doi.org/10.1172/jci116087openalex-snapshot
Role of transforming growth factor-beta in maintenance of function of cultured neonatal cardiac myocytes. Autocrine action and reversal of damaging effects of interleukin-1.
inferredcites →https://doi.org/10.1172/jci63635openalex-snapshot
Myocardin regulates BMP10 expression and is required for heart development
inferredcites →https://doi.org/10.1172/jci85782openalex-snapshot
Alternatively activated macrophages determine repair of the infarcted adult murine heart
inferredcites →https://doi.org/10.1177/002215540205000108openalex-snapshot
Morphological Characteristics of the Microvasculature in Healing Myocardial Infarcts
inferredcites →https://doi.org/10.1177/1535370219828694openalex-snapshot
Bone morphogenetic protein signaling in inflammation
inferredcites →https://doi.org/10.1177/43.10.7560881openalex-snapshot
Expression pattern of osteogenic protein-1 (bone morphogenetic protein-7) in human and mouse development.
inferredcites →https://doi.org/10.1182/blood-2011-09-377648openalex-snapshot
Platelet TGF-β1 contributions to plasma TGF-β1, cardiac fibrosis, and systolic dysfunction in a mouse model of pressure overload
inferredcites →https://doi.org/10.1189/jlb.1103539openalex-snapshot
TGF-β: the perpetrator of immune suppression by regulatory T cells and suicidal T cells
inferredcites →https://doi.org/10.1210/en.2008-0090openalex-snapshot
Bone Morphogenetic Protein-2 Antagonizes Renal Interstitial Fibrosis by Promoting Catabolism of Type I Transforming Growth Factor-β Receptors
inferredcites →https://doi.org/10.1242/dev.01094openalex-snapshot
BMP10 is essential for maintaining cardiac growth during murine cardiogenesis
inferredcites →https://doi.org/10.1242/dev.02156openalex-snapshot
Bmp2 is essential for cardiac cushion epithelial-mesenchymal transition and myocardial patterning
inferredcites →https://doi.org/10.1242/dev.078543openalex-snapshot
The regenerative capacity of the zebrafish heart is dependent on TGFβ signaling
inferredcites →https://doi.org/10.1242/dev.122.10.2977openalex-snapshot
Mice deficient for BMP2 are nonviable and have defects in amnion/chorion and cardiac development
inferredcites →https://doi.org/10.1242/jcs.00229openalex-snapshot
Making sense of latent TGFβ activation
inferredcites →https://doi.org/10.1369/jhc.3a6210.2004openalex-snapshot
Vascular Mural Cells in Healing Canine Myocardial Infarcts
inferredcites →https://doi.org/10.1371/journal.pone.0110752openalex-snapshot
Functional Cardiomyocytes Derived from Isl1 Cardiac Progenitors via Bmp4 Stimulation
inferredcites →https://doi.org/10.15252/emmm.201707604openalex-snapshot
GDF15 is a heart‐derived hormone that regulates body growth
inferredcites →https://doi.org/10.1620/tjem.244.1openalex-snapshot
Exogenous BMP-7 Facilitates the Recovery of Cardiac Function after Acute Myocardial Infarction through Counteracting TGF-β1 Signaling Pathway
inferredcites →https://doi.org/10.1634/stemcells.2007-0708openalex-snapshot
Impact of Myocardial Infarct Proteins and Oscillating Pressure on the Differentiation of Mesenchymal Stem Cells: Effect of Acute Myocardial Infarction on Stem Cell Differentiation
inferredcites →https://doi.org/10.1677/joe-07-0408openalex-snapshot
Myostatin promotes a fibrotic phenotypic switch in multipotent C3H 10T1/2 cells without affecting their differentiation into myofibroblasts
inferredcites →https://doi.org/10.18632/oncotarget.10561openalex-snapshot
TGF-β induces M2-like macrophage polarization via SNAIL-mediated suppression of a pro-inflammatory phenotype
inferredcites →https://doi.org/10.21037/jtd.2016.11.19openalex-snapshot
The role of transforming growth factor (TGF)-β in the infarcted myocardium
inferredcites →https://doi.org/10.2353/ajpath.2007.060547openalex-snapshot
Macrophage Depletion Impairs Wound Healing and Increases Left Ventricular Remodeling after Myocardial Injury in Mice
inferredcites →https://doi.org/10.2353/ajpath.2009.090393openalex-snapshot
Integrin-Mediated Transforming Growth Factor-β Activation, a Potential Therapeutic Target in Fibrogenic Disorders
inferredcites →https://doi.org/10.2353/ajpath.2010.090759openalex-snapshot
CCR5 Signaling Suppresses Inflammation and Reduces Adverse Remodeling of the Infarcted Heart, Mediating Recruitment of Regulatory T Cells
inferredcites →https://doi.org/10.2741/3573openalex-snapshot
Transforming growth factor-beta signaling and tumor angiogenesis
inferredcites →https://doi.org/10.3109/03008209109152444openalex-snapshot
Effect of Growth Factors on Collagen Metabolism in Cultured Human Heart Fibroblasts
inferredcites →https://doi.org/10.3892/ijmm.2014.1782openalex-snapshot
Hepatocyte growth factor regulates the TGF-β1-induced proliferation, differentiation and secretory function of cardiac fibroblasts
inferredcites →https://doi.org/10.3892/mmr.2018.8496openalex-snapshot
Activin A inhibition attenuates sympathetic neural remodeling following myocardial infarction in rats
inferredcites →https://doi.org/10.4049/jimmunol.1301843openalex-snapshot
TGF-β: Guardian of T Cell Function
inferredcites →https://doi.org/10.4049/jimmunol.145.3.940openalex-snapshot
Macrophage deactivating factor and transforming growth factors- beta 1 - beta 2 and - beta 3 inhibit induction of macrophage nitrogen oxide synthesis by IFN-gamma.
inferredcites →https://doi.org/10.4049/jimmunol.146.6.1849openalex-snapshot
Differential susceptibility of activated macrophage cytotoxic effector reactions to the suppressive effects of transforming growth factor-beta 1
inferredcites →https://doi.org/10.4049/jimmunol.148.4.1102openalex-snapshot
Transforming growth factor-beta enhances the M-CSF and GM-CSF-stimulated proliferation of macrophages
inferredcites →https://doi.org/10.4049/jimmunol.156.8.2964openalex-snapshot
Transforming growth factor-β 1 is the predominant paracrine inhibitor of macrophage cytokine synthesis produced by glomerular mesangial cells
inferredcites →https://doi.org/10.4049/jimmunol.157.1.360openalex-snapshot
Transforming growth factor-beta 1 inhibits the production of IL-8 and the transmigration of neutrophils through activated endothelium
inferredcites →https://doi.org/10.4049/jimmunol.176.2.1209openalex-snapshot
Oxidants Selectively Reverse TGF-β Suppression of Proinflammatory Mediator Production
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Fibroblast‐mediated intercellular crosstalk in the healthy and diseased heart
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Lefty1 Ameliorates Post-infarction Fibrosis by Suppressing p-Smad2 and p-ERK1/2 Signaling Pathways
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Key Roles of RGD-Recognizing Integrins During Cardiac Development, on Cardiac Cells, and After Myocardial Infarction
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Downregulation of histone deacetylase 8 (HDAC8) alleviated the progression of oral submucous fibrosis
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Distinct phenotypes induced by acute hypoxia and TGF-β1 in human adult cardiac fibroblasts
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Growth factors and female reproduction in vertebrates
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The role of TGF-β signaling in muscle atrophy, sarcopenia and cancer cachexia
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Fisetin protects against cardiac cell death through reduction of ROS production and caspases activity
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Boron improves cardiac contractility and fibrotic remodeling following myocardial infarction injury
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Deficiency of myostatin protects skeletal muscle cells from ischemia reperfusion injury
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The systemic deletion of interleukin-1α reduces myocardial inflammation and attenuates ventricular remodeling in murine myocardial infarction
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The role of SMAD signaling in hypertrophic obstructive cardiomyopathy: an immunohistopathological study in pediatric and adult patients
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Bone morphogenic protein-4 availability in the cardiac microenvironment controls inflammation and fibrosis in autoimmune myocarditis
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Role of B lymphocytes in the infarcted mass in patients with acute myocardial infarction
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Colchicine as potential inhibitor targeting MMP-9, NOX2 and TGF-β1 in myocardial infarction: a combination of docking and molecular dynamic simulation study
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Useful applications of growth factors for cardiovascular regenerative medicine
inferred← citeshttps://doi.org/10.1080/15384101.2023.2293595openalex-snapshot
Transforming growth factor-β and bone morphogenetic protein signaling pathways in pathological cardiac hypertrophy
inferred← citeshttps://doi.org/10.1089/jir.2022.0226openalex-snapshot
Plasma Markers for Early Prediction of Radiation-Induced Myocardial Damage
inferred← citeshttps://doi.org/10.1089/ten.tec.2020.0298openalex-snapshot
Advanced In Vitro Modeling to Study the Paradox of Mechanically Induced Cardiac Fibrosis
inferred← citeshttps://doi.org/10.1089/wound.2018.0894openalex-snapshot
A Large Mammalian Model of Myocardial Regeneration After Myocardial Infarction in Fetal Sheep
inferred← citeshttps://doi.org/10.1091/mbc.e22-02-0032openalex-snapshot
Cardiac inducing colonies halt fibroblast activation and induce cardiac/endothelial cells to move and expand via paracrine signaling
inferred← citeshttps://doi.org/10.1093/cvr/cvab181openalex-snapshot
The healing myocardium mobilizes a distinct B-cell subset through a CXCL13-CXCR5-dependent mechanism
inferred← citeshttps://doi.org/10.1093/gerona/glad135openalex-snapshot
Relationships of GDF8 and 11 and Their Antagonists With Decline of Grip Strength Among Older Adults in the Baltimore Longitudinal Study of Aging
inferred← citeshttps://doi.org/10.1093/ndt/gfaa203openalex-snapshot
The role of activin: the other side of chronic kidney disease–mineral bone disorder?
inferred← citeshttps://doi.org/10.1097/cad.0000000000001066openalex-snapshot
Stachydrine inhibits TGF-β1-induced epithelial–mesenchymal transition in hepatocellular carcinoma cells through the TGF-β/Smad and PI3K/Akt/mTOR signaling pathways
inferred← citeshttps://doi.org/10.1097/fjc.0000000000000957openalex-snapshot
Leonurine Attenuates Myocardial Fibrosis Through Upregulation of miR-29a-3p in Mice Post-myocardial Infarction
inferred← citeshttps://doi.org/10.1097/md.0000000000041976openalex-snapshot
Comprehensive analysis of GDFs as therapeutic targets and prognosis biomarkers in gastric cancer
inferred← citeshttps://doi.org/10.1097/md.0000000000047588openalex-snapshot
Mendelian randomization followed by single-cell RNA sequencing exploration identifies effector memory CD4+ T cells as a risk factor for myasthenia gravis
inferred← citeshttps://doi.org/10.1101/2020.04.28.065789openalex-snapshot
Clearance of senescent cells following cardiac ischemia-reperfusion injury improves recovery
inferred← citeshttps://doi.org/10.1101/2024.08.27.609034openalex-snapshot
Reversal of contractile defects by mediating calcium homeostasis in human mini-heart models of heart failure with preserved ejection fraction (HFpEF) leads to first-in-human gene therapy clinical trial
inferred← citeshttps://doi.org/10.1111/acel.13249openalex-snapshot
Clearance of senescent cells during cardiac ischemia–reperfusion injury improves recovery
inferred← citeshttps://doi.org/10.1111/aos.15051openalex-snapshot
Epithelial–mesenchymal transition‐related serum markers ET‐1, IL‐8 and TGF‐β2 are elevated in a Finnish wet age‐related macular degeneration cohort
inferred← citeshttps://doi.org/10.1111/jcmm.16312openalex-snapshot
GDF11 prevents the formation of thoracic aortic dissection in mice: Promotion of contractile transition of aortic SMCs
inferred← citeshttps://doi.org/10.1111/jcmm.16915openalex-snapshot
Impaired non‐canonical transforming growth factor‐β signalling prevents profibrotic phenotypes in cultured peptidylarginine deiminase 4‐deficient murine cardiac fibroblasts
inferred← citeshttps://doi.org/10.1111/jcmm.18324openalex-snapshot
Bone morphogenetic protein 10, a rising star in the field of diabetes and cardiovascular disease
inferred← citeshttps://doi.org/10.1139/cjpp-2022-0482openalex-snapshot
The myokine decorin improves the cardiac function in a rat model of isoprenaline-induced myocardial infarction
inferred← citeshttps://doi.org/10.1152/ajpcell.00120.2022openalex-snapshot
The complicated lives of GPCRs in cardiac fibroblasts
inferred← citeshttps://doi.org/10.1152/ajpcell.00209.2023openalex-snapshot
Exploring the relationship between epigenetic DNA methylation and cardiac fibrosis through Raman microspectroscopy
inferred← citeshttps://doi.org/10.1152/ajpcell.00363.2025openalex-snapshot
A human cardiomyocyte-based cellular model mimicking cardiac ischemia
inferred← citeshttps://doi.org/10.1152/ajpheart.00133.2025openalex-snapshot
Exercise training ameliorates myocardial dysfunction through fibronectin-mediated mechanotransduction in a swine model of ischemic heart disease
inferred← citeshttps://doi.org/10.1152/ajpheart.00223.2023openalex-snapshot
Divergent effects of myostatin inhibition on cardiac and skeletal muscles in a mouse model of pressure overload
inferred← citeshttps://doi.org/10.1152/ajpheart.00411.2021openalex-snapshot
Cardiac PANK1 deletion exacerbates ventricular dysfunction during pressure overload
inferred← citeshttps://doi.org/10.1152/ajpheart.00612.2021openalex-snapshot
Faster skin wound healing predicts survival after myocardial infarction
inferred← citeshttps://doi.org/10.1155/2022/1272338openalex-snapshot
Identification of Hub Genes of Keloid Fibroblasts by Coexpression Network Analysis and Degree Algorithm
inferred← citeshttps://doi.org/10.1155/2022/3455372openalex-snapshot
The Value of SII in Predicting the Mortality of Patients with Heart Failure
inferred← citeshttps://doi.org/10.1155/2022/3676444openalex-snapshot
Growth Differentiation Factor 7 Prevents Sepsis-Induced Acute Lung Injury in Mice
inferred← citeshttps://doi.org/10.1155/2023/6018375openalex-snapshot
The Carthamus tinctorius L. and Lepidium apetalum Willd. Drug Pair Inhibits EndMT through the TGF β 1/Snail Signaling Pathway in the Treatment of Myocardial Fibrosis
inferred← citeshttps://doi.org/10.1155/cdr/5632985openalex-snapshot
Exploring the Mechanism of Action of Berberine on Arrhythmia After Myocardial Infarction: A Network Pharmacology, Molecular Docking, and Cellular Experimental Study
inferred← citeshttps://doi.org/10.1159/000542728openalex-snapshot
Inactivation of Notch1-TGF-β-Smads Signaling Pathway by Atorvastatin Improves Cardiac Function and Hemodynamic Performance in Acute Myocardial Infarction Rats
inferred← citeshttps://doi.org/10.1161/circresaha.120.317146openalex-snapshot
Microfibrillar-Associated Protein 4 Regulates Stress-Induced Cardiac Remodeling
inferred← citeshttps://doi.org/10.1172/jci144336openalex-snapshot
IL-36 in chronic inflammation and fibrosis — bridging the gap?
inferred← citeshttps://doi.org/10.1177/0960327121993208openalex-snapshot
N-myc downstream regulated gene 2 ameliorates myocardial remodeling and cardiac function in heart failure rats
inferred← citeshttps://doi.org/10.1177/10760296251328361openalex-snapshot
Systemic Immune-Inflammatory Index as a Prognostic Biomarker in Patients with Heart Failure: A Comprehensive Analysis
inferred← citeshttps://doi.org/10.1186/s12864-022-08833-wopenalex-snapshot
Comprehensive analysis of RNA m6A methylation in pressure overload-induced cardiac hypertrophy
inferred← citeshttps://doi.org/10.1186/s12872-023-03464-9openalex-snapshot
Effect of Colchicine in reducing MMP-9, NOX2, and TGF- β1 after myocardial infarction
inferred← citeshttps://doi.org/10.1186/s12872-025-04517-xopenalex-snapshot
Baricitinib represses the myocardial fibrosis via blocking JAK/STAT and TGF-β1 pathways in vivo and in vitro
inferred← citeshttps://doi.org/10.1186/s12944-021-01463-3openalex-snapshot
Regulation of circulating CTRP-2/CTRP-9 and GDF-8/GDF-15 by intralipids and insulin in healthy control and polycystic ovary syndrome women following chronic exercise training
inferred← citeshttps://doi.org/10.1186/s12951-024-03047-6openalex-snapshot
An antioxidant nanozyme for targeted cardiac fibrosis therapy post myocardial infarction
inferred← citeshttps://doi.org/10.1186/s12951-025-03615-4openalex-snapshot
A fibrin gel-loaded Gouqi-derived nanovesicle (GqDNV) repairs the heart after myocardial infarction by inhibiting p38 MAPK/NF-κB p65 pathway
inferred← citeshttps://doi.org/10.1186/s12967-024-05623-8openalex-snapshot
The Abl1 tyrosine kinase is a key player in doxorubicin-induced cardiomyopathy and its p53/p73 cell death mediated signaling differs in atrial and ventricular cardiomyocytes
inferred← citeshttps://doi.org/10.1186/s12967-024-05778-4openalex-snapshot
Hyperuricemia suppresses lumican, exacerbating adverse remodeling after myocardial infarction by promoting fibroblast phenotype transition
inferred← citeshttps://doi.org/10.1186/s12967-025-06260-5openalex-snapshot
Complex regulation of cardiac fibrosis: insights from immune cells and signaling pathways
inferred← citeshttps://doi.org/10.1186/s12974-022-02647-zopenalex-snapshot
Scorpion venom peptide HsTx2 suppressed PTZ-induced seizures in mice via the circ_0001293/miR-8114/TGF-β2 axis
inferred← citeshttps://doi.org/10.1186/s13019-025-03541-4openalex-snapshot
Research progress on macrophages in cardiovascular diseases
inferred← citeshttps://doi.org/10.1186/s13075-020-02330-9openalex-snapshot
Enhanced BMP-2/BMP-4 ratio in patients with peripheral spondyloarthritis and in cytokine- and stretch-stimulated mouse chondrocytes
inferred← citeshttps://doi.org/10.1186/s13287-020-01984-1openalex-snapshot
Melphalan induces cardiotoxicity through oxidative stress in cardiomyocytes derived from human induced pluripotent stem cells
inferred← citeshttps://doi.org/10.1186/s13287-023-03560-9openalex-snapshot
Development of a thick and functional human adipose-derived stem cell tissue sheet for myocardial infarction repair in rat hearts
inferred← citeshttps://doi.org/10.1242/dmm.049856openalex-snapshot
Infiltrating circulating monocytes provide an important source of BMP4 at the early stage of spinal cord injury
inferred← citeshttps://doi.org/10.1242/jcs.258684openalex-snapshot
Titin M-line insertion sequence 7 is required for proper cardiac function in mice
inferred← citeshttps://doi.org/10.12677/acm.2023.131054openalex-snapshot
Progress in Diagnosis and Treatment of Diffuse Myocardial Fibrosis
inferred← citeshttps://doi.org/10.12794/metadc2415940openalex-snapshot
Myocardial Infarction and Its Implications for Cardiac Function and Behavior in the Zebrafish Larvae Model
inferred← citeshttps://doi.org/10.1538/expanim.24-0127openalex-snapshot
Transferrin receptor knockdown attenuates atrial fibrillation by inhibiting cardiomyocyte ferroptosis and atrial fibrosis
inferred← citeshttps://doi.org/10.1631/jzus.b2200506openalex-snapshot
Cytokine receptor-like factor 1 (CRLF1) promotes cardiac fibrosis via ERK1/2 signaling pathway
inferred← citeshttps://doi.org/10.17343/sdutfd.1793054openalex-snapshot
Electric Field-Induced Remodeling in Cardiac Tissue: Histopathological and BMP-2 Immunoreactivity Alterations Following High-Voltage Exposure in Rats
inferred← citeshttps://doi.org/10.17582/journal.pjz/20200218020203openalex-snapshot
Effect of Pilose Antler Peptide on Doxorubicin-induced H9c2 Cells Injury via TGF-β/Smad/ERK Signaling Pathway
inferred← citeshttps://doi.org/10.17650/1818-8338-2024-18-3-k709openalex-snapshot
The role of the molecular pathway of transforming growth factor β in the progression of myocardial fibrosis
inferred← citeshttps://doi.org/10.18632/aging.203127openalex-snapshot
Age-related defects in autophagy alter the secretion of paracrine factors from bone marrow mononuclear cells
inferred← citeshttps://doi.org/10.18632/aging.204901openalex-snapshot
Identification of TGF-β-related genes in cardiac hypertrophy and heart failure based on single cell RNA sequencing
inferred← citeshttps://doi.org/10.21129/nerve.2022.00248openalex-snapshot
Association of Acute Myocardial Infarction with Ossification of the Posterior Longitudinal Ligament in Korea: A Nationwide Longitudinal Cohort Study
inferred← citeshttps://doi.org/10.2147/dddt.s326024openalex-snapshot
The Synergistic Effects of Astragalus mongholicus and Salvia miltiorrhiza on Coronary Heart Disease Identified by Network Pharmacology and Experiment
inferred← citeshttps://doi.org/10.2147/jir.s324775openalex-snapshot
The Relationship Between Circulating Bone Morphogenetic Protein-4 and Inflammation Cytokines in Patients Undergoing Thoracic Surgery: A Prospective Randomized Study
inferred← citeshttps://doi.org/10.2147/jir.s356531openalex-snapshot
The Role of Bone Morphogenetic Protein 4 in Microglial Polarization in the Process of Neuropathic Pain
inferred← citeshttps://doi.org/10.21608/ejaps.2023.188530.1051openalex-snapshot
Assessment of serum levels of bone morphogenetic proteins as sensitive biomarkers for early prediction of acute myocardial infarction.
inferred← citeshttps://doi.org/10.21608/rpbs.2023.238923.1244openalex-snapshot
Signaling pathways in cardiac fibrosis; A review and future therapeutic options
inferred← citeshttps://doi.org/10.2174/0113894501421956251014101956openalex-snapshot
Role of BMP-7 in Cardiovascular Diseases: From Molecular Mechanisms to Therapeutic Horizons
inferred← citeshttps://doi.org/10.2174/0929867328666210914115411openalex-snapshot
The Roles of Signaling Pathways in Cardiac Regeneration
inferred← citeshttps://doi.org/10.22159/ijap.2025v17i5.54646openalex-snapshot
THE ROLE OF ΑIIBΒ3 RECEPTORS IN MYOCARDIAL INFARCTION: MECHANISMS AND THERAPEUTIC STRATEGIES
inferred← citeshttps://doi.org/10.2485/jhtb.31.253openalex-snapshot
Vigorous Osteoinductivity Observed in Crude Bone Morphogenetic Protein Stored for 25 Years after Extraction at Room Temperature
inferred← citeshttps://doi.org/10.25259/ijcdw_9_2024openalex-snapshot
Assessment of Inflammatory Markers among Hypertensive Women in a Nigerian Population
inferred← citeshttps://doi.org/10.29271/jcpsp.2022.07.837openalex-snapshot
The Role of Interferons in Cardiac Remodeling after Acute Myocardial Infarction
inferred← citeshttps://doi.org/10.31083/j.rcm2510368openalex-snapshot
Molecular Interplay in Cardiac Fibrosis: Exploring the Functions of RUNX2, BMP2, and Notch
inferred← citeshttps://doi.org/10.31083/rcm26421openalex-snapshot
Cytokines are the Basis of the Development and Suppression of Inflammation in Atherosclerosis
inferred← citeshttps://doi.org/10.32604/biocell.2023.026881openalex-snapshot
Role of necroptosis in spinal cord injury and its therapeutic implications
inferred← citeshttps://doi.org/10.33549/physiolres.934780openalex-snapshot
Endothelial-Mesenchymal Transition or Functional Tissue Regeneration – Two Outcomes of Heart Remodeling
inferred← citeshttps://doi.org/10.3389/fbioe.2022.1005051openalex-snapshot
Intersection of stem cell biology and engineering towards next generation in vitro models of human fibrosis
inferred← citeshttps://doi.org/10.3389/fbioe.2022.988683openalex-snapshot
Multifunctional biomaterial platforms for blocking the fibrosis process and promoting cellular restoring effects in myocardial fibrosis therapy
inferred← citeshttps://doi.org/10.3389/fcell.2025.1641584openalex-snapshot
Matrisome remodeling in the myocardium of hypertrophic cardiomyopathy; novel targets for molecular diagnostics
inferred← citeshttps://doi.org/10.3389/fcell.2026.1772507openalex-snapshot
Decoding cardiac homeostasis and injury: the evolving landscape of spatial transcriptomics
inferred← citeshttps://doi.org/10.3389/fcvm.2020.588347openalex-snapshot
Pivotal Role of TGF-β/Smad Signaling in Cardiac Fibrosis: Non-coding RNAs as Effectual Players
inferred← citeshttps://doi.org/10.3389/fcvm.2021.750604openalex-snapshot
Reawakening the Intrinsic Cardiac Regenerative Potential: Molecular Strategies to Boost Dedifferentiation and Proliferation of Endogenous Cardiomyocytes
inferred← citeshttps://doi.org/10.3389/fcvm.2022.845878openalex-snapshot
The Scientific Rationale for the Introduction of Renalase in the Concept of Cardiac Fibrosis
inferred← citeshttps://doi.org/10.3389/fcvm.2022.886553openalex-snapshot
Cardiac Fibrosis in the Pressure Overloaded Left and Right Ventricle as a Therapeutic Target
inferred← citeshttps://doi.org/10.3389/fcvm.2022.923081openalex-snapshot
Endothelial cell-derived extracellular vesicles impair the angiogenic response of coronary artery endothelial cells
inferred← citeshttps://doi.org/10.3389/fcvm.2025.1535778openalex-snapshot
Cardiac preservation using ex vivo organ perfusion: new therapies for the treatment of heart failure by harnessing the power of growth factors using BMP mimetics like THR-184
inferred← citeshttps://doi.org/10.3389/fcvm.2026.1699088openalex-snapshot
Broadening horizons: new links between cilia and heart development and disease
inferred← citeshttps://doi.org/10.3389/fcvm.2026.1729074openalex-snapshot
THR-123, a novel BMP-7 mimetic that activates Akt phosphorylation and inhibits cardiomyocyte apoptosis and inflammation, protects the heart from myocardial injury in a Rat model
inferred← citeshttps://doi.org/10.3389/fcvm.2026.1769016openalex-snapshot
Advances in targeting myocardial fibrosis: integrating mechanisms and therapeutics
inferred← citeshttps://doi.org/10.3389/fcvm.2026.1797182openalex-snapshot
Different forms of cardiomyocyte death in post-myocardial infarction ventricular remodeling: mechanisms and therapeutic strategies
inferred← citeshttps://doi.org/10.3389/fendo.2022.1080383openalex-snapshot
Mechanotransduction regulates inflammation responses of epicardial adipocytes in cardiovascular diseases
inferred← citeshttps://doi.org/10.3389/fendo.2022.895512openalex-snapshot
Intensity Dependent Effects of Interval Resistance Training on Myokines and Cardiovascular Risk Factors in Males With Obesity
inferred← citeshttps://doi.org/10.3389/fimmu.2020.01585openalex-snapshot
Alveolar Epithelial Cells Promote IGF-1 Production by Alveolar Macrophages Through TGF-β to Suppress Endogenous Inflammatory Signals
inferred← citeshttps://doi.org/10.3389/fimmu.2026.1801258openalex-snapshot
The role of the bidirectional regulatory network between immune cells and stromal cells in cardiac repair and fibrosis following myocardial infarction
inferred← citeshttps://doi.org/10.3389/fmed.2025.1678325openalex-snapshot
AAV viral vectors as therapeutic interventions for inherited or non-inherited cardiac disorders: current aspects and future prospects
inferred← citeshttps://doi.org/10.3389/fphar.2023.1157043openalex-snapshot
The role of β-catenin in cardiac diseases
inferred← citeshttps://doi.org/10.3389/fphar.2026.1758263openalex-snapshot
Regulatory mechanisms of deer antler extracellular vesicles in multilevel tissue repair: a state-of-the-art review
inferred← citeshttps://doi.org/10.3390/biom10030404openalex-snapshot
Epicardial TGFβ and BMP Signaling in Cardiac Regeneration: What Lesson Can We Learn from the Developing Heart?
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