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Metabolic Reprogramming in Breast Cancer and Its Therapeutic Implications
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IFN-γ and pro-inflammatory cytokine production by antigen-presenting cells is dictated by intracellular thiol redox status regulated by oxygen tension
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p53 as guardian of the mitochondrial genome
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Glucose deprivation inhibits multiple key gene expression events and effector functions in CD8+ T cells
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Promoter methylation of the PTEN gene is a common molecular change in breast cancer
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Mitochondrial DNA mutations and mitochondrial DNA depletion in gastric cancer
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Mitochondrial DNA mutations and mitochondrial DNA depletion in breast cancer
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IDH1mutations at residue p.R132 (IDH1R132) occur frequently in high-grade gliomas but not in other solid tumors
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Glycolysis and glucose transporter 1 as markers of response to hormonal therapy in breast cancer
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Neuromedin U alters bioenergetics and expands the cancer stem cell phenotype in HER2‐positive breast cancer
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Nuclear control of respiratory gene expression in mammalian cells
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17β‐estradiol supplementation attenuates ovariectomy‐induced increases in ATGL signaling and reduced perilipin expression in visceral adipose tissue
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Coordinate reciprocal trends in glycolytic and mitochondrial transcript accumulations during the in vitro differentiation of human myoblasts
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Intratumor lactate levels reflect HER2 addiction status in HER2‐positive breast cancer
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Somatic loss of p53 leads to stem/progenitor cell amplification in both mammary epithelial compartments, basal and luminal
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The Complete Primary Structure of Human Estrogen Receptor β (hERβ) and Its Heterodimerization with ER αin Vivoandin Vitro
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Lactate Stimulates Fibroblast Expression of Hyaluronan and CD44: The Warburg Effect Revisited
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Metabolic Reprogramming by the PI3K-Akt-mTOR Pathway in Cancer
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Acidic pH enhances the invasive behavior of human melanoma cells
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Regulation of glycolytic enzyme RNA transcriptional rates by oxygen availability in skeletal muscle cells
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Optimizing Cancer Radiotherapy with 2-Deoxy-D-Glucose
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Repression of phosphoglycerate dehydrogenase sensitizes triple-negative breast cancer to doxorubicin
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Type and frequency of IDH1 and IDH2 mutations are related to astrocytic and oligodendroglial differentiation and age: a study of 1,010 diffuse gliomas
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Inhibition of 6-phosphofructo-2-kinase (PFKFB3) suppresses glucose metabolism and the growth of HER2+ breast cancer
inferredcites →https://doi.org/10.1007/s12672-017-0298-1openalex-snapshot
Estrogen Receptor-β Modulation of the ERα-p53 Loop Regulating Gene Expression, Proliferation, and Apoptosis in Breast Cancer
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ERβ: Identification and characterization of a novel human estrogen receptor
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Human cells lacking mtDNA: repopulation with exogenous mitochondria by complementation
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Tamoxifen induced changes in MCF7 human breast cancer: In vitro and in vivo studies using nuclear magnetic resonance spectroscopy and imaging
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Mitochondrial dysfunction in human breast cancer cells and their transmitochondrial cybrids
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Luteolin as a glycolysis inhibitor offers superior efficacy and lesser toxicity of doxorubicin in breast cancer cells
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Glutathione and Thioredoxin Antioxidant Pathways Synergize to Drive Cancer Initiation and Progression
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Acetyl-CoA Synthetase 2 Promotes Acetate Utilization and Maintains Cancer Cell Growth under Metabolic Stress
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The Common Feature of Leukemia-Associated IDH1 and IDH2 Mutations Is a Neomorphic Enzyme Activity Converting α-Ketoglutarate to 2-Hydroxyglutarate
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SIRT3 Opposes Reprogramming of Cancer Cell Metabolism through HIF1α Destabilization
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Loss of FBP1 by Snail-Mediated Repression Provides Metabolic Advantages in Basal-like Breast Cancer
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Gain of Function of a p53 Hot Spot Mutation in a Mouse Model of Li-Fraumeni Syndrome
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TIGAR, a p53-Inducible Regulator of Glycolysis and Apoptosis
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Cancer Cell Metabolism: Warburg and Beyond
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Blinded by the Light: The Growing Complexity of p53
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The Tumor Suppressor p53 Regulates Polarity of Self-Renewing Divisions in Mammary Stem Cells
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A Rapid, Extensive, and Transient Transcriptional Response to Estrogen Signaling in Breast Cancer Cells
inferredcites →https://doi.org/10.1016/j.cell.2011.12.017openalex-snapshot
Mutant p53 Disrupts Mammary Tissue Architecture via the Mevalonate Pathway
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The Skp2-SCF E3 Ligase Regulates Akt Ubiquitination, Glycolysis, Herceptin Sensitivity, and Tumorigenesis
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Tumor Suppression in the Absence of p53-Mediated Cell-Cycle Arrest, Apoptosis, and Senescence
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Posttranscriptional Control of T Cell Effector Function by Aerobic Glycolysis
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Metabolic Competition in the Tumor Microenvironment Is a Driver of Cancer Progression
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Urea Cycle Dysregulation Generates Clinically Relevant Genomic and Biochemical Signatures
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Metabolic Frugality Marks Cancer Cells for Immune Targeting
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Bromodomain Protein BRD4 Is Required for Estrogen Receptor-Dependent Enhancer Activation and Gene Transcription
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Fatty Acid Oxidation-Driven Src Links Mitochondrial Energy Reprogramming and Oncogenic Properties in Triple-Negative Breast Cancer
inferredcites →https://doi.org/10.1016/j.celrep.2016.12.055openalex-snapshot
Acetate Recapturing by Nuclear Acetyl-CoA Synthetase 2 Prevents Loss of Histone Acetylation during Oxygen and Serum Limitation
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HIF-1 mediates adaptation to hypoxia by actively downregulating mitochondrial oxygen consumption
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HIF-1-mediated expression of pyruvate dehydrogenase kinase: A metabolic switch required for cellular adaptation to hypoxia
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Akt-Dependent Metabolic Reprogramming Regulates Tumor Cell Histone Acetylation
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Mitochondrial Genome Acquisition Restores Respiratory Function and Tumorigenic Potential of Cancer Cells without Mitochondrial DNA
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PDK1-Dependent Metabolic Reprogramming Dictates Metastatic Potential in Breast Cancer
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PGC-1α Promotes Breast Cancer Metastasis and Confers Bioenergetic Flexibility against Metabolic Drugs
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The biological role of estrogen receptors α and β in cancer
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Collagen Matrix Density Drives the Metabolic Shift in Breast Cancer Cells
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Pleiotropic combinatorial transcriptomes of human breast cancer cells exposed to mixtures of dietary phytoestrogens
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Hypoxia activates the capacity of tumor‐associated carbonic anhydrase IX to acidify extracellular pH
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TP53 mutation, mitochondria and cancer
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Estrogen receptor alpha negative breast cancer patients: Estrogen receptor beta as a therapeutic target
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Parenteral 17beta-estradiol decreases fasting blood glucose levels in non-obese mice with short-term ovariectomy
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Tumor suppressor p53 and estrogen receptors in nuclear–mitochondrial communication
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ROS-Mediated p53 Induction of Lpin1 Regulates Fatty Acid Oxidation in Response to Nutritional Stress
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Gain-of-Function Mutant p53 Promotes Cell Growth and Cancer Cell Metabolism via Inhibition of AMPK Activation
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Nuclear Acetyl-CoA Production by ACLY Promotes Homologous Recombination
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Metabolic Plasticity of Metastatic Breast Cancer Cells: Adaptation to Changes in the Microenvironment
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The regulation of energy metabolism and the IGF-1/mTOR pathways by the p53 protein
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Multifaceted Roles of Interleukin-6 in Adipocyte–Breast Cancer Cell Interaction
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Altered mammary gland development in the p53+/m mouse, a model of accelerated aging
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Nuclear respiratory factor-1 and bioenergetics in tamoxifen-resistant breast cancer cells
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Inhibition of glycolysis by a novel EGFR/HER2 inhibitor KU004 suppresses the growth of HER2+ cancer
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Macrophage Responses to Hypoxia
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Development of Spontaneous Mammary Tumors in BALB/c p53 Heterozygous Mice
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Transcriptional regulation of genes encoding glycolytic enzymes by hypoxia-inducible factor 1.
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Functional Differences between the Amino-Terminal Domains of Estrogen Receptors α and β
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p53 and Human Cancer: The First Ten Thousand Mutations
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Improving cancer radiotherapy with 2-deoxy-d-glucose: phase I/II clinical trials on human cerebral gliomas
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Estrogen Receptor Alpha in Human Breast Cancer: Occurrence and Significance
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Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours
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Genetic interactions between tumor suppressors Brca1 and p53 in apoptosis, cell cycle and tumorigenesis
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Depletion of the novel p53-target gene carnitine palmitoyltransferase 1C delays tumor growth in the neurofibromatosis type I tumor model
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Manganese superoxide dismutase promotes anoikis resistance and tumor metastasis
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IDH1 and IDH2 mutation analysis in chronic- and blast-phase myeloproliferative neoplasms
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Progesterone receptor by immunohistochemistry and clinical outcome in breast cancer: a validation study
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The genomic and transcriptomic architecture of 2,000 breast tumours reveals novel subgroups
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Whole-genome analysis informs breast cancer response to aromatase inhibition
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Fructose-1,6-bisphosphatase opposes renal carcinoma progression
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p53 regulates glucose metabolism through an IKK-NF-κB pathway and inhibits cell transformation
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p53 regulates biosynthesis through direct inactivation of glucose-6-phosphate dehydrogenase
inferredcites →https://doi.org/10.1038/ncb3039openalex-snapshot
PGC-1α mediates mitochondrial biogenesis and oxidative phosphorylation in cancer cells to promote metastasis
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DNAJA1 controls the fate of misfolded mutant p53 through the mevalonate pathway
inferredcites →https://doi.org/10.1038/ncomms10442openalex-snapshot
Self-renewal of CD133hi cells by IL6/Notch3 signalling regulates endocrine resistance in metastatic breast cancer
inferredcites →https://doi.org/10.1038/ncomms11479openalex-snapshot
The somatic mutation profiles of 2,433 breast cancers refine their genomic and transcriptomic landscapes
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Proline metabolism supports metastasis formation and could be inhibited to selectively target metastasizing cancer cells
inferredcites →https://doi.org/10.1038/ncomms2236openalex-snapshot
Receptor tyrosine kinase ErbB2 translocates into mitochondria and regulates cellular metabolism
inferredcites →https://doi.org/10.1038/ncomms3935openalex-snapshot
Tumour-associated mutant p53 drives the Warburg effect
inferredcites →https://doi.org/10.1038/ncomms4608openalex-snapshot
A DERL3-associated defect in the degradation of SLC2A1 mediates the Warburg effect
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Mechanisms of Disease: understanding resistance to HER2-targeted therapy in human breast cancer
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Adipocytes promote ovarian cancer metastasis and provide energy for rapid tumor growth
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Inhibition of fatty acid oxidation as a therapy for MYC-overexpressing triple-negative breast cancer
inferredcites →https://doi.org/10.1038/nm1001-1111openalex-snapshot
Ferredoxin reductase affects p53-dependent, 5-fluorouracil–induced apoptosis in colorectal cancer cells
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The current state of cancer metabolism
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The emerging roles of YAP and TAZ in cancer
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Endocrine-responsive breast cancer and strategies for combating resistance
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Cancer metabolism: a therapeutic perspective
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p53 in survival, death and metabolic health: a lifeguard with a licence to kill
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Upregulation of lactate dehydrogenase A by ErbB2 through heat shock factor 1 promotes breast cancer cell glycolysis and growth
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The gain of function of p53 cancer mutant in promoting mammary tumorigenesis
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Mitochondrial retrograde signaling induces epithelial–mesenchymal transition and generates breast cancer stem cells
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Phosphoglycerate mutase 1 promotes cancer cell migration independent of its metabolic activity
inferredcites →https://doi.org/10.1038/onc.2017.32openalex-snapshot
The branched-chain amino acid transaminase 1 sustains growth of antiestrogen-resistant and ERα-negative breast cancer
inferredcites →https://doi.org/10.1038/oncsis.2013.32openalex-snapshot
Estrogen receptor β activation impairs mitochondrial oxidative metabolism and affects malignant mesothelioma cell growth in vitro and in vivo
inferredcites →https://doi.org/10.1038/s41598-017-04626-4openalex-snapshot
Citrate Suppresses Tumor Growth in Multiple Models through Inhibition of Glycolysis, the Tricarboxylic Acid Cycle and the IGF-1R Pathway
inferredcites →https://doi.org/10.1038/s41598-017-05727-wopenalex-snapshot
The miR-29 transcriptome in endocrine-sensitive and resistant breast cancer cells
inferredcites →https://doi.org/10.1038/sj.bjc.6602302openalex-snapshot
Breast cancer risk by age at birth, time since birth and time intervals between births: exploring interaction effects
inferredcites →https://doi.org/10.1038/sj.bjc.6603384openalex-snapshot
Altered sirtuin expression is associated with node-positive breast cancer
inferredcites →https://doi.org/10.1038/sj.cdd.4401965openalex-snapshot
Regulation of AIF expression by p53
inferredcites →https://doi.org/10.1038/sj.emboj.7600461openalex-snapshot
AIF deficiency compromises oxidative phosphorylation
inferredcites →https://doi.org/10.1038/sj.onc.1202157openalex-snapshot
Delayed involution of the mammary epithelium in BALB/c-p53null mice
inferredcites →https://doi.org/10.1038/sj.onc.1203227openalex-snapshot
Epigenetic inactivation of LKB1 in primary tumors associated with the Peutz-Jeghers syndrome
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Lactate elicits vascular endothelial growth factor from macrophages: a possible alternative to hypoxia
inferredcites →https://doi.org/10.1055/s-2002-38259openalex-snapshot
Effect of Ovariectomy on Adipose Tissue of Mice in the Absence of Estrogen Receptor Alpha (ERα): a Potential Role for Estrogen Receptor Beta (ERβ)
inferredcites →https://doi.org/10.1056/nejm198612253152606openalex-snapshot
Tumors: Wounds That Do Not Heal
inferredcites →https://doi.org/10.1056/nejm199407073310102openalex-snapshot
Transient Increase in the Risk of Breast Cancer after Giving Birth
inferredcites →https://doi.org/10.1056/nejm200103153441101openalex-snapshot
Use of Chemotherapy plus a Monoclonal Antibody against HER2 for Metastatic Breast Cancer That Overexpresses HER2
inferredcites →https://doi.org/10.1056/nejmoa052122openalex-snapshot
Trastuzumab plus Adjuvant Chemotherapy for Operable HER2-Positive Breast Cancer
inferredcites →https://doi.org/10.1056/nejmoa1214091openalex-snapshot
Increased Oxidative Metabolism in the Li–Fraumeni Syndrome
inferredcites →https://doi.org/10.1073/pnas.0812029106openalex-snapshot
MUC1-induced alterations in a lipid metabolic gene network predict response of human breast cancers to tamoxifen treatment
inferredcites →https://doi.org/10.1073/pnas.1001006107openalex-snapshot
Glutaminase 2, a novel p53 target gene regulating energy metabolism and antioxidant function
inferredcites →https://doi.org/10.1073/pnas.1002459107openalex-snapshot
Phosphate-activated glutaminase (GLS2), a p53-inducible regulator of glutamine metabolism and reactive oxygen species
inferredcites →https://doi.org/10.1073/pnas.1009575107openalex-snapshot
Mechanisms of estrogen receptor antagonism toward p53 and its implications in breast cancer therapeutic response and stem cell regulation
inferredcites →https://doi.org/10.1073/pnas.1018858109openalex-snapshot
Estrogen receptor prevents p53-dependent apoptosis in breast cancer
inferredcites →https://doi.org/10.1073/pnas.1113884108openalex-snapshot
Parkin, a p53 target gene, mediates the role of p53 in glucose metabolism and the Warburg effect
inferredcites →https://doi.org/10.1073/pnas.1311249111openalex-snapshot
JMJD5 regulates PKM2 nuclear translocation and reprograms HIF-1α–mediated glucose metabolism
inferredcites →https://doi.org/10.1073/pnas.1315605111openalex-snapshot
Ribosomal protein–Mdm2–p53 pathway coordinates nutrient stress with lipid metabolism by regulating MCD and promoting fatty acid oxidation
inferredcites →https://doi.org/10.1073/pnas.1422015112openalex-snapshot
Estrogen receptor-α directly regulates the hypoxia-inducible factor 1 pathway associated with antiestrogen response in breast cancer
inferredcites →https://doi.org/10.1073/pnas.1501555112openalex-snapshot
Metabolic reprogramming in triple-negative breast cancer through Myc suppression of TXNIP
inferredcites →https://doi.org/10.1073/pnas.192561299openalex-snapshot
Involvement of estrogen receptor β in terminal differentiation of mammary gland epithelium
inferredcites →https://doi.org/10.1073/pnas.221459098openalex-snapshot
p53 is a potential mediator of pregnancy and hormone-induced resistance to mammary carcinogenesis
inferredcites →https://doi.org/10.1073/pnas.2235866100openalex-snapshot
Global analysis of ligand sensitivity of estrogen inducible and suppressible genes in MCF7/BUS breast cancer cells by DNA microarray
inferredcites →https://doi.org/10.1073/pnas.48.3.379openalex-snapshot
EXTINCTION OF EXPERIMENTAL MAMMARY CANCER, I. ESTRADIOL-17β AND PROGESTERONE
inferredcites →https://doi.org/10.1073/pnas.93.12.5925openalex-snapshot
Cloning of a novel receptor expressed in rat prostate and ovary.
inferredcites →https://doi.org/10.1074/jbc.272.36.22776openalex-snapshot
Glucose Catabolism in Cancer Cells
inferredcites →https://doi.org/10.1074/jbc.c600001200openalex-snapshot
Estrogen Receptor-α Binds p53 Tumor Suppressor Protein Directly and Represses Its Function
inferredcites →https://doi.org/10.1074/jbc.m113.529990openalex-snapshot
Estradiol Stimulates Glucose Metabolism via 6-Phosphofructo-2-kinase (PFKFB3)
inferredcites →https://doi.org/10.1074/jbc.m117.780973openalex-snapshot
Post-transcriptional regulation of ERBB2 by miR26a/b and HuR confers resistance to tamoxifen in estrogen receptor-positive breast cancer cells
inferredcites →https://doi.org/10.1074/jbc.m504652200openalex-snapshot
BRCA1 Affects Lipid Synthesis through Its Interaction with Acetyl-CoA Carboxylase
inferredcites →https://doi.org/10.1074/mcp.m400221-mcp200openalex-snapshot
Proteomic Study Reveals That Proteins Involved in Metabolic and Detoxification Pathways Are Highly Expressed in HER-2/neu-positive Breast Cancer
inferredcites →https://doi.org/10.1084/jem.189.9.1363openalex-snapshot
Inhibition of T Cell Proliferation by Macrophage Tryptophan Catabolism
inferredcites →https://doi.org/10.1093/ajcn/72.4.998openalex-snapshot
Genomic DNA methylation decreases in response to moderate folate depletion in elderly women
inferredcites →https://doi.org/10.1093/carcin/19.9.1573openalex-snapshot
Hormone-induced refractoriness to mammary carcinogenesis in Wistar- Furth rats
inferredcites →https://doi.org/10.1093/jmcb/mjs064openalex-snapshot
A new role of p53 in regulating lipid metabolism
inferredcites →https://doi.org/10.1093/jn/128.7.1204openalex-snapshot
Moderate Folate Depletion Increases Plasma Homocysteine and Decreases Lymphocyte DNA Methylation in Postmenopausal Women
inferredcites →https://doi.org/10.1093/jnci/74.4.927openalex-snapshot
Chemoprevention of N-Nitroso-N-methylurea-Induced Mammary Cancers by Pretreatment with 17β-Estradiol and Progesterone 2 3
inferredcites →https://doi.org/10.1093/jnci/82.15.1286openalex-snapshot
Human Chorionic Gonadotropin and Rat Mammary Cancer Prevention
inferredcites →https://doi.org/10.1093/jnci/djw236openalex-snapshot
Estrogen Receptor β as a Therapeutic Target in Breast Cancer Stem Cells
inferredcites →https://doi.org/10.1096/fj.201701315ropenalex-snapshot
Uncoupling effects of estrogen receptor α on LKB1/AMPK interaction upon adiponectin exposure in breast cancer
inferredcites →https://doi.org/10.1111/j.1749-6632.2010.05621.xopenalex-snapshot
Functional effects of cancer mitochondria on energy metabolism and tumorigenesis: utility of transmitochondrial cybrids
inferredcites →https://doi.org/10.1126/sciadv.1600200openalex-snapshot
Fundamentals of cancer metabolism
inferredcites →https://doi.org/10.1126/science.1126863openalex-snapshot
p53 Regulates Mitochondrial Respiration
inferredcites →https://doi.org/10.1126/science.123.3191.309openalex-snapshot
On the Origin of Cancer Cells
inferredcites →https://doi.org/10.1126/science.2649981openalex-snapshot
Chromosome 17 Deletions and p53 Gene Mutations in Colorectal Carcinomas
inferredcites →https://doi.org/10.1126/science.2814477openalex-snapshot
Human Cells Lacking mtDNA: Repopulation with Exogenous Mitochondria by Complementation
inferredcites →https://doi.org/10.1126/science.aam9305openalex-snapshot
Metabolic recycling of ammonia via glutamate dehydrogenase supports breast cancer biomass
inferredcites →https://doi.org/10.1126/scisignal.aan4667openalex-snapshot
The receptor tyrosine kinase EphA2 promotes glutamine metabolism in tumors by activating the transcriptional coactivators YAP and TAZ
inferredcites →https://doi.org/10.1128/mcb.00118-10openalex-snapshot
Genome-Wide Analysis of Estrogen Receptor α DNA Binding and Tethering Mechanisms Identifies Runx1 as a Novel Tethering Factor in Receptor-Mediated Transcriptional Activation
inferredcites →https://doi.org/10.1128/mcb.00328-17openalex-snapshot
Effect of Mutant p53 Proteins on Glycolysis and Mitochondrial Metabolism
inferredcites →https://doi.org/10.1128/mcb.00799-08openalex-snapshot
AKT Alters Genome-Wide Estrogen Receptor α Binding and Impacts Estrogen Signaling in Breast Cancer
inferredcites →https://doi.org/10.1128/mcb.06248-11openalex-snapshot
Glucose Oxidation Modulates Anoikis and Tumor Metastasis
inferredcites →https://doi.org/10.1128/mcb.17.6.3155openalex-snapshot
neu/ERBB2 Cooperates with p53-172H during Mammary Tumorigenesis in Transgenic Mice
inferredcites →https://doi.org/10.1136/jcp.2003.008599openalex-snapshot
The favourable prognostic value of oestrogen receptor β immunohistochemical expression in breast cancer
inferredcites →https://doi.org/10.1136/jcp.55.5.371openalex-snapshot
Distinct expression patterns of ER and ER in normal human mammary gland
inferredcites →https://doi.org/10.1152/ajpendo.00189.2009openalex-snapshot
Participation of ERα and ERβ in glucose homeostasis in skeletal muscle and white adipose tissue
inferredcites →https://doi.org/10.1158/0008-5472.177.65.1openalex-snapshot
Glycolytic Enzymes Can Modulate Cellular Life Span
inferredcites →https://doi.org/10.1158/0008-5472.can-06-3137openalex-snapshot
Adaptation of Energy Metabolism in Breast Cancer Brain Metastases
inferredcites →https://doi.org/10.1158/0008-5472.can-06-3724openalex-snapshot
Estrogen Receptor α Inhibits p53-Mediated Transcriptional Repression: Implications for the Regulation of Apoptosis
inferredcites →https://doi.org/10.1158/0008-5472.can-06-4447openalex-snapshot
Systemic Treatment with the Antidiabetic Drug Metformin Selectively Impairs p53-Deficient Tumor Cell Growth
inferredcites →https://doi.org/10.1158/0008-5472.can-08-3628openalex-snapshot
Transcriptional Regulation of Estrogen Receptor-α by p53 in Human Breast Cancer Cells
inferredcites →https://doi.org/10.1158/0008-5472.can-09-2608openalex-snapshot
Positive Cross-Talk between Estrogen Receptor and NF-κB in Breast Cancer
inferredcites →https://doi.org/10.1158/0008-5472.can-10-3323openalex-snapshot
Cancer-Associated Adipocytes Exhibit an Activated Phenotype and Contribute to Breast Cancer Invasion
inferredcites →https://doi.org/10.1158/0008-5472.can-11-0127openalex-snapshot
Overcoming Trastuzumab Resistance in Breast Cancer by Targeting Dysregulated Glucose Metabolism
inferredcites →https://doi.org/10.1158/0008-5472.can-11-1215openalex-snapshot
p53 Negatively Regulates Transcription of the Pyruvate Dehydrogenase Kinase Pdk2
inferredcites →https://doi.org/10.1158/0008-5472.can-12-2482openalex-snapshot
Antioxidant Enzymes Mediate Survival of Breast Cancer Cells Deprived of Extracellular Matrix
inferredcites →https://doi.org/10.1158/0008-5472.can-13-0527openalex-snapshot
Optical Metabolic Imaging Identifies Glycolytic Levels, Subtypes, and Early-Treatment Response in Breast Cancer
inferredcites →https://doi.org/10.1158/0008-5472.can-13-2779openalex-snapshot
Invasive Lobular Carcinoma Cell Lines Are Characterized by Unique Estrogen-Mediated Gene Expression Patterns and Altered Tamoxifen Response
inferredcites →https://doi.org/10.1158/0008-5472.can-14-2268openalex-snapshot
Gain of Glucose-Independent Growth upon Metastasis of Breast Cancer Cells to the Brain
inferredcites →https://doi.org/10.1158/0008-5472.can-15-2383openalex-snapshot
HER2 Signaling Drives DNA Anabolism and Proliferation through SRC-3 Phosphorylation and E2F1-Regulated Genes
inferredcites →https://doi.org/10.1158/0008-5472.can-17-2906openalex-snapshot
Transketolase Regulates the Metabolic Switch to Control Breast Cancer Cell Metastasis via the α-Ketoglutarate Signaling Pathway
inferredcites →https://doi.org/10.1158/1078-0432.ccr-06-1823openalex-snapshot
Estrogen Receptor β Expression Is Associated with Tamoxifen Response in ERα-Negative Breast Carcinoma
inferredcites →https://doi.org/10.1158/1078-0432.ccr-07-4528openalex-snapshot
Nuclear and Cytoplasmic Expression of ERβ1, ERβ2, and ERβ5 Identifies Distinct Prognostic Outcome for Breast Cancer Patients
inferredcites →https://doi.org/10.1158/1078-0432.ccr-17-0824openalex-snapshot
t-Darpp Activates IGF-1R Signaling to Regulate Glucose Metabolism in Trastuzumab-Resistant Breast Cancer Cells
inferredcites →https://doi.org/10.1158/1541-7786.mcr-17-0364openalex-snapshot
BRCA1 through Its E3 Ligase Activity Regulates the Transcription Factor Oct1 and Carbohydrate Metabolism
inferredcites →https://doi.org/10.1158/2159-8290.cd-11-0101openalex-snapshot
ERα-Dependent E2F Transcription Can Mediate Resistance to Estrogen Deprivation in Human Breast Cancer
inferredcites →https://doi.org/10.1159/000346844openalex-snapshot
Trastuzumab-Induced Hepatotoxicity: A Case Report
inferredcites →https://doi.org/10.1159/000489759openalex-snapshot
FGFR4 Links Glucose Metabolism and Chemotherapy Resistance in Breast Cancer
inferredcites →https://doi.org/10.1161/circresaha.109.205310openalex-snapshot
p53 Improves Aerobic Exercise Capacity and Augments Skeletal Muscle Mitochondrial DNA Content
showing the first 200 of 268 · page through all 268
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Hyperpolarised 13C-MRI identifies the emergence of a glycolytic cell population within intermediate-risk human prostate cancer
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Machine learning identifies SRD5A3 as a propionate-related prognostic biomarker in triple-negative breast cancer
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Systematic screening of metabolic pathways to identify two breast cancer subtypes with divergent immune characteristics
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Intratumoral and peritumoral radiomics signature based on DCE-MRI can distinguish between luminal and non-luminal breast cancer molecular subtypes
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LPLUNC1 reduces glycolysis in nasopharyngeal carcinoma cells through the PHB1 ‐p53/ c‐Myc axis
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Saikosaponin A alleviates glycolysis of breast cancer cells through repression of Akt/STAT3 pathway
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ERα down‐regulates carbohydrate responsive element binding protein and decreases aerobic glycolysis in liver cancer cells
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inferred← citeshttps://doi.org/10.11606/t.5.2023.tde-24112023-165858openalex-snapshot
Efeitos da estimulação elétrica nervosa transcutânea na angiogênese e proliferação celular do carcinoma 4T1 de glândulas mamárias em camundongas
inferred← citeshttps://doi.org/10.1166/jbt.2023.3224openalex-snapshot
Bone Marrow Mesenchymal Stem Cell (BMSC)-Derived Exosomes Regulates Growth of Breast Cancer Cells Mediated by Hedgehog Signaling Pathway
inferred← citeshttps://doi.org/10.1177/11782234241240173openalex-snapshot
Estrogen Receptor Is Required for Metformin-Induced Apoptosis in Breast Cancer Cells Under Hyperglycemic Conditions
inferred← citeshttps://doi.org/10.1186/s12885-021-08343-0openalex-snapshot
Metabolic comorbidities and the association with risks of recurrent metastatic disease in breast cancer survivors
inferred← citeshttps://doi.org/10.1186/s12885-025-13908-4openalex-snapshot
L-arginine dependence of breast cancer – molecular subtypes matter.
inferred← citeshttps://doi.org/10.1186/s12920-022-01256-2openalex-snapshot
Identification and validation of an epigenetically regulated long noncoding RNA model for breast cancer metabolism and prognosis
inferred← citeshttps://doi.org/10.1186/s12929-024-01013-wopenalex-snapshot
Metabolism-regulating non-coding RNAs in breast cancer: roles, mechanisms and clinical applications
inferred← citeshttps://doi.org/10.1186/s12935-025-03680-7openalex-snapshot
Triple-positive breast cancer: navigating heterogeneity and advancing multimodal therapies for improving patient outcomes
inferred← citeshttps://doi.org/10.1186/s12935-025-04006-3openalex-snapshot
Adaptability of lung and liver metastatic breast cancer cells to glucose
inferred← citeshttps://doi.org/10.1186/s12935-025-04135-9openalex-snapshot
Ophiobolin A impacts mitochondrial redox biology in an epithelial-mesenchymal transition (EMT)-specific manner
inferred← citeshttps://doi.org/10.1186/s12943-021-01412-9openalex-snapshot
CircMYH9 drives colorectal cancer growth by regulating serine metabolism and redox homeostasis in a p53-dependent manner
inferred← citeshttps://doi.org/10.1186/s12964-024-01917-yopenalex-snapshot
Breast cancer cells utilize T3 to trigger proliferation through cellular Ca2+ modulation
inferred← citeshttps://doi.org/10.1186/s12967-026-07905-9openalex-snapshot
A novel combination for in vivo breast cancer treatment: TAK1 inhibition combined with metformin and Lentinula edodes compounds synergistically reinvigorates CD8+ T Cells
inferred← citeshttps://doi.org/10.1186/s13045-021-01194-zopenalex-snapshot
A novel lncRNA ROPM-mediated lipid metabolism governs breast cancer stem cell properties
inferred← citeshttps://doi.org/10.1186/s13046-020-01767-9openalex-snapshot
The miR-1185-2-3p—GOLPH3L pathway promotes glucose metabolism in breast cancer by stabilizing p53-induced SERPINE1
inferred← citeshttps://doi.org/10.1186/s13046-022-02476-1openalex-snapshot
Heterogeneity of triple negative breast cancer: Current advances in subtyping and treatment implications
inferred← citeshttps://doi.org/10.1186/s13046-024-03101-zopenalex-snapshot
Shikonin blocks CAF-induced TNBC metastasis by suppressing mitochondrial biogenesis through GSK-3β/NEDD4-1 mediated phosphorylation-dependent degradation of PGC-1α
inferred← citeshttps://doi.org/10.1186/s13058-024-01813-wopenalex-snapshot
Metabolomics assisted by transcriptomics analysis to reveal metabolic characteristics and potential biomarkers associated with treatment response of neoadjuvant therapy with TCbHP regimen in HER2 + breast cancer
inferred← citeshttps://doi.org/10.1186/s13058-025-02137-zopenalex-snapshot
Pathological response and metabolites’ prognostic role in HER2-positive breast cancer treated with neoadjuvant pyrotinib, trastuzumab, nab-paclitaxel, and carboplatin: a single-arm phase II trial
inferred← citeshttps://doi.org/10.1186/s40164-025-00667-yopenalex-snapshot
Immunotherapy in breast cancer: current landscape and emerging trends
inferred← citeshttps://doi.org/10.1186/s40959-025-00433-3openalex-snapshot
Mortality differences in coronary patients with breast cancer treated with trimetazidine dihydrochloride: potential therapeutic implications
inferred← citeshttps://doi.org/10.1371/journal.pone.0278711openalex-snapshot
Ribitol alters multiple metabolic pathways of central carbon metabolism with enhanced glycolysis: A metabolomics and transcriptomics profiling of breast cancer
inferred← citeshttps://doi.org/10.1371/journal.pone.0340586openalex-snapshot
Establishment of an amino acid metabolism related signature for prognostic and therapeutic sensitivity prediction in breast cancer by machine learning
inferred← citeshttps://doi.org/10.15407/exp-oncology.2023.02.242openalex-snapshot
EFFECT OF LACTATE DEHYDROGENASE INHIBITION BY OXAMATE ON LEWIS LUNG CARCINOMA CELLS WITH DIFFERENT METASTATIC POTENTIAL
inferred← citeshttps://doi.org/10.15407/exp-oncology.2024.03.237openalex-snapshot
EFFECT OF ADHESIVE LLC CELL PRETREATMENT BY OXAMATE ON THE SURVIVAL INDEXES AFTER TRANSITION TO DE-ADHESIVE GROWTH
inferred← citeshttps://doi.org/10.15407/exp-oncology.2025.01.003openalex-snapshot
REPROGRAMMING OF GLUCOSE METABOLISM IN HUMAN BREAST CANCER CELLS AFTER CO-CULTIVATION WITH BIFIDOBACTERIUM ANIMALIS
inferred← citeshttps://doi.org/10.18632/aging.101960openalex-snapshot
Metformin induces a fasting- and antifolate-mimicking modification of systemic host metabolism in breast cancer patients
inferred← citeshttps://doi.org/10.18632/aging.203439openalex-snapshot
Mediation of circ_RPPH1 on miR-146b-3p/E2F2 pathway to hinder the growth and metastasis of breast carcinoma cells
inferred← citeshttps://doi.org/10.20517/cdr.2022.10openalex-snapshot
Molecular vulnerabilities and therapeutic resistance in hormone receptor positive and HER2 dependent breast cancer tumours
inferred← citeshttps://doi.org/10.20892/j.issn.2095-3941.2019.0210openalex-snapshot
Metabolic reprogramming in triple-negative breast cancer
inferred← citeshttps://doi.org/10.21203/rs.3.rs-11048428/v1openalex-snapshot
Early molecular changes and a pivotal transition define triple-negative breast cancer progression in a murine model: a stage-resolved transcriptomic study
inferred← citeshttps://doi.org/10.21203/rs.3.rs-1500905/v1openalex-snapshot
Circ_0000732 promotes breast cancer cell proliferation, metastasis and stemness by targeting COL11A1 through the regulation of miR-1253
inferred← citeshttps://doi.org/10.21203/rs.3.rs-2088137/v1openalex-snapshot
Breast Cancer With Increased Drug Resistance, Invasion Ability, and Cancer Stem Cell Property Through Metabolism Reprogramming
inferred← citeshttps://doi.org/10.21203/rs.3.rs-2650279/v1openalex-snapshot
Tumor necrosis factor receptor-associated protein 1 promotes aerobic glycolysis and cisplatin resistance by regulating Wnt/β-catenin signal pathway in lung cancer
inferred← citeshttps://doi.org/10.21203/rs.3.rs-3075627/v1openalex-snapshot
Dissecting fibroblast growth factor-induced endocrine therapy resistance in breast cancer cells
inferred← citeshttps://doi.org/10.2147/bctt.s284453openalex-snapshot
Genetic Influences in Breast Cancer Drug Resistance
inferred← citeshttps://doi.org/10.2147/ott.s226851openalex-snapshot
PDHA1 Gene Knockout In Human Esophageal Squamous Cancer Cells Resulted In Greater Warburg Effect And Aggressive Features In Vitro And In Vivo
inferred← citeshttps://doi.org/10.2174/0115680096303785240822155217openalex-snapshot
A Phase II Clinical Study on Apatinib Plus Vinorelbine in Refractory HER2-Negative Breast Cancer and its Metabolic Implications of Drug Resistance
inferred← citeshttps://doi.org/10.2174/1871520620666200807221047openalex-snapshot
Thymoquinone Inhibits Proliferation and Migration of MDA-MB-231 Triple Negative Breast Cancer Cells by Suppressing Autophagy, Beclin-1 and LC3
inferred← citeshttps://doi.org/10.31083/j.jmcm.2019.02.7181openalex-snapshot
Naphthol-derived Betti bases as potential SLC6A14 blockers
inferred← citeshttps://doi.org/10.3389/fendo.2020.00196openalex-snapshot
Editorial: Resistance to Endocrine Therapies in Cancer
inferred← citeshttps://doi.org/10.3389/fendo.2023.1261283openalex-snapshot
The role of the tumor microenvironment in endocrine therapy resistance in hormone receptor-positive breast cancer
inferred← citeshttps://doi.org/10.3389/fmed.2023.1140514openalex-snapshot
Application of DCE-MRI radiomics signature analysis in differentiating molecular subtypes of luminal and non-luminal breast cancer
inferred← citeshttps://doi.org/10.3389/fmicb.2022.818793openalex-snapshot
New Developments and Opportunities of Microbiota in Treating Breast Cancers
inferred← citeshttps://doi.org/10.3389/fmicb.2024.1393422openalex-snapshot
Bibliometric analysis of global research trends between gut microbiota and breast cancer: from 2013 to 2023
inferred← citeshttps://doi.org/10.3389/fmolb.2021.791927openalex-snapshot
Metabolic Reprogramming Underlying Brain Metastasis of Breast Cancer
inferred← citeshttps://doi.org/10.3389/fonc.2020.598626openalex-snapshot
Lactate Metabolism and Immune Modulation in Breast Cancer: A Focused Review on Triple Negative Breast Tumors
inferred← citeshttps://doi.org/10.3389/fonc.2020.598684openalex-snapshot
Lysine in Combination With Estradiol Promote Dissemination of Estrogen Receptor Positive Breast Cancer via Upregulation of U2AF1 and RPN2 Proteins
inferred← citeshttps://doi.org/10.3389/fonc.2020.602416openalex-snapshot
The Metabolic Mechanisms of Breast Cancer Metastasis
inferred← citeshttps://doi.org/10.3389/fonc.2021.612009openalex-snapshot
Expression of Autophagy and Mitophagy Markers in Breast Cancer Tissues
inferred← citeshttps://doi.org/10.3389/fonc.2021.612952openalex-snapshot
A 13-Gene Metabolic Prognostic Signature Is Associated With Clinical and Immune Features in Stomach Adenocarcinoma
inferred← citeshttps://doi.org/10.3389/fonc.2021.629640openalex-snapshot
The Deubiquitinating Enzyme UCHL1 Induces Resistance to Doxorubicin in HER2+ Breast Cancer by Promoting Free Fatty Acid Synthesis
inferred← citeshttps://doi.org/10.3389/fonc.2021.698951openalex-snapshot
Energy Metabolic Plasticity of Colorectal Cancer Cells as a Determinant of Tumor Growth and Metastasis
inferred← citeshttps://doi.org/10.3389/fonc.2022.1060495openalex-snapshot
Correlation between the Warburg effect and progression of triple-negative breast cancer
inferred← citeshttps://doi.org/10.3389/fonc.2022.786931openalex-snapshot
Metabolic Adaptations in an Endocrine-Related Breast Cancer Mouse Model Unveil Potential Markers of Tumor Response to Hormonal Therapy
inferred← citeshttps://doi.org/10.3389/fonc.2022.887257openalex-snapshot
Elevated BCAA Suppresses the Development and Metastasis of Breast Cancer
inferred← citeshttps://doi.org/10.3389/fonc.2022.939606openalex-snapshot
Metabolic reprogramming enables the auxiliary diagnosis of breast cancer by automated breast volume scanner
inferred← citeshttps://doi.org/10.3389/fonc.2022.960312openalex-snapshot
Targeting O-GlcNAcylation to overcome resistance to anti-cancer therapies
inferred← citeshttps://doi.org/10.3389/fonc.2022.971288openalex-snapshot
Epigenetics and environment in breast cancer: New paradigms for anti-cancer therapies
inferred← citeshttps://doi.org/10.3389/fonc.2025.1553496openalex-snapshot
Editorial: Revealing the role of mitochondrial gene defects in tumor progression and developing mitochondrial-targeted drugs
inferred← citeshttps://doi.org/10.3389/fonc.2025.1583752openalex-snapshot
Rewired glycolysis by DTL accelerates oncometabolite L-lactate generation to promote breast cancer progression
inferred← citeshttps://doi.org/10.3389/fphar.2022.942996openalex-snapshot
Dandelion extract inhibits triple-negative breast cancer cell proliferation by interfering with glycerophospholipids and unsaturated fatty acids metabolism
inferred← citeshttps://doi.org/10.3389/fphar.2024.1421905openalex-snapshot
Mitochondrial inhibitors: a new horizon in breast cancer therapy
inferred← citeshttps://doi.org/10.3389/fphar.2024.1425171openalex-snapshot
Neratinib safety evaluation: real-world adverse event analysis from the FAERS database
inferred← citeshttps://doi.org/10.3389/fphar.2025.1531667openalex-snapshot
Dual inhibitor of MDM2 and NFAT1 for experimental therapy of breast cancer: in vitro and in vivo anticancer activities and newly discovered effects on cancer metabolic pathways
inferred← citeshttps://doi.org/10.3389/fphar.2026.1887125openalex-snapshot
UNC93B1 drives IL-6/STAT3-dependent tumor–macrophage crosstalk and epithelial–mesenchymal transition in breast cancer
inferred← citeshttps://doi.org/10.3389/pore.2021.1609789openalex-snapshot
Comprehensive Analysis of Metabolic Genes in Breast Cancer Based on Multi-Omics Data
inferred← citeshttps://doi.org/10.3390/antiox15040459openalex-snapshot
Therapy as a State-Generator: Dynamic Phenotypic Landscapes and Adaptive Stress Circuits in Chemotherapy Resistance of Breast Cancer
inferred← citeshttps://doi.org/10.3390/app112210787openalex-snapshot
Molecular Profile Study of Extracellular Vesicles for the Identification of Useful Small “Hit” in Cancer Diagnosis
inferred← citeshttps://doi.org/10.3390/biom11020141openalex-snapshot
The Mitochondrial Citrate Carrier SLC25A1/CIC and the Fundamental Role of Citrate in Cancer, Inflammation and Beyond
inferred← citeshttps://doi.org/10.3390/biom11050743openalex-snapshot
Characterising the Response of Human Breast Cancer Cells to Polyamine Modulation
inferred← citeshttps://doi.org/10.3390/biom15060870openalex-snapshot
Relationship Between Thyroid Dysfunction and Ovarian Cancer
inferred← citeshttps://doi.org/10.3390/biomedicines11061709openalex-snapshot
Insights on the Role of Polyphenols in Combating Cancer Drug Resistance
inferred← citeshttps://doi.org/10.3390/biomedicines12050979openalex-snapshot
RETRACTED: High PGC-1α Expression as a Poor Prognostic Indicator in Intracranial Glioma
inferred← citeshttps://doi.org/10.3390/cancers13020188openalex-snapshot
Resveratrol’s Anti-Cancer Effects through the Modulation of Tumor Glucose Metabolism
inferred← citeshttps://doi.org/10.3390/cancers13122922openalex-snapshot
Targeting mTOR and Glycolysis in HER2-Positive Breast Cancer
inferred← citeshttps://doi.org/10.3390/cancers13194808openalex-snapshot
Nuclear Receptor-Mediated Metabolic Reprogramming and the Impact on HR+ Breast Cancer
inferred← citeshttps://doi.org/10.3390/cancers14153661openalex-snapshot
The Differential Metabolic Signature of Breast Cancer Cellular Response to Olaparib Treatment
inferred← citeshttps://doi.org/10.3390/cancers14246267openalex-snapshot
Lipid Metabolism Heterogeneity and Crosstalk with Mitochondria Functions Drive Breast Cancer Progression and Drug Resistance
inferred← citeshttps://doi.org/10.3390/cancers16173028openalex-snapshot
ASCT2 Regulates Fatty Acid Metabolism to Trigger Glutamine Addiction in Basal-like Breast Cancer
inferred← citeshttps://doi.org/10.3390/cancers16203468openalex-snapshot
The Influence of Microbiota on Breast Cancer: A Review
inferred← citeshttps://doi.org/10.3390/cancers17172756openalex-snapshot
Urinary Metabolome Study for Monitoring Prostate Cancer Recurrence Following Radical Prostatectomy
inferred← citeshttps://doi.org/10.3390/cells12172123openalex-snapshot
The Influence of Adipocyte Secretome on Selected Metabolic Fingerprints of Breast Cancer Cell Lines Representing the Four Major Breast Cancer Subtypes
inferred← citeshttps://doi.org/10.3390/cells15181630openalex-snapshot
In Vitro Glutamine Adaptability in Murine Liver- but Not Lung-Metastasizing Triple-Negative Breast Cancer Cells
inferred← citeshttps://doi.org/10.3390/cells8040293openalex-snapshot
Microbiome—Microbial Metabolome—Cancer Cell Interactions in Breast Cancer—Familiar, but Unexplored
inferred← citeshttps://doi.org/10.3390/cells8091055openalex-snapshot
The Redox Role of G6PD in Cell Growth, Cell Death, and Cancer
inferred← citeshttps://doi.org/10.3390/cells9092081openalex-snapshot
Metabolic Constrains Rule Metastasis Progression
inferred← citeshttps://doi.org/10.3390/curroncol33020129openalex-snapshot
Metabolic Vulnerabilities as a Therapeutic Target in Breast Cancer
inferred← citeshttps://doi.org/10.3390/diagnostics11081460openalex-snapshot
Prognostic Significance of O-GlcNAc and PKM2 in Hormone Receptor-Positive and HER2-Nonenriched Breast Cancer
inferred← citeshttps://doi.org/10.3390/ijms20184611openalex-snapshot
Co-Operation between Aneuploidy and Metabolic Changes in Driving Tumorigenesis
inferred← citeshttps://doi.org/10.3390/ijms21010116openalex-snapshot
Estradiol Metabolism: Crossroads in Pulmonary Arterial Hypertension
inferred← citeshttps://doi.org/10.3390/ijms22041656openalex-snapshot
Estrogen Receptor Beta: The Promising Biomarker and Potential Target in Metastases
inferred← citeshttps://doi.org/10.3390/ijms23063181openalex-snapshot
Drug Metabolism for the Identification of Clinical Biomarkers in Breast Cancer
inferred← citeshttps://doi.org/10.3390/ijms232012063openalex-snapshot
Evolutionary Origins of Metabolic Reprogramming in Cancer
inferred← citeshttps://doi.org/10.3390/ijms232112875openalex-snapshot
Breast Cancer with Increased Drug Resistance, Invasion Ability, and Cancer Stem Cell Properties through Metabolism Reprogramming
inferred← citeshttps://doi.org/10.3390/ijms232214152openalex-snapshot
Phytochemical Targeting of Mitochondria for Breast Cancer Chemoprevention, Therapy, and Sensitization
inferred← citeshttps://doi.org/10.3390/ijms241713209openalex-snapshot
Integrating Single-Cell RNA-Seq and Bulk RNA-Seq Data to Explore the Key Role of Fatty Acid Metabolism in Breast Cancer
inferred← citeshttps://doi.org/10.3390/ijms252313029openalex-snapshot
Metabolomics-Based Machine Learning Models Accurately Predict Breast Cancer Estrogen Receptor Status
inferred← citeshttps://doi.org/10.3390/ijms26041559openalex-snapshot
Adaptive Proteomic Changes in Protein Metabolism and Mitochondrial Alterations Associated with Resistance to Trastuzumab and Pertuzumab Therapy in HER2-Positive Breast Cancer
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